Log in

Relevant bibliographies by topics / Peste des petits ruminants virus

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers
Reports

Academic literature on the topic 'Peste des petits ruminants virus'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Peste des petits ruminants virus.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Peste des petits ruminants virus"

1

Saxena, Shikha, Shishir Kumar Gupta, and Satish Kumar. "Sialic Acid Activated Gold Nanoparticles as Rapid Affordable Reagent for Peste Des Petits Ruminants (PPR) Virus Detection." Journal of Nanoscience and Nanotechnology 21, no. 6 (June 1, 2021): 3630–33. http://dx.doi.org/10.1166/jnn.2021.18997.

Full text

Abstract:

Animal health issues are important for farming community in agriculture. Small ruminant populations such as goats and sheep often get affected with contagious diseases. Peste des petits ruminants caused by a virus which need to be detected quickly to isolate affected animals and stop the spread of disease. The H protein of Peste des petits ruminants virus has sialic acid specific receptor, therefore sialic acid reduce and stabilized gold-nanoparticles were synthesize by a simple one pot method and without chemically modifying the sialic acid. The gold nanoparticles showed targetspecific aggregation with viral particles via hemagglutinin-sialic acid binding. The PPR virus was readily detected at the dilution of 10−6 by sialic acid-AuNPs. While comparing with the standard monoclonal antibody based test used for the detection of Peste des petits ruminants virus, sialic acid-AuNPs gave detection faster in less than 2 minute.

APA, Harvard, Vancouver, ISO, and other styles

2

El Arbi, Ahmed Salem, Ahmed Bezeid El Mamy, Habib Salami, Ekatarina Isselmou, Olivier Kwiatek, Geneviève Libeau, Yaghouba Kane, and Renaud Lancelot. "Peste des Petits Ruminants Virus, Mauritania." Emerging Infectious Diseases 20, no. 2 (February 2014): 333–36. http://dx.doi.org/10.3201/eid2002.131345.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

KINNE, J., R. KREUTZER, M. KREUTZER, U. WERNERY, and P. WOHLSEIN. "Peste des petits ruminants in Arabian wildlife." Epidemiology and Infection 138, no. 8 (January 13, 2010): 1211–14. http://dx.doi.org/10.1017/s0950268809991592.

Full text

Abstract:

SUMMARYRecurrence of peste des petits ruminants (PPR) was diagnosed in the United Arabian Emirates in several wild ruminants confirmed by morphological, immunohistochemical, serological and molecular findings. Phylogenetic analysis revealed that the virus strain belongs to lineage IV, which is different to some previously isolated PPR strains from the Arabian Peninsula. This study shows that wild ruminants may play an important epidemiological role as virus source for domestic small ruminants.

APA, Harvard, Vancouver, ISO, and other styles

4

Sen, Arnab, Paramasivam Saravanan, Vinayagamurthy Balamurugan, Kaushal Kishor Rajak, Shashi Bhushan Sudhakar, Veerakyathappa Bhanuprakash, Satya Parida, and Raj Kumar Singh. "Vaccines against peste des petits ruminants virus." Expert Review of Vaccines 9, no. 7 (July 2010): 785–96. http://dx.doi.org/10.1586/erv.10.74.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Banyard, Ashley C., Zhiliang Wang, and Satya Parida. "Peste des Petits Ruminants Virus, Eastern Asia." Emerging Infectious Diseases 20, no. 12 (December 2014): 2176–78. http://dx.doi.org/10.3201/eid2012.140907.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Kul, O., N. Kabakci, H. T. Atmaca, and A. Özkul. "Natural Peste des Petits Ruminants Virus Infection: Novel Pathologic Findings Resembling Other Morbillivirus Infections." Veterinary Pathology 44, no. 4 (July 2007): 479–86. http://dx.doi.org/10.1354/vp.44-4-479.

