Relevant bibliographies by topics / PFN1

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'PFN1'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 June 2025

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Journal articles on the topic "PFN1"

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Gau, David M., Abigail Allen, Andrew Daoud, Jessica Kunkel, Stefan Duensing, and Partha Roy. "Abstract 4584: Genetic disruption of vascular endothelial profilin-1 impacts tumor microenvironment supressing tumorigenicity of renal cancer." Cancer Research 83, no. 7_Supplement (2023): 4584. http://dx.doi.org/10.1158/1538-7445.am2023-4584.

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Abstract Renal cell carcinoma (RCC) is estimated to result in 79,000 new cases and 13,920 deaths in 2022. Clear cell renal cell carcinoma (ccRCC) is the most common subtype of RCC and is characterized by a highly vascularized tumor microenvironment (TME). While current anti-angiogenic therapies targeting VEGF signaling are initially effective, almost all patients develop resistance to these therapies. Therefore, there is a need to identify clinically relevant alternative molecular targets to suppress tumor angiogenesis and progression in ccRCC. We previously discovered transcriptional upregula
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Teyssou, Elisa, Laura Chartier, Delphine Roussel, et al. "The Amyotrophic Lateral Sclerosis M114T PFN1 Mutation Deregulates Alternative Autophagy Pathways and Mitochondrial Homeostasis." International Journal of Molecular Sciences 23, no. 10 (2022): 5694. http://dx.doi.org/10.3390/ijms23105694.

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Mutations in profilin 1 (PFN1) have been identified in rare familial cases of Amyotrophic Lateral Sclerosis (ALS). PFN1 is involved in multiple pathways that could intervene in ALS pathology. However, the specific pathogenic role of PFN1 mutations in ALS is still not fully understood. We hypothesized that PFN1 could play a role in regulating autophagy pathways and that PFN1 mutations could disrupt this function. We used patient cells (lymphoblasts) or tissue (post-mortem) carrying PFN1 mutations (M114T and E117G), and designed experimental models expressing wild-type or mutant PFN1 (cell lines
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Schmidt, Eric J., Salome Funes, Jeanne E. McKeon, et al. "ALS-linked PFN1 variants exhibit loss and gain of functions in the context of formin-induced actin polymerization." Proceedings of the National Academy of Sciences 118, no. 23 (2021): e2024605118. http://dx.doi.org/10.1073/pnas.2024605118.

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Profilin-1 (PFN1) plays important roles in modulating actin dynamics through binding both monomeric actin and proteins enriched with polyproline motifs. Mutations in PFN1 have been linked to the neurodegenerative disease amyotrophic lateral sclerosis (ALS). However, whether ALS-linked mutations affect PFN1 function has remained unclear. To address this question, we employed an unbiased proteomics analysis in mammalian cells to identify proteins that differentially interact with mutant and wild-type (WT) PFN1. These studies uncovered differential binding between two ALS-linked PFN1 variants, G1
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Nekouei, Mina, Atousa Aliahmadi, Mahmoud Kiaei, and Ali Reza Ghassempour. "Mutant Profilin1 Aggregation in Amyotrophic Lateral Sclerosis: An in Vivo Biochemical Analysis." Basic and Clinical Neuroscience Journal 12, no. 2 (2021): 213–22. http://dx.doi.org/10.32598/bcn.12.2.1631.1.

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Introduction: Profilin1 (PFN1) is a ubiquitously expressed protein known for its function as a regulator of actin polymerization and dynamics. A recent discovery linked mutant PFN1 to Amyotrophic Lateral Sclerosis (ALS), which is a fatal and progressive motor neuron disease. We have also demonstrated that Gly118Val mutation in PFN1 is a cause of ALS, and the formation of aggregates containing mutant PFN1 may be a mechanism for motor neuron death. Hence, we were interested in investigating the aggregation of PFN1 further and searching for co-aggregated proteins in our mouse model overexpressing
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Boopathy, Sivakumar, Tania V. Silvas, Maeve Tischbein, et al. "Structural basis for mutation-induced destabilization of profilin 1 in ALS." Proceedings of the National Academy of Sciences 112, no. 26 (2015): 7984–89. http://dx.doi.org/10.1073/pnas.1424108112.

