Relevant bibliographies by topics / Pituitary gland function

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Pituitary gland function'

Author: Grafiati
Published: 19 February 2023

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Journal articles on the topic "Pituitary gland function"

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Spencer, Thomas E., Andrew M. Kelleher, and Frank F. Bartol. "Development and Function of Uterine Glands in Domestic Animals." Annual Review of Animal Biosciences 7, no. 1 (February 15, 2019): 125–47. http://dx.doi.org/10.1146/annurev-animal-020518-115321.

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All mammalian uteri contain glands that synthesize or transport and secrete substances into the uterine lumen. Uterine gland development, or adenogenesis, is uniquely a postnatal event in sheep and pigs and involves differentiation of glandular epithelium from luminal epithelium, followed by invagination and coiling morphogenesis throughout the stroma. Intrinsic transcription factors and extrinsic factors from the ovary and pituitary as well as the mammary gland (lactocrine) regulate uterine adenogenesis. Recurrent pregnancy loss is observed in the ovine uterine gland knockout sheep, providing unequivocal evidence that glands and their products are essential for fertility. Uterine gland hyperplasia and hypertrophy during pregnancy are controlled by sequential actions of hormones from the ovary and/or pituitary as well as the placenta. Gland-derived histotroph is transported by placental areolae for fetal growth. Increased knowledge of uterine gland biology is expected to improve pregnancy outcomes, as well as the health and productivity of mothers and their offspring.
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Petruska, Janet M., Maria Adamo, Jeffrey McCartney, Ahamat Aboulmali, and Thomas J. Rosol. "Evaluation of Adrenal Cortical Function in Neonatal and Weanling Laboratory Beagle Dogs." Toxicologic Pathology 49, no. 5 (June 2, 2021): 1117–25. http://dx.doi.org/10.1177/01926233211009492.

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The most common target organ for toxicity in the endocrine system is the adrenal gland, and its function is dependent upon the hypothalamus and pituitary gland. Histopathologic examination of the adrenal glands and pituitary gland is routinely performed in toxicity studies. However, the function of the adrenal gland is not routinely assessed in toxicity studies. Assessment of adrenal cortical function may be necessary to determine whether a histopathologic finding in the adrenal cortex results in a functional effect in the test species. As juvenile toxicity studies are more commonly performed in support of pediatric indications for pharmaceuticals, it is important to establish historical control data for adrenal gland function. In this study, adrenal cortical function was assessed in control neonatal and weanling beagle dogs as part of an ongoing juvenile toxicology program. Measurements of serum adrenocorticotropic hormone (ACTH), cortisol prior to and following administration of exogenous ACTH, and aldosterone were conducted beginning at 2 weeks of age continuing through 26 weeks of age. Serum electrolyte concentrations were determined at 4, 13, and 26 weeks of age. Dogs as young as 2 weeks of age synthesize and secrete adrenal cortical hormones and exhibit a functional hypothalamic pituitary adrenal axis.
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Gogakos, Apostolos I., Tasos Gogakos, Marina Kita, and Zoe A. Efstathiadou. "Pituitary Dysfunction as a Cause of Cardiovascular Disease." Current Pharmaceutical Design 26, no. 43 (December 22, 2020): 5573–83. http://dx.doi.org/10.2174/1381612824999201105165351.

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The hypothalamic-pituitary axis is responsible for the neuroendocrine control of several organ systems. The anterior pituitary directly affects the functions of the thyroid gland, the adrenal glands, and gonads, and regulates growth and milk production. The posterior hypophysis, through nerve connections with the hypothalamic nuclei, releases vasopressin and oxytocin responsible for water balance and social bonding, sexual reproduction and childbirth, respectively. Pituitary gland hormonal excess or deficiency results in dysregulation of metabolic pathways and mechanisms that are important for the homeostasis of the organism and are associated with increased morbidity and mortality. Cardiovascular (CV) disorders are common in pituitary disease and have a significant impact on survival. Hormonal imbalance is associated with CV complications either through direct effects on the heart structure and function and vasculature or indirectly by altering the metabolic profile. Optimal endocrine control can prevent or reverse CV defects and preserve survival and quality of life. In this review, we discuss the effects of pituitary hormone excess and deficiency on the CV system. Specifically, we assess the impact of Somatotroph, Corticotroph, Gonadotroph, and Lactotroph anterior pituitary axes on the CV system. The effect of posterior pituitary function on the CV system is also explored.
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Youssef, Joyce, Rohan Sadera, Dushyant Mital, and Mohamed H. Ahmed. "HIV and the Pituitary Gland: Clinical and Biochemical Presentations." Journal of Laboratory Physicians 13, no. 01 (March 2021): 084–90. http://dx.doi.org/10.1055/s-0041-1723055.

