Relevant bibliographies by topics / Peripheral vascular diseases

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Peripheral vascular diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 July 2024

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Journal articles on the topic "Peripheral vascular diseases"

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MALIK, GULZAR AHMAD, KASHIF KURSHID QURESHI, IMTIAZ AHMAD, and Muhammad Afzal. "PERIPHERAL VASCULAR DISEASES." Professional Medical Journal 14, no. 01 (March 10, 2007): 134–44. http://dx.doi.org/10.29309/tpmj/2007.14.01.3640.

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Objectives: To observe the prevalence & presentation and to review the noninvasive approaches for the evaluation and treatment of patients presenting with peripheral vascular diseases at Bahawal Victoria Hospital Bahawalpur. Design: Prospective randomized study. Place and Duration: This study was conducted from July 2003 to June 2005 at the department of Surgery Bahawal Victoria Hospital Bahawalpur. Patients & Methods: Twenty patients, 2 females and 18 males admitted with peripheral vascular diseases (PVD) fulfilling the inclusion criteria were evaluated and treated medically and surgically. A standard noninvasive approach for the evaluation of these patients was adopted by history & thorough clinical examination, Doppler USG of the vessels, Ankle Brachial Indices (ABI), Duplex Scanning and MRI in a few cases. Results: The relative frequency of PVD at BVH Bahawalpur was 1.2%. The majority of patients (60%) were in the 4th decade of life and male (90%). The smoking was exclusively the major predisposing risk factor (90%). The common (90%) presentation of patients was intermittent claudication with 60% gangrenous disease with an average duration of 4years, The lower limbs were involved in 90% cases with 70% bilateral disease. Majority (90%) of the patients was diagnosed clinically and the objective severity of the disease was assessed with Doppler sonography in all the patients. The ABI was <0.5 in 85% cases. The duplex scanning was needed only in 10% patients. The treatment procedures carried out were primary amputation in 45% followed by conservative on medicines 20%, atherectomy in 15%, lumbar sympathectomy in 10% and resection or repair of pseudoaneurysms in 10% of cases. The ultimate rate of amputation at various levels was seen to be 75%. Conclusion: The prevalence of PVD is rapidly increasing in our society which is causing a horrible threat in the form of physical disabilities at a younger age group of poor class mostly. Smoking remains exclusively the only major risk factor. Much time and money can be saved by evaluating and treating these patients by noninvasive approaches but prevention is the besttherapeutic strategy achieving by abstinence from the smoking.
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Stewart, J. H. "Peripheral Vascular Diseases." Cardiovascular Research 27, no. 5 (May 1, 1993): 891–92. http://dx.doi.org/10.1093/cvr/27.5.891a.

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Taylor, Lloyd M. "Peripheral vascular diseases." Journal of Vascular Surgery 16, no. 3 (September 1992): 507. http://dx.doi.org/10.1016/0741-5214(92)70058-s.

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Taylor, Lloyd M. "Peripheral vascular diseases." Journal of Vascular Surgery 16, no. 3 (September 1992): 505–6. http://dx.doi.org/10.1016/0741-5214(92)90406-x.

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Zarins, Christopher K. "Peripheral Vascular Diseases." JAMA: The Journal of the American Medical Association 268, no. 4 (July 22, 1992): 543. http://dx.doi.org/10.1001/jama.1992.03490040127038.

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McPherson, S. "Peripheral vascular diseases." International Journal of Cardiology 38, no. 1 (January 1993): 106. http://dx.doi.org/10.1016/0167-5273(93)90219-7.

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Robbins, Jeffrey M., and Corliss L. Austin. "COMMON PERIPHERAL VASCULAR DISEASES." Clinics in Podiatric Medicine and Surgery 10, no. 2 (April 1993): 205–19. http://dx.doi.org/10.1016/s0891-8422(23)00599-2.

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Skversky, Norman J., Allen B. Herring, and Raymond C. Baron. "THERMOGRAPHY IN PERIPHERAL VASCULAR DISEASES." Annals of the New York Academy of Sciences 121, no. 1 (December 16, 2006): 118–34. http://dx.doi.org/10.1111/j.1749-6632.1964.tb13691.x.

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Gelabert, Hugh. "Color atlas of peripheral vascular diseases." Journal of Vascular Surgery 26, no. 1 (July 1997): 174. http://dx.doi.org/10.1016/s0741-5214(97)70170-8.

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Wang, Hsin-Kai, Yi-Hong Chou, Hong-Jen Chiou, See-Ying Chiou, and Cheng-Yen Chang. "B-flow Ultrasonography of Peripheral Vascular Diseases." Journal of Medical Ultrasound 13, no. 4 (2005): 186–95. http://dx.doi.org/10.1016/s0929-6441(09)60108-9.

