Relevant bibliographies by topics / Peripheral blood inflammatory cells (PBICs)

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Peripheral blood inflammatory cells (PBICs)'

Author: Grafiati
Published: 5 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Peripheral blood inflammatory cells (PBICs)"

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Bories, Gael, Robert Caiazzo, Bruno Derudas, et al. "Impaired alternative macrophage differentiation of peripheral blood mononuclear cells from obese subjects." Diabetes and Vascular Disease Research 9, no. 3 (2011): 189–95. http://dx.doi.org/10.1177/1479164111430242.

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Visceral obesity is a chronic, low-grade inflammatory disease that predisposes people to the metabolic syndrome, type 2 diabetes and its cardiovascular complications. Adipose tissue is not a passive storehouse for fat, but an endocrine organ synthesizing and releasing a variety of bioactive molecules, some of which are produced by infiltrated immune-inflammatory cells including macrophages. Two different subpopulations of macrophages have been identified in adipose tissue: pro-inflammatory ‘classical’ M1 and anti-inflammatory ‘alternative’ M2 macrophages, and their ratio is suggested to influe
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Shulman, Iliya, Sergei Ogurcov, Alexander Kostennikov, et al. "Application of Autologous Peripheral Blood Mononuclear Cells into the Area of Spinal Cord Injury in a Subacute Period: A Feasibility Study in Pigs." Biology 10, no. 2 (2021): 87. http://dx.doi.org/10.3390/biology10020087.

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Peripheral blood presents an available source of cells for both fundamental research and clinical use. In our study, we have evaluated the therapeutic potential of peripheral blood mononuclear cells (PBMCs) excluding the preliminary sorting or mobilization of peripheral blood stem cells. We have evaluated the regenerative potential of PBMCs embedded into a fibrin matrix (FM) in a model of pig spinal cord injury. The distribution of transplanted PBMCs in the injured spinal cord was evaluated; PBMCs were shown to penetrate into the deep layers of the spinal cord and concentrate mainly in the gre
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Song, Xinqiang, Yu Zhang, Lijun Zhao, et al. "Analyzation of the Peripheral Blood Mononuclear Cells Atlas and Cell Communication of Rheumatoid Arthritis Patients Based on Single-Cell RNA-Seq." Journal of Immunology Research 2023 (August 12, 2023): 1–20. http://dx.doi.org/10.1155/2023/6300633.

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Background. Rheumatoid arthritis (RA) is a common chronic inflammatory autoimmune disease with a multifactorial etiology. Peripheral blood is the main channel of the immune system, and peripheral blood mononuclear cells (PBMCs) are the immune cells that initiate the autoimmune inflammatory process. However, there are few reports on the mechanisms of peripheral blood immunity in RA. Methods. ScRNA-seq was performed on four RA samples and integrated with single-cell transcriptome data from four healthy control samples downloaded from publicly available databases for analysis. Results. A total of
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Kaur, Kawaljit, Shahram Vaziri, Marcela Romero-Reyes, Avina Paranjpe, and Anahid Jewett. "Phenotypic and Functional Alterations of Immune Effectors in Periodontitis; A Multifactorial and Complex Oral Disease." Journal of Clinical Medicine 10, no. 4 (2021): 875. http://dx.doi.org/10.3390/jcm10040875.

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Survival and function of immune subsets in the oral blood, peripheral blood and gingival tissues of patients with periodontal disease and healthy controls were assessed. NK and CD8 + T cells within the oral blood mononuclear cells (OBMCs) expressed significantly higher levels of CD69 in patients with periodontal disease compared to those from healthy controls. Similarly, TNF-α release was higher from oral blood of patients with periodontal disease when compared to healthy controls. Increased activation induced cell death of peripheral blood mononuclear cells (PBMCs) but not OBMCs from patients
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Yadav, Brijesh, Narayan Prasad, Ravi Shanker Kushwaha, et al. "Urinary Mitochondrial DNA Induces an Inflammatory Response in Peripheral Blood Mononuclear Cells." Indian Journal of Transplantation 18, no. 2 (2024): 132–37. http://dx.doi.org/10.4103/ijot.ijot_94_23.