Full text

Abstract:

The present study describes pathologic and virologic findings in 15 sheep and 6 goats that died of natural peste des petits ruminants virus infection in Turkey. Pathologic findings included erosiveulcerative stomatitis, fibrino-necrotic tracheitis, bronchointerstitial pneumonia, multifocal coagulation necroses in the liver, and severe lymphocytolysis in lymphoid tissues. Syncytial cells were conspicuous, especially in the oral mucosa, pulmonary alveoli, liver, and lymphoid tissues. In addition to the typical tissue distribution, eosinophilic intracytoplasmic and/or intranuclear inclusions were observed in epithelial cells lining the renal pelvis and abomasal mucosa. Immunolabeling of the viral antigen was observed in the kidney, brain, rumen, abomasum, heart, and myocytes of the tongue besides its more typical locations. In this study, we report and describe in detail the first peste des petits ruminants endemic in Kirikkale Province, Central Anatolia of Turkey. In conclusion, these previously unreported pathologic findings in natural peste des petits ruminants virus infection establish a basis for resemblance to other morbillivirus infections, such as canine distemper and distemper of sea mammals. Reverse transcriptase-polymerase chain reaction analyses indicated that the 448-bp genome fragment was amplified in 18 cases (18/21, 85.7%). Phylogenetic analysis showed that viruses belong to lineage 4 in the peste des petits ruminants virus common phylogenetic tree.

APA, Harvard, Vancouver, ISO, and other styles

7

Manin, B. L., L. V. Malakhova, A. B. Sarbasov, and N. V. Moroz. "CYTOMORPHOLOGICAL TRANSFORMATIONS IN YADK-04 CELLS DURING INTERACTION WITH PESTE DE PETITS RUMINANTS VIRUS." Veterinary Science Today, no. 2 (June 28, 2019): 41–45. http://dx.doi.org/10.29326/2304-196x-2019-2-29-41-45.

Full text

Abstract:

The paper presents experimental study results of the cytopathic effect of peste de petits ruminants virus on a goat gonad continuous cell line (YaDK-04). The interaction of peste de petits ruminants virus with cells at different stages of its reproduction was shown using a combination of phase-contrast and luminescent microscopy. It was found that at the initial stage of interaction (20–24 hours) the cells became rounded and de-adhered, and the monolayer was partially loosened. On day 2 post reproduction the most part of the culture monolayer affected by the virus began to destruct, and the cell nuclei were displaced to periphery. At the terminal stage (72 hours) the destruction of monolayer cells and cytoplasmic matrix, deformation and partial lysis of the nuclei and cytoplasm, aggregation of detritus occurred. At the final stage of reproduction (96 hours) the peste de petits ruminants virus diffused into the culture medium, the fluorescence in the yellow spectrum decreased significantly, but the virus titer reached 6.89 lg TCD50/cm3.

APA, Harvard, Vancouver, ISO, and other styles

8

Muniraju, Murali, Muhammad Munir, AravindhBabu R. Parthiban, Ashley C. Banyard, Jingyue Bao, Zhiliang Wang, Chrisostom Ayebazibwe, et al. "Molecular Evolution of Peste des Petits Ruminants Virus." Emerging Infectious Diseases 20, no. 12 (December 2014): 2023–33. http://dx.doi.org/10.3201/eid2012.140684.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Sghaier, Soufien, Gian Mario Cosseddu, Sonia Ben Hassen, Salah Hammami, Héni Haj Ammar, Antonio Petrini, and Federica Monaco. "Peste des Petits Ruminants Virus, Tunisia, 2012–2013." Emerging Infectious Diseases 20, no. 12 (December 2014): 2184–86. http://dx.doi.org/10.3201/eid2012.141116.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Wang, Zhiliang, Jingyue Bao, Xiaodong Wu, Yutian Liu, Lin Li, Chunju Liu, Longciren Suo, et al. "Peste des Petits Ruminants Virus in Tibet, China." Emerging Infectious Diseases 15, no. 2 (February 2009): 299–301. http://dx.doi.org/10.3201/eid1502.080817.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Peste des petits ruminants virus"

1

Dash, Pradyot. "Development of reverse genetics for Peste des Petits Ruminants Virus." Thesis, Royal Veterinary College (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522189.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Gopilo, Abraham. "Epidemiology of peste des petits ruminants virus in Ethiopia and molecular studies on virulence." Phd thesis, Toulouse, INPT, 2005. http://oatao.univ-toulouse.fr/7414/1/gopilo.pdf.

