Relevant bibliographies by topics / Perinatal asphyxia

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  2. Dissertations / Theses
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  5. Conference papers

Academic literature on the topic 'Perinatal asphyxia'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Perinatal asphyxia"

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Lansac, Jacques. "Perinatal asphyxia." European Journal of Obstetrics & Gynecology and Reproductive Biology 52, no. 1 (November 1993): 77. http://dx.doi.org/10.1016/0028-2243(93)90230-a.

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Brouwer, O. F. "Perinatal asphyxia." Journal of the Neurological Sciences 120, no. 1 (December 1993): 121. http://dx.doi.org/10.1016/0022-510x(93)90037-y.

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Leviton, Alan. "Perinatal asphyxia." Pediatric Neurology 3, no. 2 (March 1987): 123. http://dx.doi.org/10.1016/0887-8994(87)90043-9.

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Jain, Dr Dharmendra, Dr Akash Kumar Pandey, Dr B. K. Das, and Dr Rajniti Prasad. "Cardiac Function in Perinatal Asphyxia." Scholars Journal of Applied Medical Sciences 4, no. 7 (July 2016): 2718–28. http://dx.doi.org/10.21276/sjams.2016.4.7.87.

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Gold, J. R. "Perinatal asphyxia syndrome." Equine Veterinary Education 29, no. 3 (December 1, 2015): 158–64. http://dx.doi.org/10.1111/eve.12467.

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Herrera, María Inés, Matilde Otero-Losada, Lucas Daniel Udovin, Carlos Kusnier, Rodolfo Kölliker-Frers, Wanderley de Souza, and Francisco Capani. "Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model." Neural Plasticity 2017 (2017): 1–8. http://dx.doi.org/10.1155/2017/3436943.

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Birth asphyxia also termed perinatal asphyxia is an obstetric complication that strongly affects brain structure and function. Central nervous system is highly susceptible to oxidative damage caused by perinatal asphyxia while activation and maturity of the proper pathways are relevant to avoiding abnormal neural development. Perinatal asphyxia is associated with high morbimortality in term and preterm neonates. Although several studies have demonstrated a variety of biochemical and molecular pathways involved in perinatal asphyxia physiopathology, little is known about the synaptic alterations induced by perinatal asphyxia. Nearly 25% of the newborns who survive perinatal asphyxia develop neurological disorders such as cerebral palsy and certain neurodevelopmental and learning disabilities where synaptic connectivity disturbances may be involved. Accordingly, here we review and discuss the association of possible synaptic dysfunction with perinatal asphyxia on the basis of updated evidence from an experimental model.
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Thompson, Debbie Gearner. "Consequences of Perinatal Asphyxia." AACN Advanced Critical Care 5, no. 3 (August 1, 1994): 242–45. http://dx.doi.org/10.4037/15597768-1994-3003.

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Perinatal asphyxia occurs in 3–9 of every 1,000 births. The risk for perinatal asphyxia is present in every pregnancy. When asphyxia is diagnosed in a newborn, the effects on the infant are potentially life-threatening. Management of the asphyxia focuses on initial stabilization and support based on identified organ system dysfunction as well as support for the infant’s family. Long-term outcome for the asphyxiated infant is related to the degree, duration, and resolution of organ system dysfunction
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., Siddharth, Preeti Lata Rai, and P. L. Prasad. "Urinary uric acid or urinary creatinine ratio as a non-invasive marker for perinatal asphyxia." International Journal of Contemporary Pediatrics 7, no. 6 (May 22, 2020): 1378. http://dx.doi.org/10.18203/2349-3291.ijcp20202151.

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Background: Perinatal asphyxia is amongst the common problem of neonates and there exists a significant contribution to the neonatal morbidity and mortality. It is observed as a common and a vital cause of the preventable cerebral injury. The prediction of the perinatal asphyxial outcome is very important but dreadful. There is a limited role for APGAR score to predict the immediate outcome, like HIE and the long-term neurological sequelae observational error may happen in APGAR. But the biochemical parameters can truly be relied upon. This study was done to assess urinary uric acid/urinary creatinine ratio (UA/Cr) as a non-invasive marker for perinatal asphyxia and co-relate its absolute value to the degree of the perinatal asphyxia.Methods: In this prospective case control study conducted in the Pediatrics Department of Shri Ram Murti Smarak Institute of Medical Sciences between Nov 2017 to May 2019, 42 asphyxiated and 42 non-asphyxiated newborns were included. Detailed history and assessment were for all the enrolled newborns. Spot urine samples were sent for the uric acid and creatinine estimation. Results were documented, and statistical analysis was performed.Results: Urinary uric acid to creatinine ratio used as additional non-invasive, early and easy biochemical marker of the birth asphyxia that biochemically supports severity grading and clinical diagnosis of the asphyxia by APGAR score.Conclusions: The ratio of the urinary uric acid and creatinine enables rapid and early recognition of asphyxial injury and also the evaluation of its severity and potential for short-term morbidity or death.
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Gebreheat, Gdiom, Tesfay Tsegay, Dessalegn Kiros, Hirut Teame, Natnael Etsay, Guesh Welu, and Desta Abraha. "Prevalence and Associated Factors of Perinatal Asphyxia among Neonates in General Hospitals of Tigray, Ethiopia, 2018." BioMed Research International 2018 (November 1, 2018): 1–7. http://dx.doi.org/10.1155/2018/5351010.