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Dissertations / Theses on the topic "Peripheral vascular diseases"

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Eneroth, Magnus. "Amputation for vascular disease prognostic factors for healing, long-term outcome and costs /." Lund : Lund University, Dept. of Orthopedics, Lund University Hospital, 1997. http://catalog.hathitrust.org/api/volumes/oclc/39752358.html.

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Ögren, Mats. "Vascular morbidity and mortality in men with non-invasively detected peripheral arterial disease results from the prospective population study "Men born in 1914" /." Lund : Dept. of Community Health Sciences and the Dept. of Clinical Physiology, Malmö General Hospital, Lund University, 1994. http://catalog.hathitrust.org/api/volumes/oclc/39693808.html.

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Sigvant, Birgitta. "Epidemiological aspects of peripheral arterial disease." Stockholm : Department of Molecular Medicine and Surgery, Karolinska Institutet, 2009. http://diss.kib.ki.se/2009/978-91-7409-670-5/.

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Christman, Sharon Klopfenstein. "Intervention to slow progression of peripheral arterial disease." Connect to this title online, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1054059524.

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Thesis (Ph. D.)--Ohio State University, 2003.
Title from first page of PDF file. Document formatted into pages; contains xiii, 123 p.; also includes graphics (some col.). Includes bibliographical references (p. 114-123). Available online via OhioLINK's ETD Center
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Lewis, M. H. "Peripheral arterial disease from aetiology to surgical management." Thesis, University of South Wales, 2013. https://pure.southwales.ac.uk/en/studentthesis/peripheral-arterial-disease-from-aetiology-to-surgical-management(7defd31a-6995-4fc7-9302-2fced42b5982).html.

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The work presented includes over thirty peer reviewed published manuscripts based on studies undertaken during my surgical career. As Principal Investigator, I led the study conception/design/data acquisition/analysis/interpretation and was involved with writing the final drafts of all manuscripts prior to their formal submission to high impact factor peer-reviewed specialist journals. The thesis is divided into subsections reflecting my development and different interests within surgery. The subsections start with my learning basic research principles, moving onto clinical problem solving in general surgical dilemmas, followed by a collection of papers in my subspecialty of vascular surgery. The work culminates with a group of papers focused on aneurysmal disease, specifically, abdominal aortic aneurysms (AAA), the clinical impact of which has had a bearing on the introduction of a National AAA Screening Program in Wales in 2013. I conclude these sections with a collection of papers that reflect my long term commitment to surgical training both at regional level (as Secretary and Deputy Chairman to the Higher Surgical Training Committee and Chairman of the Basic Surgical Training Committee) and national level including my involvement with the Four Royal Colleges of Surgeons for the Intercollegiate Examinations in General Surgery. This examination is undertaken at completion of junior surgical training and used to confirm a doctor's competence for safe independent practice as a consultant. In conclusion, over forty years of academic research during my career as a vascular surgeon has provided unique insight into the pathophysiology, treatment and ultimately prevention of artherosclerotic disease. These findings have improved health policies in Wales and significantly reduced patient morbidity and mortality.
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Layman, Hans Richard William. "Tissue Engineering Strategies for the Treatment of Peripheral Vascular Diseases." Scholarly Repository, 2010. http://scholarlyrepository.miami.edu/oa_dissertations/461.

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Peripheral vascular diseases such as peripheral artery disease (PAD) and critical limb ischemia (CLI) are growing at an ever-increasing rate in the Western world due to an aging population and the incidence of type II diabetes. A growing economic burden continues because these diseases are common indicators of future heart attack or stroke. Common therapies are generally limited to pharmacologic agents or endovascular therapies which have had mixed results still ending in necrosis or limb loss. Therapeutic angiogenic strategies have become welcome options for patients suffering from PAD due to the restoration of blood flow in the extremities. Capillary sprouting and a return to normoxic tissue states are also demonstrated by the use of angiogenic cytokines in conjunction with bone marrow cell populations. To this point, it has been determined that spatial and temporal controlled release of growth factors from vehicles provides a greater therapeutic and angiogenic effect than growth factors delivered intramuscularly, intravenously, or intraarterialy due to rapid metabolization of the cytokine, and non-targeted release. Furthermore, bone marrow cells have been implicated to enhance angiogenesis in numerous ischemic diseases due to their ability to secrete angiogenic cytokines and their numerous cell fractions present which are implicated to promote mature vessel formation. Use of angiogenic peptides, in conjunction with bone marrow cells, has been hypothesized in EPC mobilization from the periphery and marrow tissues to facilitate neovessel formation. For this purpose, controlled release of angiogenic peptides basic fibroblast growth factor (FGF-2) and granulocyte-colony stimulating factor (G-CSF) was performed using tunable ionic gelatin hydrogels or fibrin scaffolds with ionic albumin microspheres. The proliferation of endothelial cell culture was determined to have an enhanced effect based on altering concentrations of growth factors and method of release: co-delivery versus sequential. Scaffolds with these angiogenic peptides were implanted in young balb/c mice that underwent unilateral hindlimb ischemia by ligation and excision of the femoral artery. Endpoints for hindlimb reperfusion and angiogenesis were determined by Laser Doppler Perfusion Imaging and immunohistochemical staining for capillaries (CD-31) and smooth muscle cells (alpha-SMA). In addition to controlled release of angiogenic peptides, further studies combined the use of a fibrin co-delivery scaffold with FGF-2 and G-CSF with bone marrow stem cell transplantation to enhance vessel formation following CLI. Endpoints also included lipophilic vascular painting to evaluate the extent of angiogenesis and arteriogenesis in an ischemic hindlimb. Tissue engineering strategies utilizing bone marrow cells and angiogenic peptides demonstrate improved hindlimb blood flow compared to BM cells or cytokines alone, as well as enhanced angiogenesis based on immunohistochemical staining and vessel densities.
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Richardson, Jim. "Living with peripheral vascular disease a one-person case study : a dissertation [thesis] presented in partial fulfilment of the requirements for the Master of Health Science at Auckland University of Technology, December 2002." Full thesis. Abstract, 2002.

