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Journal articles on the topic 'Phenytoin Gingival Hyperplasia'

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Published: 4 June 2021
Last updated: 18 February 2022

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1

AlJehani, Yousef A. "Nonsurgical Management of Phenytoin-induced Gingival Hyperplasia." Journal of Contemporary Dental Practice 16, no. 4 (2015): 319–21. http://dx.doi.org/10.5005/jp-journals-10024-1683.

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ABSTRACT Introduction The aim of this report is to present a severe case of phenytoin (PHT)-induced gingival hyperplasia in a Saudi patient. Materials and methods A 12-year-old male epileptic patient, undergoing PHT therapy, was diagnosed clinically with severe gingival hyperplasia. His treatment consisted meticulous oral care and weekly professional prophylaxis. The patient was advised oral folic acid supplementation (0.5 mg/day) and was also recommended 0.2% chlorhexidine gluconate mouthwash twice daily. Results There was significant reduction in the hyperplastic tissue within 4 weeks of tre
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2

Sharma, Sanjay, and S. K. Dasroy. "Gingival Hyperplasia Induced by Phenytoin." New England Journal of Medicine 342, no. 5 (2000): 325. http://dx.doi.org/10.1056/nejm200002033420505.

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3

Saleem, Sheikh, Sawan Verma, Irfan Yousuf, Mushtaq Ahmad Wani, and Ravouf Asmi. "Diphenylhydantoin Induced Severe Gingival Hyperplasia." JMS SKIMS 20, no. 1 (2017): 44–46. http://dx.doi.org/10.33883/jms.v20i1.311.

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Gingival overgrowth (GO) is a side effect, associated with some distinct classes of drugs, such as anticonvulsants, immunosuppressant, and calcium channel blockers. One of the main drugs associated with GO is the antiepileptic, diphenylhydantoin (DPH)/ phenytoin (1), which affects gingival tissues by altering extracellular matrix metabolism. Phenytoin (DPH)-induced gingival overgrowth (PIGO) due to chronic administration remains an unsolved problem especially in cases where this drug is taken without any supervision due usually to poor follow-up. Younger age groups experience more lesions than
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4

Vahabi, Surena, Bahareh Nazemi salman, and Pouya Pourgolshani. "Effect of Phenytoin and Cyclosporine on IL-17 Production by Gingival Fibroblasts of Adults and Children." Journal of Periodontology & Implant Dentistry 7, no. 1 (2018): 1–6. http://dx.doi.org/10.15171/jpid.2015.001.

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Background and aims. Gingival hyperplasia, a relatively common side effect of antiepileptic and anticonvulsant drugs, occurs in 30‒50% of patients taking phenytoin and 25‒81% of those taking cyclosporine. Gingival hyperplasia due to lack of balance between extracellular synthesis and degradation is associated with increased production of IL-1B, IL-6 and IL-8 by gingival fibroblasts. Tissue level of IL-17 increases in inflammatory conditions. Since the role of IL-17 and patient age in gingival hyperplasia is still unclear, this study aimed to compare the level of IL-17 produced by gingival fibr
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5

Shaftic, Alison A., Lorie L. Widdup, Marie A. Abate, and Arthur I. Jacknowitz. "Nifedipine-Induced Gingival Hyperplasia." Drug Intelligence & Clinical Pharmacy 20, no. 7-8 (1986): 602–5. http://dx.doi.org/10.1177/106002808602000724.

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Gingival hyperplasia, a condition characterized by increased amounts of gingival connective tissue, has most commonly been observed in patients receiving phenytoin, but has also been noted in patients receiving cyclosporine and, as in this case report, nifedipine. Patients receiving nifedipine should be advised to practice good oral hygiene to lessen the possibility of hyperplasia occurring. If gingival hyperplasia develops in a patient taking nifedipine, the drug should be suspected as being
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6

Farook, Fathima Fazrina, Mohamed Nuzaim M. Nizam, and Abdulsalam Alshammari. "An Update on the Mechanisms of Phenytoin Induced Gingival Overgrowth." Open Dentistry Journal 13, no. 1 (2019): 430–35. http://dx.doi.org/10.2174/1874210601913010430.