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Mutations in profilin 1 (PFN1) are associated with amyotrophic lateral sclerosis (ALS); however, the pathological mechanism of PFN1 in this fatal disease is unknown. We demonstrate that ALS-linked mutations severely destabilize the native conformation of PFN1 in vitro and cause accelerated turnover of the PFN1 protein in cells. This mutation-induced destabilization can account for the high propensity of ALS-linked variants to aggregate and also provides rationale for their reported loss-of-function phenotypes in cell-based assays. The source of this destabilization is illuminated by the X-ray
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Zheng, Junke, and Chengcheng Zhang. "Profilin 1 Is Essential for Retention and Metabolism of Hematopoietic Stem Cells in Bone Marrow." Blood 120, no. 21 (2012): 1224. http://dx.doi.org/10.1182/blood.v120.21.1224.1224.

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Abstract Abstract 1224 How stem cells interact with the microenvironment to regulate their cell fates and metabolism is largely unknown. Here we show that, in a hematopoietic stem cell (HSC) -specific inducible knockout model, the cytoskeleton-modulating protein profilin 1 (pfn1) is essential for the maintenance of multiple cell fates and metabolism of HSCs. The deletion of pfn1 in HSCs led to bone marrow failure, loss of quiescence, increased apoptosis, and mobilization of HSCs in vivo. In reconstitution analyses, pfn1-deficient cells were selectively lost from mixed bone marrow chimeras. By
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Lee, Chang-Jin, Seon-Hwa Hong, Min-Ji Yoon, et al. "Endometrial profilin 1: a key player in embryo-endometrial crosstalk." Clinical and Experimental Reproductive Medicine 47, no. 2 (2020): 114–21. http://dx.doi.org/10.5653/cerm.2019.03454.

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Objective: Despite extensive research on implantation failure, little is known about the molecular mechanisms underlying the crosstalk between the embryo and the maternal endometrium, which is critical for successful pregnancy. Profilin 1 (PFN1), which is expressed both in the embryo and in the endometrial epithelium, acts as a potent regulator of actin polymerization and the cytoskeletal network. In this study, we identified the specific role of endometrial PFN1 during embryo implantation.Methods: Morphological alterations depending on the status of PFN1 expression were assessed in PFN1-deple
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Lee, Chang-Jin, Min-Ji Yoon, Dong Hyun Kim, Tae Uk Kim, and Youn-Jung Kang. "Profilin-1; a novel regulator of DNA damage response and repair machinery in keratinocytes." Molecular Biology Reports 48, no. 2 (2021): 1439–52. http://dx.doi.org/10.1007/s11033-021-06210-6.

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AbstractProfilin-1 (PFN1) regulates actin polymerization and cytoskeletal growth. Despite the essential roles of PFN1 in cell integration, its subcellular function in keratinocyte has not been elucidated yet. Here we characterize the specific regulation of PFN1 in DNA damage response and repair machinery. PFN1 depletion accelerated DNA damage-mediated apoptosis exhibiting PTEN loss of function instigated by increased phosphorylated inactivation followed by high levels of AKT activation. PFN1 changed its predominant cytoplasmic localization to the nucleus upon DNA damage and subsequently restor
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Zi, Jingjing, Jing Xu, Jintang Luo, et al. "PFN1 Inhibits Myogenesis of Bovine Myoblast Cells via Cdc42-PAK/JNK." Cells 11, no. 20 (2022): 3188. http://dx.doi.org/10.3390/cells11203188.

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Myoblast differentiation is essential for the formation of skeletal muscle myofibers. Profilin1 (Pfn1) has been identified as an actin-associated protein, and has been shown to be critically important to cellular function. Our previous study found that PFN1 may inhibit the differentiation of bovine skeletal muscle satellite cells, but the underlying mechanism is not known. Here, we confirmed that PFN1 negatively regulated the myogenic differentiation of bovine skeletal muscle satellite cells. Immunoprecipitation assay combined with mass spectrometry showed that Cdc42 was a binding protein of P
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Wittenmayer, Nina, Burkhard Jandrig, Martin Rothkegel, et al. "Tumor Suppressor Activity of Profilin Requires a Functional Actin Binding Site." Molecular Biology of the Cell 15, no. 4 (2004): 1600–1608. http://dx.doi.org/10.1091/mbc.e03-12-0873.

