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Relevant bibliographies by topics / Plasmodium falciparum

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Academic literature on the topic 'Plasmodium falciparum'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 June 2025

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Journal articles on the topic "Plasmodium falciparum"

1

Tokponnon, Filémon, Razak Osse, Jean Gille Egui, et al. "Determination of Plasmodial Species Prevalence among Patients Received at Cotonou Boni Clinic during Rainy Season in the Year 2022." International Journal of TROPICAL DISEASE & Health 44, no. 16 (2023): 9–15. http://dx.doi.org/10.9734/ijtdh/2023/v44i161464.

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Aims: Malaria is a life-threatening disease caused by parasites transmitted by bites from infected female anopheles. It is a preventable and treatable illness. It remains a recurring disease among public health diseases that exposes many people to a risk of infection, including children under the age of 05 in Benin. Methods: To determine the prevalence of malaria and different plasmodial species at the Dr Pierre BONI Clinic, we performed venous and capillary samples on 731 patients for the realization of thick drops and blood smears between June and July 2022. Results: Three plasmodial species were identified in 228 patients (31%) as malaria of the 731 patients included in the study with 3 species of plasmodium found: Plasmodium falciparum (95.5%), Plasmodium malariae (2.85%), Plasmodium ovale (1.65%). Mixed or double species was also recorded in some patients: Plasmodium falciparum+ Plasmodium malariae and Plasmodium falciparum+ Plasmodium ovale. The majority of patients have the presence of trophozoites at Plasmodium falciparum, 95.5%. The parasitic density of P. falciparum is higher than that of P. malariae and that of P. ovale. Conclusion: Although evaluated during a period of low transmission, malaria remains a real public health problem. The distribution of the disease is closely related to the presence in the blood of plasmodial species.

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MD, Dr Tapan Biswas. "Artesunate Resistant Plasmodium Falciparum." Journal of Medical Science And clinical Research 05, no. 04 (2017): 20025–27. http://dx.doi.org/10.18535/jmscr/v5i4.48.

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Anyanwu, E.B., I. Onyesom, B. Okperi, V.O Awusi, T.O Mabiaku, and D.O Umukoro. "THE VIRULENCE OF PLASMODIUM FALCIPARUM INFECTION IN HUMAN HOST." Continental J. Medical Research 2 (November 4, 2008): 35–38. https://doi.org/10.5281/zenodo.3526992.

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Malaria causes an acute febrile illness which may be characterized by periods of fibrile paroxysms. The severity and course of an attack of malaria depends on species and strains of the infecting parasite. Mankind is infected by four species of the genus Plasmodium but Plasmodium falciparum is associated with the highest degree of parasitaema due to high yields of merozoites per schizonts. The malignancy of Plasmodium falciparum is peculiar to that species. This review highlights some of the reasons why infection by Plasmodium falciparum may be malignant and more virulent than the other plasmodia species.   

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Merrick, Catherine J. "Plasmodium falciparum." Emerging Topics in Life Sciences 1, no. 6 (2017): 517–23. http://dx.doi.org/10.1042/etls20170099.

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Plasmodium falciparum is a protozoan parasite that causes the most severe form of human malaria. Five other Plasmodium species can also infect humans — P. vivax, P. malariae, P. ovale curtisi, P. ovale wallikeri and P. knowlesi — but P. falciparum is the most prevalent Plasmodium species in the African region, where 90% of all malaria occurs, and it is this species that causes the great majority of malaria deaths. These were reported by the WHO at 438 000 in 2015 from an estimated 214 million cases; importantly, however, figures for the global burden of malaria tend to have wide margins of error due to poor and inaccurate reporting. In this Perspective, features of P. falciparum that are unique among human malaria parasites are highlighted, and current issues surrounding the control and treatment of this major human pathogen are discussed.

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Maier, Alexander G., Kai Matuschewski, Meng Zhang, and Melanie Rug. "Plasmodium falciparum." Trends in Parasitology 35, no. 6 (2019): 481–82. http://dx.doi.org/10.1016/j.pt.2018.11.010.

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Fayyaz Ahmed Memon, Uzma Bukhari, and Mir Mohammad Sahito. "PREVALENCE OF MALARIA AMONG THE PATIENTS LIVING IN AREAS OF DISTRICT SBA (SHAHEED BENAZIR BHUTTO) AND MIRPURKHAS." JMMC 5, no. 1 (2014): 1–3. http://dx.doi.org/10.62118/jmmc.v5i1.345.

