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Relevant bibliographies by topics / Peritonitis model in mice

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Academic literature on the topic 'Peritonitis model in mice'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Peritonitis model in mice"

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Goswami, Manish, Deepak Sharma, Nazir M. Khan, Rahul Checker, Santosh Kumar Sandur, and Narendra Jawali. "Antioxidant supplementation enhances bacterial peritonitis in mice by inhibiting phagocytosis." Journal of Medical Microbiology 63, no. 3 (2014): 355–66. http://dx.doi.org/10.1099/jmm.0.067173-0.

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Antioxidants are known to exhibit numerous health benefits including anti-ageing, anti-apoptotic and immuno-stimulatory effects. However, we present the data showing counterproductive effects of therapeutically relevant antioxidants on bacterial clearance by the immune system in a murine peritonitic model. The antioxidants ascorbic acid, glutathione and N-acetylcysteine augmented morbidity and mortality in mice carrying Eshcerichia coli-induced acute bacterial peritonitis. Treatment of peritonitic mice with antioxidants significantly increased their bacterial load in the range of 0.3–2 logs. A

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Ishii, Yasuo, Tokihiko Sawada, Akira Shimizu, et al. "An experimental sclerosing encapsulating peritonitis model in mice." Nephrology Dialysis Transplantation 16, no. 6 (2001): 1262–66. http://dx.doi.org/10.1093/ndt/16.6.1262.

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Clark, Jessica A., Heng Gan, Alexandr J. Samocha, Amy C. Fox, Timothy G. Buchman, and Craig M. Coopersmith. "Enterocyte-specific epidermal growth factor prevents barrier dysfunction and improves mortality in murine peritonitis." American Journal of Physiology-Gastrointestinal and Liver Physiology 297, no. 3 (2009): G471—G479. http://dx.doi.org/10.1152/ajpgi.00012.2009.

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Systemic administration of epidermal growth factor (EGF) decreases mortality in a murine model of septic peritonitis. Although EGF can have direct healing effects on the intestinal mucosa, it is unknown whether the benefits of systemic EGF in peritonitis are mediated through the intestine. Here, we demonstrate that enterocyte-specific overexpression of EGF is sufficient to prevent intestinal barrier dysfunction and improve survival in peritonitis. Transgenic FVB/N mice that overexpress EGF exclusively in enterocytes ( IFABP-EGF) and wild-type (WT) mice were subjected to either sham laparotomy

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Ni, Jie, Yvette Cnops, Rachel M. McLoughlin, Nicholas Topley, and Olivier Devuyst. "Inhibition of Nitric Oxide Synthase Reverses Permeability Changes in a Mouse Model of Acute Peritonitis." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 25, no. 3_suppl (2005): 11–14. http://dx.doi.org/10.1177/089686080502503s03.

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Acute peritonitis is the most frequent complication of peritoneal dialysis. Previous studies have suggested a major role for nitric oxide (NO) in the permeability changes and loss of ultrafiltration induced by acute peritonitis. In this study, we further investigated the potential role of NO in a mouse model of peritonitis induced by Escherichia coli lipopolysaccharide (LPS). A 2-hour peritoneal equilibration test was performed in control and LPS-treated mice using 7% glucose dialysate supplemented or not with the NO synthase inhibitor NG-nitro-L-arginine methyl ester (L-NAME). The levels of N

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Liu, Shaoguang, Shaotong Zhang, Yulong Sun, and Wence Zhou. "Transcriptomics Changes in the Peritoneum of Mice with Lipopolysaccharide-Induced Peritonitis." International Journal of Molecular Sciences 22, no. 23 (2021): 13008. http://dx.doi.org/10.3390/ijms222313008.

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Peritonitis caused by LPS is a severe clinical challenge, which causes organ damage and death. However, the mechanism of LPS-induced peritonitis has not been fully revealed yet. Here, we investigated the transcriptome profile of the peritoneal tissue of LPS-induced peritonitis in mice. A model of LPS-induced peritonitis in mice was established (LPS 10 mg/kg, i.p.), and the influence of TAK 242 (TLR4 inhibitor) on the level of inflammatory cytokines in mouse peritoneal lavage fluid was investigated by using an ELISA test. Next, the peritoneal tissues of the three groups of mice (Control, LPS, a

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Steinhauser, Matthew L., Cory M. Hogaboam, Nickolas W. Lukacs, Robert M. Strieter, and Steven L. Kunkel. "Multiple Roles for IL-12 in a Model of Acute Septic Peritonitis." Journal of Immunology 162, no. 9 (1999): 5437–43. http://dx.doi.org/10.4049/jimmunol.162.9.5437.