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AbstractHuman immunodeficiency virus (HIV) can have profound impact on the function of the pituitary gland. We have performed an electronic literature search using the following database: PubMed, Medline, Scopus, and Google Scholar. These databases were searched using the keywords HIV, pituitary glands, cancer, pituitary apoplexy, and infertility. HIV can cause hypopituitarism and also can lead to diabetes insipidus. The impact can be slow and insidious, and diagnosis depends on high index of clinical suspicion. The effect on anterior pituitary gland can be associated with growth hormone deficiency, hypothyroidism, adrenal insufficiency, premature menopause, erectile dysfunction, and infertility. HIV can cause pituitary apoplexy, and this should be treated as an endocrine emergency. Importantly, HIV can be associated with pituitary lymphoma and pituitary cancer. Therefore, joined management between HIV physicians, clinical biochemists and endocrinologists may help in establishing pituitary dysfunction.
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López, B. Díaz, and L. Debeljuk. "Prenatal melatonin and its interaction with tachykinins in the hypothalamic - pituitary - gonadal axis." Reproduction, Fertility and Development 19, no. 3 (2007): 443. http://dx.doi.org/10.1071/rd06140.

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The pineal gland, through its hormone melatonin, influences the function of the hypothalamic–pituitary–gonadal axis. Tachykinins are bioactive peptides whose presence has been demonstrated in the pineal gland, hypothalamus, anterior pituitary gland and the gonads, in addition to other central and peripheral structures. Tachykinins have been demonstrated to influence the function of the hypothalamic–pituitary–gonadal axis, acting as paracrine factors at each of these levels. In the present review, we examine the available evidence supporting a role for melatonin in the regulation of reproductive functions, the possible role of tachykinins in pineal function and the possible interactions between melatonin and tachykinins in the hypothalamic–pituitary–gonadal axis. Evidence is presented showing that melatonin, given to pregnant rats, influences the developmental pattern of tachykinins in the hypothalamus and the anterior pituitary gland of the offspring during postnatal life. In the gonads, the effects of melatonin on the tachykinin developmental pattern were rather modest. In particular, in the present review, we have included a summary of our own work performed in the past few years on the effect of melatonin on tachykinin levels in the hypothalamic–pituitary–gonadal axis.
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Harary, Maya, Aislyn C. DiRisio, Hassan Y. Dawood, John Kim, Nayan Lamba, Charles H. Cho, Timothy R. Smith, Hasan A. Zaidi, and Edward R. Laws. "Endocrine function and gland volume after endoscopic transsphenoidal surgery for nonfunctional pituitary macroadenomas." Journal of Neurosurgery 131, no. 4 (October 2019): 1142–51. http://dx.doi.org/10.3171/2018.5.jns181054.