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Background: Phenytoin induced gingival overgrowth, a side effect with multifactorial aetiology, is characterized by an increase in the volume of extracellular tissues, particularly collagenous components, with varying degrees of inflammation. Objective: The aim of this paper is to review the available literature regarding the pathophysiological mechanisms of phenytoin induced gingival overgrowth. Methods: A thorough literature search of the PubMed/ Embase/ Web of science/ Cochrane central database was conducted to identify the mechanisms involved in the process of phenytoin-induced gingival ov
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7

Lafzi, Ardeshir, Ramin Mostofi Zadeh Farahani, and Mohammad Ali Mohajjel Shoja. "Phenobarbital-induced Gingival Hyperplasia." Journal of Contemporary Dental Practice 8, no. 6 (2007): 50–56. http://dx.doi.org/10.5005/jcdp-8-6-50.

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Abstract Aim The aim of this article is to present a case of a phenobarbital-induced gingival hyperplasia (GH), discuss possible etiological mechanisms of drug-induced GH, and to present a concise review of the literature. Background GH is a well-known complication associated with anticonvulsant phenytoin, antihypertensive calcium channel blockers, and immunosuppressant cyclosporine therapy. Sodium valproate and primidone has very rarely been found to cause GH. Report An extremely rare case of phenobarbital-induced GH in a 28-year-old male patient who had received this drug for three years is
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8

KASSAI, Yasuhiro, Yuri OKISHIO, Tomonobu KIDA, Shigeyuki EBISU, and Hiroshi OKADA. "Immunopathological studies of phenytoin-induced gingival hyperplasia." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 27, no. 1 (1985): 197–205. http://dx.doi.org/10.2329/perio.27.197.

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9

Millichap, J. Gordon. "Folic Acid Supplement Prevents Phenytoin-Induced Gingival Hyperplasia." Pediatric Neurology Briefs 25, no. 5 (2011): 35. http://dx.doi.org/10.15844/pedneurbriefs-25-5-3.

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10

Butler, Rex T., Kenneth L. Kalkwarf, and Wayne B. Kaldahl. "Drug-induced gingival hyperplasia: phenytoin, cyclosporine, and nifedipine." Journal of the American Dental Association 114, no. 1 (1987): 56–60. http://dx.doi.org/10.14219/jada.archive.1987.0050.

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11

Stinnett, Elizabeth, Brad Rodu, and William E. Grizzle. "New developments in understanding phenytoin-induced gingival hyperplasia." Journal of the American Dental Association 114, no. 6 (1987): 814–16. http://dx.doi.org/10.14219/jada.archive.1987.0162.

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12

Perlík, F., M. Kolínová, J. Zvárová, and V. Patzelová. "Phenytoin as a Risk Factor in Gingival Hyperplasia." Therapeutic Drug Monitoring 17, no. 5 (1995): 445–48. http://dx.doi.org/10.1097/00007691-199510000-00002.

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13

Takada, Kazuko, Hitoshi Sugiyama, Koji Umezawa, Junichi Mega, and Masatomo Hirasawa. "The subgingival microflora in phenytoin-induced gingival hyperplasia." Journal of Periodontal Research 38, no. 5 (2003): 477–81. http://dx.doi.org/10.1034/j.1600-0765.2003.00676.x.

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14

Verma, Pushpendra, Anju Gautam, Karuna Lodhi, and Ruchi Srivastava. "Esthetic Correction of Phenytoin Induced Gingival Hyperplasia- Case Reports." Health Care 1, no. 2 (2013): 28. http://dx.doi.org/10.12966/hc.08.01.2013.

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15

MARSHALL, WILLIAM R., DAVID A. LATTANZI, G. SCOTT WEAVER, and LARRY J. SCHMITT. "Laser excision of type III phenytoin-induced gingival hyperplasia." Special Care in Dentistry 7, no. 5 (1987): 207–10. http://dx.doi.org/10.1111/j.1754-4505.1987.tb00648.x.

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16

Lin, Katia, Laura M. F. F. Guilhoto, and Elza Márcia Targas Yacubian. "Drug-induced gingival enlargement - Part II. Antiepileptic drugs: not only phenytoin is involved." Journal of Epilepsy and Clinical Neurophysiology 13, no. 2 (2007): 83–88. http://dx.doi.org/10.1590/s1676-26492007000200009.

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INTRODUCTION: Gingival enlargement is the term now used to describe medication-related gingival overgrowth or gingival hyperplasia, a common reactionary phenomenon that occurs with the use of several types of therapeutic agents, including antiepileptic drugs. This disorder has been recognized since 1939, shortly after the introduction of phenytoin. METHODS: Review of literature concerning etiology, pathogenesis and management of antiepileptic drug induced gingival enlargement. CONCLUSIONS: It is important that neurologists become aware of the potential etiologic agents of antiepileptic drug in
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17

Kafadar, Ihsan, Burcu Tufan, and Huriye Elif Sinan. "Gingival hyperplasia occurs due to using phenytoin: a case report." Medical Journal of Okmeydani Training and Research Hospital 28, no. 1 (2012): 49–51. http://dx.doi.org/10.5222/otd.2012.049.

