Relevant bibliographies by topics / Pilawin

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Pilawin'

Author: Grafiati
Published: 4 June 2021
Last updated: 6 February 2022

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Journal articles on the topic "Pilawin"

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Graupner, Stefan, and Wilfried Wackernagel. "Pseudomonas stutzeri Has Two Closely RelatedpilA Genes (Type IV Pilus Structural Protein) with Opposite Influences on Natural Genetic Transformation." Journal of Bacteriology 183, no. 7 (2001): 2359–66. http://dx.doi.org/10.1128/jb.183.7.2359-2366.2001.

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ABSTRACT Pseudomonas stutzeri has type IV pili for which the pilA gene (here termed pilAI) provides the structural protein and which are required for DNA uptake and natural genetic transformation. Downstream of pilAIwe identified a gene, termed pilAII, coding for a deduced protein with a size similar to that of PilAI with 55% amino acid sequence identity and with a typical leader peptide including a leader peptidase cleavage site. Fusions to lacZ revealed that pilAII is expressed only about 10% compared topilAI, although the genes are cotranscribed as shown by reverse transcription-PCR. Surprisingly, insertional inactivation ofpilAII produced a hypertransformation phenotype giving about 16-fold-increased transformation frequencies. Hypertransformation also occurred in pilAI pilAII double mutants expressing heterologous pilA genes of nontransformable bacteria, like Pseudomonas aeruginosa or Dichelobacter nodosus. The overexpression of pilAII decreased transformation up to 5,000-fold compared to that of thepilAII mutant. However, neither inactivation ofpilAII nor its overexpression affected the amounts of [3H]thymidine-labeled DNA that were competence-specifically bound and taken up by the cells. In thepilAII mutant, the transformation by purified single-stranded DNA (which depends on comA andexbB, as does transformation by duplex DNA) was also increased 17-fold. It is concluded that PilAII suppresses a step in transformation after the uptake of duplex DNA into the cell and perhaps before its translocation into the cytoplasm. The idea that the degree of the transformability of cells could be permanently adjusted by the expression level of an antagonistic protein is discussed.
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HEIM, J., T. ANKE, U. MOCEK, B. STEFFAN, and W. STEGLICH. "Antibiotics from Basidiomycetes. XXIX: Pilatin, a new antibiotically active marasmane derivative from cultures of Flagelloscypha pilatii agerer." Journal of Antibiotics 41, no. 12 (1988): 1752–57. http://dx.doi.org/10.7164/antibiotics.41.1752.

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Gódor, Ferenc, Judit Hársing, Norbert Wikonkál, and Sarolta Kárpáti. "Pityriasis rubra pilaris." Bőrgyógyászati és Venerológiai Szemle 92, no. 1 (2016): 22–25. http://dx.doi.org/10.7188/bvsz.2016.92.1.4.

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Bajtel, Nóra, Linda Nagy, Alíz Kovács, and Zita Battyáni. "Pityriasis rubra pilaris." Bőrgyógyászati és Venerológiai Szemle 93, no. 1 (2017): 17–21. http://dx.doi.org/10.7188/bvsz.2017.93.1.4.

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Kubanov, Alexey, and Yulia Gallyamova. "Diagnosis and Treatment of Pityriasis Rubra Pilaris." Serbian Journal of Dermatology and Venereology 6, no. 4 (2014): 167–73. http://dx.doi.org/10.2478/sjdv-2014-0014.

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AbstractThe article deals with clinical diagnosis and treatment of pityriasis rubra pilaris (PRP). The authors analyze the diagnostic errors, present literature review, and their own observations. The clinical study included 23 patients with pityriasis rubra pilaris: 18 women and 5 men, average age of 54 ± 7.2. The clinical diagnosis of all examined patients was subsequently confirmed by histological analysis of the skin. The primary clinical diagnosis was psoriasis in 15 (65.2%) patients, 6 (26%) patients received treatment for toxic exanthema, and only 2 (8.8%) patients were presumptively diagnosed with pityriasis rubra pilaris. In conclusion, pityriasis rubra pilaris was initially misdiagnosed in 91.2% of patients. Considering the great number of diagnostic errors, we analyzed the main diagnostic and differential diagnostic features of PRP. The most effective of all synthetic retinoids in PRP treatment is acitretin. Although symptomatic improvement in PRP occurs within a month, substantial improvement, even clearing is possible within 4 - 6 months.
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Khan, Umair, Nahla Shihab, Robert G. Phelps, and Mark Lebwohl. "Successful Treatment of Pityriasis Rubra Pilaris with Brodalumab." SKIN The Journal of Cutaneous Medicine 4, no. 2 (2020): 139. http://dx.doi.org/10.25251/skin.4.2.7.

