Relevant bibliographies by topics / Plasmid-mediated-resistance

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Academic literature on the topic 'Plasmid-mediated-resistance'

Author: Grafiati

Published: 10 September 2021

Last updated: 29 July 2025

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Journal articles on the topic "Plasmid-mediated-resistance"

Martínez-Martínez, Luis, María Eliecer Cano, José Manuel Rodríguez-Martínez, Jorge Calvo, and Álvaro Pascual. "Plasmid-mediated quinolone resistance." Expert Review of Anti-infective Therapy 6, no. 5 (2008): 685–711. http://dx.doi.org/10.1586/14787210.6.5.685.

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DRABU, Y. J., P. H. BLAKEMORE, D. M. COX, and A. McLAREN. "Plasmid-mediated ceftazidime resistance." Journal of Antimicrobial Chemotherapy 23, no. 2 (1989): 287–90. http://dx.doi.org/10.1093/jac/23.2.287.

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PHILLIPS, IAN, ANNA KING, and KEVIN SHANNON. "Plasmid-mediated ceftazidime resistance." Journal of Antimicrobial Chemotherapy 24, no. 1 (1989): 85. http://dx.doi.org/10.1093/jac/24.1.85.

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Schultsz, Constance, and Suzanne Geerlings. "Plasmid-Mediated Resistance in Enterobacteriaceae." Drugs 72, no. 1 (2012): 1–16. http://dx.doi.org/10.2165/11597960-000000000-00000.

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Piekarska, Katarzyna. "PLASMID-MEDIATED QUINOLONE RESISTANCE - PMQR." Postępy Mikrobiologii - Advancements of Microbiology 57, no. 1 (2019): 47–57. http://dx.doi.org/10.21307/pm-2018.57.1.047.

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Starliper, Clifford E., Richard K. Cooper, Emmett B. Shotts, and Peter W. Taylor. "Plasmid-Mediated Romet Resistance ofEdwardsiella ictaluri." Journal of Aquatic Animal Health 5, no. 1 (1993): 1–8. http://dx.doi.org/10.1577/1548-8667(1993)005<0001:pmrroe>2.3.co;2.

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Rodriguez-Martinez, Jose-Manuel, Laurent Poirel, Alvaro Pascual, and Patrice Nordmann. "Plasmid-Mediated Quinolone Resistance in Australia." Microbial Drug Resistance 12, no. 2 (2006): 99–102. http://dx.doi.org/10.1089/mdr.2006.12.99.

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Tran, J. H., and G. A. Jacoby. "Mechanism of plasmid-mediated quinolone resistance." Proceedings of the National Academy of Sciences 99, no. 8 (2002): 5638–42. http://dx.doi.org/10.1073/pnas.082092899.

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Jacoby, George A., Nancy Chow, and Ken B. Waites. "Prevalence of Plasmid-Mediated Quinolone Resistance." Antimicrobial Agents and Chemotherapy 47, no. 2 (2003): 559–62. http://dx.doi.org/10.1128/aac.47.2.559-562.2003.

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ABSTRACT Quinolone resistance encoded by the qnr gene and mediated by plasmid pMG252 was discovered in a clinical strain of Klebsiella pneumoniae that was isolated in 1994 at the University of Alabama at Birmingham Medical Center. The gene codes for a protein that protects DNA gyrase from quinolone inhibition and that belongs to the pentapeptide repeat family of proteins. The prevalence of the gene has been investigated by using PCR with qnr-specific primers with a sample of more than 350 gram-negative strains that originated in 18 countries and 24 states in the United States and that included

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Dolejska, Monika, and Costas C. Papagiannitsis. "Plasmid-mediated resistance is going wild." Plasmid 99 (September 2018): 99–111. http://dx.doi.org/10.1016/j.plasmid.2018.09.010.

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Dissertations / Theses on the topic "Plasmid-mediated-resistance"

BERTINI, ALESSIA. "Plasmid-mediated antimicrobial resistance in Gram-negative bacteria." Doctoral thesis, Università degli Studi di Roma "Tor Vergata", 2008. http://hdl.handle.net/2108/641.

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L’epidemiologia dei plasmidi di resistenza è il principale mezzo per descrivere la diffusione delle resistenze agli antimicrobici. L’identificazione di plasmidi correlati associati a specifici geni di resistenza può aiutare a tracciare la disseminazione di plasmidi epidemici, aprendo nuovi scenari epidemiologici sui meccanismi di diffusione delle resistenze agli antimicrobici. Questo aspetto è particolarmente evidente quando si considerano plasmidi che sono associati alla diffusione delle Beta Lattamasi a Spettro Esteso (ESBL) come gli enzimi CTX-M, SHV, TEM. Lo scopo di questa tesi è st

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Bottery, Michael J. "The sociality and evolution of plasmid-mediated antimicrobial resistance." Thesis, University of York, 2017. http://etheses.whiterose.ac.uk/19016/.

