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Dissertations / Theses on the topic 'Peritonitis model in mice'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Diedrich, Stephan, der Linde Julia van, Michael Nielson, et al. "The MRI Sepsis Score: An Innovative Tool for the Evaluation of Septic Peritonitis in Mice Using 7-Tesla Small Animal MRI." Karger, 2018. https://tud.qucosa.de/id/qucosa%3A38909.

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Background: Magnetic resonance imaging (MRI) techniques are rarely used in the context of abdominal sepsis and in sepsis research. This study investigates the impact of MRI for monitoring septic peritonitis in an animal model (colon ascendens stent-induced peritonitis, CASP). The CASP model closely mimics that of human disease and is highly standardized. The most frequently employed readout parameter in mouse CASP studies is prolonged or decreased rate of survival. Monitoring the progression of peritonitis via MRI could provide a helpful tool in the evaluation of severity. The use of alternati

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2

Gallimore, Barbara. "Characterization of experimental Staphylococcus epidermidis peritonitis in chronically uremic mice." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=75686.

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A mouse model of surgically induced renal failure was utilized to investigate the pathogenesis of Staphylococcus epidermidis peritonitis which is a frequent and serious complication of continuous ambulatory peritoneal dialysis (CAPD). Compared to sham-operated controls, chronically uremic mice were more susceptible to intraperitoneal S. epidermidis inoculation, presenting decreased survival time and survival (10$ sp9$ cfu, 10$ sp8$ cfu), delayed bacterial clearance and attenuated peritoneal inflammatory response (10$ sp6$ cfu). In mice bearing a peritoneal catheter implant, the catheter was a

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3

Su, FUHONG. "Adjunctive therapies in an ovine model of septic shock due to fecal peritonitis." Doctoral thesis, Universite Libre de Bruxelles, 2007. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210580.

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Sepsis remains a severe issue in critically ill patients. Adjunctive therapies might play important role to decrease morbidity and mortality. The aim of this thesis is to investigate new adjunctive therapies role in the treatment of sepsis and septic shock.<br>Doctorat en Sciences biomédicales et pharmaceutiques<br>info:eu-repo/semantics/nonPublished

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4

Brant, Jamet Ann. "Devleopment of an in vitro model of peritonitis complicating continuous ambulatory peritoneal dialysis." Thesis, University of Brighton, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337400.

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5

Wang, YueYi. "Ca2+ handling in a mice model of CPVT." Thesis, Université Paris-Saclay (ComUE), 2016. http://www.theses.fr/2016SACLS156/document.

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Le canal calcique de libération du Ca2+, appelé récepteur à la ryanodine (RyR) est localisé dans la membrane du réticulum sarcoplasmique des cardiomyocytes, en incluant ceux du pacemaker, et a un rôle important dans le couplage excitation contraction et la génération du rythme cardiaque. Des mutations dans leur gène sont responsables de la tachycardie catécholergique (CPVT), qui est une maladie létale, manifestée par des syncopes ou mort subite lors de stress émotionnel ou physique. Au repos, ces patients ont un électrocardiogramme normal, mais une tendance plus importante à la bradycardie.Nos

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6

Luiza, Batista Camilla. "Humanized mice as model for Plasmodium vivax infection." Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS232.

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Le paludisme est une maladie transmise par les moustiques causée par des espèces de Plasmodium, parmi lesquelles P. falciparum et P. vivax représentent problème de santé majeur avec plus de 400 000 victimes et 250 millions de cas cliniques signalés chaque année. P. vivax est reconnu comme une maladie débilitante et terrible. La mauvaise compréhension de la biologie des parasites est due au manque de culture à long terme et de modèles murins fidèles, ce qui reste difficile en raison de son tropisme pour les globules rouges (GR) immatures CD71+. Le nouveau modèle chimérique appelé HERYHIS pour H

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7

Swartz, Daniel E. "Alterations of polymorphonuclear neutrophi, PMN, recruitment in a murine model of peritonitis and a secondary injury." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0021/MQ55091.pdf.

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8

Swartz, Daniel E. "Alterations of Polymorphonuclear neutrophil (PMN) recruitment in a murine model of peritonitis and a secondary injury." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=29924.