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Objective:Malaria is a major cause of morbidity in the tropics and about 300 million causes were reported word wide in2006 among the 100 species of genous plasmodia, the four species such as PL: falciparum, vivax, ovaule andmalaria causes malaria. The malaria is transmitted by the bite of female anopheles mosquitoes. .Methodology: This descriptive and experimental study was carried out at department of pathology, People'sUniversity of Medical & Health Science (PUMHS), Nawabshah. The cases were collected from paediatrics &Medical outpatients departments of PUMHS Hospital Nawabshah and also from Muhammad Medical College(MMC) Hospital & Civil hospital Mirpurkhas (CHM) from January 2010 to December 2011. A total of 1200 patientswere included. The prevalence of malaria on the basis of age, sex, areas of resident, and clinical finding of allpatients were recorded and blood tests performed.Results: Plasmodium Vivax in 70.8% of cases and Plasmodium Falciparum in29.2%of cases.Conclusion: In the areas (Nawabshah and Mirpurkhas), Plasmodium Vivax and Plasmodium Falciparum are thecause of Malaria.Keywords: Malaria, Plasmodium, Pakistan, Nawabshah, Mirpurkhas.

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Trasi, Reqgi First. "Plasmodium Resistance to Artemisinin Derivates due to Kelch-3 Gene Mutation." Indonesian Journal of Pharmaceutical and Clinical Research 4, no. 2 (2021): 39–44. http://dx.doi.org/10.32734/idjpcr.v4i2.6332.

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Artemisinin class of antimalarial drugs play an important role in controlling falciparum malaria after the emergence of resistance of Plasmodium falciparum to other antimalarial drugs such as chloroquine, sulfadoxine-pyrimethamine and mefloquine. Therefore, the presence of Plasmodium falciparum resistance to this class of drugs is threat to global efforts to eliminate this disease. Resistance of Plasmodium falciparum to artemisinin recently known to be associated with mutations in the propeller domain of the kelch-13 (K13) Plasmodium falciparum gene. The incidence of Plasmodium falciparum resistance due to mutations in the K13 gene, among others, can be found in Cambodia, Laos, Vietnam, China, Myanmar, Thailand and Africa. The presence of mutations in this gene will change the response of Plasmodium falciparum against oxidative stress induced by artemisinin by involving the proteasome-ubiquitin pathway. In addition, mutation K13 will also change the levels of PI3K and PI3P in the body of Plasmodium falciparum. PI3K and PI3P are lipids that essential for the development of Plasmodium falciparum from ring stage to schizont. Resistance to artemisinin will also provide phenotypic changes in the life cycle of Plasmodium falciparum in the form of elongation at the stage ring and transient shortening in trophozoite development. This resistance incident can be overcome, among others by prolonging the duration of treatment (from a 3-day regimen to a 4-day regimen) and combining artemisinin with proteasome inhibitors.

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ARICAN ÇELİK, Aslı, Rıfat TAMALI, Gülruhsar YILMAZ, Pınar KARABACAK, and Mustafa Soner ÖZCAN. "A Rare Cause of Fever in ICU; Malaria." Acta Medica Nicomedia 5, no. 3 (2022): 235–37. http://dx.doi.org/10.53446/actamednicomedia.1103400.

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Sıtma, anofel cinsi sivrisineklerin insanları sokması sonucu bulaşan paraziter bir hastalıktır. Plasmodium falciparum (P Falciparum) en ölümcül tabloya yol açan türüdür, ülkemizde nadir görülür ve genellikle yurtdışı kaynaklıdır. Yoğun bakımda yüksek ateş pek çok klinik durumda karşımıza çıksa da nadir görülen bir durum olan P. Falciparuma bağlı sıtma aklımızda bulunmalıdır Bu olgu sunumunda, yurt dışı seyahat öyküsü bulunan P. Falciparum sıtma tanısı konulan hastanın yoğun bakım takip ve tedavi süreci sunulmuştur.

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Umar, Khairuddin Zainuddin, Yuri P.Utami, and Muhammad A.Y. Ardi. "Antimalarial Activity of Ethanol Extract of Sampare Leaves (Glochidion sp var. Biak) Against Plasmodium falciparum In Vitro." Indonesian Journal of Pharmaceutical Science and Technology 10, no. 1 (2023): 10. http://dx.doi.org/10.24198/ijpst.v10i1.29477.