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Hou, Xiang-Yu. "Exercise performance and mitochondrial function in peripheral arterial disease." Thesis, Queensland University of Technology, 2002. https://eprints.qut.edu.au/36778/1/36778_Digitised%20Thesis.pdf.

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Peripheral arterial disease (PAD) is an atherosclerotic disease in the peripheral arteries, which reduces blood supply to the lower extremities. Intermittent claudication is the symptom that develops early in PAD patients and is accompanied by the haemodynamic finding of a fall in systolic blood pressure at the ankles following exercise. In PAD patients, exercise performance is not well correlated with haemodynamic measurements and other mechanisms have been suggested to account for the impairment. There have been reports about the impaired mitochondrial metabolism (eg, decreased activities of mitochondrial enzymes) and abnormal mitochondrial structure in the skeletal muscle in PAD patients. It is not known, however, whether mitochondrial ATP production is impaired in the skeletal muscle in PAD. Whether the mitochondrial function is impaired in PAD patients, and whether the impaired mitochondrial function in the muscle contributes to the impaired exercise performance in PAD patients is unknown. The object of this work is to explore the mitochondrial function in the skeletal muscle of PAD patients and its relationship to exercise performance. Impaired exercise performance in PAD patients is evaluated using a treadmill walking performance test, which is closely correlated to patients' daily activity performance. Treadmill walking however, in addition to being influenced by local muscle factors, is influenced by central contributions, such as cardiac output and the central nervous system. As walking is limited by intermittent claudication in PAD patients localized in the legs, it would be valuable to develop a local calf muscle performance test to better understand the underlying pathophysiology in PAD. Such a protocol has not been used previously in experiments involving PAD patients. Hence, the research aim for Study 1 was to establish a calf muscle performance test protocol and to investigate its variability. Fourteen healthy control subjects and eight PAD patients undertook the maximal plantar flexion test once a week for five weeks using a Kin-Com Dynamometer. In the traditional assessment, the total impulse and peak impulse are the variables that were measured as representing the calf muscle performance. Both these variables are significantly lower in PAD patients than in controls. Alternatively, by applying simple mathematical models, the muscle function dimensions of endurance, strength and fatigability can be investigated in a single test. Compared with control subjects the PAD patients had lower muscle endurance, lower muscle strength, higher fatigue index, but no difference were found in magnitude or rate-of-fatigue. The variability of the test was different for different estimated parameters of the models, with the highest variability in muscle fatigability (rate of fatigue, CV=75% in controls) and the lowest variability in muscle strength (CV=16% in controls). The variability of the traditional assessment parameters, which included total impulse and peak impulse, was around 13% in controls and 18- 24% in PAD patients for the five tests. Based on these findings the calf muscle performance test can be applied in PAD patients to investigate different muscle function dimensions. While many of the dimensions were impaired in PAD patients compared with controls, the high variability of some of the parameters have to be considered during its application. Having established a local calf muscle performance test, the aim of Study 2 was to explore the relationship between the calf muscle performance and the traditional treadmill walking performance. Seventeen PAD patients and fourteen control subjects were tested using both the calf muscle performance test described earlier and walking performance test. The walking performance was tested using a graded treadmill protocol. The total walking time was significantly lower in PAD than that in control subjects. No variable of calf muscle performance correlated with walking performance in control subjects. However, in PAD patients, a number of calf muscle performance variables correlated with walking performance. The total impulse and the peak impulse in the best legs (higher ABI) tended to correlate with pain-time. In simple mathematical models, the muscle endurance in the worst legs (lower ABI) correlated positively with pain-walking time, and the muscle fatigue-index in the worst legs correlated negatively with total walking time. In conclusion, in PAD patients, some dimensions of calf muscle performance correlated with walking performance. This suggests that some factors of local calf muscle performance might contribute to the impaired walking performance in PAD patients. The research aim for Study 3 was to investigate a number of calf muscle physiological factors, and to ascertain their relationship with calf muscle and walking performance in PAD patients and control subjects. The