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Journal articles on the topic 'Perisynaptic Schwann cell'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Sugiura, Yoshie, and Weichun Lin. "Neuron–glia interactions: the roles of Schwann cells in neuromuscular synapse formation and function." Bioscience Reports 31, no. 5 (2011): 295–302. http://dx.doi.org/10.1042/bsr20100107.

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The NMJ (neuromuscular junction) serves as the ultimate output of the motor neurons. The NMJ is composed of a presynaptic nerve terminal, a postsynaptic muscle and perisynaptic glial cells. Emerging evidence has also demonstrated an existence of perisynaptic fibroblast-like cells at the NMJ. In this review, we discuss the importance of Schwann cells, the glial component of the NMJ, in the formation and function of the NMJ. During development, Schwann cells are closely associated with presynaptic nerve terminals and are required for the maintenance of the developing NMJ. After the establishment

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2

Weis, J., S. M. Fine, C. David, S. Savarirayan, and J. R. Sanes. "Integration site-dependent expression of a transgene reveals specialized features of cells associated with neuromuscular junctions." Journal of Cell Biology 113, no. 6 (1991): 1385–97. http://dx.doi.org/10.1083/jcb.113.6.1385.

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After skeletal muscle is denervated, fibroblasts near neuromuscular junctions proliferate more than fibroblasts distant from synaptic sites, and they accumulate adhesive molecules such as tenascin (Gatchalian, C. L., M. Schachner, and J. R. Sanes. 1989. J. Cell Biol. 108:1873-1890). This response could reflect signals that arise perisynaptically after denervation, preexisting differences between perisynaptic and extrasynaptic fibroblasts, or both. Here, we describe a line of transgenic mice in which patterns of transgene expression provide direct evidence for differences between perisynaptic a

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3

Alvarez-Suarez, Paloma, Marta Gawor, and Tomasz J. Prószyński. "Perisynaptic schwann cells - The multitasking cells at the developing neuromuscular junctions." Seminars in Cell & Developmental Biology 104 (August 2020): 31–38. http://dx.doi.org/10.1016/j.semcdb.2020.02.011.

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4

Young, Paul, Jing Nie, Xueyong Wang, C. Jane McGlade, Mark M. Rich, and Guoping Feng. "LNX1 is a perisynaptic Schwann cell specific E3 ubiquitin ligase that interacts with ErbB2." Molecular and Cellular Neuroscience 30, no. 2 (2005): 238–48. http://dx.doi.org/10.1016/j.mcn.2005.07.015.

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5

Brill, Monika S., Jeff W. Lichtman, Wesley Thompson, Yi Zuo, and Thomas Misgeld. "Spatial constraints dictate glial territories at murine neuromuscular junctions." Journal of Cell Biology 195, no. 2 (2011): 293–305. http://dx.doi.org/10.1083/jcb.201108005.

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Schwann cells (SCs), the glial cells of the peripheral nervous system, cover synaptic terminals, allowing them to monitor and modulate neurotransmission. Disruption of glial coverage leads to axon degeneration and synapse loss. The cellular mechanisms that establish and maintain this coverage remain largely unknown. To address this, we labeled single SCs and performed time-lapse imaging experiments. Adult terminal SCs are arranged in static tile patterns, whereas young SCs dynamically intermingle. The mechanism of developmental glial segregation appears to be spatial competition, in which glia

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6

Covault, J., and J. R. Sanes. "Distribution of N-CAM in synaptic and extrasynaptic portions of developing and adult skeletal muscle." Journal of Cell Biology 102, no. 3 (1986): 716–30. http://dx.doi.org/10.1083/jcb.102.3.716.

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Previous studies of denervated and cultured muscle have shown that the expression of the neural cell adhesion molecule (N-CAM) in muscle is regulated by the muscle's state of innervation and that N-CAM might mediate some developmentally important nerve-muscle interactions. As a first step in learning whether N-CAM might regulate or be regulated by nerve-muscle interactions during normal development, we have used light and electron microscopic immunohistochemical methods to study its distribution in embryonic, perinatal, and adult rat muscle. In embryonic muscle, N-CAM is uniformly present on t

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7

Kapitza, Christopher, Rittika Chunder, Anja Scheller, et al. "Murine Esophagus Expresses Glial-Derived Central Nervous System Antigens." International Journal of Molecular Sciences 22, no. 6 (2021): 3233. http://dx.doi.org/10.3390/ijms22063233.