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Perinatal asphyxia is one of the most important causes of morbidity and mortality in neonates. Perinatal asphyxia occurs in association with maternal, fetal, and maternofetal factors. However, the magnitude and associated factors of perinatal asphyxia are not well studied in Tigray, Ethiopia. Therefore, our study is conducted to determine the prevalence and factors associated with perinatal asphyxia among neonates in general hospitals of Tigray. An observational hospital-based cross-sectional study was conducted in randomly selected general hospitals. A semistructured questionnaire was used to collect data from 421 randomly selected neonates with their mothers and medical records. The data was entered into epidata version 3.5 and exported to Statistical Package for Social Sciences (SPSS) version 20 for analysis. Finally, the presence of an association between a dependent variable and an independent variables has been declared at P-value ≤0.05, or adjusted odds ratio (AOR), 95% confidence interval (CI). Accordingly, the result of this study showed that the prevalence of perinatal asphyxia among the selected general hospitals was 22.1%. Neonates born with cesarean section are seven times more likely to have perinatal asphyxia than those who are born spontaneously through the vagina (AOR, 6.97; CI (2.87-16.93)). In addition, neonates who are born meconium stained are 8.55 times more likely to have perinatal asphyxia than those who had not stained with meconium (AOR, 8.55; CI (4.20-17.39)). Neonates who are weighed less than 2.5 kg are 12.75 times more likely to have perinatal asphyxia than those who are weighed 2.5-4 kg (AOR, 12.75; CI (4.05-40.08)). Prolonged duration of labour was also associated statistically with perinatal asphyxia (AOR, 3.33, CI (1.32-8.38)). In conclusion, the magnitude of perinatal asphyxia in general hospitals of Tigray remains high. Low birth weight, meconium-stained amniotic fluid, cesarean section, and prolonged maternal labour have been associated with perinatal asphyxia.
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Leone, C. R., and N. O. E. Barbosa. "Magnesium and Perinatal Asphyxia." NeoReviews 8, no. 9 (August 31, 2007): e387-e393. http://dx.doi.org/10.1542/neo.8-9-e387.

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Dissertations / Theses on the topic "Perinatal asphyxia"

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Chandrasekaran, M. "Cerebral magnetic resonance spectroscopy biomarkers and outcome in perinatal asphyxia." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1387843/.

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Background: Neonatal encephalopathy is a common clinical condition affecting 2-3 per 1,000 neonates in the developed world. 10-15% of cases will die in the neonatal period. Therapeutic hypothermia has become the standard of care in the developed world for infants with moderate to severe neonatal encephalopathy only recently, after 2 decades of experimental and clinical studies. Approximately half the infants who receive therapeutic hypothermia still have an abnormal outcome. Research is now being focused on pre-clinical and Phase II clinical studies of drugs, which act synergistically or additively with therapeutic hypothermia. Magnetic resonance spectroscopy (MRS) techniques provide translational biomarkers, which may be used to speed up the development of safe and effective neuroprotective interventions. The precise relation between MRS and brain histology is unknown and it is unclear whether cooling itself alters the prognostic efficacy of MRS. Aim: (i) To explore the relation between MRS biomarkers and the degree of brain pathology observed in our pre-clinical piglet model of perinatal asphyxia; (ii) To assess the predictive value of MRS biomarkers in infants with neonatal encephalopathy Methods: In our piglet study, data from 2 large piglet asphyxia studies investigating neuroprotective agents were analysed. 1. The first was a study of Xenon-augmented hypothermia. Following transient hypoxia-ischemia, 36 piglets were randomised into 4 groups (each n=9), with intervention from 2-26 h: Group (i) normothermia (38.5oC); Group (ii) normothermia (38.5oC) + 24 h 50% inhaled xenon; Group (iii) 24 h therapeutic hypothermia (rectal temperature (Trectal) 33.5oC) or Group (iv) 24 h 50% inhaled xenon +24 h therapeutic hypothermia (Trectal 33.5oC). 2. The second was a study investigating Amiloride as a neuroprotective agent. Following transient hypoxia-ischemia, 18 male piglets (<24 h of age) were randomized to 2 groups (each n=9) (1) normothermia; or (2) 2.5mg/kg of methyl isobutyl amiloride (MIA) at 10 minutes after resuscitation and 8 hourly thereafter. Both studies were performed on a Bruker 4.7 Tesla MR system. Following resuscitation after hypoxia-ischemia, proton (1H) magnetic resonance (MR) spectra were acquired with repetition time (TR) 5 sec, 128 summed transients, and echo times (TE) 25 ms, 144 ms, and 288 ms. (we measured the following peak area ratios: Lactate/N acetyl aspartate (Lac/NAA) and Lactate/Creatine (Lac/Cr) in both white matter and thalamus). Phosphorus (31P) MR spectra were acquired from whole brain using single-pulse acquire with TR 10 s (we measured inorganic phosphate (Pi)/exchangeable phosphate pool (EPP = Pi + PCr + (2γ +β)-NTP), NTP/EPP). Immunohistochemistry was performed on brain sections for activated Caspase 3, TUNEL positive cells and Iba I (activated microglia) to quantify cell death and microglial activation. For the clinical study, we assessed 45 infants (median gestational age – 40 weeks) with moderate to severe neonatal encephalopathy admitted over a 3 year period to the neonatal unit at UCH. Their neurodevelopmental outcome was assessed at 18 months. Results: (i) Experimental Study - Early Biomarkers Early biomarkers (acquired between 2 and 4 hours after hypoxia-ischemia) in particular thalamic Lac/NAA, predicted the 1H MRS area under the curve values from 0-48h and quantitative immunohistochemistry at 48 hours. Late Biomarkers: WM Lac/Cr at 40-48h after HI demonstrated the highest positive correlation with Tunel positive cell death (R20.98, p = 0.01) and NTP/EPP with microglial ramification (R20.68, p = 0.006); this correlation was present in both treated and untreated piglets. (ii) In the clinical study, deep gray matter Lac/NAA was the most accurate predictor of long term adverse neurological outcome following NE; importantly the predictive accuracy of Lac/NAA was unaltered by preceding therapeutic hypothermia. Conclusions: (i) Early thalamic Lac/NAA predicted the subsequent trajectory of energy disruption; this has importance in understanding the relevance of very early MRS studies in babies. White matter lactate/Cr at 40-48h correlated best with quantitative immunohistochemistry (especially TUNEL positive cells) and this relationship was present with and without preceding therapeutic intervention. (ii) In babies with neonatal encephalopathy, the predictive accuracy of Lac/NAA was unaltered by therapeutic hypothermia.
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Nguyen, Quyen. "Quantitative diffusion weighted imaging : techniques and applications to perinatal asphyxia." Thesis, University College London (University of London), 2001. http://discovery.ucl.ac.uk/1382401/.