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Profilin 1 (PFN1) is a regulator of the microfilament system and is involved in various signaling pathways. It interacts with many cytoplasmic and nuclear ligands. The importance of PFN1 for human tissue differentiation has been demonstrated by the findings that human cancer cells, expressing conspicuously low PFN1 levels, adopt a nontumorigenic phenotype upon raising their PFN1 level. In the present study, we characterize the ligand binding site crucial for profilin's tumor suppressor activity. Starting with CAL51, a human breast cancer cell line highly tumorigenic in nude mice, we establishe
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Dissertations / Theses on the topic "PFN1"

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Schmidt, Eric J. "Investigation of a Misfolded, Destabilized Profilin-1 Species as a Toxic Molecule in ALS Pathogenesis." eScholarship@UMMS, 2019. https://escholarship.umassmed.edu/gsbs_diss/1043.

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Dominant mutations in profilin-1 (PFN1) are associated with amyotrophic lateral sclerosis (ALS), a fatal neurodegenerative disease characterized by motor neuron loss, paralysis, and death from respiratory failure. Our lab recently demonstrated that PFN1 mutant proteins are destabilized—they unfold at milder conditions during thermal and chemical denaturation. Furthermore, we and others have shown that mutant PFN1 is more prone to misfold and aggregate. This misfolding alters PFN1’s protein-protein interactions, as demonstrated by an affinity purification-mass spectrometry screen. While ALS-ass
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Wu, Chi-Hong. "Functional Characterization of Novel PFN1 Mutations Causative for Familial Amyotrophic Lateral Sclerosis: A Dissertation." eScholarship@UMMS, 2015. https://escholarship.umassmed.edu/gsbs_diss/815.

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Amyotrophic lateral sclerosis (ALS) is a progressive adult neurodegenerative disease that causes death of both upper and lower motor neurons. Approximately 90 percent of ALS cases are sporadic (SALS), and 10 percent are inherited (FALS). Mutations in the PFN1 gene have been identified as causative for one percent of FALS. PFN1 is a small actin-binding protein that promotes actin polymerization, but how ALS-linked PFN1 mutations affect its cognate functions or acquire gain-of-function toxicity remains largely unknown. To elucidate the contribution of ALS-linked PFN1 mutations to neurodegenerati
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Wu, Chi-Hong. "Functional Characterization of Novel PFN1 Mutations Causative for Familial Amyotrophic Lateral Sclerosis: A Dissertation." eScholarship@UMMS, 2012. http://escholarship.umassmed.edu/gsbs_diss/815.

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Amyotrophic lateral sclerosis (ALS) is a progressive adult neurodegenerative disease that causes death of both upper and lower motor neurons. Approximately 90 percent of ALS cases are sporadic (SALS), and 10 percent are inherited (FALS). Mutations in the PFN1 gene have been identified as causative for one percent of FALS. PFN1 is a small actin-binding protein that promotes actin polymerization, but how ALS-linked PFN1 mutations affect its cognate functions or acquire gain-of-function toxicity remains largely unknown. To elucidate the contribution of ALS-linked PFN1 mutations to neurodegenerati
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Devecioglu, Semira [Verfasser], and Kerstin [Akademischer Betreuer] Kutsche. "Molekulargenetische Analysen bei Patienten mit Kleinhirnfehlbildungen : Sequenzanalyse der Gene SHH, CNPY1, EN2 und PFN1 / Semira Devecioglu. Betreuer: Kerstin Kutsche." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2014. http://d-nb.info/1053811942/34.

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Ingre, Caroline. "On the aetiology of ALS : a comprehensive genetic study." Doctoral thesis, Umeå universitet, Institutionen för farmakologi och klinisk neurovetenskap, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-67460.

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Introduction: Amyotrophic lateral sclerosis (ALS) is a deadly, progressive neuromuscular disease that affects individuals all over the world. About 10% of the patients have a familial predisposition (FALS) while the remainder of cases are isolated or sporadic (SALS) and of unknown cause. To date, the principal recognized risk factors for ALS are higher age, male gender, slim figure (BMI<23) and a family history of ALS. In 1993, Rosen et al. observed that some FALS cases were associated with mutations in the gene encoding the CuZn superoxide dismutase enzyme (SOD1). Since then, several mutat
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Weyrich, Joy [Verfasser]. "Ergebnisse der Versorgung pertrochantärer Frakturen mit dem PFNA / Joy Weyrich." Ulm : Universität Ulm. Medizinische Fakultät, 2014. http://d-nb.info/105682364X/34.

