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Dissertations / Theses on the topic 'Peripheral neuropathies'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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1

Wilmshurst, Jo. "Peripheral neuropathies of childhood." Master's thesis, University of Cape Town, 2009. http://hdl.handle.net/11427/10746.

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Includes synopsis.<br>Incldues bibliographical references (p. 195-220).<br>Peripheral nerve disease was described by Galen (AD 130-200) over a thousand years ago.(3) Detailed anatomical illustrations were documented by Andreas Vesalius in his major work 'De humani corporis fabrica' in 1543.(4) Over the last two centuries an explosion in knowledge in the area has occurred, with a further exponential increase in the last 20 years mostly related to understandings in the field of molecular genetics.(5) Although some degree of diagnostic closure was possible for a number of the hereditary periphera
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2

MELFI, SIMONA. "NEW SIGNALING PATHWAYS REGULATING SCHWANN CELLS OF THE PERIPHERAL NERVOUS SYSTEM: IMPLICATIONS IN PERIPHERAL NEUROPATHIES." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/564819.

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The origin, development and maturation of Schwann cells (SCs), the main glial cells of the peripheral nervous system (PNS), are a set of complicated and intriguing processes. These multifactorial processes take place following a precise and unique coordination between different molecules and intracellular signaling, that interact with a complex of endogenous and exogenous signals. Among these, there are integrins, neuregulins, growth factors, hormones, neurotransmitters and intracellular pathway, including protein kinase A and protein kinase C (PKA and PKC), serine/threonine kinase 1 (A
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3

Fortun, Jenny. "Protein aggregation in peripheral myelin protein 22 (pmp22)-associated neuropathies." [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0010065.

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Thesis (Ph.D.)--University of Florida, 2005.<br>Typescript. Title from title page of source document. Document formatted into pages; contains 123 pages. Includes Vita. Includes bibliographical references.
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4

Lunn, Michael Peter Thomas. "The role of antibodies in paraproteinaemic and inflammatory peripheral neuropathies." Thesis, King's College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246321.

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5

Ferdousi, Maryam. "Assessment of corneal pathology using corneal confocal microscopy in peripheral neuropathies." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/assessment-of-corneal-pathology-using-corneal-confocal-microscopy-in-peripheral-neuropathies(53cb63fb-b645-4389-9fed-f9a028eaea90).html.

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The validity of corneal confocal microscopy (CCM) in assessing peripheral neuropathy has been studied extensively in several studies with a large cohort of subjects with diabetes and in a handful of studies with small sample sizes in subjects with other systemic conditions. The non-invasive nature of this technique as well as its high reproducibility, moderate to high sensitivity and specificity, and ease of use make it an ideal biomarker for diagnosing onset, severity and progression of peripheral neuropathy. This thesis aims to further investigate the potential of CCM by evaluating abnormali
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6

Drew, Alexander Peter. "Genetics of distal hereditary motor neuropathies." Thesis, The University of Sydney, 2012. http://hdl.handle.net/2123/8652.

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The distal hereditary motor neuropathies (dHMN) are a clinically and genetically heterogeneous group of disorders that primarily affect motor neurons, without significant sensory involvement. Using genome wide linkage analysis in a large Australian family (CMT54), a form of dHMN was previously mapped by this laboratory, to a 12.98 Mb interval on chromosome 7q34-q36. The axonal neuropathy seen in this family was classified as dHMN1; with autosomal dominant inheritance, early but variable age of onset, and muscle weakness and wasting affecting the lower limbs. In this project, genetic linkage a
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7

Walters, David Paul. "The prevalence of diabetic foot disease." Thesis, University of London, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320402.

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During a surveillance programme all the known diabetics (1150) were identified from a general population of 97,034 representing all patients registered with 10 general practices. A control group of 751 non-diabetic subjects were also drawn from the same general population. A single observer reviewed 1077 (93.6%) of the diabetics and 480 (69%) of the controls. Peripheral vascular disease was detected using doppler ankle/brachial pressure index in 20.6% (95% CI 18.2-23.0) of diabetics and 9.6% (95% CI 7.0-11.2) of controls. There was no significant difference between the prevalence in non-insuli
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8

Gnonlonfoun, Dieudonné. "Epidémiologie des Neuropathies périphériques au Bénin (EPINEUB)." Electronic Thesis or Diss., Limoges, 2023. http://www.theses.fr/2023LIMO0102.