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Abstract The present study addressed the role of IL-12 in a murine model of septic peritonitis, induced by cecal ligation and puncture (CLP). Although CLP surgery induced IL-12 production at 6 and 24 h after surgery, IL-12 immunoneutralization was clearly deleterious in this model: 54% of CLP mice receiving preimmune serum survived, whereas mice administered IL-12 antisera prior to CLP experienced a 25% survival rate. IL-12 immunoneutralization not only led to increased mortality, but also appeared to promote a shift away from IL-12 and IFN-γ, in favor of IL-10. This cytokine shift corresponde

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Lan, Jing, Hong Zhang, Hui Zhao, et al. "Cord Blood Natural Killer Cells Inhibit Sepsis Caused by Feces-Induced Acute Peritonitis via Increasing Endothelium Integrity." Cell Transplantation 31 (January 2022): 096368972210902. http://dx.doi.org/10.1177/09636897221090257.

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Sepsis is associated with acute peritonitis, which can be induced by lipopolysaccharide exposure and feces. Generally, lipopolysaccharide induces mono-microbial peritonitis, whereas feces cause poly-microbial peritonitis; the latter is a more complicated and closer to the clinical diseases. Although several reports have discussed the mechanism of immune response in peritonitis-induced sepsis, however, the role of natural killer (NK) cells in sepsis, especially the relationship between NK cells and stabilization of the vascular endothelial barrier, is still unclear. Accordingly, in this study,

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Kang, Xu-Qi, Yue Qiao, Xiao-Yang Lu, et al. "Tocopherol polyethylene glycol succinate-modified hollow silver nanoparticles for combating bacteria-resistance." Biomaterials Science 7, no. 6 (2019): 2520–32. http://dx.doi.org/10.1039/c9bm00343f.

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Diedrich, Stephan, Julia van der Linde, Michael Nielson, et al. "The MRI Sepsis Score: An Innovative Tool for the Evaluation of Septic Peritonitis in Mice Using 7-Tesla Small Animal MRI." European Surgical Research 59, no. 3-4 (2018): 126–42. http://dx.doi.org/10.1159/000490663.

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Background: Magnetic resonance imaging (MRI) techniques are rarely used in the context of abdominal sepsis and in sepsis research. This study investigates the impact of MRI for monitoring septic peritonitis in an animal model (colon ascendens stent-induced peritonitis, CASP). The CASP model closely mimics that of human disease and is highly standardized. The most frequently employed readout parameter in mouse CASP studies is prolonged or decreased rate of survival. Monitoring the progression of peritonitis via MRI could provide a helpful tool in the evaluation of severity. The use of alternati

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Woods, Dustin R., Susan Heffley, James C. Peyton, and William G. Cheadle. "Antibiotic Modulation in a Clinically Relevant Model of Chronic Intraabdominal Infection." American Surgeon 72, no. 7 (2006): 655–60. http://dx.doi.org/10.1177/000313480607200717.

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Continuous and twice-daily cefoxitin dosing was used in a highly lethal model of acute peritonitis in mice using intraperitoneal (IP) Klebsiella pneumoniae (Kpn). The purpose was to use antibiotics to create a model of chronic infection. Male Balb/c mice (averaging 20 g body weight) were inoculated IP with 103 colony-forming units (CFU) Kpn serotype 2. Controls received subcutaneous saline either twice daily or continuously. Antibiotic groups received 300 mg/kg per day of cefoxitin either twice daily or continuously. Survival and daily weight losses were determined. Another group was inoculate

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Dissertations / Theses on the topic "Peritonitis model in mice"

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Diedrich, Stephan, der Linde Julia van, Michael Nielson, et al. "The MRI Sepsis Score: An Innovative Tool for the Evaluation of Septic Peritonitis in Mice Using 7-Tesla Small Animal MRI." Karger, 2018. https://tud.qucosa.de/id/qucosa%3A38909.

