Relevant bibliographies by topics / Plasma membrane

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Plasma membrane'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 December 2024

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Journal articles on the topic "Plasma membrane"

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Ahmed, Sadiq, and Andre Kaplan. "Therapeutic Plasma Exchange Using Membrane Plasma Separation." Clinical Journal of the American Society of Nephrology 15, no. 9 (April 20, 2020): 1364–70. http://dx.doi.org/10.2215/cjn.12501019.

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Therapeutic plasma exchange is a blood purification technique designed for the removal of large molecular weight toxins such as pathogenic antibodies and lipoproteins. Plasma exchange can be performed either by membrane separation or centrifugation. Centrifugal plasma exchange is more common in the United States, while membrane separation is more popular in Germany and Japan. The membrane separation technique is similar to the ultrafiltration procedures performed with a standard dialysis machine but in which the membrane’s pores are large enough to allow removal of all circulating molecules while retaining the cellular components. The current availability of plasma separation membranes compatible with CRRT systems has dramatically increased the potential for almost all nephrologists to perform these treatments. This review describes the membrane separation techniques available in the United States, the practical aspects of ordering and operating a membrane separation plasma exchange procedure, and its possible complications.
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Bakouche, O., Y. Ichinose, R. Heicappell, I. J. Fidler, and L. B. Lachman. "Plasma membrane-associated tumor necrosis factor. A non-integral membrane protein possibly bound to its own receptor." Journal of Immunology 140, no. 4 (February 15, 1988): 1142–47. http://dx.doi.org/10.4049/jimmunol.140.4.1142.

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Abstract Purified plasma membranes from LPS-activated human blood monocytes produced significant lysis and growth inhibition of the TNF-sensitive L929 murine fibroblast cell line. Unactivated human monocyte plasma membranes did not display either activity. Anti-TNF serum specifically inhibited the anti-tumor activity of activated monocyte membranes whereas anti-IL-1 serum or non-specific rabbit serum decreased neither the lysis nor growth inhibition of L929 cells. Membrane-associated TNF did not behave as an integral protein as it could be eluted from the plasma membranes by either high salt or low pH treatment. Plasma membranes cleared of membrane-associated TNF by high salt treatment were able to bind TNF, and this binding was specifically inhibited by preincubation of rTNF with specific anti-TNF serum. Western blot analysis of plasma membranes showed a membrane-associated TNF with a m. w. of approximately 17 kDa present only in the activated monocytes. When the plasma membranes were preincubated with the cross-linker agent dissuccinimidyl suberate, Western blot analysis revealed the presence of a TNF-binding protein with a Mr of approximately 102 kDa. These studies indicate that unlike IL-1, membrane-associated TNF is not an integral membrane protein and that TNF may be associated with the monocyte membrane by occupying the TNF R.
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Matthes, Bernd, and Peter Böger. "Chloroacetamides Affect the Plasma Membrane." Zeitschrift für Naturforschung C 57, no. 9-10 (October 1, 2002): 843–52. http://dx.doi.org/10.1515/znc-2002-9-1015.

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In the present study membrane fatty acids were analyzed to find a link between the biosynthesis inhibition of very-long-chain fatty acids and the phytotoxic effects of herbicidal chloroacetamides. Accordingly,we have isolated membranes of cucumber seedlings (Cucumis sativus) by two-phase partitioning and analyzed their fatty acid content. Saturated VLCFAs ranging from C20 to C26 were found in high amounts (22%) in the plasma membrane fraction. Nonmodified VLCFAs were predominantly present in phospholipids, while saturated 2-hydroxylated VLCFAs were identified in cerebrosides. Treatment of intact seedlings with chloroacetamides markedly reduced the VLCFA content in the plasma membrane. This result could be specified by fatty-acid labeling using [14C]malonate as a substrate for fatty acid elongation. De novo incorporation of VLCFAs into the plasma membrane and into microsomal membranes, respectively, was severely impaired by chloroacetamides with I50 values between 10 to 100 nm. These results confirm the previous finding that chloroacetamides inhibit VLCFA biosynthesis localized in the microsomes (Böger et al., Pest Manage. Sci. 56, 497D508, 2000). The direct consequence of this inhibition is a strong decrease of VLCFAs required as constituents of the plasma membrane and the substitution by shorter acyl chains. Apparently, physical properties and function of the plasma membrane are affected eventually leading to death of the plant.
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Taylor, C. W., and O. Dellis. "Plasma membrane IP3 receptors." Biochemical Society Transactions 34, no. 5 (October 1, 2006): 910–12. http://dx.doi.org/10.1042/bst0340910.

