Relevant bibliographies by topics / Physiological

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Physiological'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 July 2025

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Journal articles on the topic "Physiological"

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Chen, Wujun, Shuai Wang, Yudong Wu, Xin Shen, Shutan Xu, Zhu Guo, Renshuai Zhang, and Dongming Xing. "The Physiologic Activity and Mechanism of Quercetin-Like Natural Plant Flavonoids." Current Pharmaceutical Biotechnology 21, no. 8 (July 8, 2020): 654–58. http://dx.doi.org/10.2174/1389201021666200212093130.

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The term “vitamin P” is an old but interesting concept. Most substances in this category belong to the family of flavonoids. “Vitamin P” has also been used to define the activity of some flavonoids, including quercetin, myricetin, and rutin. According to experimental studies, the “quercetin-like natural plant flavonoids” are beneficial to the body due to their various physiological and pharmacological activities in large doses (5 μM in vitro, 50 mg/kg in mice and 100 mg/kg in rats). The physiologically achievable concentration is 10 to 100 nM, which is quite high and hard to achieve from a normal diet. Thus, the physiologic activity and mechanism of "vitamin P" are still not clear. It should be noted that the quercetin-like natural plant flavonoids are physiological co-factors of cyclooxygenases (COXs), which are the rate-limiting key enzymes of prostaglandins. These quercetin-like natural plant flavonoids can strongly stimulate prostaglandin levels at lower doses (10 nM in vitro and in 0.1 mg/kg in vivo in rats). Although these "vitamin P" substances are not original substances in the body, their physiological functions affect the body. This review is focused on the most compelling evidence regarding the physiologic role and mechanism of quercetin-like natural plant flavonoids, which may be useful in understanding the physiological functions of "vitamin P", with the goal of focusing on the role of flavonoids in human physiological health.
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Brown, Ronald P., Michael D. Delp, Stan L. Lindstedt, Lorenz R. Rhomberg, and Robert P. Beliles. "Physiological Parameter Values for Physiologically Based Pharmacokinetic Models." Toxicology and Industrial Health 13, no. 4 (July 1997): 407–84. http://dx.doi.org/10.1177/074823379701300401.

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Cavasin, Pedro Yuri, Wilson Vicente Souza Pereira, and Heloisa Oliveira dos Santos. "Physiological quality of Capsicum chinense 'Adjuma' seeds during development." Bioscience Journal 39 (May 5, 2023): e39075. http://dx.doi.org/10.14393/bj-v39n0a2023-64823.

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Once “bode” pepper plants (Capsicum) have continuous development and fructification, it is not possible to determine a specific season for seed physiologic maturation and harvest. This research aimed to evaluate physiological quality of “bode” pepper (Capsicum chinense 'Adjuma’) during fruit development and enzymes expression on seed germination. Manually extracted seeds were submitted to physiological tests being evaluated germination at first count (FC), final (G), and germination speed index (GSI), additionally, enzymatic analyses were carried. The lowest values for the physiological tests were observed for initial development stages. The greatest for FC, G and IVG were observed for seeds from fruits 70 days after anthesis (DAA). Superoxide dismutase enzyme has the highest values on seeds harvested at 49 DAA while malate dehydrogenase has more expression at 70 DAA. Catalase, alcohol dehydrogenase, and esterase have the higher expression at 63 DAA. The maturation stage influences bode pepper seed physiological quality, being seeds harvested at 70 DAA those with the better results on the evaluated parameters, thus, considered physiologically mature and the indicated time for harvest.
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Fiserova-Bergerova, Vera. "Extrapolation of physiological parameters for physiologically based simulation models." Toxicology Letters 79, no. 1-3 (September 1995): 77–86. http://dx.doi.org/10.1016/0378-4274(95)03359-s.

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KRAUSE, E. "Cardiac energetics: Physiological and patho-physiological aspects." Journal of Molecular and Cellular Cardiology 23 (July 1991): S52. http://dx.doi.org/10.1016/0022-2828(91)90669-d.

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Pandey, Sandhya, Ravi Kumar Chittoria, Mohapatra Devi Prasad, Friji M.T., Dinesh Kumar Sivakumar, and Bibilash Babu Suseela. "Conventional Palatoplasty Versus Physiological Palatoplasty." New Indian Journal of Surgery 7, no. 2 (2016): 95–102. http://dx.doi.org/10.21088/nijs.0976.4747.7216.2.