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Background: Mitochondria are semiautonomous cell organelles having its own nucleic acid. Mitochondrial DNA (Mt-DNA) remain in hypomethylated (CpG) state and impose an immunogenic response by binding to the toll-like receptor (TLR-9) through the NF-kB pathway. Innate immune cells recognize the hypomethylated pattern of mt-DNA and quickly trigger the innate immune response. The immunomodulatory effects of urinary mt-DNA derived from renal transplant recipients with COVID-19-associated acute kidney injury (AKI) have not been studied. Materials and Methods: Healthy donor peripheral blood mononucle
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Yin, Xuejiao, Yi Liu, Hongyan Tong та Liangshun You. "Pro-Inflammatory Monocytes As Key Effector Cells in Peripheral Circulatory Inflammatory Storms Driven By IFN-γ and IL-6 Pathways in Imcd". Blood 144, Supplement 1 (2024): 4685. https://doi.org/10.1182/blood-2024-207774.

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Objective: Dysfunctional cytokine-driven inflammatory storms in patients with idiopathic multicentric Castleman disease (iMCD) are a recurring issue affecting both symptoms and mortality, yet a thorough understanding of the involved immune cells remains incomplete. A comprehensive and detailed peripheral immune landscape is crucial for effectively treating iMCD. Design: We employed single-cell RNA sequencing of 150936 cells from a cohort of 19 individuals, including 15 iMCD patients with mild or severe disease, as well as 4 healthy controls. For validation, we also enrolled a second cohort of
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Krishnamoorthy, Rajapandiyan, Abdulraheem R. Adisa, Vaiyapuri Subbarayan Periasamy, Jegan Athinarayanan, Subash-Babu Pandurangan, and Ali A. Alshatwi. "Colonic Bacteria-Transformed Catechin Metabolite Response to Cytokine Production by Human Peripheral Blood Mononuclear Cells." Biomolecules 9, no. 12 (2019): 830. http://dx.doi.org/10.3390/biom9120830.

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Human gut microbes are a profitable tool for the modification of food compounds into biologically active metabolites. The biological properties of catechins have been extensively investigated. However, the bioavailability of catechin in human blood plasma is very low. This study aimed to determine the biotransformed catechin metabolites and their bioactive potentials for modulating the immune response of human peripheral blood mononuclear cells (PBMCs). Biotransformation of catechin was carried out using in-vitro gut microbial biotransformation method, the transformed metabolites were identifi
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Prasetio, Ardi, Luthfiana Mifta Syafitri, Adam Prabowo, Iffan Alif, and Aldan Nurichsan. "Priming of Mesenchymal Stem Cells for Enhanced Interleukin-10 Secretion via Conditioned Medium from Lipopolysaccharide-Activated Peripheral Blood Mononuclear Cells." International Journal of Cell and Biomedical Science 2, no. 5 (2024): 167–74. https://doi.org/10.59278/cbs.v2i5.39.

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Background: Mesenchymal stem cells (MSCs) are known for their immunomodulatory properties, particularly their ability to secrete anti-inflammatory cytokines such as interleukin-10 (IL-10). Enhancing the secretion of IL-10 by MSCs could have significant therapeutic potential for treating inflammatory diseases. Objective: This study aimed to prime the secretion of IL-10 by MSCs through the use of conditioned medium (CM) derived from lipopolysaccharide (LPS)-induced peripheral blood mononuclear cells (PBMCs). Methods: MSCs were isolated from Wharton’s Jelly and characterized using flow cytometry
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Szafran, Brittany, Jung Lee, Abdolsamad Borazjani, et al. "Characterization of Endocannabinoid-Metabolizing Enzymes in Human Peripheral Blood Mononuclear Cells under Inflammatory Conditions." Molecules 23, no. 12 (2018): 3167. http://dx.doi.org/10.3390/molecules23123167.