Full text

Abstract:

Peste des petits ruminants (PPR) is an acute and highly contagious viral disease of small ruminants, which is characterised by high fever, ocular and nasal discharge, pneumonia, necrosis and ulceration of the mucuous membrane and inflammation of the gastro-intestinal tract leading to severe diarrhoea and high mortality. In Africa, goats are severely affected while sheep undergo a mild form or rarely suffer clinical disease. PPR is one of the most important economical diseases in Ethiopia. Clinical PPR is confirmed in Ethiopian goats, however, its circulation in other animals has never been described. In the present work, we showed that the antibody seroprevalence in camel, cattle, goat and sheep confirmed natural transmission in these animals without clinical disease. The apparent absence of pathogenicity in these animals may have been due to host resistance or loss of virulence of the virus strain. We have further investigated the latter point by in vitro studies on PPRV comparing strains from Ethiopia and other countries with the vaccine strain which has been attenuated after several cell culture passages. In a first approach, virulence of PPRV was monitored in cell culture system and the use of virus specific monoclonal antibodies enabled to detect differences in virulence between PPRV and RPV. Vero (primate origin) and 293T (human) cell lines supported virus replication permitting the in vitro growth of both PPRV and RPV. In contrast to RPV, B95a (marmoset B) cells infected with PPRV were non permissive. The capability of cells to support active virus replication, which may result in intercellular spread and induce damages in infected cells, has implications on the pathogenesis and epidemiology. Cellular receptors are major determinants of host range and tissue tropism of a virus. The difference in infectivity of PPRV and RPV may have depended on the H protein epitopes and their cellular receptors. Therefore, we decided to compare the amino acid epitope of H protein of PPRV with that of other morbilliviruses. As part of our investigation of virulence factors, we have sequenced and compared genome and antigenome promoters of a vaccine strain with field strains of PPRV. The promoters contain the polymerase binding sites to initiate and generate the positive-strand replication and transcription of mRNAs. Nucleotide base change differences between vaccine strain and field strains would provide molecular basis for attenuation. Alignment of the genome promoter sequences revealed seven nucleotide mutations at certain positions. Our finding on nucleotide mutation on PPRV are in agreement with the nucleotide changes in rinderpest virus and other morbillivirus promoter regions between vaccine strain and wild type virus. Certain mutations were specific to PPRV. The promoter sequences were clustered around the geographic origin of the viruses and were lineage specific. Phylogenetic analysis of PPRV promoters was used for PPR phylogeograhy, and for comparison with other paramyxoviruses. The thesis is divided in 6 chapters. The first chapter deals with the natural history of PPR including the virus, the genome, epidemiology, transmission, clinical signs, immunology, diagnosis, control and its economic cost in the low income subsistence farming systems in sub-Saharan Africa. The second chapter is about comparative biology of PPRV with regard to other groups of morbillivirus genus in the Paramyxoviridae family. The third chapter deals with field study and observations on epidemiology of PPR in Ethiopia. In chapter four, PPRV virulence was monitored in cell culture system and comparison of H protein epitopes. In chapter five, sequence analysis of genome and antigenome promoters of PPRV was described In chapter six, general discussion and recommendations were forwarded.

APA, Harvard, Vancouver, ISO, and other styles

3

Sanz, Bernardo Beatriz. "Control of host innate immune (interferon) responses by peste des petits ruminants virus (PPRV)." Thesis, St George's, University of London, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703281.

Full text

Abstract:

Peste des petits ruminants virus (PPRV) produces clinical disease in goats and sheep. PPRV is a morbillivirus, others of which are known to affect the activation of the innate immune response by the actions of their accessory proteins (V&C). A crucial part of normal activation is the induction of interferon β (IFNβ) after pathogen recognition by pattern recognition receptors (PRRs). The presence of viral RNA can be sensed by the cytosolic proteins MDA-5 and RIG-I, starting a signalling cascade that leads to the activation of the IFNβ promoter and the synthesis of IFNβ. The production of IFNβ is a defence mechanism to control the spread of infection to neighbouring cells. Many viruses have evolved to antagonize this cell response. In this thesis I present a study of the induction of IFNβ following PPRV infection in both goat and primate cells, and the effects of infection with PPRV on the induction of IFNβ following MDA-5 and RIG-I mediated activation. Using both reporter assays and direct measurement of IFNβ mRNA, I found that PPRV infection does not induce IFNβ and can block the activation of expression of IFNβ. A study of the interaction of the PPRV accessory proteins with MDA-5 and RIG-I was carried out, including the cloning of goat RIG-I and LGP2. I also generated mutant viruses that lack expression of either accessory protein to characterize the role of these proteins in IFNβ induction during virus infection. Overexpression of V blocks MDA-5 mediated induction of IFNβ, but PPRV lacking V can still block MDA-5 mediated activation of IFNβ. PPRV C bound to neither MDA-5 nor RIG-I, but PPRV lacking C lost the ability to block MDA-5 and RIG-I mediated activation of IFNβ. These results shed new light on the inhibition of the induction of IFNβ by PPRV.

