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Relevant bibliographies by topics / Placenta proteins

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Academic literature on the topic 'Placenta proteins'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Placenta proteins"

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Tang, Yunhui, Katie Groom, Larry Chamley, and Qi Chen. "Melatonin, a Potential Therapeutic Agent for Preeclampsia, Reduces the Extrusion of Toxic Extracellular Vesicles from Preeclamptic Placentae." Cells 10, no. 8 (2021): 1904. http://dx.doi.org/10.3390/cells10081904.

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Preeclampsia, characterised by maternal endothelial cell activation, is triggered by toxic factors, such as placental extracellular vesicles (EVs) from a dysfunctional placenta. The increased oxidative stress seen in the preeclamptic placenta links to endoplasmic reticulum (ER) stress. The ER regulates protein folding and trafficking. When the ER is stressed, proteins are misfolded, and misfolded proteins are toxic. Misfolded proteins can be exported from cells, via EVs which target to other cells where the misfolded proteins may also be toxic. Melatonin is a hormone and antioxidant produced b

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Wu, Zhimin, Guangling Hu, Yiyu Zhang, and Zheng Ao. "IGF2 May Enhance Placental Fatty Acid Metabolism by Regulating Expression of Fatty Acid Carriers in the Growth of Fetus and Placenta during Late Pregnancy in Pigs." Genes 14, no. 4 (2023): 872. http://dx.doi.org/10.3390/genes14040872.

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Fatty acids (FAs) are essential substances for the growth and development of the fetus and placenta. The growing fetus and placenta must obtain adequate FAs received from the maternal circulation and facilitated by various placental FA carriers, including FA transport proteins (FATPs), FA translocase (FAT/CD36), and cytoplasmic FA binding proteins (FABPs). Placental nutrition transport was regulated by imprinted genes H19 and insulin-like growth factor 2 (IGF2). Nevertheless, the relationship between the expression patterns of H19/IGF2 and placental fatty acid metabolism throughout pig pregnan

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Kulikov, I. A., K. A. Artemyeva, A. P. Aleksankin, et al. "Placental and peripheral blood changes in functional morphology and immunology in patients with placenta accreta spectrum." CLINICAL AND EXPERIMENTAL MORPHOLOGY 13, no. 3 (2024): 42–52. http://dx.doi.org/10.31088/cem2024.13.3.42-52.

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Introduction. Placenta accreta spectrum is an abnormal invasion of villous trophoblast into the myometrium and one of the most severe complications of pregnancy. We aimed to study morphofunctional, molecular, and immunological changes in the placenta and peripheral blood in various types of placenta accreta spectrum.Materials and methods. The study involved 45 pregnant women who underwent ultrasound examination at weeks 35–38. According to the ultrasound data, 15 women had placenta accreta and 15 women developed placenta increta. The comparison group included 15 pregnant women without placenta

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Szendzielorz, Ewelina, and Radoslaw Spiewak. "Placental Extracts, Proteins, and Hydrolyzed Proteins as Active Ingredients in Cosmetic Preparations for Hair Loss: A Systematic Review of Available Clinical Evidence." Applied Sciences 14, no. 22 (2024): 10301. http://dx.doi.org/10.3390/app142210301.

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Placentae and their derivatives have been used in both traditional and modern medicine, as well as in cosmetic sciences. Although hair loss is frequently mentioned among problems for which the placenta is supposed to be a remedy, the evidence seems rather scarce. The aim of this study was to highlight the clinical evidence for the efficacy of placenta products against baldness and hair loss. Methods: This systematic review was performed according to PRISMA and PICO guidelines. Database searches were conducted in PubMed, Google Scholar and Scopus. Results: Among the 2922 articles retrieved by t

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Avissar, N., C. Eisenmann, J. G. Breen, S. Horowitz, R. K. Miller, and H. J. Cohen. "Human placenta makes extracellular glutathione peroxidase and secretes it into maternal circulation." American Journal of Physiology-Endocrinology and Metabolism 267, no. 1 (1994): E68—E76. http://dx.doi.org/10.1152/ajpendo.1994.267.1.e68.