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Endocannabinoid-metabolizing enzymes are downregulated in response to lipopolysaccharide (LPS)-induced inflammation in mice, which may serve as a negative feedback mechanism to increase endocannabinoid levels and reduce inflammation. Increased plasma levels of the pro-inflammatory cytokine interleukin-6 (IL-6) and decreased fatty acid amide hydrolase (FAAH) activity in peripheral lymphocytes from individuals diagnosed with Huntington’s disease (HD) suggests that a similar negative feedback system between inflammation and the endocannabinoid system operates in humans. We investigated whether Cp
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Connolly, Peter H., Vincent J. Caiozzo, Frank Zaldivar, et al. "Effects of exercise on gene expression in human peripheral blood mononuclear cells." Journal of Applied Physiology 97, no. 4 (2004): 1461–69. http://dx.doi.org/10.1152/japplphysiol.00316.2004.

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Exercise leads to increases in circulating levels of peripheral blood mononuclear cells (PBMCs) and to a simultaneous, seemingly paradoxical increase in both pro- and anti-inflammatory mediators. Whether this is paralleled by changes in gene expression within the circulating population of PBMCs is not fully understood. Fifteen healthy men (18–30 yr old) performed 30 min of constant work rate cycle ergometry (∼80% peak O2 uptake). Blood samples were obtained preexercise (Pre), end-exercise (End-Ex), and 60 min into recovery (Recovery), and gene expression was measured using microarray analysis
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Dissertations / Theses on the topic "Peripheral blood inflammatory cells (PBICs)"

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Toschka, Robert. "Identification of human peripheral blood monocyte derived pro-inflammatory dendritic cells." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2014. http://dx.doi.org/10.18452/17040.

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Dendritische Zellen (DZ) sind essentiell für die Aktivierung von Immunantworten. Drei Flt3-abhängige DZ Populationen aus dem Blut bestehend aus konventionellen (kDZ) BDCA1+ DZs und BDCA3+ DZs und plasmazytoide DZs wurden bereits beschrieben. Hier wurden zum ersten Mal sich aus Monozyten entwickelnde DZ (moDZ), genauer BDCA1+CD14+ pro-inflammatorische DZ (pro-iDZ) aus periphärem Blut unter homöostatischen Bedingungen identifiziert. Isolierte pro-iDZ sekretierten spontan große Mengen an pro-inflammatorischen Zytokinen, die kDZ reifen ließen und T Zell Proliferation unterstützten. Sie waren BDCA1
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Toschka, Robert [Verfasser], Andreas [Akademischer Betreuer] Radbruch, Alexander [Akademischer Betreuer] Scheffold, and Achim [Akademischer Betreuer] Leutz. "Identification of human peripheral blood monocyte derived pro-inflammatory dendritic cells / Robert Toschka. Gutachter: Andreas Radbruch ; Alexander Scheffold ; Achim Leutz." Berlin : Humboldt Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2014. http://d-nb.info/1059801116/34.

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Chasty, Richard. "Analysis of the differential responsiveness of CD34 enriched mononuclear cells from normal bone marrow, and chronic granulocytic leukaemia peripheral blood, to the myeloinhibitory molecule macrophage inflammatory protein-lα (LD78)." Thesis, King's College London (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265852.

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Frémond, Marie-Louise. "Clinical and molecular characterisation of the type I interferonopathies and approaches to therapy Efficacy of the Janus kinase 1/2 inhibitor ruxolitinib in the treatment of vasculopathy associated with TMEM173-activating mutations in three children Blockade of TANK-binding kinase 1/IKKε mutant stimulator of interferon genes (STING)-mediated inflammatory responses in human peripheral blood mononuclear cells". Thesis, Sorbonne Paris Cité, 2018. http://www.theses.fr/2018USPCB098.

