Relevant bibliographies by topics / Plasmablastic lymphoma

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Plasmablastic lymphoma'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Plasmablastic lymphoma.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Plasmablastic lymphoma"

1

Dupin, Nicolas, Tim L. Diss, Paul Kellam, Micheline Tulliez, Ming-Qing Du, Didier Sicard, Robin A. Weiss, Peter G. Isaacson, and Chris Boshoff. "HHV-8 is associated with a plasmablastic variant of Castleman disease that is linked to HHV-8–positive plasmablastic lymphoma." Blood 95, no. 4 (February 15, 2000): 1406–12. http://dx.doi.org/10.1182/blood.v95.4.1406.004k26_1406_1412.

Full text
Abstract:
Castleman disease (CD) is a lymphoproliferative disorder of unknown etiology that is associated with the development of secondary tumors, including B-cell lymphoma. Human herpesvirus 8 (HHV-8) (Kaposi's sarcoma–associated herpesvirus) sequences have been described in some cases of multicentric Castleman disease (MCD). Using a monoclonal antibody against an HHV-8–latent nuclear antigen, we show that HHV-8 is specifically associated with a variant of MCD in which HHV-8–positive plasmablasts that show λ light-chain restriction localize in the mantle zone of B-cell follicles and coalesce to form microscopic lymphomas in some cases. Furthermore, we show that the frank plasmablastic lymphoma that develops in patients with this plasmablastic variant of MCD is also positive for HHV-8 and λ light chain. Plasmablastic lymphoma associated with MCD is a new disease entity associated with HHV-8 infection.
APA, Harvard, Vancouver, ISO, and other styles
2

Goedhals, Jacqueline, Leriska Haupt, and Deborah Jafta. "Chronic lymphocytic leukaemia with concomitant plasmablastic lymphoma." South African Journal of Oncology 1 (March 3, 2017): 3. http://dx.doi.org/10.4102/sajo.v1i0.8.

Full text
Abstract:
Plasmablastic lymphomas are high-grade lymphomas that usually occur in immune-suppressed patients. Occasionally, they can arise from an underlying haematolymphoid malignancy, termed ‘transformed plasmablastic lymphoma’. In this report, we describe a case of chronic lymphocytic leukaemia and plasmablastic lymphoma in an elderly HIV-negative patient.
APA, Harvard, Vancouver, ISO, and other styles
3

Fernández-Álvarez, Rubén, Juan-Manuel Sancho, and Josep-María Ribera. "Plasmablastic lymphoma." Medicina Clínica (English Edition) 147, no. 9 (November 2016): 399–404. http://dx.doi.org/10.1016/j.medcle.2016.11.027.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Han, Xiao, Minghui Duan, Lixing Hu, Daobin Zhou, and Wei Zhang. "Plasmablastic lymphoma." Medicine 96, no. 9 (March 2017): e5981. http://dx.doi.org/10.1097/md.0000000000005981.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

&NA;. "Plasmablastic lymphoma." Advances in Anatomic Pathology 4, no. 6 (November 1997): 373. http://dx.doi.org/10.1097/00125480-199711000-00006.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Ahn, Janice S., Ryan Okal, Jeffrey A. Vos, Matthew Smolkin, Abraham S. Kanate, and Flavia G. Rosado. "Plasmablastic lymphoma versus plasmablastic myeloma: an ongoing diagnostic dilemma." Journal of Clinical Pathology 70, no. 9 (March 1, 2017): 775–80. http://dx.doi.org/10.1136/jclinpath-2016-204294.

Full text
Abstract:
AimsTo determine the utility of clinical, morphological and phenotypical features in the differential diagnosis of plasmablastic lymphoma and myeloma with plasmablastic features.MethodsAll plasmablastic neoplasms identified from a 15-year retrospective search were reviewed and classified into ‘lymphoma’, ‘myeloma’ or ‘indeterminate’. The classification was then compared with the previously established clinical diagnosis. Lessons learned from this review were used to design a diagnostic algorithm for pathologists to use in the absence of known clinical history.ResultsThe classification was possible in 10 of 11 cases, 8 lymphomas and 2 myelomas (n=2). No distinctive morphological or phenotypical features were identified. The most useful histopathological parameter was a positive Epstein-Barr virus in situ hybridisation. Presence of associated lymphadenopathy and/or oral mass in the absence of complete myeloma-defining signs was used to favour a diagnosis of lymphoma in 4 of 8 cases.ConclusionsThe distinction between plasmablastic lymphoma from plasmablastic myeloma warrants detailed knowledge of clinical, radiological and laboratorial findings. New studies identifying distinctive phenotypical or genetic features are needed to improve the histopathological differentiation of plasmablastic neoplasms.
APA, Harvard, Vancouver, ISO, and other styles
7

Bhardwaj, Parul, and Shivbrat Sharma. "A Case of Isolated Plasmacytoid Lymphoma Presenting as Hard Palate Mass: A Case Report." International Journal of Science and Healthcare Research 6, no. 2 (June 29, 2021): 178–80. http://dx.doi.org/10.52403/ijshr.20210432.