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Overuse and misuse of antibiotics has led to the global spread of antimicrobial resistance, threatening our ability to treat bacterial infections. The horizontal acquisition of multidrug resistance (MDR) plasmids, from other bacterial lineages, has been instrumental in spreading resistance. Newly acquired plasmids are often poorly adapted to hosts causing intragenomic conflicts, reducing the competitiveness of plasmid-carrying strains. Costs can be overcome by positive selection for plasmid-encoded adaptive traits in the short-term, or ameliorated by compensatory evolution in the long-term. Ho

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Risley, Claire. "The population dynamics of plasmid-mediated antibiotic resistance in salmonella typhimurium in chickens." Thesis, University of Oxford, 2002. http://ora.ox.ac.uk/objects/uuid:a70bda98-533f-41d0-b9a0-630457b3f982.

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A model of growth and plasmid transfer between strains of Escherichia coli and Salmonella typhimurium was developed with reference to the literature. This was the organising principle for the collection of a complete set of in vitro life history parameters of one S. typhimurium and one E. coli strain. In the course of estimating these parameters two results of note were obtained. Fits of the Lotka-Volterra competition model were obtained for data on S. typhimuiurm growing in competition with E. coli. The first noteworthy discovery was the failure of this model to account for several characteri

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Le, Vien Thi Minh. "Plasmid-mediated quinolone resistance determinants in Enterobacteriaceae isolated in Ho Chi Minh City, Vietnam." Thesis, University of Oxford, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.559782.

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Fluoroquinolones are amongst the most effective antimicrobials used to treat infectious diseases caused by members of the Enterobacteriaceae. The rapid spread of fluoroquinolone resistant Enterobacteriaceae threatens the future effectiveness of this widely used group of antimicrobial compounds. Fluoroquinolone resistance in Enterobacteriaceae in Vietnam highlights an approaching crisis in antimicrobial therapy of Enterobacteriaceae infections as fluoroquinolone resistant infections may require expensive alternative treatments. Furthermore, a lack of alternative drugs for treating fluoroquinolo

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Guerreiro, Lara Sofia Fernandes. "Influência do uso de enrofloxacina no aparecimento de resistência às quinolonas mediada por plasmídeos em Escherichia coli de vitelos." Master's thesis, Universidade Técnica de Lisboa. Faculdade de Medicina Veterinária, 2012. http://hdl.handle.net/10400.5/4589.

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Dissertação de Mestrado em Segurança Alimentar O conhecimento sobre a presença e frequência de genes de resistência às quinolonas mediada por plasmídeos (RQMP) em estirpes comensais de Escherichia coli de origem bovina é escasso a nível mundial. Foram estudadas um total de 237 amostras de E. coli de vitelos saudáveis previamente isoladas após pressão selectiva in vivo de enrofloxacina (ENR) e caracterizadas quanto à resistência aos antibióticos: 101, 79 e 57 isolados relativos a T0, T1 (6 semanas após administração de ENR) e T2 (10 semanas após administração de ENR), respectivamente. Os iso

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Sousa, Rafaela Rogério Floriano de. "Pesquisa de genes de resistência a quinolonas em bacilos Gram negativos de origem clínica e ambiental." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/6/6135/tde-27032014-091736/.

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Introdução. Quinolonas são antimicrobianos sintéticos que inibem as enzimas DNA-girase e topoisomerase IV resultando na morte bacteriana. São altamente eficazes no tratamento de infecções bacterianas, especialmente causadas por bactérias Gram negativas, e portanto amplamente utilizados na medicina humana e veterinária, na qual também são empregados como profiláticos. Porém, o uso indiscriminado e inadequado levou ao aumento de bactérias resistentes a estes compostos. Esta resistência pode ocorrer devido a mutações nas enzimas DNA-girase e topoisomerase IV, e também por genes contidos em plasmí

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KOCSIS, Bela. "Plasmid-mediated fluoroquinolone resistance in Enterobacteriaceae." Doctoral thesis, 2012. http://hdl.handle.net/11562/393920.

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In tutto il mondo si sta assistendo ad un aumento dell’incidenza di isolati clinici di Enterobacteriaceae resistenti ai fluorochinoloni. La resistenza può essere dovuta ad un accumulo di mutazioni cromosomiche a livello dei geni che codificano le topoisomerasi, bersaglio d’azione dei fluorochinoloni; mediata da determinanti plasmidici, in particolare quelli che codificano per proteine di protezione Qnr, per l’enzima di acetilazione aac(6’)-Ib-cr e per le pompe di efflusso QepA e OqxAB. Tutti questi determinanti da soli conferiscono al batterio bassi livelli di resistenza ma consentono un aume

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Wang, Ting-Yin, and 王亭茵. "Characterization of plasmid-mediated fosfomycin resistance determinants in carbapenem-resistant Klebsiella pneumoniae isolates in Taiwan." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/40027960002821028863.