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Extracellular glutathione peroxidase (eGPX) is a selenoglycoprotein distinct from cellular glutathione peroxidase (cGPX). The cDNA for eGPX has recently been cloned from human placenta. To determine whether human placenta makes both cGPX and eGPX and secretes eGPX, we used specific immunoprecipitations of 75Se metabolically labeled proteins from full-term placental explants in culture and perfused placental lobules. Placental explants and metabolically active, dually perfused placental lobules synthesized and contained both cGPX and eGPX and secreted eGPX. Perfused tissue secreted eGPX into th

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Kim, H. R., K. Naruse, H. R. Lee, T. Wakayama, C. S. Park, and D. I. Jin. "51 PROTEOMICS ANALYSIS OF PLACENTOMEGALY IN CLONED MICE." Reproduction, Fertility and Development 19, no. 1 (2007): 143. http://dx.doi.org/10.1071/rdv19n1ab51.

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A variety of mammalian species have been cloned during the past few years. However, the success rate of somatic cell nuclear transfer in animals has been extremely low with many problems. Particularly, placentomegaly is a frequent finding in cloned mice and cattle (Wakayma et al. 1999 PNAS USA 96, 14 984–14 989; Niemann et al. 2000 Theriogenology 53, 21–34). To assess protein expression profile in the placentomagaly of cloned mice produced by nuclear transfer of embryonic stem cells, we have used global proteomics approach by 2-D gel electrophoresis and mass-spectrometry with the differential

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Weber, Milena, Ivona Baričević-Jones, Romana Masnikosa, Dejan Filimonović, Željko Miković, and Olgica Nedić. "Receptors and Binding Proteins for Insulin and Insulin-Like Growth Factors in the Placenta of Healthy Mothers and Mothers with Insulin-Dependent Diabetes Mellitus." Journal of Medical Biochemistry 28, no. 1 (2009): 30–35. http://dx.doi.org/10.2478/v10011-008-0030-3.

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Receptors and Binding Proteins for Insulin and Insulin-Like Growth Factors in the Placenta of Healthy Mothers and Mothers with Insulin-Dependent Diabetes Mellitus The IGF system of human placenta consists of insulin-like growth factors (IGF)-I and -II, their receptors (IGF-1R and IGF-2R), and binding proteins (IGFBP-1 to -6). Due to many structural and metabolic similarities with insulin, the IGF system cannot be examined separately from insulin and its receptor (IR). In this study gel filtration was used to detect solubilized membrane proteins of the placenta obtained from healthy mothers and

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Taher, Shèdy, Yamilette Borja, Lucía Cabanela, et al. "Cholecystokinin, gastrin, cholecystokinin/gastrin receptors, and bitter taste receptor TAS2R14: trophoblast expression and signaling." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 5 (2019): R628—R639. http://dx.doi.org/10.1152/ajpregu.00153.2018.

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We investigated expression of cholecystokinin (CCK) in humans and mice, and the bitter taste receptor TAS2R14 in the human placenta. Because CCK and gastrin activate the CCKBR receptor, we also explored placental gastrin expression. Finally, we investigated calcium signaling by CCK and TAS2R14. By RT-PCR, we found CCK/Cck and GAST/Gast mRNA expression in both normal human and mouse placentas, as well as in human trophoblast cell lines (TCL). Although both Cckar and – br mRNA were expressed in the mouse placenta, only CCKBR mRNA was detected in the human placenta and TCL. mRNA expression for TA

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Selvaratnam, Johanna, Haiyan Guan, James Koropatnick, and Kaiping Yang. "Metallothionein-I- and -II-deficient mice display increased susceptibility to cadmium-induced fetal growth restriction." American Journal of Physiology-Endocrinology and Metabolism 305, no. 6 (2013): E727—E735. http://dx.doi.org/10.1152/ajpendo.00157.2013.

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Maternal cadmium exposure induces fetal growth restriction (FGR), but the underlying mechanisms remain largely unknown. The placenta is the main organ known to protect the fetus from environmental toxins such as cadmium. In this study, we examine the role of the two key placental factors in cadmium-induced FGR. The first is placental enzyme 11β-hydroxysteroid dehydrogenase type 2 (11β-HSD2), which is known to protect the fetus from exposure to high cortisol levels and subsequently FGR, and the second the cadmium binding/sequestering proteins metallotheionein (MT)-I and -II. Using the MT-I/II −

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Kumar K. V., Anil, Kavitha S., and Sreekanth K. S. "Regulatory proteins in placental angiogenesis." Biomedicine 41, no. 4 (2021): 694–700. http://dx.doi.org/10.51248/.v41i4.944.