APA, Harvard, Vancouver, ISO, and other styles

4

Gopilo, Abraham Picavet Dominique-Pierre. "Epidemiology of peste des petits ruminants virus in ethiopia and molecular studies on virulence." Toulouse : INP Toulouse, 2006. http://ethesis.inp-toulouse.fr/archive/00000226.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Woma, Timothy Yusufu. "Epidemiology of peste-des-petits-ruminants virus in sheep goats and camels in Nigeria." Thesis, University of Pretoria, 2015. http://hdl.handle.net/2263/53316.

Full text

Abstract:

Peste-des-petits-ruminants (PPR), caused by the peste-des-petits ruminants virus (PPRV) is endemic in Nigeria and is a major constraint to the improvement of small ruminant production, thereby affecting the rural poor who depends on these livestock species as their source of livelihoods. The major objectives of this work were: 1) to conduct a seroprevalence of PPRV in camels, sheep and goats in Nigeria; 2) to isolate PPRV from naturally infected animals; and 3) to characterize the isolates using molecular techniques and compare the profile of the current isolates with the worldwide vaccine (Nig75/1) and other existing strains from other parts of the world. The major contribution of this work to current knowledge were: finding that more than one lineage of PPRV was circulating in Nigeria, the emerging Asian lineage IV gradually replacing the traditional lineage II in the country; the isolation in cell cultures and full genome sequencing of current field viruses which was last done in 1976; the unique amino acid residues in the individual proteins of the various lineages of PPRV were identified and used to develop a lineage-specific diagnostic test for the virus; and the current seroprevalence report of PPR in camels, sheep and goats from all the agro-ecological zones of the country. This work has also increased the sequences of PPRV available in GenBank. A total of 6,065 field serum samples from camels (1,517), goats (3,489) and sheep (1,059) were collected from all the six agro-ecological zones of Nigeria. The study subjects cut across all ages and sexes. The samples were subjected to both the N and H protein based c-ELISA. In addition, 140 clinical samples from 16 sheep and 63 goats with symptoms suggestive of PPRV infection were collected from different (15) states of Nigeria during a four year period (2010 2013). Published primers were used to amplify a 351 bp segment of the PPRV nucleoprotein (N) gene and a 370 bp segment of the fusion (F) gene. Monkey CV 1 cell line expressing the sheep-goat SLAM protein was used to isolate PPRV from RT-PCR positive samples. Thirty primer pairs were used to amplify and sequence two full genome isolates of the current circulating PPRV in Nigeria. Primer express software was used to develop a lineage-specific real time PCR for PPRV. The overall prevalence estimate of serum positive results for PPRV in sheep and goats was 23.16 % (n = 1,018/4,548, 95 % confidence interval [CI]: 21.79 - 24.57. There were significant differences in prevalences between the states (p = 0.001), zones (p = 0.058) and age (p = 0.032). Taraba State had the highest seroprevalence rate of 27.97 % while the lowest rate of 14.76 % was observed in Cross River State. There were no significant differences in the PPRV prevalences between male and female animals (p = 0.571) and between species (p = 0.639). The overall prevalence in camels was 3.36 % (51/1517, 95 % CI: 2.51 4.39). There were no significant differences in prevalences between states (p = 0.892) and between male and female camels (p = 0.742). The prevalence differed significantly (p < 0.001) by body condition scores - camels with poor body condition score have a higher (16.67 %) antibody seroprevalences to PPRV compared to those with fair and good body condition scores. There was a statistically significant difference between camels aged ? 5 years and those > 5 years (p = 0.004). The clinical samples subjected to RT-PCR showed that 33 (42 %) animals were positive for PPRV nucleic acid, which included two sheep (13 %) and 31 goats (49 %). The amplicons were sequenced and phylogenetic analysis using the neighbour joining, maximum parsimony and Bayesian analysis of the sequences with those available in GenBank showed that isolates from the current study belonged to lineage II and lineage IV. The lineage II viruses from this study grouped into two clades, one closely related to the vaccine virus (Nigeria75/1) and the other clade group with other wild-type viruses from Mali, Senegal and Sierra Leone. The lineage IV isolates also grouped into two sub-clades, one closely related to a 2011 strain from Gabon and the other closely related to a 1997 strain from Cameroon. Analysis of two full genomes of PPRV from Nigeria representing the two lineages of the virus circulating currently in the country showed that the lineage II representative isolated in 2012 has an identity of 96 % at the nucleotide level with the lineage II Nigeria75/1 virus isolated in 1975 and used as a vaccine. The lineage IV representative isolated in 2013 revealed an identity of 96 % with Sungri/96 from India. The unique amino acid residues in the individual proteins of the various lineages of PPRV were identified and used to develop a one-step TaqMan® real-time RT-PCR assay targeting the H gene of PPRV. This study reports for the first time, the presence of PPRV lineage IV, the Asian lineage in Nigeria. As part of the study, a lineage-specific assay was also developed, which once validated, could be included in the panel of assays recommended by the World Organization for Animal Health (OIE). The seroprevalence studies showed occasional transient PPRV infection of camels and the current antibody seroprevalence to PPRV in the small ruminants population in Nigeria. There is the need to include camels among species to be studied in elucidating the epidemiology of the disease in sheep and goats. The seroprevalence studies may be helpful in reaching a decision on the vaccination strategy for the control of the disease in the country.
Thesis (PhD)--University of Pretoria, 2015.
tm2016
Veterinary Tropical Diseases
PhD