physiological factors examined include ankle brachial pressure index (ABI), calf muscle weight, calf blood flow, and skeletal muscle mitochondrial ATP production rate (MAPR) in vitro. The calf muscle weight in PAD patients was significantly lower than that in control subjects. In PAD patients, the calf muscle weight was significantly lower in the worst legs than that in the best legs. The ABI was lower in PAD than in controls and significantly lower in PAD worst legs than in PAD best legs. The leg blood flow (measured by venous-occlusion plethysmography) was lower in PAD than that in controls, but there was no significant difference between PAD best legs and PAD worst legs. The MAPR was measured using different substrate combinations. The MAPR (PM, pyruvate + malate), MAPR (PCM, palmitoyl-carnitine + malate) and MAPR (PPKM, pyruvate + palmi_toyl-carnitine + alpha-ketoglutarate + malate) shows the capacity of mitochondria to produce ATP by oxidising glucose or fatty acids or both of these substrates respectively. The MAPR for the three substrate combinations in PAD patients was no different from controls. The relationship between these physiological measurements and exercise performance differed between PAD and controls. In control subjects, the calf muscle weight, ABI, leg blood flow and MAPR were not significantly correlated with walking performance, but correlated with some variables of local calf muscle performance. In PAD patients, the calf muscle weight, ABI and blood flow did not correlate with walking performance. However, the MAPR (PMkg) was positively correlated with total walking time, and MAPR (PPKMg) was positively correlated with pain-free walking time. In calf muscle performance, in the best legs, the body weight was positively correlated with total impulse and peak impulse; the calf muscle weight was positively correlated with contraction number and peak impulse; and the blood flow correlated with peak impulse. In the worst legs, the calf muscle weight and ABI were not significantly correlated with any variables; the leg blood flow was negatively correlated with contraction number; the mitochondrial protein content correlated with total impulse; the MAPR (PM) tended to correlate with peak impulse. These results suggest the importance of all these local muscle physiological factors in calf muscle performance. However, only MAPR was important in the walking performance in PAD patients. The aim of Study 4 was to further explore the relationship between MAPR and exercise performance in PAD patients after exercise training. The effect of 16 weeks of treadmill exercise training on exercise performance and MAPR was evaluated in five PAD patients. In the treadmill walking performance test, total walking time increases ranged from 100 to 150% in these five patients. The pain-free walking time increased in three patients but did not change in the other two. In the calf muscle performance test, the total impulse and contraction number increased in both legs of four patients and decreased in both legs of one patient, but the magnitude of improvement was less than 5% and the peak impulse did not change in a consistent trend. The changes in body weight, calf muscle weight, and ABI in these five patients were less than 5%. However, the increased blood flow measured by venousocclusion plethysmography in both legs ranged from 100 to 150%. The MAPR by oxidising glucose was significantly higher in trained patients than that in untrained patients, which suggested a possible change in mitochondrial function in response to exercise training. Such change in mitochondrial function may have a potential role in contributing to calf muscle performance and walking performance after exercise training. In summary, for the first time, a local calf muscle performance test has been established to allow better understanding of calf muscle pathophysiology in PAD patients. Using this test, it has been shown that calf muscle performance is significantly impaired in PAD patients compared with control subjects. The impairment is characterised by lower muscle endurance, lower muscle strength and higher fatigability. The impaired local calf muscle performance might contribute to the impaired overall walking performance in PAD patients. The MAPR, especially 5 through oxidising glucose, contributed to walking performance. In this pilot exercise training study, a 20 weeks exercise training program failed to improve the calf muscle performance and walking performance in PAD patients. The higher MAPR in oxidising glucose in trained PAD patients again suggested the importance of muscle glucose oxidation as a contributing factor in the exercise performance in PAD patients.
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Collins, Patrick William Hugh. "Assessing the severity of lower limb ischaemia and the thrombo-inflammatory response to surgery and exercise in peripheral arterial disease." Thesis, Available from the University of Aberdeen Library and Historic Collections Digital Resources. Restricted contains 3rd party material and therefore cannot be made available electronically until Jan. 1, 2012, 2008. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=53369.

