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Journal articles on the topic 'Periodontal diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Hanisch, Marcel, Thomas Hoffmann, Lauren Bohner, Lale Hanisch, Korbinian Benz, Johannes Kleinheinz, and Jochen Jackowski. "Rare Diseases with Periodontal Manifestations." International Journal of Environmental Research and Public Health 16, no. 5 (March 9, 2019): 867. http://dx.doi.org/10.3390/ijerph16050867.

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Background: The object of this paper was to provide an overview of rare diseases (RDs) with periodontal manifestations and allocate them to relevant categories. Methods: In ROMSE, a database for “Rare Diseases with Orofacial Involvement”, all 541 entities were analyzed with respect to manifestations of periodontal relevance. Inclusion criteria were periodontally relevant changes to the oral cavity, in accordance with the 2018 version of the Classification of Periodontal and Peri-Implant Diseases and Conditions. Rare diseases were recorded, using the methodology described, and subsequently compared with the Orphanet Classification of Rare Diseases. Results: A total of 76 RDs with periodontal involvement were recorded and allocated in accordance with the Classification of Periodontal and Peri-Implant Diseases and Conditions. Of the 541 RDs analyzed as having known orofacial manifestations, almost 14 percent indicated a periodontally compromised dentition. Conclusions: Around 14 percent of RDs with an orofacial involvement showed periodontally relevant manifestations, which present not only as a result of gingivitis and periodontitis, but also gingival hyperplasia in connection with an underlying disease. Thus, dentists play an important role in therapy and early diagnoses of underlying diseases based on periodontally relevant manifestations.

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2

Dorri, M. "Periodontal diseases: New classification for periodontal diseases." British Dental Journal 225, no. 8 (October 2018): 686. http://dx.doi.org/10.1038/sj.bdj.2018.941.

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3

Dr.NupurSah, Dr NupurSah, and Dr Hemant Bhutani. "Proteomics and Periodontal Diseases." Paripex - Indian Journal Of Research 2, no. 2 (January 15, 2012): 242–44. http://dx.doi.org/10.15373/22501991/feb2013/86.

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4

Pihlstrom, Bruce L., Bryan S. Michalowicz, and Newell W. Johnson. "Periodontal diseases." Lancet 366, no. 9499 (November 2005): 1809–20. http://dx.doi.org/10.1016/s0140-6736(05)67728-8.

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5

Manstein, Carl H. "Periodontal diseases." Plastic and Reconstructive Surgery 90, no. 4 (October 1992): 733. http://dx.doi.org/10.1097/00006534-199210000-00050.

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6

Nokhbehsaim, Marjan, Anna Damanaki, Andressa Vilas Boas Nogueira, Sigrun Eick, Svenja Memmert, Xiaoyan Zhou, Shanika Nanayakkara, et al. "Regulation of Ghrelin Receptor by Periodontal Bacteria In Vitro and In Vivo." Mediators of Inflammation 2017 (2017): 1–11. http://dx.doi.org/10.1155/2017/4916971.

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Ghrelin plays a major role in obesity-related diseases which have been shown to be associated with periodontitis. This study sought to analyze the expression of the functional receptor for ghrelin (GHS-R1a) in periodontal cells and tissues under microbial conditions in vitro and in vivo. The GHS-R1a expression in human periodontal cells challenged with the periodontopathogen Fusobacterium nucleatum, in gingival biopsies from periodontally healthy and diseased individuals, and from rats with and without ligature-induced periodontitis was analyzed by real-time PCR, immunocytochemistry, and immunofluorescence. F. nucleatum induced an initial upregulation and subsequent downregulation of GHS-R1a in periodontal cells. In rat experimental periodontitis, the GHS-R1a expression at periodontitis sites was increased during the early stage of periodontitis, but significantly reduced afterwards, when compared with healthy sites. In human gingival biopsies, periodontally diseased sites showed a significantly lower GHS-R1a expression than the healthy sites. The expression of the functional ghrelin receptor in periodontal cells and tissues is modulated by periodontal bacteria. Due to the downregulation of the functional ghrelin receptor by long-term exposure to periodontal bacteria, the anti-inflammatory actions of ghrelin may be diminished in chronic periodontal infections, which could lead to an enhanced periodontal inflammation and tissue destruction.