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OBJECTIVELoss of pituitary function due to nonfunctional pituitary adenoma (NFPA) may be due to compression of the pituitary gland. It has been proposed that the size of the gland and relative perioperative gland expansion may relate to recovery of pituitary function, but the extent of this is unclear. This study aims to assess temporal changes in hormonal function after transsphenoidal resection of NFPA and the relationship between gland reexpansion and endocrine recovery.METHODSPatients who underwent endoscopic transsphenoidal surgery by a single surgeon for resection of a nonfunctional macroadenoma were selected for inclusion. Patients with prior pituitary surgery or radiosurgery were excluded. Patient characteristics and endocrine function were extracted by chart review. Volumetric segmentation of the pre- and postoperative (≥ 6 months) pituitary gland was performed using preoperative and long-term postoperative MR images. The relationship between endocrine function over time and clinical attributes, including gland volume, were examined.RESULTSOne hundred sixty eligible patients were identified, of whom 47.5% were female; 56.9% of patients had anterior pituitary hormone deficits preoperatively. The median tumor diameter and gland volume preoperatively were 22.5 mm (interquartile range [IQR] 18.0–28.8 mm) and 0.18 cm3 (IQR 0.13–0.28 cm3), respectively. In 55% of patients, endocrine function normalized or improved in their affected axes by median last clinical follow-up of 24.4 months (IQR 3.2–51.2 months). Older age, male sex, and larger tumor size were associated with likelihood of endocrine recovery. Median time to recovery of any axis was 12.2 months (IQR 2.5–23.9 months); hypothyroidism was the slowest axis to recover. Although the gland significantly reexpanded from preoperatively (0.18 cm3, IQR 0.13–0.28 cm3) to postoperatively (0.33 cm3, IQR 0.23–0.48 cm3; p < 0.001), there was no consistent association with improved endocrine function.CONCLUSIONSRecovery of endocrine function can occur several months and even years after surgery, with more than 50% of patients showing improved or normalized function. Tumor size, and not gland volume, was associated with preserved or recovered endocrine function.
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Feigl, Günther Christian, Christine Maria Bonelli, Andrea Berghold, and Michael Mokry. "Effects of gamma knife radiosurgery of pituitary adenomas on pituitary function." Journal of Neurosurgery 97 (December 2002): 415–21. http://dx.doi.org/10.3171/jns.2002.97.supplement_5.0415.

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Object. The authors undertook a retrospective analysis of the incidence and time course of pituitary insufficiency following gamma knife radiosurgery (GKS) for pituitary adenomas. Methods. Pituitary adenomas in 92 patients were analyzed. There were 61 hormonally inactive tumors, 18 prolactinomas, and nine somatotropic and four adrenocorticotropic adenomas. The mean tumor volume was 3.8 cm3 (range 0.2–14.6 cm3). The mean prescription dose was 15 Gy. The mean prescription isodose was 50.7%. The mean follow-up time was 4.6 years (range 1.2–10 years). The following new or deteriorating insufficiencies that did not require treatment were recorded for the different pituitary axes: follicle-stimulating hormone (FSH)/luteinizing hormone (LH) 19 (20.6%), thyroid-stimulating hormone (TSH) 32 (34.8%), adenocorticotropic hormone (ACTH) 10 (10.9%), and growth hormone (GH) 26 (28.3%). For new insufficiencies or deterioration requiring replacement therapy, the figures were as follows: FSH/LH 20 (21.7%), TSH 22 (23.9%), ACTH eight (8.7%), and GH 12 (13%). Spot dosimetry was performed in 59 patients in the hypothalamic region, the pituitary gland, and pituitary stalk. The pituitary stalks in patients with deterioration of pituitary function received a statistically higher dosage of radiation, 7.7 ± 3.7 Gy compared with 5.5 ± 3 Gy (p = 0.03). Conclusions. The function of the residual normal pituitary gland is less affected following GKS of pituitary adenomas than after fractionated radiotherapy. Nonetheless, increased attention needs to be exercised to reduce the dose to the stalk and pituitary gland to minimize the incidence of these complications.
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Thody, A. J., and S. Shuster. "Control and function of sebaceous glands." Physiological Reviews 69, no. 2 (April 1, 1989): 383–416. http://dx.doi.org/10.1152/physrev.1989.69.2.383.

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This review describes the various types of sebaceous glands, their locations, and where possible their different functions. All sebaceous glands are similar in structure and secrete sebum by a holocrine process. However, the nature of this secretion and the regulation of the secretory process seem to differ among the various types of glands. Methods for measuring sebum secretion and assessing sebaceous gland activity are also described. The area of major interest during the last 20 years has undoubtedly been the mechanisms that control sebaceous gland function. Most studies have focused on the endocrine control and in particular on the role of androgens and pituitary hormones, although evidence suggests that nonendocrine factors may also be important. However, many questions remain and during the next few years attention will certainly be given to the role of retinoids and their mode of action in the treatment of acne.
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Van de Kar, LD, and MS Brownfield. "Serotonergic Neurons and Neuroendocrine Function." Physiology 8, no. 5 (October 1, 1993): 202–7. http://dx.doi.org/10.1152/physiologyonline.1993.8.5.202.