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Verma, S. S. "Regression models in physiological research." Zeitschrift für Morphologie und Anthropologie 83, no. 1 (March 1, 2001): 129–38. http://dx.doi.org/10.1127/zma/83/2001/129.

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Shahnawaz, Fatima. "Spirituality and Psycho-Physiological Health." Global Journal For Research Analysis 3, no. 2 (June 15, 2012): 190–91. http://dx.doi.org/10.15373/22778160/february2014/61.

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Aslam, Muhammad Sohail, and Muhammad Jabran Javaid Sidhu. "PHYSIOLOGICAL EFFECTS OF MUSIC INHUMANBEINGS." Journal of Akhtar Saeed Medical & Dental College 04, no. 03 (September 30, 2022): 140–44. https://doi.org/10.51127/jamdcv4i3ra01.

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Music and ancient art arebeing used as a mode of entertainment. There isevidenceof its effectiveness as a therapy fordifferent diseases.Listening to rock music (440 Hz) results inincreased cortisol levels appetite, blood glucose, lipids and body weight. Whereaslistening to classical music (432 Hz) leads to a significant lowering of cortisol level due to its soothing effect. It also decreases blood glucose, lipids, appetite,and body weight.Classical music exposure helps to control anxiety.Conclusion:Listening to classical music helpsto relieve anxiety and reduces blood glucose, lipids,and body weight.Key Words:Music, Human body, Anxiety
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Lagos, Leah M. "The Physiologically Gifted Child." Biofeedback 41, no. 2 (June 1, 2013): 62–65. http://dx.doi.org/10.5298/1081-5937-41.2.06.

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The Physiologically Gifted Child model proposes that sensitive children can turn their vulnerability to stress into strength through physiological training. When the child's sensitivity is viewed as a physiologically modifiable trait, the child is less likely to engage in self-blame and more likely to engage in self-care strategies to manage emotions. The author emphasizes that cardiovascular reactivity is significantly higher in a child with physiological giftedness. Because the physiologically gifted child is wired to react more intensely to stress, he or she is unlikely to be able to reduce sensitivity without addressing its physiological origin. The author proposes biofeedback and self-care activities as methods to reduce a physiologically gifted child's vulnerability to stress while simultaneously allowing the child to benefit from his or her extraordinary gifts of feeling and perceiving. A description of a physiologically gifted child and optimal performance strategies illustrate this concept.
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Dissertations / Theses on the topic "Physiological"

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Brauns, Seth Clint Aron. "Physiological and non-physiological induction of gastrointestinal differentiation." Thesis, University of Port Elizabeth, 1999. http://hdl.handle.net/10948/d1015521.

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The human colonic carcinoma cell lines HT-29 and Caco-2 both exhibit structural and functional differentiation under appropriate culture conditions. HT-29 can be induced to differentiate by treatment with short-chain fatty acids or acetoacetate. Caco-2 cells differentiate spontaneously upon contact inhibition. In this study HT-29 cells were treated with 5 mM acetate, propionate, butyrate and acetoacetate (physiological inducers) to assess their effects on the expression of carbonic anhydrase 1, sucrase-isomaltase and alkaline phosphatase which are reported to be markers of gastrointestinal differentiation. The maturation induction observed was compared to that of the spontaneous differentiation observed in Caco-2 cells. Assays were performed over an 18 day period. Results showed a close correlation (p < 0.05) between HT-29 and Caco-2 cell on days 4 and 12. These results indicate that differentiation reported in both cell lines is comparable and can be used as a basis for further comparative studies. In addition, parallel experiments to the above were conducted using a selection of nine rationally designed cyclic dipeptides (CDPs) potential drug entities which were chosen as non-physiological inducers. The results showed that the cyclic dipeptides were able to induce the gastrointestinal phenotype as observed in HT-29 cells treated with physiological inducers. Studies on the effects of energy-related metabolism in HT-29 and Caco-2 cells as induced by physiological and non-physiological inducers indicated that energy metabolism is a significant role player in gastrointestinal differentiation. The results reported show a decrease in ATP concentrations indicating that the cyclic dipeptides, like physiological inducers, affect the energy state of the HT-29 cells and thus may effect the differentiation of these cells. A positive correlation was found between histone phsophorylation and differentiation confirming that histone phsophorylation was partly responsible for the decrease in ATP concentrations. It is suggested that the induction of differentiation in HT- 29 cells could be either due to non-specific transcription of genes by activation of a chromatin switch or specific by the activation of signal transduction pathways based on the flux of ATP through the cells. Differential display RT-PCR is probably the most sensitive method that could be used to validate the suggestion of either a nonspecific transcription of genes or a specific differentiation reported for HT-29 cells.
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Andersson, Kennet. "Effects of Physiological Variations." Thesis, Umeå universitet, Institutionen för matematik och matematisk statistik, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-51310.

