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1

van der Meij, E. H., and J. G. A. M. de Visscher. "A periapical lesion, not always an inflammation. Periapical radiopacities." Nederlands Tijdschrift Voor Tandheelkunde 129, no. 10 (2022): 454–64. http://dx.doi.org/10.5177/ntvt.2022.10.22054.

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2

van der Meij, E. H., and J. G. A. M. de Visscher. "A periapical lesion, not always an inflammation. Periapical radiolucencies." Nederlands Tijdschrift Voor Tandheelkunde 129, no. 6 (2022): 295–305. http://dx.doi.org/10.5177/ntvt.2022.06.22013.

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3

Guo, Hongmei, Wei Lu, Qianqian Han, Shubo Li, and Pishan Yang. "Rare Periodontal Ligament Drainage for Periapical Inflammation of an Adjacent Tooth: A Case Report and Review of the Literature." Case Reports in Dentistry 2014 (2014): 1–6. http://dx.doi.org/10.1155/2014/879562.

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Aim. To report a case with an unusual drainage route of periapical inflammation exiting through the gingival sulcus of an adjacent vital tooth and review probable factors determining the diversity of the discharge routes of periapical inflammation.Summary. An 18-year-old male patient presented with periodontal abscess of tooth 46, which was found to be caused by a periapical cyst with an acute abscess of tooth 45. During endodontic surgery, a rarely reported drainage route for periapical inflammation via the gingival sulcus of an adjacent vital tooth was observed for the first time. Complete p
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4

Ma, Junqing, Wei Chen, Lijie Zhang, et al. "RNA Interference-Mediated Silencing ofAtp6iPrevents Both Periapical Bone Erosion and Inflammation in the Mouse Model of Endodontic Disease." Infection and Immunity 81, no. 4 (2013): 1021–30. http://dx.doi.org/10.1128/iai.00756-12.

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ABSTRACTDental caries is one of the most prevalent infectious diseases in the United States, affecting approximately 80% of children and the majority of adults. Dental caries may lead to endodontic disease, where the bacterial infection progresses to the root canal system of the tooth, leading to periapical inflammation, bone erosion, severe pain, and tooth loss. Periapical inflammation may also exacerbate inflammation in other parts of the body. Although conventional clinical therapies for this disease are successful in approximately 80% of cases, there is still an urgent need for increased e
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5

Hestiana, Celine, Ria Noerianingsih Firman, and Deddy Firman. "Tulang alveolar pasca perawatan jembatan ditinjau dari radiografi periapikalAlveolar bone after bridge treatment in terms of periapical radiography." Padjadjaran Journal of Dental Researchers and Students 4, no. 1 (2020): 9. http://dx.doi.org/10.24198/pjdrs.v4i1.24043.

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Pendahuluan: Radiografi periapikal dapat digunakan untuk mendeteksi tulang alveolar pada perawatan gigi tiruan jembatan. Pembuatan gigi tiruan jembatan harus memenuhi syarat-syarat biologis. Apabila adaptasi marginal buruk, penempatan margin intracrevicular terlalu dalam, permukaan restorasi yang kasar dan restorasi yang overkontur dapat menyebabkan peradangan lokal seperti inflamasi gingiva, peningkatan kedalaman probing dan kehilangan tulang alveolar. Tujuan penelitian ini untuk mengetahui bagaimana deskripsi tulang alveolar pasca perawatan jembatan di RSGM UNPAD ditinjau dari radiografi per
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6

Gao, B., W. Chen, L. Hao, et al. "Inhibiting Periapical Lesions through AAV-RNAi Silencing of Cathepsin K." Journal of Dental Research 92, no. 2 (2012): 180–86. http://dx.doi.org/10.1177/0022034512468757.

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Dental caries, one of the most prevalent infectious diseases worldwide, affects approximately 80% of children and the majority of adults. Dental caries may result in endodontic disease, leading to dental pulp necrosis, periapical inflammation and bone resorption, severe pain, and tooth loss. Periapical inflammation may also increase inflammation in other parts of the body. Although many studies have attempted to develop therapies for this disease, there is still an urgent need for effective treatments. In this study, we applied a novel gene therapeutic approach using recombinant adeno-associat
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7

Boeddinghaus, R., and A. Whyte. "The many faces of periapical inflammation." Clinical Radiology 75, no. 9 (2020): 675–87. http://dx.doi.org/10.1016/j.crad.2020.06.009.

