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Journal articles on the topic 'Perinatal damage'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

RUTH, VINETA J., and KARL O. RAIVIO. "Perinatal Brain Damage." Obstetrical & Gynecological Survey 44, no. 4 (1989): 267–68. http://dx.doi.org/10.1097/00006254-198904000-00014.

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2

Dammann, Olaf, and Alan Leviton. "Perinatal Brain Damage Causation." Developmental Neuroscience 29, no. 4-5 (2007): 280–88. http://dx.doi.org/10.1159/000105469.

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3

Leviton, Alan, and Pierre Gressens. "Neuronal damage accompanies perinatal white-matter damage." Trends in Neurosciences 30, no. 9 (2007): 473–78. http://dx.doi.org/10.1016/j.tins.2007.05.009.

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4

IKEDA, Tomoaki. "Prevention of Perinatal Brain Damage." TRENDS IN THE SCIENCES 15, no. 4 (2010): 8–14. http://dx.doi.org/10.5363/tits.15.4_8.

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5

Evrard, Philippe. "Pathophysiology of Perinatal Brain Damage." Developmental Neuroscience 23, no. 3 (2001): 171–74. http://dx.doi.org/10.1159/000046138.

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6

Berger, Richard, and Yves Garnier. "Pathophysiology of perinatal brain damage." Brain Research Reviews 30, no. 2 (1999): 107–34. http://dx.doi.org/10.1016/s0165-0173(99)00009-0.

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7

Kjellmer, Ingemar. "Mechanisms of Perinatal Brain Damage." Annals of Medicine 23, no. 6 (1991): 675–79. http://dx.doi.org/10.3109/07853899109148102.

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8

Silverman, William A. "'Collateral damage' in perinatal warfare." Paediatric and Perinatal Epidemiology 16, no. 2 (2002): 98–99. http://dx.doi.org/10.1046/j.1365-3016.2002.00400.x.

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9

Dammann, Olaf, and T. Michael O'Shea. "Cytokines and Perinatal Brain Damage." Clinics in Perinatology 35, no. 4 (2008): 643–63. http://dx.doi.org/10.1016/j.clp.2008.07.011.

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10

H., Ziyadullaeva, Turaeva I., Nizamova D., Ravshanova B., and Sharipova M. "The state of neonatal neurosonography in infants with perinatal nervous system damage." International Journal of Medical Sciences And Clinical Research 5, no. 1 (2025): 75–78. https://doi.org/10.37547/ijmscr/volume05issue01-12.

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In this study, the authors conducted ultrasound examinations of the central nervous system in newborns with perinatal nervous system damage. It was observed that infants born to mothers with a complicated obstetric history and pathological course of delivery constitute a high-risk group for perinatal nervous system damage. To early detect structural brain changes, neurosonographic examinations of the central nervous system are recommended for all newborns in the high-risk group.

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11

Florio, Pasquale. "Biochemical markers of perinatal brain damage." Frontiers in Bioscience S2, no. 1 (2010): 47–72. http://dx.doi.org/10.2741/s45.

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12

Symonds, E. M. "Antenatal, perinatal, or postnatal brain damage?" BMJ 294, no. 6579 (1987): 1046–47. http://dx.doi.org/10.1136/bmj.294.6579.1046.

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13

Savage, W. "Antenatal, perinatal, or postnatal brain damage." BMJ 294, no. 6585 (1987): 1488. http://dx.doi.org/10.1136/bmj.294.6585.1488.

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14

Hagberg, Henrik, A. David Edwards, and Floris Groenendaal. "Perinatal brain damage: The term infant." Neurobiology of Disease 92 (August 2016): 102–12. http://dx.doi.org/10.1016/j.nbd.2015.09.011.

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15

Ohlsson, Arne. "Preterm birth and perinatal brain damage." International Journal of Gynecology & Obstetrics 70 (2000): D8. http://dx.doi.org/10.1016/s0020-7292(00)82509-6.

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16

Degos, Vincent, Géraldine Favrais, Angela M. Kaindl, et al. "Inflammation processes in perinatal brain damage." Journal of Neural Transmission 117, no. 8 (2010): 1009–17. http://dx.doi.org/10.1007/s00702-010-0411-x.