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Malaria is one of the health problems in the world and in Indonesia. This disease is caused by theprotozoan parasite, the genus Plasmodium. Plasmodium falciparum is the most important parasiticdisease with high morbidity and mortality in the world and tropical countries such as Indonesia inparticular. This study aims to determine the activity of sampare leaf extract in inhibiting the growth ofthe FCR-3 strain of the P. falciparum parasite which causes malaria. Sampare leaves were extracted bymaceration method using 70% ethanol. Extract the results extraction were tested against P. falciparumby in vitro method. 32,67% Yield extract resulted from the extraction process. Phytochemical screeningshows the presence of alkaloid compounds, flavonoids, quinones, saponins, and tannins. The ethanolextract of sampare leaves had IC50 antimalarial activity of 0,125 μg/mL and was categorized very well.Keywords: Antimalarial, Glochidion sp var. Biak, IC50, Plasmodium falciparum.

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Ibraheem, Zaid O., R. Abd Majid, S. Mohd Noor, H. Mohd Sedik, and R. Basir. "Role of Different Pfcrt and Pfmdr-1 Mutations in Conferring Resistance to Antimalaria Drugs in Plasmodium falciparum." Malaria Research and Treatment 2014 (November 11, 2014): 1–17. http://dx.doi.org/10.1155/2014/950424.

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Emergence of drugs resistant strains of Plasmodium falciparum has augmented the scourge of malaria in endemic areas. Antimalaria drugs act on different intracellular targets. The majority of them interfere with digestive vacuoles (DVs) while others affect other organelles, namely, apicoplast and mitochondria. Prevention of drug accumulation or access into the target site is one of the mechanisms that plasmodium adopts to develop resistance. Plasmodia are endowed with series of transporters that shuffle drugs away from the target site, namely, pfmdr (Plasmodium falciparum multidrug resistance transporter) and pfcrt (Plasmodium falciparum chloroquine resistance transporter) which exist in DV membrane and are considered as putative markers of CQ resistance. They are homologues to human P-glycoproteins (P-gh or multidrug resistance system) and members of drug metabolite transporter (DMT) family, respectively. The former mediates drifting of xenobiotics towards the DV while the latter chucks them outside. Resistance to drugs whose target site of action is intravacuolar develops when the transporters expel them outside the DVs and vice versa for those whose target is extravacuolar. In this review, we are going to summarize the possible pfcrt and pfmdr mutation and their role in changing plasmodium sensitivity to different anti-Plasmodium drugs.

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Dissertations / Theses on the topic "Plasmodium falciparum"

1

Krishna, S. "Cation atpases in Plasmodium falciparum." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.293442.

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Holland, Zoe. "Plasmodium falciparum protein kinase CK2." Thesis, University of Glasgow, 2008. http://theses.gla.ac.uk/606/.

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Malaria, caused by infection with intracellular protozoan parasites of the genus Plasmodium, is responsible for 300 to 600 million clinical cases annually (Snow et al., 2005), resulting in the deaths of up to three million people every year (Breman, 2001, Breman et al., 2004). There is a clear need for further research aimed at identifying novel drug targets (Ridley, 2002). Reversible phosphorylation of proteins is a major regulatory mechanism in most cellular processes, and protein kinases are considered promising drug targets, comprising as much as 30% of all protein targets under investigation (Cohen, 2002). The divergences between human and plasmodial protein kinases suggest that specific inhibition of the latter is an achievable goal (Doerig, 2004, Doerig and Meijer, 2007). This study investigates protein kinase CK2 of Plasmodium falciparum, seeking to establish by reverse genetics and biochemical approaches whether it represents a possible antimalarial drug target. Protein-kinase CK2, formerly known as Casein Kinase II, is a dual-specificity (Serine/Threonine and Tyrosine) protein kinase ubiquitously expressed in eukaryotes. It has over 300 cellular substrates catalogued to date (Meggio and Pinna, 2003). Consistent with its multiple substrates, the enzyme plays a crucial role in many cellular processes, and is essential to viability in yeast and slime mould (Padmanabha et al., 1990, Kikkawa et al., 1992). The human CK2 holoenzyme consists of two catalytic a or a’ subunits and two regulatory b subunits, and recent evidence indicates that the latter interact with several protein kinases in addition to CK2a (reviewed in (Bibby and Litchfield, 2005)), pointing to a likely role in the integration of numerous signalling pathways. A putative CK2a orthologue and two predicted CK2b subunits were identified in the P. falciparum genome (Ward et al., 2004, Anamika et al., 2005). Here we present the biochemical characterisation of the PfCK2a orthologue and both PfCK2b orthologues, and demonstrate by using a reverse genetics approach that each of the three subunits is essential for completion of the erythrocytic asexual cycle of the parasite, thereby validating the enzyme as a possible drug target. Recombinant PfCK2a possesses protein kinase activity, exhibits similar substrate and co-substrate preferences to those of CK2a subunits from other organisms, and interacts with both of the PfCK2b subunits in vitro. PfCK2a is amenable to inhibitor screening, and we report differential susceptibility between the human and P. falciparum CK2a enzymes to a small molecule inhibitor. Taken together, the data indicate that PfCK2a is an attractive, validated target for antimalarial chemotherapeutic intervention.