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Secondary peritonitis is a significant cause of morbidity and mortality in the ICU and ICU patients as a group have the highest rate of nosocomial infections. Once recruited to the site of injury, the PMN interacts with endothelial cells (ECs) via rolling adhesion, firm adhesion, and transendothelial migration. Using a murine cecal ligation and puncture peritonitis model and either skin or cremaster injury as the secondary site in a two-front injury model, we examined the role of injury severity on the triage of PMNs to competing sites of injury. We demonstrated that a finite pool of PMNs was

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9

So, Chi-leung. "Transgenic mouse model of human chondrodysplasia /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19161347.

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10

Bishop, Katherine Mary. "A threshold model for development of the corpus callosum in normal and acallosal mice." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq22952.pdf.

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11

Agarwal, Rajat. "A model for minimizing cost for housing laboratory mice." [Gainesville, Fla.] : University of Florida, 2003. http://purl.fcla.edu/fcla/etd/UFE0001241.

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12

Balastik, Martin. "Trim2 mutant mice as a model for cerebellar ataxia." Doctoral thesis, [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=975117025.

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13

蘇志良 and Chi-leung So. "Transgenic mouse model of human chondrodysplasia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237678.

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14

衛永剛 and Wing-kong Wai. "Abnormal chondrocyte differentiation: a transgenic model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31237800.

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15

Wai, Wing-kong. "Abnormal chondrocyte differentiation : a transgenic model /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19656439.

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16

Ou, Linda Ye. "Hyperactive p53 leads to age-related phenotypes in mice model." Diss., [La Jolla, Calif.] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p1460672.

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17

Makinen, Kimmo. "Spontaneous model of experimental autoimmune uveoretinitis : IRBP-HEL transgenic mice." Thesis, University of Aberdeen, 2006. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU490269.

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To generate a spontaneous EAU model, transgenic mice expressing membrane-bound hen egg lysozyme (HEL) under control of the IRBP promoter were generated. The mice expressed HEL in the photoreceptors, and in the thymus as measured by immunofluorescent confocal microscopy and real-time RT-PCR, respectively. When crossed with the 3A9 TCR-transgenic mice whose CD4+ T cells are HEL-specific, double-transgenic mice developed spontaneous EAU with 100% incidence and an onset age of 22 days post-partum. The ocular inflammation was multifocal and affected the posterior segment of the eye, and led to comp

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18

Siu, Kwan-yin. "The development and characterization of a knockout model for secretin." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B40887674.

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19

Clifford, Adrianne Brown. "Tumor Associated Macrophages in a MaFIA Mouse Model." Diss., CLICK HERE for online access, 2006. http://contentdm.lib.byu.edu/ETD/image/etd1427.pdf.

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20

Nguyen, Duy Cuong. "A new long-term porcine model of fecal peritonitis-induced septic shock hemodynamics, gas exchange, metabolism, and organ function." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-65005.

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21

Teich, Joshua P. "Generation of a femoral non-union model utilizing phosphate deficient mice." Thesis, Boston University, 2013. https://hdl.handle.net/2144/12237.

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Thesis (M.A.)--Boston University<br>Fracture non-unions occur when the biological process of fracture healing fails to complete. The events that occur in the fracture healing process of mice are similar to the events that occur in humans. Inorganic phosphate is essential for maintaining healthy bones. In this study mice are placed on a phosphate deficient diet two days prior to fracture of the right femur. The mice were then maintained for 5, 10, 15, and 20 days of restricted diet and compared to control animals. The progression of fracture healing was assessed by plain film x-ray radiography

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22

Siu, Kwan-yin, and 蕭君言. "The development and characterization of a knockout model for secretin." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B40887674.

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23

Ganesan, Poo Balan. "Development of an image-based anatomical network model and modelling of circulation of mouse retinal vasculature." Thesis, University of Aberdeen, 2010. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=210068.

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24

Chung, Chi-kin Samuel. "The development and characterization of a gene-knockout mouse model for secretin receptor /." View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31491121.

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25

Eastgard, Rebecca Lugar. "Diet-induced hyperhomocysteinemia in a mouse model /." Thesis, Connect to this title online; UW restricted, 2002. http://hdl.handle.net/1773/6601.

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26

Lau, John C. Y. "Developmental regulation of metals by metallothionein, genetically altered mice as a model." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq28603.pdf.