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Thesis (M.D.)--Aberdeen University, 2008.
With: Surgical revascularisation in patients with severe limb ischaemia induces a pro-thrombotic state / P. Collins ... et al. Platelets. 2006: 17(5), 311-317. With: A preliminary study on the effects of exercising to a maximum walking distance on platelet and endothelial function in patients with intermittent claudication / P. Collins ... et. Eur. J. Vasc. Endovasc. Surg. 2006: 31, 266-273. Includes bibliographical references.
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Wilson, Alasdair. "The effects of combination antiplatelet therapy on smooth muscle mitogenesis after angioplasty for claudication." Thesis, University of Aberdeen, 2010. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=165239.

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peripheral arterial disease (PAD), a limiting factor in the success of percutaneous transluminal angioplasty (PTA) is the development of restenosis secondary to vascular smooth muscle cell (SMC) proliferation. The aim of this study was to determine the effect of combination antiplatelet therapy on the ability of plasma, from patients undergoing PTA, to stimulate SMCs in vitro. We aimed to investigate the effect of combination treatment on levels of circulating adhesion molecules and factors which mediate SMC proliferation in experimental models. We also sought to demonstrate any association between changes in measured markers and the development of restenosis or vascular events. Methods Fifty patients were randomised to receive clopidogrel or placebo, for thirty days, in addition to their daily 75mg aspirin. To measure proliferative capacity, diluted plasma was incubated with 24h-growth-arrested rat vascular SMCs, and Extracellular-regulated-kinase (ERK)1/2 activation was analysed by Western blotting at baseline, 1-hour pre-PTA, and at 1-hour, 24-hours and 30-days post-PTA. Plasma platelet-derived growth factor (PDGF-BB), soluble (s)E-selectin, sICAM-1 (intracellular adhesion molecule-1) and von Willebrand factor (vWF) were measured by ELISA (Enzyme-linked immunosorbent assay), at the same time-points. Platelet activation was measured by flow cytometry of ADP-stimulated platelet fibrinogen binding at baseline and 1-hour post-PTA. Patients’ notes and all investigations were reviewed for 2 years post-PTA to record restenosis or vascular events. Results Samples were available for all 50 patients at baseline, 1-hour pre-PTA and 1-hour post-PTA timepoints. In this cohort ERK1/2 activation was significantly increased post-PTA in both the aspirin/clopidogrel and aspirin/placebo groups. Those who developed a symptomatic restenosis had a significantly higher level of SMC activation at the 1-hour post-PTA time-point. There was a statistically significant decrease in PDGF-BB, and increase in vWF, following loading with clopidogrel. sICAM-1 levels significantly decreased in the aspirin/placebo group following PTA. ADP-stimulated platelet fibrinogen binding was significantly inhibited by clopidogrel therapy post-PTA. Conclusions This is the first study to show in-vitro ERK1/2 activation (a marker of SMC proliferation) increases post-PTA. Patients developing a symptomatic restenosis had a significantly higher level of SMC activation at the 1-hour post-PTA time-point. Clopidogrel therapy had no significant effect on ERK1/2 activation, although it did reduce PDGF-BB in the larger cohort of patients. Further work is required to evaluate potential therapeutic treatments which may reduce peripheral PTA-induced smooth muscle cell activation.
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Books on the topic "Peripheral vascular diseases"

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1928-, Young Jess R., ed. Peripheral vascular diseases. St. Louis: Mosby Year Book, 1991.

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1928-, Young Jess R., Olin Jeffrey W, and Bartholomew John R, eds. Peripheral vascular diseases. 2nd ed. St. Louis: Mosby, 1996.

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Alonso, Alvaro. Peripheral vascular disease. Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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Alonso, Alvaro. Peripheral vascular disease. Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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R, Mohler Emile, Jaff Michael R, and American College of Physicians, eds. Peripheral arterial disease. Philadelphia: American College of Physicians, 2008.

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Zeitler, Eberhard, ed. Radiology of Peripheral Vascular Diseases. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2.

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1930-, Zeitler E., and Ammann Ernst, eds. Radiology of peripheral vascular diseases. Berlin: Springer, 2000.

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R, Heuser Richard, and Biamino Giancarlo, eds. Peripheral vascular stenting. 2nd ed. London: Taylor & Francis, 2005.

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Lippincott Williams & Wilkins, ed. Peripheral vascular system. 4th ed. [Philadelphia]: Lippincott Williams & Wilkins, 2005.

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Robert, Felix W., ed. Noninvasive diagnosis of peripheral vascular disease. New York: Raven Press, 1988.

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Book chapters on the topic "Peripheral vascular diseases"

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Fagrell, Bengt. "Peripheral Vascular Diseases." In Laser-Doppler Blood Flowmetry, 201–13. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-2083-9_11.