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Patzewitz, Eva-Maria. "Glutathione metabolism of Plasmodium falciparum." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/913/.

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Apicomplexan parasites of the genus Plasmodium are the causative agent of malaria, one of the most prevalent infectious diseases worldwide. Five different Plasmodium species can cause malaria in humans, leading to a total of approximately 500 million cases each year and of these, P. falciparum causes the most deadly form of the disease and is responsible for more than 1 million deaths annually. A major problem in the global fight against malaria is the widespread resistance of the parasites against the currently available drugs. It is of great importance to identify new drug target as well as to understand the mechanisms that lead to drug resistance in the first instance in order to potentially reverse the resistant phenotypes and to avoid the development of resistance in the future. The tripeptide glutathione (GSH) or γ-glutamylcysteinyl-glycine is the most abundant low molecular weight thiol in most eukaryotic organisms and serves a number of important functions as sulfhydryl-buffer, cofactor for enzymes and for the detoxification of xenobiotics and drugs. GSH is an important component of the antioxidant machinery and because malaria parasites live in an environment rich in iron and oxygen and thus increased oxidative stress, they depend on functional antioxidant systems. The biosynthesis pathway for GSH, consisting of γ-glutamylcysteine synthetase (γGCS) and glutathione synthetase (GS) is present in malaria parasites as well as in their host cells. Previous studies have shown that depletion of GSH has an antimalarial effect, but it remained unclear whether parasites were killed directly or died because their host cell could not survive the depletion of GSH. To address this question, the knockout of both genes encoding the enzymes of the GSH biosynthesis pathway in P. falciparum was attempted. While both gene loci were targeted by control constructs, the knockout of either pfγgcs or pfgs was impossible, indicating both genes are essential for parasite survival in the erythrocytic stages. To analyse the localization of γGCS and GS, GFP-tagged recombinant fusion proteins were expressed in the parasites and showed that GSH biosynthesis is cytosolic. Apart form its other functions GSH has previously been suggested to be involved in resistance to the antimalarial drug chloroquine (CQ). CQ was for a long time the first line antimalarial drug due to its high efficiency, low cost and low toxicity, but is now widely inefficient in the treatment of the disease. CQ resistance is associated with mutations in the CQ resistance transporter (PfCRT), a membrane protein of the digestive vacuole that allows the efflux of the drug form its site of action. However, PfCRT mutations alone cannot explain the full array of phenotypes found in resistant parasites. GSH is able to degrade heme, the target of CQ, in vitro and it has been suggested that elevated GSH levels contribute to CQ resistance. However, analyses of isogenic parasite lines bearing different forms of PfCRT in this study revealed lower GSH levels and higher susceptibility to inhibition of GSH biosynthesis in the CQ resistant lines. These changes did not correlate with changes in the expression of enzymes involved in the de novo biosynthesis or consumption of GSH. However, the cellular accumulation ratio for CQ indicated a decrease of free heme in the resistant parasites. Mutant forms of PfCRT expressed in oocytes of Xenopus laevis were able to transport GSH, while the sensitive wild-type form did not transport the tripeptide. The findings of this study suggest that in parasites bearing mutant PfCRT, GSH is transported into the digestive vacuole where it is able to contribute to resistance by degrading heme, before the tripeptide itself is degraded by peptidases inside the vacuole, consistent with the overall reduction of GSH levels in CQ resistant parasites.

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Quashie, Neils Benjamin. "Purine transport in plasmodium falciparum." Thesis, Thesis restricted. Connect to e-thesis to view abstract, 2008. http://theses.gla.ac.uk/165/.