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18

POPPELL, TIMOTHY D., and J. FRANK COLLINS. "Phenytoin-induced gingival hyperplasia of edentulous spaces: a case report." Special Care in Dentistry 7, no. 3 (1987): 106–7. http://dx.doi.org/10.1111/j.1754-4505.1987.tb00616.x.

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19

Byun, Sang Kil, Hee-Kyung Lee, Byung-Rho Chin, and Meung Chul Oh. "Surgical Treatment of Phenytoin Induced Gingival Hyperplasia: A Report of Case." Yeungnam University Journal of Medicine 3, no. 1 (1986): 383. http://dx.doi.org/10.12701/yujm.1986.3.1.383.

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20

Kinane, D. F., J. R. Drummond, and D. M. Chisholm. "Langerhans cells in human chronic gingivitis and phenytoin-induced gingival hyperplasia." Archives of Oral Biology 35, no. 7 (1990): 561–64. http://dx.doi.org/10.1016/0003-9969(90)90088-r.

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21

Gaba, Saurabh, Samiksha Gupta, Monica Gupta, and Gautam Jesrani. "Concurrent gingival hyperplasia, hirsutism, and megaloblastic anemia due to phenytoin use." Apollo Medicine 17, no. 4 (2020): 299. http://dx.doi.org/10.4103/am.am_117_20.

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22

Calin, Alina Mihaela, Mihaela Debita, Raluca Dragomir, Ovidiu Mihail Stefanescu, Cristian Budacu, and Andreea Silvana Szalontay. "Treatment Methods Conditioned by the Gravity of Drug-Induced Gingival Hyperplasias." Revista de Chimie 68, no. 11 (2017): 2618–22. http://dx.doi.org/10.37358/rc.17.11.5941.

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The first drug discovered to be involved in the development of gingival hyperplasia is phenytoin, which is indicated in the treatment of epileptic patients. The other drugs are calcium channel blockers with vasodilating effect. The most important one is Nifedipine, while Ciclosporin A, which is used as an immunosuppressant in the prevention of transplant rejection, causes gingival hyperplasia as a secondary effect. Gingival hyperplasia can reach an impressive volume, completely covering the dental crown and affecting the masticatory and physiognomic functions. The elucidation of the mechanism,
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23

SASAKI, Toshiaki, Eikichi MATTA, and Hiroshi HORIUCHI. "A clinical examination of phenytoin-induced gingival hyperplasia in severely handicapped patients." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 32, no. 4 (1990): 1077–85. http://dx.doi.org/10.2329/perio.32.1077.

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24

Zhou, Ling Xiang, Bruce Pihlstrom, James P. Hardwick, Sang S. Park, Steven A. Wrighton, and Jordan L. Holtzman. "Metabolism of phenytoin by the gingiva of normal humans: The possible role of reactive metabolities of phenytoin in the initiation of gingival hyperplasia*." Clinical Pharmacology & Therapeutics 60, no. 2 (1996): 191–98. http://dx.doi.org/10.1016/s0009-9236(96)90135-6.

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25

LIMA, VICTOR GUEDES DE. "GINGIVAL HYPERPLASIA ASSOCIATED WITH THE USE OF CYCLOSPORINE, PHENYTOIN, AND NIFEDIPINE SYSTEMIC ACTION MEDICINES." Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology 130, no. 3 (2020): e266. http://dx.doi.org/10.1016/j.oooo.2020.04.717.

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26

Tigaran, S. "A 15-year follow-up of phenytoin-induced unilateral gingival hyperplasia: a case report." Acta Neurologica Scandinavica 90, no. 5 (2009): 367–70. http://dx.doi.org/10.1111/j.1600-0404.1994.tb02739.x.

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27

Monterde-Coronel, M. E., and J. Asbun-Bojalil. "A case of gingival hyperplasia secondary to sodium phenytoin treatment and its homeopathic treatment." La Revue d'Homéopathie 5, no. 2 (2014): 84. http://dx.doi.org/10.1016/j.revhom.2014.04.012.