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This thesis describes the results of a study to investigate early cerebral changes in a piglet model of perinatal asphyxia using quantitative diffusion-weighted imaging (DWI) and subsequent work to develop a robust DWI technique to enable similar studies to be performed in neonates. 31Phophorus magnetic resonance spectroscopy and quantitative diffusion and T2 imaging of the cerebrum were performed in a piglet model of perinatal asphyxia. A significant decline in the ratio of phosphocreatine to inorganic phosphate concentrations ([PCr]/[Pi]) was observed during the 48 hours following the transient hypoxicischaemic (H-I) insult. The global directionally averaged apparent diffusion coefficient (ADCav) also declined significantly during the same period and a strong correlation between the [PCr]/[Pi] and ADCav was found. Strong regional and temporal variations in the cerebral response were observed following the H-I insult. In the basal ganglia and parasagittal cortex, significant decline in ADCs was seen approximately 8 hours after the H-I insult. In the thalamus, internal capsule, periventricular white matter and medial cortex, significant ADCs decline was not observed until 32 hours following the H-I insult. A significant T2 increase was observed in the internal capsule but not in the other regions of interest. To enable clinical DWI to be performed in neonates a novel `reacquisition' technique that overcomes the problem of motion artefact in DWI is presented. The reacquisition technique involves the automatic detection and reacquisition of motion-corrupted data in real-time. Computer simulations were used to demonstrate that motion-corrupted data may be detected accurately and reacquired in a time efficient manner. The reacquisition technique was implemented on a Bruker AVANCE scanner in combination with a spinecho 2DFT DWI sequence and an interleaved EPI DWI sequence. The effectiveness of the technique was demonstrated with both a computer-controlled motion phantom and neonates from an ongoing study of perinatal asphyxia.
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Hoque, Nicholas Nadir. "Selective head cooling for perinatal asphyxia: effects on physiology and brain injury." Thesis, University of Bristol, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.654564.

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Perinatal asphyxia remains a major cause of morbidity and mortality throughout the world. Despite improvements in obstetric and neonatal care there has been little improvement in rates of death and disability. Therapeutic hypothermia has recently been shown to be the only effective treatment following perinatal asphyxia. Selective Head Cooling (SHe) and Whole Body Cooling (WBe) are methods of applying therapeutic hypothermia. Yet the optimum method of cooling for maximum neuroprotection and minimum adverse effects remains unclear. We retrospectively compared four methods of cooling newborns following perinatal asphyxia. One method of SHC and three methods of WBC, using water-filled gloves, a coolant- filled mattress, and a water- filled body wrap. All methods maintained rectal t emperature within narrow target range. We found that WBC using a servo- controlled system minimised initial over-cooling and reduced subsequent fluctuation in rectal t emperature compared with manually-controlled WBC or SHe. We found similar variation in rectal temperature, heart rate, and mean arterial blood pressure using SHC and manually controlled WBe. We developed a technique of applying SHC with minimal systemic hypothermia as a t reatment for perinatal asphyxia in our established experimental model. We conducted a study to assess the neuroprotective and physiological effects of our technique compared to standard care under normothermic conditions. We did not find any difference in brain injury between treatment groups but increased mortality in the hypothermia group. We have.previously used a neuropathology score in our established model of perinatal asphyxia in order to assess treatment effects. Our neuropathology score incorporates subjective assessments of area injure'd and morphological cellular changes in several brain regions. We conducted quantitative cell counting to assess brain injury following perinatal asphyxia. Cell count correlated with neuropathology score in the putamen and hippocampus CA1 and we were able to validate our neuropathology score to assess outcome.
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Berg, Wilma Dorethea Johanna van de. "Cell death and synaptic remodelling as a consequence of perinatal asphyxia implications of hypothermia /." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 2003. http://arno.unimaas.nl/show.cgi?fid=6331.

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Suarez, Olivia Adayr Xavier [UNESP]. "O monitoramento dos movimentos corporais do sono e do ritmo de sono-vigília como indicador das alterações provocadas pela asfixia em ratos recém-nascidos." Universidade Estadual Paulista (UNESP), 2006. http://hdl.handle.net/11449/96121.

