Relevant bibliographies by topics / Plague Plague Plague Yersinia pestis

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Academic literature on the topic 'Plague Plague Plague Yersinia pestis'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Journal articles on the topic "Plague Plague Plague Yersinia pestis"

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Theilmann, John, and Frances Cate. "A Plague of Plagues: The Problem of Plague Diagnosis in Medieval England." Journal of Interdisciplinary History 37, no. 3 (2007): 371–93. http://dx.doi.org/10.1162/jinh.2007.37.3.371.

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Recent works by historians and biologists have called into doubt whether the great epidemic of 1348/49 in England was the plague. Examination of the biological evidence, however, shows their arguments to be faulty. The great epidemic of 1348/49 may have included other diseases, but it was clearly yersinia pestis.
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Daniszewski, Piotr. "Pestis (Yersinia pestis) - As Biological Weapons." International Letters of Social and Humanistic Sciences 9 (September 2013): 84–94. http://dx.doi.org/10.18052/www.scipress.com/ilshs.9.84.

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Yersinia pestis (formerly Pasteurella pestis) is a type of bacterium. It is believed to have been responsible for plagues of the early 1300s. More accurately, it is a Gram-negative rod-shaped coccobacillus. It is a facultative anaerobe that can infect humans and other animals. Human Y. pestis infection takes three main forms: pneumonic, septicemic, and bubonic plagues. All three forms are widely believed to have been responsible for a number of high-mortality epidemics throughout human history, including the Justinianic Plague of the sixth century and the Black Death that accounted for the death of at least one-third of the European population between 1347 and 1353. It has now been shown conclusively that these plagues originated in rodent populations in China. More recently, Y. pestis has gained attention as a possible biological warfare agent and the CDC has classified it as a category A pathogen requiring preparation for a possible terrorist attack. Every year, thousands of cases of plague are still reported to the World Health Organization, although, with proper treatment, the prognosis for victims is now much better. A five- to six-fold increase in cases occurred in Asia during the time of the Vietnam war, possibly due to the disruption of ecosystems and closer proximity between people and animals. Plague also has a detrimental effect on non-human mammals. In the United States of America, animals such as the black-tailed prairie dog and the endangered black-footed ferret are under threat from the disease.
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Titball, R. W., J. Hill, D. G. Lawton, and K. A. Brown. "Yersinia pestis and plague." Biochemical Society Transactions 31, no. 1 (2003): 104–7. http://dx.doi.org/10.1042/bst0310104.

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Yersinia pestis is the aetiological agent of plague, a disease of humans that has potentially devastating consequences. Evidence indicates that Y. pestis evolved from Yersinia pseudotuberculosis, an enteric pathogen that normally causes a relatively mild disease. Although Y. pestis is considered to be an obligate pathogen, the lifestyle of this organism is surprisingly complex. The bacteria are normally transmitted to humans from a flea vector, and Y. pestis has a number of mechanisms which allow survival in the flea. Initially, the bacteria have an intracellular lifestyle in the mammalian host, surviving in macrophages. Later, the bacteria adopt an extracellular lifestyle. These different interactions with different host cell types are regulated by a number of systems, which are not well characterized. The availability of the genome sequence for this pathogen should now allow a systematic dissection of these regulatory systems.
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Titball, Richard W., and E. Diane Williamson. "Yersinia pestis (plague) vaccines." Expert Opinion on Biological Therapy 4, no. 6 (2004): 965–73. http://dx.doi.org/10.1517/14712598.4.6.965.

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Sun, Wei, Kenneth L. Roland, and Roy Curtiss III. "Developing live vaccines against plague." Journal of Infection in Developing Countries 5, no. 09 (2011): 614–27. http://dx.doi.org/10.3855/jidc.2030.