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Le concept d'interféronopathie de type I émerge en 2011 et fait référence à un ensemble de pathologies Mendéliennes caractérisées par une hyperactivation des interférons (IFN) de type I. Tous les gènes associés au syndrome d'Aicardi-Goutières (SAG), la première interféronopathie de type I décrite, sont impliqués dans la détection ou le métabolisme des acides nucléiques. Les autres protéines mutées associées aux interféronopathies de type I modifient toutes la voie de signalisation des acides nucléiques, de manière directe, indirecte ou encore non définie. Les IFN de type I se fixent à un récep
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Sturm, Andreas. "Modulation intestinaler Wundheilungsvorgänge und Erhaltung der mukosalen Immunhomöostase." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2004. http://dx.doi.org/10.18452/13916.

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Die intestinale Mukosa bildet eine biologisch wichtige Barriere zwischen dem Organismus und den schädigenden Faktoren im intestinalen Lumen. Diese komplexe Aufgabe wird durch eine hochdifferenzierte intestinale Mukosa bewältigt, die eine strukturelle sowie funktionelle intestinale Barriere bildet. Das Ziel der vorliegenden Arbeiten war, ausgewählte Aspekte der Regulations- und Reparaturmechanismen der intestinalen mukosalen Barriere weitergehend zu charakterisieren. Unsere Untersuchungen zeigen, dass das Phospholipid Lysophosphatidsäure (LPA) die intestinale epitheliale Zellmigration stimulier
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Books on the topic "Peripheral blood inflammatory cells (PBICs)"

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Albert, Tyler J., and Erik R. Swenson. The blood cells and blood count. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0265.

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Blood is a dynamic fluid consisting of cellular and plasma components undergoing constant regeneration and recycling. Like most physiological systems, the concentrations of these components are tightly regulated within narrow limits under normal conditions. In the critically-ill population, however, haematological abnormalities frequently occur and are largely due to non-haematological single- or multiple-organ pathology. Haematopoiesis originates from the pluripotent stem cell, which undergoes replication, proliferation, and differentiation, giving rise to cells of the erythroid, myeloid, and
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Breban, Maxime, and Hill Gaston. Immune mechanisms: adaptive immunity. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0008.

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The role of adaptive immunity (i.e. the involvement of B and T lymphocytes) in the pathogenesis of axial spondyloarthritis has been investigated in both human disease and relevant animal models. Studies of B cell responses have not generally implicated an autoantibody in the disease, but there are abnormalities of antibody responses, particularly increased titres of antibodies to various gut bacteria. T cells are critical to the disease in animal models other than those where overexpression of a cytokine is engineered, suggesting that they are the drivers of the inflammatory response. There is
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Guzik, Tomasz J., and Rhian M. Touyz. Vascular pathophysiology of hypertension. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0019.

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Hypertension is a multifactorial disease, in which vascular dysfunction plays a prominent role. It occurs in over 30% of adults worldwide and an additional 30% are at high risk of developing the disease. Vascular pathology is both a cause of the disease and a key manifestation of hypertension-associated target-organ damage. It leads to clinical symptoms and is a key risk factor for cardiovascular disease. All layers of the vascular wall and the endothelium are involved in the pathogenesis of hypertension. Pathogenetic mechanisms, whereby vascular damage contributes to hypertension, are linked
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Book chapters on the topic "Peripheral blood inflammatory cells (PBICs)"

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Homayouni, Vida, Fariba Dehghan, and Roya Sherkat. "The Role of NK Cells in Recurrent Miscarriage (Abortion)." In Natural Killer Cells - Lessons and Challenges [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.109572.