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17

Kjellmer, Ingemar. "Etiology and Pathophysiology of Postasphyxial Brain Damage." International Journal of Technology Assessment in Health Care 7, S1 (1991): 106–9. http://dx.doi.org/10.1017/s0266462300012605.

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In spite of major developments in prenatal supervision, perinatal asphyxia remains an important reason for the development of brain damage (18). Epidemiological investigations suggest that perinatal asphyxia actually represents a factor of increasing frequency as a cause of severe cerebral injury (9).

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18

Bersani, Iliana, Francesca Pluchinotta, Andrea Dotta, et al. "Early predictors of perinatal brain damage: the role of neurobiomarkers." Clinical Chemistry and Laboratory Medicine (CCLM) 58, no. 4 (2020): 471–86. http://dx.doi.org/10.1515/cclm-2019-0725.

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AbstractThe early detection of perinatal brain damage in preterm and term newborns (i.e. intraventricular hemorrhage, periventricular leukomalacia and perinatal asphyxia) still constitute an unsolved issue. To date, despite technological improvement in standard perinatal monitoring procedures, decreasing the incidence of perinatal mortality, the perinatal morbidity pattern has a flat trend. Against this background, the measurement of brain constituents could be particularly useful in the early detection of cases at risk for short-/long-term brain injury. On this scenario, the main European and

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19

Ishchenko, L. S., E. E. Voropaeva, E. L. Kazachkov, et al. "Features of placental damage in patients with perinatal losses during premature birth and acute COVID-19." YAKUT MEDICAL JOURNAL 88, no. 4 (2024): 76–79. https://doi.org/10.25789/ymj.2024.88.18.

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A morphological study of the placenta in patients with perinatal losses in preterm labor and acute COVID-19 of varying severity, manifested in the II and III trimesters of gestation, was performed. It was found that the features of placental damage in patients with perinatal losses are statistically significant predominance of thrombosis, inflammatory changes and manifestations of placentitis SARS-CoV-2 in comparison with placentas of patients without perinatal losses. Keywords: pregnancy, novel coronavirus infection, COVID-19, preterm labor, perinatal mortality, placental damage

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20

Mamatkulova, Shaxzoda Shuxratovna. "PERINATAL LESIONS OF THE CENTRAL NERVOUS SYSTEM." «Zamonaviy dunyoda innovatsion tadqiqotlar: Nazariya va amaliyot» nomli ilmiy, masofaviy, onlayn konferensiya 1, no. 24 (2022): 448–49. https://doi.org/10.5281/zenodo.7198439.

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21

Dekker, G., H. B. Louw, S. Andronikou, et al. "In utero and perinatal hypoxic brain damage." South African Journal of Radiology 10, no. 3 (2006): 15. http://dx.doi.org/10.4102/sajr.v10i3.164.

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22

Edwards, A. D., and H. Mehmet. "Apoptosis in perinatal hypoxic-ischaemic cerebral damage." Neuropathology and Applied Neurobiology 22, no. 6 (1996): 494–98. http://dx.doi.org/10.1111/j.1365-2990.1996.tb01122.x.

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23

Vannucci, Robert C., Robert M. Brucklacher, and Susan J. Vannucci. "Glycolysis and Perinatal Hypoxic-Ischemic Brain Damage." Developmental Neuroscience 27, no. 2-4 (2005): 185–90. http://dx.doi.org/10.1159/000085991.

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24

Jensen, Arne, Yves Garnier, Johannes Middelanis, and Richard Berger. "Perinatal brain damage—from pathophysiology to prevention." European Journal of Obstetrics & Gynecology and Reproductive Biology 110 (September 2003): S70—S79. http://dx.doi.org/10.1016/s0301-2115(03)00175-1.

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25

van der Knaap, M. S., and J. Valk. "Selective brain damage in perinatal hypoxia-ischemia." Clinical Neurology and Neurosurgery 93, no. 4 (1991): 349. http://dx.doi.org/10.1016/0303-8467(91)90108-2.

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26

Nabieva, Shoxista. "CLINICAL MANIFESTATIONS OF CENTRAL NERVOUS SYSTEM DAMAGE IN NEWBORNS, DEPENDING ON THE SEVERITY OF PERINATAL ENCEPHALOPATHY." Medical science of Uzbekistan, no. 3 (June 28, 2025): 59–63. https://doi.org/10.56121/2181-3612-2025-3-59-63.