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The vasculature of the placenta plays a crucial role during the course of pregnancy in order to maintain the growing need of the fetus. Abnormal placental structure and function significantly increase the risk of stillbirth. Various growth factors and cytokines play an important role in the vasculogenesis and angiogenesis of placenta. These processes are stimulated by various pro-angiogenic factors. The activities of these factors are also stimulated by hypoxia. In some of the physiological phenomenon like ovulation, embryogenesis as well as in wound healing intense blood vessel growth can be

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Dissertations / Theses on the topic "Placenta proteins"

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Sealey, Amy Lynn. "Loss of the murine TATA-binding protein N terminus leads to placental labyrinth defects but not maternal adaptive immune responses." Thesis, Montana State University, 2007. http://etd.lib.montana.edu/etd/2007/sealey/SealeyA0507.pdf.

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Leavenworth, Jonathan Dean. "Investigations of cellular stress-responsive proteins and PGF gene expression in human trophoblast." OpenSIUC, 2009. https://opensiuc.lib.siu.edu/dissertations/296.

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Placenta growth factor (PGF) expression is downregulated in preeclampsia (PE), a leading cause of maternal morbidity and mortality. The pathophysiology of PE is thought to be manifested by a poorly perfused placenta hampered by hypoxic stress. Two stress-mediate angiogenic responses include post-transcriptional regulation of mRNA stability, and regulation of PML sequestering protein. We investigate whether these mechanisms occur in hypoxic stressed trophoblast and preeclamptic placenta. Methods: To determine transcript stability, PGF mRNA was measured in normal vs. stressed conditions, and the

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Monteiro, Janaína Munuera. "Imunolocalização das Heat Shock Proteins (HSPs) 60 e 70 na placenta bovina." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-27062006-105146/.

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As Heat Shock Proteins (HSPs) ou proteínas do choque térmico são encontradas em todas as células e são classificadas de acordo com seu peso molecular. Dentre elas encontram-se as de 27, 60, 70, 90 e 110 kDa, sendo as mais estudadas no contexto da reprodução as da família 60 e 70. Essas proteínas são ditas como chaperoninas, em razão do seu importante papel no dobramento e desdobramento de outras proteínas celulares sem alterar sua conformação final, e são expressas frente a qualquer tipo de estresse como calor, vírus, bactéria, hormônios, diferenciação celular, etc, e influenciam nas respostas

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Sterle, Jodi A. "Effect of recombinant porcine somatotropin (rpST) on placental and fetal growth in gilts /." free to MU campus, to others for purchase, 1998. http://wwwlib.umi.com/cr/mo/fullcit?p9901288.

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Campbell, Fiona M. "Long-chain fatty acid transport by the human placenta : the role of fatty acid-binding proteins." Thesis, University of Aberdeen, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363738.

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The placenta is thought to play a vital role in the transfer of essential fatty (EFA) and their long-chain polyunsaturated derivatives (LCPUFA) from mother to the fetus. There is a preferential accumulation of these fatty acids from maternal to fetal tissues. However, little was known about the manner in which these nutrients preferentially traversed the placenta. This study investigated part of this placental transport mechanism. The results from these investigations demonstrated that the preferential transport of LCPUFA to the fetal circulation may at least be partially mediated by a prefere

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Bebington, Catherine. "Ubiquitin and ubiquitin-like proteins in the uterus and placenta of the human and non-human primate." Thesis, University of Nottingham, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312211.

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White, Lloyd. "Characterisation of caspase- 14 in the human placenta : evidence for trophoblast-specific inhibition of differentiation by caspase- 14." University of Western Australia. School of Anatomy and Human Biology, 2009. http://theses.library.uwa.edu.au/adt-WU2009.0160.

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[Truncated abstract] The placenta forms a barrier regulating the transfer of gases, nutrients and wastes between the mother and the developing conceptus, and also produces hormones affecting both the fetus and the mother. This barrier is formed by the differentiation of the outer layer of the blastocyst- the trophoblast- to facilitate implantation and subsequent invasion of the uterus. The trophoblast consists of an underlying proliferative pool of cytotrophoblasts, which differentiate to replenish the overlying continuous, multi-nucleated syncytiotrophoblast that forms the barrier between the

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Anandavijayan, Sothinathan. "Purification and characterisation of serum retinol binding proteins and the transport of Vitamin A across the human placenta." Thesis, University of Aberdeen, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394651.