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Background: Magnetic resonance imaging (MRI) techniques are rarely used in the context of abdominal sepsis and in sepsis research. This study investigates the impact of MRI for monitoring septic peritonitis in an animal model (colon ascendens stent-induced peritonitis, CASP). The CASP model closely mimics that of human disease and is highly standardized. The most frequently employed readout parameter in mouse CASP studies is prolonged or decreased rate of survival. Monitoring the progression of peritonitis via MRI could provide a helpful tool in the evaluation of severity. The use of alternati

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Gallimore, Barbara. "Characterization of experimental Staphylococcus epidermidis peritonitis in chronically uremic mice." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75686.

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A mouse model of surgically induced renal failure was utilized to investigate the pathogenesis of Staphylococcus epidermidis peritonitis which is a frequent and serious complication of continuous ambulatory peritoneal dialysis (CAPD). Compared to sham-operated controls, chronically uremic mice were more susceptible to intraperitoneal S. epidermidis inoculation, presenting decreased survival time and survival (10$ sp9$ cfu, 10$ sp8$ cfu), delayed bacterial clearance and attenuated peritoneal inflammatory response (10$ sp6$ cfu). In mice bearing a peritoneal catheter implant, the catheter was a

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Su, FUHONG. "Adjunctive therapies in an ovine model of septic shock due to fecal peritonitis." Doctoral thesis, Universite Libre de Bruxelles, 2007. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210580.

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Sepsis remains a severe issue in critically ill patients. Adjunctive therapies might play important role to decrease morbidity and mortality. The aim of this thesis is to investigate new adjunctive therapies role in the treatment of sepsis and septic shock.<br>Doctorat en Sciences biomédicales et pharmaceutiques<br>info:eu-repo/semantics/nonPublished

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Brant, Jamet Ann. "Devleopment of an in vitro model of peritonitis complicating continuous ambulatory peritoneal dialysis." Thesis, University of Brighton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337400.

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Wang, YueYi. "Ca2+ handling in a mice model of CPVT." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS156/document.

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Le canal calcique de libération du Ca2+, appelé récepteur à la ryanodine (RyR) est localisé dans la membrane du réticulum sarcoplasmique des cardiomyocytes, en incluant ceux du pacemaker, et a un rôle important dans le couplage excitation contraction et la génération du rythme cardiaque. Des mutations dans leur gène sont responsables de la tachycardie catécholergique (CPVT), qui est une maladie létale, manifestée par des syncopes ou mort subite lors de stress émotionnel ou physique. Au repos, ces patients ont un électrocardiogramme normal, mais une tendance plus importante à la bradycardie.Nos

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Luiza, Batista Camilla. "Humanized mice as model for Plasmodium vivax infection." Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS232.

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Le paludisme est une maladie transmise par les moustiques causée par des espèces de Plasmodium, parmi lesquelles P. falciparum et P. vivax représentent problème de santé majeur avec plus de 400 000 victimes et 250 millions de cas cliniques signalés chaque année. P. vivax est reconnu comme une maladie débilitante et terrible. La mauvaise compréhension de la biologie des parasites est due au manque de culture à long terme et de modèles murins fidèles, ce qui reste difficile en raison de son tropisme pour les globules rouges (GR) immatures CD71+. Le nouveau modèle chimérique appelé HERYHIS pour H

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Swartz, Daniel E. "Alterations of polymorphonuclear neutrophi, PMN, recruitment in a murine model of peritonitis and a secondary injury." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0021/MQ55091.pdf.

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Swartz, Daniel E. "Alterations of Polymorphonuclear neutrophil (PMN) recruitment in a murine model of peritonitis and a secondary injury." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29924.

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Secondary peritonitis is a significant cause of morbidity and mortality in the ICU and ICU patients as a group have the highest rate of nosocomial infections. Once recruited to the site of injury, the PMN interacts with endothelial cells (ECs) via rolling adhesion, firm adhesion, and transendothelial migration. Using a murine cecal ligation and puncture peritonitis model and either skin or cremaster injury as the secondary site in a two-front injury model, we examined the role of injury severity on the triage of PMNs to competing sites of injury. We demonstrated that a finite pool of PMNs was

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So, Chi-leung. "Transgenic mouse model of human chondrodysplasia /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19161347.

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Bishop, Katherine Mary. "A threshold model for development of the corpus callosum in normal and acallosal mice." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq22952.pdf.

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Books on the topic "Peritonitis model in mice"

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Martin, Hrabé de Angelis, Chambon Pierre, and Brown Stephen D. M, eds. Standards of mouse model phenotyping. Wiley-VCH, 2006.