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120 newborns with moderate and severe perinatal CNS lesions who were in the neonatal pathology department were examined. The study showed a high reliable incidence of perinatal CNS damage in newborns in the studied groups. An analysis of studies of the clinical manifestations of damage to the nervous system of a newborn in perinatal encephalopathy has shown that changes in the state of the central nervous system can further affect the health of the child, and with late diagnosis and severe cases, the disease can occur with a complicated long-term course of the disease perinatal encephalopathy

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27

Khan, Maya A., Maria G. Degtyareva, Natalya A. Mikitchenko, Ekaterina L. Vakhova, Evgeniya A. Troyanovskaya, and Elizaveta U. Gomenyuk. "Physical rehabilitation of children with perinatal lesion of the central nervous system." Russian Journal of Physiotherapy, Balneology and Rehabilitation 22, no. 3 (2023): 199–207. http://dx.doi.org/10.17816/625323.

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The relevance and social significance of the problem of medical rehabilitation of children with perinatal damage to the central nervous system is determined by the high prevalence of this pathology and the possibility of severe disabling diseases in such children. In the complex treatment of such patients, preference is given to non-drug methods. The leading place in the system of rehabilitation measures for children with perinatal central nervous system damage belongs to kinesiotherapy technologies, as the most gentle and pathogenetically justified. To study the results of research cond

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28

Student. "ICONOCLASTS AT WORK." Pediatrics 83, no. 1 (1989): A64. http://dx.doi.org/10.1542/peds.83.1.a64.

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To assess the predictive value for perinatal brain damage of acidosis at birth, alone or in combination with the Apgar score at 5 minutes, a cohort of 982 liveborn infants delivered over the two months was studied prospectively. Metabolic acidosis determined from the umbilical artery at birth [was] a poor predictor of perinatal brain damage.

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29

Student. "ICONOCLASTS AT WORK." Pediatrics 84, no. 2 (1989): A54. http://dx.doi.org/10.1542/peds.84.2.a54a.

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To assess the predictive value for perinatal brain damage of acidosis at birth, alone or in combination with the Apgar score at 5 minutes, a cohort of 982 liveborn infants delivered over the two months was studied prospectively.... Metabolic acidosis determined from the umbilical artery at birth [was] a poor predictor of perinatal brain damage.

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30

Khan, M. A., N. A. Mikitchenko, M. G. Degtyareva, E. A. Troyanovskaya, Z. Kh Shungarova, and I. A. Lomaga. "Neurodevelopmental technologies in the medical rehabilitation of children with the consequences of perinatal damage to the central nervous system." Fizioterapevt (Physiotherapist), no. 4 (August 12, 2024): 59–65. http://dx.doi.org/10.33920/med-14-2404-06.

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Abstract. Modern medicine focuses on the development of effective methods of medical rehabilitation of children with perinatal damage to the central nervous system, which is due to the high risk of the formation of persistent motor disorders and disability of such patients. A promising area of physical rehabilitation of children with the consequences of perinatal CNS damage is the development and scientific substantiation of neurodevelopmental technologies. The purpose. Analyze scientific publications on the use of neurodevelopmental technologies in the medical rehabilitation of children with

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31

Bratanović, Sadmela, Amela Teskeredžić, and Hurma Begić. "DEVELOPMENT OF FUNCTIONAL VISION IN CHILDREN WITH PERINATAL BRAIN DAMAGE." Research in Education and Rehabilitation 4, no. 2 (2021): 109–19. http://dx.doi.org/10.51558/2744-1555.2021.4.2.109.

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The sense of sight plays a very important role in the life of every individual, since we receive most of the information from the environment with the help of sight. Visually impaired children have difficulty receiving information from the world around them. Lack of visual experience can negatively affect their development. Timely examinations and assessments will indicate the occurrence of various neurological disorders in children, if any are present. A very common cause of neurological disorders is perinatal brain damage. Children with perinatal brain damage often have difficulties in visua

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32

Alimović, Sonja, Nikolina Jurić, and Vlatka Mejaški Bošnjak. "Functional vision in children with perinatal brain damage." Journal of Maternal-Fetal & Neonatal Medicine 27, no. 14 (2013): 1491–94. http://dx.doi.org/10.3109/14767058.2013.863863.