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8

Nah, Kyung Soo. "A roentgenographic study of periapical inflammation." Oral Radiology 3, no. 2 (1987): 99–104. http://dx.doi.org/10.1007/bf02351541.

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9

Andonovska, Biljana, Cena Dimova, and Saso Panov. "Matrix metalloproteinases (MMP-1, -8, -13) in chronic periapical lesions." Vojnosanitetski pregled 65, no. 12 (2008): 882–86. http://dx.doi.org/10.2298/vsp0812882a.

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Background/Aim. Matrix metalloproteinases (MMPs) are proteolytic enzymes capable of degrading almost all extracellular matrix and basement membrane components in many destructive pathological processes, such as chronic inflammation and bone-destructive lesions. The aim of this study was to determinate the correlation between concentration of collagenases (MMP-1, -8, -13) in chronic periapical lesions and their dimension calculated with software predilection through X-ray. Metods. Chronic periapical tissues were collected by periapical surgery from 60 teeth with clinically and radiographically
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10

Pattan, S. R., P. S. Bykod, P. U. Shetkar, S. V. Pattewar, S. V. Kothiwale, and J. S. Pattan. "SIGNIFICANCE AND NEED OF TREATMENT OF PERIAPICAL LESION." INDIAN DRUGS 51, no. 07 (2014): 5–13. http://dx.doi.org/10.53879/id.51.07.p0005.

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Dental caries, one of the most prevalent infectious diseases worldwide, affects approximately 80% of children and the majority of adults. Dental caries may result in endodontic disease, leading to dental pulp necrosis, periapical inflammation and bone desorption, severe pain, and tooth loss. Periapical inflammation may also increase inflammation in other parts of the body. Although many studies have attempted to develop therapies for this disease, there is still an urgent need for effective treatments.
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11

Matsumoto, Mariza Akemi, and Daniel Araki Ribeirob. "Inducible Nitric Oxide Expression Correlates with the Level of Inflammation in Periapical Cysts." European Journal of Dentistry 01, no. 04 (2007): 212–15. http://dx.doi.org/10.1055/s-0039-1698341.

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ABSTRACTObjectives: In an attempt to elucidate if inducible nitric oxide expression (iNOS) is correlated with the level of inflammation in periapical cysts with accuracy, the goal of this study was to evaluate the expression of iNOS in these ones.Methods: 30 cases were included in this study being iNOS evaluated by means of immunohistochemistry. Statistical analysis was performed by Kruskal-Wallis non-parametric test followed by the post-hoc Dunn’s test.Results: iNOS stain was detected throughout the epithelium, subepithelial fibroblasts and macrophages in all cases, indistinctly. Nevertheless
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12

Stashenko, P., R. Teles, and R. D'Souza. "Periapical Inflammatory Responses and Their Modulation." Critical Reviews in Oral Biology & Medicine 9, no. 4 (1998): 498–521. http://dx.doi.org/10.1177/10454411980090040701.

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Periapical inflammatory responses occur as a consequence of bacterial infection of the dental pulp, as a result of caries, trauma, or iatrogenic insult. Periapical inflammation stimulates the formation of granulomas and cysts, with the destruction of bone. These inflammatory responses are complex and consist of diverse elements. Immediate-type responses-including vasodilatation, increased vascular permeability, and leukocyte extravasation-are mediated by endogenous mediators, including prostanoids, kinins, and neuropeptides. Non-specific immune responses-including polymorphonuclear leukocyte a
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13

Saund, D., S. Kotecha, J. Rout, and T. Dietrich. "Non-resolving periapical inflammation: a malignant deception." International Endodontic Journal 43, no. 1 (2010): 84–90. http://dx.doi.org/10.1111/j.1365-2591.2009.01644.x.

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14

Fernández, A., P. Veloso, J. Astorga, et al. "Epigenetic regulation of TLR2‐mediated periapical inflammation." International Endodontic Journal 53, no. 9 (2020): 1229–37. http://dx.doi.org/10.1111/iej.13329.

