Relevant bibliographies by topics / Perisynaptic Schwann cell

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Academic literature on the topic 'Perisynaptic Schwann cell'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Perisynaptic Schwann cell"

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Sugiura, Yoshie, and Weichun Lin. "Neuron–glia interactions: the roles of Schwann cells in neuromuscular synapse formation and function." Bioscience Reports 31, no. 5 (2011): 295–302. http://dx.doi.org/10.1042/bsr20100107.

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The NMJ (neuromuscular junction) serves as the ultimate output of the motor neurons. The NMJ is composed of a presynaptic nerve terminal, a postsynaptic muscle and perisynaptic glial cells. Emerging evidence has also demonstrated an existence of perisynaptic fibroblast-like cells at the NMJ. In this review, we discuss the importance of Schwann cells, the glial component of the NMJ, in the formation and function of the NMJ. During development, Schwann cells are closely associated with presynaptic nerve terminals and are required for the maintenance of the developing NMJ. After the establishment
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Weis, J., S. M. Fine, C. David, S. Savarirayan, and J. R. Sanes. "Integration site-dependent expression of a transgene reveals specialized features of cells associated with neuromuscular junctions." Journal of Cell Biology 113, no. 6 (1991): 1385–97. http://dx.doi.org/10.1083/jcb.113.6.1385.

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After skeletal muscle is denervated, fibroblasts near neuromuscular junctions proliferate more than fibroblasts distant from synaptic sites, and they accumulate adhesive molecules such as tenascin (Gatchalian, C. L., M. Schachner, and J. R. Sanes. 1989. J. Cell Biol. 108:1873-1890). This response could reflect signals that arise perisynaptically after denervation, preexisting differences between perisynaptic and extrasynaptic fibroblasts, or both. Here, we describe a line of transgenic mice in which patterns of transgene expression provide direct evidence for differences between perisynaptic a
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Alvarez-Suarez, Paloma, Marta Gawor, and Tomasz J. Prószyński. "Perisynaptic schwann cells - The multitasking cells at the developing neuromuscular junctions." Seminars in Cell & Developmental Biology 104 (August 2020): 31–38. http://dx.doi.org/10.1016/j.semcdb.2020.02.011.

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Young, Paul, Jing Nie, Xueyong Wang, C. Jane McGlade, Mark M. Rich, and Guoping Feng. "LNX1 is a perisynaptic Schwann cell specific E3 ubiquitin ligase that interacts with ErbB2." Molecular and Cellular Neuroscience 30, no. 2 (2005): 238–48. http://dx.doi.org/10.1016/j.mcn.2005.07.015.

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Brill, Monika S., Jeff W. Lichtman, Wesley Thompson, Yi Zuo, and Thomas Misgeld. "Spatial constraints dictate glial territories at murine neuromuscular junctions." Journal of Cell Biology 195, no. 2 (2011): 293–305. http://dx.doi.org/10.1083/jcb.201108005.

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Schwann cells (SCs), the glial cells of the peripheral nervous system, cover synaptic terminals, allowing them to monitor and modulate neurotransmission. Disruption of glial coverage leads to axon degeneration and synapse loss. The cellular mechanisms that establish and maintain this coverage remain largely unknown. To address this, we labeled single SCs and performed time-lapse imaging experiments. Adult terminal SCs are arranged in static tile patterns, whereas young SCs dynamically intermingle. The mechanism of developmental glial segregation appears to be spatial competition, in which glia
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Covault, J., and J. R. Sanes. "Distribution of N-CAM in synaptic and extrasynaptic portions of developing and adult skeletal muscle." Journal of Cell Biology 102, no. 3 (1986): 716–30. http://dx.doi.org/10.1083/jcb.102.3.716.

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Previous studies of denervated and cultured muscle have shown that the expression of the neural cell adhesion molecule (N-CAM) in muscle is regulated by the muscle's state of innervation and that N-CAM might mediate some developmentally important nerve-muscle interactions. As a first step in learning whether N-CAM might regulate or be regulated by nerve-muscle interactions during normal development, we have used light and electron microscopic immunohistochemical methods to study its distribution in embryonic, perinatal, and adult rat muscle. In embryonic muscle, N-CAM is uniformly present on t
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Kapitza, Christopher, Rittika Chunder, Anja Scheller, et al. "Murine Esophagus Expresses Glial-Derived Central Nervous System Antigens." International Journal of Molecular Sciences 22, no. 6 (2021): 3233. http://dx.doi.org/10.3390/ijms22063233.

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Multiple sclerosis (MS) has been considered to specifically affect the central nervous system (CNS) for a long time. As autonomic dysfunction including dysphagia can occur as accompanying phenomena in patients, the enteric nervous system has been attracting increasing attention over the past years. The aim of this study was to identify glial and myelin markers as potential target structures for autoimmune processes in the esophagus. RT-PCR analysis revealed glial fibrillary acidic protein (GFAP), proteolipid protein (PLP), and myelin basic protein (MBP) expression, but an absence of myelin oli
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Dowdall, M., A. Green, and C. Richardson. "Dynamic imaging of functional nerve terminals and Schwann cells in presynaptic 'nerve plates' isolated from the skate electric organ." Journal of Experimental Biology 200, no. 1 (1997): 161–71. http://dx.doi.org/10.1242/jeb.200.1.161.