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33

Speidel, B. D. "Perinatal Events and Brain Damage in Surviving Children." Archives of Disease in Childhood 64, no. 5 (1989): 767–68. http://dx.doi.org/10.1136/adc.64.5.767-b.

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34

Berger, Richard, Yves Garnier, and Arne Jensen. "Perinatal Brain Damage: Underlying Mechanisms and Neuroprotective Strategies." Journal of the Society for Gynecologic Investigation 9, no. 6 (2002): 319–28. http://dx.doi.org/10.1177/107155760200900601.

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35

Yager, Jerome Y., and Stephen Ashwal. "Animal Models of Perinatal Hypoxic-Ischemic Brain Damage." Pediatric Neurology 40, no. 3 (2009): 156–67. http://dx.doi.org/10.1016/j.pediatrneurol.2008.10.025.

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36

VANNUCCI, ROBERT C., and SUSAN J. VANNUCCI. "A model of Perinatal Hypoxic-Ischemic Brain Damage." Annals of the New York Academy of Sciences 835, no. 1 Frontiers of (1997): 234–49. http://dx.doi.org/10.1111/j.1749-6632.1997.tb48634.x.

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37

Wirrell, Elaine C., Edward A. Armstrong, Lyndon D. Osman, and Jerome Y. Yager. "Prolonged Seizures Exacerbate Perinatal Hypoxic-Ischemic Brain Damage." Pediatric Research 50, no. 4 (2001): 445–54. http://dx.doi.org/10.1203/00006450-200110000-00005.

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38

Berger, R. "Perinatal brain damage: underlying mechanisms and neuroprotective strategies." Journal of the Society for Gynecologic Investigation 9, no. 6 (2002): 319–28. http://dx.doi.org/10.1016/s1071-5576(02)00164-8.

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39

Jaenke, Roger S., and G. M. Angleton. "Perinatal Radiation-Induced Renal Damage in the Beagle." Radiation Research 122, no. 1 (1990): 58. http://dx.doi.org/10.2307/3577583.

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40

Bueter, Wolfgang, Olaf Dammann, and Alan Leviton. "Endoplasmic Reticulum Stress, Inflammation, and Perinatal Brain Damage." Pediatric Research 66, no. 5 (2009): 487–94. http://dx.doi.org/10.1203/pdr.0b013e3181baa083.

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41

Hagberg, H., C. I. Rousset, X. Wang, and C. Mallard. "Mechanisms of perinatal brain damage and protective possibilities." Drug Discovery Today: Disease Mechanisms 3, no. 4 (2006): 397–407. http://dx.doi.org/10.1016/j.ddmec.2006.11.003.

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42

Kumar, Kusum. "Hypoxic-ischemic brain damage in perinatal age group." Indian Journal of Pediatrics 66, no. 4 (1999): 475–82. http://dx.doi.org/10.1007/bf02727151.

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43

Vannucci, Robert C., and Susan J. Vannucci. "Glucose, acidosis, and perinatal hypoxic-ischemic brain damage." Mental Retardation and Developmental Disabilities Research Reviews 3, no. 1 (1997): 69–75. http://dx.doi.org/10.1002/(sici)1098-2779(1997)3:1<69::aid-mrdd9>3.0.co;2-s.

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44

Vannucci, R. C., J. R. Connor, D. T. Mauger, et al. "Rat model of perinatal hypoxic-ischemic brain damage." Journal of Neuroscience Research 55, no. 2 (1999): 158–63. http://dx.doi.org/10.1002/(sici)1097-4547(19990115)55:2<158::aid-jnr3>3.0.co;2-1.

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45

Michetti, Fabrizio, and Diego Gazzolo. "S100B Protein in Biological Fluids: A Tool for Perinatal Medicine." Clinical Chemistry 48, no. 12 (2002): 2097–104. http://dx.doi.org/10.1093/clinchem/48.12.2097.

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Abstract The diagnosis of perinatal insults currently relies on adequate documentation of general medical and obstetric factors and on radiologic and laboratory assessments. The measurement of brain constituents such as S100B protein may offer an alternative and direct indicator of cell damage in the nervous system when clinical and radiologic assessments are still silent and has the additional advantage of providing a quantitative indicator of the extent of brain lesions. S100B protein has been measured by several immunoassays in biological fluids (i.e., cerebrospinal fluid, blood, amniotic f

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46

Van Steenwinckel, Juliette, Anne-Laure Schang, Stéphanie Sigaut, et al. "Brain damage of the preterm infant: new insights into the role of inflammation." Biochemical Society Transactions 42, no. 2 (2014): 557–63. http://dx.doi.org/10.1042/bst20130284.