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The release of serotonin (5-HT) from nerve terminals in the hypothalamus increases secretion of adrenocorticotropic hormone and prolactin from the anterior pituitary, vasopressin and oxytocin from the posterior pituitary gland, and renin secretion from the kidneys. Activation of 5-HT1 and/or 5-HT2 receptors stimulates the secretion of these hormones.
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Francis, Karen, B. Mary Lewis, Peter N. Monk, and Jack Ham. "Complement C5a receptors in the pituitary gland: expression and function." Journal of Endocrinology 199, no. 3 (December 2008): 417–24. http://dx.doi.org/10.1677/joe-08-0110.

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Communication between the immune and endocrine system is important for the control of inflammation that is primarily mediated through the hypothalamic–pituitary–adrenal axis. The innate immune system rapidly responds to pathogens by releasing complement proteins that include the anaphylatoxins C3a and C5a. We previously reported the existence of C3a receptors in the anterior pituitary gland and now describe the presence of C5a receptors in the gland. C5a and its less active derivative (C5adR) can bind to its own receptor and to another receptor called C5L2. Using RT-PCR and immunocytochemistry, C5a receptors and C5L2 were demonstrated in the rat anterior pituitary gland and in several rodent anterior pituitary cell lines. Western blotting analysis showed that C5a stimulated the phosphorylation of MAPK and AKT but not p38; C5adR on the other hand, had no effect on any of the signal molecules investigated. The effects of C5a and C5adR on the secretion of the inflammatory molecule, macrophage migration inhibitory factor (MIF) were investigated by ELISA. Both compounds showed a dose-dependent inhibition of MIF release, 30–40% inhibition at around 35–70 nM agonist with IC50 values of around 20 nM. C5a and C5adR also stimulated ACTH secretion (up to 25%) from AtT-20DV16 cells. These data show that functional C5a receptors (C5a and C5L2) are present in the anterior pituitary gland and they may play a role in dampening down inflammation by inhibiting the release of MIF and stimulating the release of ACTH.
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Dissertations / Theses on the topic "Pituitary gland function"

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Stallings, Caitlin. "Forkhead Factors FOXO1 and FOXO3 in Pituitary Gland Development and Function." OpenSIUC, 2019. https://opensiuc.lib.siu.edu/dissertations/1763.

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The differentiation of growth hormone producing somatotropes in the pituitary gland is dependent upon signaling and transcription factors produced both within and outside the gland. In order to better understand this fundamental process, we have focused on investigating the contribution of forkhead factors FOXO1 and FOXO3. We sought to elucidate whether FOXO1 is sufficient to drive somatotrope differentiation in an over-expression mouse model, identify potential functional redundancy between closely related forkhead family members, and to specify the genetic targets of FOXO1 binding in the pituitary gland. Using a combination of mouse models and molecular techniques we have established a role for Foxo1 in early embryonic development, generated a somatotrope-specific FOXO1 binding enrichment library, revealed functional redundancy between FOXO1 and FOXO3 in the pituitary gland, and discovered a novel protein-protein interaction with YBX1. These results demonstrate FOXO factors are important for pituitary gland formation and function during both pre- and postnatal periods.
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Rosenberg, Lewis A. "CYSTIC FIBROSIS IN MICE ELICITS MULTIPLE CHANGES IN PITUITARY GLAND FUNCTION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=case1127498814.

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Deeb, Asma. "The regulation of somatotroph function by growth hormone releasing hormone and its receptor in vitro." Thesis, University of Newcastle upon Tyne, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246715.

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Nye, Elisabeth Jane. "Dynamic stimulation tests in the assessment of hypothalamic-pituitary-adrenal axis function in pituitary disease and obesity /." St. Lucia, Qld, 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17166.pdf.

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Kapali, Jyoti. "DELETION OF THE Foxo1 GENE IN MOUSE PITUITARY GLAND AND THE EFFECTS ON SOMATOTROPE DIFFERENTIATION AND FUNCTION." OpenSIUC, 2017. https://opensiuc.lib.siu.edu/dissertations/1485.