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Thesis (Ph.D.) -- University of Glasgow, 2008.<br>Ph.D. thesis submitted to the Division of Infection and Immunity, Institute of Biomedical and Life Sciences, University of Glasgow, 2008. Includes bibliographical references. Print version also available.

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Jones, Matthew L. "Erythrocyte invasion by Plasmodium falciparum." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2009. https://www.mhsl.uab.edu/dt/2009r/jonesm.pdf.

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Vetelet, Philippe. "Paludisme grave à plasmodium falciparum." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25294.

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Mejia, Pedro. "Amélioration et utilisation d'un modèle murin des stades érythrocytaires du paludisme humain." Rouen, 2006. http://www.theses.fr/2006ROUES062.

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Paul, Richard E. L. "The genetic diversity of Plasmodium falciparum." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318788.

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Hayward, Rhian Elizabeth. "The biology of Plasmodium falciparum gametocytes." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.360296.

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Beveridge, Susan Elizabeth. "Metabolite transport pathways of Plasmodium falciparum." Thesis, University of Liverpool, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.590055.

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Metabolite transport pathways of the malaria parasite, Plasmodium falciparum, are an important area for study in order to further the understanding of the parasite's biology. Identification and characterisation of the transporters involved in these pathways may also provide potential novel drug targets or drug delivery mechanisms. This is especially valuable as chemotherapy remains one of the main management strategies in the fight against malaria and the usefulness 0 f the current range 0 f antimalarial drugs is seriously threatened by the emergence and spread of resistance . .' In this thesis the Xenopus laevis oocyte heterologous expression system was used to functionally characterise a gene-specific cDNA library of 48 putative membrane proteins and the previously annotated putative amino acid transporter PFF1430c for the uptake of several amino acids. This screening failed to identify any definite amino acid transport by the cDNA library or PFF1430c, however this could have been due to the fact that uptake of a relatively narrow range of amino acids was tested and these were used at concentrations lower than found physiologically. Inherent issues with the X laevis expression system may also have been an issue, including the expression of endogenous transporters for the substrates being investigated.

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Books on the topic "Plasmodium falciparum"

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Costanzo, Marna Schutte. Constraints and trade-offs in enzyme evolution of Plasmodium falciparum. Harvard University, 2010.

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Tarique, Mohammed. Drug Targets for Plasmodium Falciparum: Historic to Future Perspectives. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-19-4484-0.

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Murithi, James Muriungi. Dissecting the mechanisms of antiplasmodial resistance in Plasmodium falciparum. [publisher not identified], 2021.

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Golightly, Edmond Kwashie Odartey. Interaction between nutritional deficiencies and Plasmodium Falciparum malaria in the Gambia. Brunel University, 1988.

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Iqbal, Jamshaid. Erythrocyte membrane associated Plasmodium falciparum antigens, involvement in immunity and sequestration. Department of Immunology, the Wenner-Gren Institute, Stockholm University, 1994.

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Chu, Yen. Rheological beheaviour[sic] of erythrocyte rosettes induced by Plasmodium falciparum in flow. University of Birmingham, 1995.

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Patel, Samir. CD36-mediated clearance of Plasmodium falciparum-infected erythrocytes by rodent monocytes/macrophages. National Library of Canada, 2003.

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Scanfeld, Daniel. Exploring the Plasmodium falciparum Transcriptome Using Hypergeometric Analysis of Time Series (HATS). [publisher not identified], 2013.

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Verrall, B. Expresssion of the TATA box binding protein of plasmodium falciparum in heterologous systems. UMIST, 1997.

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Lee, Andrew Hojin. Investigating mutability and the plasmodium falciparum chloroquine resistance transporter in drug resistant malaria parasites. [publisher not identified], 2016.

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Book chapters on the topic "Plasmodium falciparum"

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Keenihan, Sarah Hudson, Robert Gramzinksi, Sutanti Ratiwayanto, et al. "Plasmodium Falciparum." In Tropical Diseases. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0059-9_7.

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Ringelmann, R., and Beate Heym. "Plasmodium falciparum." In Parasiten des Menschen. Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85397-5_72.

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Mehlhorn, Heinz. "Plasmodium falciparum: Endothelial Receptors." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4778.

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Mehlhorn, Heinz. "Plasmodium falciparum: Phylogenetic Origins." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4782.

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Mehlhorn, Heinz. "Plasmodium falciparum: Endothelial Receptors." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4778-1.