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Short, Nicholas J. "The DNA sequence of the filamentous bacteriophage Pf1." Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305822.

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Antiga, Enrico <1994&gt. "PFN attiva: i suoi vantaggi nelle imprese moderne." Master's Degree Thesis, Università Ca' Foscari Venezia, 2018. http://hdl.handle.net/10579/13645.

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L'elaborato si incentra sull'analisi delle disponibilità finanziarie (cassa, banca e titoli a breve termine) e della PFN: dapprima attraverso il loro significato teorico all'interno delle teorie di corporate finance susseguitesi nel XX secolo; successivamente attraverso un'analisi statistica dei trend su quei parametri relativa al Veneto, all'Italia e agli USA con differenti campioni. Nella conclusione si spiegano i motivi della crescita della liquidità (riscontrata nell'analisi) assieme al miglioramento della PFN, sottolineando i vantaggi della PFN attiva.
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Nunes, Patrique Nelson Ramos. "Dynamic and interaction of cytochrome c with Pf1 virus." Master's thesis, Faculdade de Ciências e Tecnologia, 2011. http://hdl.handle.net/10362/5662.

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Dissertação apresentada na Faculdade de Ciências e Tecnologia da Universidade Nova de Lisboa para obtenção do grau de Mestre em BioOrgânica<br>Cytochrome c is a positive protein and the Pf1 virus surface is negative forging strong electrostatic complex. When a critical ratio concentration of Cytochrome c and Pf1 virus is achieved a spontaneous complex is formed. The maximum association upon addition of cytochrome c to Pf1 solutions is about 1700 cytochrome c molecules to one Pf1 virion particle. The effect of univalent salt concentration on protein polyelectrolyte complex formation was measur
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Zwick, Eva [Verfasser], and Thomas [Akademischer Betreuer] Kretsch. "Analysen zur Funktion von PFT1, LAF1 und HY5 in der Lichtsignaltransduktion von Arabidopsis thaliana." Freiburg : Universität, 2013. http://d-nb.info/1123475377/34.

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Books on the topic "PFN1"

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D, Knight T., Metzger Vera, Los Alamos National Laboratory, and U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Accident Evaluation., eds. TRAC-PF1 independent assessment. Los Alamos National Laboraotory, 1985.

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U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Systems Research. and Los Alamos National Laboratory, eds. TRAC-PF1/MOD2 code manual. Division of Systems Research, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Systems Research. and Los Alamos National Laboratory, eds. TRAC-PF1/MOD2 code manual. Division of Systems Research, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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Bochenek, Dariusz. Technologia wytwarzania i właściwości multiferroikowej ceramiki typu PFN: Manufacturing technology and properties of the multiferroic PFN ceramics = [Tekhnologii︠a︡ poluchenii︠a︡ i svoĭstva mulʹtiferroika na primere keramiki tipa PFN]. Wydawnictwo Uniwersytetu Ślaskiego, 2012.

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1973-, Andraos Amale, Wood Dan 1967-, and P.S. 1 Contemporary Art Center., eds. Above the pavement-- the farm!: Architecture & agriculture at PF1. Princeton Architectural Press, 2010.

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A, Siebe Donald, Boyack B. E, U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Systems Research., and Los Alamos National Laboratory, eds. Posttest analysis of MIST test 3109AA using TRAC-PF1/MOD1. Division of Systems Research, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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C, Gill, U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research, and Winfrith Technology Centre, eds. TRAC-PF1/MOD1 calculations of LOFT experiment LP-02-6. Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research. Division of Systems Research. and Los Alamos National Laboratory, eds. Posttest analysis of MIST test 330302 using TRAC-PF1/MOD1. Division of Systems Research, Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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Colin, Gill, U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research., and Winfrith Technology Centre, eds. TRAC-PF1/MOD1 calculations of LOFT experiment LP-02-6. Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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Colin, Gill, U.S. Nuclear Regulatory Commission. Office of Nuclear Regulatory Research., and Winfrith Technology Centre, eds. TRAC-PF1/MOD1 calculations of LOFT experiment LP-02-6. Office of Nuclear Regulatory Research, U.S. Nuclear Regulatory Commission, 1992.