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Lamoreux, M. Lynn. The colors of mice: A model genetic network. Wiley-Blackwell, 2010.

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Lynn, Lamoreux M., ed. The colors of mice: A model genetic network. Wiley-Blackwell, 2010.

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A, Goffinet, ed. The reeler mouse as a model of brain development. Springer, 1998.

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Ferrick, David A. Transgenic mice as a in vivo model for self reactivity. R.G. Landes Co., 1994.

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1947-, McQueen Charlene A., ed. In vitro toxicology: Model systems and methods. Telford Press, 1989.

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1931-, Takeda Toshio, ed. The SAM model of senescence: Proceedings of the first International Conference on Senescence, the SAM model, Kyoto, 17-18 March 1994. Excerpta Medica, 1994.

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Brakebusch, Cord. Mouse as a Model Organism: From Animals to Cells. Springer Science+Business Media B.V., 2011.

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International Conference on Senescence (2nd 2003 Sapporo-shi, Japan). The senescence-accelerated mouse (SAM): An animal model of senescence : proceedings of the 2nd International Conference on Senescence, the SAM model, held in Sapporo, Japan between 21 and 23 July 2003. Edited by Nomura Yasuyuki Ph D, Takeda Toshio 1931-, and Okuma Yasunobu Ph D. Elsevier, 2004.

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Stilton, Gerónimo. Top model per un giorno. Piemme, 2011.

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Book chapters on the topic "Peritonitis model in mice"

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Giraud, Antoine. "Axenic Mice Model." In Innate Immunity. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-570-1_19.

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Kyuwa, S., Y. Tagawa, K. Machii, et al. "MHV-Induced Fatal Peritonitis in Mice Lacking IFN-γ." In Advances in Experimental Medicine and Biology. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5331-1_56.

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Sasaki, Takeshi, Kae Nakamura, and Masafumi Kuzuya. "Plaque Rupture Model in Mice." In Methods in Molecular Medicine™. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-571-8_3.

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Heljasvaara, Ritva, and Taina Pihlajaniemi. "Experimental Tumour Models in Mice." In Mouse as a Model Organism. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0750-4_5.

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Ray Banerjee, Ena. "Aseptic Peritonitis Model for Drug Discovery (As Therapy)." In Perspectives in Translational Research in Life Sciences and Biomedicine. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0989-1_3.

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Ray Banerjee, Ena. "Aseptic Peritonitis Model for Drug Discovery (for Prophylaxis)." In Perspectives in Translational Research in Life Sciences and Biomedicine. Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-0989-1_4.

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Quan, D., Z. Zhang, A. Jevnikar, R. Zhong, and D. Grant. "Intestinal Transplantation in the Murine Model." In Organtransplantation in Rats and Mice. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-72140-3_66.

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Vennema, Harry, Raoul J. de Groot, David A. Harbour, et al. "Immunogenicity of Recombinant Feline Infectious Peritonitis Virus Spike Protein in Mice and Kittens." In Advances in Experimental Medicine and Biology. Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-5823-7_30.

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Grimm, H., P. Mages, and G. Lindemann. "Technique of Plasmapheresis in the Xenogeneic Rat Model." In Organtransplantation in Rats and Mice. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-72140-3_59.

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Cesano, Alessandra, and Daniela Santoli. "SCID Mice as a Model for Human Leukemias." In Human Hematopoiesis in SCID Mice. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22008-5_9.

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Conference papers on the topic "Peritonitis model in mice"

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Lee, Yu-Hsuan, Yi-Ke Lin, Ta-Ching Chen, and Lih-Chu Chiou. "Novel glaucoma therapy rescuing excitotoxicity in an ischemia-reperfusion mice model." In Mechanisms of Photobiomodulation Therapy XIX, edited by James D. Carroll, Ann Liebert, and Jeri-Anne Lyons. SPIE, 2025. https://doi.org/10.1117/12.3054809.

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Subramani, Nishant, Jason Eisner, Justin Svegliato, Benjamin Van Durme, Yu Su, and Sam Thomson. "MICE for CATs: Model-Internal Confidence Estimation for Calibrating Agents with Tools." In Proceedings of the 2025 Conference of the Nations of the Americas Chapter of the Association for Computational Linguistics: Human Language Technologies (Volume 1: Long Papers). Association for Computational Linguistics, 2025. https://doi.org/10.18653/v1/2025.naacl-long.615.