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15

De Jesus, Sidinéia Feitoza, Yasmim Santos Meneses, Rafaela Cardoso De-Sá, et al. "Cirurgia do periápice para remoção de lesões granulomatosas: relato de caso." Revista da Faculdade de Odontologia de Porto Alegre 61, no. 2 (2020): 129–35. http://dx.doi.org/10.22456/2177-0018.102764.

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Periapical granuloma is a histological term that refers to the formation of a mass of granulomatous tissue around the apex of a tooth resulting from inflammation and necrosis of the pulp tissue. It consists of an infiltrate of inflammatory cells such as macrophages, plasmocytes and lymphocytes, as well as fibroblasts, collagen fibers and capillaries. Periapical surgery is an alternative for preserving the tooth in the oral cavity in cases where periapical inflammation is persistent even after treatment of root canals. The objective of this study was to report a clinical case in which the surgi
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16

SALIHU FAZLIU, Afërdita, Marina KACARSKA, Uran HALIMI, Blerina KOLGECI, and Gazmend FAZLIU. "PERIAPICAL INFLAMMATORY LESIONS AND DIABETES MELLITUS TYPE II– A REVIEW." International Journal of Medical Sciences - ACTA MEDICA BALKANICA 9, no. 17-18 (2024): 21–27. http://dx.doi.org/10.62792/ut.amb.v9.i17-18.p2546.

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Periapical periodontitis is a chronic inflammatory disease, caused by endodontic infection, and its development is regulated by the host immune/inflammatory response. Metabolic disorders, which are largely dependent on life style such as eating habits, have been interpreted as a “metabolically-triggered” low-grade systemic inflammation and may interact with periapical periodontitis by triggering immune modulation. Diabetes mellitus (DM) is a group of complex multisystem metabolic disorders due to a deficiency in insulin secretion caused by pancreatic β-cell dysfunction and/or insulin resistanc
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17

Yang, Wenbin, Zheng Zhu, Longjiang Li, et al. "Silencing of Ac45 Simultaneously Inhibits Osteoclast-Mediated Bone Resorption and Attenuates Dendritic Cell-Mediated Inflammation through Impairing Acidification and Cathepsin K Secretion." Infection and Immunity 89, no. 1 (2020): e00436-20. http://dx.doi.org/10.1128/iai.00436-20.

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ABSTRACTEndodontic disease is characterized by inflammation and destruction of periapical tissues, leading to severe bone resorption and tooth loss. ATP6AP1 (Ac45) has been implicated in human immune diseases, yet the mechanism underlying how Ac45 regulates immune response and reaction in inflammatory diseases remains unknown. We generated endodontic disease mice through bacterial infection as an inflammatory disease model and used adeno-associated virus (AAV)-mediated Ac45 RNA interference knockdown to study the function of Ac45 in periapical inflammation and bone resorption. We demonstrated
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18

González Herrera, Daniela, and Mabel Campos Castro. "Surgical-Endodontic Management of Periapical Cyst — A Clinical Case Report." SVOA Dentistry 6, no. 1 (2025): 41–44. https://doi.org/10.58624/svoade.2025.06.006.

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Introduction: Dental pulp is a sterile connective tissue protected by enamel, dentin and cementum. A significant injury to the dental pulp causes inflammation which can lead to pulp necrosis if left untreated. Persistent inflammation can result in periapical radiolucency observed on radiological images. Periapical cysts are cysts that affect teeth with pulp necrosis. Most root cysts develop slowly and don't grow much. Patients do not experience symptoms, so lesions are often only detected during routine radiographic examination. Case Presentation: Male patient, 56 years old, with no relevant s
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19

Ostroverkhova, N. V. "Endodontic treatment or extraction: search for compromise." Public health of the Far East Peer-reviewed scientific and practical journal 1, no. 99 (2024): 27–30. http://dx.doi.org/10.33454/1728-1261-2024-1-27-30.