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La neuropathie périphérique (NP) est une complication fréquente qui jalonne l’évolution des maladies chroniques. Mais son caractère polymorphe et hétérogène, avec une myriade de classifications et des critères diagnostiques rendent difficile l’accès à des données précises sur l'épidémiologie des NP. L’objectif général de cette thèse était d’évaluer l’état actuel des connaissances, les facteurs associés, la prise en charge et les interventions potentielles pour les NP au Bénin. Pour répondre à cet objectif plusieurs travaux ont été réalisés.A partir des données disponibles, la fréquence hospita
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9

Song, Zhentao, and 宋震濤. "Effects of Schwann cell-specific over-expression of aldose reductase on diabetic and galactosemic neuropathy." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31240951.

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10

Jimenez-Andrade, Juan, Monica Herrera, Joseph Ghilardi, Marina Vardanyan, Ohannes Melemedjian, and Patrick Mantyh. "Vascularization of the dorsal root ganglia and peripheral nerve of the mouse: Implications for chemical-induced peripheral sensory neuropathies." BioMed Central, 2008. http://hdl.handle.net/10150/610211.

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Although a variety of industrial chemicals, as well as several chemotherapeutic agents used to treat cancer or HIV, preferentially induce a peripheral sensory neuropathy what remains unclear is why these agents induce a sensory vs. a motor or mixed neuropathy. Previous studies have shown that the endothelial cells that vascularize the dorsal root ganglion (DRG), which houses the primary afferent sensory neurons, are unique in that they have large fenestrations and are permeable to a variety of low and high molecular weight agents. In the present report we used whole-mount preparations, immunoh
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11

Tavakoli, Mitra. "Corneal confocal microscopy : a novel non-invasive diagnostic tool for assessing peripheral neuropathies." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492280.

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Corneal confocal microscopy (GCM) is a novel non-invasive in vivo imaging clinical technique for the study of corneal cellular structure. It provides images of all layers of the cornea which are comparable to in-vitro histochemical techniques. In the series of studies presented in this thesis we have evaluated the considerable potential of GGM to quantify corneal nerve morphology in a range of clinical conditions characterised by small fibre damage. The results indicate that the widest application of GCM may well be in the field of peripheral metabolic (diabetes, Fabry disease) and other cause
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12

Miressi, Federica. "Hereditary Peripheral Neuropathies : from Molecular Genetics to a cellular model of hiPSC-derived motor neurons." Thesis, Limoges, 2020. http://aurore.unilim.fr/theses/nxfile/default/56675caf-59b3-4af2-ae86-c5e356784128/blobholder:0/2020LIMO0053.pdf.

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La maladie de Charcot-Marie-Tooth (CMT) est la neuropathie périphérique héréditaire la plus fréquente. Actuellement plus de 80 gènes ont été identifiées comme étant à l’origine des CMT, mais le diagnostic génétique est posé seulement dans 30 à 40% des cas. Cette étude avait deux objectifs principaux : dans un premier temps, nous nous sommes intéressés aux CMT et neuropathies périphériques associées via une approche moléculaire et bioinformatique, pour optimiser leur caractérisation génétique ; dans un second temps, nous avons étudié les mécanismes altérés dans une forme axonale de CMT, par la
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13

Cerles, Olivier. "Prévention des neuropathies périphériques induites par les chimiothérapies par une modulation pharmacologique des dérives des formes réactives de l'oxygène et des récepteurs muscariniques." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB106.