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Kao, LeslieAnn, Deena Chihade, and Guillermo A. Escobar. "Peripheral Vascular Disease." In Anatomic, Physiologic, and Therapeutic Principles of Surgical Diseases, 581–601. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-25596-0_31.

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Narins, Craig R., Jason D. Pacos, and Theodore I. Hirokawa. "Vascular Diseases – Peripheral/Aorta." In Manual of Outpatient Cardiology, 325–46. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-944-4_12.

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Zeitler, Eberhard, D. Raithel, Peter Heilberger, C. Schunn, and David M. Williams. "Aortic Diseases." In Radiology of Peripheral Vascular Diseases, 321–400. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_17.

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Zeitler, Eberhard. "Paraaortic Diseases." In Radiology of Peripheral Vascular Diseases, 401–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_18.

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Zeitler, Eberhard. "Arterosclerotic Diseases." In Radiology of Peripheral Vascular Diseases, 469–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_25.

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Zeitler, Eberhard. "Diabetic Vascular Disease." In Radiology of Peripheral Vascular Diseases, 479–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_26.

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Schellong, Sebastian, and Thomas Schwarz. "Peripheral Venous Diseases: Nonsurgical Approach." In Pan Vascular Medicine, 1497–526. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/978-3-642-56225-9_94.

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Katzen, Barry T., and Gary J. Becker. "The Vascular Center." In Radiology of Peripheral Vascular Diseases, 291–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_15.

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Zeitler, Eberhard. "Blood-Cell Diseases." In Radiology of Peripheral Vascular Diseases, 509–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-56956-2_29.

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Conference papers on the topic "Peripheral vascular diseases"

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Elcin, Huseyn. "EARLY IDENTIFICATION OF THE NEUROLOGICAL COMPLICATIONS OF DIABETES MELLITUS." In International Trends in Science and Technology. RS Global Sp. z O.O., 2021. http://dx.doi.org/10.31435/rsglobal_conf/30032021/7474.

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Diabetes mellitus is still a very common disease in the world and affects the daily lives of patients negatively. Diabetes is also known to be associated with neurological diseases such as peripheral nerve diseases, stroke and dementia. Among these, the most common disease is a peripheral nerve disease, and it has been reported that poor diabetic control increases the risk of development and can be prevented by education of the patients. Vascular dementia is more common in patients with diabetes than Alzheimer's disease, and it is thought that cerebrovascular diseases may berelated to cognitive impairment in diabetes. Although the mechanisms by which diabetes affects the brain are not clearly revealed, it is thought that changes in vascular structure, insulin resistance, glucose toxicity, oxidative stress, accumulation of glycation end products, hypoglycemic episodes and amyloid metabolism are effective.The aim of this article is to describe the neurological complications of diabetes and to emphasize the importance of patient education, good diabetes control and early diagnosis in preventing these complications.
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Aragão, José Aderval, João Víctor Santos Gomes, Larissa Santos Silva, Felipe Matheus Sant'Anna Aragão, Iapunira Catarina Sant'Anna Aragão, Bárbara Costa Lourenço, and Francisco Prado Reis. "Occurrence of anxiety and depression in patients with peripheral arterial obstructive disease." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-112.

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Context: neuropsychiatric diseases correlate with biological risk factors, such as systemic arterial hypertension, diabetes mellitus, stroke and cardiovascular diseases, which can lead to suffering, physical and labor disability of the individual. Within this context, there is a high number of cases of peripheral obstructive arterial disease (poad) within a population affected by some depressive disorder, so if untreated, there is a tendency to have a disability and even mortality of this individual, whether for psychic or physical reasons. From this perspective, the theme is extremely relevant due to few studies that address the correlation between anxiety or depression with poad. Objective: to determine the occurrence of anxiety and depression in patients with poad. Methodology: this was a descriptive, observational, cross-sectional study conducted in the vascular surgery department of a tertiary hospital. All patients with poad included in the study for clinical or surgical treatment were evaluated through clinical history, physical examination and through the ankle-brachial index-itb, where systolic blood pressure was measured in the brachial, posterior tibial and pedicle arteries, with the patient in the supine position and at room temperature to avoid peripheral arterial vasoconstriction. The hospital anxiety and depression scale (hads) was used to assess anxiety and depression. Results: the prevalence of anxiety in patients with poad was 24.3%, while depression was 27.6%. It was observed that there was an association of both anxiety and depression in patients with low monthly family income, smoking, systemic arterial hypertension, and were more prevalent in females. Conclusion: there is a high prevalence of anxiety and depression among patients with poad. These when not adequately treated, increase their secondary risks, mainly by increasing the chances of chronic vascular diseases, often predisposing to suicide.
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Pittella, Erika, Stefano Pisa, Emanuele Piuzzi, Emanuele Rizzuto, and Zaccaria Del Prete. "Combined impedance plethysmography and spectroscopy for the diagnosis of diseases of peripheral vascular system." In 2017 IEEE International Symposium on Medical Measurements and Applications (MeMeA). IEEE, 2017. http://dx.doi.org/10.1109/memea.2017.7985904.