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Multiple sclerosis (MS) has been considered to specifically affect the central nervous system (CNS) for a long time. As autonomic dysfunction including dysphagia can occur as accompanying phenomena in patients, the enteric nervous system has been attracting increasing attention over the past years. The aim of this study was to identify glial and myelin markers as potential target structures for autoimmune processes in the esophagus. RT-PCR analysis revealed glial fibrillary acidic protein (GFAP), proteolipid protein (PLP), and myelin basic protein (MBP) expression, but an absence of myelin oli

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8

Dowdall, M., A. Green, and C. Richardson. "Dynamic imaging of functional nerve terminals and Schwann cells in presynaptic 'nerve plates' isolated from the skate electric organ." Journal of Experimental Biology 200, no. 1 (1997): 161–71. http://dx.doi.org/10.1242/jeb.200.1.161.

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The cholinergic innervation and its glial support were isolated in a functional state from the electric organ of the skate (Raja species) using a combined enzymatic and mechanical dissociation technique. Examination using light and electron microscopy showed that this 'nerve plate' is a disc-shaped structure several hundred micrometres in diameter consisting of a dense plexus of nerve terminals attached to branching nerve fibrils with numerous associated myelinating and perisynaptic Schwann cells. In unfixed nerve plates, depolarisation and Ca2+-dependent staining of the nerve terminals was se

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9

Ko, Chien-Ping, and Richard Robitaille. "Perisynaptic Schwann Cells at the Neuromuscular Synapse: Adaptable, Multitasking Glial Cells." Cold Spring Harbor Perspectives in Biology 7, no. 10 (2015): a020503. http://dx.doi.org/10.1101/cshperspect.a020503.

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10

Duregotti, Elisa, Samuele Negro, Michele Scorzeto, et al. "Mitochondrial alarmins released by degenerating motor axon terminals activate perisynaptic Schwann cells." Proceedings of the National Academy of Sciences 112, no. 5 (2015): E497—E505. http://dx.doi.org/10.1073/pnas.1417108112.

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An acute and highly reproducible motor axon terminal degeneration followed by complete regeneration is induced by some animal presynaptic neurotoxins, representing an appropriate and controlled system to dissect the molecular mechanisms underlying degeneration and regeneration of peripheral nerve terminals. We have previously shown that nerve terminals exposed to spider or snake presynaptic neurotoxins degenerate as a result of calcium overload and mitochondrial failure. Here we show that toxin-treated primary neurons release signaling molecules derived from mitochondria: hydrogen peroxide, mi

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11

Voigt, Tilman, Wolfgang Dauber, and Ulrike Kohler. "Perisynaptic Schwann cells of the vertebrate motor endplate bear modified cilia." Microscopy Research and Technique 63, no. 3 (2004): 149–54. http://dx.doi.org/10.1002/jemt.20023.

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12

Jahromi, Babak S., Richard Robitaille, and Milton P. Charlton. "Transmitter release increases intracellular calcium in perisynaptic schwann cells in situ." Neuron 8, no. 6 (1992): 1069–77. http://dx.doi.org/10.1016/0896-6273(92)90128-z.

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13

Bourque, Marie-Josée, and Richard Robitaille. "Endogenous peptidergic modulation of perisynaptic Schwann cells at the frog neuromuscular junction." Journal of Physiology 512, no. 1 (1998): 197–209. http://dx.doi.org/10.1111/j.1469-7793.1998.197bf.x.

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14

Georgiou, John, and Milton P. Charlton. "Non-myelin-forming perisynaptic Schwann cells express protein zero and myelin-associated glycoprotein." Glia 27, no. 2 (1999): 101–9. http://dx.doi.org/10.1002/(sici)1098-1136(199908)27:2<101::aid-glia1>3.0.co;2-h.

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15

Negro, Samuele, Francesca Lessi, Elisa Duregotti та ін. "CXCL 12α/ SDF ‐1 from perisynaptic Schwann cells promotes regeneration of injured motor axon terminals". EMBO Molecular Medicine 9, № 8 (2017): 1000–1010. http://dx.doi.org/10.15252/emmm.201607257.

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16

Koirala, Samir, Huahong Qiang, and Chien-Ping Ko. "Reciprocal interactions between perisynaptic Schwann cells and regenerating nerve terminals at the frog neuromuscular junction." Journal of Neurobiology 44, no. 3 (2000): 343–60. http://dx.doi.org/10.1002/1097-4695(20000905)44:3<343::aid-neu5>3.0.co;2-o.