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7

Papapanou, Panos N. "Periodontal Diseases: Epidemiology." Annals of Periodontology 1, no. 1 (November 1996): 1–36. http://dx.doi.org/10.1902/annals.1996.1.1.1.

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8

Armitage, Gary C. "Periodontal Diseases: Diagnosis." Annals of Periodontology 1, no. 1 (November 1996): 37–215. http://dx.doi.org/10.1902/annals.1996.1.1.37.

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9

Offenbacher, Steven. "Periodontal Diseases: Pathogenesis." Annals of Periodontology 1, no. 1 (November 1996): 821–78. http://dx.doi.org/10.1902/annals.1996.1.1.821.

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10

Barrington, Erwin P., and Myron Nevins. "Diagnosing Periodontal Diseases." Journal of the American Dental Association 121, no. 4 (October 1990): 460–64. http://dx.doi.org/10.14219/jada.archive.1990.0210.

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11

Stenchláková, Barbora, and Tomáš Siebert. "Necrotising periodontal diseases." Stomatológ 32, no. 1 (June 15, 2022): 17–21. http://dx.doi.org/10.69658/sto.2022.004.

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12

Santos, Bruna Rafaela Martins dos, Clarissa Favero Demeda, Eutália Elizabeth Novaes Ferreira da Silva, Maria Helena Marques Fonseca de Britto, Kenio Costa Lima, and Maria Celeste Nunes de Melo. "Prevalence of Subgingival Staphylococcus at Periodontally Healthy and Diseased Sites." Brazilian Dental Journal 25, no. 4 (2014): 271–76. http://dx.doi.org/10.1590/0103-6440201302285.

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Staphylococci are considered members of the transient oral microbiota and are seldom isolated from the oral cavity. The aim of this study was to establish the prevalence of subgingival staphylococci in healthy and periodontal disease sites. Sterile endodontic paper points were used to isolate subgingival staphylococci in periodontally healthy and periodontally diseased sites in 30 adult subjects (n=540 sites). Staphylococcus spp were identified by an automated method and confirmed by conventional biochemical tests. All the samples were identified as coagulase-negative staphylococci. The results were analyzed using Mann-Whitney U, chi-square and Fisher's exact test at 5% significance level. A total of 86.7% of the subjects harbored these microorganisms in 11.7% of their periodontal sites. The most frequently isolated species was S. auricularis, which was isolated from 31.4% of the periodontal sites, followed by S. epidermidis, isolated from 21.4% of them. There was no statistically significant difference between the frequencies of these species isolated either from the healthy and the diseased sites (p>0.153). Although staphylococci are present in the subgingival environment and contribute to the pathogenic synergism involved in periodontal diseases, the results suggest that they do not participate directly in the pathogenesis of these diseases.

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13

Jasim Khalaf, Salim, Mahdi Salih Hamad, and Entedhar Rifaat Sarhat. "Salivary Biomarkers in Periodontal Diseases: A Review." Tikrit Journal for Dental Sciences 11, no. 1 (October 19, 2023): 105–8. http://dx.doi.org/10.25130/tjds.11.1.12.

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Saliva represents an important biological material for diagnosis of oral problems. Salivary biomarkers like enzymes, proteins, or oxidative stress are used for early screening of oral diseases and evaluating of disease activity, and also measure the efficacy of therapy, therefore; it is used for determination of periodontal disease activity and prognosis. Salivary biomarkers are potentially important for determining the presence, risk, and progression of periodontal disease, therefore; it is not only reflecting the current status of periodontal diseases but may predict their progression and response to treatment. The prevalence of periodontitis is about 5–15% worldwide. There are some factors that may increase the incidenc e of periodontitis like smoking, obesity, diabetes, and heart diseases. Method of assessing and monitoring periodont al disease must be quick, easy and reliable and should provide important diagnostic information that improves and speeds treatment decisions.

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14

Armitage, Gary C. "Periodontal diagnoses and classification of periodontal diseases." Periodontology 2000 34, no. 1 (February 2004): 9–21. http://dx.doi.org/10.1046/j.0906-6713.2002.003421.x.