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Various mouse models have allowed for identification of several transcription factors that are necessary for pituitary development. Lesions in the transcription factor genes result in pituitary hormone deficiency. Hormone deficiencies occur in approximately one in 4000 live births. Pituitary hormone deficiency may occur due to loss of a single hormone causing isolated hormone deficiency or several hormones leading to combined pituitary hormone deficiency (CPHD). Defects in genes such as LHX3, LHX4, RPX, PROP1, and PIT1 are known to contribute to CPHD in humans. FOXO1 is a member of a large family of forkhead transcription factors. FOXO1 is expressed in various tissues where it functions to regulate metabolism, maintenance of cell differentiation, vascular development, cell cycle progression and apoptosis. Previous studies in our laboratory found that FOXO1 is expressed in different subsets of pituitary cells during embryonic pituitary development, with almost 50% of GH positive somatotropes also immunopositive for FOXO1. However, the roles of FOXO1 during pituitary development have not been extensively explored. Therefore, this research focuses on the contributions of FOXO1 to pituitary development and exploration of FOXO1 as a candidate gene for CPHD. In this study, a mouse model (Foxo1 cKO) is used wherein the Foxo1 gene has been deleted in the pituitary gland by Cre-LoxP recombination system. First, expression of several genes was examined that might be associated with loss of FOXO1 in the pituitary with an aim to place FOXO1 within the hierarchy of transcription factors critical for pituitary development. The early pituitary organizers, PITX2, PITX3, LHX3, LHX4, are not affected due to deletion of Foxo1 suggesting that FOXO1 is not critical for the initial induction of oral ectoderm to form Rathke’s pouch during the early stages of pituitary development. PIT1 marks the progenitors committed to becoming somatotropes, thyrotropes or lactotropes. No apparent difference in Pit1 mRNA level as well as PIT1 immunostaining between cKO and wildtype embryos suggests that FOXO1 does not affect the commitment of progenitors to cells of the PIT1 lineage. The most significant effects of Foxo1 deletion in the pituitary gland was observed in somatotrope differentiation. There was a drastically decreased mRNA level of Ghrhr, a marker of terminally differentiated somatotropes as well as reduced expression of Neurod4 in Foxo1 cKO embryos compared to wildtype littermates. NEUROD4 is downstream of FOXO1. Another study suggests that Neurod4 deletion in the pituitary gland affects maturation of somatotrope while preserving other cell types of anterior pituitary. NEUROD4 is essential for expression of Ghrhr during embryonic development as Neurod4 deletion results in fewer somatotropes and a complete lack of Ghrhr. Therefore, it can be implied that NEUROD4 may act as an intermediate in FOXO1 mediated terminal somatotrope differentiation and loss of FOXO1 in pituitary tissue is impeding somatotrope differentiation. We also assessed the functional consequences of loss of FOXO1 in postnatal mice. The delay in differentiation of somatotropes that was evident during embryonic development seems to have recovered by P10. Therefore, we suggest that FOXO1 is important for somatotrope differentiation embryonically. FOXO1 is important for somatotrope function postnatally also. Gh1 expression, GH pituitary content and serum IGF1 levels are significantly reduced at P21. However, the cKO mice do not exhibit any growth deficit indicating that FOXO1 is dispensable for postnatal somatotrope expansion and growth. Our results show that the embryonic somatotrope phenotype associated with deletion of Foxo1 does not result in any morphological changes in postnatal cKO mice. A gene expression profiling study was done to ascertain the changes in transcriptome of the embryonic pituitary lacking FOXO1. We identified Slc25a33 and Deptor as differentially expressed genes. SLC25A33 is involved with transport of pyrimidine nucleotides across mitochondrial membrane and such transport is essential for mitochondrial DNA and RNA metabolism. Studies involving overexpression of Slc25a33 in human cells have shown it enhances cell size and mitochondrial thymidine triphosphate level but decreases ROS. Its knockdown causes depletion of mitochondrial DNA, reduced oxidative phosphorylation, cell size, and mitochondrial UTP levels, and increased ROS levels. SLC25A33 essentially maintains mitochondrial function as it regulates mitochondrial DNA replication and the ratio of transcription of mitochondrial genes relative to nuclear genes. DEPTOR acts as an intermediate in BAF60c-induced AKT activation that results in a metabolic switch from oxidative phosphorylation to glycolysis in fast-twitch myofiber. Such a switch is considered to protect mice from diet induced insulin resistance and glucose intolerance in diabetic state. In differentiating cells, significant oxidative damage occurs, which can be attributed to higher mitochondrial activity. Embryonic stem cells undergoing differentiation exhibit increased mitochondrial activity associated with mitochondrial DNA replication to encode mitochondrial electron transport chain components. During osteogenic differentiation, there is a significant increase in expression of Slc25a33, with concomitant increase in mitochondrial oxygen consumption. Similar changes have been reported during spontaneous embryonic stem cell differentiation with increase in ROS production associated with differentiation. Identification of Slc25a33 and Deptor brings to our attention, a possible mechanism which might explain the delayed somatotrope differentiation phenotype in Foxo1 cKO pituitary. We hypothesize that loss of FOXO1 and resulting suppression of Slc25a33 and Deptor results in perturbation in mitochondrial replication and imbalances ROS homeostasis, which thereby affects mitochondrial function. This hinders the somatotrope’s ability to switch to more energetically demanding metabolic pathways that are essential during terminal differentiation. A metabolic switch may be the key to the terminal differentiation of mature somatotropes from their committed progenitor cells. Our findings thus provide new insights and opens avenues for future research to investigate mechanisms of somatotrope development.
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Yarney, Thaddeus A. "Sexual maturational changes in the pituitary and testes of ram lambs and predictability of adult reproductive function." Thesis, McGill University, 1985. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=72049.