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Pityriasis Rubra Pilaris (PRP) is a rare inflammatory skin disease with highly variable clinical appearance. Treatment of PRP remains a challenge and has been mostly guided by case reports and case series. We report the first case of pityrisis rubra pilaris that is successfully treated with combination therapy of brodalumab and methotrexate.
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F, Maruma. "Imatinib-Induced Keratosis Pilaris in a Patient with Chronic Myeloid Leukemia." Asploro Journal of Biomedical and Clinical Case Reports 3, no. 3 (2020): 221–24. http://dx.doi.org/10.36502/2020/asjbccr.6216.

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We discuss the case of a 23 years old female with chronic myeloid leukemia. This patient developed keratosis pilaris nearly three months after she was treated with imatinib mesylate for her chronic myeloid leukemia. She was then referred to our dermatology outpatient clinic for assessment of the skin lesions. Prior to initiating the tyrosinase kinase inhibitor, the patient had no skin lesions at all. The skin lesions were widespread and diffuse in distribution and featured a primary morphology of follicular keratotic papules. This clinical picture was in keeping with keratosis pilaris. The further histological evaluation also confirmed features compatible with this diagnosis. The tyrosine kinase inhibitors are considered first-line therapy for the treatment of chronic myeloid leukemia. Imatinib mesylate belongs to the first generation of oral tyrosine kinase inhibitors and has a long-term control on the chronic myeloid leukemia and good safety profile. Most cases of keratosis pilaris are reported to arise from the second generation of tyrosine kinase inhibitors such as Nilotinib. We report a case of keratosis pilaris associated with imatinib mesylate in a patient with chronic myeloid leukemia.
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Hanfstingl, Kathrin, Agnes Pekar-Lukacs, Reinhard Motz, Emmanuella Guenova, and Wolfram Hoetzenecker. "Successful Treatment of Pityriasis Rubra Pilaris with Ixekizumab." Case Reports in Dermatology 10, no. 2 (2018): 97–100. http://dx.doi.org/10.1159/000488902.

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Pityriasis rubra pilaris is an inflammatory dermatologic disorder of unknown cause and often confounded with psoriasis. It is characterised by hyperkeratotic follicular papules, scaly erythematous plaques, palmoplantar keratoderma, and a progression to generalised erythroderma. Here, we report the case of a 68-year-old man with pityriasis rubra pilaris, who was successfully treated with ixekizumab, an interleukin-17A inhibitor.
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Arnold, Andreas W., and Stanislaw A. Buechner. "Keratosis pilaris and keratosis pilaris atrophicans faciei." JDDG 4, no. 4 (2006): 319–23. http://dx.doi.org/10.1111/j.1610-0387.2006.05933.x.

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Arnold, Andreas W., and Stanislaw A. Buechner. "Keratosis pilaris und Keratosis pilaris atrophicans faciei." JDDG 4, no. 4 (2006): ———. http://dx.doi.org/10.1111/j.1610-0387.2006.05933suppl.x.

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Dissertations / Theses on the topic "Pilawin"

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Pilari, Sabine [Verfasser]. "Novel approaches to mechanistic pharmacokinetic/pharmacodynamic modeling : lumping of PBPK models and systems biology / Sabine Pilari." Berlin : Freie Universität Berlin, 2011. http://d-nb.info/1025551605/34.

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Waibel, Dirk [Verfasser], and B. [Akademischer Betreuer] Pilawa. "Der Berezinskii-Kosterlitz-Thouless-Übergang von BaNi2V2O8 untersucht mit magnetischer Resonanz / Dirk Waibel. Betreuer: B. Pilawa." Karlsruhe : KIT-Bibliothek, 2014. http://d-nb.info/1060425475/34.