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Les chimiothérapies à base de sels de platine exercent leurs effets anti-tumoraux en compromettant l'intégrité de l'ADN. Cette cytotoxicité conduit à une augmentation du stress oxydant qui, à son tour, favorise les processus de mort cellulaire. L'oxaliplatine indiquée dans les cancers métastatiques secondaires du colon et dans les cancers colorectaux, induit une augmentation des espèces réactives de l’oxygène en diminuant le glutathion réduit dans les cellules cancéreuses. Similairement aux autres chimiothérapies à base de sels de platine, elle doit être utilisée avec précaution. En effet, la
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14

Lare, Banfantini. "Prise en charge du diabète et impact sur les neuropathies périphériques au Togo." Electronic Thesis or Diss., Limoges, 2024. http://www.theses.fr/2024LIMO0010.

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PRISE EN CHARGE DU DIABETE ET IMPACT SUR LES NEUROPATHIES PERIPHERIQUES AU TOGO Il s'agit d'une thèse en cotutelle entre les universités de Limoges en France et de Lomé au Togo. L'objectif général de cette thèse est d'améliorer les connaissances épidémiologiques du diabète et des neuropathies périphériques au Togo Cette thèse s'articule en cinq études. ETUDE1: Valeurs normales de Conductance Electrochimique Cutanée mesurer par sudoscan en population africaine Objectif principale de cette étude est de définir les valeurs normales du Sudoscan® en population africaine Critère de jugement principa
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15

Kawasaki, Teruaki. "Oct6, a transcription factor controlling myelination, is a marker for active nerve regeneration in peripheral neuropathies." Kyoto University, 2003. http://hdl.handle.net/2433/148771.

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16

Ng, Wing Tin Sophie. "Douleur et caractérisation neurophysiologique de l'atteinte des petites fibres dans les neuropathies périphériques." Thesis, Paris Est, 2013. http://www.theses.fr/2013PEST0117.

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L'objectif de notre travail était dans un premier temps d'étudier les liens entre l'altération des fibres nerveuses de petit diamètre et la présence de douleurs chez des patients ayant une neuropathie périphérique. Notre deuxième objectif était d'évaluer la pertinence de certaines techniques neurophysiologiques pour mettre en évidence l'atteinte de ces petites fibres nerveuses. Notre première étude réalisée sur une large cohorte de patients présentant divers types de neuropathie a montré, grâce à une étude quantifiée de la sensibilité, qu'il n'y avait pas de corrélation entre la perte ou perte
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17

Howard, Heidi C. "Identification of the gene responsible for peripheral neuropathy associated with agenesis of the corpus callosum." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84259.

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Peripheral neuropathy associated with agenesis of the corpus callosum (ACCPN or HMSN/ACC) is a severe polyneuropathy affecting both the peripheral nervous system and the central nervous system. It is transmitted as an autosomal recessive trait and is particularly frequent in the French Canadian population of Quebec (Canada). The disease was linked to chromosome 15 in 1996 by Dr. Rouleau's team.<br>We genotyped polymorphic markers in the ACCPN candidate region on chromosome 15 in over 67 patients and 200 control individuals. Observation of affected haplotypes confirmed the presence of a
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18

Bouchenaki, Hichem. "Evaluation de l'effet de molécules candidates dans des modèles murins de neuropathies périphériques induites par la chimiothérapie anticancéreuse." Thesis, Limoges, 2020. http://www.theses.fr/2020LIMO0035.

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Les neuropathies périphériques induites par la chimiothérapie (NPIC) sont un effet indésirable commun des agents anticancéreux. Les symptômes des NPIC sont principalement sensitifs : douleurs, fourmillements, sensations d’engourdissement, altérations de la sensibilité thermique et mécanique. La symptomatologie des NPIC est associée à une forte altération de la qualité de vie des patients, et peut dans certains cas conduire à une diminution des doses et des cycles de chimiothérapie, voire à un arrêt du traitement. La prise en charge actuelle des NPIC repose principalement sur l’utilisation de m
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19

Lerat, Justine. "Neuropathies Périphériques Génétiques et Surdité : Etude des Relations Génétiques et Mécanistiques." Thesis, Limoges, 2018. http://www.theses.fr/2018LIMO0055.