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As limitações dos métodos diagnósticos da asfixia perinatal levaram-nos a investigar se o monitoramento dos movimentos corporais do sono (MCS) e do ritmo de sono-vigília (S/V) pode ser utilizado como marcador diagnóstico deste processo patológico. Para tanto, os registros eletromiográfico e eletrocardiográfico de 8 ratos recém-nascidos, Wistar, com 6 a 48h de vida, foram obtidos durante 4 períodos experimentais: período controle - com ratos respirando ar atmosférico, por 30min; período de asfixia - com ratos submetidos a dois períodos de asfixia de 30min (T1 e T2), com seus corpos inteiramente envolvidos por filme de polivinil (PVC); e período de recuperação - com ratos respirando ar ambiente novamente, por 30min. A freqüência cardíaca diminuiu significantemente durante a asfixia, em relação à fase controle. Nós concluímos que o monitoramento dos MCS e do ritmo de sono-vigília se mostrou um parâmetro promissor no diagnóstico da asfixia perinatal e de suas complicações. Este fato, todavia, requer novas investigações para se estabelecer a real viabilidade do método para o uso rotineiro na prática clínica.
The limitations of current diagnostic methods of perinatal asphyxia induced to investigate if monitoring of body movements and of the sleep-wake rhythm can be used as diagnostic marker of this pathologic process. So, electromyogram and electrocardiogram records of 8 newborn Wistar rats, 6 to 48 hours postnatal, were obtained throughout 4 experimental periods: control period - with rats breathing room air for 30 min (ARpre); asphyxia periods - with rats submitted to two 30 min asphyxia periods (T1 and T2) with Its entire body covered with a polyvinyl sheet (PVC); and recovering period - with rats breathing room air for 30 min again. Asphyxia Heart rate was significantly decreased all over the asphyxia period compared with the control phase. In conclusion the monitoring of SBM, and of the sleep-wake rhythm seems to be of utmost value to the perinatal asphyxia diagnosis and its complications. This fact, however, requires new investigations to establish the real viability of the method for clinical practice routine.
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Suarez, Olivia Adayr Xavier. "O monitoramento dos movimentos corporais do sono e do ritmo de sono-vigília como indicador das alterações provocadas pela asfixia em ratos recém-nascidos /." Botucatu : [s.n.], 2006. http://hdl.handle.net/11449/96121.

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Orientador: Katsumasa Hoshino
Banca: Lígia Maria Suppo de Souza Sugolo
Banca: Mônica Levy Andersen
Resumo: As limitações dos métodos diagnósticos da asfixia perinatal levaram-nos a investigar se o monitoramento dos movimentos corporais do sono (MCS) e do ritmo de sono-vigília (S/V) pode ser utilizado como marcador diagnóstico deste processo patológico. Para tanto, os registros eletromiográfico e eletrocardiográfico de 8 ratos recém-nascidos, Wistar, com 6 a 48h de vida, foram obtidos durante 4 períodos experimentais: período controle - com ratos respirando ar atmosférico, por 30min; período de asfixia - com ratos submetidos a dois períodos de asfixia de 30min (T1 e T2), com seus corpos inteiramente envolvidos por filme de polivinil (PVC); e período de recuperação - com ratos respirando ar ambiente novamente, por 30min. A freqüência cardíaca diminuiu significantemente durante a asfixia, em relação à fase controle. Nós concluímos que o monitoramento dos MCS e do ritmo de sono-vigília se mostrou um parâmetro promissor no diagnóstico da asfixia perinatal e de suas complicações. Este fato, todavia, requer novas investigações para se estabelecer a real viabilidade do método para o uso rotineiro na prática clínica.
Abstract: The limitations of current diagnostic methods of perinatal asphyxia induced to investigate if monitoring of body movements and of the sleep-wake rhythm can be used as diagnostic marker of this pathologic process. So, electromyogram and electrocardiogram records of 8 newborn Wistar rats, 6 to 48 hours postnatal, were obtained throughout 4 experimental periods: control period - with rats breathing room air for 30 min (ARpre); asphyxia periods - with rats submitted to two 30 min asphyxia periods (T1 and T2) with Its entire body covered with a polyvinyl sheet (PVC); and recovering period - with rats breathing room air for 30 min again. Asphyxia Heart rate was significantly decreased all over the asphyxia period compared with the control phase. In conclusion the monitoring of SBM, and of the sleep-wake rhythm seems to be of utmost value to the perinatal asphyxia diagnosis and its complications. This fact, however, requires new investigations to establish the real viability of the method for clinical practice routine.
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Andersen, Danielle Louise. "Development of neurotransmitter receptors in the human brain and vulnerability to perinatal asphyxia and sudden infant death syndrome /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17412.pdf.

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Vargas, Nadia Sandra Orozco. "Marcadores prognósticos de evolução neonatal de recém-nascidos de termo portadores de asfixia perinatal." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-29082012-135124/.