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The cholinergic innervation and its glial support were isolated in a functional state from the electric organ of the skate (Raja species) using a combined enzymatic and mechanical dissociation technique. Examination using light and electron microscopy showed that this 'nerve plate' is a disc-shaped structure several hundred micrometres in diameter consisting of a dense plexus of nerve terminals attached to branching nerve fibrils with numerous associated myelinating and perisynaptic Schwann cells. In unfixed nerve plates, depolarisation and Ca2+-dependent staining of the nerve terminals was se
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Ko, Chien-Ping, and Richard Robitaille. "Perisynaptic Schwann Cells at the Neuromuscular Synapse: Adaptable, Multitasking Glial Cells." Cold Spring Harbor Perspectives in Biology 7, no. 10 (2015): a020503. http://dx.doi.org/10.1101/cshperspect.a020503.

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Duregotti, Elisa, Samuele Negro, Michele Scorzeto, et al. "Mitochondrial alarmins released by degenerating motor axon terminals activate perisynaptic Schwann cells." Proceedings of the National Academy of Sciences 112, no. 5 (2015): E497—E505. http://dx.doi.org/10.1073/pnas.1417108112.

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An acute and highly reproducible motor axon terminal degeneration followed by complete regeneration is induced by some animal presynaptic neurotoxins, representing an appropriate and controlled system to dissect the molecular mechanisms underlying degeneration and regeneration of peripheral nerve terminals. We have previously shown that nerve terminals exposed to spider or snake presynaptic neurotoxins degenerate as a result of calcium overload and mitochondrial failure. Here we show that toxin-treated primary neurons release signaling molecules derived from mitochondria: hydrogen peroxide, mi
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Dissertations / Theses on the topic "Perisynaptic Schwann cell"

1

Xin, Ling. "Stability of the frog motor nerve terminal roles of perisynaptic Schwann cells and muscle fibers /." Connect to this title online, 2008. http://scholarworks.umass.edu/theses/101/.

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Provost, Frédéric. "Analyse comparative pour comprendre la résistance des jonctions neuromusculaires des muscles extraoculaires dans la sclérose latérale amyotrophique." Thesis, 2020. http://hdl.handle.net/1866/24531.

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La sclérose latérale amyotrophique (SLA) est une maladie touchant spécifiquement les motoneurones (MN) qui se caractérise par la perte précoce des jonctions neuromusculaires (JNMs) et menant à une paralysie musculaire. La dénervation des JNMs des muscles squelettiques se produit en amont de la mort des MN de la moelle épinière. Des études récentes publiées ont révélé une altération de la transmission synaptique, une instabilité de la morphologie des JNMs ainsi que des mécanismes de réparations de la JNMs inappropriés dans le modèle SOD1, et ce avant l’apparition des symptômes moteurs. De maniè
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Arbour, Danielle. "Altérations des cellules de Schwann périsynaptiques à la jonction neuromusculaire : implications pour la sclérose latérale amyotrophique." Thèse, 2016. http://hdl.handle.net/1866/19334.

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Moeckel, Cole Stephanie Anne. "Organization and maintenance of the motor nerve terminal: Roles for presynaptic actin and perisynaptic Schwann cells." 2008. https://scholarworks.umass.edu/dissertations/AAI3336977.

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At the adult frog neuromuscular junctions (Rana pipien and Rana catesbiana), F-actin microfilaments are enriched in the nonrelease domains of nerve terminals, outside the vesicle-rich release sites. The development of this defined F-actin cytoskeleton may be critical for nerve terminal function as microfilaments may play a role in synaptic vesicle release and recycling and/or synaptic maintenance. I used cutaneous pectoris muscles of adult frogs (Rana pipiens) and bullfrog larvae (Rana catesbiana) stained with markers of synaptic and cytoskeletal components to ask how elements of the neuronal
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Bélair, Eve-Lyne. "Plasticité présynaptique et gliale à long-terme en réponse à un changement chronique de l’activité synaptique,à la jonction neuromusculaire d’amphibien." Thèse, 2008. http://hdl.handle.net/1866/2780.

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La plasticité synaptique est une importante propriété du système nerveux, impliquée dans l’intégration de l’information. Cette plasticité a généralement été décrite par des changements aux niveaux pré et postsynaptiques. Notamment, l’efficacité présynaptique, soit la probabilité de libération de neurotransmetteurs associée au contenu quantique d’une synapse, peut être augmentée ou diminuée selon l’activité antérieure de la synapse. Malgré cette caractérisation, les mécanismes à l’origine de la détermination de l’efficacité présynaptique demeurent obscurs. Également, la plasticité synaptiqu
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Moustaine, Ayman. "Implication des cellules gliales dans la modulation de l’activité synaptique à la jonction neuromusculaire sénescente." Thèse, 2018. http://hdl.handle.net/1866/22147.

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Krief, Noam. "L’altération des interactions neurone-glie à la jonction neuromusculaire de souris âgées." Thèse, 2014. http://hdl.handle.net/1866/12056.

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Durant le vieillissement, l’ensemble des fonctions de l’organisme se détériore, que ce soit aussi bien au niveau moteur que cognitif. Le vieillissement s’accompagne d’une diminution de la force, ainsi que de la masse musculaire. Des études récentes tendent à montrer que cette perte de masse musculaire que l’on appelle sarcopénie aurait pour origine un dérèglement de la jonction neuromusculaire. Les changements au niveau du présynaptique et du post synaptiques lors du vieillissement normal font l’objet de plusieurs études, mais les changements relatifs aux cellules de Schwann périsynaptique son
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Martineau, Éric. "Dégénérescence locale et réparation anormale de la jonction neuromusculaire dans un modèle de la sclérose latérale amyotrophique." Thèse, 2018. http://hdl.handle.net/1866/22602.

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Journal articles
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