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Les neuropathies périphériques héréditaires (NP) sont caractérisées par des phénotypes très divers et une hétérogénéité génétique importante. La maladie de Charcot-Marie-Tooth (CMT) représente la majeure partie des neuropathies périphériques sensitivo-motrices. D’autres symptômes peuvent être associés, telle que la surdité. A l’heure actuelle, aucune estimation précise de la surdité n’existe dans cette population et la pathogénicité est incertaine. L’objectif de cette thèse était de mieux comprendre la physiopathologie de la surdité chez les patients atteints de neuropathies périphériques. Pou
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20

Castelnovo, L. F. "MOLECULAR BASIS FOR THE DEVELOPMENT OF INNOVATIVE THERAPIES FOR PERIPHERAL NEUROPATHIES TREATMENT: ROLE AND CROSS-REGULATION OF THE GABAERGIC SYSTEM AND NEUROACTIVE STEROIDS." Doctoral thesis, Università degli Studi di Milano, 2017. http://hdl.handle.net/2434/465187.

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Peripheral neuropathies are a heterogeneous group of pathologies with a high prevalence worldwide, which are characterized by alterations of peripheral nerves structure and function. Their treatment is currently a challenge for clinicians. Indeed, even if continuous progresses are made in the study of the basic mechanisms underlying these pathologies, etiology is still unknown in a significant number of cases. Different compounds, such as growth factors, adhesion proteins, neurotransmitters, enzymes, peptides and neuroactive steroids, have been proposed to play important roles in the patho-phy
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21

Jamali, Reza. "Peripheral Hypoglycaemic Neuropathy in Type 1 Diabetic Rats : Morphologic and Metabolic Studies." Doctoral thesis, Linköping : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-7978.

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22

El, Bazzal Lara. "Etude des mécanismes physiopathologiques des neuropathies périphériques dues à des mutations dans FRABIN (CMT4H) et VRK1." Thesis, Aix-Marseille, 2018. http://www.theses.fr/2018AIXM0736.

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Les Neuropathies Périphériques Héréditaires (IPN) constituent l’une des causes les plus fréquentes de maladies neurologiques héréditaires. Parmi elles, la maladie de Charcot-Marie-Tooth (CMT), constitue le groupe plus large. Durant ma thèse, j’ai étudié les bases physiopathologiques de deux formes d’IPN.1)J’ai étudié CMT4H, une forme rare de CMT démyélinisante, à transmission autosomique récessive, due à des mutations dans FGD4, qui code la protéine FRABIN. J’ai ainsi validé trois partenaires de FRABIN impliqués dans la voie du trafic vésiculaire. J’ai mis au point un modèle de myélinisation i
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23

Munné, Rodríguez Carles. "Neurodinámica: movilizaciones del sistema nervioso estudio descriptivo observacional." Doctoral thesis, Universitat de Vic - Universitat Central de Catalunya, 2022. http://hdl.handle.net/10803/673701.

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Introducción: La neurodinámica se considera una técnica de terapia manual donde las fuerzas aplicadas van orientadas a la acción sobre las estructuras nerviosas a partir de la movilización y posicionamiento de las articulaciones, integrando las funciones biomecánicas y fisiológicas del sistema nervioso. Objetivos: Analizar y cuantificar el desplazamiento del nervio ciático en la región isquiosural mediante ecografía mediante la técnica del “Frame-by-frame cross correlation system” en función de la rama terminal seleccionada así como observar si hay una restricción del desplazamiento en pers
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24

Saad, Mehdi. "Détection précoce et quantification objective par mesures chronoampérometriques de l’atteinte neurologique périphérique chez des patients recevant une chimiothérapie neurotoxique." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS060/document.

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Introduction : La chimiothérapie cytotoxique constitue une modalité thérapeutique de nombreux cancers. L’amélioration de la durée de survie des patients a fait apparaître des complications de ces traitements notamment sur le nerf périphérique. Il s’agit d’une complication fréquente et potentiellement sévère qui peut avoir un impact durable. Pourtant, bien que les chimiothérapies neurotoxiques soient connues, il n’existe pas de données précises permettant de prédire la tolérance individuelle. La détection précoce des polyneuropathies chimio-induites (PNCI) est donc capitale pour évaluer les fac
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Matta, Célia. "Neuropathies induites par chimiothérapie néo-adjuvante du cancer du sein : atteintes périphériques et centrales, mécanismes impliqués et perspectives thérapeutiques." Thesis, Strasbourg, 2018. http://www.theses.fr/2018STRAJ080/document.