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27

Chan, Chu-fung, and 陳柱峰. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B40687284.

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28

Schirm, Sibylle, Peter Ahnert, Sandra Wienhold, et al. "A biomathematical model of pneumococcal lung infection and antibiotic treatment in mice." Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-204153.

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Pneumonia is considered to be one of the leading causes of death worldwide. The outcome depends on both, proper antibiotic treatment and the effectivity of the immune response of the host. However, due to the complexity of the immunologic cascade initiated during infection, the latter cannot be predicted easily. We construct a biomathematical model of the murine immune response during infection with pneumococcus aiming at predicting the outcome of antibiotic treatment. The model consists of a number of non-linear ordinary differential equations describing dynamics of pneumococcal population, t

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29

Chan, Chu-fung. "Neuroprotective effects of granulocyte-colony stimulating factor in a mice stroke model." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B40687284.

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30

Antognoli, Agnese <1981&gt. "Rag2-/-;gammac-/- immunodeficient mice, a new preclinical model to study antitumor approaches." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2010. http://amsdottorato.unibo.it/2333/1/Antognoli_Agnese_tesi.pdf.

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Animal models have been relevant to study the molecular mechanisms of cancer and to develop new antitumor agents. Anyway, the huge divergence in mouse and human evolution made difficult the translation of the gained achievements in preclinical mouse based studies. The generation of clinically relevant murine models requires their humanization both concerning the creation of transgenic models and the generation of humanized mice in which to engraft a functional human immune system, and reproduce the physiological effects and molecular mechanisms of growth and metastasization of human tumors. In

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31

Antognoli, Agnese <1981&gt. "Rag2-/-;gammac-/- immunodeficient mice, a new preclinical model to study antitumor approaches." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2010. http://amsdottorato.unibo.it/2333/.

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Animal models have been relevant to study the molecular mechanisms of cancer and to develop new antitumor agents. Anyway, the huge divergence in mouse and human evolution made difficult the translation of the gained achievements in preclinical mouse based studies. The generation of clinically relevant murine models requires their humanization both concerning the creation of transgenic models and the generation of humanized mice in which to engraft a functional human immune system, and reproduce the physiological effects and molecular mechanisms of growth and metastasization of human tumors. In

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32

Price, David Alan. "Neuroprotective effects of cannabinoids in a mouse model of Parkinson's disease : a dissertation /." San Antonio : UTHSC, 2007. http://proquest.umi.com/pqdweb?did=1317324441&sid=1&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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33

Stinnett, Hilary M. "OSTEOACTIVIN IN SKELETON: CHARACTERIZATION OF OSTEOACTIVIN KNOCKOUT MICE & THERAPEUTIC IMPLICATIONS." Kent State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=kent1428859108.

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34

Brown, Steven J. "Immunological studies of a glycosylation based mouse model of colitis /." Connect to thesis, 2004. http://eprints.unimelb.edu.au/archive/00000788.

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Thesis (Ph.D.)--University of Melbourne, Dept. of Gastroenterology and the Immunology Research Centre St. Vincents Hospital & Dept of Medicine, 2004.<br>Typescript (photocopy). Includes bibliographical references (leaves 309-343).

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35

Tillotson, Anne Rebekah. "Identifying the key functions of MeCP2 via genetic manipulation in mice." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/28917.

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MeCP2 was identified by its ability to bind DNA in a methylation-specific manner. Yet, how it interprets the DNA methylome remains unclear. Several mechanisms have been proposed, including a role in transcriptional repression. MeCP2 is highly abundant in the brain, and loss-of-function mutations result in a neurological disorder called Rett syndrome (RTT). Strikingly, RTT-causing missense mutations are almost all located in either the methyl-CpG-binding domain (MBD) or a region that has been shown to bind the NCoR/SMRT co-repressor complex (NID). This suggests that the MBD and the NID are the

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36

Boman, Erik. "Olfactory and cognitive abilities in two strains of Alzheimer`s disease model mice." Thesis, Linköping University, Department of Physics, Chemistry and Biology, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-19200.