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Since retinol is insoluble in aqueous media, transport of vitamin A to epithelial tissues from its storage sites in the liver, is mediated by a specific plasma carrier protein called Retinol-Binding Protein (RBP). There have been controversial views expressed by different workers as to the mechanism of active transfer of retinol through the lipid bilayer of target cell membrane. The supply of Vitamin A and its derivatives from mother to foetus is of vital importance for fetal growth and development. Placenta plays an important role in the supply of these nutrients to the fetus. Since little is

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Basir, Ghazala Sikandar. "Fetoplacental circulation and the role of IGF-1 in placental remodelling by apoptosis and proliferation in diabetic pregnancies." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B30496457.

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Dinant, Soraya. "Rôle des protéines prion au cours de l'infection par le virus influenza A chez la souris." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASB072.

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Les infections respiratoires virales constituent un problème de santé public majeur. Les virus influenza A (VIA) sont des virus zoonotiques figurant parmi les agents étiologiques responsables de ces pathologies. Dans le but d'identifier des facteurs de restriction à même de lutter contre ces agents pathogènes, nous avons réalisé plusieurs analyses qui nous ont permis d'identifier les gènes de la famille prion comme facteur d'atténuation des symptômes grippaux en modèle souris.La famille Prion est composée des trois gènes : Prnp, Prnd et Sprn qui codent respectivement les protéines PrPC, Doppel

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Books on the topic "Placenta proteins"

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P, Bischof, and Klopper Arnold, eds. Proteins of the placenta: Biochemistry, biology, and clinical application. Karger, 1985.

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International Conference on Placenta (1990 Tokyo, Japan). Placenta: Basic research for clinical application. Edited by Soma H. Karger, 1991.

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Lise, Cédard, and Firth Anthony, eds. Placental signals: Autocrine and paracine control of pregnancy. University of Rochester Press, 1992.

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Matsuto, Mochizuki, and Hussa Robert O, eds. Placental protein hormones: Proceedings of the Satellite Symposium on Placental Protein Hormones, Kobe, Japan, 14-15 July 1988. Excerpta Medica, 1988.

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International Congress on Placental and Endometrial Proteins (6th 1987 Nagoya-shi, Japan). Placental and endometrial proteins: Basic and clinical aspects : proceedings of the 6th International Congress on Placental and Endometrial Proteins, Nagoya, Japan, 13 December 1987. VSP, 1988.

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(Editor), A. Klopper, and P. Bischof (Editor), eds. Proteins of the Placenta. S. Karger AG (Switzerland), 1985.

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Placental Signals Autocrine and Paracine Control of Pregnancy (Trophoblast Research). University of Rochester Press, 1993.

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Klopper, A., and T. Chard. Placental Proteins. Springer London, Limited, 2012.

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Placental Proteins. Springer, 2012.

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(Editor), Matsuto Mochizuki, and Robert Hussa (Editor), eds. Placental Protein Hormones (International Congress). Elsevier, 1988.

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Book chapters on the topic "Placenta proteins"

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Björkman, N. "COMPARATIVE STRUCTURAL AND FUNCTIONAL FEATURES OF THE MAMMALIAN PLACENTA." In Pregnancy Proteins in Animals, edited by Jann Hau. De Gruyter, 1986. http://dx.doi.org/10.1515/9783110858167-002.

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S. Raikar, R., Janet F. Bank, and S. G. Joshi. "PROGESTAGEN-ASSOCIATED ENDOMETRIAL PROTEIN: DETECTION AND BINDING IN IMMATURE HUMAN PLACENTA." In Pregnancy Proteins in Animals, edited by Jann Hau. De Gruyter, 1986. http://dx.doi.org/10.1515/9783110858167-015.

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Cole, Laurence A. "O-Glycosylation of Proteins in the Normal and Neoplastic Trophoblast." In Cellular Biology and Pharmacology of the Placenta. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4757-1936-9_10.

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Schneider, H., A. Malek, R. Duft, and N. Bersinger. "Evaluation of an In Vitro Dual Perfusion System for the Study of Placental Proteins: Energy Metabolism." In Placenta as a Model and a Source. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0823-2_4.