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IP3Rs (inositol 1,4,5-trisphosphate receptors) are expressed in the membranes of non-mitochondrial organelles in most animal cells, but their presence and role within the plasma membrane are unclear. Whole-cell patch–clamp recording from DT40 cells expressing native or mutated IP3Rs has established that each cell expresses just two or three functional IP3Rs in its plasma membrane. Only approx. 50% of the Ca2+ entry evoked by stimulation of the B-cell receptor is mediated by store-operated Ca2+ entry, the remainder appears to be carried by the IP3Rs expressed in the plasma membrane. Ca2+ entering the cell via just two large-conductance IP3Rs is likely to have very different functional consequences from the comparable amount of Ca2+ that enters through the several thousand low-conductance store-operated channels.
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Beaulieu, Nadine, Bari Zahedi, Rebecca E. Goulding, Ghazaleh Tazmini, Kira V. Anthony, Stephanie L. Omeis, Danielle R. de Jong, and Robert J. Kay. "Regulation of RasGRP1 by B Cell Antigen Receptor Requires Cooperativity between Three Domains Controlling Translocation to the Plasma Membrane." Molecular Biology of the Cell 18, no. 8 (August 2007): 3156–68. http://dx.doi.org/10.1091/mbc.e06-10-0932.

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RasGRP1 is a Ras-activating exchange factor that is positively regulated by translocation to membranes. RasGRP1 contains a diacylglycerol-binding C1 domain, and it has been assumed that this domain is entirely responsible for RasGRP1 translocation. We found that the C1 domain can contribute to plasma membrane-targeted translocation of RasGRP1 induced by ligation of the B cell antigen receptor (BCR). However, this reflects cooperativity of the C1 domain with the previously unrecognized Plasma membrane Targeter (PT) domain, which is sufficient and essential for plasma membrane targeting of RasGRP1. The adjacent suppressor of PT (SuPT) domain attenuates the plasma membrane-targeting activity of the PT domain, thus preventing constitutive plasma membrane localization of RasGRP1. By binding to diacylglycerol generated by BCR-coupled phospholipase Cγ2, the C1 domain counteracts the SuPT domain and enables efficient RasGRP1 translocation to the plasma membrane. In fibroblasts, the PT domain is inactive as a plasma membrane targeter, and the C1 domain specifies constitutive targeting of RasGRP1 to internal membranes where it can be activated and trigger oncogenic transformation. Selective use of the C1, PT, and SuPT domains may contribute to the differential targeting of RasGRP1 to the plasma membrane versus internal membranes, which has been observed in lymphocytes and other cell types.
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Teiwes, Nikolas K., Ingo Mey, Phila C. Baumann, Lena Strieker, Ulla Unkelbach, and Claudia Steinem. "Pore-Spanning Plasma Membranes Derived from Giant Plasma Membrane Vesicles." ACS Applied Materials & Interfaces 13, no. 22 (May 27, 2021): 25805–12. http://dx.doi.org/10.1021/acsami.1c06404.

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Roth, J., M. J. Lentze, and E. G. Berger. "Immunocytochemical demonstration of ecto-galactosyltransferase in absorptive intestinal cells." Journal of Cell Biology 100, no. 1 (January 1, 1985): 118–25. http://dx.doi.org/10.1083/jcb.100.1.118.

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Galactosyltransferase immunoreactive sites were localized in human duodenal enterocytes by the protein A-gold technique on thin sections from low temperature Lowicryl K4M embedded biopsy specimens. Antigenic sites detected with affinity-purified, monospecific antibodies were found at the plasma membrane of absorptive enterocytes with the most intense labeling appearing along the brush border membrane. The lateral plasma membrane exhibited a lower degree of labeling at the level of the junctional complexes but the membrane interdigitations were intensely labeled. The labeling intensity decreased progressively towards the basal part of the enterocytes and reached the lowest degree along the basal plasma membrane. Quantitative evaluation of the distribution of gold-particle label proved its preferential orientation to the outer surface of the plasma membrane. In addition to this membrane-associated labeling, the glycocalyx extending from the microvillus tips was heavily labeled. Occasionally, cells without plasma membrane labeling were found adjacent to positive cells. The demonstration of ecto-galactosyltransferase on membranes other than Golgi membranes precludes its general use as a marker for Golgi membrane fractions. The possible function of galactosyltransferase on a luminal plasma membrane is unclear at present, but a role in adhesion appears possible on the basolateral plasma membrane.
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Ji, Zuohui, Yue Zhao, Min Zhang, Xiaopeng Li, and Heguo Li. "Surface Modification of ETFE Membrane and PTFE Membrane by Atmospheric DBD Plasma." Membranes 12, no. 5 (May 10, 2022): 510. http://dx.doi.org/10.3390/membranes12050510.

