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Relevant bibliographies by topics / Persistent developmental stuttering

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Journal articles
Dissertations / Theses
Book chapters

Academic literature on the topic 'Persistent developmental stuttering'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

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Journal articles on the topic "Persistent developmental stuttering"

1

Kumar, Amardeep, and Sabish Balan. "Fluoxetine for Persistent Developmental Stuttering." Clinical Neuropharmacology 30, no. 1 (2007): 58–59. http://dx.doi.org/10.1097/01.wnf.0000240950.18821.19.

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Ranjan, Sanjeev, Vinit Sawhney, and Prabha S. Chandra. "Persistent developmental stuttering: treatment with risperidone." Australian and New Zealand Journal of Psychiatry 40, no. 2 (2006): 193. http://dx.doi.org/10.1111/j.1440-1614.2006.01774_1.x.

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3

Sommer, Martin, Kathrin Knappmeyer, Evke Jane Hunter, Alexander Wolffvon Gudenberg, Nicole Neef, and Walter Paulus. "Normal interhemispheric inhibition in persistent developmental stuttering." Movement Disorders 24, no. 5 (2009): 769–73. http://dx.doi.org/10.1002/mds.22383.

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Tran, Nancy L., Gerald A. Maguire, David L. Franklin, and Glyndon D. Riley. "Case Report of Aripiprazole for Persistent Developmental Stuttering." Journal of Clinical Psychopharmacology 28, no. 4 (2008): 470–72. http://dx.doi.org/10.1097/jcp.0b013e31817ea9ad.

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5

Corbera, S., M. J. Corral, C. Escera, and M. A. Idiazabal. "Abnormal speech sound representation in persistent developmental stuttering." Neurology 65, no. 8 (2005): 1246–52. http://dx.doi.org/10.1212/01.wnl.0000180969.03719.81.

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Mock, Jeffrey R., Anne L. Foundas, and Edward J. Golob. "Speech preparation in adults with persistent developmental stuttering." Brain and Language 149 (October 2015): 97–105. http://dx.doi.org/10.1016/j.bandl.2015.05.009.

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Foundas, A. L., D. M. Corey, V. Angeles, A. M. Bollich, E. Crabtree-Hartman, and K. M. Heilman. "Atypical cerebral laterality in adults with persistent developmental stuttering." Neurology 61, no. 10 (2003): 1378–85. http://dx.doi.org/10.1212/01.wnl.0000094320.44334.86.

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Dias, Fernando Machado Vilhena, Patrícia Matos Pereira, Flavia Costa de Proença Doyle, and Antônio Lúcio Teixeira. "Psychiatric Disorders in a Patient With Persistent Developmental Stuttering." Clinical Neuropharmacology 34, no. 5 (2011): 199–200. http://dx.doi.org/10.1097/wnf.0b013e31822b9ead.

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Frigerio-Domingues, Carlos E., Zoi Gkalitsiou, Alexandra Zezinka, et al. "Genetic factors and therapy outcomes in persistent developmental stuttering." Journal of Communication Disorders 80 (July 2019): 11–17. http://dx.doi.org/10.1016/j.jcomdis.2019.03.007.

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10

Kronfeld-Duenias, Vered, Ofer Amir, Ruth Ezrati-Vinacour, Oren Civier, and Michal Ben-Shachar. "Dorsal and ventral language pathways in persistent developmental stuttering." Cortex 81 (August 2016): 79–92. http://dx.doi.org/10.1016/j.cortex.2016.04.001.

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Dissertations / Theses on the topic "Persistent developmental stuttering"

1

Unger, Julia [Verfasser], and Christian [Akademischer Betreuer] Glück. "The Immediate and Long-term Effects of Altered Auditory Feedback (AAF) on the Characteristics of Persistent Developmental Stuttering / Julia Unger. Betreuer: Christian Glück." Heidelberg : Pädagogische Hochschule Heidelberg, 2012. http://d-nb.info/107028579X/34.

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2

Frankford, Saul Alexander. "Internal and external speech timing mechanisms in persistent developmental stuttering." Thesis, 2021. https://hdl.handle.net/2144/42559.