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Spring-born ram lambs were used to examine: (1) sexual maturational changes in LH, FSH and prolactin (PRL) secretion, testicular gonadotropin receptors, and testicular size and function; (2) predictability of yearling ram reproductive function from juvenile testicular size and reproductive hormone measurements. Despite continuous increases in testis size, serum LH-profile characteristics became greatest between 2 and 4 months and declined thereafter. However, LH-peak frequency increased by about 2-fold between 6 and 7 months; this was associated with marked increases in testosterone (T) secretion and spermatogenic function. Mean FSH and PRL levels were maximum at 2 months and 3 to 5 months, respectively, and decreased thereafter. Increases in steroidogenic and spermatogenic function were due partly to increases in testicular content of LH and FSH receptors. Yearling ram testis size and spermatogenic function were predictable from testis size at 5 to 6 months, neonatal (50 days) secretion of LH and T, and pubertal (150 days) secretion of T. However, combinations of testicular size and reproductive hormone measurements provided greater predictive power.
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Hasan, Nouma [Verfasser], and Stephan [Akademischer Betreuer] Philipp. "Expression and function of TRPM3 proteins in the pituitary gland of the mouse / Nouma Hasan. Betreuer: Stephan Philipp." Saarbrücken : Saarländische Universitäts- und Landesbibliothek, 2015. http://d-nb.info/1065232624/34.

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Ivanova, Irena [Verfasser], Horst-Werner [Akademischer Betreuer] Korf, and Manfred [Akademischer Betreuer] Schubert-Zsilavecz. "Influence of endocannabinoids on the function of the pituitary gland with focus on folliculo-stellate, corticotroph, and lactotroph cell lines / Irena Ivanova. Gutachter: Horst-Werner Korf ; Manfred Schubert-Zsilavecz." Frankfurt am Main : Univ.-Bibliothek Frankfurt am Main, 2015. http://d-nb.info/107268697X/34.

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Ivanova, Irena Verfasser], Horst-Werner [Akademischer Betreuer] [Korf, and Manfred [Akademischer Betreuer] Schubert-Zsilavecz. "Influence of endocannabinoids on the function of the pituitary gland with focus on folliculo-stellate, corticotroph, and lactotroph cell lines / Irena Ivanova. Gutachter: Horst-Werner Korf ; Manfred Schubert-Zsilavecz." Frankfurt am Main : Univ.-Bibliothek Frankfurt am Main, 2015. http://d-nb.info/107268697X/34.

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Fung, Sai-kit, and 馮世傑. "Functional studies of pituitary activin/follistatin system in grass carp." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hdl.handle.net/10722/192776.