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Rattanasirivilai, Palinee. "The histopathology, direct immunofluorescence and immunoperoxidase staining in the distinction between lichen plano-pilaris and central centrifugal cicatricial alopecia." Thesis, Boston University, 2013. https://hdl.handle.net/2144/12201.

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Thesis (Sc.D.)--Boston University
Lichen planopilaris (LPP) and central centrifugal cicatricial alopecia (CCCA) are lymphocytic scarring alopecias. They share overlapping clinical and histopathologic findings. The goal of this dissertation has been to fmd reliably distinguishing features between these two conditions. Toward this goal, three different studies were conducted. Study 1 was a retrospective cross-sectional data analytic review of histologic features from patients identified by diagnosis of LPP or CCCA. Horizontal sections at level of the infundibulum, isthmus and inferior from scalp biopsies of 24 patients (19 CCCA and 5 LPP) were analyzed. The findings of unaffected follicular units, retained sebaceous glands and mild perifollicular inflammation were found to favor the diagnosis of CCCA. Dilated eccrine glands and heavy perifollicular inflammation were found to favor the diagnosis of LPP. Study 2 was a prospective cross-sectional data analysis study designed to identify and compare direct immunohistochemical findings in patients with LPP or CCCA. Vertical frozen sections of scalp biopsies from eleven patients (4 CCCA and 7 LPP) were stained with IgG, IgA, IgM, C3 and fibrinogen. No DIF finding that reliably distinguishes LPP from CCCA was found. The presence of a positive DIF was significantly correlated with the amount of inflammation. Study 3 investigated and compared T lymphocyte subsets, including T helper cells, cytotoxic T cells, Th 17 lymphocytes and regulatory T cells between LPP and CCCA cases. Subjects in this study were identical to study 2. There were no significantly distinctive T lymphocyte populations that differentiate between CCCA and LPP. There were higher numbers and percentages of CD8 positive cells in LPP compared to CCCA. The CD4:CD8 ratios were decreased in LPP and increased with duration of disease approaching proportions found in the normal hair follicle and CCCA. There were higher CD1a:CD3 ratios in LPP compared to CCCA. The CD1a:CD3 ratios in LPP decrease over time, approaching those found in CCCA. Results of this study confirm role of Langerhans cells as antigen presenting cells and role of cytotoxic lymphocytes in pathogenesis of LPP in early disease. Th17 lymphocytes and Tregs may have a role in both CCCA and LPP.
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Kamenskyi, Dmytro [Verfasser], Joachim [Akademischer Betreuer] Wosnitza, and Bernd [Akademischer Betreuer] Pilawa. "Electron spin resonance studies of frustrated quantum spin systems / Dmytro Kamenskyi. Gutachter: Joachim Wosnitza ; Bernd Pilawa. Betreuer: Joachim Wosnitza." Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://d-nb.info/1068151889/34.

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Ozerov, Mykhaylo [Verfasser], Joachim [Akademischer Betreuer] Wosnitza, and Bernd [Akademischer Betreuer] Pilawa. "High-field electron spin resonance in low-dimensional spin systems / Mykhaylo Ozerov. Gutachter: Joachim Wosnitza ; Bernd Pilawa. Betreuer: Joachim Wosnitza." Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2011. http://d-nb.info/1067189319/34.

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Ehlen, Oliver. "Leitbilder und romanhafte Züge in apokryphen Evangelientexten : Untersuchungen zur Motivik und Erzählstruktur : anhand des Protevangelium Jacobi und der Acta Pilati Graec. B /." Stuttgart : F. Steiner, 2004. http://catalogue.bnf.fr/ark:/12148/cb40210243v.

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Bizeto, Marcos Augusto. "Niobatos lamelares: síntese, caracterização, reatividade e estudo das propriedades luminescentes." Universidade de São Paulo, 2003. http://www.teses.usp.br/teses/disponiveis/46/46134/tde-04102006-110550/.

