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1

G, Rosselin, ed. International Symposium on Vasoactive Intestinal Peptide Pituitary Adenylate Cyclase Activating Polypeptide & Related Regulatory Peptides: From molecular biology to clinical applications : Euroconference, Strasbourg (Bischenberg), 19-23, September 1993. World Scientific, 1994.

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2

Fischer, Kirk D. Intestinal growth in models of glucagon-like peptide-2 overexpression. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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3

International Symposium on VIP and Related Peptides (5th 1991 Shizuoka, Japan). Vasoactive intestinal peptide and related peptides: Proceedings of the Fifth International Symposium on VIP and Related Peptides, Shizuoka, Japan, November 12-15, 1991. Edited by Yanaihara Noboru. Biomedical Research Foundation, 1992.

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4

1930-, Bevan John A., ed. Vascular neuroeffector mechanisms: Receptors, ion-channels, second messengers, and endogenous mediators : proceedings of the Sixth International Symposium on Vascular Neuroeffector Mechanisms, Melbourne, Australia, August 30-September 2, 1987. Published for the ICSU Press by IRL Press, 1988.

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5

Nicklin, Paul Leslie. Amino acid, peptide and drug transport across monolayers of human intestinal (CAC0-2) cells in vitro. Aston University. Department of Pharmaceutical Sciences., 1993.

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6

International Symposium on "Vasoactive Intestinal Peptide (VIP) and Related Peptides" (2nd 1985 Cap d'Agde, France). Abstracts of the second International Symposium on "Vasoactive Intestinal Peptide (VIP) and Related Peptides": Cap d'Agde, Hérault, France, june 18-22, 1985. Elsevier Science Publishers, 1985.

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7

Hubert, Vaudry, and Laburthe Marc, eds. VIP, PACAP, and related peptides: From gene to therapy. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

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8

Indigestion: Living better with upper intestinal problems from heartburn to ulcers and gallstones. Consumer Reports Books, 1992.

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9

Indigestion: Living better with upper intestinal problems from heartburn to ulcers and gallstones. Oxford University Press, 1992.

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10

Davis, Micheal D. The isolation of novel insulin-releasing peptides from the small intestine of the obese hyperglycaemic (ob/ob)mouse. The Author], 1994.

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11

Sāmī, Saʻīd, Mutt Viktor, and New York Academy of Sciences., eds. Vasoactive intestinal peptide and related peptides. New York Academy of Sciences, 1988.

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12

E, Klusha V., and Organiskās sintēzes instutūts (Latvijas psr ZinAtn̜u akadm̄ija), eds. Central and peripheral peptidergic regulation. The Institute, 1991.

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13

D, Bataille, Saʻīd Sāmī, and International Symposium on "Vasoactive Intestinal Peptide (VIP) and Related Peptides" (1985 : Cap d'Agde, France), eds. VIP and related substances: Second international symposium, held in Cap D'Agde, France, June 18-22, 1985. Ankho International, 1986.

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14

Khatri, Ismat A. *. Biochemical studies of the peptide backbone of intestinal mucin. 1988.

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15

Akira, Arimura, Saʻīd Sāmī, and International Symposium on VIP, PACAP, and Related Peptides (2nd : 1995 : New Orleans, La.), eds. VIP, PACAP, and related peptides: Second international symposium. New York Academy of Sciences, 1996.

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16

Rosselin, Gabriel. International Symposium on Vasoactive Intestinal Peptide, Pituitary Adenylate Cyclase Activating Polypeptide & Related Regulatory Peptides: From Mol. World Scientific Pub Co Inc, 1994.

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17

PACAP, GLUCAGON, and Related Peptides (4th : 1999 : Elsinore, Denmark) International Symposium on VIP. Vip, Pacap, Glucagon, and Related Peptides: Fourth International Symposium (Annals of the New York Academy of Sciences). 3rd ed. New York Academy of Sciences, 2001.

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18

PACAP, GLUCAGON and Related Peptides (4th :. 1999 :. Elsinore Denmark) International Symposium on VIP. Vip, Pacap, Glucagon, and Related Peptides: Fourth International Symposium (Annals of the New York Academy of Sciences, V. 921). New York Academy of Sciences, 2000.

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19

1939-, Forssmann W. G., Said Sami, and International Symposium on VIP, PACAP, and Related Peptides: (3rd : 1997 : Freiburg, Germany), eds. VIP, PACAP, and related peptides: Third international symposium. New York Academy of Sciences, 1998.

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20

(Editor), J. A. Bevan, H. Majewski (Editor), R. A. Maxwell (Editor), and D. F. Story (Editor), eds. Vascular Neuroeffector Mechanisms: Receptors, Ion-Channels, Second Messengers and Endogenous Mediators (Icsu Symposium Series, Vol 10). Oxford University Press, USA, 1988.

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21

Shin, Eric D. Intestinal mucosal adaptation in re-fed mice is dependent on the physiological actions of glucagon-like peptide-2 (GLP-2). 2004.