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The article, using the example of clinical cases from dental practice, analyzes the causes, symptoms and tactics of dental treatment in case of periapical inflammation, an algorithm of instrumental and drug treatment. A tooth caused an apical granuloma or cysts often has to be removed. The aim of endodontic treatment is the prolonged preservation of the tooth as a functional unit of the chewing apparatus, the restoration of the health of periapic tissues and the prevention of autoinfection and sensitization of the body
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20

Brown, Jackie. "Can we improve radiological diagnosis of periapical inflammation?" British Dental Journal 189, no. 1 (2000): 31. http://dx.doi.org/10.1038/sj.bdj.4800592.

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21

Brown, Jackie. "Can we improve radiological diagnosis of periapical inflammation?" British Dental Journal 189, no. 1 (2000): 31. http://dx.doi.org/10.1038/sj.bdj.4800592a.

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22

Andrada, Ana Cristina, Mariane Maffei Azuma, Hisako Furusho, et al. "Immunomodulation Mediated by Azithromycin in Experimental Periapical Inflammation." Journal of Endodontics 46, no. 11 (2020): 1648–54. http://dx.doi.org/10.1016/j.joen.2020.07.028.

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23

Hernández Vigueras, Scarlette, Manuel Donoso Zúñiga, Enric Jané-Salas, et al. "Viruses in pulp and periapical inflammation: a review." Odontology 104, no. 2 (2015): 184–91. http://dx.doi.org/10.1007/s10266-015-0200-y.

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24

Hirai, K., H. Furusho, N. Kawashima, et al. "Serum Amyloid A Contributes to Chronic Apical Periodontitis via TLR2 and TLR4." Journal of Dental Research 98, no. 1 (2018): 117–25. http://dx.doi.org/10.1177/0022034518796456.

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In the current concept of bacterial infections, pathogen-associated molecular patterns (PAMPs) derived from pathogens and damage-associated molecular patterns (DAMPs) released from damaged/necrotic host cells are crucial factors in induction of innate immune responses. However, the implication of DAMPs in apical and marginal periodontitis is unknown. Serum amyloid A (SAA) is a DAMP that is involved in the development of various chronic inflammatory diseases, such as rheumatoid arthritis. In the present study, we tested whether SAA is involved in the pathogenesis of periapical lesions, using hu
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25

Siregar, Fazwishni, and Rini Damayanti. "Acute Toxicity of Jatropha Curcas L. Latex and Its Histopathological Effects on Dental Pulp and Periapical Tissues." Majalah Obat Tradisional 25, no. 1 (2020): 15. http://dx.doi.org/10.22146/mot.46303.

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Traditional uses of Jatropha curcas L. latex,among others, are to cure toothache. This study aimed to evaluate the acute toxicity of J. curcas latex by oral route and to assess the histopathological effects of J. curcas latex on dental pulp and periapical tissues. For acute toxicity, the latex of 5 g/kg BW was administered intragastrically to the test groups of albino mice and water to the control groups. The assessment was based upon mortality, sign of toxicity, body weight, and histological evaluation of organs. Results showed no mortality in mice up to 5 g/kg body weight, no sign of toxicit
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26

Hennet, Ph. "Endodontic Treatment Including Apexification in a Chow Chow with a Necrotic Immature Mandibular Canine Tooth." Journal of Veterinary Dentistry 15, no. 1 (1998): 21–25. http://dx.doi.org/10.1177/089875649801500102.

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A necrotic immature mandibular, canine tooth in a two year-old, male, intact Chow Chow was endodontically treated. This tooth had an open apex, wide root canal, thin dentinal walls, and there was periapical bone resorption. An apexification procedure was used to induce apical closure by calcified tissue formation, with resolution of the periapical inflammation.
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27

Qin, Han, and Jun Cai. "Clinical study of iRoot-BP combined with collagen sponge in treatment of periapical inflammation." Materials Express 14, no. 4 (2024): 677–82. http://dx.doi.org/10.1166/mex.2024.2625.