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O estudo apresentado nesta Tese diz respeito à química dos niobatos lamelares e aborda a síntese, caracterização, avaliação da reatividade intracristalina e das propriedades luminescentes desses materiais. Os niobatos lamelares utilizados foram o hexaniobato K4Nb6O17, o triniobato KNb3O8 e as perovskitas lamelares K1-xLnxCa2-xNb3O10 (Ln = La, Eu e x = 0,02; 0,25; 0,50; 0,75 e 1,00). Esses materiais são constituídos de lamelas que apresentam cargas negativas e a região interlamelar é preenchida por íons de potássio que mantêm a neutralidade dos sistemas. A reatividade intracristalina dos niobatos lamelares foi avaliada frente à intercalação de espécies simples como a butilamina e volumosas como o macrociclo porfirínico, o polioxocátion de alumínio e compostos orgânicos de silício. A alta densidade de carga lamelar dos niobatos lamelares dificulta a intercalação direta de espécies volumosas, o que fez com que novas rotas sintéticas fossem desenvolvidas a fim de permitir a imobilização de tais espécies na região interlamelar. As metodologias sintéticas desenvolvidas foram baseadas, principalmente, no uso de dispersões coloidais dos niobatos esfoliados que, a partir da reestruturação na presença da espécie convidada de interesse, tornou possível a intercalação de espécies volumosas. As propriedades luminescentes dos niobatos lamelares são extremamente dependentes da estrutura do material. Os niobatos com estrutura tipo perovskita não apresentam emissão enquanto que o hexaniobato apresenta emissão apenas a 77 K e o triniobato, à temperatura ambiente. Neste estudo foram avaliadas as propriedades luminescentes dos niobatos EuxK4-3xNb6O17, EuxK1-3xNb3O8 e KCa2Nb3O10 (intercalado com Eu3+ e dopado com 1 % de Eu3+ ou La3+). Foram observados processos de transferência de energia tanto nos niobatos intercalados com Eu3+ quanto nos dopados. A dopagem também provocou mudanças nas propriedades fotofísicas dos niobatos com estrutura perovskita, os quais passaram a apresentar emissão da matriz de niobato mesmo à temperatura ambiente.
The study described in this Thesis is related to the synthesis and evaluation of some chemical properties of layered niobates with formulas K4Nb6O17 (hexaniobate), KNb3O8 (triniobate) and K1-xLnxCa2-xNb3O10 (layered perovskites - Ln = La, Eu and x = 0.02; 0.25; 0.50; 0.75 and 1.00). These niobates are constituted of negative layers and an interlayer region filled with potassium ions that maintain the system charge neutrality. The reactivity of these niobates was evaluated through intercalation reactions of simple species such as butylamine and bulky species such as porphyrin, aluminum polyoxocation and organosilanes. The high charge density of the niobate layer makes the direct intercalation of bulky guest species more difficult. Therefore, to overcome this situation, new synthetic routes were developed. The intercalation of bulky species was achieved by using colloidal dispersions of exfoliated niobates that, upon restaking, incorporate the guest species into the interlayer region. The luminescent properties of the lamellar niobates are very dependent on the structure. Niobates that present a perovskite structure do not show emission even at liquid helium temperature. The hexaniobate presents emission at nitrogen liquid temperature and triniobate at both room and 77 K temperatures. In this study the luminescent properties of EuxK4-3xNb6O17, EuxK1-3xNb3O8 e KCa2Nb3O10 (intercalated with Eu3+ and doped with 1 % of Eu3+ or La3+) were evaluated. Charge transfers processes were observed in both intercalated and doped niobates with Eu3+ ion. The lanthanide doping also promoted changes in the photophysical properties of niobates with perovskite structure, which become to show emission of the niobate group even at room temperature.
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"A Pedagogical Approach to the Teaching of Six Selected Formative Euphonium Recital Pieces: Annotations, Exercises and Recording." Doctoral diss., 2010. http://hdl.handle.net/2286/R.I.8695.