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17

Cunningham, Madeleine E., Gavin R. Meehan, Sophie Robinson, Denggao Yao, Rhona McGonigal, and Hugh J. Willison. "Perisynaptic Schwann cells phagocytose nerve terminal debris in a mouse model of Guillain‐Barré syndrome." Journal of the Peripheral Nervous System 25, no. 2 (2020): 143–51. http://dx.doi.org/10.1111/jns.12373.

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18

Robitaille, Richard, Babak S. Jahromi, and Milton P. Charlton. "Muscarinic Ca2+responses resistant to muscarinic antagonists at perisynaptic schwann cells of the frog neuromuscular junction." Journal of Physiology 504, no. 2 (1997): 337–47. http://dx.doi.org/10.1111/j.1469-7793.1997.337be.x.

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19

Noronha‐Matos, José B., Laura Oliveira, Ana R. Peixoto та ін. "Nicotinic α7 receptor‐induced adenosine release from perisynaptic Schwann cells controls acetylcholine spillover from motor endplates". Journal of Neurochemistry 154, № 3 (2020): 263–83. http://dx.doi.org/10.1111/jnc.14975.

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20

Herrera, Albert A., Huahong Qiang, and Chien-Ping Ko. "The role of perisynaptic Schwann cells in development of neuromuscular junctions in the frog (xenopus laevis)." Journal of Neurobiology 45, no. 4 (2000): 237–54. http://dx.doi.org/10.1002/1097-4695(200012)45:4<237::aid-neu5>3.0.co;2-j.

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21

Tam, S. L., and T. Gordon. "Neuromuscular activity impairs axonal sprouting in partially denervated muscles by inhibiting bridge formation of perisynaptic Schwann cells." Journal of Neurobiology 57, no. 2 (2003): 221–34. http://dx.doi.org/10.1002/neu.10276.

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22

Auld, Daniel S., and Richard Robitaille. "Perisynaptic Schwann Cells at the Neuromuscular Junction: Nerve- and Activity-Dependent Contributions to Synaptic Efficacy, Plasticity, and Reinnervation." Neuroscientist 9, no. 2 (2003): 144–57. http://dx.doi.org/10.1177/1073858403252229.

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23

Halstead, S. K. "Anti-disialoside antibodies kill perisynaptic Schwann cells and damage motor nerve terminals via membrane attack complex in a murine model of neuropathy." Brain 127, no. 9 (2004): 2109–23. http://dx.doi.org/10.1093/brain/awh231.

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24

Heredia, Dante J., Cheng-Yuan Feng, Grant W. Hennig, Robert B. Renden, and Thomas W. Gould. "Activity-induced Ca2+ signaling in perisynaptic Schwann cells of the early postnatal mouse is mediated by P2Y1 receptors and regulates muscle fatigue." eLife 7 (January 31, 2018). http://dx.doi.org/10.7554/elife.30839.

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Perisynaptic glial cells respond to neural activity by increasing cytosolic calcium, but the significance of this pathway is unclear. Terminal/perisynaptic Schwann cells (TPSCs) are a perisynaptic glial cell at the neuromuscular junction that respond to nerve-derived substances such as acetylcholine and purines. Here, we provide genetic evidence that activity-induced calcium accumulation in neonatal TPSCs is mediated exclusively by one subtype of metabotropic purinergic receptor. In P2ry1 mutant mice lacking these responses, postsynaptic, rather than presynaptic, function was altered in respon

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25

Castro, Ryan, Thomas Taetzsch, Sydney K. Vaughan, et al. "Specific labeling of synaptic schwann cells reveals unique cellular and molecular features." eLife 9 (June 25, 2020). http://dx.doi.org/10.7554/elife.56935.

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Perisynaptic Schwann cells (PSCs) are specialized, non-myelinating, synaptic glia of the neuromuscular junction (NMJ), that participate in synapse development, function, maintenance, and repair. The study of PSCs has relied on an anatomy-based approach, as the identities of cell-specific PSC molecular markers have remained elusive. This limited approach has precluded our ability to isolate and genetically manipulate PSCs in a cell specific manner. We have identified neuron-glia antigen 2 (NG2) as a unique molecular marker of S100β+ PSCs in skeletal muscle. NG2 is expressed in Schwann cells alr

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