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The purpose of this research was to observe the clinical effect of iRoot-BP combined with collagen sponge for the treatment of periapical inflammation with lesions larger than 10 mm and undeveloped apical foramen. 26 teeth from 25 patients with periapical inflammation meeting the inclusion criteria were randomly assigned into two groups with 13 teeth per group. In collagen sponge group, the apical barrier was performed using iRoot-BP combined with collagen sponge, and in non-collagen sponge group was only treated with iRoot-BP. Patients in two groups were followed up at 3, 6 and 12 months resp
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28

Gupta, Geetika, Abhilasha Agarwal, Afroz Alam Ansari, and Rajeev Kumar Singh. "Non-surgical management of a large periapical lesion with internal resorption using PRF, hydroxyapatite and MTA." BMJ Case Reports 15, no. 9 (2022): e248907. http://dx.doi.org/10.1136/bcr-2022-248907.

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Periapical lesions of endodontic origin are caused by microbial infection of pulp. According to various studies, it is known that necrosis of pulp provides a favourable habitat for microbes to replicate and release various toxins into the periapical tissue leading to inflammation and formation of a periapical lesion. A variety of non-invasive methods to manage such lesions include conservative root canal treatment, aspiration-irrigation technique, decompression technique, calcium hydroxide therapy, lesion sterilisation and tissue repair therapy, and the apexum procedure. We present a case repo
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29

Nalini, MS, Balasubramanya Kumar, and Sunil Shroff. "Management of an Infected Maxillary Periapical (Radicular) Cyst through Nasal Antrostomy." Journal of Health Sciences & Research 7, no. 2 (2016): 58–62. http://dx.doi.org/10.5005/jp-journals-10042-1036.

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ABSTRACT The radicular cyst is an inflammatory cyst that is formed as a consequence of pulpal necrosis mostly due to caries, which is often found and associated with periapical inflammation. Most of the times, radicular cysts resemble the preexisting chronic periapical periodontitis lesions radiographically. In this paper, a case of radicular cyst in the maxillary anterior region is discussed. How to cite this article Nalini MS, Kumar B, Shroff S. Management of an Infected Maxillary Periapical (Radicular) Cyst through Nasal Antrostomy. J Health Sci Res 2016;7(2):58-62.
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30

Wahlgren, J., T. Salo, O. Teronen, H. Luoto, T. Sorsa, and L. Tjaderhane. "Matrix metalloproteinase-8 (MMP-8) in pulpal and periapical inflammation and periapical root-canal exudates." International Endodontic Journal 35, no. 11 (2002): 897–904. http://dx.doi.org/10.1046/j.1365-2591.2002.00587.x.

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31

Al Gashaamy, Zainab Jamal, Tiba Alomar, Linah Al-Sinjary, Mohammad Wazzan, Musab Hamed Saeed, and Natheer H. Al-Rawi. "MicroRNA expression in apical periodontitis and pulpal inflammation: a systematic review." PeerJ 11 (March 3, 2023): e14949. http://dx.doi.org/10.7717/peerj.14949.

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Background The aim of this systematic review is to determine microRNAs (miRs) that are differently expressed between diseased pulpal and periapical tissues. Design This systematic review used PubMed, Scopus, EBSCO, ProQuest, Cochrane database as well as manual searching to extract studies from January 2012 up to February 2022. Results A total of 12 studies met the eligibility criteria were included. All selected studies were of case-control type. Twenty-four miRNAs associated with apical periodontitis, 11 were found to be upregulatedand 13 were downregulated. Four out of the 44 miRs associated
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32

Krawczyk, Przemysław, Daniel Majszyk, Antoni Bruzgielewicz, and Kazimierz Niemczyk. "Inflammation of periapical tissues of the teeth, complicated by a fistula of the cheek skin." Polski Przegląd Otorynolaryngologiczny 7, no. 4 (2018): 1–5. http://dx.doi.org/10.5604/01.3001.0012.7840.

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Inflammation of the periapical tissue may lead to the development of complications involving cooperation between many medical specialists. The cutaneous fistula is a rare complication of chronic alveolus inflammation being diagnostic and therapeutic challenge due to unspecific symptoms. The correct diagnostic protocol may influence correct diagnosis, localization of the primary site of inflammation and the appropriate treatment.
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33

Takahara, Shintaro, Naoki Edanami, Razi Saifullah Ibn Belal, et al. "An Evaluation of the Biocompatibility and Chemical Properties of Two Bioceramic Root Canal Sealers in a Sealer Extrusion Model of Rat Molars." Journal of Functional Biomaterials 16, no. 1 (2025): 14. https://doi.org/10.3390/jfb16010014.