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abstract: The purpose of this project was to provide a pedagogical resource for students and teachers to utilize when preparing six standard formative pieces from the euphonium repertoire. The guided practice sections are written in plain English with several instances of first person writing to explain certain concepts in a less formal way. This was done so that any teacher, regardless of level could help a younger, more inexperienced student. In addition, the sections of guided practice were written to help those teachers who may or may not be intimately familiar with the works chosen. The recording was designed to present the music in current published format, with no improvisation by the soloist. The solos that were chosen are either college preparatory pieces, or formative works for the younger collegiate musician. All of the pieces included are published, and as of September 2010, available for purchase. The works included are: Six Studies in English Folk Song, by Ralph Vaughan Williams, ed. Paul Droste, Introduction and Dance, by J. Edouard Barat, ed. Glenn Smith, Andante et Allegro, by Joseph- Guy Ropartz, ed. Shapiro, Sonata for Unaccompanied Euphonium (or Trombone), by Fred L. Clinard, Jr., Suite for Baritone, by Don Haddad, and Andante and Rondo, by Antonio Capuzzi, ed. Philip Catelinet.
Dissertation/Thesis
D.M.A. Music 2010
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Almeida, Guilherme João Calado dos Santos Portas de. "Pitiríase Rubra Pilar - novas perspetivas de tratamento." Master's thesis, 2019. http://hdl.handle.net/10316/89894.

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Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina
A Pitiríase Rubra Pilar (PRP) é uma dermatose papulodescamativa, rara, crónica e de etiologia desconhecida. A sua prevalência exata permanece desconhecida, variando de 1/5000 até 1/50000.A classificação atualmente mais aceite é a de Griffiths modificada, que define a PRP em 6 diferentes subtipos, de acordo com as suas manifestações clínicas, idade de início dos sintomas e associação à infeção pelo Vírus da Imunodeficiência Humana (VIH). Normalmente, a PRP surge de forma esporádica, mas também pode surgir como forma familiar. A patogénese da PRP pode estar eventualmente ligada a mutações de ganho de função do gene caspase recruitment domain family, member 14 (CARD14). Também se coloca a possibilidade de a PRP estar relacionada com distúrbios na metabolização e no transporte da Vitamina A para a pele, através da Retinol Binding Protein (RBP), com infeções e com reações idiossincráticas a fármacos e a radiação ultravioleta (UV).A PRP pode estar associada, ainda que de forma mal-esclarecida, a condições autoimunes e neoplásicas, podendo manifestar-se raramente como uma Síndrome Paraneoplásica. A apresentação clínica da PRP sugere o seu diagnóstico, nomeadamente a presença de pápulas hiperqueratósicas foliculares, rodeadas por halo esbranquiçado, que ao coalescerem formam placas de bordos elevados, com progessão cefalocaudal, associadas ao aparecimento de escamas espessadas, quebradiças em tom de laranja/salmão. À dermatoscopia, o aspeto é muito típico, sendo observáveis rolhões queratósicos esbranquiçados, rodeados por halos amarelo-alaranjados, associados a vasos organizados em clusters à periferia. A análise histopatológica da biópsia de pele é o gold-standard para o diagnóstico, permitindo-nos achar características classicamente atribuídas à PRP: paraqueratose e ortoqueratose, hiperqueratose irregular, acantose epidérmica e plugging folicular. A PRP é uma condição que, dada a sua baixa frequência na população e a sua particular semelhança a uma dermatose bem mais comum, a Psoríase, bem como a tantas outras doenças dermatológicas, requer um alto índice de suspeição para ser devidamente diagnosticada, especialmente em fases iniciais da doença.O tratamento inicial da PRP passa pela aplicação de terapias tópicas, como corticosteroides, emolientes, queratolíticos, retinóides tópicos e inibidores de calcineurina como o Pimecrolimus e o Tacrolimus. Como as terapias tópicas são normalmente insuficientes, em formas mais difusas da doença os retinóides sistémicos, como a isotretinoína, a alitretinoína e a acitretina surgem como terapêutica de 1ª linha. Se estiverem