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碩士 高雄醫學大學 醫學檢驗生物技術學系碩士班 104 Carbapenem-resistant Klebsiella pneumoniae which are highly resistant to many antibiotics have been recognized as emerging problems worldwide. Recently, there has been a renewed interest in reviving older antimicrobial agents, such as fosfomycin, for multidrug-resistant pathogens. Fosfomycin is a bactericidal antibiotic with broad-spectrum activity against wide range of Gram-positive and Gram-negative bacteria. Fosfomycin has been used for the successful treatment of urinary tract infections for a long time. It could be considered as medical treatment of

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"Detection and significance of plasmid-mediated quinolone resistance (qnr) genes in Enterobacteriaceae isolates from bacteraemic patients in Hong Kong." 2010. http://library.cuhk.edu.hk/record=b5894440.

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Lee, Ching Ching. Thesis (M.Phil.)--Chinese University of Hong Kong, 2010. Includes bibliographical references (leaves 90-103). Abstracts in English and Chinese. Acknowledgments --- p.i Abstract --- p.ii 論文摘要 --- p.iv Table of Contents --- p.vi List of Tables --- p.x List of Figures --- p.xi Chapter Chapter 1 --- Introduction Chapter 1.1. --- Quinolone Antimicrobial Agents --- p.1 Chapter 1.1.1. --- Development --- p.1 Chapter 1.1.2. --- Mode of action --- p.3 Chapter 1.1.3. --- Mechanisms of resistance to quinolones --- p.4 Chapter 1.1.3.

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LO, PICCOLO FRANCESCO. "Multidrug Resistance and Production of Extended Spectrum β-lactamases and Plasmid-mediated AmpC β-lactamases in Enterobacteriaceae isolates from diseased cats in Sicily". Doctoral thesis, 2017. http://hdl.handle.net/11570/3104461.

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Introduction: In human and veterinary medicine, Enterobacteriaceae are common causes of enteric and extra-intestinal opportunistic infections and their resistance to multiple antimicrobials is a major global threat. Multidrug-resistant (MDR) Enterobacteriaceae are increasingly reported in companion animals, thus raising great concerns for animal and public health (Bogaerts et al., 2015). The β-lactam resistance in Enterobacteriaceae is associated mainly with production of enzymes hydrolyzing these antibiotics, among which the extended-spectrum β-lactamases (ESBLs), Plasmid-mediated AmpC β-lact

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Books on the topic "Plasmid-mediated-resistance"

Barrett, Siobhán. Studies on plasmid-mediated copper resistance in Escherichia coli. Universityof Birmingham, 1994.

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Huovinen, Saara. Plasmid-mediated [beta]-lactamase resistance in gram-negative bacilli: Detection of [beta]-lactamase genes and [beta]-lactamases by using DNA-hybridization and isoelectric focusing. S. Huovinen, 1988.

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Book chapters on the topic "Plasmid-mediated-resistance"

Jacoby, George A. "Plasmid-Mediated Quinolone Resistance." In Antimicrobial Drug Resistance. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-180-2_17.

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Jacoby, George A. "Plasmid-Mediated Quinolone Resistance." In Antimicrobial Drug Resistance. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46718-4_17.

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Jacoby, George A., Jacob Strahilevitz, and David C. Hooper. "Plasmid-Mediated Quinolone Resistance." In Plasmids. ASM Press, 2015. http://dx.doi.org/10.1128/9781555818982.ch25.

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Cullen, D. W., I. M. Packer, and D. J. Platt. "Plasmid Mediated Resistance to Hexavalent Chromium in Agrobacterium." In The Release of Genetically Modified Microorganisms—REGEM 2. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4613-0493-7_47.

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Jacoby, George A. "Study of Plasmid-Mediated Quinolone Resistance in Bacteria." In Methods in Molecular Biology. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7459-7_22.

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Fernández-Beros, M. E., C. González, V. Kissel, et al. "Plasmid-Mediated Complement and Phagocytosis Resistance in E. Coli." In Bacteria, Complement and the Phagocytic Cell. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-85718-8_24.

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Schwarz, Stefan, Jianzhong Shen, Sarah Wendlandt, et al. "Plasmid-Mediated Antimicrobial Resistance in Staphylococci and Other Firmicutes." In Plasmids. ASM Press, 2015. http://dx.doi.org/10.1128/9781555818982.ch22.

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Ramirez, Maria S., German M. Traglia, David L. Lin, Tung Tran, and Marcelo E. Tolmasky. "Plasmid-Mediated Antibiotic Resistance and Virulence in Gram-Negatives: The Klebsiella pneumoniae Paradigm." In Plasmids. ASM Press, 2015. http://dx.doi.org/10.1128/9781555818982.ch24.