Full text
Abstract:
Plasmablastic lymphoma (PBL) is classified under the category of 'Mature B-cell neoplasms'. Plasmablastic lymphomas are the most common and account for 30% of all the cases type. PBL is an uncommon B-cell tumor which is limited to the jaw and oral cavity at presentation but can spread to distant sites at later stages type. Here we case report a 35 year old male presented to OPD as hard palate mass which turns to plasmablastic lymphoma after investigation. Keywords: Plasmablastic Lymphoma (PBL), Triglycerides (TG), Low density lipoproteins (LDL).
APA, Harvard, Vancouver, ISO, and other styles
8

Lin, Yuan, Gilberto D. Rodrigues, John F. Turner, and Mohammad A. Vasef. "Plasmablastic Lymphoma of the Lung." Archives of Pathology & Laboratory Medicine 125, no. 2 (February 1, 2001): 282–85. http://dx.doi.org/10.5858/2001-125-0282-plotl.

Full text
Abstract:
Abstract Non-Hodgkin lymphomas associated with acquired immunodeficiency syndrome are heterogeneous. Recently, a novel subtype of non-Hodgkin lymphoma occurring mostly in patients with acquired immunodeficiency syndrome has been described and designated as plasmablastic lymphoma. The histomorphologic and immunophenotypic findings of this distinct subtype of non-Hodgkin lymphoma have been characterized previously. Most patients present with oral cavity involvement. We report a case of plasmablastic lymphoma presenting as a lung tumor. To our knowledge, this is the first case report of this unusual subtype of diffuse large B-cell lymphoma in this location.
APA, Harvard, Vancouver, ISO, and other styles
9

Vaubell, Jalaludin I., Yetish Sing, Amsha Ramburan, Vikash Sewram, Rajendra Thejpal, Nadine Rapiti, and Pratistadevi K. Ramdial. "Pediatric Plasmablastic Lymphoma." International Journal of Surgical Pathology 22, no. 7 (April 25, 2014): 607–16. http://dx.doi.org/10.1177/1066896914531815.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Bloch, Edward, and Fiona Robinson. "Orbital plasmablastic lymphoma." Clinical Case Reports 6, no. 1 (November 24, 2017): 222–23. http://dx.doi.org/10.1002/ccr3.1281.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Plasmablastic lymphoma"

1

Elamin, Hassan Elzain. "Incidence of plasmablastic lymphoma in HIV positive and negative patients at a tertiary hospital in South Africa (2005-2017)." University of the Western Cape, 2018. http://hdl.handle.net/11394/6627.

Full text
Abstract:
Magister Scientiae Dentium - MSc(Dent)
The aim of the study was to investigate and describe the incidence of Plasmablastic Lymphoma (PBL) diagnosed at the Divisions of Anatomical Pathology and Haematopathology at Tygerberg Hospital from 2005 to 2017, and to ascertain a possible correlation with HIV infection, by identifying the number of HIV positive and negative patients diagnosed with Plasmablastic Lymphoma. Method: This was a retrospective study using the case records of all newly diagnosed PBL patients from 2005 to 2017. Results: Fifty-seven cases of PBL were diagnosed from 2005-2017. The overall result shows an increasing incidence of PBL in the intended period with the maximum incidence occurring in 2017. Most of the cases, 40.4%, were diagnosed in the age range 40-49-years. Forty-five patients were HIV-positive (78.9%) with (P value 0.011) and the majority of the patients were males (66.7%).
APA, Harvard, Vancouver, ISO, and other styles
2

Chiyapo, Sebathu Phillip. "Retrospective study of patients treated for Plasmablastic Lymphoma at Groote Schuur Hospital between 2004 and 2009." Master's thesis, University of Cape Town, 2014. http://hdl.handle.net/11427/5932.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Kriel, Raymond Frank. "An investigation of Epstein-Barr Virus (EBV) latency type and MYC gene aberrations in plasmablastic lymphoma diagnosed at Groote Schuur Hospital, Cape Town, South Africa." Master's thesis, Faculty of Health Sciences, 2020. http://hdl.handle.net/11427/32714.