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Recurrent miscarriage is an early pregnancy complication that affects about 1–3% of couples. There are specific characteristics of natural killer (NK) cells associated with miscarriage. In patients with recurrent miscarriage, a lack of inhibition of decidua natural killer cells can be observed, which leads to a more activated state and presentation of NK cell dim that is characterized by higher levels of pro-inflammatory cytokines and cytotoxicity effect. In peripheral blood, a dysfunctional cytokine production by natural killer cells has been also reported, with an increase of interferon-γ le
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Giannini, Caterina, and Bernd W. Scheithauer. "Tumors of the Peripheral Nervous System (Including the Craniospinal Nerve Roots)." In Principles And Practice Of Neuropathology. Oxford University PressNew York, NY, 2003. http://dx.doi.org/10.1093/oso/9780195125894.003.0019.

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Abstract The pathologic spectrum of tumors of the peripheral nervous system (PNS) is broad, despite their derivation from a rather limited repertoire of nerve sheath cells. Putative precursor cells subject to neoplastic transformation reside in all three nerve compartments (endo-, peri-, and epineurium). These include: (1) Schwann cells, which ensheath myelinated and unmyelinated axons, (2) perineurial cells that comprise the perineurium and are an integral part of the blood–nerve barrier, and (3) fibroblasts, endothelial cells, and pericytes, as well as adipose cells. Tumors of peripheral ner
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A.G. Lokossou, Gatien, and Maximin Senou. "Role of Cytotrophoblast Cells and Placenta-Derived Exosomes in Regulatory B Cell Differentiation and Function during Pregnancy." In Immunosuppression and Immunomodulation [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.108335.

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Pregnancy is a particular physiologic stage during which immune regulation is essential. A successful placentation and subsequent fetal development depend on the delicate balance between moderate pro-inflammatory response and immune tolerance. Findings have pointed out a crucial role for regulatory B cells (Bregs) in establishing an immunomodulatory (IM) environment relevant to pregnancy. In a steady state, Bregs represent 10% of B cells in peripheral blood, a proportion that increases during pregnancy, with the highest rate being observed in post-partum. In the context of pregnancy, Bregs see
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Edeer Karaca, Neslihan. "Neutropenia in Primary Immunodeficiency Diseases." In Innate Immunity in Health and Disease. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97297.

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Phagocytes including neutrophil granulocytes and macrophages are important cells of the innate immune system whose primary function is to ingest and destroy microorganisms. Neutrophils help their host fight infections by phagocytosis, degranulation, and neutrophil extracellular traps. Neutrophils are the most common type of circulating white blood cells and the principal cell type in acute inflammatory reactions. A total absence of neutrophils or a significant decrease in their number leads to severe immunodeficiency that renders patients vulnerable to recurrent infections by Staphylococcus au
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Strauchen, James A. "Noninfectious Granulomas." In Diagnostic Histopathology of the Bone Marrow. Oxford University PressNew York, NY, 1996. http://dx.doi.org/10.1093/oso/9780195097566.003.0024.

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Abstract Several types of noninfectious granulomas may be found in the bone marrow biopsy. These include lipogranulomas, sarcoidosis, and reactive (sarcoid-like) granulomas in association with systemic disorders. Lipogranulomas are focal granulomatous reactions to lipid. Lipogranulomas are the most common type of granulomatous reaction occurring in the bone marrow and may be found in up to 9% of bone marrow specimens (Rywlin and Ortega, 1972). Lipogranulomas are a reaction to lipid, the origin of which is not established; exogenous mineral oil has been suggested to be the cause of similar lipo
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Mesquita, Carolina, Bruna Lopes, Patrícia Sousa, et al. "Application of Cell-Based Therapies in Veterinary Dermatology." In Wound Healing - Recent Advances and Future Opportunities [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.111553.