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La chimiothérapie néoadjuvante (CNA) est une avancée majeure dans les traitements des cancers du sein. La CNA réduit nettement la tumeur primaire et permet une chirurgie conservatrice à tous les stades du cancer. Malheureusement, elle s’accompagne de dysfonctionnements neurologiques qui limitent le succès du traitement anti-tumoral. La prise en charge médicale de ces troubles neurologiques est problématique compte tenu de l’inefficacité des neuroprotecteurs disponibles. De plus, la recherche de nouvelles thérapeutiques est handicapée par l’absence de modèles expérimentaux reproduisant fidèleme
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Toledano, Sierra Pilar. "Afectación neurológica en el lupus eritematoso sistémico: Alteraciones en la neuroimagen y caracterización de la afectación del sistema nervioso periférico." Doctoral thesis, Universitat de Barcelona, 2017. http://hdl.handle.net/10803/585890.

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Los síndromes neuropsiquiátricos son una complicación grave del lupus eritematoso sistémico (LES) que contribuyen de forma considerable a una disminución de la calidad de vida y a un aumento de la morbilidad y mortalidad de dichos pacientes. No obstante, su diagnóstico continúa siendo un desafío por la ausencia de una prueba de oro que permita la atribución correcta de estas manifestaciones a la enfermedad. La resonancia magnética nuclear (RM) es la prueba de imagen de elección en los casos de afectación neuropsiquiática de localización central, pero su validez e importancia en el diagnóstico
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Lamarca, Dams Aloa. "Extracellular UTP signalling in Schwann cell migration: A novel role of the P2Y2 receptor." Doctoral thesis, Universitat Internacional de Catalunya, 2013. http://hdl.handle.net/10803/121635.

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Peripheral neuropaties are one of the major complications of the Peripheral Nervous System. Depsite the big importance of those pathologies, less drugs are effective for their treatment, and most of the prescribed drugs are based on the inhibition of pain. Nucleo CMP forte is a drug mainly composed of nucleotides (UMP, UDP, UTP and CMP), prescribed to patients with Peripheral Nervous System disorders. But their exact mechanism of action is still unknown. Because extracellular nucleotides can act as signalling molècules, our research group focus the studies in which are the effects of the Nucle
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Wallace, Victoria C. J. "Peripheral nerve demyelination and neuropathic pain." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/27605.

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Chronic neuropathic pain, characterised by allodynia (perception of innocuous stimuli as painful) and hyperalgesia (facilitated responses to painful stimuli), is poorly understood and is usually resistant to classical analgesics. Such abnormal pain phenomena can be associated with human demyelinating conditions such as Charcot-Marie-Tooth disease. Using mouse models of peripheral nerve demyelination, we have provided evidence for the consequent establishment of neuropathic pain and investigated possible underlying mechanisms. The first model investigated was the Prx-null mouse. The murine peri
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29

Taylor, Anna. "Peripheral mechanisms of trigeminal neuropathic pain." Thesis, McGill University, 2011. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=97105.

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Trigeminal neuropathic pain is a unique type of neuropathic pain resulting from damage to primary afferents of the trigeminal nerve that innervates the head and neck region. It is characterized by intractable, unremitting pain in the absence of any overt tissue damage, and represents a significant clinical and societal burden. A thorough understanding of the mechanisms driving this condition is required to adequately treat, and ideally cure, this condition. The skin of the lower lip is innervated by several classes of primary afferents, including myelinated Aβ fibers, thinly myelinated Aδ f
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30

Eid, Stéphanie A. "Neuropathic injury : new insights on NADPH oxidases in diabetic neuropathy." Electronic Thesis or Diss., Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB130.