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Fluorine resin membranes with excellent chemical resistance have great potential for the application of high-performance chemical protective clothing. However, it is difficult to integrate fluorine resins into other materials such as fabrics due to their lower surface energy and poor bondability, making the fabrication of composite fabrics and the further seal splicing challenging. In this study, atmospheric pressure dielectric barrier discharge (DBD) plasma in helium (He) and helium/acrylic acid (He/AA) mixture atmospheres were used to modify two kinds of fluorine resins, ethylene tetrafluoroethylene (ETFE) and polytetrafluoroethylene (PTFE) membrane. The surface chemical properties, physical morphology, hydrophilicity and adhesion strength of the fluororesin membranes before and after plasma treatments were systematically analyzed. The results showed that the plasma treatment can modify the membrane surface at the nanoscale level without damaging the main body of the membrane. The hydrophilicity of the plasma-treated membrane was improved with the water contact angle decreasing from 95.83° to 49.9° for the ETFE membrane and from 109.9° to 67.8° for the PTFE membrane, respectively. The He plasma creates active sites on the membrane surface as well as etching the membrane surface, increasing the surface roughness. The He/AA plasma treatment introduces two types of polyacrylic acid (PAA)—deposited polyacrylic acid (d-PAA) and grafted polyacrylic acid (g-PAA)—on the membrane surface. Even after ultrasonic washing with acetone, g-PAA still existed stably and, as a result, improved the polarity and adhesion strength of fluororesin membranes. This work provides useful insights into the modification mechanism of DBD plasma on fluorine resins, with implications for developing effective strategies of integrating fluorine resin membrane to chemical protective clothing fabrics.
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Kwan, Chiu-Yin. "Aggregation of smooth muscle membranes and its use in the preparation of plasma membrane enriched fraction from gastric fundus smooth muscle." Biochemistry and Cell Biology 64, no. 6 (June 1, 1986): 535–42. http://dx.doi.org/10.1139/o86-075.

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Microsomal membranes isolated from rat gastric fundus smooth muscle by differential centrifugation aggregate substantially in the presence of the divalent metal ion Mg2+ or Ca2+. The magnitude of cation-induced membrane aggregation is higher for Ca2+ than for Mg2+, but the ion concentration required for half-maximum membrane aggregation (K0.5 value) is similar for Mg2+ and Ca2+. Cation-induced membrane aggregation is suppressed by high ionic strength and low pH of the medium. Cation-induced membrane aggregation of mitochondrial membrane and plasma membrane enriched fractions differ in the rate of aggregate formation, metal ion concentration dependence, and pH dependence. Such different properties of membrane aggregation were used to prepare a plasma membrane enriched fraction by conventional differential centrifugation. Subfractionation of the heterogenous microsomal membranes by free-flow electrophoresis indicated that smooth muscle plasma membranes showed a higher electrophoretic mobility than the intracellular membranes. These results suggest that ionic interactions on the cell membrane surfaces differ from those on the intracellular membrane surfaces and that induction of membrane aggregation by Ca2+ or Mg2+ is a useful procedure for an effective and rapid preparation of plasma membrane enriched fraction from smooth muscle.
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Block, E. R., J. M. Patel, and D. Edwards. "Mechanism of hypoxic injury to pulmonary artery endothelial cell plasma membranes." American Journal of Physiology-Cell Physiology 257, no. 2 (August 1, 1989): C223—C231. http://dx.doi.org/10.1152/ajpcell.1989.257.2.c223.