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Stuttering is a developmental speech disorder characterized by interruptions of fluency. A large body of research suggests that stuttering occurs due to a reduced ability to generate timing signals in order to sequence speech sounds. One piece of supporting evidence for this is that when speaking along with an external timing source like a metronome, disfluencies suddenly and significantly decrease. The aim of this dissertation was to characterize the effects of using auditory cues to time speech on neural activation and auditory feedback processing, and how these effects may contribute to fluency in adults who stutter (AWS). Two studies were carried out to examine these effects. In the first study, functional magnetic resonance imaging was used to measure brain activity while AWS and adults who do not stutter (ANS) read sentences aloud either using natural speech timing or aligning each syllable to the beat of a metronome. Consistent with previous literature, AWS produced fewer disfluent trials in the externally paced condition than in the normal condition. Collapsing across the AWS and ANS groups, participants had greater activation in the metronome-timed condition in regions associated with speech sequencing, sensory feedback control, and timing perception. AWS also demonstrated increased functional connectivity among cerebellar regions during externally paced speech. In the second study, responses to online spectral and timing perturbations of auditory feedback were measured while AWS and ANS read sentences with and without metronome pacing. Results indicated that AWS showed no responses to spectral perturbations during the non-paced condition and significant compensatory responses during the paced condition along with fewer disfluencies, while responses in ANS showed the opposite effect. For the timing perturbation, no significant differences were found between groups in either condition. Together, these studies indicate that the deficit in stuttering is related to spectral processing rather than purely temporal processing, and that externally paced speech recruits compensatory neural regions that may help resolve this deficit.

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3

"Atypical anatomy in children and adults with persistent developmental stuttering." Tulane University, 2007.

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A larger right prefrontal and a larger left occipital lobe (lobar asymmetry) and a larger left planum temporale (PT) are consistent asymmetries found in the human brain. Reduced or reversed asymmetries from these typical configurations are considered atypical and may be markers of atypical function. Atypical lobar and PT asymmetries have been found in adults with persistent developmental stuttering (PDS). These atypical asymmetries may represent a neural risk for developing PDS. To further understand the development of these asymmetries; volumes, ratios and asymmetry quotients were investigated in three groups: (1) healthy right-handed boys and girls ages 8--13 (2) righthanded boys with PDS and matched controls ages 8--13 (3) right-handed adult men with PDS and matched controls ages 21--49. The healthy boys and girls displayed sex-linked differences. Boys had larger total brain and total gray matter volumes than girls. Due to sex-linked gray matter differences, boys had larger right prefrontal and left occipital volumes which lead to a greater magnitude of brain torque. The PT was leftward in both sexes. The boys with PDS differed in multiple anatomical areas compared to controls. The PDS group had more total white matter and a smaller gray-to-white matter ratio in the right hemisphere compared to controls. The PDS group had a smaller right prefrontal region due to decreased prefrontal inferior gray matter. The occipital volume and asymmetry patterns also differed between groups. Controls had the expected leftward asymmetry while the PDS group was more atypical. Thus, the overall brain torque was more symmetrical in the PDS group. The stuttering severity scores positively correlated with right prefrontal white matter volume. The PT was leftward in the boys with no group difference. Right-handed men with PDS and controls did not differ in lobar volumes and asymmetries. The PT was more symmetrical in the PDS group than controls. Interestingly, in all PDS subjects and controls, the prefrontal and occipital white matter asymmetries followed the typical brain torque configuration while the gray matter asymmetries were more variable. This observation indicates that white matter volumes may be responsible for the typical brain torque configuration<br>acase@tulane.edu

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4

Whillier, Alexander Sean. "Investigating the neural processes underpinning the production and anticipation of stuttering in Adults who Stutter." Doctoral thesis, 2018. http://hdl.handle.net/11858/00-1735-0000-002E-E580-A.

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Knappmeyer, Kathrin. "Untersuchung der funktionellen Konnektivität zwischen dem links-und rechtshemisphärischen primärmotorischen Kortex bei Stotternden mit Hilfe der transkraniellen Magnetstimulation." Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-0006-B22F-1.

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Metzger, Friederike Luise. "Funktionelle Konnektivität der Substantia nigra in einem generellen Aufmerksamkeitstest bei idiopathischem Stottern – eine klinische Studie mittels funktioneller Magnetresonanztomografie." Doctoral thesis, 2020. http://hdl.handle.net/21.11130/00-1735-0000-0005-14D2-7.

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Book chapters on the topic "Persistent developmental stuttering"

1

Prado-Velasco, Manuel, and Carlos Fernández-Peruchena. "An Advanced Concept of Altered Auditory Feedback as a Prosthesis-Therapy for Stuttering Founded on a Non-Speech Etiologic Paradigm." In Handbook of Research on Personal Autonomy Technologies and Disability Informatics. IGI Global, 2011. http://dx.doi.org/10.4018/978-1-60566-206-0.ch006.

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Persistent Developmental Stuttering affects 1-2% of the world adult population. Its etiology is still unknown, although modern neuroimaging techniques have shown a new and exciting perspective of earlier ideas and hypotheses. However, it is now clear that a new approach to understand the true nature of the disorder is needed. We present a new etiological model of persistent developmental stuttering based on a deep analysis of earlier models and on the stuttering phenomenology, described in basic, clinical, and even ethnographic sources. One of the more stimulating conclusions has been the suggestion that stuttering is a non-speech based disorder, in opposition to the accepted belief. The implications of this model have guided the design of a new adaptive AAF device for prosthetic and therapeutic functions. It is supported by a wearable multimodal intelligent system, which evolves from a preliminary proposal presented in (Prado & Roa, 2007).

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