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Stem cells have been extensively studied in the field of veterinary medicine due to their unique characteristics. The last are undifferentiated cells with self-renewal, anti-inflammatory, and immunomodulatory capacity. Mesenchymal stem cells (MSCs) are widely used due to its simple isolation and expansion, being collected from different sources such as adipose tissue, bone marrow, peripheral blood, and umbilical cord. For that reason, MSCs have been studied and used as innovative therapies in the treatment of several diseases, such as tendinitis, bone regeneration, osteoarthritis, neuromuscula
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J. Cordato, Dennis, Wissam Soubra, Sameer Saleem, and Kaneez Fatima Shad. "Are ABO Gene Alleles Responsible for Cardiovascular Diseases and Venous Thromboembolism and Do They Play a Role in COVID?" In Blood Groups - More Than Inheritance of Antigenic Substances [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.100479.

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Cardiovascular diseases (CVD) including coronary heart disease and stroke are leading causes of death and disability globally. Studies of the association between ABO blood groups and CVD have consistently demonstrated an increased risk of coronary heart disease, myocardial infarction, cerebral ischaemic stroke, peripheral arterial disease and venous thromboembolism (VTE) including deep vein thrombosis and pulmonary thromboembolism in patients who possess a non-O blood group type. The most likely mechanism is thought to be the increase in von Willebrand Factor (vWF) and factor VIII levels seen
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Jänig, Wilfrid. "The Autonomic Nervous System and its Coordination by the Brain." In Handbook of Affective Sciences. Oxford University PressNew York, NY, 2002. http://dx.doi.org/10.1093/oso/9780195126013.003.0009.

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Abstract The internal milieu of the body is controlled to keep the component cells, tissues, and organs (including the brain and skeletal muscles) maintained in an optimal environment for their function. This enables the organism to adjust its performance to the varying internal and external demands placed on the organism. In the short term the mechanisms involved include the control of: constancy of the fluid matrix of the body (fluid volume regulation, osmoregulation) gas exchange with the environment (regulation of airway resistance and the pulmonary circulation) ingestion and digestion of
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Conference papers on the topic "Peripheral blood inflammatory cells (PBICs)"

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Al-Haidose, Amal. "Effect of Omega-3 Polyunsaturated Fatty Acids on Inflammatory Biomarkers in Chronic Obstructive Pulmonary Disease." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0144.

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Chronic obstructive pulmonary disease (COPD) is a chronic progressive inflammatory disease characterised by airflow limitation. Several pro-inflammatory markers are thought to be involved in the pathogenesis of COPD. Cigarette smoking is a major risk factor for COPD, and diet may be a modifiable risk factor for its progression & management. Dietary supplementation with omega3 polyunsaturated fatty acids (omega-3 PUFAs) may be effective therapeutically in patient COPD. Aim: To determine the plasma basal level of inflammatory biomarkers in the study population, to determine the inflammatory
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Tannheimer, Stacey L., Aaron C. Haran, Jyoti Velayudhan, E. A. Sorensen, Cliff D. Wright, and Michael Salmon. "Complementary Anti-Inflammatory Interactions Of Roflumilast And Salmeterol In Human Primary Peripheral Blood Mononuclear Cells." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5399.

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Borges, Isabella Sabião, João Victor Aguiar Moreira, Eustaquio Costa Damasceno Junior, et al. "Chronic inflammatory demyelinating polyradiculoneuropathy induced by paclitaxel." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.413.

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Background: Peripheral neuropathies in cancer are most often due to neurotoxic chemotherapeutic agents. Approximately 30% of patients receiving neurotoxic chemotherapy (CTX) will suffer from chemotherapy-induced peripheral neuropathy (CIPN). Paclitaxel is an extremely effective chemotherapeutic agent for the treatment of breast, ovarian, and lung cancer. However, paclitaxel-induced peripheral neuropathy occurs in 59-87% of patients who receive this drug. Paclitaxel is an anti-tubulin drug that causes microtubule stabilization, resulting in distal axonal degeneration, secondary demyelination an
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Lea, Simon, Jian Li, Kieran Littolff, Kajaluckcika Vijayacumaran, and Dave Singh. "Anti-inflammatory effects of PDE3/4 inhibitor ensifentrine in peripheral blood mononuclear cells from COPD patients." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.oa2775.