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We exposed monolayer cultures of pulmonary artery endothelial cells or plasma membranes derived from these cells to hypoxic (0 and 5% O2) and normoxic (20% O2; control) conditions and measured cellular contents of malondialdehyde and conjugated dienes, plasma membrane fluidity and lipid composition, and plasma membrane-dependent transport of 5-hydroxytryptamine (5-HT). Hypoxia caused significant increases in malondialdehyde and conjugated dienes, in fluidity, and in 5-HT transport. Hypoxia also caused a significant decrease in plasma membrane total phospholipids and a marked increase in plasma membrane free fatty acids that appeared to be due to release of fatty acids from the plasma membrane phospholipids. The increases in fluidity and 5-HT transport and the alterations in fatty acids were reversible after return to control conditions. These results indicate that hypoxia alters the physical state, lipid composition, and function of endothelial cell plasma membranes by a combination of stimulation of membrane lipid peroxidation and accelerated degradation of membrane phospholipids, the latter probably secondary to activation of membrane phospholipases.
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Dissertations / Theses on the topic "Plasma membrane"

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Terry, Matthew. "Purification of wheat leaf plasma membranes and characterization of plasma membrane ATPase activity and phytochrome binding." Thesis, University of Southampton, 1990. https://eprints.soton.ac.uk/411150/.

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Großmann, Guido. "Plasma membrane compartmentation in Saccharomyces cerevisiae." kostenfrei, 2008. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1152/.

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Crooks, Kim Chantelle. "Turnover of plant plasma membrane proteins." Thesis, Oxford Brookes University, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363720.

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Stanworth, Marie Helen. "Plasma membrane ATPase of Phytophthora cactorum." Thesis, University of the West of England, Bristol, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284886.

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Osman, Sangar Mahmoud. "Control of plasma membrane invagination systems." Thesis, University of Leicester, 2015. http://hdl.handle.net/2381/40507.

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The demarcation membrane system (DMS) is an extensive plasma membrane invagination system of the megakaryocyte (MK) that provides a membrane reserve for platelet generation. The properties of the DMS are poorly understood, particularly in living MKs. In this study, advanced electron microscopy, live cell confocal imaging and pharmacological tools were used to study the DMS and its connections with the extracellular environment. Confocal imaging of membrane-impermeant extracellular fluorescent indicators (HPTS, FITC-dextrans between 4 and 2000 kDa, and quantum dots) provided evidence for a discrete molecular cut-off (≈110kDa, estimated to be ≈11.0 nm by dynamic light scatter (DLS) measurements) for access to the DMS, which will prevent entry of large adhesion molecules (e.g. fibrinogen). Using extra-high resolution scanning electron microscopy (SEM) of fixed MKs, membrane invagination pores (MIPs) were observed on the MK surface that were variable in size but generally larger than predicted from live cell imaging experiments. A neck constriction was observed beneath the surface opening of the DMS using Gatan 3-view serial block face EM and may explain the rejection of molecules smaller than the surface pore. After inhibition of Cdc42, the DMS allowed entry of all indicators tested (up to 2000 kDa = reported to be ≈ 54.0 nm diameter). Using membrane-impermeant fluorescent indicators, electrophysiological capacitance measurements and transmission EM, multiple cationic amphiphilic drugs (CADs) were shown to cause complete surface detachment of the DMS. These compounds included the calmodulin inhibitor W-7, the phospholipase-C inhibitor U-73122 and anti-psychotic phenothiazines (trifluoperazine and chlorpromazine). CADs also caused loss of T tubules in cardiac ventricular myocytes and the open canalicular system of platelets. A possible explanation for this action is the ability of CADs to interfere with the interaction between PIP2 and binding of BAR domain containing proteins or the cytoskeleton. This work provides new insights into plasma membrane invagination systems.
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Kessler, Felix Ernst. "Isoforms of the plasma membrane Ca²⁺-ATPase /." Zürich, 1991. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=9630.

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Berchtold, Doris. "TOR complex 2 regulates plasma membrane homeostasis." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-146485.

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Ansa-Addo, Ephraim Abrokwa. "Plasma Membrane-derived Vesicles : Composition and Function." Thesis, London Metropolitan University, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.536717.

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Quiroga, Álvarez Xarxa. "Plasma membrane mechanosensing upon stretch-induced topography remodelling." Doctoral thesis, Universitat de Barcelona, 2021. http://hdl.handle.net/10803/672367.