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15

Wanderley, H. F., F. M. S. Costa, A. B. G. Quirino, N. E. C. Nobre, F. B. Da Silva, L. V. B. Holanda, D. M. M. De Paula, and M. V. S. Lemos. "Irrigação Subgengival com Antimicrobianos como Adjunto à Terapia Periodontal não-Cirúrgica: uma Revisão da Literatura." Journal of Health Sciences 19, no. 5 (February 23, 2018): 110. http://dx.doi.org/10.17921/2447-8938.2017v19n5p110.

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O tratamento instituído para doenças periodontais baseia-se na eliminação dos patógenos através de abordagem mecânica não cirúrgica (considerado padrão no tratamento das doenças periodontais). Entretanto, o tratamento mecânico pode não eliminar previsivelmente os patógenos existentes na área subgengival. Este trabalho tem como objetivo apresentar antimicrobianos que podem auxiliar no tratamento de bolsas periodontais, complementando assim, a abordagem mecânica. Para tanto revisou-se a literatura nas bases de dados PubMed e Science Direct, utilizando-se os descritores (inglês/português): Antimicrobials, Periodontal Diseases e Chlorhexidine. Foram encontrados 26 artigos entre 2006 e 2016, no qual foram selecionados 11. Como critérios de inclusão foram selecionados artigos que descreviam diferentes substâncias antimicrobianas utilizadas em bolsas após raspagem subgengival. Foram excluídos artigos que apresentassem metodologia incompleta. Muitas soluções antimicrobianas são utilizadas como irrigantes de bolsa periodontal, como: iodo, peróxido de hidrogênio, tetraciclina, metronidazol e clorexidina. A solução de digluconato de clorexidina 0,12% é a mais estudada e utilizada. Entretanto, o iodo tem se mostrado promissor na periodontia, devido suas características farmacológicas e seu uso frequente e comprovado pela medicina. A abordagem mecânica do biofilme periodontal ainda é o método mais utilizado, porém o uso de métodos coadjuvantes a esse tratamento básico pode ser de grande ajuda para atingir áreas de difícil acesso.Palavras-chave: Antimicrobianos. Doenças Periodontais. Clorexidina.

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16

Sauvetre, EJ, MS Farid, and CV Diji. "Cardiovascular Diseases and Periodontal Treatment." Journal of Oral Health and Community Dentistry 2, no. 2 (2008): 25–29. http://dx.doi.org/10.5005/johcd-2-2-25.

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ABSTRACT A Safe and effective periodontal treatment requires knowledge and understanding of the diseases specifically the cardiovascular ones, and the necessary modifications to periodontal therapy accordingly. Considering the high incidence of periodontal diseases in elderly individuals, the periodontist must be prepared to provide periodontal therapeutic support for an increasing number of cardiovascular patients. In this review, common cardiovascular disorders and associated periodontal issues would be discussed briefly.

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17

Herrera, David, Belén Retamal-Valdes, Bettina Alonso, and Magda Feres. "Acute periodontal lesions (periodontal abscesses and necrotizing periodontal diseases) and endo-periodontal lesions." Journal of Clinical Periodontology 45 (June 2018): S78—S94. http://dx.doi.org/10.1111/jcpe.12941.

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18

Herrera, David, Belén Retamal-Valdes, Bettina Alonso, and Magda Feres. "Acute periodontal lesions (periodontal abscesses and necrotizing periodontal diseases) and endo-periodontal lesions." Journal of Periodontology 89 (June 2018): S85—S102. http://dx.doi.org/10.1002/jper.16-0642.

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19

Ван дер Бийль, Петер, and Peter Van der Biyl'. "PERIODONTAL AND CARDIOVASCULAR DISEASES." Actual problems in dentistry 10, no. 6 (December 25, 2014): 4–9. http://dx.doi.org/10.18481/2077-7566-2014-0-6-4-9.

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<p>The relationship between periodontal and cardiovascular diseases is addressed in this article. Both these diseases have an inflammatory basis. Because periodontal disease is a risk factor for developing atherosclerotic vascular disease, diagnosis of the former is important. Particular attention must be paid to patients who have periodontal disease with other risk factors for atherosclerotic vascular disease. Recommendations managing these patients have been made included. </p>

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20

Kouanda, Bakey, Zeeshan Sattar, and Patrick Geraghty. "Periodontal Diseases: Major Exacerbators of Pulmonary Diseases?" Pulmonary Medicine 2021 (November 2, 2021): 1–10. http://dx.doi.org/10.1155/2021/4712406.