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Heart ischemia, the precursor to an infarction, is one of the most common diseases in the western world. Today, the electrocardiogram or ECG is the most widely used tool to diagnose the disease. However, it often fails to detect the ischemia or to give an adequate picture of the size and location. Therefore, the potential of increasing knowledge obtained through mathe- matical models is very high. In this thesis the bidomain model is used to describe the electrical activity in the heart and body with ischemia incorporated into the model. To solve the equations set up by the bidomain model, the finite element method is used. Different physiological variations have been made to the body, these include changing the location of the heart and varying the conductivities in the body. The solution to the equations is then studied at the body surface. The main question asked is whether it is possible to detect the location and size of different types of ischemia by analyzing the solution. The methods used for this have been Singular Value Decomposition and Su- pervised learning. The different vectors obtained from the decomposition are used to distinguish the location and size of the ischemia for different physiolog- ical variations. The results show that it is possible to distinguish the location of the ischemia but that it probably will be more difficult to find the correct size since the change in size is harder to separate from other physiological variations, such as the conductivity of the body. Although relatively simple methods have been used, they indicate that, with further development, they can be used for the purpose of detecting the different ischemia.
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Aiken, Simon Piers. "Physiological transport of zinc." Thesis, University of Southampton, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278677.

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Lørup, Lise. "Physiological patterns in pregnancy." Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:d14b1507-140e-4647-889f-d6b98237100d.

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The Confidential Enquiries into Maternal Deaths in the UK have highlighted an urgent need for development of a national Modified Early Obstetric Warning System (MEOWS) to aid the more timely recognition and treatment of complications in pregnancy. Six vital signs are used to assess the clinical status of women presenting acutely during and immediately after pregnancy: heart rate, oxygen saturation, systolic and diastolic blood pressure, temperature and respiratory rate. In the case of MEOWS, these define the thresholds that determine if a woman requires further medical review. Dynamic changes in maternal vital sign physiology associated with pregnancy complicate the use of vital signs to detect physiological deterioration. These changes are currently poorly described in the literature. The aim of this work is to establish the normal physiological response to pregnancy in low-risk pregnant women, in terms of expected changes in vital signs. Using this knowledge, we will propose the first evidence-based, gestation-specific MEOWS chart. To achieve this, we have set up the Prediction of Physiological Patterns in Pregnancy (4P) study. Using a custom-designed digital health system, healthcare professionals and women self-monitoring at home collect large numbers of vital sign measurements electronically. In particular, as respiratory rate currently is the only vital sign measured manually in routine clinical care, we propose two methods for electronic measurement of respiratory rate: using the in-built accelerometer of a smartphone and using the photoplethysmogram signal output by a pulse oximeter. These methods are validated against the current reference standard derived from a 15-second nurse count of breaths. This thesis uses a dataset of 2,645 sets of vital sign measurements, collected from 439 women during pregnancy, in the 4P study. Using the electronic methods for measurement of respiratory rate, we obtain respiratory rate estimates with a mean absolute error of 2.9 respirations per minute compared to the reference measurement. From these data, we design reference ranges for changes in all six vital signs conditional on gestational age. Using a centile-based approach, we propose gestation-specific thresholds of normality for each vital sign, which will form the basis of a new MEOWS score to alert for physiological deterioration. Defining normality is the first step in recognising abnormality.
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Laubacher, Timothy Charles. "Physiological measures of presence." The Ohio State University, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=osu1407237660.

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Pratt, David Andrew. "Physiological studies of thrombospondin." Thesis, University of Edinburgh, 1991. http://hdl.handle.net/1842/20119.