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Five years ago, I started walking this path that now seems like an entire life. Although everyone around tried to explain how this would feel, none of their explanations could have match what it has been in the end. And this is exactly how living systems are, at all levels. The harder the scientists try to feed our curiosity taking closer looks to them, inspecting the question from a different angle, and despite all the previous knowledge that we could gather; the more they surprise us and reveal new ways of sensing, reacting and adapting to the environment. And I think this is exactly what drove me here. I wanted to understand how this is done. I wanted to “see” it. How is it possible that a cell “understands” what is going on around? Which are the parameters that they sense? Biochemistry alone does not answer the question. In a crowded environment, such as it is our body, cells are exposed to thousands of mechanical stimuli too. And those can be also harnessed and transformed into chemical responses as a way of signalling. While the classical biochemical inputs have long been studied, loads of questions remain open about how cells interpret those physical stimuli around them, and microscopy comes as a powerful technique to try to answer these queries. In that sense, this thesis represents a small approach in trying to unravel how the plasma membrane, the first boundary between the cell and the extracellular media, can receive mechanical stimuli and convert them into biochemical signals amenable for the cell. To try to answer this question, this work starts with an introduction to the structure and physicochemical characteristics of the plasma membrane. An overview of the cortical component of the cytoskeleton, intrinsically interconnected to this structure, is also provided. Next, a summary of the literature available on how the plasma membrane can perceive mechanical stimuli and which are the associated biochemical responses triggered by them is included as well. This part is based on a review article published by my colleague and co-supervisor Dr. Le Roux and myself at Philosophical Transactions B as part of the 2019 issue “Forces in cancer” [1]. After the introduction, chapter 2 describes the objectives of this study, which can be summarised as trying to unravel the way in which cells couple mechanical signals at their plasma membrane to biochemical cascades that mediate a response to those. Following, chapters 3 and 4 compose the main body of this thesis, including the methods and the experimental results coming from this research work. Both sections constitute a scientific article that has been recently submitted for publication. In chapter 3 the simplified model chosen to study the question of how cells sense and integrate mechanical stimuli at their plasma membrane is described. This consisted in submitting fibroblast to a controlled stretch-release cycle, forcing them to quickly adapt their shape, mimmicking a highly-relevant scenario in physiology. Chapter 4 recapitulates the way in which plasma membrane reacted to this mechanical perturbation. In the first place, the structure reacted by passively forming evaginations on the nanometric scale of homogeneous size and shape. These evaginations are next recognised by the I- BAR protein IRSp53, which subsequently organizes a node of actin polymerisation dependent on Rac1 and Arp2/3 that mediates the flattening of the structures. Absence of IRSp53 results, thus, in an impaired recovery of homeostasis after stretch. To reinforce the obtained experimental results, theoretical framework to model the mechanics of the system was generated in collaboration with the group of Dr. Arroyo at the Centre Internacional de Mètodes Numèrics en Enginyeria (CIMNE). The aim of this model was to describe how a network generated by the Arp2/3 complex, until now described to push, is able to generate in-plane forces that mediate the ironing of the evagination. Chapter 5 includes a discussion about the limitations of the technique, the novelty of the presented findings, the possible physiological scenarios where the described mechanochemical pathway can be of relevance and, finally, some exciting and unexplored questions that remained open after this work. Additionally, other scientific production obtained during this thesis consisting in unexplored results or work belonging to other publications, has been added at the end of this manuscript in four appendixes. On the first one, I describe all the efforts made during the first 1 year and a half of the PhD to improve immunostaining technique for plasma membrane bound proteins in order to try to stain endogenous BAR proteins. The second appendix gathers the findings obtained from the silencing assay of BAR candidates likely to recognise the curved shape of the stretch-release generated evaginations. The third appendix contains experimental results part of a different publication from Le Roux et al. now under review in Nature Communications [2]. Here, I studied the response of the N-BAR protein Amphiphysin after mechanical stimulation in cells. A fourth appendix including scanning electron microscopy representative images of stretch-release generated evaginations in other cell lines is also added. Finally, I included two more appendix containing the sequencing of all the IRSp53 plasmids used for the body of the work of this thesis and the MATLAB code used for analysis of evaginations flattening dynamics after stretch.
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Blixt, Ylva. "Early interaction between adenovirus type 2 and HeLa cells significance of the plasma membrane constitution /." Lund : Dept. of Microbiology, University of Lund, 1992. http://books.google.com/books?id=DzhrAAAAMAAJ.

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Books on the topic "Plasma membrane"

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Murphy, Angus S., Burkhard Schulz, and Wendy Peer, eds. The Plant Plasma Membrane. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-13431-9.

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Larsson, Christer, and Ian M. Møller, eds. The Plant Plasma Membrane. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74522-5.

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Crooks, Kim Chantelle. Turnover of plant plasma membrane proteins. Oxford: Oxford Brookes University, 1996.

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J, Garrahan Patricio, ed. The Ca2+ pump of plasma membranes. Boca Raton, Fla: CRC Press, 1986.

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Wilcock, Carol. The effects of nitrogen mustard on plasma membrane function. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1987.

