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Relevant bibliographies by topics / Phagosom

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Academic literature on the topic 'Phagosom'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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Journal articles on the topic "Phagosom"

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Lerm, M., Å Holm, Å Seiron, E. Särndahl, K. E. Magnusson, and B. Rasmusson. "Leishmania donovani Requires Functional Cdc42 and Rac1 To Prevent Phagosomal Maturation." Infection and Immunity 74, no. 5 (2006): 2613–18. http://dx.doi.org/10.1128/iai.74.5.2613-2618.2006.

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ABSTRACT Leishmania donovani promastigotes survive inside macrophage phagosomes by inhibiting phagosomal maturation. The main surface glycoconjugate on promastigotes, lipophosphoglycan (LPG), is crucial for survival and mediates the formation of a protective shell of F-actin around the phagosome. Previous studies have demonstrated that this effect involves inhibition of protein kinase Cα. The present study shows that functional Cdc42 and Rac1 are required for the formation of F-actin around L. donovani phagosomes. Moreover, we present data showing that phagosomes containing LPG-defective L. do

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Lu, Nan, Qian Shen, Timothy R. Mahoney, Xianghua Liu, and Zheng Zhou. "Three sorting nexins drive the degradation of apoptotic cells in response to PtdIns(3)P signaling." Molecular Biology of the Cell 22, no. 3 (2011): 354–74. http://dx.doi.org/10.1091/mbc.e10-09-0756.

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Apoptotic cells are swiftly engulfed by phagocytes and degraded inside phagosomes. Phagosome maturation requires phosphatidylinositol 3-phosphate [PtdIns(3)P], yet how PtdIns(3)P triggers phagosome maturation remains largely unknown. Through a genome-wide PtdIns(3)P effector screen in the nematode Caenorhabditis elegans, we identified LST-4/SNX9, SNX-1, and SNX-6, three BAR domain-containing sorting nexins, that act in two parallel pathways to drive PtdIns(3)P-mediated degradation of apoptotic cells. We found that these proteins were enriched on phagosomal surfaces through association with Ptd

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Rupper, A. C., J. M. Rodriguez-Paris, B. D. Grove, and J. A. Cardelli. "p110-related PI 3-kinases regulate phagosome-phagosome fusion and phagosomal pH through a PKB/Akt dependent pathway in Dictyostelium." Journal of Cell Science 114, no. 7 (2001): 1283–95. http://dx.doi.org/10.1242/jcs.114.7.1283.

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The Dictyostelium p110-related PI 3-kinases, PIK1 and PIK2, regulate the endosomal pathway and the actin cytoskeleton, but do not significantly regulate internalization of particles in D. discoideum. Bacteria internalized into (Δ)ddpik1/ddpik2 cells or cells treated with PI 3-kinase inhibitors remained intact as single particles in phagosomes with closely associated membranes after 2 hours of internalization, while in control cells, bacteria appeared degraded in multi-particle spacious phagosomes. Addition of LY294002 to control cells, after 60 minutes of chase, blocked formation of spacious p

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Alvarez-Dominguez, C., R. Roberts, and P. D. Stahl. "Internalized Listeria monocytogenes modulates intracellular trafficking and delays maturation of the phagosome." Journal of Cell Science 110, no. 6 (1997): 731–43. http://dx.doi.org/10.1242/jcs.110.6.731.

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Previous studies have shown that early phagosome-endosome fusion events following phagocytosis of Listeria monocytogenes are modulated by the live organism. In the present study, we have characterized more fully the intracellular pathway of dead and live Listeria phagosomes. To examine access of endosomal and lysosomal markers to phagosomes containing live and dead Listeria, quantitative electron microscopy was carried out with intact cells using internalized BSA-gold as a marker to quantify transfer of solute from endosomal and lysosomal compartments to phagosomes. To monitor the protein comp

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Clemens, Daniel L., Bai-Yu Lee, and Marcus A. Horwitz. "Francisella tularensis Phagosomal Escape Does Not Require Acidification of the Phagosome." Infection and Immunity 77, no. 5 (2009): 1757–73. http://dx.doi.org/10.1128/iai.01485-08.