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Karamchandani, S., M. Y. Dixit, R. K. Jain, and M. Bhowmick. "Application of Neural Networks in the Interpretation of Impedance Cardiovasograms for the Diagnoses of Peripheral Vascular Diseases." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1616256.

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Terminelli, L., F. Carpignano, J. M. May, S. Merlo, and P. A. Kyriacou. "Photoplethysmography and electrocardiography for real time evaluation of pulse transit time A diagnostic marker of peripheral vascular diseases." In 2014 Fotonica AEIT Italian Conference on Photonics Technologies (Fotonica AEIT). IEEE, 2014. http://dx.doi.org/10.1109/fotonica.2014.6843951.

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Ulutin, O. N., G. Cizmeci, U. Cornelli, and J. Fareed. "CLINICAL AND LABORATORY STUDIES ON THE EFFECTIVENESS OF DEFIBROTIDE IN PERIPHERAL VASCULAR DISEASE OF ATHEROSCLEROTIC ORIGIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643148.

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Defibrotide is a derivative of polydeoxyribonucleotide extracted from bovine lung which has been found to produce profound activation of endothelial cells as evidenced by the increase of tissue plasminogen activator (t-PA) and prostacyclin levels in animal models. Administration of a 600 mg dose daily for six weeks to patients with peripheral obliterative diseases resulted in significant clinical improvements in post-treatment exercise tests and radionuclide arteriography. Results were consistent with improvements in the circulation. Ex vivo blood analysis showed marked increases in the production of t-PA and prostacyclin levels. Other laboratory tests were also suggestive of activated endothelial states. Platelet C-AMP levels were increased, whereas MDA and TXB2 levels were reduced. No systemic anticoagulation was observed in terms of alterations of the global coagulant tests (PT, APTT, etc.). These studies suggest that Defibrotide mainly acts via the modulation of the endothelial function and acts in a novel fashion in contrast to other drugs used in this area.
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Kuprina, N. I., E. V. Ulanovskaya, and V. V. Shilov. "CHANGES IN THE FOREARM VESSELS OF PATIENTS WITH VIBRATION DISEASE AFTER CORONAVIRUS INFECTION." In The 16th «OCCUPATION and HEALTH» Russian National Congress with International Participation (OHRNC-2021). FSBSI “IRIOH”, 2021. http://dx.doi.org/10.31089/978-5-6042929-2-1-2021-1-300-303.

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Abstract. A lot of scientific literature has already been written on the study of the consequences of a new coronavirus infection. The real long-term effects of this disease on population health are yet to be studied in the coming years. Vibration disease is a leading occupational pathology in the Russian Federation, characterized by vegetative-vascular disorders of the extremities and is manifested by cold extremities, cyanosis, paresthesias, and a violation of regional blood circulation. It is necessary to take into account the concomitant chronic diseases in patients who are particularly dangerous in the post-ovoid period. The aim of the study was to study the features of hemodynamics in patients with vibration disease after a new coronavirus infection. In the clinic of occupational pathology, 28 patients were examined with a previously established diagnosis of vibration disease after a coronavirus infection. Ultrasound examination of the arteries and veins of the upper extremities was performed. In patients with ultrasound of the vessels of the upper extremities, moderate expansion of the radial and ulnar veins, insufficiency of the valvular apparatus during functional tests, and increased venous outflow were revealed. There was also an increase in peripheral vascular resistance, which indicates violations of the tonic properties of the vessels of the upper extremities and violations of vasodilation. Inference. In patients with vibration disease in the post-ovoid period, ultrasound examination reveals violations of the tonic properties of blood vessels, venous dyscirculation in the veins of the forearm.
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Kerdjoudj, H., V. Moby, N. Berthelemy, J. C. Voegel, P. Menu, and J. F. Stoltz. "Use of Polyelectrolyte Films in Vascular Tissue Engineering." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192538.

