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Relevant bibliographies by topics / Pin 1 synaptic transmission and remodelling

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Journal articles
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Academic literature on the topic 'Pin 1 synaptic transmission and remodelling'

Author: Grafiati

Published: 14 March 2023

Last updated: 31 July 2025

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Journal articles on the topic "Pin 1 synaptic transmission and remodelling"

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Zaitseva, Nina V., Olga Yu Ustinova, Marina A. Zemlyanova, et al. "Etiopathogenetic programs aimed at preventing in children risk-associated diseases caused by exposure to adverse components in emissions from a large non-ferrous metallurgic production." Hygiene and sanitation 104, no. 1 (2025): 84–95. https://doi.org/10.47470/0016-9900-2025-104-1-84-95.

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Introduction. Non-ferrous metallurgy holds a specific place among various productions that create potential health risks due to environmental pollution. Total emissions from the branch enterprises into ambient air reach three million tons (aluminum, fluorine, nickel, manganese, benzo(a)pyrene, sulfur dioxide and trioxide, hydrocarbon, etc.) per year, which accounts for 18% of the total emissions from all stationary sources. Implementation of etiopathogenetic prevention programs is a promising trend in preventing in children actual diseases associated with poor quality of the environment. The a

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Brancato, Anna, Valentina Castelli, Gianluca Lavanco, et al. "Social stress under binge-like alcohol withdrawal in adolescence: evidence of cannabidiol effect on maladaptive plasticity in rats." Psychological Medicine, September 6, 2022, 1–13. http://dx.doi.org/10.1017/s0033291722002744.

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Abstract Background Alcohol binge drinking may compromise the functioning of the nucleus accumbens (NAc), i.e. the neural hub for processing reward and aversive responses. Methods As socially stressful events pose particular challenges at developmental stages, this research applied the resident–intruder paradigm as a model of social stress, to highlight behavioural neuroendocrine and molecular maladaptive plasticity in rats at withdrawal from binge-like alcohol exposure in adolescence. In search of a rescue agent, cannabidiol (CBD) was selected due to its favourable effects on alcohol- and str

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Li, Minghua, Michelle Sorensen, Morgan A. Johnson, Susan L. Ingram, Michael C. Andresen, and Beth A. Habecker. "Hypertension increases sympathetic neuron activity by enhancing intraganglionic cholinergic collateral connections." Journal of Physiology, June 21, 2024. http://dx.doi.org/10.1113/jp286601.

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AbstractAutonomic dysregulation, including sympathetic hyperactivity, is a common feature of hypertension (HT) and other cardiovascular diseases. The CNS plays a role in driving chronic sympathetic activation in disease, but several lines of evidence suggest that neuroplasticity in the periphery may also contribute. The potential contribution of postganglionic sympathetic neurons to sustained sympathetic hyperactivity is not well understood. We recently discovered that noradrenergic sympathetic neurons in the stellate ganglion (SG) have excitatory cholinergic collateral connections to other ne

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Popek, Mariusz, Marta Obara-Michlewska, Łukasz Mateusz Szewczyk, et al. "Oedematic-atrophic astrocytes in hepatic encephalopathy." Acta Neuropathologica Communications 13, no. 1 (2025). https://doi.org/10.1186/s40478-025-02045-5.

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Abstract Hepatic encephalopathy (HE) following acute liver failure (ALF) is considered as a primary toxic astrocytopathy, but in-depth characterisation of astrocytic contribution to the pathogenesis of this disease is far from complete. Using transmission electron microscopy, confocal fluorescent microscopy, and 3D reconstruction, we found complex morphological alterations of cortical astrocytes in mice with azoxymethane-induced ALF and post-mortem cortical tissue of patients at grade IV of HE. In both mice and post-mortem human tissues astrocytic primary branches demonstrated the territory oc

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Huang, Li, Francesca Hardyman, Megan Edwards, and Elisa Galliano. "Deprivation-induced plasticity in the early central circuits of the rodent visual, auditory, and olfactory systems." eneuro, January 9, 2024, ENEURO.0435–23.2023. http://dx.doi.org/10.1523/eneuro.0435-23.2023.

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Activity-dependent neuronal plasticity is crucial for animals to adapt to dynamic sensory environments. Traditionally, it has been investigated using deprivation approaches in animal models primarily in sensory cortices. Nevertheless, emerging evidence emphasizes its significance in sensory organs and in sub-cortical regions where cranial nerves relay information to the brain. Additionally, critical questions started to arise. Do different sensory modalities share common cellular mechanisms for deprivation-induced plasticity at these central entry-points? Does the deprivation duration correlat

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Dissertations / Theses on the topic "Pin 1 synaptic transmission and remodelling"

1

Antonelli, Roberta. "The role of prolyl-isomerase PIN1 in GABAergic and glutamatergic synaptic transmission." Doctoral thesis, SISSA, 2015. http://hdl.handle.net/20.500.11767/4893.

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The correct functioning of the central nervous system relies on the rapid and efficient communication between neurons. This occurs at highly specialized functional points of contact called synapses. Synapses are extremely plastic in structure and function, strongly influenced by their own histories of impulse traffic and by signals from nearby cells. Synaptic contacts are fundamental to the development, homeostasis and remodeling of neural circuits. All these events are achieved through different mechanisms operating at both pre- and postsynaptic sites. At the level of the post synaptic densit

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