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Books on the topic "Pituitary gland function"

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Istvan, Berczi, ed. Pituitary function and immunity. Boca Raton, Fla: CRC Press, 1986.

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Fujio, Yoshimura, and Gorbman Aubrey 1914-, eds. Pars distalis of the pituitary gland: Structure, function, and regulation : proceedings of the First International Symposium on the Pituitary Gland, Tokyo, Japan, November 14-17, 1984. Amsterdam: Excerpta Medica, 1986.

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Regulation of pituitary function. Basel ; New York: Karger, 1985.

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Wimersma Greidanus, Tj. B. van., ed. and Lamberts, Steven W. J., ed., eds. Regulation of pituitary function. Basel: Karger, 1985.

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T. Van Wimersma Greidanus (Editor) and W. J. Lamberts (Editor), eds. Regulation of Pituitary Function (Frontiers of Hormone Research). S. Karger AG (Switzerland), 1985.

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Berczi, Istvan. Pituitary Function and Immunity. Taylor & Francis Group, 2019.

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Berczi, Istvan. Pituitary Function and Immunity. Taylor & Francis Group, 2019.

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Berczi, Istvan. Pituitary Function and Immunity. Taylor & Francis Group, 2019.

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Berczi, Istvan. Pituitary Function and Immunity. Taylor & Francis Group, 2020.

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Berczi, Istvan. Pituitary Function and Immunity. Taylor & Francis Group, 2019.

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Book chapters on the topic "Pituitary gland function"

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Villarreal, Jesús Z., Guillermo Elizondo, Rafael Real, José G. Gonzalez, and Homero Náñez. "Magnetic Resonance Imaging (MRI) of the Pituitary Gland in Sheehan’s Syndrome." In Cell Function and Disease, 445–55. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0813-3_39.

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Chester-Jones, I., P. M. Ingleton, and J. G. Phillips. "The Structure and Function of the Hypothalamus and Pituitary Gland." In Fundamentals of Comparative Vertebrate Endocrinology, 285–409. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4899-3617-2_9.

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Sullivan, David A., L. Alexandra Wickham, Eduardo M. Rocha, Robin S. Kelleher, Lilia Aikawa da Silveira, and Ikuko Toda. "Influence of Gender, Sex Steroid Hormones, and the Hypothalamic-Pituitary Axis on the Structure and Function of the Lacrimal Gland." In Lacrimal Gland, Tear Film, and Dry Eye Syndromes 2, 11–42. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5359-5_2.

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Baiocco, Annamaria, Claude E. Boujon, Gilberto E. Bestetti, and Giovanni L. Rossi. "Function and Morphology of the Rat Pituitary Gland, Combined Investigations by Means of an In Vitro Model." In Endocrine System, 42–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60996-1_3.

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Al-Suhaimi, Ebtesam A., and Firdos Alam Khan. "The Pituitary Gland: Functional Relationship with the Hypothalamus, Structure, and Physiology." In Emerging Concepts in Endocrine Structure and Functions, 73–131. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-9016-7_4.

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Liu, H. M., Y. W. Li, and Ch T. Su. "Perfusion enhancement of anterior pituitary gland with MRI: a functional study." In Proceedings of the XV Symposium Neuroradiologicum, 355–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79434-6_171.

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Capen, Charles C. "Functional and Pathologic Interrelationships of the Pituitary Gland and Hypothalamus in Animals." In Endocrine System, 3–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60996-1_1.

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Koch, Bernard, and Bernadette Lutz-Bucher. "Characterization, Regulation and Functional Activity of Specific Vasopressin Receptors in the Anterior Pituitary Gland." In Neuroendocrine Molecular Biology, 249–60. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5131-3_22.

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Nagy, Eva. "The Pituitary Gland Eva Nagy." In Pituitary Function and Immunity, 27–40. CRC Press, 2019. http://dx.doi.org/10.1201/9780429279737-2.

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Turner, Helen E., Richard Eastell, and Ashley Grossman. "Posterior pituitary gland." In Endocrinology (Oxford Desk Reference), 122–29. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199672837.003.0004.