Full text
Abstract:
Introduction: Plasmablastic lymphoma (PBL) is a rare, aggressive, AIDS-associated non-Hodgkin lymphoma. The pathogenesis of PBL is incompletely understood, however association with the Epstein-Barr virus (EBV) and the MYC gene, have been identified as important pathogenic mechanisms. Aims and objectives: To characterise the EBV latency in a cohort of patients diagnosed with PBL at Groote Schuur Hospital (GSH), by means of immunohistochemistry. To determine MYC gene aberrations using fluorescent in situ hybridisation (FISH). Materials and methods: The cohort comprised PBL cases diagnosed from 2005-2017. EBER ISH was used to confirm EBV infection. Manual immunohistochemistry using three monoclonal antibodies for EBV latent proteins, (EBNA1, EBNA2 and LMP1) was used to determine the latency type. Manual MYC FISH was performed on all PBL cases using a dual colour break apart rearrangement probe. Results: Forty-nine cases of PBL were included in this study. Forty-one cases were positive for EBER ISH. Thirty-seven (78.7%) cases showed HIV/EBV coinfection. Latency 0 was observed in 29 (70.7%) cases, latency 1 in 8 (19.5%) and latency 2 in 4 (9.8%) cases. MYC FISH was performed on all 49 PBL cases, of which 30 (61.2%) yielded a result. MYC was intact in 11 (36.7%), translocated in 8 (26.7%) and 11 (36.7 %) cases showed copy number variations. Conclusion: Our research demonstrated 37 (90.2%) of the EBV positive PBL cases showed a restricted latency pattern of 0 or 1. Furthermore we found that MYC gene aberrations consisting of translocations and copy number variations occurred in 19 cases (63.3%) , with copy number variations being higher than cited in current literature. Our study is also the first to investigate PBL EBV latency in SA. An uncommon finding was the existence of MYC gene aberrations in HIV positive, EBV negative PBL cases.
APA, Harvard, Vancouver, ISO, and other styles
4

Santamaria, Kathleen. "Etude de l’hétérogénéité des centrocytes humains à travers l’expression du CD23 : différenciation en plasmablastes et expression d’une signature minimale transcriptionnelle au niveau cellule-unique comportant DEC2." Thesis, Rennes 1, 2020. http://www.theses.fr/2020REN1B038.

Full text
Abstract:
La différenciation terminale des lymphocytes B à travers le centre germinatif conduit à la production de cellules sécrétrices d’anticorps : les plasmocytes (PC) à longue durée de vie et de haute affinité pour l’antigène. Cette réaction implique la formation d’une microstructure anatomique qui comprend une zone sombre : lieu d’intenses proliférations des centroblastes et de maturation d’affinité de leur BCR, et une zone claire dans laquelle ont lieu les commutations de classes isotypiques du BCR et la sélection des centrocytes (CC). Ce processus est finement contrôlé par les lymphocytes T folliculaires auxiliaires (Tfh) notamment à travers la production de molécules comme l’IL-4, le CD40L et l’IL-21. Ce travail de thèse s'intéresse à l'expression du CD23 à la surface des CC lors de leur métamorphose en PC. J'ai d’abord montré que l'expression de ce récepteur de faible affinité aux IgE est régulée par les signaux Tfh : CD40L et IL-4. De plus j'ai mis en évidence que les CC humains exposés aux signaux Tfh mais négatifs pour le CD23 sont capables de se différencier en PC. Dans ce cadre, la signature transcriptionnelle de ces progéniteurs de PC a été étudiée à l'échelle de la cellule unique et a permis d’identifier un gène jusqu’alors jamais décrit dans les PC qui code pour le facteur de transcription DEC2 et dont la fonction reste à déterminer. J'ai par ailleurs étudié l'expression du CD23 dans le lymphome folliculaire et mis en évidence qu’il existe des différences entre les populations tumorales positives et négatives pour le CD23, suggérant que les cellules CD23neg ont un avantage de survie et une capacité de différenciation supérieure en culture que les cellules CD23pos
Terminal B cell differentiation through the germinal center leads to the production of antibody-secreting cells: plasma cells (PC) with a long lifespan and high affinity for the antigen. This reaction involves the formation of an anatomical microstructure that includes a dark zone: site of intense proliferation and affinity maturation of the BCR of the centroblasts, and a light zone in which the class switch recombination of the BCR and centrocyte (CC) selection take place. This differentiation is finely controlled by follicular helper T cells (Tfh) that produce molecules such as IL-4, CD40L and IL-21. This phD work focus on the CD23 expression on the surface of CC during their metamorphosis into PC. Firstly, I showed that the expression of the low affinity IgE receptor is regulated by Tfh derived IL-4 and CD40L. Moreover, I showed that CC exposed to Tfh signals and which do not express the CD23 were the ones that have the ability to differentiate into PC. In this context, I identified at the single cell level a transcriptional signature expressed specifically by these cells which contains a gene never described in PC, encoding the transcription factor DEC2 whose function remains to be determined. In addition, I studied CD23 expression in follicular lymphoma and showed the existence of differences between these two populations, suggesting a preferential survival and differentiation of CD23neg cells compared to CD23pos cells
APA, Harvard, Vancouver, ISO, and other styles
5