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Periodontal diseases are a range of polymicrobial infectious disorders, such as gingivitis and periodontitis, which affect tooth-supporting tissues and are linked to playing a role in the exacerbation of several pulmonary diseases. Pulmonary diseases, such as pneumonia, chronic obstructive pulmonary disease (COPD), asthma, tuberculosis, COVID-19, and bronchiectasis, significantly contribute to poor quality of life and mortality. The association between periodontal disease and pulmonary outcomes is an important topic and requires further attention. Numerous resident microorganisms coexist in the oral cavity and lungs. However, changes in the normal microflora due to oral disease, old age, lifestyle habits, or dental intervention may contribute to altered aspiration of oral periodontopathic bacteria into the lungs and changing inflammatory responses. Equally, periodontal diseases are associated with the longitudinal decline in spirometry lung volume. Several studies suggest a possible beneficial effect of periodontal therapy in improving lung function with a decreased frequency of exacerbations and reduced risk of adverse respiratory events and morbidity. Here, we review the current literature outlining the link between the oral cavity and pulmonary outcomes and focus on the microflora of the oral cavity, environmental and genetic factors, and preexisting conditions that can impact oral and pulmonary outcomes.

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21

Shirmohammadi, Adileh, Masoumeh Faramarzi, Ashkan Salari, Mehrnoosh Sadighi Shamami, Amir Reza Babaloo, and Zohreh Mousavi. "Effect of non-surgical periodontal treatment on serum albumin levels in patients with chronic periodontitis." Journal of Advanced Periodontology & Implant Dentistry 10, no. 1 (October 10, 2018): 18–23. http://dx.doi.org/10.15171/japid.2018.004.

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Background. Albumin is a protein whose serum levels decrease in inflammatory conditions such as periodontal diseases. This study was undertaken to evaluate changes in serum albumin levels in patients with and without periodontal diseases prior and subsequent to non-surgical periodontal treatment and its relationship with clinical parameters of periodontal disease.Methods. Twenty patients diagnosed as having chronic periodontitis and 20 periodontally healthy subjects, referring to Tabriz Faculty of Dentistry, were selected. Serum albumin levels and clinical variables of periodontal disease (probing pocket depth, gingival index, bleeding index, clinical attachment level and plaque index) were determined before treatment and three months subsequent to non-surgical periodontal treatment. Data were subjected to descriptive statistical analyses (mean ± SD). Serum levels of albumin and clinical parameters were compared between the two groups with independent-samples t-test. Paired-samples t-test was applied to compare the variables before and after treatment in the case group. Statistical significance was defined at P<0.05.Results. The mean serum albumin level of chronic periodontitis patients (3.62±0.11 mg/dL) exhibited a significantly lower value compared to subjects who were periodontally healthy (4.17±0.29 mg/dL), with the serum albumin levels increasing significantly three months postoperatively (3.78±0.33 mg/dL), approaching the level in subjects who were periodontally healthy (P<0.05).Conclusion. Decreases and increases in serum albumin levels under the effect of periodontal disease and its treatment indi-cated an inverse relationship between the albumin levels of serum and chronic periodontitis.

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22

Torkzaban, Parviz, Zahra Khalili, and Narges Ziaei. "Smoking and Periodontal Diseases." Avicenna Journal of Dental Research 5, no. 2 (December 25, 2013): 49–55. http://dx.doi.org/10.17795/ajdr-20218.

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23

Zambon, Joseph J. "Periodontal Diseases: Microbial Factors." Annals of Periodontology 1, no. 1 (November 1996): 879–925. http://dx.doi.org/10.1902/annals.1996.1.1.879.

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24

Gutteridge, D. H. "Periodontal diseases and smoking." Medical Journal of Australia 143, no. 6 (September 1985): 241. http://dx.doi.org/10.5694/j.1326-5377.1985.tb122959.x.

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25

Higgins, T. J. "Periodontal diseases and smoking." Medical Journal of Australia 143, no. 6 (September 1985): 241. http://dx.doi.org/10.5694/j.1326-5377.1985.tb122960.x.

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26

Joseph, Shiny. "Probiotics in Periodontal Diseases." IOSR Journal of Dental and Medical Sciences 13, no. 3 (2014): 36–39. http://dx.doi.org/10.9790/0853-13343639.