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22

Carton, James. Gastrointestinal pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0007.

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This chapter discusses gastrointestinal pathology, including gastrointestinal malformations, oesophagitis, oesophageal polyps and nodules, oesophageal carcinoma, gastritis, gastric polyps, gastric carcinoma, gastrointestinal stromal tumours, peptic duodenitis, coeliac disease, small bowel infarction, intestinal infections, intestinal obstruction, acute appendicitis, Crohn’s disease, ulcerative colitis, colorectal polyps, colorectal carcinoma, diverticular disease, and anal pathology.
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23

The catabolism of glucagon-like peptidea 2: A novel intestinal growth factor. National Library of Canada, 1998.

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24

Nemeroff, Charles B., and Artur J. Prang. Neurotensin (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1993.

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25

A, Johnston Craig, and Barnes Charles D. 1935-, eds. Brain-gut peptides and reproductive function. CRC Press, 1991.

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26

Burton, Derek, and Margaret Burton. Food procurement and processing. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0004.

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Fish display a wide range of adaptations of the mouth and pharynx for specific feeding patterns including planktivory, fin-biting, picking and scraping. Appetite control is complex, involving stimulatory and inhibitory hormones. The gut has a linear plan similar to other vertebrates but with considerable variation between taxa, and a stomach may be absent. Many bony fish possess pyloric caeca, containing digestive enzymes, and may increase surface area for digestion. In chondrichthyes (sharks, etc.), a ‘spiral valve’ increases surface area of the intestine. Smooth muscle contractions in the gu
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27

Janowitz, Henry D. Indigestion: Living Better with Upper Intestinal Problems from Heartburn to Ulcers and Gallstones. Oxford University Press, USA, 1994.

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28

Sedlack, Robert E., Conor G. Loftus, Amy S. Oxentenko, and Thomas R. Viggiano. Gastroenterology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0210.

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Part 1 reviews the major portions of the gastrointestinal system (esophagus, stomach, small intestine, colon, and pancreas), their function (motility, acid production, enzymatic function, and absorption), and various disorders associated with them (dysmotility, ulceration, malabsorption, inflammation, and dysplasia). Symptoms, diagnostic testing, and treatment of common gastrointestinal conditions, such as gastroesophageal reflux disease, peptic ulcer disease, diarrhea, constipation, inflammatory bowel disease, and pancreatitis, are reviewed.
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29

Ulcer, Cancer of Esophagus and Intestinal Disease (CRC series in gastrointestinal disease). CRC Press Inc, 2000.

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30

(Editor), Daniel Hollander, and Andrzej S. Tarnawski (Editor), eds. Gastric Cytoprotection: A Clinician's Guide. Springer, 1989.

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31

Gastric cytoprotection: A clinician's guide. Plenum Medical Book Co., 1989.

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32

Sohn, Woon-Mok, and Jong-Yil Chai. Anisakiosis (Anisakidosis). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0070.

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The term ‘anisakiosis (anisakidosis)’ or ‘anisakiasis’ collectively defines human infections caused by larval anisakids belonging to the nematode family Anisakidae or Raphidascarididae. Anisakis simplex, Anisakis physeteris, and Pseudoteranova decipiens are the three major species causing human anisakiosis. Various kinds of marine fish and cephalopods serve as the second intermediate hosts and the infection source. Ingestion of viable anisakid larvae in the fillet or viscera of these hosts is the primary cause of infection. The parasite does not develop further in humans as they are an acciden
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33

Malyszko, Jolanta, and Iain C. Macdougall. Iron metabolism in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0125.

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While whole-body (‘absolute’) iron deficiency is common and probably increased in frequency in chronic kidney disease (CKD), functional iron deficiency is a particular problem in CKD. Absolute iron deficiency is likely to be present in advanced CKD when the ferritin falls below 100 ng/mL and the TSAT falls below 20%. Functional iron deficiency is characterized by the presence of adequate iron stores (as defined by conventional criteria), but with an inability to mobilize this iron rapidly enough to adequately support erythropoiesis with the administration of erythropoietin. Among such patients
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34

(Editor), Henry Parkman, and Robert S. Fisher (Editor), eds. The Clinician's Guide to Acid/Peptic Disorders and Motility Disorders of the Gastrointestinal Tract (The Clinician's Guide to GI Series). Slack Incorporated, 2006.

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35

Keshav, Satish, and Alexandra Kent. Immunology and genetics in gastrointestinal and hepatic medicine. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0196.

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The gut has a pivotal role in immune homeostasis. It is constantly exposed to a wide array of antigens in food, and resident and consumed microorganisms. It is estimated that the number of bacterial cells in the gastrointestinal tract is tenfold greater than the number of cells in the human body. The gut needs to recognize harmful bacteria, and consequently contains the largest number of immune cells in the body. However, it must remain tolerant to commensal bacteria. Bacteria express antigens that stimulate an immunological response via the gut-associated lymphoid tissue (GALT). The GALT incl
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