Perner, Yvonne. "EBV status in extra-oral plasmablastic lymphomas." Thesis, 2016. http://hdl.handle.net/10539/21413.

Full text
Abstract:
A research report submitted to the Faculty of Health Sciences, University of the Witwatersrand, Johannesburg, in partial fulfilment of the requirements for the degree of Master of Medicine in the branch of Anatomical Pathology Johannesburg, 2016
Introduction: Plasmablastic lymphoma (PBL) is an uncommon variant of aggressive B-cell non-Hodgkin lymphoma that occurs in immune-compromised individuals, most commonly secondary to HIV infection. This tumour classically occurs in the oral cavity but has also been described in a variety of extra-oral locations. This is a clinicopathological study of 45 cases of extra-oral PBL (EPBL). Aim: To define the clinical parameters, histology and immunophenotypic features of extra-oral plasmablastic lymphoma (EPBL) and assess the extent to which Epstein–Barr virus (EBV) is associated with this tumour. Materials and Methods: This retrospective study on archival cases of EPBL included the patients‟ age, gender, race, HIV status where available and the site of tumour presentation. Of 49 archival cases retrieved, 4 were discounted owing to reclassification as diffuse large B-cell lymphoma (1 case), multiple myeloma or extramedullary plasma cell tumour (3 cases). The remaining 45 cases were reviewed histologically and classified according to whether they displayed a pure plasmablastic (PBm) morphology or a plasmablastic morphology with plasmacytic differentiation (PBm+PCd), and assessed immunohistochemically with CD45 (LCA), CD20, CD79a, PAX5, CD138, MUM1, BLIMP1, VS38c, Ki-67, BCL6, CD10, HHV8 and CyclinD1 using standard automated procedures. The presence of EBV was assessed by chromogenic in-situ hybridisation. Ethical clearance was obtained (M10750 and M120993). Results: 27of the 45 cases had a pure plasmablastic morphology. The remaining 18 cases showed plasmacytic differentiation. There was no site predilection according to histological pattern. 60% of the tumours were reported in males and 40% in females and all were black African patients. The anus was the favoured extra-oral site of presentation (13 of 45 cases, 28%), followed by soft tissue (11 of 45 cases, 24%). There was no significant difference in the age of presentation between males (38.5 years) and females (35.4 years). Of the 18 patients of known HIV status, 17 were HIV positive (94%). The immunohistochemical profile of EPBL recapitulated that found for both oral and extra-oral PBL in the literature, except for CD45 (leucocyte common antigen) which signalled positively in a higher percentage of cases. 36 of 42 cases (85.7%) were positive for CD45. The positive membrane signal for CD45 was of variable intensity, between 5 and 100% of tumour cells. EBV was positive by in situ hybridisation in 37 of 40 cases tested (92.5%). Conclusion: EPBL is identical to its oral counterpart in gender and age distribution, HIV status, morphological appearances, immunophenotypic profile and association with EBV. The high association with EBV as assessed by in-situ hybridisation studies mirrors that of oral-based PBL reported in the literature. EPBL should be regarded as the same tumour as that arising within the oral cavity. A peculiarity observed within this case cohort is the high level of expression of CD45 (leucocyte common antigen). This has been reported to be of low or near absent expression in most cases of PBL, as defined by the 2008 WHO Classification of Tumours of the Haematopoietic and Lymphoid Tissues.
MT2016
APA, Harvard, Vancouver, ISO, and other styles
6

Boy, Sonja Catharina. "The molecular profile of oral plasmablastic lymphomas in a South African population sample." Thesis, 2011. http://hdl.handle.net/2263/28880.