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This study assessed the biocompatibility and chemical properties of two bioceramic root canal sealers, EndoSequence BC Sealer (EBC) and Nishika Canal Sealer BG (NBG), using a sealer extrusion model. Eight-week-old male Wistar rats were used. The mesial root canals of the upper first molars were pulpectomized and overfilled with EBC, NBG, or, as reference, epoxy resin-based AH Plus (AHP). After 28 days, periapical tissue reactions were assessed using microcomputed tomography and histological staining. The elemental composition and chemical composition of the extruded EBC and NBG were analyzed a
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34

Harjunmaa, U., R. Doyle, J. Järnstedt, et al. "Periapical infection may affect birth outcomes via systemic inflammation." Oral Diseases 24, no. 5 (2018): 847–55. http://dx.doi.org/10.1111/odi.12817.

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35

Heling, I., M. Morag-Hezroni, E. Marva, N. Hochman, Z. Zakay-Rones, and A. Morag. "Is herpes simplex virus associated with pulp/periapical inflammation?" Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology 91, no. 3 (2001): 359–61. http://dx.doi.org/10.1067/moe.2001.113162.

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36

Yamauchi, Shizuko, Guy Shipper, Thomas Buttke, Mitsuo Yamauchi, and Martin Trope. "Effect of Orifice Plugs on Periapical Inflammation in Dogs." Journal of Endodontics 32, no. 6 (2006): 524–26. http://dx.doi.org/10.1016/j.joen.2005.09.005.

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37

Penarrocha, Miguel, Berta Garcia, Eva Marti, and Jose Balaguer. "Pain and Inflammation After Periapical Surgery in 60 Patients." Journal of Oral and Maxillofacial Surgery 64, no. 3 (2006): 429–33. http://dx.doi.org/10.1016/j.joms.2005.11.014.

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38

Popović, Jelena, Tatjana Cvetković, Tanja Džopalić, et al. "The role of interleukin-8 in the development and clinical progression of chronic periapical lesions." Acta Facultatis Medicae Naissensis 41, no. 3 (2024): 355–63. http://dx.doi.org/10.5937/afmnai41-48765.

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Introduction/Aim. Chronic apical periodontitis represents dynamic continuation of the presence of endodontic infection in the root canal system of the tooth, when the innate and acquired immune responses are activated and various cells and inflammatory mediators are recruited, which cause the consequent destruction of periapical tissues and the development of periapical lesions. The aim of the study was to analyze the concentration of IL-8 in tissue homogenates of periapical lesions and to compare the obtained results with the symptomatology of the patients and the size of the lesion. Methods.
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39

Pandey, Apoorva Kumar, Fatma Anjum, Saqib Ahmed, and Aparna Bhardwaj. "Peri-apical granuloma presenting as a chronic cystic swelling in the hard palate: a case report." Journal of Otolaryngology-ENT Research 16, no. 3 (2024): 58–60. http://dx.doi.org/10.15406/joentr.2024.16.00551.

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A periapical granuloma, also known as a radicular or apical granuloma, is an inflammatory condition at the tip of a nonvital (dead) tooth. This lesion, which often begins as an epithelial-lined cyst, develops into granulation tissue inflammation at the root apex of the affected tooth, usually caused by dental decay or a bacterial infection of the tooth's pulp. While it is a relatively uncommon condition with a wide range of prevalence, the term "periapical granuloma" is used even though it does not contain granulomatous inflammation. This being a rare occurrence in usual clinical otolaryngolog
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40

Migas, Klaudia, and Joanna Marchlewska. "Influence of Periapical Lesion on Healing in Sinus after Endodontics." European Journal of General Dentistry 11, no. 01 (2022): 046–50. http://dx.doi.org/10.1055/s-0041-1739509.