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Bersinger, Niklaus A., A. Malek, and H. Schneider. "De Novo Synthesis of Pregnancy-Specific and Pregnancy-Associated Proteins by the In Vitro Perfused Human Term Placenta." In Placenta as a Model and a Source. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0823-2_5.

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Murthi, Padma, and Lynda K. Harris. "Liposome-Encapsulated Anti-inflammatory Proteins for Targeted Delivery to the Placenta to Treat Fetal Growth Restriction." In Methods in Molecular Biology. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3495-0_14.

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Bährle-Rapp, Marina. "Placental Protein." In Springer Lexikon Kosmetik und Körperpflege. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_8032.

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Bährle-Rapp, Marina. "Animal Placental Protein." In Springer Lexikon Kosmetik und Körperpflege. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_651.

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Bährle-Rapp, Marina. "Hydrolyzed Placental Protein." In Springer Lexikon Kosmetik und Körperpflege. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_4991.

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Burch, Rebecca L. "Placental Proteins in Semen." In Encyclopedia of Evolutionary Psychological Science. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-16999-6_2009-1.

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Conference papers on the topic "Placenta proteins"

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Vieira, Enayle Soares, Cecília Mariana Gontijo, Emanuel Nicolas Dias Pereira, Lucas Teodoro Vieira Campos, and Douglas Roberto Guimarães Silva. "EFEITOS DE HEMODERIVADOS SINTÉTICOS NO TRATAMENTO DE HEMORRAGIA AGUDA." In Anais do I Congresso Brasileiro de Urgência e Emergência. Even3, 2025. https://doi.org/10.29327/1484146.1-2.

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A perda de sangue do sistema circulatório é, inicialmente, compensada fisiologicamente pelo organismo. Contudo, o prolongamento do quadro de hemorragia aguda pode levar ao choque hemorrágico grave, no qual o comprometimento da função cardíaca associado a arritmias letais culminam em um desfecho fatal de falência de órgãos. O tratamento atual de transfusão com hemoderivados naturais, como concentrado de hemácias (PRBCs), plasma fresco congelado (FFP) e plaquetas, possui a limitação de depender diretamente da disponibilidade nos bancos de sangue, os quais estão linearmente associados à doação de

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Ny, T., L. Hansson, and B. Åstedt. "ISOLATION OF cDNA FOR TYPE-2 PLASMINOGEN ACTIVATOR INHIBITOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642855.

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The placental type plasminogen activator inhibitor (PAI-2) has been purified from extracts of human placenta and from a histiocytic lymphoma cell line. It is mainly an uPA inhibitor but it also inhibits the two-chain form of tPA.In order to determine the factors regulating PAI-2 gene expression and thereby clarify the physiological role of PAI-2 we have undertaken the molecular cloning of PAI-2 cDNA. A λgt11 expression library prepared from placental mRNA, was screened, immunologically using a monoclonal antibody probe developed against PAI-2 purified from human placenta. When 1.7×105 recombin

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Bukowski, Michael, Brij Singh, James Roemmich, and Kate Larson. "Lipidomic analysis of TRPC1 Ca2+-permeable channel-knock out mouse demonstrates a vital role in placental tissue sphingolipid and triacylglycerol homeostasis under high-fat diet." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/tjdt4839.

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Placental function including oxygen delivery and nutrient transport are critical determinants of fetal growth, moderating the risks of obesity and metabolic diseases later in life. Previously, we demonstrated in a mouse model that parental diet and exercise play important roles in placental lipid content and inflammation. Transient receptor potential canonical channel 1 (TRPC1) is a Ca2+-permeable integral membrane protein. We have demonstrated that TRPC1 increases total body adiposity in mice by decreasing the efficacy of exercise to limit adipose accumulation under a high fat (HF) diet. Impo

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Fujikawa, K., T. Funakoshi, J. F. Tait, and R. L. Heimark. "PLACENTAL ANTICOAGULANT PROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643912.