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Mehlhorn, Heinz. "Plasmodium falciparum: Phylogenetic Origins." In Encyclopedia of Parasitology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4782-1.

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Rug, Melanie, and Alexander G. Maier. "Transfection of Plasmodium falciparum." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-026-7_6.

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Noor, Rana. "Pathogenesis of Plasmodium falciparum." In Drug Targets for Plasmodium Falciparum: Historic to Future Perspectives. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-19-4484-0_2.

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Petter, Michaela, and Michael F. Duffy. "Antigenic Variation in Plasmodium falciparum." In Results and Problems in Cell Differentiation. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20819-0_3.

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Cohen, Scott J. "Plasmodium Falciparum Malaria in Children." In Updates in Emergency Medicine. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0155-8_10.

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Conference papers on the topic "Plasmodium falciparum"

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Verma, Ved Vrat, Yugandhar P. Reddy, Aditi Tomar, Chetan Kumar Sharma, and Varun Kumar Sharma. "Computational Approaches Based Evolutionary Analysis to Identify the Genetic Variation Among Kelch 13 Gene Linked to Artemisinin Resistance in Plasmodium Falciparum." In 2025 International Conference on Cognitive Computing in Engineering, Communications, Sciences and Biomedical Health Informatics (IC3ECSBHI). IEEE, 2025. https://doi.org/10.1109/ic3ecsbhi63591.2025.10991235.

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Quevedo, Viviana, and Bernabe Ortega-Tenezaca. "IF for the dataset of Plasmodium Falciparum." In MOL2NET 2019, International Conference on Multidisciplinary Sciences, 5th edition. MDPI, 2019. http://dx.doi.org/10.3390/mol2net-05-06253.

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Borges, Fabiana Bazaga, Kamila Cristina Silva, Maria Inês de Souza, et al. "Diagnóstico laboratorial de infecção mista por Plasmodium falciparum e Plasmodium vivax: importância do treinamento técnico para leitura da gota espessa." In Resumos do 56º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2024. https://doi.org/10.5327/1516-3180.142s1.10713.

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Objetivo: O diagnóstico da malária em áreas nas quais coexistem as espécies de Plasmodium falciparum e vivax merece especial atenção devido à possibilidade de infecções mistas (espécies diferentes no exame de sangue, não necessariamente na mesma coleta). O objetivo deste trabalho é mostrar a importância do treinamento técnico de excelência para a leitura da “gota espessa”, por meio de um diagnóstico de infecção mista por Plasmodium, realizado pelo laboratório de parasitologia clínica. Método: Um paciente, gênero masculino, 27 anos, foi admitido no serviço com dor abdominal, dor lombar, inapetência e febre há 5 dias. Na anamnese, informou viagem recente à Venezuela e histórico de sete infecções prévias por Plasmodium. A amostra de sangue total foi encaminhada ao laboratório para realização dos exames de triagem: teste imunocromatográfico, gota espessa e esfregaço sanguíneo, corados com giemsa. Conclusão: No teste imunocromatográfico, foi observada positividade na linha T3, específica para Plasmodium vivax. No exame de gota espessa, observou-se parasitemia de formas em anéis (trofozoítos jovens) e gametócitos de Plasmodium vivax, associada a formas irregulares (trofozoítos maduros) e esquizontes, com granulações de Schuffner bem visíveis, em maior grau de parasitemia. Dessa forma, o diagnóstico da triagem foi infecção por Plasmodium vivax. Na segunda amostra, (6º dia, lâmina de verificação de cura), foram visualizadas formas em anel, em vírgula e sem granulações de Schuffner. O teste imunocromatográfico foi realizado novamente, mostrando positividade para Plasmodium falciparum (T2). Aqui, observamos um caso no qual a parasitemia de vivax na triagem ocultou a presença de falciparum, que, quando observado na lâmina de controle de cura, mudou a conduta do tratamento. Diante disso, gostaríamos de evidenciar a importância do treinamento técnico de excelência para a leitura da gota espessa, primordial para a decisão da conduta clínica, bem como o comparecimento do paciente para o acompanhamento do tratamento.

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Cai, Hong, Maribel Sanchez, Yufeng Wang, and Jianying Gu. "Putative Cell Cycle Related Genes in Plasmodium Falciparum." In 2009 International Joint Conference on Bioinformatics, Systems Biology and Intelligent Computing. IEEE, 2009. http://dx.doi.org/10.1109/ijcbs.2009.44.