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A asfixia perinatal e sua mais grave complicação, a encefalopatia hipóxicoisquêmica (EHI) são causas de elevada mortalidade e de sequelas neurológicas no recém-nascido (RN). Evidencias de comprometimento cerebral podem ser detectadas logo na primeira semana de vida e o diagnóstico precoce é de grande importância para o tratamento e seguimento. OBJETIVOS: Verificar a prevalência de asfixia perinatal e de EHI; analisar a utilidade de quatro marcadores sanguíneos: transaminase glutâmica oxalacética (TGO), transaminase glutâmica pirúvica (TGP), desidrogenasse láctica (DHL) e Creatina Quinase MB (CK-MB) coletados ao nascimento, com 24 e 72 horas de vida, como sinalizadores de lesões cerebrais nos RN asfixiados; identificar a presença de alterações neurológicas clínicas pelo Escore de Sarnat e Sarnat (1976) com 24 e 72 horas e com 28 dias de vida e identificar e descrever a presença de lesões cerebrais detectadas pela ultrassonografia de crânio com 24 e 72 horas e com 28 dias de vida. MÉTODO: Estudo de coorte prospectivo no qual foram incluídos RN de termo que apresentaram asfixia perinatal pelo critério de Buonocore (presença ao nascimento de pelo menos dois das seguintes condições clínico-laboratoriais: acidose metabólica com níveis de pH 7,20, Boletim de Apgar no quinto minuto de vida < 6, necessidade de fração inspirada de oxigênio 0,40 para manter uma saturação de oxigênio de 86%). Para realização do pH e dos marcadores bioquímicos foi coletado sangue logo ao nascimento, com 24 e 72 horas de vida. O exame clínico segundo critérios de Sarnat e Sarnat foi realizado com 24 e 72 horas e com 28 dias de vida e a ultrassonografia de crânio nos mesmos momentos. RESULTADOS: De 2.989 nascidos vivos durante o período do estudo, 28 recém-nascidos apresentaram asfixia perinatal (1% do total de nascimentos) e a EHI foi evidenciada em 21,42%. A enzima CK-MB se mostrou um bom marcador de asfixia, pois todos os valores foram superiores a 5,10 ng/ml, e se correlacionaram positivamente com as alterações apresentadas no exame clínico de Sarnat e com a ultrassonografia transfontanela. Os valores das outras enzimas como TGO (24h), TGO e TGP (72h) também se correlacionaram positivamente com as alterações ultrassonográficas as quais mostraram alteração em 3,5% dos pacientes com 24 horas de vida, 25% com 72 horas e 28,6% com 28 dias. A ultrassonografia de crânio com 28 dias mostrou aumento estatisticamente significante, no percentual de resultados anormais quando comparado com o observado com 24h (p=0,039), apesar dos estágios de Sarnat terem melhorado, com maior número de pacientes com estágio I no decorrer dos 28 dias. CONCLUSOES: A prevalência de asfixia perinatal e da EHI estão dentro da faixa citada na literatura. O melhor marcador bioquímico nesta casuística foi a isoenzima CK-MB e se correlacionou com as alterações apresentadas no exame clínico de Sarnat e na USG de crânio. A curva ROC mostrou: os valores de CK-MB de 24 horas em relação à USG de crânio de 72 horas Sensibilidade de 85,7%, Especificidade de 85,7% e Acurácia de 85,7%. O escore clínico de Sarnat não se alterou depois de 72 horas e apresentou correlação com as alterações na USG de crânio e este método de imagem foi adequado para detecção de lesões crebrais precoces e com 28 dias de vida
Perinatal asphyxia and its most serious complication, hypoxic-ischemic encephalopathy (HIE) are causes of high mortality and neurological sequelae in the newborn (NB). Evidence of cerebral impairment can be detected in the first week of life and early diagnosis is very important for the treatment and follow-up. OBJECTIVES: To assess the prevalence of perinatal asphyxia and HIE; consider the usefulness of four blood markers: glutamic oxaloacetic transaminase (GOT), glutamic pyruvic transaminase (GPT), lactate dehydrogenase (LDH) and creatine kinase MB (CK-MB) collected the birth, with 24 and 72 hours of life, as markers of brain damage in asphyxiated infants, to identify the presence of neurological clinical score by Sarnat and Sarnat (1976) with 24 and 72 hours and 28 days of life and describe the presence of brain lesions detected by cranial ultrasound with 24 and 72 hours and 28 days. METHOD: A prospective cohort study which included fullterm infants who had perinatal asphyxia by Buonocore criteria (presence at birth of at least two of the following clinical and laboratory conditions: metabolic acidosis with pH levels of the umbilical vein 7.20, Apgar score in the fifth minute of life <6, need for inspired oxygen fraction (FiO2) 0.40 to maintain an oxygen saturation of 86%). To perform the pH and biochemical markers of blood was collected at birth, 24 and 72 hours of life. Clinical examination according to criteria of Sarnat and Sarnat was performed with 24 and 72 hours and 28 days of life and cranial ultrasound was performed in the same time. RESULTS: Of 2989 live births during the study period, 28 had asphyxia (1% of total births) and HIE was found in 21.42%. The CK-MB showed that all above normal values (<5.10ng/ml) and correlated with the changes presented in the clinical examination of Sarnat and the USG transfontanelle. The values of other enzymes such as GOT (24h), GOT and GPT (72h) also correlated positively with the brain lesins detected by cranial ultrasound in 3.5% of patients with 24 hours of life, 25% at 72 hours and 28 6% after 28 days. Ultrasonography of brain at 28 days showed a statistically significant increase in the percentage of abnormal results when compared with that observed at 24h (p = 0.039), despite the Sarnat stages have improved, with larger numbers of patients with stage I during the 28 days. CONCLUSION: The prevalence of perinatal asphyxia and HIE is within the range cited in literature. The best biochemical marker in this series was the CK-MB and correlated with the changes presented in the clinical examination of Sarnat and ultrasound transfontanelle. The ROC curve showed: values of CK-MB of 24 hours and USG 72 hours sensitivity of 85.7%, specificity of 85.7% and accuracy of 85.7%. The clinical Sarnat score did not change after 72 hours and correlated with changes in ultrasound transfontanelle and this imaging method proved to be an appropiate study to detect brain lesions early and with 28 days of life
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Freitas, Záira Moura da Paixão. "Influência da avaliação neurológica seriada e seus reflexos no prognóstico funcional de recém-nascidos a termo com asfixia perinatal." Universidade Federal de Sergipe, 2016. http://ri.ufs.br:8080/xmlui/handle/123456789/3634.