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27

Trivedi, Shilpa, Nand Lal, and Rameshwari Singhal. "Periodontal diseases and pregnancy." Journal of Orofacial Sciences 7, no. 1 (2015): 67. http://dx.doi.org/10.4103/0975-8844.157408.

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28

Kaneko, Takashi. "Peptidoglycan and periodontal diseases." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 61, no. 2 (June 28, 2019): 73–80. http://dx.doi.org/10.2329/perio.61.73.

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29

Greenstein, Gary. "Periodontal Diseases Are Curable." Journal of Periodontology 73, no. 8 (August 2002): 950–53. http://dx.doi.org/10.1902/jop.2002.73.8.950.

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30

Vieira, Reinaldo Wilson. "Cardiovascular and periodontal diseases." Revista Brasileira de Cirurgia Cardiovascular 29, no. 1 (2014): VII—IX. http://dx.doi.org/10.5935/1678-9741.20140003.

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31

RANNEY, RICHARD R. "Classification of periodontal diseases." Periodontology 2000 2, no. 1 (June 1993): 13–25. http://dx.doi.org/10.1111/j.1600-0757.1993.tb00216.x.

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32

Slots, Jorgen. "Herpesviruses in periodontal diseases." Periodontology 2000 38, no. 1 (June 2005): 33–62. http://dx.doi.org/10.1111/j.1600-0757.2005.00109.x.

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33

Genco, Robert J. "Pharmaceuticals and Periodontal Diseases." Journal of the American Dental Association 125, no. 1 (January 1994): 11S—19S. http://dx.doi.org/10.14219/jada.archive.1994.0256.

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34

Hirsch, R. S., and N. G. Clarke. "Infection and Periodontal Diseases." Clinical Infectious Diseases 11, no. 5 (September 1, 1989): 707–15. http://dx.doi.org/10.1093/clinids/11.5.707.

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35

Wilson, Thomas G. "Periodontal Diseases and Diabetes." Diabetes Educator 15, no. 4 (August 1989): 342–45. http://dx.doi.org/10.1177/014572178901500416.

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36

Migliorati, Cesar A. "Periodontal diseases and cancer." Lancet Oncology 9, no. 6 (June 2008): 510–12. http://dx.doi.org/10.1016/s1470-2045(08)70138-4.

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37

Dentino, Andrew R., Moawia M. Kassab, and Erica J. Renner. "Prevention of Periodontal Diseases." Dental Clinics of North America 49, no. 3 (July 2005): 573–94. http://dx.doi.org/10.1016/j.cden.2005.03.005.

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38

Goldman, Robert S., and Leslie Z. Taynor. "Odontologic and periodontal diseases." Clinics in Dermatology 5, no. 2 (April 1987): 59–65. http://dx.doi.org/10.1016/0738-081x(87)90008-3.

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39

Chow, Jack P. "PERIODONTAL AND CARDIOVASCULAR DISEASES." Journal of the American Dental Association 129, no. 4 (April 1998): 406. http://dx.doi.org/10.14219/jada.archive.1998.0221.

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40

Ranney, R. R. "Diagnosis of Periodontal Diseases." Advances in Dental Research 5, no. 1 (December 1991): 21–36. http://dx.doi.org/10.1177/08959374910050010201.

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This paper reviews current (Fall, 1990) information related to the diagnosis of periodontal diseases. As background, principles of diagnostic decision-making and conceptual shifts during the 1970's and 1980's are reviewed in brief. "Diseases" that appeared in many classification schemes for periodontal diseases in the early 1970's—for example, "periodontosis" and "occlusal trauma"-do not appear in most current classifications. A recent (1989a) classification recommended by the American Academy of Periodontology holds that "periodontitis" includes several different diseases. There is, indeed, evidence for several different forms of periodontitis, but the AAP's classification does not conform to the principles of diagnostic decision-making because of the significant overlap between and heterogeneities within its suggested "diseases". An alternative classification is suggested, based on a concept that the periodontal diseases are mixed infections whose outcome is modified by relative effectiveness of host response. This view suggests that the most usual forms, gingivitis and adult periodontitis, normally occur in persons with essentially normal defense systems. Variation in extent or severity of disease can be understood as a function of the local infection in hosts with various degrees of compromised resistance to the infection. Early-onset periodontitis (EOP) cases could be accounted for by those where host response is abnormal to some significant degree. The greater the abnormality, the greater the extent and severity of disease might be. Localized EOP cases would be those where a relatively effective specific response intervenes to ameliorate progress of disease after the initially rapid progression. Other issues are detection of disease activity and assessment of risk for disease progression. Non-cultural bacteriological tests are available, but have not yet been shown to detect or predict activity or risk. One difficulty in reaching such proof for those or other tests has been the lack of an appropriate "gold standard" for disease activity or progression. This is being remedied by development of improved automated probes and imaging technologies. Considerable effort is being devoted to determining whether factors in gingival crevicular fluid may have diagnostic utility. More evidence is needed before clinical utility is known, but several enzymes and cytokines have potential for aiding diagnostic decisions.