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Vascular diseases with their high morbidity and mortality are a major challenge for medical science, engaging the best minds in modern medicine. The development of antithrombogenic surfaces still remains a huge challenge in the vascular tissue engineering field. Various researchers have expanded surface coating procedures allowing endothelial cells (EC) adhesion and retention on vascular substitutes or by incorporating some of the mechanisms employed by vascular endothelial cells i.e. heparin. The short in vivo patency of these grafts is related. Our group study evaluates a new surface modification based on polyelectrolyte building. The layer by layer self assembly and the result in polyelectrolyte multilayer films (PEM) became also in a recent past a challenging, simple and versatile way to engineer surfaces with highly specific properties. Previous studies indicated that the poly(sodium-4 styrene sulfonate)/poly (allylamine hydrochloride) PSS/PAH multilayered films when ended by PAH induce strong adhesion and retention of mature EC which spread and keep their phenotype as well on glass [1,2], on expanded polytetrafluoroethylene ePTFE [3] and on cryopreserved arteries [4,5]. The mechanical properties (compliance), leading to early intimal hyperplasia and graft failure, were lost after artery cryopreservation. We have demonstrated the compliance restoration of PEM treated cryopreserved close to native arteries [5]. The use of an autologous EC source avoids the immunological rejections of the grafts. With an autologous origin, high proliferation capacity and potentialities to proliferate and differentiate into matures EC, the endothelial progenitor cells (EPC) have raised huge interest and offer new opportunities in vascular engineering. Currents protocols for isolation and differentiation of EPC from peripheral blood requires at least 1 month to observe an endothelium-like morphology and about 2 months for confluent EC monolayer. The EPC cultivated on PEM treated glasses showed a monolayer development after only 14 days of culture. The morphological appearance and mature phenotype markers expression and repartition of the monolayer cells are close to mature EC [6]. These main results have led to French patent deposit in June 2007[7].
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De Crée, J., V. Roels, and H. Verhaegen. "HYPERACTIVITY OF PLATELETS TO 5-HYDROXYTRYPTAMINE IN PATIENTS WITH CARDIOVASCULAR DISEASES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643472.

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Controversy still exists about the exact role of platelets in the pathogenesis of atherosclerosis. However, patients with cardiovascular (CV) diseases have been reported to have enhanced platelet activity and to show hyperaggregability in response to common aggregators. Very little is known on 5-hydroxytryptamine (5-HT)-induced platelet aggregation in these patients. In a previous preliminary study we observed an increased sensitivity of platelets in response to 5-HT in patients with CV diseases. We further showed that ketanserin, a selective 5-HT receptor antagonist both on platelets and vascular tissue, abolished the 5-HT dependent hyperreactivity of platelets in patients with CV diseases. In a prospective study we investigated platelet aggregation in response to 5-HT at 2 x 10-5Mol in 405 patients with various CV diseases as compared with an age-matched control group of 110 apparently healthy donors. Evaluation of the results was based on the presence of a second irreversible aggregation in response to 5-HT. The control subjects responded to 5-HT with a shape change and a weak, reversible aggregation, except for 9 of the 110 volunteers, where a second irreversible wave occurred (8%). In contrast, it was found that 119 out of 405 patients with CV diseases had a biphasic irreversible aggregation (29%) (Chi-square test : p < 0.0001). From these 405 patients 129 patients suffered from an acute myocardial infarction (AMI), between 4 and 14 days after the onset of symptoms, 78 patients suffered from ischemic heart disease (IHD), 99 patients from peripheral arterial obstructive disease (PAOD) and 99 patients from diabetes, without clinical symptoms of atherosclerosis. A secondary irreversible platelet aggregation to 5-HT was observed in 36% of patients with AMI, in 22% of patients with IHD, in 25% of patients with PAOD and in 31% of patients with diabetes, all the subgroups being significantly different from the control subjects (p <0.01). These findings suggest that platelets may play a role in the propagation and manifestations of CV diseases and that in diabetes the enhanced platelet activity may be a contributing risk factor in de development of atherosclerosis. Finally, since 5-HT is a potent mediator of vasospasm, treatment with ketanserin might be of therapeutic value in atherosclerosis, where platelet activation is thought to be involved.
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Swaminathan, Ganesh, Suraj Thyagaraj, Francis Loth, Susan McCormick, and Hisham Bassiouny. "Influence of Intermittent Pneumatic Compression on Wall Shear Stress and Nitric Oxide Levels." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53670.

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Wall shear stress (WSS) in blood vessels has been shown to play an important role in the development of atherosclerosis. In particular, regions of low and oscillating WSS have been shown to correlate with the localization of atherosclerosis. Thus, we hypothesize that increasing the WSS for patients with peripheral vascular diseases (PVD) will either reduce PVD severity or slow its progression. We analyzed WSS changes from a study by Delis et al. on 32 limbs of PVD patients [1]. Results show that intermittent pneumatic compression (IPC) increases mean WSS by 170% and 240% in PVD patients and healthy subjects, respectively. Peak WSS was found to increase by 93% and 40% in PVD patients and healthy subjects, respectively. In addition, we examined changes in NOX level with use of IPC on five limbs from PVD patients. Our study demonstrated increased NOx levels in subjects after IPC. Further research is needed to determine the benefits of IPC for PVD patients.
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