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Background: According to the World Health Organization, between four to nine million newborns develop birth asphyxia. It is estimated that 1.2 million fatal outcomes and at least the same number develop disabling neurological sequelae and developmental delay. Early neurological evaluation promotes the improvement of the life expectancy of serious asphyxiated babies and It can be incorporated into the routine of neonatal intensive care units. Glasgow Coma Scale adapted for children can be an assessment tool used in high-risk newborns. Objective: To compare the clinical outcome of term infants with perinatal asphyxia (PA) moderate and severe that underwent a neurological neonatal serial evaluation protocol during hospital stay, with those not undergoing evaluation. Method: This is an epidemiologic, observational and analytical study, using a quantitative approach. The study considered two groups, intervention and control. For the control group, at first, it was made a survey data in charts and then the longitudinal, prospective, observational approach was adopted during the period of outpatient care. For the intervention group, a longitudinal approach, prospective, observational was used during the hospital stay and follow-up clinic. We sought to investigate the neurological clinical course of children, the length of stay in hospital and delay presence in the development of neuromotor skills. Results: The sample consisted of 112 newborns, 86 infants in the control group and 26 infants in the intervention group. The intervention group showed a median of less hospital stay (p<0.001) than the control group. Full-term newborns diagnosed with PA, which remained for a shortest time in hospital length and were subjected to a serial neurological evaluation protocol, incorporated into standard clinical procedures in the NICU for the management of PA, did not show disturbances in neuromotor development. Conclusion: The use of serial neurological evaluation protocol influenced significantly, the prevalence of delayed neuromotor development.
Introdução: Segundo a Organização Mundial da Saúde, cerca de quatro a nove milhões de recém-nascidos (RN) desenvolvem asfixia ao nascer. Estima-se que 1,2 milhão evoluem para óbito e, pelo menos, o mesmo número desenvolve sequelas neurológicas incapacitantes e atraso no desenvolvimento. A avaliação neurológica precoce promove melhora da perspectiva de vida dos anoxiados graves, podendo, esta, ser incorporada à rotina das unidades de terapia intensiva neonatal. A escala de coma de Glasgow adaptada para crianças poderá ser utilizada em RN de alto risco. Objetivo: Comparar a evolução clínica neurológica dos RN a termo com asfixia perinatal (AP) moderada e grave que foram submetidos a um protocolo de avaliação neurológica neonatal seriada durante período de internamento hospitalar, com aqueles não submetidos à avaliação. Métodos: Trata-se de um estudo epidemiológico, observacional, analítico, com abordagem quantitativa. O estudo considerou dois grupos, intervenção e controle. Para o grupo controle foi feito um levantamento de dados em prontuários e, em seguida, adotada a abordagem longitudinal, prospectiva, observacional, durante o período de atendimento ambulatorial. Para o grupo intervenção, foi utilizada uma abordagem longitudinal, prospectiva, observacional, durante o período intra-hospitalar e em ambulatório de seguimento. Buscou-se investigar a evolução clínica neurológica da criança, o tempo de permanência no serviço hospitalar e presença de atraso no desenvolvimento das habilidades neuromotoras. Resultados: A amostra foi constituída de 112 RN, sendo 86 RN do grupo controle e 26 RN do grupo intervenção. O grupo intervenção mostrou uma mediana de tempo de internamento hospitalar menor (p<0,001) que o grupo controle. RN a termo, diagnosticados com AP, que permaneceram por menor tempo em internamento hospitalar e foram submetidos a um protocolo de avaliação neurológica seriada, incorporado às condutas clínicas padronizadas na UTIN para a gestão da AP, não apresentaram distúrbios no desenvolvimento neuromotor. Conclusão: A utilização do protocolo de avaliação neurológica seriada influenciou a prevalência de atraso no desenvolvimento neuromotor.
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Cruz, Ana Cristina Silvestre da. "Prevalência de asfixia perinatal e encefalopatia hipóxico-isquêmica em recém-nascidos de termo considerando dois critérios diagnósticos e o tipo de assistência obstétrica." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-17122008-104415/.