APA, Harvard, Vancouver, ISO, and other styles

6

Buczkowski, Hubert. "Development of marker vaccines for rinderpest (RPV) and peste des petits ruminants (PPRV) viruses." Thesis, Royal Veterinary College (University of London), 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558958.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Bodjo, Sanne Charles. "Etude de la nucleocapside des virus de la peste bovine et peste des petits ruminants : caractérisation moléculaire des interactions protéiques et des sites antigénique." Montpellier 2, 2007. http://www.theses.fr/2007MON20073.

Full text

Abstract:

L'étude des structures antigéniques des nucléoprotéines (N) des virus de la peste bovine et de la peste des petits ruminants a montré que la région N-terminale est immunodominante. Les études de cartographie d’anticorps monoclonaux anti-N ont permis de localiser dans cette région des épitopes communs mais aussi des épitopes spécifiques à chacun des deux virus. Les tests de compétition ELISA (cELISA), développés avec certains de ces monoclonaux spécifiques à chaque virus détectent malheureusement aussi bien des sérums anti-PPR et qu'anti-peste bovine. Ce croisement serait probablement la résultante d’encombrement stérique engendré par des anticorps fixés sur les épitopes communs aux deux virus mais proches des sites spécifiques. Une partie de nos travaux a porté sur l'analyse des interactions protéine-protéine du virus PPR. Ils ont permis de localiser le domaine d'interaction N-N dans la zone couverte par les 240 premiers aminoacides de N. Quatre régions capables de se lier à la protéine M ont été identifiées sur la protéine N. Les séquences de ces zones d’interaction N-M sont conservées au sein groupe Morbillivirus

APA, Harvard, Vancouver, ISO, and other styles

8

Mantip, Samuel Elias Lashat. "Molecular characterisation of peste des petits ruminants viruses in sheep and goats from Nigeria." Diss., University of Pretoria, 2013. http://hdl.handle.net/2263/40708.

Full text

Abstract:

Peste des petits ruminants virus (PPRV) belongs to the family Paramyxoviridae and genus morbillivirus. It is a highly contagious, fatal and economically important viral disease of small ruminants that is still endemic and militate against the production of sheep and goats in Nigeria. It is a notifiable disease according to the World Organization for Animal Health (Office International des Epizooties). In this study, a molecular analysis of PPRV from sheep and goats from recent outbreaks across the different regions of Nigeria was carried out. The aim was to describe the viral strains and the movement of the virus within the country compared to other endemic areas of the world. This was carried out through tissue and swab samples collected from sheep and goats in various agro-ecological zones of Nigeria.The evolution and relationship of earlier PPRV strains/isolates and those circulating and causing recent outbreaks was determined by sequencing of the nucleoprotein (N)-gene. Twenty tissue and swab samples from apparently healthy and sick sheep and goats were collected randomly from each of three states of each of the six agro-ecological zones visited. A total of 360 samples were collected. A total of 35 samples of 360 (9.7 %) tested positive by RT-PCR, of which 25 were from oculo-nasal swabs and 10 were from tissue samples (Table 4.2). Phylogenetic analysis was carried out using the N-gene sequences of the PPRV amplicons. Alignment of the sequences and related sequences from GenBank and neighbor-joining phylogenetic analysis using PAUP identified four different lineages, i.e. lineages I, II, III and IV. Interestingly, the Nigerian strains described in this study grouped in two separate major lineages i.e. lineages II and IV. Strains from Sokoto, Oyo, Plateau and Ondo states grouped according to the historical distribution of PPRV together with the Nigerian 75/1 strain of lineage II, while other strains from Sokoto, Oyo, Plateau, Akwa-Ibom, Adamawa, Kaduna, Lagos, Bauchi, Niger and Kano states grouped together with the East-African and Asian strains of lineage IV. This finding suggests that both lineages II and IV strains of PPRVs are circulating presently in Nigeria, contrary to an earlier publication which indicated that only strains of lineage II were circulating in the country (Shamaki, 2002).
Dissertation (MSc)--University of Pretoria, 2013.
gm2014
Veterinary Tropical Diseases
unrestricted