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Le diabète, un problème majeur de santé publique, est associé à un certain nombre de facteurs de risque métaboliques qui contribuent à la progression de maladies micro- et macrovasculaires. Les mécanismes qui contribuent à l'apparition et au développement de ces complications sont encore mal déterminés. La neuropathie diabétique (DN) est la complication la plus fréquente du diabète dans le monde, touchant jusqu'à 50% de tous les patients diabétiques. La neuropathie périphérique diabétique (DPN), le type le plus courant de neuropathie diabétique est associé à des changements histologiques et de
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31

Hausner, Robert Michael. "Degenerative periphere Neuropathien bei Hund und Katze." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-93213.

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32

Caspani, Ombretta [Verfasser]. "Cold transduction mechanisms during peripheral neuropathic pain / Ombretta Caspani." Berlin : Freie Universität Berlin, 2008. http://d-nb.info/1023169932/34.

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33

Ramer, Matthew Stephen. "Sympathetic and sensory neuronal plasticity, peripheral substrates of neuropathic pain." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ31950.pdf.

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34

Liu, Xue Jun. "Peripheral regulation of inflammatory pain and neuropathic pain by adenosine." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/NQ66636.pdf.

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35

Bian, Di. "Peripheral and spinal mechanisms of neuropathic pain in the rat." Diss., The University of Arizona, 2000. http://hdl.handle.net/10150/284087.

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The Chung peripheral nerve injury model shows consistent allodynia and thermal hyperalgesia, which represent the most common clinical neuropathic pain symptoms. Also clinically relevant, in the Chung model spinal morphine was inactive against tactile allodynia and diminished in potency in acute nociception without spinal/supraspinal antinociceptive synergy. Further, there are increased levels of dynorphin in multiple segmental regions of the spinal cord. This loss of spinal/supraspinal synergy and the spinal antiallodynic effect of morphine is restored by spinal MK-801 or antiserum to dynorphi
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36

Erichsen, Helle Kirstein. "Characterisation of the spared nerve injury model of neuropathic pain /." Cph. : The Danish University of Pharmaceutical Scienes, Department of Pharmacology, NeuroSearch, Kongl. Carolinska Medico Chirurgiska Institutet, 2003. http://www.dfh.dk/phd/defences/Hellekirsteinerichsen.htm.

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37

McCormick, Barry. "Antioxidant protection in mitochondria in chemotherapy-induced neuropathic pain." Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229728.

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Neuropathic pain is a common and dose-limiting adverse effect of several cancer chemotherapeutic agents including paclitaxel. Current treatments for chemotherapy-induced peripheral neuropathy (CIPN) are largely ineffective and the pain can persist long after the cessation of the chemotherapy regimen. Whilst the specific underlying mechanisms are not fully understood, oxidative stress and mitochondrial damage are thought to be involved in the development of CIPN. Antioxidants which protect mitochondria may inhibit oxidative stress and protect mitochondrial function more effectively than antioxi
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Rode, Frederik. "Pharmacological testing in the spared nerve injury model of neuropathic pain /." Cph. : The Danish University of Pharmaceutical Sciences, Department of Pharmacology, 2005. http://www.dfuni.dk/index.php/Frederik_Rode/1938/0/.

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39

Evans, Laura Jane. "Peripheral changes in NGF in a spared fibre model of neuropathic pain." Thesis, University of Sheffield, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574620.

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The experiments described in this thesis have investigated the role of nerve growth factor (NGF) acting on spared nerve fibres in the pathophysiology of neuropathic pain, caused by a partial nerve injury of the mental nerve, a branch of the trigeminal nerve that innervates the skin and mucosa of the lower lip. The experiments have tested the hypothesis that an over-supply of NGF acts on a reduced population of mental nerve fibres and causes phenotypic changes in the population that could underlie symptoms of neuropathic pain. A behavioural testing protocol was developed to investigate the pres
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Wieczorek, Lara Alexa. "Nervenzupfpräparation (nerve fiber teasing) in der Diagnostik peripherer Neuropathien beim Tier." Diss., lmu, 2002. http://nbn-resolving.de/urn:nbn:de:bvb:19-7107.