contraindicados, pelo alto risco de teratogenicidade ou pelo seu perfil de feitos adversos, ou se não se obtiver resposta terapêutica, as terapias sistémicas imunossupressoras como o metotrexato (MTX) e a ciclosporina surgem como alternativas de tratamento. A fototerapia, ao contrário do que acontece na psoríase, não é uma terapêutica consensual e eficaz, pelo que está reservada apenas para doentes que comprovadamente terão algo a beneficiar dela.Os biológicos constituem terapêuticas de última linha. Os antagonistas do Tumor Necrosis Factor Alpha (TNFα) foram, historicamente, os primeiros biológicos a ser usados na PRP, com resultados variáveis. Recentemente duas novas classes de agentes biológicos, os antagonistas da interleucina 12/23 e os da interleucina 17A, têm demonstrado resultados bastante promissores no tratamento de casos de PRP recalcitrante.
Pityriasis Rubra Pilaris (PRP) is a rare, chronic, papulosquamous dermatosis of unknown etiology. Its exact prevalence remains unknown, ranging from 1/5000 to 1/50000.The most widely accepted classification is Griffiths modified, which defines PRP in 6 different subtypes, according to their clinical manifestations, age of onset of symptoms and association with Human Immunodeficiency Virus (HIV) infection. PRP usually arises sporadically, but it can also arise as a family form.The pathogenesis of PRP may be eventually linked to gain-of-function mutations of the caspase recruitment domain family member, member 14 gene (CARD14). It also arises the possibility that PRP might be related to disorders in the metabolism and transport of Vitamin A to the skin through Retinol Binding Protein (RBP), with infections and with idiosyncratic reactions to drugs and ultraviolet (UV) radiation.PRP may be associated, albeit poorly understood, with autoimmune and neoplastic conditions, and may rarely manifest as a Paraneoplastic Syndrome.The clinical presentation of the PRP suggests its diagnosis, namely the presence of follicular hyperkeratotic papules, surrounded by whitish halo, plaques with cephalo-caudal progession, associated with the appearance of thickened and orange-hued scales. At dermoscopy, the appearance is very typical, with whitish keratotic plugs surrounded by yellow-orange halos, associated with vessels organized in clusters to the periphery. The histopathological analysis of skin biopsy is the gold standard for diagnosis, allowing us to find characteristics classically attributed to PRP: parakeratosis and orthokeratosis, irregular hyperkeratosis, epidermal acanthosis and follicular plugging. PRP is a condition that, given its low frequency in the population and its particular resemblance to a much more common dermatosis, psoriasis, as well as to many other dermatological diseases, requires a high index of suspicion to be properly diagnosed, especially in stages of the disease.Initial treatment of PRP involves the application of topical therapies such as corticosteroids, emollients, keratolytics, topical retinoids and calcineurin inhibitors such as Pimecrolimus and Tacrolimus. As topical therapies are usually insufficient, systemic retinoids such as isotretinoin, alitretinoin and acitretin appear as first-line therapy in more diffuse forms of the disease. If they are contraindicated because of the high risk of teratogenicity or their profile of adverse events, or if no therapeutic response is obtained, immunosuppressive therapies such as methotrexate (MTX) and cyclosporin appear as treatment alternatives. Phototherapy, on the contrary to what happens in psoriasis, is not a consensual and effective therapy, so it is reserved only for patients who are proven to have something to benefit from it.Biologicals constitute last-line therapies. Tumor Necrosis Factor Alpha (TNFα) antagonists were historically the first biological to be used in PRP, with variable results. Recently two new classes of biological agents, interleukin 12/23 antagonists and interleukin 17A antagonists have shown very promising results in the treatment of recalcitrant PRP cases.
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Pilawa, Sandra [Verfasser]. "Entwicklung der chemisch-technischen Grundlagen einer automatisierten Synthese von Molekülbibliotheken und des intelligenten Screenings von Leitstrukturen / von Sandra Pilawa." 2001. http://d-nb.info/961762683/34.