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Thrombospondin (TSP), a glycoprotein of wide cellular distribution, exhibits several activities important in platelet aggregation, haemostasis and cell adhesion. The aim of this project was to investigate the production and role of TSP in human tissues and fluids; particularly its source in the breast and relation to malignant disease. Most circulating TSP was contained within platelet α-granules and released upon activation of these cells. Infused TSP appeared to be rapidly bound, whilst its clearance from the circulation was relatively slow. Extra-platelet sources contributed substantially to basal plasma levels; platelet-associated TSP may therefore be a better indicator of platelet activation than plasma concentration. TSP concentration in breast cyst fluids varied according to cyst type and correlated inversely with epidermal growth factor. TSP was present in milk at high levels compared with plasma and its pattern of secretion resembled that of IgA; it may be that white cells which infiltrate the mammary gland are a major source of TSP in breast secretions. Very high levels of TSP were found in malignant breast tissue compared with non-malignant breast and were associated with the centre of the tumour mass. Positive correlation between TSP and von Willebrand factor suggested that endothelium contributes to the high levels of both proteins in malignant breast, whereas lack of correlation between TSP and tissue plasminogen activator argued against epithelium being the source of TSP in breast cancer. In conclusion, the results presented in this thesis support current knowledge of TSP as an adhesive glycoprotein of platelets, vessel walls and connective tissues. Novel studies in the breast have revealed a marked association of TSP with cancerous tissue and some cyst fluids, which could be due to production by vascular endothelium and certain white blood cells. Whilst the role of TSP in the breast remains to be defined, its relation to disease states may be of particular physiological significance.
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Ramsey, Michael W. "Physiological Technology for Coaches." Digital Commons @ East Tennessee State University, 2012. https://dc.etsu.edu/etsu-works/4105.

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Schadock, Ines Claudia. "Physiological role of prolylcarboxypeptidase." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2011. http://dx.doi.org/10.18452/16412.