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Epidemiological studies have shown a strong association between perinatal infection/inflammation and brain damage in preterm infants and/or neurological handicap in survivors. Experimental studies have shown a causal effect of infection/inflammation on perinatal brain damage. Infection including inflammatory factors can disrupt programmes of brain development and, in particular, induce death and/or blockade of oligodendrocyte maturation, leading to myelin defects. Alternatively, in the so-called multiple-hit hypothesis, infection/inflammation can act as predisposing factors, making the brain m

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47

Rey-Funes, Manuel, Ignacio M. Larrayoz, Juan C. Fernández, et al. "Methylene blue prevents retinal damage in an experimental model of ischemic proliferative retinopathy." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 310, no. 11 (2016): R1011—R1019. http://dx.doi.org/10.1152/ajpregu.00266.2015.

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Perinatal asphyxia induces retinal lesions, generating ischemic proliferative retinopathy, which may result in blindness. Previously, we showed that the nitrergic system was involved in the physiopathology of perinatal asphyxia. Here we analyze the application of methylene blue, a well-known soluble guanylate cyclase inhibitor, as a therapeutic strategy to prevent retinopathy. Male rats ( n = 28 per group) were treated in different ways: 1) control group comprised born-to-term animals; 2) methylene blue group comprised animals born from pregnant rats treated with methylene blue (2 mg/kg) 30 an

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48

Cappellini, A. "Encefalopatia ipossico-ischemica." Rivista di Neuroradiologia 16, no. 3 (2003): 345–48. http://dx.doi.org/10.1177/197140090301600304.

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The term hypoxic-ischaemic encephalopathy, often used to cover the whole spectrum of perinatal brain damage, should be confined to neonatal suffering at term presenting specific physiopathological, clinical and radiological features. Most perinatal brain lesions are secondary to perinatal or post-natal hypoxic-ischaemic injury and can be classified on the basis of the predominant morphological characteristics (table 1). The different locations and morphological expression of brain damage in the premature infant and the neonate born at term reflect the different levels of brain maturation (tabl

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49

Vasiljevic, Brankica, Svjetlana Maglajlic-Djukic, Miroslava Gojnic, and Sanja Stankovic. "The role of oxidative stress in perinatal hypoxic-ischemic brain injury." Srpski arhiv za celokupno lekarstvo 140, no. 1-2 (2012): 35–41. http://dx.doi.org/10.2298/sarh1202035v.

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Introduction. The pathogenesis of perinatal hypoxic-ischemic brain damage is highly complex. Objective. The aim of this study was to assess the role of oxidative stress in hypoxic-ischemic brain injury and subsequent abnormal neurological outcome in infants with perinatal hypoxic-ischemic encephalopathy (HIE). We estimated perinatal oxidative brain damage measuring activity of glutathione peroxidase (GPX) in cerebrospinal fluid (CSF) as an indirect biomarker of free radical production during cerebral hypoxia-ischemia in correlation with the level of intracellular enzyme neuron specific enolase

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50

Saidovich, Ergashev Suxrob, Mamurova Mavludaxon Mirxamzayevna, and Turayev Bobir Temirpulotovich. "CLINICAL-NEUROPHYSIOLOGICAL AND MORPHOLOGICAL STUDIES OF CHILDREN WITH CONSEQUENCES OF HYPOXIC." Journal of Medical Genetics and Clinical Biology 2, no. 2 (2025): 117–25. https://doi.org/10.61796/jmgcb.v2i2.1214.

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Objective: The problem of perinatal brain injury of hypoxic-ischemic genesis remains a critical issue, with a focus on its high mortality rates, significant disability, and impact on neuropsychic and somatic development in children. These injuries lead to difficulties in social adaptation, diagnosis, and treatment. Methods: The study aims to explore current therapeutic measures applied to children with perinatal hypoxic-ischemic damage to the central nervous system (CNS), evaluating their effectiveness in addressing these challenges. Various medical and social factors are examined to understan

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