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41

Baelum, Vibeke, and Flemming Scheutz. "Periodontal diseases in Africa." Periodontology 2000 29, no. 1 (April 2002): 79–103. http://dx.doi.org/10.1034/j.1600-0757.2002.290105.x.

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42

Sheiham, Aubrey, and Gopalakrishnan S. Netuveli. "Periodontal diseases in Europe." Periodontology 2000 29, no. 1 (April 2002): 104–21. http://dx.doi.org/10.1034/j.1600-0757.2002.290106.x.

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43

Victor, DJ, MaryAnie Paul, and DelonTeh Chai Liu. "Biomarkers of periodontal diseases." SRM Journal of Research in Dental Sciences 1, no. 3 (2010): 266. http://dx.doi.org/10.4103/0976-433x.121703.

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44

Wang, Hom-Lay, and Jason Cooke. "Periodontal Regeneration Techniques for Treatment of Periodontal Diseases." Dental Clinics of North America 49, no. 3 (July 2005): 637–59. http://dx.doi.org/10.1016/j.cden.2005.03.004.

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45

Dankevych-Kharchyshyn, Iryna S., Olena M. Vynogradova, Natalia V. Malko, Roman M. Gnid, Andriana P. Skalat, Lidiya Y. Minko, Oleg I. Mrochko, Yurij L. Bandrivsky, and Orysia O. Bandrivska. "PERIODONTAL DISEASES AND ATHEROSCLEROSIS (LITERATURE REVIEW)." Wiadomości Lekarskie 72, no. 3 (2019): 462–65. http://dx.doi.org/10.36740/wlek201903127.

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Introduction: The relationship between periodontal diseases and atherosclerosis is addressed in this article. Both these diseases have an inflammatory basis. Because periodontal disease is a risk factor for developing atherosclerotic vascular disease, diagnosis of the former is important. Particular attention must be paid to patients who have periodontal disease with other risk factors for atherosclerotic vascular disease. Recommendations managing these patients have been made included. The aim: The paper is aimed at familiarization of broad medical public with the presence of the relationship between diseases of periodontal tissues and atherosclerosis. Materials and methods: A thorough comprehensive analysis and generalization of scientific achievements elucidated in the fundamental and periodical publications, relating to diseases of the periodontal tissues and atherosclerosis, has been carried out. Review: The article consists of many researchers regarding the prevalence and intensity of periodontal tissue diseases in people of all ages. Problems associated with the state of periodontal tissues in people under study as dentists and general practitioners. Proven role in the pathogenesis of inflammatory diseases of the periodontal tissues in people with atherosclerosis. In the modern concept of the etiology and pathogenesis of periodontal diseases in people is extremely important role for the immune system and resistance to periodontal bacterial invasion. Analyzed common changes important for pathogenesis of periodontal tissue diseases and atherosclerosis. Conclusions: Consequently, recent studies have shown a clear, directly proportional relationship between periodontal tissue diseases and atherosclerosis, but mechanisms for their development and interaction are not fully disclosed.

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46

Al-Ghutaimel, Hayat, Hisham Riba, Salem Al-Kahtani, and Saad Al-Duhaimi. "Common Periodontal Diseases of Children and Adolescents." International Journal of Dentistry 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/850674.