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<p>The present study assessed olfactory and cognitive abilities in two strains of Alzheimer’s disease (AD) model mice and in healthy control mice over a four month time period. To this end an operant conditioning paradigm using an automated olfactometer and a spatial learning test with non-olfactory cues were employed and data on olfactory learning and memory, discrimination, and sensitivity as well as spatial learning and memory were collected. The mice were between 6 to 7 month old at the beginning of the study and 9 to 10 months old at the end of the data collection, that is, in the age ran

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37

Hässler, Signe. "Autoimmune Regulator Deficient Mice, an Animal Model of Autoimmune Polyendocrine Syndrome Type I." Doctoral thesis, Uppsala University, Department of Medical Sciences, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7218.

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<p>Autoimmune diseases develop when the immune system fails to distinguish self from non-self or when the immune system is hypersensitive to endogenous or exogenous danger signals, or when a tissue erroneously sends a danger signal to the immune system. The education of the immune system to distinguish self from non-self is mainly carried out in the thymus and gives rise to central tolerance, whereas the ability to sense a danger or a healthy tissue constitutes peripheral tolerance. In these studies we have investigated the peripheral tolerance mechanisms controlled by the autoimmune regulator

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38

Österman, Hanna. "Olfactory performance and neuropathology in the Tg6799 strain of Alzheimer’s disease model mice." Thesis, Linköpings universitet, Institutionen för fysik, kemi och biologi, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-56816.

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The present study evaluated olfactory and cognitive abilities of the Tg6799 (also called 5xFAD) strain of Alzheimer’s disease (AD) model mice of two different age groups (2-3 and 8-10 months of age), and one group of healthy control mice (9-10 months). Employment of an operant conditioning paradigm using an automated olfactometer, an olfactory habituation/dishabituation test and a spatial learning test with non olfactory cues resulted in data showing that the 5xFAD mice develop olfactory impairments already at 2-3 months of age. The impairments consisted in a robust impairment in olfactory sen

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39

Thorn, Catherine Elisabeth. "Pasteurella haemolytica induced bronchopneumonia in Scid/bg mice, a model for bovine pneumonia." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/NQ31905.pdf.

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40

Hässler, Signe. "Autoimmune regulator deficient mice : an animal model of autoimmune polyendocrine syndrome type I /." Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7218.

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41

Lindsay, Karen. "Serotonin transporter knockout mice and maternal stress a potential animal model of autism /." Connect to resource, 2008. http://hdl.handle.net/1811/32183.

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42

Pham, Thu-Trang. "Functional analysis of DJ-1 deficient mice - a mouse model for Parkinsons disease." Thesis, Open University, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504328.

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43

Kanba, Tomomi. "Failure of ureteric bud invasion a new model of renal agenesis in mice." Kyoto University, 2003. http://hdl.handle.net/2433/148695.

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44

Göhring, Claudia [Verfasser]. "Hepcidin knockout mice constitute a suitable mouse model for hereditary hemochromatosis / Claudia Göhring." Ulm : Universität Ulm. Medizinische Fakultät, 2015. http://d-nb.info/1076828523/34.

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45

Tsang, Kwok-yeung. "Molecular pathogenesis of abnormal chondrocyte differentiation in a transgenic mouse model /." View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B35132796.

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46

Hogmalm, Anna. "Mechanisms of lung injury in a mouse model of Bronchopulmonary dysplasia /." Göteborg : The Sahlgrenska Academy, Institute of Clinical Sciences, Department of Pediatrics, University of Gothenburg, 2009. http://hdl.handle.net/2077/21084.

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47

Cheng, Yin-wo. "Molecular basis for the increased osteoblast activity in a mouse model with hyperostosis." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B34612981.

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48

Chung, Chi-kin Samuel, and 鍾志堅. "The development and characterization of a gene-knockout mouse model for secretin receptor." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B45014759.

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49

Phan, Tri Giang. "The SW hel model for studying B cell responses in tolerance and immunity." Connect to full text, 2004. http://hdl.handle.net/2123/626.

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Thesis (Ph. D.)--University of Sydney, 2005.<br>Title from title screen (viewed 22 May 2008). Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the Discipline of Experimental Medicine, Faculty of Medicine. Degree awarded 2005; thesis submitted 2004. Includes bibliographical references. Also available in print form.

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50

Van, Andel Roger A. "The immunopathogenesis of clostridium piliforme evaluated in a murine model /." free to MU campus, to others for purchase, 1997. http://wwwlib.umi.com/cr/mo/fullcit?p9842572.

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