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An anticoagulant protein was purified from the soluble fraction of human placenta by ammonium sulfate precipitation and column chromatography on DEAE-Sepharose, Sephadex G-75, and mono S (Pharmacia). Approximately 30 mg of the protein was purified from one placenta. The purified protein gave a single band on SDS polyacrylamide gel and had a molecular weight of 36,500. This protein inhibited both kaolin and thromboplastin induced clotting times of normal human plasma. It also inhibited the clotting time of platelet-rich plasma induced by factor Xa, but did not affect thrombin activity of fibrin

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Kruithof, E. KO, W. D. Schleuning, and F. Bachman. "PLASMINOGEN ACTIVATOR INHIBITOR BIOCHEMICAL AND CLINICAL ASPECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644764.

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Plasminogen activator (PAs) are enzymes that convert the zymogen plasminogen into the trypsin-like protease plasmin, which degrades extracellular matrix proteins and fibrin in the course of fibrinolysis, embryogenesis, tissue remodeling and in tumor metastasis. Plasminogen activator inhibitors (PAIs) are important modulators of PA activity. Several proteins have been identified which inhibit at fast rates urokinase (u-PA) and tissue-type PA (t-PA). In the order of inhibition rate constants these are: a) PAI-1, present in human plasma and platelet extracts and purified from human endothelial ce

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Fujikawa, K., T. Funakoshi, R. L. Heimark, and J. F. Tait. "HUMAN PLACENTAL ANTICOAGULANT PROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642949.

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Endothelium is important to maintain blood fluidity preventing coagulation. Glycosaminoglycan in the endothelial cell plasma membrane has been thought to prevent activation of blood coagulation. Heparin-like compound, which is a potent anticoagulant activity, has been localized on the surface of the cultured endothelial cells. Anticoagulant action associated with thrombomodulin, which is present in endothelial cells, is another mechanism to provide hemostatic nature of endothelial cells.We wondered whether any other intracellular protein(s) is involved in coagulation. We looked for such a prot

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JIAN, Mariana, Oleg SOLOMON, Andrei MOSTOVEI, et al. "Complexul ombilico-placentar – sursă de collagen pentru aplicații în chirurgia oro-maxilo-facială." In International Congress "Research – Innovation – Innovative Entrepreneurship". Ion Creangă Pedagogical State University, 2024. https://doi.org/10.46727/c.17-18-05-2024.p183-187.

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This paper is focused on the description of sources of collagen such as the umbilical cord, placenta and the types of collagen found in them. It is necessary to mention that, collagen is a fibrous structural protein that is found in abundance in all organs of the human body. In the composition of the bone is found type I collagen, which forms its organic phase and is widely used in the creation of grafts for the regeneration of soft and hard tissues. At the moment, the lack of autologous grafts for oral-maxillo-facial surgery determines the use of bone substitutes, which are a promising trend

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Sadler, J. Evan. "THE MOLECULAR BIOLOGY OF VON WILLEBRAND FACTOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643930.

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Human von Willebrand factor (vWF) is a plasma glycoprotein that is synthesized by endothelial cells and megakaryocytes, and perhaps by syncytiotrophoblast of placenta. The biosynthesis of vWF is very complex, involving proteolytic processing, glycosyla-tion, disulfide bond formation, and sulfation. Mature vWF consists of a single subunit of ∼ 250,000 daltons that is assembled into multimer ranging from dimers to species of over 10 million daltons. vWF performs its essential hemostatic function through several binding interactions, forming a bridge between specific receptors on the platelet sur

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Tanaka, H., N. Narahara, H. Sadakata, K. Andoh, N. Kobayashi, and T. Maekawa. "ANALYSIS OF LEUKEMIA PELT. TISSUE FACTOR BY WESTERN BLOTTING TECHNIQUE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643285.

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It has been reported that the tissue factor(TF) of monocytes and leukemic leukocytes is one of the trigger substances of disseminated intravascular coagulation(DIC) in leukemia patients. To assess the properties of TF of leukemia cells, their TF was analyzed by the method of Western blotting. Placenta TF was purified using Concanavalin-A affinity chromatography. Briefly, human placenta TF was extracted from placenta acetone powder using Triton X-100 extraction and purified by Concanavalin-A affinity chromatography and SDS-preparative PAGE. Hie final product of the purified placenta TF-apoprote

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Wiesel, M. L., R. Spaethe, J.-M. Freyssinet, et al. "DETECTION AND EFFECTS OF THROMBOMODULIN ACTIVITY IN CRUDE THROMBOPLASTIN PREPARATIONS FROM PLACENTA AND LUNG." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644300.