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This chapter discusses disorders specific to the posterior pituitary gland. It includes diabetes insipidus, describing its epidemiology, pathophysiology, including vasopressin and renal water loss, aetiology, and common investigations such as water deprivation test, pituitary function tests, and MRI. The chapter continues with sections on hyponatraemia and syndrome of inappropriate anti-diuretic hormone secretion (often abbreviated as SIADH), describing the disorders clinical assessment, causes, and management. It also enumerates disorders of hypothalamic dysfunction, including craniopharyngiomas, ependymomas, sarcoidosis, and basilar meningitis. It lists neurological manifestations of these hypothalamic dysfunction including food intake-related, fluid intake-related, sleep-related, temperature-regulation-related, and periodicity-related syndromes and disorders.
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Conference papers on the topic "Pituitary gland function"

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Kenning, Tyler, Maria Peris-Celda, and Carlos Pinheiro-Neto. "Safety and Efficacy of a Side-Cutting Aspiration Device for the Resection of the Gland–Tumor Interface in Optimizing Surgical Treatment of Functional Pituitary Adenomas." In 29th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679824.

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Reports on the topic "Pituitary gland function"

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Ori, Naomi, and Sarah Hake. Similarities and differences in KNOX function. United States Department of Agriculture, March 2008. http://dx.doi.org/10.32747/2008.7696516.bard.

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Hypothalamic gonadotropinreleasing hormone (GnRH1) is the key hormone in the control of gametogenesis and gonadal growth in vertebrates. Developmentally, hypothalamic GnRHproducing neurons originate from the olfactory placode, migrate along olfactory axons into the forebrain, and continue to the preoptic area and hypothalamus where they function to stimulate gonadotropin secretion from the pituitary gland. An appropriate location of GnRH neurons within the hypothalamus is necessary for normal reproductive function in the adult; abnormal migration and targeting of GnRH neurons during embryogenesis results in hypogonadism and infertility. The developmental migration of GnRH neurons and axonal pathfinding in mammals are modulated by a plethora of factors, including receptors, secreted molecules, adhesion molecules, etc. Yet the exact mechanism that controls these developmental events is still unknown. We investigated these developmental events and the underlying mechanisms using a transgenic zebrafish model, Tg(gnrh1: EGFP), in which GnRH1 neurons and axons are fluorescently labeled. The role of factors that potentially affect the development of this system was investigated by testing the effect of their knockdown and mutation on the development of the GnRH1 system. In addition, their localization in relation to GnRH1 was described during development. These studies are expected to generate the scientific foundation that will lead to developing innovative technologies, based on the disruption of the early establishment of the GnRH system, for inducing sterility in farmed fish, which is highly desirable for economical and environmental reasons.
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Gothilf, Yoav, Yonathan Zohar, Susan Wray, and Hanna Rosenfeld. Inducing sterility in farmed fish by disrupting the development of the GnRH System. United States Department of Agriculture, October 2007. http://dx.doi.org/10.32747/2007.7696512.bard.

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Hypothalamic gonadotropinreleasing hormone (GnRH1) is the key hormone in the control of gametogenesis and gonadal growth in vertebrates. Developmentally, hypothalamic GnRHproducing neurons originate from the olfactory placode, migrate along olfactory axons into the forebrain, and continue to the preoptic area and hypothalamus where they function to stimulate gonadotropin secretion from the pituitary gland. An appropriate location of GnRH neurons within the hypothalamus is necessary for normal reproductive function in the adult; abnormal migration and targeting of GnRH neurons during embryogenesis results in hypogonadism and infertility. The developmental migration of GnRH neurons and axonal pathfinding in mammals are modulated by a plethora of factors, including receptors, secreted molecules, adhesion molecules, etc. Yet the exact mechanism that controls these developmental events is still unknown. We investigated these developmental events and the underlying mechanisms using a transgenic zebrafish model, Tg(gnrh1: EGFP), in which GnRH1 neurons and axons are fluorescently labeled. The role of factors that potentially affect the development of this system was investigated by testing the effect of their knockdown and mutation on the development of the GnRH1 system. In addition, their localization in relation to GnRH1 was described during development. These studies are expected to generate the scientific foundation that will lead to developing innovative technologies, based on the disruption of the early establishment of the GnRH system, for inducing sterility in farmed fish, which is highly desirable for economical and environmental reasons.
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