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INTRODUÇÃO: A asfixia perinatal é uma das principais causa de óbito nos recémnascidos (RN) de termo acima de 2500g no Brasil, sendo também a causa mais importante de encefalopatia e lesão cerebral permanente em crianças. Não existindo ainda um consenso acerca de qual seria o melhor critério para seu diagnóstico. OBJETIVOS: Verificar a prevalência de asfixia e de encefalopatia hipóxico-isquêmica segundo dois critérios diagnósticos, avaliar influência do tipo de parto e a evolução neurológica. MÉTODO: Corte transversal prospectivo, onde foram incluídos 30 recém-nascidos que apresentaram asfixia segundo dois critérios diagnósticos: critério 1 foi preconizado pela AAP/ACOG de 1996 (pH de cordão 7,0, disfunção múltipla de órgãos, manifestações neurológicas na primeira semana de vida além do Apgar entre 0-3 no quinto minuto); o critério 2 foi de Buonocore em 2002, modificado (pH de cordão 7,2, Apgar de 4-6 no quinto minuto e necessidade de fração de oxigênio inspirada 0,40 manter saturação de 86%); num período de dois anos (2004/2006) sendo excluídos aqueles que pudessem apresentar encefalopatia por outras causas como malformações, infecções congênitas, erro inato do metabolismo. Para realizar o diagnóstico foram colhida gasometria de cordão dos recémnascidos a termo que apresentaram Apgar de quinto minuto 6 e feitas provas de função cardíaca, hepática, renal e controle hematológico além da avaliação neurológica pelos critérios clínicos de Sarnat e Sarnat de 1976, para verificar o grau de encefalopatia. RESULTADOS: Durante este período a prevalência observada de asfixia foi de 3,2 por 1000 nascimentos a termo (IC a 95% - [2,1 por mil; 4,5 por mil]) e de encefalopatia de 1,7 por 1000 nascimentos a termo (IC a 95% - [0,8 por mil; 2,5 por mil]). A taxa de mortalidade foi de 16,7% e 36,7% evoluíram com encefalopatia grave. Não houve correlação estatística da asfixia e nem da encefalopatia quanto às características maternas, exceto uma tendência maior nas nulíparas e primíparas com parto normal. Quanto à indicação do parto 46,7% apresentava trabalho de parto sem intercorrências, mas pelo critério 1 houve maior número com sofrimento fetal relacionado à maior gravidade da asfixia. O sexo mais freqüente foi o masculino e em ambos os grupos apresentaram acidose metabólica e respiratória e alterações enzimáticas principalmente cardíacas e hepáticas e, função renal com aumento da creatinina. Foi observado que para Sarnat estágios 1 e 2, leve e moderada, houve uma maior proporção de recém-nascidos no critério 2 enquanto que para o Sarnat estágio 3, grave, a maior proporção foi com o critério 1 Resumo (p = 0,016). Enquanto o Apgar de primeiro minuto não mostrou correlação com a gravidade da encefalopatia, 85% dos recém-nascidos com encefalopatia leve/moderada tiveram Apgar de quinto minuto entre 4-6 e a maioria com quadro grave o Apgar foi entre 0-3 (p = 0,018). Houve uma tendência de acordo com o aumento da gravidade da encefalopatia a uma redução do dióxido de carbono sanguíneo, bicarbonato e aumento negativo do excesso de base além do aumento de enzima cardíaca (creatina fosfoquinase), mas não foi estatisticamente significante. CONCLUSÃO: Não houve correlação estatística entre asfixia e a gravidade da encefalopatia com fatores maternos. Todos os recém-nascidos apresentaram acidose respiratória e metabólica e entre as alterações enzimáticas cardíacas foram as mais importante. Em relação ao índice de Apgar, a nota de quinto minuto mostrou melhor correlação com a gravidade evolutiva dos pacientes. Com o critério 1 (Academia Americana de Pediatria) houve melhor correlação com a mortalidade, no entanto por ser muito rigoroso acaba por excluir recém-nascidos que evoluem com quadros de encefalopatia grave
INTRODUCTION: The perinatal asphyxia is one of the main causes of death in newborns and also the most important cause of encephalopathy and permanent cerebral lesion in children. OBJECTIVES: To check the prevalence of asphyxia and of hypoxic-ischemic encephalopathy in term newborns, using two diagnostic criteria; to assess whether the diagnostic criterion used and the type of obstetric assistance are related to the grade of seriousness of the asphyxia and of the encephalopathy. Methods: Prospective transversal cut study carried out in a public hospital in the East Zone of São Paulo, in which 30 term newborns with perinatal asphyxia were included and classified in two groups, according to two diagnostic criteria adopted: criterion 1 recommended by American Academy of Pediatrics (1996), and which considers as bearer of perinatal asphyxia the newborn presenting: cord pH 7.0, multiple organ dysfunction, neurological manifestations in the first week of life and Apgar value in the fifth minute of life between 0-3. Criterion 2 defined by Buonocore in 2002 and which consists in: cord pH 7.2, Apgar value in the fifth minute of life between 4-6 and fraction inspired of oxygen need 0.40 to maintain a saturation of 86%. To confirm the diagnosis, the following laboratorial examinations were carried out: gasometry, hepatic, renal and cardiac function tests, besides the hematological control. To assess the neurological function and verify the grade of hypoxic-ischemic encephalopathy, the clinical criteria of Sarnat and Sarnat were used. RESULTS: The prevalence of perinatal asphyxia observed in this case was of 3.2 per 1,000 term births (IC at 95% - [2.1 per one thousand; 4.5 per one thousand]) and of hypoxic-ischemic encephalopathy was of 1.7 per 1,000 term births (IC at 95% - [0.8 per one thousand; 2.5 per one thousand]). As regards the criteria used, the newborns of criterion 1 statistically presented more fetal suffering when compared to those of criterion 2, and this fact was also related to the grade of seriousness of the asphyxia. The newborns of the two groups presented cardiac changes with elevation of the specific enzyme, hepatic changes with elevation of the glutamic pyruvic and oxaloacetic transaminases and renal changes proven by elevation of creatinine, besides the relevant respiratory and metabolic acidosis. The newborns with serious metabolic acidosis and high levels of creatine phosphokinase had a greater degree of neurological impairment. In 85% of newborns with light/moderate encephalopathy was verified an Apgar value at fifth minute of life between 4-6, and in newborns with serious encephalopathy this value was between 0-3 (p = 0.018). A positive trend for Summary the presence of asphyxia and encephalopathy was found in children of primiparous mothers and born during normal parturition. When assessing the degree of neurological impairment through the criteria of Sarnat and Sarnat, A greater proportion of newborns of criterion 2 were found in the lighter degrees. In degree 3, which is the most serious, a greater proportion of newborns of criterion 1 (p = 0,016) was found. The mortality rate in these cases was of 16.7%, and most of the newborn were of criterion 1. CONCLUSION: The prevalence of perinatal asphyxia and hypoxic -ischemic encephalopathy is as mentioned in the world literature, and smaller than found in Brazil. Criterion 1 was the one that showed a better correlation with the mortality of patients. However, as it is too rigorous, it may exclude the newborn that survive and develop hypoxic-ischemic encephalopathy. As regards the type of obstetric assistance, despite the fact that no statistically significant difference was observed, there was a positive trend to the presence of asphyxia and encephalopathy in children of primiparous mothers born during normal parturition
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Books on the topic "Perinatal asphyxia"

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Haddad, Joseph, and Elie Saliba, eds. Perinatal Asphyxia. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77896-4.

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Lacoius-Petruccelli, Alberto. Perinatal Asphyxia. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1.