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The activation of protein C (PC) by thrombin requires the presence of an endothelial membrane cofactor, thrombomodulin (TM). Activated PC (APC) exerts its anticoagulant activity by degrading factors (F) Va and Villa in the presence of phospholipids and of a vitamin K-dependent cofactor, protein S. Tissue factor (TF) is the essential cofactor of factor Vll/VIIain the activation of factor X. TF is synthetized by several cell lines including endothelial cells. Using a specific TM assay, up to 0.85 units of TM activity could be detected in commercial thromboplastin (TP) preparations from human pla

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Reports on the topic "Placenta proteins"

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Splitter, Gary A., Menachem Banai, and Jerome S. Harms. Brucella second messenger coordinates stages of infection. United States Department of Agriculture, 2011. http://dx.doi.org/10.32747/2011.7699864.bard.

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Aim 1: To determine levels of this second messenger in: a) B. melitensiscyclic-dimericguanosinemonophosphate-regulating mutants (BMEI1448, BMEI1453, and BMEI1520), and b) B. melitensis16M (wild type) and mutant infections of macrophages and immune competent mice. (US lab primary) Aim 2: To determine proteomic differences between Brucelladeletion mutants BMEI1453 (high cyclic-dimericguanosinemonophosphate, chronic persistent state) and BMEI1520 (low cyclicdimericguanosinemonophosphate, acute virulent state) compared to wild type B. melitensisto identify the role of this second messenger in esta

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Bremel, Robert D., and Arieh Gertler. Effect of Bovine Placental Lactogen and other Placental Proteins on Growth and Differentiation of Cultured Bovine Mammary Cells. United States Department of Agriculture, 1986. http://dx.doi.org/10.32747/1986.7566755.bard.

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Bazer, Fuller W., Arieh Gertler, and Elisha Gootwine. Role of Placental Lactogen in Sheep. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574339.bard.

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Central problems in sheep and dairy cattle production are reproductive failure due to embryonic/fetal mortality and low birth weights, especially in prolific breeds, and reduced milk yields which adversely affect neonatal survival and economy of production. The sheep placenta expresses lactogenic (ovine placental lactogen, oPL) and somatogenic (ovine placental growth hormone, oGH) hormones. Our research has focused on the biological roles of oPL and oGH in function of the uterine endometrium during gestation and the mammary gland during pregnancy and lactation. Major conclusions were that: ( 1

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Tayyab, Muhammad, Shakeela Parveen, Ayesha Khan, Sarwar Jahan, and Mehwish Sultana. Identification, Genomic Characterization, and Phylogenetic Relationship of the Heat Shock Protein Beta-1 (HSPB1) in Placental Mammals Authors. ResearchHub Technologies, Inc., 2025. https://doi.org/10.55277/researchhub.r0ldewvg.1.

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Splitter, Gary, and Menachem Banai. Microarray Analysis of Brucella melitensis Pathogenesis. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7709884.bard.

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Original Objectives 1. To determine the Brucella genes that lead to chronic macrophage infection. 2. To identify Brucella genes that contribute to infection. 3. To confirm the importance of Brucella genes in macrophages and placental cells by mutational analysis. Background Brucella spp. is a Gram-negative facultative intracellular bacterium that infects ruminants causing abortion or birth of severely debilitated animals. Brucellosis continues in Israel, caused by B. melitensis despite an intensive eradication campaign. Problems with the Rev1 vaccine emphasize the need for a greater understand

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Baszler, Timothy, Igor Savitsky, Christopher Davies, Lauren Staska, and Varda Shkap. Identification of bovine Neospora caninum cytotoxic T-lymphocyte epitopes for development of peptide-based vaccine. United States Department of Agriculture, 2006. http://dx.doi.org/10.32747/2006.7695592.bard.

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The goal of the one-year feasibility study was to identify specific cytotoxic T-lymphocyte (CTL) epitopes to Neosporacaninum in the natural bovine host in order to make progress toward developing an effective peptide-based vaccine against bovine neosporosis. We tested the hypothesis that: N. caninum SRS2 peptides contain immunogenicCTLepitope clusters cross-presented by multiple bovine MHC-I and MHC-IIhaplotypes. The specific objectives were: (1) Map bovine CTLepitopes of N. caninum NcSRS-2 and identify consensus MHC-I and class-II binding motifs; and (2) Determine if subunit immunization with

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