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Book chapters on the topic "PFN1"

1

Narizzano, Allison M. "Perfluorononanoic Acid (PFNA)." In Understanding Risk to Wildlife from Exposures to Per- and Polyfluorinated Alkyl Substances (PFAS). CRC Press, 2021. http://dx.doi.org/10.1201/9781003162476-6.

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De Zwart, P. M., D. Höntzsch, T. Krackhardt, and E. Schwab. "PFN - Proximaler Femurnagel." In Hefte zur Zeitschrift „Der Unfallchirurg“. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60913-8_495.

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Schwab, E., T. Krackhardt, F. Maurer, and K. Weise. "Implantationstechnik des proximalen Femurnagels (PFN)." In Deutsche Gesellschaft für Chirurgie. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59573-8_478.

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Xiaochao, Yin, Han Guodong, and Gu Zeyu. "Modified PFNF Fault-Tolerant Routing Algorithm for WUDN Structure." In Advances in Intelligent Systems and Computing. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8944-2_64.

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Roux, Benoît, and Thomas B. Woolf. "Molecular Dynamics of Pf1 Coat Protein in a Phospholipid Bilayer." In Biological Membranes. Birkhäuser Boston, 1996. http://dx.doi.org/10.1007/978-1-4684-8580-6_17.

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Kornilov, N., T. Massey, S. Grimes, and A. Voinov. "New Experimental Proposal for235U PFNS Measurement to Answer a Fifty Year Old Question." In Reactor Dosimetry: 14th International Symposium. ASTM International, 2012. http://dx.doi.org/10.1520/stp155020120055.

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Kornilov, N., T. Massey, S. Grimes, and A. Voinov. "New Experimental Proposal for235U PFNS Measurement to Answer a Fifty Year Old Question." In Reactor Dosimetry: 14th International Symposium. ASTM International, 2012. http://dx.doi.org/10.1520/stp49652t.

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Jia, Qifei, Shikui Wei, and Yufeng Zhao. "R-PFN: Towards Precise Object Detection by Recurrent Pyramidal Feature Fusion." In Pattern Recognition and Computer Vision. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-60633-6_47.

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Messmer, P., R. Babst, and P. Regazzoni. "Der Proximale Femurnagel (PFN) der AO. Erste klinische Erfahrungen bei 81 Patienten." In Hefte zur Zeitschrift „Der Unfallchirurg“. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60913-8_218.

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Holzman, P., and R. Ruckert. "Der proximale Femurnagel (PFN) der AO — erste Erfahrungen und Nachkontrolle in der Gerontotraumatologie." In Vielfalt und Einheit der Chirurgie Humanität und Wissenschaft. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-45774-6_492.

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Conference papers on the topic "PFN1"

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Bu, Zhaoxiong, Li Ma, Jihui Li, Linjiu Guo, Yong Zhong, and Zhongrong Zeng. "Access Control Attribute Mining Model Based on ALBERT-PFN." In 2024 IEEE International Conference on Cognitive Computing and Complex Data (ICCD). IEEE, 2024. https://doi.org/10.1109/iccd62811.2024.10843602.

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He, Y., T. Ishiyama, K. Nagao, T. Sugai, and W. Jiang. "Glow Discharge Using Spiker-Sustainer Circuit Consisting of PFN and SOS." In 2024 IEEE International Conference on Plasma Science (ICOPS). IEEE, 2024. http://dx.doi.org/10.1109/icops58192.2024.10626734.

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Ou, J., Y. Liu, L. Li, Y. Xu, and F. Lin. "Research on hybrid pulse power supply based on Marx-PFN distributed hybrid power topology." In 2024 IEEE International Conference on Plasma Science (ICOPS). IEEE, 2024. http://dx.doi.org/10.1109/icops58192.2024.10625818.

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Gau, David M., Jamie Lesnock, Thomas Krivak, Robert Edwards, and Partha Roy. "Abstract LB-70: BRCA1 impacts ovarian cancer cell migration through modulating Pfn1 expression." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-lb-70.

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Beneckv, M. J., C. G. Kolvenbach, D. L. Amrani, and M. W. Mosesson. "EVIDENCE THAT THE C-TERMINAL HEPARIN BINDING DOMAIN ("HEP II") DOMINATES HEPARIN-FIBRONECTIN INTERACTIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643631.