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Background.Since 2000, studies, experiments, and clinical observations revealed high prevalence of periodontal diseases among children and adolescents. Therefore, this paper was designed to provide an update for dental practitioners on epidemiology, microbiology, pathology, prevention, diagnosis, and treatment of periodontal diseases in children and adolescents.Methods.This paper reviews the current literature concerning periodontal diseases in pediatric dentistry. It includes MEDLINE database search using key terms: “periodontal diseases in children,” “Periodontal diseasesin adolescents,” “periodontal diseases risk factors,” “microbiology of periodontal diseases,” “classification of periodontal diseases,” “epidemiology of periodontal diseases,” and “treatment of periodontal diseases.” Articles were evaluated by title and/or abstract and relevance to pediatric dentistry. Sixty-five citations were selected by this method and by the references within the chosen articles. A review of the comprehensive textbooks on pediatric dentistry and periodontology was done. Some recommendations were based on the opinions of experienced researchers and clinicians, when data were inconclusive.

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47

Jagelavičienė, Eglė, Evaldas Padervinskis, and Erika Valienė. "THE DIAGNOSTIC RELATIONSHIP BETWEEN HELICOBACTER PYLORI, PERIODONTAL DISEASES AND GASTROESOPHAGEAL REFLUX." Health Sciences 33, no. 1 (January 1, 2023): 56–63. http://dx.doi.org/10.35988/sm-hs.2023.013.

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Background and objectives. The development of the digestive tract and periodontal diseases is influenced by several general risk factors. One of them is the tooth loss, due to periodontal diseases, which results in poor mastication of food and latter gastroesophageal reflux. The second major risk factor is bacterial microflora. Current research is relevant due to the controversial nature of the opinions on the general bacterial causes of the aforementioned diseases and their correlations. The aim of the study is to confirm the hypothesis that the development of periodontal and gastroesophageal reflux diseases is influenced by the simultaneous colony deve-lopment of Helicobacter pylori in periodontal pockets and the digestive tract. Material and Methods. A total of 70 respondents were studied. Social and demographic indicators were analyzed based on the data of a questionnaire-based survey. The periodontium was studied clinically; community periodontal index of treatment needs was determined, a rapid urease test (Pronto Dry) was performed to detect bacteria. The gastroesophageal reflux disease was analyzed using an endoscope and via survey. The respondents were distributed into three test and three age groups. IBM SPSS Statistics 22 software was used for statistical analysis. Results. A statistically significant correlation was found between the community periodontal index of treatment needs and Reflux symptom index (p=0.004), and urease test (p<0.001) and oral hygiene (p<0.05). No statistically significant correlations were determined between indices (RSI; RFS), age, gender, urease test (p>0.05). Conclusions. The colony formation of Helicobacter pylori in periodontium has no impact on the gastroesophageal reflux disease but is significantly related to periodontal diseases. Thus, the oral cavity is the primary reservoir of internal H. pylori colonization.

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Weidlich, Patrícia, Renata Cimões, Claudio Mendes Pannuti, and Rui Vicente Oppermann. "Association between periodontal diseases and systemic diseases." Brazilian Oral Research 22, suppl 1 (August 2008): 32–43. http://dx.doi.org/10.1590/s1806-83242008000500006.

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Storto Soares, Larissa Marques, Ana Emilia Farias Pontes, Fernanda de Oliveira Bello Corrêa, and Cleverton Corrêa Rabelo. "Influence of Psychological Stress on Periodontal Diseases." American Research Journal of Dentistry 2, no. 1 (December 24, 2020): 1–5. http://dx.doi.org/10.21694/2578-1448.20001.

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Introduction: The association between periodontal disease and stress has been questioned for a almost a century, however, it still represents an unexplored field of research with several orphaned questions of conclusive answers. Objective: To evaluate the relationship between periodontal disease and stress. Methodology: Searches were performed with descriptors related to periodontal diseases and psychological factors in the following databases: Pubmed, Embase, Lilacs. Were identified and included studies that deal with the relationship between stress and periodontal disease and /or that emphasize the role of this psychosocial factor in the progression of periodontal disease. Conclusion: Most studies have shown a positive relationship between periodontal disease and stress, however, further research needs to be developed to confirm stress as a risk factor for periodontal disease

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Hartanti, Hartanti. "DIASTEMA CORRECTION OF PERIODONTAL DISEASES FOLLOWING PERIODONTAL THERAPY ALONE." B-Dent: Jurnal Kedokteran Gigi Universitas Baiturrahmah 8, no. 3 (December 29, 2021): 242–48. http://dx.doi.org/10.33854/jbd.v8i3.884.

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