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Prolylcarboxypeptidase (PRCP, EC3.4.16.2) ist ein ubiquitär exprimiertes Enzym, mit höchster Expression im Maushirn. Es spaltet spezifisch die letzte carboxyterminale Aminosäure von Substraten, deren vorletzte Aminosäure ein Prolin ist. Seine bisher publizierten Substrate Angiotensin II (AngII) und alpha Melanocortin Stimulierendes Hormone (alphaMSH) legen eine Rolle von PRCP in der Entwicklung von kardiovaskulären und metabolischen Krankheiten nahe. Um die in vivo Funktion von PRCP zu studieren, wurde eine Knockout Maus generiert (PRCP-/-). Metabolischer Phänotyp: PRCP-/- Mäuse zeigten generell ein reduziertes Körpergewicht, selbst wenn sie über Monate mit einer Hochfettdiät versorgt wurden. Erhöhte Plasmaleptin Werte und Proopiomelanocortin (pomc) Expression in knockout Hypothalami wiesen auf eine wichtige Rolle von PRCP in der Regulation von Futteraufnahme und Energiehomöostase hin. Eines der Genprodukte von pomc ist alphaMSH, welches im Hypothalamus die Futteraufnahme terminiert. Die carboxyterminale Struktur dieses Neuropeptids erfüllt alle Voraussetzungen, um von PRCP gespalten zu werden. Zudem konnte prcp Promotoraktivität in den selben Hirnstrukturen gezeigt werden, in denen auch alphaMSH-Wirkung beschrieben wurde. Eine mögliche Funktion von PRCP wäre somit die Inaktivierung des Appetitzüglers alphaMSH im Hypothalamus. Kardiovasculärer Phänotyp: Zunächst erwiesen sich zirkulierende Ang-Peptide in PRCP-/- Mäusen als unverändert. Jedoch konnte ein erhöhtes Niveau des Degradationsproduktes Ang1-7 in der Niere gezeigt werden. Die Entdeckung einer erhöhten Enzymaktivität von Angiotensin Converting Enzyme 2 (ACE2) in PRCP-/- Nieren, wurde als Kompensation der fehlenden PRCP Aktivität in PRCP-/- Nieren interpretiert. bot einen Erklärungsansatz für dieses Ergebnisse. Es wird davon ausgegangen, daß ACE2 die fehlende PRCP Aktivität in knockout Mäusen kompensiert. Das es sich hierbei um eine lokale begrenzte Kompensation handeln muß, zeigten der erhöhte Blutdruck und Herzrate, sowie die milde Herzhypertrophie. Da spezifische prcp Promotoraktivität in Hirnnuclei gefunden wurde, die in die Kontrolle der Herzfrequenz und des Blutdrucks involviert sind, wird eine regulatorische Funktion von Hirnstamm-PRCP auf Herzrhytmus und Blutdruck vermutet.<br>Prolylcarboxypeptidase (PRCP, EC3.4.16.2) is an enzyme specifically cleaving the last carboxy-terminal amino acid from substrates containing a penultimate proline. Its known potential substrates are linked to cardiovascular and metabolic phenomenon. To analyse the in vivo function of this enzyme a PRCP knockout mouse was generated. Homozygous knockout mice are viable but show tendency of decreased life span. In mice prcp expression is present in all tissues tested with very specific localizations of prcp promotor activity to distinct brain areas within the cortex, hippocampus, hypothalamus and the brain stem. The metabolic phenotype of PRCP deficient mice is characterized by low body weight even when feeding the animals a high fat diet. The increased plasma leptin levels and elevated expression of proopiomelanocortin gene (pomc) found in knockout hypothalami suggests an involvement of PRCP in the regulation of food intake and energy homeostasis. One of the gene products of pomc is alpha-melanocortin stimulating hormone (alphaMSH) that is terminating feeding when released from hypothalamic POMC neurons. Its carboxy-terminal structure is fitting the cleavage preferences of PRCP. Prcp promotor activities are localized in arcuate nucleus and paraventricular nucleus, brain areas of known alphaMSH signalling, supporting a role of PRCP in the degradation of central alphaMSH. The impact of PRCP on angiotensin II (AngII) metabolism was studied by determining the level of AngII and its degradation product Ang1-7 in blood and tissues. But instead of increased AngII levels due to the missing degradation enzyme in knockout mice, Ang1-7 levels were increased in kidney. These results were explainable by the increased activity of angiotensin converting enzyme 2 (ACE2) found in kidney. Probably ACE2 is compensating the lack of PRCP in the knockout mouse. Nevertheless, blood pressure and heart rate of PRCP knockout mice was increased. The mild hypertension was accompanied by mild hypertrophy of the hearts. Prcp promotor activity was found in brain stem an area important for regulation of blood pressure and heart rate suggesting that central PRCP regulates blood pressure.
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Veluvolu, Kalyana [Verfasser]. "Real-time Filtering of Physiological Tremor for Microsurgery. Physiological Tremor Robotic Compensation / Kalyana Veluvolu." München : GRIN Verlag, 2020. http://d-nb.info/1220832847/34.

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Carrithers, John A. "Effects of post-exercise carbohydrate-protein feedings on muscle glycogen restoration." Virtual Press, 1999. http://liblink.bsu.edu/uhtbin/catkey/1133741.

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The purpose of this investigation was to determine the effects of post-exercise carbohydrate-protein feedings on muscle glycogen restoration following exhaustive cycle ergometer exercise. Seven male collegiate cyclist (age=25.6±3.3y, ht.=180.9±8.5cm, wt.=75.4±10.7kg, VO2max=4.20±0.4 1•miri 1) performed three trials, each separated by -lwk, 1) 100% (x-D glucose (CHO), 2) 70% carbohydrate-20% protein-10% fat (CHOPRO), and 3) 86% carbohdyrate-14% amino acid (CHO-AA). All feedings were eucaloric, based upon 1.0 g•kgb.W.'1•hr"1 of carbohydrate, and administered every half hour during a four hour muscle glycogen restoration period in an 18% wt./vol. solution. Muscle biopsies were obtained immediately and four hours post exercise. Following the exhaustive exercise and every half hour for four hours a blood sample was drawn. Muscle glycogen concentrations increased 53%, 47%, and 57% for the CHO, CHO-PRO, and CHO-AA feedings, respectively, however no differences among the feedings were apparent in muscle glycogen restoration. The plasma glucose and insulin concentrations demonstrated no differences throughout the restoration period among the three feedings. These results suggest that muscle glycogen restoration does not appear to be enhanced with the addition of either protein or amino acids to an eucaloric carbohydrate feeding following an exhaustive cycle exercise. However, it appears that if adequate amounts of carbohydrates are consumed (greater than 0.70 g•kgb,W,."'•hf' carbohydrate) following exhaustive exercise, maximal muscle glycogen restoration occurs.<br>School of Physical Education
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Books on the topic "Physiological"

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Lynn, Sayer, and South Bank University. Distance Learning Centre., eds. Physiological hurdles. London: Distance Learning Centre, South Bank University, 1993.