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Three great plague pandemics caused by the gram-negative bacterium Yersinia pestis have killed nearly 200 million people and it has been linked to biowarfare in the past. Plague is endemic in many parts of the world. In addition, the risk of plague as a bioweapon has prompted increased research to develop plague vaccines against this disease. Injectable subunit vaccines are being developed in the United States and United Kingdom. However, the live attenuated Y. pestis-EV NIIEG strain has been used as a vaccine for more than 70 years in the former Soviet Union and in some parts of Asia and provides a high degree of efficacy against plague. This vaccine has not gained general acceptance because of safety concerns. In recent years, modern molecular biological techniques have been applied to Y. pestis to construct strains with specific defined mutations designed to create safe, immunogenic vaccines with potential for use in humans and as bait vaccines to reduce the load of Y. pestis in the environment. In addition, a number of live, vectored vaccines have been reported using attenuated viral vectors or attenuated Salmonella strains to deliver plague antigens. Here we summarize the progress of live attenuated vaccines against plagu
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Rollins, MD, Sarah E., Sean M. Rollins, PhD, and Edward T. Ryan, MD. "Yersinia pestis and the Plague." Pathology Patterns Reviews 119 (June 1, 2003): 78–85. http://dx.doi.org/10.1309/dqm93r8qnqwbfyu8.

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Bai, Guangchun, Andrey Golubov, Eric A. Smith, and Kathleen A. McDonough. "The Importance of the Small RNA Chaperone Hfq for Growth of Epidemic Yersinia pestis, but Not Yersinia pseudotuberculosis, with Implications for Plague Biology." Journal of Bacteriology 192, no. 16 (2010): 4239–45. http://dx.doi.org/10.1128/jb.00504-10.

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ABSTRACT Yersinia pestis, the etiologic agent of plague, has only recently evolved from Yersinia pseudotuberculosis. hfq deletion caused severe growth restriction at 37°C in Y. pestis but not in Y. pseudotuberculosis. Strains from all epidemic plague biovars were similarly affected, implicating Hfq, and likely small RNAs (sRNAs), in the unique biology of the plague bacillus.
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Drancourt, Michel, and Didier Raoult. "Genotyping Yersinia pestis in historical plague." Lancet Infectious Diseases 11, no. 12 (2011): 894–95. http://dx.doi.org/10.1016/s1473-3099(11)70292-4.

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Perry, R. D., and J. D. Fetherston. "Yersinia pestis--etiologic agent of plague." Clinical Microbiology Reviews 10, no. 1 (1997): 35–66. http://dx.doi.org/10.1128/cmr.10.1.35.

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Plague is a widespread zoonotic disease that is caused by Yersinia pestis and has had devastating effects on the human population throughout history. Disappearance of the disease is unlikely due to the wide range of mammalian hosts and their attendant fleas. The flea/rodent life cycle of Y. pestis, a gram-negative obligate pathogen, exposes it to very different environmental conditions and has resulted in some novel traits facilitating transmission and infection. Studies characterizing virulence determinants of Y. pestis have identified novel mechanisms for overcoming host defenses. Regulatory systems controlling the expression of some of these virulence factors have proven quite complex. These areas of research have provide new insights into the host-parasite relationship. This review will update our present understanding of the history, etiology, epidemiology, clinical aspects, and public health issues of plague.
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Perry, R. D., and J. D. Fetherston. "Yersinia pestis--etiologic agent of plague." Clinical microbiology reviews 10, no. 1 (1997): 35–66. http://dx.doi.org/10.1128/cmr.10.1.35-66.1997.

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Dissertations / Theses on the topic "Plague Plague Plague Yersinia pestis"

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Malek, Maliya Alia. "Plague in Maghreb." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5021/document.