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Perinatal asphyxia. New York: Plenum Medical Book Co., 1986.

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Ruth, Vineta. Perinatal asphyxia: Biochemical parameters as indices of asphyxia at birth and predictors of brain damage, and a trial of preventing damage by phenobarbital. Helsinki: University of Helsinki, 1988.

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L, Wright Linda, Merenstein Gerald B, Hirtz Deborah, National Institute of Child Health and Human Development (U.S.), National Institute of Neurological Disorders and Stroke (U.S.), American Academy of Pediatrics, and American College of Obstetricians and Gynecologists., eds. Report of the Workshop on Acute Perinatal Asphyxia in Term Infants: August 30-31, 1993, Rockville, Maryland. [Washington, D.C.?]: The Institute, 1996.

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Workshop on Acute Perinatal Asphyxia in Term Infants (1993 Rockville, Md.). Report of the Workshop on Acute Perinatal Asphyxia in Term Infants: August 30-31, 1993, Rockville, Maryland. [Washington, D.C.?]: The Institute, 1996.

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Symposium on Perinatal Asphyxia (1988 Toronto, Ont.). Perinatal asphyxia: Its role in developmental deficits in children : papers and discussion based on a symposium held October 26, 1988, in Toronto, Ontario, Canada, jointly with the 42nd Annual Meeting of the American Academy for Cerebral Palsy and Developmental Medicine and sponsored by the Canadian Medical Protective Association. Ottawa, Ontario: Canadian Medical Protective Association, 1988.

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Lacoius-Petruccelli, Alberto. Perinatal Asphyxia. Vantage Press, 2006.

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J, Haddad, Saliba E. 1950-, and Arbeille Ph, eds. Perinatal asphyxia. Berlin: Springer-Verlag, 1993.

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Haddad, Joseph, and Elie Saliba. Perinatal Asphyxia. Springer, 2011.

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Book chapters on the topic "Perinatal asphyxia"

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Lacoius-Petruccelli, Alberto. "Asphyxia." In Perinatal Asphyxia, 11–15. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_1.

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Pierre, F. "Perinatal Management." In Perinatal Asphyxia, 229–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77896-4_17.

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Braun-Falco, Markus, Henry J. Mankin, Sharon L. Wenger, Markus Braun-Falco, Stephan DiSean Kendall, Gerard C. Blobe, Christoph K. Weber, et al. "Perinatal Asphyxia." In Encyclopedia of Molecular Mechanisms of Disease, 1611–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_154.

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Lacoius-Petruccelli, Alberto. "The Placenta." In Perinatal Asphyxia, 77–86. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_10.

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Lacoius-Petruccelli, Alberto. "Fetal Circulation." In Perinatal Asphyxia, 87–89. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_11.

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Lacoius-Petruccelli, Alberto. "Placental Insufficiency or Dysfunction." In Perinatal Asphyxia, 91–95. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_12.

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Lacoius-Petruccelli, Alberto. "Fetal Breathing Movements." In Perinatal Asphyxia, 97–99. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_13.

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Lacoius-Petruccelli, Alberto. "Adjustment in the Mechanism of Oxygenation at Birth." In Perinatal Asphyxia, 101–11. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_14.

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Lacoius-Petruccelli, Alberto. "Birth Asphyxia in the Full-Term Infant." In Perinatal Asphyxia, 113–17. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_15.

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Lacoius-Petruccelli, Alberto. "Detecting Fetal Distress." In Perinatal Asphyxia, 119–20. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1807-1_16.

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Conference papers on the topic "Perinatal asphyxia"

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Calabrese, Clementina, Alessandra Marinari, Lucia Taurino, Alessandra Catucci, Lorenza Chiossi, Salvatore Cringoli, Francesca Lotti, et al. "P114 Also adipose tissue pays consequencies of perinatal asphyxia." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.202.

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Onu, Charles C., Jonathan Lebensold, William L. Hamilton, and Doina Precup. "Neural Transfer Learning for Cry-Based Diagnosis of Perinatal Asphyxia." In Interspeech 2019. ISCA: ISCA, 2019. http://dx.doi.org/10.21437/interspeech.2019-2340.

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Onu, Charles C. "Harnessing infant cry for swift, cost-effective diagnosis of Perinatal Asphyxia in low-resource settings." In 2014 IEEE Canada International Humanitarian Technology Conference (IHTC). IEEE, 2014. http://dx.doi.org/10.1109/ihtc.2014.7147559.

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Hardie, Morgan, Amy Lesneski, Satyan Lakshminrusimha, and payam vali. "Umbilical Cord Milking (UCM) During Positive Pressure Venitlation (PPV) Stablizes Hemoglobin but Increases Systemic Blood Pressure and Heart Rate In Perinatal Asphyxia." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.747.

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Hütten, S., R. Schlößer, and L. Schrod. "Asphyxie und therapeutische Hypothermie im klinischen Alltag." In 28. Deutscher Kongress für Perinatale Medizin. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1607689.

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Zorbach, C., B. Wittekindt, and R. Schlößer. "Neonatale Asphyxie: Retrospektive Analyse der Behandlungsfälle eines tertiären Zentrums." In 29. Deutscher Kongress für Perinatale Medizin. Deutsche Gesellschaft für Perinatale Medizin (DGPM) – „Hinterm Horizont geht's weiter, zusammen sind wir stark“. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-3401261.

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Sankaran, Deepika, Peggy Chen, Ziad Alhassen, Amy Lesneski, Morgan Hardie, Satyan Lakshminrusimha, and payam vali. "Optimal Inspired Oxygen Weaning Strategy Following Return of Spontaneous Circulation After Perinatal Asphyxial Arrest." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.757.

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