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Abstract Objective The goal of primary and secondary endodontic treatment is to eliminate the cause of inflammation inside the tooth and in the surrounding tissues. When the inflammation from the root canal system of the tooth spreads beyond the apex of the root, periapical changes in the bone tissue and, in the case of upper premolars and molars, inflammatory changes in the Schneider membrane may occur. Materials and Methods In a retrospective documentation analysis of root canal treatments, three-dimensional (3D) computed tomography images before and after endodontic treatment were assessed
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41

Hao, Liang, Wei Chen, Matthew McConnell, et al. "A Small Molecule, Odanacatib, Inhibits Inflammation and Bone Loss Caused by Endodontic Disease." Infection and Immunity 83, no. 4 (2015): 1235–45. http://dx.doi.org/10.1128/iai.01713-14.

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Periapical disease, an inflammatory disease mainly caused by dental caries, is one of the most prevalent infectious diseases of humans, affecting both children and adults. The infection travels through the root, leading to inflammation, bone destruction, and severe pain for the patient. Therefore, the development of a new class of anti-periapical disease therapies is necessary and critical for treatment and prevention. A small molecule, odanacatib (ODN), which is a cathepsin K (Ctsk) inhibitor, was investigated to determine its ability to treat this disease in a mouse model of periapical disea
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42

EldhoJijy, Varghese. "Non-Surgical Management of Periapical Lesion with Antibiotic Paste and Water Based Calcium Hydroxide Paste: A Case Report." Journal of International Case Reports 2, no. 3 (2023): 189–91. https://doi.org/10.5281/zenodo.8283460.

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Abstract For many years, calcium hydroxide has been utilised extensively as an intracanal medication in endodontic. It is utilised in a variety of clinical settings, including controlling root resorption, promoting apexification, repairing perforation, improving healing of periapical lesions, and reducing exudation in teeth with persistent periapical inflammation. This case report describes the use of Ca(OH)2 and double antibiotic paste as an intracanal medication to treat periradicular cystic lesion in upper maxillary central incisor of a female patient.
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43

Maria, Tanumihardja, Natsir Nurhayaty, K. Mattulata Indrya, and M. Lukman. "Potent Anti-Inflammatory Effect of Root of Sidaguri (Sida rhombifolia L) on Rat Periapical Lesion Model." International Journal of Toxicological and Pharmacological Research 8, no. 6 (2017): 412–15. https://doi.org/10.5281/zenodo.12760851.

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Sidaguri (<em>S. rhombifolia</em>&nbsp;L) is one of the most important species of medicinal part in Indonesia as anti-inflammation. To clarify this, we investigated the anti-inflammatory effect of the root of&nbsp;<em>S. rhombifolia</em>&nbsp;on rat periapical lesion model. The incisivus teeth were drilled to expose the dental pulp to the oral cavity and apical lesions were induced with LPS isolated from Porphyromonas gingivalis. Pour plate method was used to determine the bacterial CFU of gingival crevicular fluid (GCF) and C-reactive protein (CRP) serum was examined by enzyme-linked immunoso
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44

Dixit, Seema, Ashutosh Dixit, and Pravin Kumar. "Nonsurgical Treatment of Two Periapical Lesions with Calcium Hydroxide Using Two Different Vehicles." Case Reports in Dentistry 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/901497.

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Calcium hydroxide is used extensively as an intracanal medicament in endodontics for many years. It is used in various clinical situations such as to promote apexification, to repair perforation, to enhance healing of periapical lesions, to control root resorption, and to control exudation in teeth with persistent periapical inflammation. This paper presents a case report in which Ca(OH)2was used as an intracanal medicament for treatment of periradicular lesions using two different vehicles in two different teeth of same patient.
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45

Chandwani, Neelam D., Unnati Devanand Gedam, Ranjana Deshmukh, Darshan M. Dakshindas, and Manish Shrigiriwar. "Mines of cytokine: A treasure trove in pulpal and periapical diseases." Journal of Conservative Dentistry and Endodontics 27, no. 3 (2024): 227–32. http://dx.doi.org/10.4103/jcde.jcde_289_23.