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Evans, Krystal J., Dedreia Tull, Michael C. Hewitt, Malcolm J. McConville, Peter H. Seeberger, and Louis Schofield. "PURIFICATION OF THE GLYCOSYLPHOSPHATIDYLINOSITOL TOXIN OF PLASMODIUM FALCIPARUM." In XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.690.

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Silva, Kamila Cristina, Fabiana Bazaga Borges, Rodrigo Nogueira Angerami, Mariângela Ribeiro Resende, Daniela Maira Cardoso, and Mariangela Ribeiro Resende. "Importância do diagnóstico preciso na malária: um caso de Plasmodium ovale diagnosticado por microscopia de “gota espessa”." In Resumos do 56º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2024. https://doi.org/10.5327/1516-3180.142s1.11025.

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Objetivo: A malária é uma doença infecciosa febril aguda, causada por protozoários do gênero Plasmodium, transmitidos pela picada da fêmea do mosquito do gênero Anopheles. No Brasil, as espécies de Plasmodium vivax, falciparum e malariae apresentam maior prevalência. Já a espécie ovale tem uma prevalência muito baixa no país e será investigada caso o paciente tenha estado em áreas endêmicas, localizadas em determinadas regiões do continente africano. O objetivo do presente trabalho foi destacar a importância do exame parasitológico da “gota espessa” como teste para o diagnóstico diferencial na detecção do Plasmodium ovale no Brasil. Método: Um paciente, sexo masculino, 39 anos, proveniente de área endêmica, foi recebido no pronto atendimento com os sintomas de cefaleia, alteração do estado mental, e com histórico de infecção prévia de malária. A amostra de sangue total foi encaminhada ao Laboratório de Patologia Clínica e submetida à análise por três diferentes metodologias: teste imunocromatográfico, gota espessa e esfregaço sanguíneo corado com giemsa. Conclusão: O resultado do teste imunocromatográfico mostrou positividade para a linha T2 (Plasmodium falciparum). Porém, na visualização da gota espessa, foram identificados parasitas semelhantes ao Plasmodium vivax, no entanto com presença de numerosos pigmentos maláricos e formas menores de trofozoítos e gametócitos. Diante dos achados laboratoriais na gota espessa, foi possível concluir que o paciente estava infectado pelo Plasmodium da espécie ovale, demonstrando, assim, a importância de um correto diagnóstico laboratorial, primordial para um rápido e correto início de tratamento.

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Yumang, Analyn N., Ericson D. Dimaunahan, Jennifer Dela Cruz, Geraldo C. Talisic, Gabriel Avelino R. Sampedro, and Maricor N. Soriano. "Real-Time Plasmodium Falciparum Parasitemia using Natural Neighbor Interpolation." In 2018 IEEE 10th International Conference on Humanoid, Nanotechnology, Information Technology,Communication and Control, Environment and Management (HNICEM). IEEE, 2018. http://dx.doi.org/10.1109/hnicem.2018.8666280.

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Rocha, Maura G. R., Rodrigo M. S. Veras, Maíla L. Claro, Laurindo S. Britto Neto, and Kelson R. T. Aires. "Análise Comparativa de Versões YOLO na Detecção e Identificação de Parasitas da Malária." In Simpósio Brasileiro de Computação Aplicada à Saúde. Sociedade Brasileira de Computação - SBC, 2021. http://dx.doi.org/10.5753/sbcas.2021.16066.

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A malária é uma doença endêmica causada pelo parasita Plasmodium que pode ser fatal em muitas regiões do mundo. Alguns pesquisadores estão utilizando conceitos de aprendizagem de máquina para detectar e classiﬁcar células infectadas pelo parasita Plasmodium. Este trabalho apresenta um estudo comparativo de três versões recentes da rede neural convolucional You Only Look Once (YOLO), são elas a: YOLOv4, Scaled-YOLOv4 e YOLOv5. Foi utilizado a base de dados MP-IDB que possui 210 imagens com o parasita Plasmodium. Os modelos alcançaram excelentes resultados, tendo o melhor resultado com mAP e precisão de 94,8% e 93,3%, respectivamente, para a classiﬁcação em dois tipos de espécies do Plasmodium falciparum e vivax.