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1945-, Larsson C., and Møller I. M. 1950-, eds. The plant plasma membrane: Structure, function and molecular biology. Berlin: Springer-Verlag, 1990.

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1945-, Larsson C., and Møller I. M. 1950-, eds. The Plant plasma membrane: Structure, function, and molecular biology. Berlin: Springer-Verlag, 1989.

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Crane, Frederick L., D. James Morré, and Hans Löw, eds. Plasma Membrane Oxidoreductases in Control of Animal and Plant Growth. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-8029-0.

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NATO Advanced Research Workshop on Plasma Membrane Oxidoreductases in Control of Animal and Plant Growth (1988 Córdoba, Spain). Plasma membrane oxidoreductases in control of animal and plant growth. New York: Plenum Press, 1988.

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Murphy, Christopher R. The plasma membrane of uterine epithelial cells: Structure and histochemistry. Stuttgart: G. Fischer, 1993.

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Book chapters on the topic "Plasma membrane"

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Bryjak, Marek, and Irena Gancarz. "Membrane Prepared via Plasma Modification." In Membranes for Membrane Reactors, 549–68. Chichester, UK: John Wiley & Sons, Ltd, 2011. http://dx.doi.org/10.1002/9780470977569.ch25.

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Thiriet, Marc. "Plasma Membrane." In Biomathematical and Biomechanical Modeling of the Circulatory and Ventilatory Systems, 383–484. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9758-6_8.

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Roualdes, Stephanie. "Plasma Membrane." In Encyclopedia of Membranes, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-40872-4_1227-4.

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Baker, Julien S., Fergal Grace, Lon Kilgore, David J. Smith, Stephen R. Norris, Andrew W. Gardner, Robert Ringseis, et al. "Plasma Membrane." In Encyclopedia of Exercise Medicine in Health and Disease, 711. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2879.

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Gooch, Jan W. "Plasma Membrane." In Encyclopedic Dictionary of Polymers, 915. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14513.

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Minami, Anzu, Daisuke Takahashi, Yukio Kawamura, and Matsuo Uemura. "Isolation of Plasma Membrane and Plasma Membrane Microdomains." In Isolation of Plant Organelles and Structures, 199–212. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6533-5_16.

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Pavelka, Margit, and Jürgen Roth. "The Plasma Membrane." In Functional Ultrastructure, 154–55. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_81.

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Palmgren, Michael G., Lone Bækgaard, Rosa Laura López-Marqués, and Anja Thoe Fuglsang. "Plasma Membrane ATPases." In The Plant Plasma Membrane, 177–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-13431-9_7.

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Alexandersson, Erik, Niklas Gustavsson, Katja Bernfur, Per Kjellbom, and Christer Larsson. "Plasma Membrane Proteomics." In Plant Proteomics, 186–206. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-72617-3_13.

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Roualdes, Stephanie. "Plasma Polymerized Membrane." In Encyclopedia of Membranes, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-40872-4_1229-4.

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Conference papers on the topic "Plasma membrane"

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Russo, Michael J., Simon H. Friedman, Jens O. M. Karlsson, and Mehmet Toner. "A Two-Compartment Membrane Limited Model of Molecular Transport Through Nano-Scale Pores With a Metal-Actuated Switch." In ASME 1997 International Mechanical Engineering Congress and Exposition, 9–14. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-1306.

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Abstract We have previously demonstrated that we can reversibly alter the transport selectivity of the plasma membrane to small molecules (∼1000 Da) by treating cells with H5, a genetic mutant of the pore-forming protein Staphylococcus aureus α-toxin, designed to be equipped with a metal-actuated switch. Toward the development of a plasma membrane permeabilization technique for both clinical and basic research applications, we have developed a simple model of molecular transport through the H5 pore. This model in combination with hindered transport models predicts the rate of transport of our marker molecules, carboxycalcein blue (CCB) and sucrose, and provides an approximation of the number of pores in the plasma membrane. Model predictions are also in reasonable agreement with experimental measurements of CCB and sucrose transport through the H5 treated plasma membranes of 3T3 fibroblasts. This model allows us to analyze quantitatively the selectivity of the H5 pore and by extension to use genetically designed pore forming proteins to advance the understanding of hindered transport in pores.
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Wang, T., J. Sheng, Z. H. Deng, L. P. Shi, S. L. Chen, Z. Q. Chen, and S. X. Rao. "Atmospheric Cold Plasma Assisted Preparation of Cellulose-based Janus Membrane for Biomedical Application." In 2024 IEEE International Conference on Plasma Science (ICOPS), 1. IEEE, 2024. http://dx.doi.org/10.1109/icops58192.2024.10625782.