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B, Graham Robert. Physiological psychology. Belmont, Calif: Wadsworth Pub. Co., 1990.

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Hinchliff, Susan M. Physiological hurdles. London: DistanceLearning Centre, South Bank Polytechnic, 1990.

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Gould, Dinah. Physiological insights. London: Distance Learning Centre, South Bank University, 1993.

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Gould, Dinah. Physiological insights. London: Distance Learning Centre, South Bank Polytechnic, 1990.

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L, Leiman Arnold, ed. Physiological psychology. 2nd ed. New York: Random House, 1989.

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Symposium Pharmaco-Clinique Roussel Uclaf (16th 1993 Paris, France). Nitric oxide, a physiological and patho-physiological mediator. Romainville: Institut scientifique Roussel, 1993.

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da Silva, Hugo Plácido, Andreas Holzinger, Stephen Fairclough, and Dennis Majoe, eds. Physiological Computing Systems. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-45686-6.

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Khoo, Michael. Physiological Control Systems. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2018. http://dx.doi.org/10.1002/9781119058786.

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Lambers, Hans, F. Stuart Chapin, and Thijs L. Pons. Plant Physiological Ecology. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-78341-3.

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Book chapters on the topic "Physiological"

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Leavens, Teresa. "Physiological Models." In Comparative Pharmacokinetics, 225–40. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9780470959916.ch11.

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Epstein, Scott K. "Physiological Barriers." In A Practical Guide to Mechanical Ventilation, 263–75. Chichester, UK: John Wiley & Sons, Ltd, 2011. http://dx.doi.org/10.1002/9780470976609.ch20.

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Schubert, Hendrik, Irena Telesh, Mikko Nikinmaa, and Sergei Skarlato. "Physiological adaptations." In Biological Oceanography of the Baltic Sea, 255–78. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-007-0668-2_7.

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Laurent, Francois, and Michel Montaudon. "Physiological Interactions." In Integrated Cardiothoracic Imaging with MDCT, 109–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-72387-5_8.

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Binley, Katie. "Physiological Targeting." In Vector Targeting for Therapeutic Gene Delivery, 505–25. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2003. http://dx.doi.org/10.1002/0471234303.ch23.

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Sharp, Peter F., and Russell Philips. "Physiological Optics." In The Perception of Visual Information, 1–32. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-1836-4_1.

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Benjamin, Rachele. "Physiological Needs." In Encyclopedia of Personality and Individual Differences, 3925–27. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_1495.

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Zotz, Gerhard. "Physiological Ecology." In Plants on Plants – The Biology of Vascular Epiphytes, 95–148. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39237-0_5.

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Thompson, Jack George. "Physiological Models." In The Psychobiology of Emotions, 265–82. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4899-2121-5_13.

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Grierson, W. "Physiological Disorders." In Fresh Citrus Fruits, 361–78. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-8792-3_14.

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Conference papers on the topic "Physiological"

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Robinson, Raquel, Katelyn Wiley, Amir Rezaeivahdati, Madison Klarkowski, and Regan L. Mandryk. ""Let's Get Physiological, Physiological!"." In CHI PLAY '20: The Annual Symposium on Computer-Human Interaction in Play. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3410404.3414227.

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Allanson, Jennifer, and Gillian M. Wilson. "Physiological computing." In CHI '02 extended abstracts. New York, New York, USA: ACM Press, 2002. http://dx.doi.org/10.1145/506443.506655.

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Ghassemi, Marzyeh, Tristan Naumann, Finale Doshi-Velez, Nicole Brimmer, Rohit Joshi, Anna Rumshisky, and Peter Szolovits. "Unfolding physiological state." In KDD '14: The 20th ACM SIGKDD International Conference on Knowledge Discovery and Data Mining. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2623330.2623742.