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Fogel, Gary B., Carla Islas, and David Hecht. "Modeling the evolution of drug resistance in Plasmodium falciparum DHFR." In 2013 IEEE Congress on Evolutionary Computation (CEC). IEEE, 2013. http://dx.doi.org/10.1109/cec.2013.6557572.

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Setianingrum, Anif Hanifa, Luh Kesuma Wardhani, A. Faisal Ridwan, and Silvia F. Nasution. "Identification of Plasmodium falciparum Stages Using Support Vector Machine Method." In 2019 7th International Conference on Cyber and IT Service Management (CITSM). IEEE, 2019. http://dx.doi.org/10.1109/citsm47753.2019.8965413.

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Reports on the topic "Plasmodium falciparum"

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สืบหลินวงศ์, ธาดา, та จุฑาพันธุ์ พิณสวัสดิ์. รูปแบบของโปรตีนใน Plasmodium falciparum โดยอิเล็คโตรฟอรีซิสสองมิติ : รายงานวิจัยฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 1988. https://doi.org/10.58837/chula.res.1988.10.

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โปรตีนในแต่ละเซลล์จะถูกสังเคราะห์ขึ้นอย่างรัดกุม มีการควบคุมในระดับยีน จึงมีคุณสมบัติเฉพาะตัวสำหรับแต่ละเซลล์หรือแต่ละชนิดของสัตว์และพืช ดังนั้น การศึกษารูปแบบของโปรตีนซึ่งถือเป็น gene product จึงอาจใช้เป็น marker ของเซลล์หรือสัตว์แต่ละชนิดได้ วัตถุประสงค์ของการวิจัยครั้งนี้เพื่อนำรูปแบบโปรตันซึ่งศึกษาโดยเทคนิค 2-D electrophoresis มาเป็น marker ของแต่ละสายพันธุ์บริสุทธิ์ของ P.falciparum โดยได้ศึกษารูปแบบโปรตีนของเม็ดเลือดแดงที่ติดเชื้อ P.falciparum ในตัวอย่างที่เป็น isolate และสายพันธุ์บริสุทธิ์รวม 70 ตัวอย่าง และศึกษาโปรตีนใน P. falciparum โดยใช้ [superscript 3 5]S-methionine incorporation, 2-D electrophoresis และ autoradiography และ autoradiography นั้น ไวกว่าการย้อมด้วย Coomassie blue R โดยแยกโปรตีนของ P. falciparum ออกเป็น 14 กลุ่มและแต่ละกลุ่มยังแยกย่อย ๆออกไปอีก 1-7 ชนิดย่อย และพบว่า รูปแบบโปรตีนใน P. falciparum ซึ่งเลี้ยงในห้องปฏิบัติการเป็นเวลานานกว่า 3 ปี ยังคงที่ จึงสรุปว่าความแตกต่างในรูปแบบโปรตีนของ P. falciparum สามารถใช้เป็นเครื่องแสดงลักษณะเฉพาะประจำตัวปรสิตได้

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Rossan, Richard N. Drug Evaluation in the Plasmodium Falciparum-Aotus Model. Defense Technical Information Center, 1989. http://dx.doi.org/10.21236/ada210494.

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Rossan, Richard N. Drug Evaluation in the Plasmodium Falciparum - Aotus Model. Defense Technical Information Center, 1986. http://dx.doi.org/10.21236/ada211392.

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Rossan, Richard N. Drug Evaluation in the Plasmodium falciparum - Aotus Model. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada232854.

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Wirth, Dyann F. New Strategies for Drug Discovery and Development for Plasmodium Falciparum. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada375802.

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Piper, Robert C. Parasite Lactate Dehydrogenase for Diagnosis of Plasmodium Falciparum. Phase II. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/adb230017.

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Obaldia, Nicanor, and III. Drug and Vaccine Evaluation in the Human-Aotus Plasmodium Falciparum Model. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada405377.

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Rollwagen, Florence M., Nancy D. Pacheco, Jr Wistar, and Richard. Proliferative Responses of Mice to a Cloned Plasmodium Falciparum Sporozoite Antigen. Defense Technical Information Center, 1988. http://dx.doi.org/10.21236/ada205098.

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Makler, Michael T. Method for Evaluation of Drug Resistance for Plasmodium Falciparum. Phase 2. Defense Technical Information Center, 1991. http://dx.doi.org/10.21236/adb158380.

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Obaldia, III, and Nocanor. Drug and Vaccine evaluation in the Human Aotus Plasmodium falciparum Model. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada547285.

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