APA, Harvard, Vancouver, ISO, and other styles

9

Salami, Habib. "Diffusion d'un virus et évolution de son génome dans les populations de ruminants domestiques : application à l'épidémiosurveillance de la "Peste des petits ruminants"." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONTS155/document.

Full text

Abstract:

La peste des petits ruminants (PPR), causée par un Morbillivirus, est l'infection virale la plus grave des caprins et ovins. Elle est largement répandue en Asie, au Moyen Orient et en Afrique. En Afrique elle est en émergence au nord et au sud du continent et représente un facteur majeur d'insécurité alimentaire pour la population agricole (70% des populations pauvres des régions considérées). La PPR est un modèle d'étude des maladies transfrontalières ; sa diffusion est très liée aux mouvements régionaux d'animaux vivants. La compréhension de cette diffusion est une condition essentielle à la mise en place de mesures de contrôle efficaces (vaccination, quarantaine, contrôle aux frontières,…). A notre connaissance aucune étude n'a été entreprise pour connaître l'ampleur de la diversité génétique du PPRv au cours d'infections naturelles de petits ruminants et l'accumulation des mutations virales dans un circuit de diffusion. Or dans les pays d'élevages extensifs tropicaux l'identification et la traçabilité animale sont inexistantes, ce qui rend difficile reconstruction des circuits de diffusion des animaux et du virus. Dans ces conditions, la diversité génétique du virus peut être utilisée comme marqueur de diffusion épidémiologique. L'objectif de cette thèse est d'utiliser la variabilité génétique du PPRV pour caractériser les lignées virales circulantes et retracer les processus de transmission du virus à travers un large territoire centré sur le Sénégal. En analysant 2 gènes de PPR nous avons estimé la vitesse d'évolution du virus sur une période de 4 années comprise en 2010 et 2014.Les résultats montrent que les premières souches de la lignée 2 de PPRv ont été introduites en 2005 au Sénégal et dans les pays voisins. L'horloge moléculaire et l'arbre phylogéographique rapportés ici indiquent clairement que la lignée II maintenant enzootique en Afrique de l'ouest prend son origine au Nigeria. Les mouvements trans-africains à l'origine du déplacement est-ouest de la lignée II trouvent leur origine dans le commerce de bétail à la croisée des frontières, une évidence économique et culturelle en Afrique de l'Ouest.Mots clés : peste des petits ruminants ; gène viral ; mutation virale ; circuit de transmission ; phylogénie ; phylogéographie ; surveillance épidémiologique, Sénégal
Peste des petits ruminants (PPR), caused by a Morbillivirus is one of the most important viral infections in sheep and goats. It is widely spread in Asia, Middle East and Africa. In Africa, it is an emerging disease in the north and the south of the continent. It is a major factor of food insecurity for the farming population (70% of the poor population in the tropical regions). PPR is a study model of transboundary diseases; its spread is highly related to regional movements of livestock. Understanding the spread of PPR is an essential condition for the implementation of efficient control measures (vaccination, quarantine, border controls etc.). Up to our knowledge, no studies have investigated the range of genetic diversity of PPR virus (PPRv) during natural infections in small ruminants and the accumulation of virus mutations during its spread. Further on, in tropical countries with extensive farming, animal identification and traceability are a current problem. In such conditions, the genetic diversity of the PPRv can be used as a marker of animal movement and spread of the virus. The objective of this study was to investigate the genetic diversity of the PPRv in order to characterise the actual viral lineages and to retrace the transmission of the virus in Senegal and its surrounding countries. Analyzing two complete viral genes of the PPR, we have estimated the rate of evolution of this virus, in a four year period, between 2010 and 2014. The results of the study show that the first strains of lineage II of PPRv have been introduced in 2005 in Senegal and its surrounding countries. Molecular clock analysis and phylogeographical reconstitution of the PPRv indicate that the lineage II, actually enzootique in western Africa, has its origins in Nigeria. This viral introduction from the direction east towards west, corresponds to the transboundary movement and commerce of livestock in the countries of western Africa, which represents the economic and cultural tradition of the people of this region.Key words: Peste des petits ruminants, viral gene, virus mutation, transmission, phylogeny, phylogéographie, epidemiosurveillance, Senegal, West Africa