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Guo, P., and R. Wang. "preparation of mof 808/Ag-based biochemical protection fiber membrane by plasma graft modification." In 2024 IEEE International Conference on Plasma Science (ICOPS), 1. IEEE, 2024. http://dx.doi.org/10.1109/icops58192.2024.10626061.

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Wu, Pei-Chen, Yen-Liang Chen, Chien-Min Lee, Shy-Jay Lin, Wen-Wei Wu, and Yen-Lin Huang. "Characteristics of CNT pellicle membrane under hydrogen plasma torture test via TEM and SEM images." In Photomask Technology 2024, edited by Lawrence S. Melvin and Seong-Sue Kim, 35. SPIE, 2024. http://dx.doi.org/10.1117/12.3034672.

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Merchant, Fatima A., and Mehmet Toner. "Spatial and Dynamic Characterization of the Interaction of Staphylococcus Aureus Alpha-Toxin With Cell Membranes." In ASME 1997 International Mechanical Engineering Congress and Exposition, 3–8. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-1305.

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Abstract Genetically engineered pore-forming proteins such as the H5 mutant of the staphylococcal aureus α-toxin, have been specially designed to achieve controlled and reversible plasma membrane permeabilization. Hence, quantitative information regarding the dynamics of poration is critical for designing applications employing α-toxins for the permeabilization of cell membranes. We have employed immunofluorescence imaging techniques in conjunction with viability assays to elucidate the spatial and temporal interactions of α-toxin with living cells. This information would aid in the design and development of better permeabilization protocols that effectively utilize the toxin to achieve efficient poration of a large number of cells.
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Faiz, Mehwish, Areej Ahmed, and Sumaya Abid. "Discriminating plasma membrane, internal membrane, and organelle membrane proteins by SVM." In 2021 4th International Conference on Computing & Information Sciences (ICCIS). IEEE, 2021. http://dx.doi.org/10.1109/iccis54243.2021.9676407.

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Burrage, P. M., K. Burrage, K. Kurowski, M. Lorenc, D. V. Nicolau, M. Swain, and M. A. Ragan. "A Parallel Plasma Membrane Simulation." In 2009 International Workshop on High Performance Computational Systems Biology (HiBi 2009). IEEE, 2009. http://dx.doi.org/10.1109/hibi.2009.18.

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Enouf, J., R. Breadux, N. Bourdeau, and S. Levy-Toledano. "EVIDENCE FOR TWO DIFFERENT Ca2+TRANSPORT SYSTEMS ASSOCIATED WITH PLASMA AND INTRACELLULAR HUMAN PLATELET MEMBRANES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644490.

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The regulation of Ca2+ concentration in different cells involves two Ca2+ pumps. The presence of such mechanisms in human platelets is still controverted. We then investigated this question by using plasma and intracellular membranes obtained after simultaneous isolation by centrifugation ca 40% sucrose from a mixed 100,000 g membrane fraction.The Ca2+ uptake by the different membrane vesicles has been studied. Both membrane fractions took up Ca2+ and the Ca2+ transport systems exhibited a high affinity towards Ca 2+.However, the two associated Ca2+ transport systems showed a different time course and exhibited different oxalate sensitivity. The plasma membranes are not permeable to potassium oxalate, while the Ca2+ uptake was stimulated by potassium oxalate in intracellular membranes.Two Ca2+ activated ATPase activities are associated with the isolated membrane fractions and appeared different for the following parameters : 1) a different time course of the two enzyme activities; 2)a similar apparent affinity towards Ca2+ (10−7 M), though inhibition of the Ca2+ ATPase activity was only observed in intracellular membranes at 10−6 M Ca2+ ; 3)a different pH dependence with a maximum at pH 7 for the enzyme of intracellular membranes and pH 8 for the enzyme of plasma membranes; 4)a 10 fold difference in the ATP requirement of the enzymes, thus the maximal response was obtained with 20 uM for the intracellular membrane enzyme and with 200 uM for the plasma membrane enzyme ; 5) a different affinity for various nucleotides as energy donors with a higher specificity of the plasma membrane enzyme towards ATP ; 6) a different vanadate inhibition-dose reponse which did not exceed 60% for the plasma enzyme while it reached 100% for the intracellular enzyme.Taken together, these studies agree with the possible role of both a plasma membrane and a dense tubular system Ca2+ -ATPases in the regulation of Ca2+ concentration in human platelets.
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Wolf, Erich W., Cedric F. Walker, and Charles F. Ide. "Survey for magnetically induced plasma membrane damage." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5760876.