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Alrhmoun, S., R. Y. Perik-Zavodskii, O. I. Perik-Zavodskaia, et al. "INVESTIGATING ACTIVATION THRESHOLDS OF PERIPHERAL BLOOD MONONUCLEAR CELLS TO TNF BASED ON ITS SURFACE RECEPTOR EXPRESSION." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-11.

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Tumor necrosis factor alpha (TNF-α) is a key pro-inflammatory cytokine in rheumatoid arthritis (RA) pathogenesis, with its overexpression driving systemic effects characteristic of RA [1, 2]. Targeting TNF-α with biologic therapies has revolutionized RA management [3, 4], highlighting its role in disease progression and severity. Our study revealed that monocytes from patients with RA exhibit lower activation thresholds for TNF-α compared to healthy donors.
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Zhang, Yuan, Jonathan Aldridge, Anna-Carin Lundell, Anna Rudin, and Cristina Maglio. "AB0083 ADIPONECTIN STIMULATES PRO-INFLAMMATORY CYTOKINE PRODUCTION BY PERIPHERAL BLOOD MONONUCLEAR CELLS FROM PATIENTS WITH EARLY UNTREATED RHEUMATOID ARTHRITIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.7355.

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Tsekhanovich, D., A. Starastsin, A. Dybau, and D. Nizheharodava. "Y6T-LYMPHOCYTES PHENOTYPE IN PATIENTS WITH INFLAMMATORY BOWEL DISEASES." In SAKHAROV READINGS 2022: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2022. http://dx.doi.org/10.46646/sakh-2022-2-68-71.

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YST-сells role in inflammatory bowel disease is still not fully investigated: on the one hand, they are thought to be involved in dysregulation of the immune response to gastrointestinal commensal bacteria in genetically susceptible individuals and, on the other hand, YST-cells may initiate repair of damaged intestinal epithelium and exhibit immunoreg-ulatory effects. The composition of T-lymphocytes subsets (авТ-cells, YST-cells) and the expression of functional molecules (TLR4+, CD314+, CD8+, CD45RO+) on circulating YST-cells in patients with inflammatory bowel disease were characterized in
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Cao, D., Y. Yin, H. Shen, et al. "Application of MIZ1 in Peripheral Blood Mononuclear Cells in Severe Community-Acquired Pneumonia and the Relationship Between MIZ1 and Inflammatory Factors." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3877.

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Sulaiti, Asma Al, Mohammed El Anbari, Mohammed Toufiq, et al. "226 ‘Off-the-shelf’ CD19-CAR-T cells generated from umbilical cord blood display superior anti-tumor fitness and lower pro-inflammatory activity as compared to peripheral blood-derived engineered T cells." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0226.

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Klings, Elizabeth S., Daniel A. Dworkis, Surinder Safaya, et al. "Altered Inflammatory Gene Expression Occurs In Peripheral Blood Mononuclear Cells Of Sickle Cell Disease Patients With An Elevated Tricuspid Regurgitant Jet Velocity And Pulmonary Hypertension." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5519.

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Reports on the topic "Peripheral blood inflammatory cells (PBICs)"

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Schwartz, Bertha, Vaclav Vetvicka, Ofer Danai, and Yitzhak Hadar. Increasing the value of mushrooms as functional foods: induction of alpha and beta glucan content via novel cultivation methods. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7600033.bard.

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Abstract:
During the granting period, we performed the following projects: Firstly, we differentially measured glucan content in several pleurotus mushroom strains. Mushroom polysaccharides are edible polymers that have numerous reported biological functions; the most common effects are attributed to β-glucans. In recent years, it became apparent that the less abundant α-glucans also possess potent effects in various health conditions. In our first study, we explored several Pleurotus species for their total, β and α-glucan content. Pleurotuseryngii was found to have the highest total glucan concentrati
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