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Abstract Pulpitis is a special disease of dental pulp. It causes localized inflammation, due to various inflammatory mediators such as cytokines and chemokines. These inflammatory mediators are responsible for various reparative and resorptive processes in the dental pulp. The balance between these processes ultimately determines the viability of the tooth. Due to the important properties of various inflammatory markers, the correlation of cytokinin gene expression in various stages of inflammation becomes necessary to focus on. Several studies in the past have focused on the importance of suc
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46

Nazakat Hussain Memon, Seemi Tanvir, Ayesha Munawar, Majid Ali Abbasi, Adil Khan, and Mahwish Ashraf. "Diabetes in the Pathogenesis of Periapical Lesions." Journal of Health and Rehabilitation Research 4, no. 3 (2024): 1–5. https://doi.org/10.61919/jhrr.v4i3.1646.

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Background: Diabetes mellitus is a chronic metabolic disorder associated with impaired wound healing and increased susceptibility to infections. Its impact on the development and severity of periapical lesions is clinically significant due to altered immune response and inflammatory profiles.Objective: To investigate the relationship between diabetes mellitus and the pathogenesis of periapical lesions and compare lesion characteristics between diabetic and non-diabetic patients.Methods: A comparative cross-sectional study was conducted involving 100 patients divided into diabetic (n=50) and no
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Farias, Zilda Betânia Barbosa Medeiros de, Jade Souza Cavalcante, Anne Caroline de Lima, Emanuel Savio de Souza Andrade, Márcia Maria Fonseca da Silveira, and Ana Paula Veras Sobral. "Immunohistochemical expression of macrophages in chronic periapical lesions." Research, Society and Development 10, no. 7 (2021): e35310716622. http://dx.doi.org/10.33448/rsd-v10i7.16622.

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Objective: To analyze the expression of macrophages in periapical granulomas (PGs) and radicular cysts (RCs). Methodology: We selected 264 cases of chronic periapical lesions stored at the Laboratory of Pathology, Dental School of Pernambuco (FOP)/UPE, including 89 PGs and 175 RCs. Seventy-nine cases, 23 PGs and 56 RCs, were submitted to immunohistochemical analysis by the streptavidin-biotin method using anti-CD68 antibody. The chi-square and Fisher’s exact tests were used to determine the existence of associations with categorical and clinical variables, adopting a 95% confidence level (p≤0.
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Varma, K. Meghana, Rajulapati Kalyan Satish, Girija S Sajjan, K. Madhu Varma, Bhavani Panitini, and Naveena Ponnada. "Advances in the management of post-endodontic pain: a review." International Journal of Dental Materials 05, no. 01 (2023): 13–16. http://dx.doi.org/10.37983/ijdm.2023.5104.

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This review describes the management of post-endodontic pain pharmacologically and non-pharmacologically. Acute periapical inflammation brought on by endodontic treatment-related chemical, mechanical, and/or microbiological damage to the periapical region causes postoperative pain, which is multifactorial in nature. A wide range of technical and pharmacological techniques are taken into consideration and proposed to control post-operative endodontic pain, the present study primarily comprised an overview of systematic reviews, randomized clinical trials,and invitrostudies summarizing advanceme
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49

Varma, K. Meghana, Rajulapati Kalyan Satish, Girija S. Sajjan, K. Madhu Varma, Bhavani Panitini, and Naveena Ponnada. "Advances in the management of post-endodontic pain: a review." International Journal of Dental Materials 05, no. 01 (2023): 13–16. http://dx.doi.org/10.37983/ijdm.2023.5103.

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This review describes the management of post-endodontic pain pharmacologically and non-pharmacologically. Acute periapical inflammation brought on by endodontic treatment-related chemical, mechanical, and/or microbiological damage to the periapical region causes postoperative pain, which is multifactorial in nature. A wide range of technical and pharmacological techniques are taken into consideration and proposed to control post-operative endodontic pain, the present study primarily comprised an overview of systematic reviews, randomized clinical trials, and invitro studies summarizing advance
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50

van der Meij, E. H., and J. G. A. M. de Visscher. "A periapical lesion, not always an inflammation. Overprojection of neoplastic pathology." Nederlands Tijdschrift Voor Tandheelkunde 129, no. 9 (2022): 395–407. http://dx.doi.org/10.5177/ntvt.2022.09.22052.

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