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Lopes, Pedro, Lewis L. Chuang, and Pattie Maes. "Physiological I/O." In CHI '21: CHI Conference on Human Factors in Computing Systems. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3411763.3450407.

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Wang, Weinan, and Laleh Najafizadeh. "Imaging Physiological Signals." In 2022 56th Asilomar Conference on Signals, Systems, and Computers. IEEE, 2022. http://dx.doi.org/10.1109/ieeeconf56349.2022.10051933.

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Boudaya, Amal, Siwar Chaabene, Bassem Bouaziz, Hela Zouari, Sana ben Jemea, and Lotfi Chaari. "Physiological/Non-physiological artifacts classification using EEG signals based on CNN." In 2022 International Conference on Technology Innovations for Healthcare (ICTIH). IEEE, 2022. http://dx.doi.org/10.1109/ictih57289.2022.10112088.

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"Physiological Signal Processing for Emotional Feature Extraction." In International Conference on Physiological Computing Systems. SCITEPRESS - Science and and Technology Publications, 2014. http://dx.doi.org/10.5220/0004727500400047.

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Escourrou, P. "Non-invasive physiological measurements." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.95111.

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Kingsley, Stuart A., Sriram Sriram, Andrea Pollick, and John Marsh. "Photrodes for physiological sensing." In Biomedical Optics 2004, edited by Israel Gannot. SPIE, 2004. http://dx.doi.org/10.1117/12.529473.

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Deniskin, V. V. "PHYSIOLOGICAL BASES Kettlebell Lifting." In Всероссийская научно-практическая конференция "ОПТИМИЗАЦИЯ УЧЕБНО-ВОСПИТАТЕЛЬНОГО И ТРЕНИРОВОЧНОГО ПРОЦЕССА В УЧЕБНЫХ ЗАВЕДЕНИЯХ ВЫСШЕГО ПРОФЕССИОНАЛЬНОГО ОБРАЗОВАНИЯ. ЗДОРОВЫЙ ОБРАЗ ЖИЗНИ КАК ФАКТОР ПРОФИЛАКТИКИ НАРКОМАНИИ". Сибирский юридический институт Министерства внутренних дел Российской Федерации, 2016. http://dx.doi.org/10.51980/2016_143_157.

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Reports on the topic "Physiological"

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Wiwanitkit, Viroj, and Viroj Wiwanitkit. PHYSIOLOGICAL GENOMICS IN MANIA. Buenos Aires: siicsalud.com, September 2017. http://dx.doi.org/10.21840/siic/148771.

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Tyack, Peter L., Andreas Fahlman, Michael Moore, Warren Zapol, and Richard Anderson. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2010. http://dx.doi.org/10.21236/ada541814.

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SPACE COMMAND PETERSON AFB CO. Air Force Physiological Training Program. Fort Belvoir, VA: Defense Technical Information Center, January 1996. http://dx.doi.org/10.21236/ada335011.

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Fahlman, Andreas, Peter L. Tyack, Michael Moore, Warren Zapol, Richard Anderson, and Steve Trumble. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2012. http://dx.doi.org/10.21236/ada573474.

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Fahlman, Andreas, Peter L. Tyack, Michael Moore, Warren Zapol, Richard Anderson, and Steve Trumble. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2013. http://dx.doi.org/10.21236/ada598516.

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Tyack, Peter L., Andreas Fahlman, Michael Moore, Warren Zapol, and Richard Anderson. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada598816.

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Tyack, Peter L., Andreas Fahlman, Michael Moore, Warren Zapol, and Richard Anderson. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada551257.

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Fahlman, Andreas, Peter L. Tyack, Michael Moore, Warren Zapol, Richard Anderson, and Steve Trumble. Physiological Monitoring in Diving Mammals. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada617976.

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Epstein, E., T. W.-M. Fan, R. M. Higashi, and W. K. Silk. Plant Physiological Aspects of Silicon. Office of Scientific and Technical Information (OSTI), July 2002. http://dx.doi.org/10.2172/761913.

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Matzen, Laura, Mallory Stites, Christina Ting, Breannan Howell, and Kyra Wisniewski. Physiological Characterization of Language Comprehension. Office of Scientific and Technical Information (OSTI), September 2021. http://dx.doi.org/10.2172/1821528.

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You might also be interested in the extended bibliographies on the topic 'Physiological' for particular source types:
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