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Knapp, J. S., J. M. Zenilman, J. W. Biddle, et al. "Distribution and frequency of strains of Neisseria gonorrhoeae with plasmid-mediated, high-level resistance to tetracycline (TRNG) in the United States." In Gonococci and Meningococci. Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1383-7_21.

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Li, Xian-Zhi, and Manisha Mehrotra. "Role of Plasmid-Encoded Drug Efflux Pumps in Antimicrobial Resistance." In Efflux-Mediated Antimicrobial Resistance in Bacteria. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-39658-3_23.

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Conference papers on the topic "Plasmid-mediated-resistance"

Liao, Pinyu. "Exploiting Plasmid-Mediated Resistance: Design of Small-Molecule Inhibitors for the Disruption of the Kid-Kis Toxin-Antitoxin System in Plasmid R1." In 2022 IEEE 12th International Conference Nanomaterials: Applications & Properties (NAP). IEEE, 2022. http://dx.doi.org/10.1109/nap55339.2022.9934272.

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Hassan, Teba Shaker, and Mouna Akeel Hamed Al-Oebady. "Prevalence of plasmid-mediated quinolone resistance (PMQR) genes in Klebsiella pneumoniae isolates from Iraqi patients." In FIFTH INTERNATIONAL CONFERENCE ON APPLIED SCIENCES: ICAS2023. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0209739.

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Shams, Noora, Hanin AlHiraky, Nabila Moulana, et al. "Comparison of Available Methods for Investigating The in vitro Activity of Colistin Against Different Gram-Negative Bacilli." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0121.

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Background: The surge in the prevalence of multidrug-resistant (MDR) Gram-negative bacterial infections with limited treatment options and the decrease in the development of new antibiotics are challenges that lead to the reuse of colistin to treat infections caused by MDR pathogens. This study aimed to determine economical, simple, and reliable colistin susceptibility testing methods as an alternative to the time and effort-consuming microdilution technique and identify the colistin resistance's genetic determinants to find if it affects the testing method. Material and Methods: Seven colisti

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Reports on the topic "Plasmid-mediated-resistance"

Trezona, Thomas. Plasmid-mediated resistance to arsenite and arsenate in Escherichia coli. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.3125.

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Buaboocha, Teerapong, and Supachitra Chadchawan. Effect of transgenic expression in rice of a calmodulin on salt stress response. Chulalongkorn University, 2009. https://doi.org/10.58837/chula.res.2009.31.

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Calcium signaling has been implicated in transducing signals from environmental changes into adaptive responses in plants. Calmodulin (CaM), members of the EF-hand family of Ca2+-binding proteins, represent important relays in plant calcium signals. Here, transgenic rice plants containing OsCam1-1 gene under the control of 35SCaMV promoter (35SCaMV-OsCam1-1) were constructed by Agrobacterium-mediated transformation via pCAMBIA1301 plasmid. Three putative transgenic rice lines harboring the 35SCaMV-OsCam1-1 and two putative transgenic lines harboring the T-DNA from pCAMBIA1301 alone as negative

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Freeman, Stanley, and Russell J. Rodriguez. The Interaction Between Nonpathogenic Mutants of Colletotrichum and Fusarium, and the Plant Host Defense System. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7573069.bard.

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The intent of this proposal was to study the interaction between nonpathogenic mutants of Colletotrichum magna and Fusarium oxysporum, and the cucurbit host defense system. We had shown previously that a nonpathogenic endophytic mutant path- 1 of C. magna, caused no visible disease symptoms but protected watermelon seedlings from disease caused by the wildtype isolate and F. o. niveum. Objectives were: 1) Determine the microscopic, biochemical and molecular genetic interaction between "protected" (path- 1 colonized) cucurbit hosts and wildtype isolates of C. magna; 2) Isolate non-pathogenic mu

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Coplin, David L., Shulamit Manulis, and Isaac Barash. roles Hrp-dependent effector proteins and hrp gene regulation as determinants of virulence and host-specificity in Erwinia stewartii and E. herbicola pvs. gypsophilae and betae. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7587216.bard.

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Gram-negative plant pathogenic bacteria employ specialized type-III secretion systems (TTSS) to deliver an arsenal of pathogenicity proteins directly into host cells. These secretion systems are encoded by hrp genes (for hypersensitive response and pathogenicity) and the effector proteins by so-called dsp or avr genes. The functions of effectors are to enable bacterial multiplication by damaging host cells and/or by blocking host defenses. We characterized essential hrp gene clusters in the Stewart's Wilt of maize pathogen, Pantoea stewartii subsp. stewartii (Pnss; formerly Erwinia stewartii)

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