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Yersinia pestis, agent causal de la peste, persiste dans la nature maintenu par un cycle enzootique dans des foyers conduisant à la réémergence de la maladie. En Afrique du Nord, où une réémergence a eu lieu après des années de ‘silence’, nous avons répertorié les différents épisodes ainsi que le nombre de cas en sur six pays à compter de 1940 en mettant en évidence l’importation de la maladie et un mode de contamination négligé, la transmission par voie orale. Une étude en Algérie sur 237 micromammifères confirme deux foyers et en revèle trois nouveaux porteurs d’un nouveau génotype (MST) de biotype Orientalis. Apodemus sylvaticus est par la même ajouté à la liste des rongeurs pestiférés. La projection des foyers de peste ainsi actualisés sur une carte géographique et écologique met en évidence la proximité des foyers de peste aux points d’eau saumâtre. Une étude statistique a confirmé une corrélation significative entre foyer de peste/eau salée à une proximité minimale &lt;3 km en comparaison à des zones d’eau douce. Des échantillons environnementaux salés ont permis l’isolement d’une souche Y. pestis Algeria 3. Cette découverte confortée par l’observation expérimentale de la résistance de Y. pestis à un milieu hyper salé à 150g/L NaCl se traduisant par un protéome spécifique en réponse à ce stress avec une forme d’adaptation de type forme L de la bactérie dans ce type d’environnement. Notre travail éclaire de façon originale un facteur méconnu de persistance tellurique de Y. pestis, conditionnant la réémergence de la peste dans des foyers séculaires au Maghreb contrairement aux rivages Nord de la Méditerranée où la peste autochtone a disparu depuis un siècle<br>Yersinia pestis, the causal agent of plague, persists in nature maintained by an enzootic cycle in foci leading to the re-emergence of the disease. In North Africa, where re-emergence took place after years of 'silence', we have listed the various episodes and the number of cases in six countries from 1940 onwards, highlighting the importation of the disease and A method of neglected contamination, oral transmission. A study in Algeria on 237 micromammals confirms two foci and reveals three new carriers of a new genotype (MST) of orientalis biotype. Apodemus sylvaticus is by the same added to the list of plague rodents. The projection of the plague foci thus updated on a geographical and ecological map highlights the proximity of plague foci to brackish water points. A statistical study confirmed a significant correlation between plague / salt water at a minimal proximity &lt;3 km compared to freshwater areas. Saline environmental samples allowed the isolation of a Y. pestis Algeria 3 strain. This discovery was confirmed by the experimental observation of the resistance of Y. pestis to a hyper-saline medium at 150 g / L NaCl resulting in a specific proteome In response to this stress with an adaptation form of form L of the bacterium in this type of environment. Our work illuminates in an original way an unknown factor of telluric persistence of Y. pestis, conditioning the re-emergence of the plague in secular centers in the Maghreb unlike the northern shores of the Mediterranean where the indigenous plague has disappeared for a century
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Isherwood, Karen Elizabeth. "Quorum sensing in Yersinia pestis." Thesis, University of Nottingham, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364667.

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Ayyadurai, Saravanan. "Specificity of the Yersinia Pestis biotype orientalis in the natural history of plague." Thesis, Aix-Marseille 2, 2010. http://www.theses.fr/2010AIX20674/document.