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ABSTRACT Following uptake, Francisella tularensis enters a phagosome that acquires limited amounts of lysosome-associated membrane glycoproteins and does not acquire cathepsin D or markers of secondary lysosomes. With additional time after uptake, F. tularensis disrupts its phagosomal membrane and escapes into the cytoplasm. To assess the role of phagosome acidification in phagosome escape, we followed acidification using the vital stain LysoTracker red and acquisition of the proton vacuolar ATPase (vATPase) using immunofluorescence within the first 3 h after uptake of live or killed F. tulare

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Lee, Warren L., Moo-Kyung Kim, Alan D. Schreiber, and Sergio Grinstein. "Role of Ubiquitin and Proteasomes in Phagosome Maturation." Molecular Biology of the Cell 16, no. 4 (2005): 2077–90. http://dx.doi.org/10.1091/mbc.e04-06-0464.

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Phagosomes undergo multiple rounds of fusion with compartments of the endocytic pathway during the course of maturation. Despite the insertion of vast amounts of additional membrane, the phagosomal surface area remains approximately constant, implying active ongoing fission. To investigate the mechanisms underlying phagosomal fission we monitored the fate of Fcγ receptors (FcγR), which are known to be cleared from the phagosome during maturation. FcγR, which show a continuous distribution throughout the membrane of nascent phagosomes were found at later times to cluster into punctate, vesicula

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Lennon-Duménil, Ana-Maria, Arnold H. Bakker, René Maehr, et al. "Analysis of Protease Activity in Live Antigen-presenting Cells Shows Regulation of the Phagosomal Proteolytic Contents During Dendritic Cell Activation." Journal of Experimental Medicine 196, no. 4 (2002): 529–40. http://dx.doi.org/10.1084/jem.20020327.

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Here, we describe a new approach designed to monitor the proteolytic activity of maturing phagosomes in live antigen-presenting cells. We find that an ingested particle sequentially encounters distinct protease activities during phagosomal maturation. Incorporation of active proteases into the phagosome of the macrophage cell line J774 indicates that phagosome maturation involves progressive fusion with early and late endocytic compartments. In contrast, phagosome biogenesis in bone marrow–derived dendritic cells (DCs) and macrophages preferentially involves endocytic compartments enriched in

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Sullivan, Jonathan Tabb, Ellen F. Young, Jessica R. McCann, and Miriam Braunstein. "The Mycobacterium tuberculosis SecA2 System Subverts Phagosome Maturation To Promote Growth in Macrophages." Infection and Immunity 80, no. 3 (2012): 996–1006. http://dx.doi.org/10.1128/iai.05987-11.

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The ability ofMycobacterium tuberculosisto grow in macrophages is critical to the virulence of this important pathogen. One wayM. tuberculosisis thought to maintain a hospitable niche in macrophages is by arresting the normal process of phagosomes maturing into acidified phagolysosomes. The process of phagosome maturation arrest byM. tuberculosisis not fully understood, and there has remained a need to firmly establish a requirement for phagosome maturation arrest forM. tuberculosisgrowth in macrophages. Other intracellular pathogens that control the phagosomal environment use specialized prot

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Levin, Roni, Gerald R. V. Hammond, Tamas Balla, Pietro De Camilli, Gregory D. Fairn, and Sergio Grinstein. "Multiphasic dynamics of phosphatidylinositol 4-phosphate during phagocytosis." Molecular Biology of the Cell 28, no. 1 (2017): 128–40. http://dx.doi.org/10.1091/mbc.e16-06-0451.