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Klein, Johann, Sahr Sandi-Gahun, Gabriele Schackert, and Tareq A. Jratli. "Peripheral nerve field stimulation for trigeminal neuralgia, trigeminal neuropathic pain, and persistent idiopathic facial pain." Sage, 2015. https://tud.qucosa.de/id/qucosa%3A35439.

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Objective: Peripheral nerve field stimulation (PNFS) is a promising modality for treatment of intractable facial pain. However, evidence is sparse. We are therefore presenting our experience with this technique in a small patient cohort. Methods: Records of 10 patients (five men, five women) with intractable facial pain who underwent implantation of one or several subcutaneous electrodes for trigeminal nerve field stimulation were retrospectively analyzed. Patients’ data, including pain location, etiology, duration, previous treatments, long-term effects and complications, were evaluated. Resu
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Gurney, Jason Kevin. "The effect of an exercise intervention on peripheral sensorimotor function in neuropathic diabetics." Thesis, University of Auckland, 2011. http://hdl.handle.net/2292/9081.

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Peripheral neuropathy is the first and most common microvascular complication of diabetes mellitus, and involves the distal-to-proximal necrosis of peripheral nerves. Loss of peripheral sensory function, combined with confounding autonomic and neuromuscular complications, leads to an increase in the risk of plantar ulceration due to traumatic or repetitive loading. The evidence for the effectiveness of an exercise intervention in improving sensorimotor function in this population is building, but care is needed when implementing these interventions due to the risk of exacerbating associa
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Wiendieck, Kurt Rolf Winhold. "Punktmutationen im Connexin32-, PMP22- und anderen Genen als Ursache hereditärer peripherer Neuropathien /." Berlin : Rhombos, 2008. http://d-nb.info/987600079/04.

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Wiendieck, Kurt Rolf Winhold. "Punktmutationen im Connexin32-, PMP22- und anderen Genen als Ursache hereditärer peripherer Neuropathien." Berlin Rhombos, 2007. http://d-nb.info/987600079/04.

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Ntogwa, Mpumelelo. "Mechanisms of HIV-induced peripheral neuropathic pain by focusing on Schwann cell-macrophage interaction." Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263602.

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Cruz, Mariana Neiva. "Doença de Charcot-Marie-Tooth ligado ao X em crianças: série de casos tipo 1 de pacientes do HC-FMRP." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17160/tde-24042018-172820/.

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Entre as neuropatias periféricas hereditárias, a Doença de Charcot Marie Tooth (CMT) é a mais prevalente, sendo o Charcot Marie Tooth Lidado ao X tipo 1 (CMTX1) o segundo subtipo mais comum, causado por mutações no gene GJB1 e de herança ligada ao X. A sintomatologia de fraqueza, atrofia e alteração de sensibilidade progressiva, de padrão simétrico e distal é característica da CMT e, no CMTX1, o acometimento do sistema nervoso central pode estar associado ao quadro típico. Com relação à eletroneurofisiologia, há redução dos parâmetros de velocidade de condução nervosa, com prolongamento da lat
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XIE, WENRUI. "BLOCKADE OF ECTOPIC ACTIVITY AT THE INITIAL STAGE OF PERIPHERAL NERVE INJURY PREVENTS NEUROPATHIC PAIN." University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1057691866.

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Stötzner, Philip [Verfasser]. "Opioids in neuropathic pain - the role of potassium channels in peripheral sensory neurons / Philip Stötzner." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2018. http://d-nb.info/1160515018/34.

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Durrenberger, Pascal F. "Peripheral and central mechanisms in chronic human inflammatory and neuropathic pain and associated animal models." Thesis, Imperial College London, 2007. http://hdl.handle.net/10044/1/11344.

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Lieber, Sascha [Verfasser]. "Chemotherapie-induzierte periphere Neuropathie bei Kindern und Jugendlichen nach akuter lymphoblastischer Leukämie / Sascha Lieber." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2019. http://d-nb.info/1202042082/34.

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