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28

Brown, Ronald S., Phillip T. Di Stanislao, William T. Beaver, and William K. Bottomley. "The administration of folic acid to institutionalized epileptic adults with phenytoin-induced gingival hyperplasia." Oral Surgery, Oral Medicine, Oral Pathology 71, no. 5 (1991): 565–68. http://dx.doi.org/10.1016/0030-4220(91)90363-h.

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29

Lin, Chun-Jung, Ming-Fang Yen, Oliver Yoa-Pu Hu, et al. "Association of Galactose Single-Point Test Levels and Phenytoin Metabolic Polymorphisms with Gingival Hyperplasia in Patients Receiving Long-Term Phenytoin Therapy." Pharmacotherapy 28, no. 1 (2008): 35–41. http://dx.doi.org/10.1592/phco.28.1.35.

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30

Saito, Keiichi, Shiro Mori, Naoko Tanda, and Seizaburo Sakamoto. "Expression of p53 Protein and Ki-67 Antigen in Gingival Hyperplasia Induced by Nifedipine and Phenytoin." Journal of Periodontology 70, no. 6 (1999): 581–86. http://dx.doi.org/10.1902/jop.1999.70.6.581.

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31

Roed-Petersen, Bjarne. "The potential use of CO2-laser ginagivectomy for phenytoin-induced gingival hyperplasia in mentally retarded patients." Journal of Clinical Periodontology 20, no. 10 (1993): 729–31. http://dx.doi.org/10.1111/j.1600-051x.1993.tb00698.x.

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32

TREVISOL-BITTENCOURT, PAULO CÉSAR, VICTOR REIS DA SILVA, MÁRCIO ALCIDES MOLINARI, and ANDRÉ RIBEIRO TROIANO. "Phenytoin as the first option in female epileptic patients?" Arquivos de Neuro-Psiquiatria 57, no. 3B (1999): 784–86. http://dx.doi.org/10.1590/s0004-282x1999000500008.

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OBJECTIVE: Phenytoin (PHT) is one of the first-choice drugs in several epileptic syndromes, mostly in partial epilepsies, in which case it is effective as carbamazepine and phenobarbital. However, like any other anti-epileptic drug (AED), unpleasant side-effects are not rare. The aim of this study is the evaluation of dermatological troubles related to chronic PHT usage in female patients. METHOD: Between 1990-93, 731 new patients underwent investigation for epilepsy at the Multidisciplinary Clinic for Epilepsy in our State. In this sample 283 were AED users at the time of the first assessment
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33

Gunasekaran, Vijay, Saravanakumar Subbaraj, and Tirou Aroul. "The efficacy of phenytoin dressing in healing of diabetic ulcer: a randomized control study." International Surgery Journal 4, no. 10 (2017): 3311. http://dx.doi.org/10.18203/2349-2902.isj20174174.

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Background: Diabetic foot infection constitutes up to 10 percent of diabetes-related hospital admissions and the prevalence of diabetes is 2.4% in rural and 12-17% in urban settings. The quest for better wound healing agents for diabetic ulcers is perhaps one of the oldest challenges for medical practice. One such agent that has been tried in wound healing is phenytoin. A common side effect of phenytoin (diphenylhydantoin) treatment for epilepsy is gingival hyperplasia. This stimulatory effect of phenytoin on connective tissue suggested a possibility for its use in wound healing.Methods: 60 pa
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34

Johnson, Bradley D., A. Sampath Narayanan, Helen P. Pieters, and Roy C. Page. "Effect of cell donor age on the synthetic properties of fibroblasts obtained from phenytoin-induced gingival hyperplasia." Journal of Periodontal Research 25, no. 2 (1990): 74–80. http://dx.doi.org/10.1111/j.1600-0765.1990.tb00895.x.

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35

Saito, Keiichi, Shiro Mori, Naoko Tanda, and Seizaburo Sakamoto. "Immunolocalization of c-Myc and bcl-2 Proto-Oncogene Products in Gingival Hyperplasia Induced by Nifedipine and Phenytoin." Journal of Periodontology 71, no. 1 (2000): 44–49. http://dx.doi.org/10.1902/jop.2000.71.1.44.

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36

Saito, K., S. Mori, M. Iwakura, and S. Sakamoto. "Immunohistochemical localization of transforming growth factor beta, basic fibroblast growth factor and heparan sulphate glycosaminoglycan in gingival hyperplasia induced by nifedipine and phenytoin." Journal of Periodontal Research 31, no. 8 (1996): 545–55. http://dx.doi.org/10.1111/j.1600-0765.1996.tb00519.x.