APA, Harvard, Vancouver, ISO, and other styles

10

Bailey, Dalan. "Development of reverse genetics for peste des petets ruminants virus (PPRV)." Thesis, University of Reading, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.494788.

Full text

Abstract:

Peste des petits ruminants virus (PPRV), a member of the Morbillivims genus in the family Paramyxoviridae, is the cause of a serious and emerging plague of small ruminants, mostly affecting sheep and goats. It is endemic in many developing countries in Africa and southern Asia and is highly contagious, with the mortality rate approaching 90% in some outbreaks. Development of reverse genetics systems for other morbilliviruses such as rinderpest and measles has allowed more focused research into replication, pathogenicity and marker vaccines.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Peste des petits ruminants virus"

1

Munir, Muhammad, ed. Peste des Petits Ruminants Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45165-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Munir, Muhammad, Siamak Zohari, and Mikael Berg. Molecular Biology and Pathogenesis of Peste des Petits Ruminants Virus. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-31451-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Food and Agriculture Organization of the United Nations., ed. Recognizing peste des petits ruminants: A field manual. Rome: Food and Agriculture Organization of the United Nations, 1999.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

US DEPARTMENT OF AGRICULTURE. Keeping America free from foreign animal diseases. 6. Rinderpest, peste des petits ruminants. [Washington, D.C.]: U.S. Dept. of Agriculture, 1997.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

Munir, Muhammad. Peste des Petits Ruminants Virus. Springer, 2016.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Munir, Muhammad. Peste des Petits Ruminants Virus. Springer Berlin / Heidelberg, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

Munir, Muhammad. Peste des Petits Ruminants Virus. Springer, 2014.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

Thomas, Barrett, Pastoret Paul-Pierre, and Taylor W. P, eds. Rinderpest and peste des petits ruminants: Virus plagues of large and small ruminants. London: Academic, 2006.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

Barrett, Thomas, William P. Taylor, and Paul-Pierre Pastoret. Rinderpest and Peste des Petits Ruminants: Virus Plagues of Large and Small Ruminants. Elsevier Science & Technology Books, 2005.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

Molecular Biology and Pathogenesis of Peste des Petits Ruminants Virus. Springer, 2012.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Book chapters on the topic "Peste des petits ruminants virus"

1

de Haan, N. C., T. Kimani, J. Rushton, and J. Lubroth. "Why Is Small Ruminant Health Important—Peste des Petits Ruminants and Its Impact on Poverty and Economics?" In Peste des Petits Ruminants Virus, 195–226. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_12.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Munir, Muhammad. "Peste des Petits Ruminants: An Introduction." In Peste des Petits Ruminants Virus, 1–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Parida, Satya, Emmanuel Couacy-Hymann, Robert A. Pope, Mana Mahapatra, Medhi El Harrak, Joe Brownlie, and Ashley C. Banyard. "Pathology of Peste des Petits Ruminants." In Peste des Petits Ruminants Virus, 51–67. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Kul, Oguz, Hasan Tarık Atmaca, and Muhammad Munir. "Pathology of Peste des Petits Ruminants Virus Infection in Small Ruminants and Concurrent Infections." In Peste des Petits Ruminants Virus, 119–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Renukaradhya, Gourapura J., and Melkote S. Shaila. "Host Immune Responses Against Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 171–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_10.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Singh, R. K., K. K. Rajak, D. Muthuchelvan, Ashley C. Banyard, and Satya Parida. "Vaccines Against Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 183–94. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_11.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Dhinakar Raj, G., A. Thangavelu, and Muhammad Munir. "Strategies and Future of Global Eradication of Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 227–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_13.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Baron, Michael D. "The Molecular Biology of Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 11–38. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_2.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Balamurugan, Vinayagamurthy, Habibur Rahman, and Muhammad Munir. "Host Susceptibility to Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 39–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Banyard, Ashley C., and Satya Parida. "Molecular Epidemiology of Peste des Petits Ruminants Virus." In Peste des Petits Ruminants Virus, 69–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45165-6_5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Peste des petits ruminants virus"