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Wolf, Walker, and Ide. "Survey For Magnetically Induced Plasma Membrane Damage." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.589522.

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Reports on the topic "Plasma membrane"

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Steponkus, P. L. Effects of freezing and cold acclimation on the plasma membrane of isolated protoplasts. Office of Scientific and Technical Information (OSTI), January 1991. http://dx.doi.org/10.2172/7302592.

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Hussey, Stephen L., and Blake Peterson. Novel Synthetic Antiestrogens That Block Nuclear Estrogen Receptor Function Through Plasma Membrane Localization. Fort Belvoir, VA: Defense Technical Information Center, May 2003. http://dx.doi.org/10.21236/ada415782.

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Steponkus, P. L. Effects of freezing and cold acclimation on the plasma membrane of isolated protoplasts. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/6551768.

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Steponkus, P. Effects of freezing and cold acclimation on the plasma membrane of isolated protoplasts. Office of Scientific and Technical Information (OSTI), January 1990. http://dx.doi.org/10.2172/7190629.

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Delmer, Deborah P., and Prem S. Chourey. The Importance of the Enzyme Sucrose Synthase for Cell Wall Synthesis in Plants. United States Department of Agriculture, October 1994. http://dx.doi.org/10.32747/1994.7568771.bard.

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The goal of this work was to understand the role of the enzyme sucrose synthase (SuSy) in synthesis of cellulose and callose in plants. The work resulting from the this grant leads to a number of conclusions. SuSy clearly plays diverse roles in carbon metabolism. It can associate with the plasma membrane of cells undergoing rapid cellulose deposition, such as cotton fibers, developing maize endosperm, gravistimulated pulvini, and transfer cells of the cotton seed. It is also concentrated at sites of high callose deposition (tapetal cells; cell plates). When SuSy levels are lowered by mutation or by anti-sense technology, cell walls undergo degeneration (maize endosperm) and show reduced levels of cellulose (potato tubers). In sum, our evidence has very much strengthened the concept that SuSy does function in the plasma membrane to channel carbon from sucrose via UDP-glucose to glucan synthase complexes. Soluble SuSy also clearly plays a role in providing carbon for starch synthesis and respiration. Surprisingly, we found that the cotton seed is one unique case where SuSy apparently does not play a role in starch synthesis. Current evidence in sum suggests that no specific SuSy gene encodes the membrane-associated form, although in maize the SS 1 form of SuSy may be most important for cell wall synthesis in the early stages of endosperm development. Work is still in progress to determine what does control membrane localization - and the current evidence we have favors a role for Ca2+, and possibly also protein phosphorylation by differentially regulated protein kinases. Finally, we have discovered for the first time, a major new family of genes that encode the catalytic subunit of the cellulose synthase of plants - a result that has been widely cited and opens many new approaches for the study of this important plant function.
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Pencheva, Mina, Yvetta Koeva, Ilian Dimitrov, and Nina Atanassova. Angiotensin Converting Enzyme (ACE) in Seminal Plasma and Sperm Membrane as a Marker for Male Infertility. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, May 2020. http://dx.doi.org/10.7546/crabs.2020.05.05.

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Staiger, Christopher. Regulation of Cell Wall Assembly: Myosin and Exocyst Involvement in Cellulose Synthase Delivery to the Plasma Membrane. Office of Scientific and Technical Information (OSTI), January 2022. http://dx.doi.org/10.2172/1840725.

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VanHouten, Joshua N. Does Increased Expression of the Plasma Membrane Calcium-ATPase Isoform 2 Confer Resistance to Apoptosis on Breast Cancer Cells? Fort Belvoir, VA: Defense Technical Information Center, September 2008. http://dx.doi.org/10.21236/ada493700.

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Ben-Hayyim, Gozal, Frances DuPont, Robert Lundin, John Windle, and William Hurkman. The Role of the Plasma Membrane and Tonoplast in Salt-Tolerance: Characterization of the Atpases and Ion Transport Properties. United States Department of Agriculture, November 1987. http://dx.doi.org/10.32747/1987.7566871.bard.

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Steponkus, P. L. Effects of freezing and cold acclimation on the plasma membrane of isolated protoplasts. Progress report, May 16, 1992--January 9, 1993. Office of Scientific and Technical Information (OSTI), May 1993. http://dx.doi.org/10.2172/10148698.

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