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We analyzed the distribution, fate, and functional role of phosphatidylinositol 4-phosphate (PtdIns4P) during phagosome formation and maturation. To this end, we used genetically encoded probes consisting of the PtdIns4P-binding domain of the bacterial effector SidM. PtdIns4P was found to undergo complex, multiphasic changes during phagocytosis. The phosphoinositide, which is present in the plasmalemma before engagement of the target particle, is transiently enriched in the phagosomal cup. Soon after the phagosome seals, PtdIns4P levels drop precipitously due to the hydrolytic activity of Sac2

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Steinberg, B. E., K. K. Huynh, and S. Grinstein. "Phagosomal acidification: measurement, manipulation and functional consequences." Biochemical Society Transactions 35, no. 5 (2007): 1083–87. http://dx.doi.org/10.1042/bst0351083.

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Phagocytosis holds a central position in the development of a successful innate immune response and in the initiation of the corresponding adaptive response. The destruction of invading pathogens and the presentation of their antigens to lymphoid cells require acidification of the phagosomal lumen. The present review discusses the mechanism of phagosome acidification, with particular reference to the two components of the protonmotive force: the chemical (pH) gradient and the electrical potential across the phagosomal membrane. A method for the in situ measurement of the electrical potential a

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Dissertations / Theses on the topic "Phagosom"

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Lührmann, Anja. "Analyse der Reifung von Afipien- und Rhodokokken-enthaltenden Phagosomen in Makrophagen." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=966000323.

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Guo, Manman. "Phagosome proteomes in activated macrophages." Thesis, University of Dundee, 2015. https://discovery.dundee.ac.uk/en/studentTheses/c5a94702-2164-4acc-9763-bdfcaf1229dc.

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Macrophages play key roles in innate and adaptive immune systems not only in the response to pathogens but also in tissue homeostasis. They are extremely plastic and recognize external stimuli such as cytokines with substantial changes to the proteome and molecular functions. One major macrophage function altered by cytokine activation is phagocytosis and phagosome maturation, through which macrophages engulf foreign material such as microbes or apoptotic cells to form phagosomes which then fuse with endosome and finally lysosomes, where the particles are finally degraded. My project aims at i

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Smith, Leanne May. "Investigating phagosome dynamics of microbial pathogens." Thesis, University of Birmingham, 2015. http://etheses.bham.ac.uk//id/eprint/4937/.

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Many microbial pathogens are able to evade killing by phagocytes of the innate immune system. This thesis focuses on two pathogens: the fungal pathogen \(Cryptococcus\) \(neoformans\) and the bacterial pathogen \(Streptococcus\) \(agalactiae\). \(C\). \(neoformans\) causes severe cryptococcal meningitis in mostly immunocompromised hosts, such as those with HIV infection. In contrast, \(S\). \(agalactiae\) is the leading cause of neonatal sepsis and meningitis. The interaction between macrophages and these pathogens is likely to be critical in determining dissemination and outcome of disease in

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Boucherit, Nicolas. "Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii." Thesis, Aix-Marseille, 2013. http://www.theses.fr/2013AIXM5061/document.

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Pour survivre et se multiplier dans leurs cellules hôtes les bactéries intracellulaires ont élaboré divers mécanismes d'échappement à la réponse immunitaire. L’altération du trafic intracellulaire est une des stratégies utilisée par ces agents pathogènes. Ainsi Coxiella burnetii, bactérie intracellulaire stricte responsable chez l’homme de la fièvre Q, bloque la maturation du phagosome afin de résider et se multiplier dans un compartiment incapable de fusionner avec les lysosomes. Ce défaut de fusion est associé à la virulence bactérienne. Pour analyser le défaut de maturation du phagosome de

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Pietersen, Ray-Dean Donovan. "Proteomic analysis of the Mycobacterium avium-containing phagosome membrane." Master's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/12226.