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Obanina, Natalia, and Nataliya Bgatova. "Effect of Lithium and Chloroquine on Neuronal Ultrastructure in a Melanoma Mice Model." In 2024 IEEE International Multi-Conference on Engineering, Computer and Information Sciences (SIBIRCON). IEEE, 2024. http://dx.doi.org/10.1109/sibircon63777.2024.10758443.

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Seeley, Eric J., Sophia S. Kim, Michael A. Matthay, and Paul J. Wolters. "Endogenous Catecholamines Impair Innate Immune Responses And Worsen Survival During Septic Peritonitis In Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3852.

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Du, Liping, Hongyan Niu, Xuekuang Zhang, and Dongguang Xiao. "Preparation of Glucan Sulfates with Different Degree of Substitution and Their Immunoprophylaxis Potentials in Escherichia coli Induced Mice Peritonitis." In 2010 4th International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2010. http://dx.doi.org/10.1109/icbbe.2010.5516040.

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Stergar, Jost, Katja Lakota, Martina Perseˇ, Matija Tomsic, and Matija Milanic. "Vasculature-based biomarkers and segmentation from hyperspectral images of murine peritonitis model." In Translational Biophotonics: Diagnostics and Therapeutics, edited by Lothar D. Lilge and Zhiwei Huang. SPIE, 2021. http://dx.doi.org/10.1117/12.2614589.

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Wahyudin, Nanang, Sandy Pratama, and Echo Perdana Kusumah. "MICE Model: Artificial Tourism Potential." In Proceedings of the International Conference on Maritime and Archipelago (ICoMA 2018). Atlantis Press, 2019. http://dx.doi.org/10.2991/icoma-18.2019.56.

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Tackett, S., R. Zamora, C. Clifford, et al. "Respiratory and Neuro-Inflammatory Control Networks in a Rat Model of E. Coli Peritonitis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7610.

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Ueda-Arima, Tomomi, Koichi Fukunaga, Jun Miyata, Hiroshi Seki, Junzo Takeda, and Koichiro Asano. "The Effect Of Anti-Inflammatory Lipid Mediator, Lipoxin A4, In A Murine Peritonitis Model." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4678.

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Plotkin, Dmitry, Tatiana Vinogradova, Mikhail Reshetnikov, Mikhail Sinitsyn, and Sergey Okovityi. "IDDF2021-ABS-0013 The method for obtaining a reproducible model of tuberculous peritonitis in rabbits." In Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 4–5 September 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-iddf.21.

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Reports on the topic "Peritonitis model in mice"

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ศิริวิชยกุล, สุณี, เอกชัย พรหมเพชร, ภัทรวัจน์ ตันติวรสิทธิ์, นิธิรา อนัคกุล та เกียรติ รักษ์รุ่งธรรม. โครงการ การแยกเซลล์ต้นกำเนิดจากสายสะดือเพื่อสร้าง humanized mice เพื่อใช้ในการทดสอบวัคซีนเอดส์ในระดับก่อนการทดสอบในมนุษย์ : รายงานการวิจัยฉบับสมบูรณ์. คณะแพทยศาสตร์ จุฬาลงกรณ์มหาวิทยาลัย, 2016. https://doi.org/10.58837/chula.res.2016.24.

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ในความพยายามที่จะหาสัตว์ทดลองที่จะใช้ทดสอบวัคซีนเพื่อให้ได้ผลการตอบสนองทางภูมิคุ้มกันที่ใกล้เคียงกับคนที่สุดนั้น humanized mice model เป็นทางเลือกที่สามารถให้การตอบสนองทางภูมิคุ้มกันที่ใกล้เคียงกับการตอบสนองในคนมากที่สุด กล่าวคือ humanized mice เป็นหนูทดลองที่ได้รับการปลูกถ่ายเซลล์ต้นกำเนิดของคน (C034+ human hematopoietic stem cells) ที่แยกได้จากเลือดที่เจาะจากสายสะดือทารกแรกคลอด เพื่อให้เซลล์ต้นกำเนิดของคนไปเจริญเติบโตเป็นเซลล์ที่รับผิดชอบต่อการตอบสนองทางภูมิคุ้มกัน เช่น lymphocytes และ macrophages ในหนู โดยถือว่าการปลูกถ่ายเซลล์ต้นกำเนิดประสบความสำเร็จ ต่อเมื่อสามารถตรวจพบเซลล์ที่เกี่ยวกับระ

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Robins, Diane M. Humanized Androgen Receptor Mice: A Genetic Model for Differential Response to Prostate Cancer Therapy. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada582175.