1

Li, Guili, Yin Wang, Xueping Yao, and Ling Hu. "Establishment of a duplex RT-PCR assay for simultaneous detection of Rift valley fever virus and peste des petits ruminants virus." In 2015 International Conference on Materials, Environmental and Biological Engineering. Paris, France: Atlantis Press, 2015. http://dx.doi.org/10.2991/mebe-15.2015.92.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Peste des petits ruminants virus"

1

Lignes directrices pour le contrôle et la prévention de la peste des petits ruminants (PPR) dans les populations de faune sauvage. OIE (World Organisation for Animal Health), December 2021. http://dx.doi.org/10.20506/ppr.3274.

Full text

Abstract:

La peste des petits ruminants (PPR) est une maladie animale des petits ruminants domestiques et des artiodactyles sauvages, très répandue, virulente et dévastatrice, causée par le virus de la peste des petits ruminants, un morbillivirus. Le taux de mortalité peut dépasser 90 %, en particulier dans les populations naïves au plan immunologique, souffrant de malnutrition et soumises à des stress. Ces lignes directrices sont destinées à aider les pays à élaborer et à mettre en œuvre leur programme d’éradication de la PPR ; elles incluent des objectifs, des politiques et des stratégies qui sont adaptables à l’ensemble des besoins nationaux et qui favorisent l’intégration du secteur en charge de la faune sauvage dans le plan stratégique national.

APA, Harvard, Vancouver, ISO, and other styles

2

Guidelines for the control and prevention of peste des petits ruminants (PPR) in wildlife populations. OIE, 2021. http://dx.doi.org/10.20506/ppr.2943.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Le déploiement de la stratégie mondiale de contrôle et d’éradication de la peste des petits ruminants en Afrique. O.I.E (World Organisation for Animal Health), 2017. http://dx.doi.org/10.20506/tt.2656.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

12th Meeting of the Global Steering Committee of the Global Framework for the Progressive Control of Transboundary Animal Diseases (GF-TADs). Report of the meeting, 2 November and 2 December 2021. WOAH (World Organisation for Animal Health), September 2022. http://dx.doi.org/10.20506/gftads.3327.

Full text

Abstract:

This report provides an overview of the main activities on global priority transboundary animal diseases (TADs) since the last Global steering Committee in November and December 2020. It covers, by alphabetic order, African Swine Fever (ASF), Foot and Mouth disease (FMD), Peste des Petits Ruminants (PPR) and Rinderpest post eradication programme (RP). Sources of funding (financial information) are presented in Annex I and a follow up of action plan, adopted following the 3rd external evaluation of GF-TADs, is presented in Annex II. Information on specific activities can also be found through the dedicated sections of the GF-TADs website or on request to the GF-TADs global secretariat. The GF-TADs global secretariat thanks the members of these disease working groups and secretariat for their support to prepare this document.

APA, Harvard, Vancouver, ISO, and other styles

5

12th Meeting of the Global Steering Committee of the Global Framework for the Progressive Control of Transboundary Animal Diseases (GF-TADs). Activity report. O.I.E (World Organisation for Animal Health), October 2022. http://dx.doi.org/10.20506/gftads.3204.

Full text

Abstract:

This report provides an update on the progress made in the control of global priority transboundary animal diseases (TADs), which was presented by the disease working groups and secretariats at the Global Steering Committee meeting (GSC12), held during two sessions in November and December 2021. It presents a concise summary of activities carried out under the Global Framework for the Progressive Control of Transboundary Animal Diseases (GF-TADs) umbrella from November 2020 to October 2021. The TADs covered in the report include African swine fever, foot-and-mouth disease and peste des petits ruminants, as well as the rinderpest post-eradication programme. The main areas of focus include a summary of TADs control strategies, the epidemiological situation in the past two years, as well as the progress and challenges faced in the last year. Regional activities for the five main geographical regions of the GF-TADs are also highlighted in the report. Funding support for TADs control is presented in Annex I and a follow-up of the action plan developed following the recommendations of the third external evaluation of GF-TADs is presented in Annex II. Information on specific activities mentioned in the report can also be found through the dedicated sections of the GF-TADs website or by request from the GF-TADs global secretariat.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!