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Includes abstract.<br>Includes bibliographical references (leaves 139-158).<br>Pathogenic mycobacteria, like [Mycobacterium tuberculosis] M.tb and Mycobacterium avium (M.av), reside intracellularly in phagosomes where they are able to survive, because they block the process of phagosome maturation. ... An approach of characterizing M.av-containing phagosomes in terms of cell surface-derived glycoconjugates, revealed that these phagosomes show a 3- to 4-fold depletion of these glycoconjugates as compared to early endosomes with which the phagosomes continuously fuse and exchange membrane molecu

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Abnave, Prasad. "Exploring mammalian immunity against intracellular bacteria through planarian flatworms." Thesis, Aix-Marseille, 2014. http://www.theses.fr/2014AIXM5049.

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Les interactions hôte-pathogène sont un jeu vaste et complexe entre agent pathogène et hôtepour la victoire de la bataille de la pathogenèse. Plusieurs organismes modèles sont étudiéspour illustrer les mécanismes impliqués dans ces interactions. Dans ma thèse, j'ai utilisé lesplanaires comme un organisme modèle pour explorer les interactions hôte-pathogène. Comme les différents organismes modèles peuvent mettre enévidence les différentes caractéristiques de l'immunité, j'ai décidé de tirer avantage del'absence de connaissances sur l'immunité des planaires en explorant l'inexplorée. Dans monpro

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Geffken, Anna Christina [Verfasser]. "Mycobacterium tuberculosis – host-cell interactions in the phagosome / Anna Christina Geffken." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2016. http://d-nb.info/1098170482/34.

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Hackam, David Joel. "Mechanisms of phagosome formation, maturation and acidification, implications for intracellular infection." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0003/NQ41434.pdf.

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Bajno, Lydia. "Focal exocytosis of VAMP3-containing vesicles at sites of phagosome formation." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0018/MQ53328.pdf.

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Blocker, Ariel. "Analyse des interactions entre phagosomes et microtubules." Paris 11, 1995. http://www.theses.fr/1995PA112390.

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Le cytosquelette microtubulaire est une structure filamenteuse et dynamique qui participe a l'organisation des cellules, en particulier au positionnement des organelles intracellulaires. Il est requis dans de nombreuses etapes du routage des proteines via le transport vesiculaire, qu'il facilite par des mechanoenzymes, telles les moteurs microtubulaire kinesine et dyneine cytoplasmique. La phagocytose est l'engloutissement par des cellules d'autres cellules ou de particles de taille importantes (>0. 3 micron de diametre). Elle remplit diverses fonctions dans le monde vivant. Chez les vertebres

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Books on the topic "Phagosom"

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Deretic, Vojo, ed. Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4.

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Botelho, Roberto, ed. Phagocytosis and Phagosomes. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6581-6.

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Bajno, Lydia. Focal exocytosis of VAMP3-containing vesicles at sites of phagosome formation. National Library of Canada, 2000.

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Vojo, Deretic, ed. Autophagosome and phagosome / edited by Vojo Deretic. Humana Press, 2008.

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Scott, Cameron. Control of phagocytosis and phagosome maturation by phosphoinositides. 2005.

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Phagocytosis and Phagosomes: Methods and Protocols. Humana, 2018.

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Lee, Warren Lester. Membrane remodelling and signal transduction during phagosome formation and maturation. 2006.

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Hackam, David Joel. Mechanisms of phagosome formation, maturation and acidification: Implications for intracellular infection. 1998.

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9

Tan, Tracy. The ESAT-6/CFP-10 secretion system of Mycobacterium marinum modulates phagosome maturation. 2006.

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Paroutis, Paul. Quantitative and dynamic assessment of the contribution of the endoplasmic reticulum to phagosome formation. 2005.

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Book chapters on the topic "Phagosom"

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Gooch, Jan W. "Phagosome." In Encyclopedic Dictionary of Polymers. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14473.

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Härtlova, Anetta, Julien Peltier, Orsolya Bilkei-Gorzo, and Matthias Trost. "Isolation and Western Blotting of Latex-Bead Phagosomes to Track Phagosome Maturation." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6581-6_16.