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Robins, Diane. Humanized Androgen Receptor Mice: A Genetic Model for Differential Response to Prostate Cancer Therapy. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada547343.

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Pilitkul, Trairak, Prapaporn Pisitkun, Asada Leelahavanichkul, and Poorichaya Somparn. Systems-investigation of aberrant signaling in immune cells of SLE mouse model. Faculty of Medicine, Chulalongkorn University, 2020. https://doi.org/10.58837/chula.res.2020.21.

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Autoimmune diseases occur when the immune cells react against self antigens and subsequently lead to inflammation in the tissues. The interactions between genetics and environmental triggers regulate the phenotypes and outcome of the diseases. Type I interferon has been shown as one of the most crucial cytokines involving in the pathogenesis of autoimmune diseases such as systemic lupus erythematosus (SLE) and rheumatoid arthritis (RA). SLE is a chronic systemic autoimmune disease which can result in autoantibody production and fatal glomerulonephritis. Activation via nucleic acid sensors can

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Pilitkul, Trairak, Prapaporn Pisitkun, and Asada Leelahavanichkul. Systems-investigation of aberrant signaling in immune cells of SLE mouse model. Faculty of Medicine, Chulalongkorn University, 2015. https://doi.org/10.58837/chula.res.2015.22.

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Autoimmune diseases occur when the immune cells react against self-antigens and subsequently lead to inflammation in the tissues. The interactions between genetics and environmental triggers regulate the phenotypes and outcome of the diseases. Type I interferon has been shown as one of the most crucial cytokines involving in the pathogenesis of autoimmune diseases such as systemic lupus erythematosus (SLE) and rheumatoid arthritis (RA). SLE is a chronic systemic autoimmune disease which can result in autoantibody production and fatal glomerulonephritis. Activation via nucleic acid sensors can

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Matory, Yvedt L. Evaluation of a Mouse Model for Prophylactic Mastectomy in Mice Genetically Predisposed to Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada400613.

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Deng, Yingjun, ShengJing Liu, Ming Zhao, Feng Zhao, Jun Guo, and Bin Yan. Diet-induced male infertility in mice models: a systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.5.0116.

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Review question / Objective: In order to compare the different high energy diet such as high-fat diet and high sugar diet how to damage the male mice model in metabolize and fertility,and explore a reliable mice model method in the study of obesity with male infertility. P：obesity mice model with male infertility. I: High energy diet such as High-fat or High-sugar diet. C:High-fat diet,High-sugar diet, compared with normal diet in mice model. O:High energy diet induce male mice obesity model and damage their fertility. S: Use network meta-analysis. Condition being studied: The relationship bet

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Li, Xiao-Nan. Harnessing Autopsied DIPG Tumor Tissues for Orthotopic Xenograft Model Development in the Brain Stems of SCID Mice. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada568355.

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Alves-Borba, Laryssa, Verónica Espinosa-Fernández, Ania Canseco Rodriguez, and Ana María Sánchez-Pérez. ABA Supplementation Rescues IRS2 and BDNF mRNA Expression in a Triple-Transgenic Mice Model of Alzheimer’s Disease. Universitat Jaume I, 2023. http://dx.doi.org/10.6035/uji_b201801.

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Tumwasorn, Somying, and Asada Leelahavanichkul. Effect of Lactobacillus and Bifidobacterium on the inhibition of Clostridium difficile in mouse infection model. Chulalongkorn University, 2018. https://doi.org/10.58837/chula.res.2018.25.

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Clostridium difficile is a major cause antibiotic-associated diarrhea and colitis in patients with broad-spectrum antibiotic therapy. A disruption of the gut microbiota by using antibiotics results in colonization with C. difficile and release of toxins that cause leaky gut and the production inflammatory cytokines. We identified six specific strains of Lactobacillus or Bifidobacterium are able to reduce leaky gut and inflammation caused by C. difficile in vitro. In this study, we aim to investigate the effect of these strains either alone or in combination on the inhibition of C. difficile in

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