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Yesinia pestis est l'agent de la peste, maladie infectieuse spontanément mortelle, et une bactérie classée parmi les agents de bioterrorisme de groupe A [http://www.bt.cd.gov/agent/plague]. Les cas sporadiques ont été rapportés dans plusieurs pays d'Asie, d'Afrique, et d'Amérique et la peste reste endémique en Afrique (République Démocratique du Congo; Madagascar) qui déclare le plus grand nombre de cas annuels. La majorité de cas de peste chez les humains et les animaux sauvages se manifeste dans les régions délimitées géographiquement et appelées communément les foyers de la peste. Les mécanismes de la résistance de la peste dans le sol des foyers reste de nos jours un sujet de recherche alors que la peste est maintenant considérée comme une maladie re-émergente. Au cours de notre travail, nous avons développé un outil pour l'identification de Y. pestis par spectrométrie de masse MALDI-TOF MS. Cette méthode s'est avérée très simple et efficace pour l'identification au niveau des espèces, et constitue une méthode de première ligne d'identification. Nous avons ensuite montré que Y. pestis survivait et maintenait sa virulence pendant au moins neuf mois dans le sol stérilisé par la vapeur et humidifié, dépourvu d'éléments nutritifs ajoutés et d'invertébrés du sol. Afin de contribuer à l'étude de l'épidémiologie de la peste, nous avons démontré que seul le biovar Oriantalis est transmis dans un modèle animal par les poux d'homme (Pediculus humanus), les biovars Antiqua et Medievalis de Y. pestis n'étant pas transmissibles par les poux de corps. Le mécanisme impliqué dans la transmission de la peste par les poux de corps reste inconnu, ce qui voudrait dire que le mécanisme de l'adaptation de Y. pestis Orientalis à des nouveaux vecteurs qui sont corrélés aux circonstances de l'épidémie mortelle provoquée par la peste bubonique, reste aussi inconnu. Au cours d'un dernier travail, nous avons étudié des nouveaux composés pour la prophylaxie de la peste. Notamment, nous avons évalué le potentiel du lovastatine dans la prévention de la mortalité pendant la peste. Il a été démontré sur un modèle d'expérimentation avec les souris que la lovastatine réduisait considérablement le taux de mortalité associée à la peste. Toutes les données que nous avons rapportées dans ce rapport de thèse sont destinées à mieux comprendre le cycle épidémiologique de la peste<br>Yersinia pestis is the agent of deadly plague and a bacterium listed in the group A of potential bioterrorism agents [http://www.bt.cdc.gov/agent/plague/]. Sporadic cases are reported in several countries in Asia, Africa and America. Majority of human plague cases and enzootic animals occur in the geographical areas of so-called plague foci. The mechanisms sustaining geographical foci of plague remain poorly understood and plague been classified as a currently re-emerging disease. As first step, we established new front line tool for Y. pestis identification by using MALDI-TOF MS. This method was demonstrated to be simple and effective for Y. pestis identification at species level. Second step, we demonstrated that Y. pestis survived fully virulent for at least 9 months in a steam sterilized, humidified soil devoid of any nutritional supplements or any soil invertebrates. In third step we successfully demonstrated that the human louse (Pediculus humanus) as vector of plague and the body lice transmission of plague was restricted to Orientalis biovar; Antiqua and Medievalis biovars of Y. pestis were not able to transmit by body lice. This result shows that a un- explained mechanism is involved in the body lice transmission of plague and Y. pestis Orientalis adaptation to newly described vectors which effectively correlates the mass death caused by bubonic plague in Black Death individuals. Finally we conclude our study by exploring new compounds for the plague prophylaxis. The potential role of lovastatin in the prevention of mortality during plague was assessed. Lovastatin could significantly reduce the mortality associated with plague in an experimental mouse model. All These data herein we reported in our study may help to better understanding the epidemiology of plague
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Bubeck, Sarah S. "Mechanism by which Yersinia pestis blocks pro-inflammatory host responses : a dissertation /." San Antonio : UTHSC, 2007. http://proquest.umi.com/pqdweb?did=1394663471&sid=4&Fmt=2&clientId=70986&RQT=309&VName=PQD.

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Griffin, Kate Frances. "An investigation of the V antigen of Yersinia pestis as a potential vaccine antigen." Thesis, University of Newcastle Upon Tyne, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324934.

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Reddin, Karen Margaret. "Purification, immunogenicity and protective potency of the F1 antigen from Yersinia pestis." Thesis, Open University, 1998. http://oro.open.ac.uk/54548/.

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The area of self-esteem in people with learning disabilities has been largely neglected, and previous researchers have employed a variety of approaches. It is important to further our understanding in the context of providing appropriate clinical interventions and in monitoring the effect of social policy developments on the individuals at the receiving end of service provision. The study aimed to assess the reliability and validity of a set of measures devised specifically for use with learning disabled people, by Szivos-Bach (1993). The measures assess social comparisons, perception of stigma and aspirations and expectations. The study was carried out with 30 adults with mild and moderate learning disabilities between the ages of 18 and 65. The results provide initial support for the social comparisons test as a measure of self-esteem. Less evidence was found for the stigma questionnaire and the aspirations-expectations test. The results are discussed in the light of comparable research into self-esteem measures with non-learning disabled populations. Further research is required, and the most profitable way forward seems to be development of multi-dimensional measures of self-esteem.
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Chapman, David A. G. "The role of the immunoglobulin like periplasmic chaperone Caf1M in the export of the F1 capsular antigen of Yersinia pestis." Thesis, University of Reading, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312433.