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Deretic, Vojo. "Autophagosome and Phagosome." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_1.

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Young, Andrew, and Sharon Tooze. "Protein Trafficking into Autophagosomes." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_10.

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Lavieu, Grégory, Francesca Scarlatti, Giusy Sala, et al. "Sphingolipids in Macroautophagy." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_11.

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Ciechomska, Iwona A., Christoph G. Goemans, and Aviva M. Tolkovsky. "Molecular Links Between Autophagy and Apoptosis." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_12.

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Ravikumar, Brinda, Sovan Sarkar, and David C. Rubinsztein. "Clearance of Mutant Aggregate-Prone Proteins by Autophagy." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_13.

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Schmid, Dorothee, and Christian Münz. "Localization and MHC Class II Presentation of Antigens Targeted for Macroautophagy." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_14.

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Kaushik, S., and A. M. Cuervo. "Chaperone-Mediated Autophagy." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_15.

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Uttenweiler, Andreas, and Andreas Mayer. "Microautophagy in the Yeast Saccharomyces cerevisiae." In Autophagosome and Phagosome. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-157-4_16.

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Conference papers on the topic "Phagosom"

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Crouser, E. D., L. W. Locke, M. W. Julian, S. Bicer, P. White, and L. S. Schlesinger. "Phagosome-Regulated-mTOR Signaling During Sarcoidosis Granulomagenesis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3102.

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Cheng, Wenhao, Sundol Kim, Sandra Zivkovic, Yi Ren, Hoyong Chung, and Jingjiao Guan. "Tracking Phagosome-Derived Vesicles in Macrophages with Microfabricated Microparticles." In The 6th World Congress on Recent Advances in Nanotechnology. Avestia Publishing, 2021. http://dx.doi.org/10.11159/nddte21.lx.203.

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Daniel, Rebekah, Andrew T. Koll, and David Altman. "Force dependence of phagosome trafficking in retinal pigment epithelial cells." In SPIE NanoScience + Engineering, edited by Kishan Dholakia and Gabriel C. Spalding. SPIE, 2014. http://dx.doi.org/10.1117/12.2062055.

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O'Leary, SM, M. O'Sullivan, DM Kelly, R. Ryan, and J. Keane. "IL-10 Blocks Phagosome Maturation in Human Macrophages Infected with M. tuberculosis." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5103.

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Alhousseiny, Sara. "The role of the autophagy pathway in phagosome formation in macrophages interacting with adipocytes." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2018. http://dx.doi.org/10.5339/qfarc.2018.hbpd154.

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Yoon, Jonghee, Kyoohyun Kim, Jaehwang Jung, and YongKeun Park. "3-D quantitative tracking of phagosomes using quantitative phase microscopy." In Asia Communications and Photonics Conference. OSA, 2014. http://dx.doi.org/10.1364/acpc.2014.ath1d.5.

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Oberley-Deegan, RE, Y. Lee, GE Morey, DM Cook, ED Chan, and JD Crapo. "MnTE-2-PyP, an Antioxidant Mimetic, Reduces Intracellular Growth ofMycobacterium abscessusby Enhancing Phagosome-Lysosome Fusion." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5104.

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Jubrail, Jamil, Kshanti Africano-Gomez, Floriane Herit, et al. "Human rhinovirus 16 infection of human macrophages impairs their clearance ability by perturbing phagosome maturation." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4977.

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Ufimtseva, Elena, Natalya Eremeeva, Diana Vakhrusheva, and Sergey Skornyakov. "Mycobacterium tuberculosis reside only inside phagosomes in alveolar macrophages of tuberculosis patients." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4730.

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Sengupta, Debrup, and Peter Cresswell. "Abstract B059: Slow maturation of early phagosomes promotes a TAP independent antigen cross-presentation pathway." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b059.

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