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37

Lipovskyte, Kamile, Anna Burrows, and Vijay Kale. "Identify effects of phenytoin and losartan on TGF‐beta using human gingival fibroblast‐1 (HGF‐1) cells and establish HGF‐1 cell culture as a model to study drug‐induced gingival hyperplasia." FASEB Journal 34, S1 (2020): 1. http://dx.doi.org/10.1096/fasebj.2020.34.s1.07522.

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38

Iacopino, Anthony M., Deborah Doxey, Christopher W. Cutler, et al. "Phenytoin and Cyclosporine A Specifically Regulate Macrophage Phenotype and Expression of Platelet-Derived Growth Factor and Interleukin-1 In Vitro and In Vivo: Possible Molecular Mechanism of Drug-Induced Gingival Hyperplasia." Journal of Periodontology 68, no. 1 (1997): 73–83. http://dx.doi.org/10.1902/jop.1997.68.1.73.

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39

DEGUCHI, Shinji, Akira OOYAMA, Mitsuo FUKUNO, et al. "Biochemical characterization of phenytoin-induced hyperplastic human gingival fibroblasts. Non-collagenous proteins biosynthesis." Nihon Shishubyo Gakkai Kaishi (Journal of the Japanese Society of Periodontology) 30, no. 4 (1988): 1047–54. http://dx.doi.org/10.2329/perio.30.1047.

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40

Dill, Russell E., and Anthony M. Iacopino. "Myofibroblasts in Phenytoin-Induced Hyperplastic Connective Tissue in the Rat and in Human Gingival Overgrowth." Journal of Periodontology 68, no. 4 (1997): 375–80. http://dx.doi.org/10.1902/jop.1997.68.4.375.

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41

Narayanan, A. S., D. F. Meyers, and R. C. Page. "Regulation of collagen production in fibroblasts cultured from normal and phenytoin-induced hyperplastic human gingiva." Journal of Periodontal Research 23, no. 2 (1988): 118–21. http://dx.doi.org/10.1111/j.1600-0765.1988.tb01343.x.

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42

GUNTURU, LAKSHMI NARASIMHA. "CASE REPORT ON PHENYTOIN-INDUCED IATROGENIC GINGIVAL HYPERPLASIA." Asian Journal of Pharmaceutical and Clinical Research, July 15, 2020, 1–2. http://dx.doi.org/10.22159/ajpcr.2020.v13i10.38761.

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Phenytoin is a diphenylhydantoin which is mostly used in the treatment of epilepsies and arrhythmias. Chronic usage of phenytoin leads to a number of undesirable effects. Most of the adverse effects associated with phenytoin are hirsutism, gingival hyperplasia, osteomalacia, cleft lip, and hypoplastic phalanges. Gingival hyperplasia is an increase in the number of cells or an increase in the size of gingiva is observed due to a defect in collagen metabolism. Here, we report a case of a 20-year-old male patient who was diagnosed with gingival hyperplasia due to the prolonged use of phenytoin fo
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43

Bakshi, Satvinder Singh. "Phenytoin-induced gingival hyperplasia." Erciyes Medical Journal, 2019. http://dx.doi.org/10.14744/etd.2019.86422.

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44

Cláudio, Marina Módolo, João Victor Soares Rodrigues, Valdir Gouveia Garcia, and Leticia Helena Theodoro. "Prevalence of Gingival Hyperplasia Induced by Anticonvulsants: A Systematic Review." Brazilian Dental Science 24, no. 1 (2020). http://dx.doi.org/10.14295/bds.2021.v24i1.2112.

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Objective: Gingival hyperplasia (GH) is one of the side effects of anticonvulsant drugs. The aim of this study was to verify the prevalence of GH associated with the use of anticonvulsant, through a systematic review. Material and Methods: Systematic search was done at databases Pubmed and Embase between January 1984 and March of 2020 for identification of articles addressing the prevalence of GH associated with the use of anticonvulsant drugs. The methodological index for non-randomized studies (MINORS) was independently assessed for quality in the selected papers. Results: The search identif
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45

Ieiri, I., W. Goto, S. Higuchi, et al. "Effect of 5-(p-hydroxyphenyl)-5-phenylhydantoin (p-HPPH) enantiomers, major metabolites of phenytoin, on the occurrence of chronic-gingival hyperplasia: in vivo and in vitro study." European Journal of Clinical Pharmacology 49, no. 1-2 (1995). http://dx.doi.org/10.1007/bf00192358.

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