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Spyrou, Maria Alexandra [Verfasser]. "The evolutionary history of plague as revealed through the analysis of ancient Yersinia pestis genomes / Maria Alexandra Spyrou." Tübingen : Universitätsbibliothek Tübingen, 2020. http://d-nb.info/1218073055/34.

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Palace, Samantha G. "Plague and the Defeat of Mammalian Innate Immunity: Systematic Genetic Analysis of Yersinia pestis Virulence Factors: A Dissertation." eScholarship@UMMS, 2016. http://escholarship.umassmed.edu/gsbs_diss/836.

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Yersinia pestis, the causative agent of plague, specializes in causing dense bacteremia following intradermal deposition of a small number of bacteria by the bite of an infected flea. This robust invasiveness requires the ability to evade containment by the innate immune system. Of the various mechanisms employed by Y. pestis to subvert the innate immune response and to proliferate rapidly in mammalian tissue, only a few are well-characterized. Here, I present two complementary genetic analyses of Y. pestis adaptations to the mammalian environment. In the first, genome-wide fitness profiling for Y. pestis by Tn-seq demonstrates that the bacterium has adapted to overcome limitation of diverse nutrients during mammalian infection. In the second, a series of combinatorial targeted mutations disentangles apparent functional redundancy among the effectors of the Y. pestis type III secretion system, and we report that YpkA, YopT, and YopJ contribute to virulence in mice. We have also begun to investigate a novel relationship between Y. pestis and mammalian platelets, a highly abundant cell type in plasma. I present evidence that Y. pestis has evolved specific mechanisms to interfere with platelet activation, likely in order to evade immune responses and promote maintenance of bacteremia by undermining platelet thrombotic and innate immune functions. The principles guiding this work – systematic genetic analysis of complex systems, coupled with rational modification of in vitro assays to more closely mimic the in vivo environment – are a generalizable approach for increasing the efficiency of discovering new virulence determinants in bacterial pathogens.
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Bilich, Richard Christian. "Climate Change and the Great Plague Pandemics of History: Causal Link between Global Climate Fluctuations and Yersinia Pestis Contagion?" ScholarWorks@UNO, 2007. http://scholarworks.uno.edu/td/632.

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The two great bubonic plague outbreaks of history, Justinian's Plague and the Black Death were responsible for the deaths of over one hundred million individuals across Eurasia and Africa. Both occurrences of the plague coincided with climatic shifts that are well documented by both literary and physical evidence. This thesis explores the possibility that both Justinian's Plague and the Black Death were precipitated by climatic shifts preceding their respective eras and that these changes also contributed to disappearance of each pandemic. A scientific analysis investigating the climatic changes including the anomalous weather of 535-536 A.D., the Medieval Warm Period, and the Little Ice Age are correlated with literary evidence recording the transmission and dormancy sequence of the plague. Although distinct differences exist between the origins of climate change in the periods preceding each plague, the effects of such changes clearly resulted in conditions ideal for the resulting pandemics.
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Books on the topic "Plague Plague Plague Yersinia pestis"

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Abbott, Rachel C. Plague. U.S. Department of the Interior, U.S. Geological Survey, 2012.

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Deville, Patrick. Plague and cholera. ABACUS, 2015.

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Horányi, Ildikó. Démoni ragály, a pestis: Kiállításvezető : Semmelweis Orvostörténeti Múzeum, 2007. október 30-2008. szeptember 3. Semmelweis Orvostörténeti Múzeum, Könyvtár és Levéltár, 2007.

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1956-, Magyar László András, and Semmelweis Orvostörténeti Múzeum, eds. Démoni ragály, a pestis: Kiállításvezető : Semmelweis Orvostörténeti Múzeum, 2007. október 30-2008. szeptember 3. Semmelweis Orvostörténeti Múzeum, Könyvtár és Levéltár, 2007.