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The interaction of glycosaminoglycans with plasma fibronectin (PFn) may play a role in the conversion of PFn from an “inert” dimeric circulating form to an “activated” multimeric form deposited on the cell surface or in the extracellular matrix. We carried out a quantitative comparison of heparin affinity for PFn and its proteolytic fragments in order to assess the relative importance of heparin interactions with PFn’s various reported heparin-binding domains. We employed affinity chromatography on PFn-sepharose to prepare a subset of fluorescein-labelled heparin molecules with high affinity f
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Hafsah, Anna. "DESAIN E-MODUL INTERAKTIF BERBASIS DISCOVERY LEARNING PADA MATERI FLUIDA STATIS." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.pf21.

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Estiningsih, Asri. "PENGEMBANGAN PROTOTYPE BUKU PENGAYAAN PENGETAHUAN FISIKA TENTANG KARAKTERISTIK GELOMBANG MEKANIK BERBASIS CONTEXTUAL TEACHING AND LEARNING (CTL) UNTUK SISWA SMA." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.pf11.

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Putria, Sarah Febriyana. "PENGEMBANGAN E-LKPD BERBANTUAN LIVEWORKSHEETS DENGAN MODEL POE (PREDICT-OBSERVE-EXPLAIN) PADA MATERI TEORI KINETIK GAS UNTUK MELATIH KETERAMPILAN BERPIKIR TINGKAT TINGGI." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.pf41.

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Rahmatika, Fadia Fatasya. "RANCANGAN MODUL DIGITAL “PHYQSURE” BERBASIS STAD PADA MATERI BESARAN FISIKA DAN PENGUKURAN." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.pf31.

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Serevina, Vina. "INTERACTIVE DIGITAL MODULE OF GLOBAL WARMING (IDMOGW) BERBASIS STEM-PROJECT BASED LEARNING UNTUK SISWA SMA." In SEMINAR NASIONAL FISIKA 2016 UNJ. PRODI Pendidikan Fisika dan Fisika UNJ, 2024. http://dx.doi.org/10.21009/03.1201.pf01.

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Reports on the topic "PFN1"

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Neudecker, Denise. 239Pu(n,f) PFNS evaluation: potential release candidate. Office of Scientific and Technical Information (OSTI), 2021. http://dx.doi.org/10.2172/1779660.

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Lovell, Amy, and Denise Neudecker. Correcting the PFNS for more consistent fission modeling. Office of Scientific and Technical Information (OSTI), 2021. http://dx.doi.org/10.2172/1878094.

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Taddeucci, Terry. MCNP modeling of the Starostov et al. PFNS measurements. Office of Scientific and Technical Information (OSTI), 2014. http://dx.doi.org/10.2172/1150688.

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4

Denise, Neudecker, Keegan Kelly, Robert Little, Amy Lovell, and Matthew Devlin. Evaluating the 238U PFNS Including Chi-Nu Experimental Data. Office of Scientific and Technical Information (OSTI), 2024. http://dx.doi.org/10.2172/2372659.

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5

White, Morgan Curtis, Matthew James Devlin, Jaime A. Gomez, et al. Milestone 5431: Chi-Nu Measurements of Prompt Fission Neutron Spectra (PFNS). Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1329700.

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Neudecker, Denise. Final evaluated 239Pu(n500keV,f) PFNS and uncertainties and test evaluations. Office of Scientific and Technical Information (OSTI), 2014. http://dx.doi.org/10.2172/1150662.

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Wang, Ju, and G. J. Volk. Design of kicker/bumper magnet and PFN for. Office of Scientific and Technical Information (OSTI), 1990. http://dx.doi.org/10.2172/91969.

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8

Slater, C. O. Irradiation Effects for the Pulsed Fast Neutron Analysis (PFNA) Cargo Interrogation System. Office of Scientific and Technical Information (OSTI), 2001. http://dx.doi.org/10.2172/777672.

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9

Neudecker, Denise. <sup>238</sup>U PFNS Evaluation Update [Slides]. Office of Scientific and Technical Information (OSTI), 2023. http://dx.doi.org/10.2172/2208742.

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Gruen, G. E., and J. E. Fisher. TRAC-PF1/MOD1 US/Japanese PWR conservative LOCA prediction. Office of Scientific and Technical Information (OSTI), 1987. http://dx.doi.org/10.2172/5746401.

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