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Books on the topic "Pilawin"

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Lechowski, Leszek. Herb Pilawa i Pilawici z Łachowa. L. Lechowski, 1995.

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Mejía, Abigail. Sueña, Pilarín--: Novela. Instituto del Libro, 1992.

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Lübcke, Wolfgang. Die Wacholderdrossel (Turdus pilaris). A. Ziemsen, 1985.

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Borek, Piotr. Od Pilawiec do Humania [studia staropolskie]. Wydawnictwo Naukowe Akademii Pedagogicznej, 2005.

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Gucht, Karel de. Het einde der pilaren: Een Toscaans gesprek. Houtekiet, 2001.

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Agliati, Mario. I Problemi del Professor Pilati: Racconti. Edizioni del Cantonetto, 1987.

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Pilati, Giacomo. Le siciliane: Quindici storie vere / Giacomo Pilati. Coppola, 1998.

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Aḥmadiyān, ʻAbd Allāh. Gelḧo le ser sutuwan r̄ahatuwe: Tews u pilarî komełayetî. A. Aḥmadiyān, 1991.

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Armellini, Serenella. Libertà e organizzazione: Il riformismo di Carlantonio Pilati. Jaka book, 1991.

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Bhargava, Harsh, Kinnera Murthy, and Anu Khendry. BITS of success: Inspiring stories of BITS Pilani alumni. Universities Press (India), 2014.

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Book chapters on the topic "Pilawin"

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Vaccaro, Mario, Claudio Guarneri, Richard F. Ambinder, et al. "Keratosis Pilaris." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_3240.

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Plewig, Gerd, and Albert M. Kligman. "Keratosis Pilaris." In ACNE and ROSACEA. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-97234-8_67.

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Plewig, Gerd, and Albert M. Kligman. "Keratosis Pilaris." In ACNE and ROSACEA. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59715-2_73.

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Oranje, Arnold P., and Dirk Van Gysel. "Keratosis Pilaris." In Harper's Textbook of Pediatric Dermatology. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444345384.ch123.

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Bakker, Annemarie. "Keratosis pilaris." In Kleine kwalen in de huisartsenpraktijk. Bohn Stafleu van Loghum, 2018. http://dx.doi.org/10.1007/978-90-368-2251-0_43.

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Plewig, Gerd, and Albert M. Kligman. "Keratosis pilaris." In Akne und Rosazea. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-57960-8_52.

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Gloster, Hugh Morris, Lauren E. Gebauer, and Rachel L. Mistur. "Keratosis Pilaris." In Absolute Dermatology Review. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-03218-4_4.

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Petrov, Andrej, and Vesna Pljakoska. "Pityriasis Rubra Pilaris." In European Handbook of Dermatological Treatments. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-45139-7_77.

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Pacha, Omar, and Adelaide Hebert. "Keratosis Pilaris Atrophicans." In Acneiform Eruptions in Dermatology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8344-1_50.

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Chitgopeker, Pooja. "Pityriasis Rubra Pilaris." In Inpatient Dermatology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-18449-4_63.

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Conference papers on the topic "Pilawin"

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Srivastava, Harsh, and Anupam Singhal. "Design and Analysis of Optimized Water Distribution Network at BITS Pilani, Pilani Campus." In The 3rd World Congress on Civil, Structural, and Environmental Engineering. Avestia Publishing, 2018. http://dx.doi.org/10.11159/icesdp18.117.

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Utami, Hanif Sri, Benny Nelson, Eyleny Meisyah Fitri, Windy Keumala Budianti, and Endi Novianto. "Oral Vitamin A as an Adjuvant Treatment for Refractory Pityriasis Rubra Pilaris (PRP)." In The 2nd International Conference on Tropical Medicine and Infectious Disease. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009987102950298.

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"Effect of Biomass on Wind Reduction Pattern in BITS PILANI DUBAI CAMPUS." In International Conference on Biological, Civil and Environmental Engineering. International Institute of Chemical, Biological & Environmental Engineering, 2014. http://dx.doi.org/10.15242/iicbe.c0314170.

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Simpson, Angus R. "Comparing the Q-Equations and Todini-Pilati Formulation for Solving the Water Distribution System Equations." In 12th Annual Conference on Water Distribution Systems Analysis (WDSA). American Society of Civil Engineers, 2011. http://dx.doi.org/10.1061/41203(425)6.

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Bhattacharya, Basabdatta Sen, and Teresa Serrano-Gotarredona. "On- and Off-centre Pathways in a Retino-Geniculate Spiking Neural Network on SpiNNaker**This work is supported by the Science and Engineering Research Board of India (SERB) Core Research Grant CRG/2019/003534, BITS Pilani Institutional Research Grants GOA/ACG/2019-20/Oct/02 and BPGC/RIG/2018-19. TSG is supported by EU grant PCI2019-111826-2 “APROVIS3D”, by Spanish grant from the Ministry of Science and Innovation PID2019-105556GB-C31 “NANOMIND” (with support from the European Regional Development Fund) and by the CSIC 2018-50E008 AVE project." In 2021 10th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2021. http://dx.doi.org/10.1109/ner49283.2021.9441462.

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