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1863-1943, Yersin Alexandre, ed. Alexandre Yersin, un passe-muraille (1863-1943): Vainqueur de la peste et de la diphtérie, explorateur des hauts plateaux d'Indochine. Connaissances et savoirs, 2007.

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Deville, Patrick. Plague and Cholera. Little, Brown Book Group, 2014.

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Butler, Thomas. Plague and Other Yersinia Infections. Springer, 2012.

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United States. Indian Health Service. Navajo Area. Division of Environmental Health Services, ed. Plague: Dlóǫ́'̨ binaałniih. Navajo Area Indian Health Service, Division of Environmental Health Services, 2000.

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United States. Indian Health Service. Navajo Area. Division of Environmental Health Services., ed. Plague: Dlóǫ́'̨ binaałniih. Navajo Area Indian Health Service, Division of Environmental Health Services, 2000.

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Bartone, John C. Bioterrorism of Plague Yersinia Infections: Index of New Information and Guide-Book for Consumers, Reference and Research. Abbe Pub Assn of Washington Dc, 2001.

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Book chapters on the topic "Plague Plague Plague Yersinia pestis"

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Laudisoit, Anne, Werner Ruppitsch, Anna Stoeger, and Ariane Pietzka. "Yersinia pestis: Plague." In BSL3 and BSL4 Agents. Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch21.

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Laudisoit, Anne, Werner Ruppitsch, Anna Stoeger, and Ariane Pietzka. "Yersinia Pestis: Plague." In BSL3 and BSL4 Agents. Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch7.

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Murphy, Brian S., Susan C. Straley, Beth A. Garvy, and Christine R. Wulf. "Yersinia pestis YadC: A Novel Vaccine Candidate Against Plague." In Advances In Experimental Medicine And Biology. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72124-8_37.

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Dubyanskiy, Vladimir M., and Aidyn B. Yeszhanov. "Ecology of Yersinia pestis and the Epidemiology of Plague." In Advances in Experimental Medicine and Biology. Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-024-0890-4_5.

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Tao, Pan, Marthandan Mahalingam, and Venigalla B. Rao. "Highly Effective Soluble and Bacteriophage T4 Nanoparticle Plague Vaccines Against Yersinia pestis." In Vaccine Design. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3387-7_28.

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Hinnebusch, B. Joseph. "Transmission Factors: Yersinia pestis Genes Required to Infect the Flea Vector of Plague." In Advances in Experimental Medicine and Biology. Springer US, 2004. http://dx.doi.org/10.1007/0-306-48416-1_11.

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Hinnebusch, B. J., and D. L. Erickson. "Yersinia pestis Biofilm in the Flea Vector and Its Role in the Transmission of Plague." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-75418-3_11.

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Grosfeld, Haim, Tamar Bino, Yehuda Flashner, et al. "Vaccination with Plasmid DNA Expressing the Yersinia pestis Capsular Protein F1 Protects Mice Against Plague." In Advances in Experimental Medicine and Biology. Springer US, 2004. http://dx.doi.org/10.1007/0-306-48416-1_84.

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Anderson, Paul E., Rachel M. Olson, Joshua L. Willix, and Deborah M. Anderson. "Standardized Method for Aerosol Challenge of Rodents with Yersinia pestis for Modeling Primary Pneumonic Plague." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9541-7_3.

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Branger, Christine G., Roy Curtiss III, Robert D. Perry, and Jacqueline D. Fetherston. "Oral Vaccination with Different Antigens from Yersinia pestis KIM Delivered by Live Attenuated Salmonella Typhimurium Elicits a Protective Immune Response Against Plague." In Advances In Experimental Medicine And Biology. Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72124-8_36.

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Reports on the topic "Plague Plague Plague Yersinia pestis"

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Welkos, S., M. L. Pitt, M. Martinez, A. Friedlander, and P. Vogel. Determination of the Virulenec of the Pigmentation-Deficient and Pigmentation-/Plasminogen Activator-Deficient Strains of Yersinia pestis in Non-Human Primate and Mouse Models of Pneumonic Plague. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407896.

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