Full text
Abstract:
Plasmablastic lymphoma (PBL) was originally described in 1997 as an AIDS associated tumour although cases have been described in individuals not infected with HIV. Due to the high number of people living with HIV in South Africa, a substantial number of cases are diagnosed annually and 45 cases were included in this study. This represented the largest cohort of PBL affecting the oral mucosa published to date. Three main aspects of PBL were investigated: pathological features, viral status and certain genetic characteristics. The results from the genetic studies were the most important and interesting. These included rearrangements of the IGH gene in 63% and MYC- rearrangements in 62% of PBL’s. Seven of 43 cases (16%) showed rearrangement of both the IGH gene alleles, a finding never described before. New genetic findings also included increased CCND1 gene copy numbers in 17/41 (42%) and increased IGH gene copy numbers in 6/41 (15%) of cases. The exact role of MYC-rearrangements in the development of PBL is unclear. Many factors may be responsible for MYC deregulation but in the case of PBL of the oral cavity the possible role of Epstein Barr Virus (EBV) infection was considered. All but one of the patients with known HIV-status (32/45) was HIV positive and I supported the proposal that the diagnosis of PBL should serve as a sign of immunodeficiency, either as diagnostic thereof or as a predictor of a progressive state of immunodeficiency in patients with known HIV/AIDS status. The HIV-negative patient in this study was the only one that presented with an EBV-negative PBL on in situ hybridisation. The clinico-pathological features of the current study therefore strongly suggested an association between EBV, PBL and HIV/AIDS although the exact nature thereof remains uncertain. Routine genetic evaluation of tumours diagnosed as PBL should be introduced, as this may have prognostic and eventually treatment implications in the future. The exact panel of genes to be evaluated with a possible diagnosis of PBL should still be determined but examination of IGH and MYC for rearrangements should be included. This study proved the histomorphological features including the degree of plasmacytic differentiation not to have any diagnostic role although its prognostic value should be determined. The results of the immunohistochemical investigations performed in this study confirmed PBL always to be negative for CD20 but proved PBL not to be a morphological or immunohistochemical diagnosis by any means. In conclusion, it became clear that PBL should never be diagnosed without thorough clinical, systemic, pathological and genetic investigations, especially in the backdrop of HIV/AIDS. No pathologist should make the diagnosis of PBL and no clinician should accept such a diagnosis or decide on the treatment modality for the patient involved unless all other possibilities of systemic plasma cell disease have been excluded.
Thesis (PhD)--University of Pretoria, 2011.
Oral Pathology and Oral Biology
unrestricted
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Plasmablastic lymphoma"

1

Wang, Huan-You, Ida Wong-Sefidan, and Erin Reid. "Plasmablastic Lymphoma." In Encyclopedia of AIDS, 1–8. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9610-6_28-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Carbone, Antonino, and Annunziata Gloghini. "Plasmablastic Lymphoma." In Encyclopedia of Pathology, 414–17. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-95309-0_3834.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Wang, Huan-You, Ida Wong-Sefidan, and Erin Reid. "Plasmablastic Lymphoma." In Encyclopedia of AIDS, 1644–50. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7101-5_28.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Wang, Huan-You, Ida Wong-Sefidan, and Erin Reid. "Plasmablastic Lymphoma." In Cancers in People with HIV and AIDS, 223–34. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0859-2_16.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Carbone, Antonino, and Annunziata Gloghini. "Plasmablastic Lymphoma." In Encyclopedia of Pathology, 1–5. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-28845-1_3834-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Miranda, Roberto N., Joseph D. Khoury, and L. Jeffrey Medeiros. "Plasmablastic Lymphoma." In Atlas of Lymph Node Pathology, 265–68. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7959-8_56.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Rubinstein, Paul G., and Christoph Wyen. "AIDS-Related Plasmablastic Lymphoma." In HIV-associated Hematological Malignancies, 73–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-26857-6_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Guerrero-Garcia, Thomas A., and Jorge J. Castillo. "Plasmablastic Lymphoma and Primary Effusion Lymphoma." In Novel Therapeutics for Rare Lymphomas, 101–18. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-25610-4_7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

"Plasmablastic Lymphoma." In Diagnostic Pathology: Lymph Nodes and Extranodal Lymphomas, 500–509. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-47779-6.50074-0.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

"Plasmablastic Lymphoma." In Diagnostic Pathology: Cytopathology, 362–63. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-54763-5.50133-1.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Plasmablastic lymphoma"

1

Leeman-Neill, Rebecca J., Craig Soderquist, Murty Vundavalli, David Park, Susan Hsiao, Mahesh Mansukhani, Bachir Alobeid, and Govind Bhagat. "Abstract 171: Genomic and phenotypic analysis of post-transplant plasmablastic lymphomas." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-171.

Full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Plasmablastic lymphoma' for particular source types:
Journal articles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography