Relevant bibliographies by topics / Plasmodium parasites

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Plasmodium parasites'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 July 2025

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Journal articles on the topic "Plasmodium parasites"

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HANADA, Kentaro, Toshihide MITAMURA, Masayoshi FUKASAWA, Pamela A. MAGISTRADO, Toshihiro HORII, and Masahiro NISHIJIMA. "Neutral sphingomyelinase activity dependent on Mg2+ and anionic phospholipids in the intraerythrocytic malaria parasite Plasmodium falciparum." Biochemical Journal 346, no. 3 (2000): 671–77. http://dx.doi.org/10.1042/bj3460671.

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Sphingolipid metabolism and metabolites are important in various cellular events in eukaryotes. However, little is known about their function in plasmodial parasites. Here we demonstrate that neutral sphingomyelinase (SMase) involved in the sphingomyelin (SM) catabolism is retained by the intraerythrocytic parasite Plasmodium falciparum. When assayed in a neutral pH buffer supplemented with Mg2+ and phosphatidylserine, an activity for the release of the phosphocholine group from SM was detected in parasite-infected, but not in uninfected, erythrocyte ghosts. The SMase activity in the parasite-
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Sá e Cunha, Cláudia, Britta Nyboer, Kirsten Heiss, et al. "Plasmodium berghei EXP-1 interacts with host Apolipoprotein H during Plasmodium liver-stage development." Proceedings of the National Academy of Sciences 114, no. 7 (2017): E1138—E1147. http://dx.doi.org/10.1073/pnas.1606419114.

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The first, obligatory replication phase of malaria parasite infections is characterized by rapid expansion and differentiation of single parasites in liver cells, resulting in the formation and release of thousands of invasive merozoites into the bloodstream. Hepatic Plasmodium development occurs inside a specialized membranous compartment termed the parasitophorous vacuole (PV). Here, we show that, during the parasite’s hepatic replication, the C-terminal region of the parasitic PV membrane protein exported protein 1 (EXP-1) binds to host Apolipoprotein H (ApoH) and that this molecular intera
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Jaramillo Ponce, José R., and Magali Frugier. "Plasmodium, the Apicomplexa Outlier When It Comes to Protein Synthesis." Biomolecules 14, no. 1 (2023): 46. http://dx.doi.org/10.3390/biom14010046.

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Plasmodium is an obligate intracellular parasite that has numerous interactions with different hosts during its elaborate life cycle. This is also the case for the other parasites belonging to the same phylum Apicomplexa. In this study, we bioinformatically identified the components of the multi-synthetase complexes (MSCs) of several Apicomplexa parasites and modelled their assembly using AlphaFold2. It appears that none of these MSCs resemble the two MSCs that we have identified and characterized in Plasmodium. Indeed, tRip, the central protein involved in the association of the two Plasmodiu
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Kalanon, Ming, and Geoffrey I. McFadden. "Malaria, Plasmodium falciparum and its apicoplast." Biochemical Society Transactions 38, no. 3 (2010): 775–82. http://dx.doi.org/10.1042/bst0380775.

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Malaria, which is caused by species of the parasite genus Plasmodium, remains a major global health problem. A vestigial plastid homologous with the chloroplasts of plants and algae was discovered in malaria and related parasites from the phylum Apicomplexa and has radically changed our view of the evolutionary origins of these disease-causing protists. We now recognize that this large group of parasites had a photosynthetic ancestry and were converted into parasitism early in the evolution of animals. Apicomplexans have probably been parasitizing the animal kingdom for more than 500 million y
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Perkins, Susan L., and Jos J. Schall. "A molecular phylogeny of malarial parasites recovered from cytochrome b gene sequences." Journal of Parasitology 88, no. 5 (2002): 972–78. https://doi.org/10.5281/zenodo.13524290.

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(Uploaded by Plazi for the Bat Literature Project) A phylogeny of haemosporidian parasites (phylum Apicomplexa, family Plasmodiidae) was recovered using mitochondrial cytochrome b gene sequences from 52 species in 4 genera (Plasmodium, Hepatocystis, Haemoproteus, and Leucocytozoon), including parasite species infecting mammals, birds, and reptiles from over a wide geographic range. Leucocytozoon species emerged as an appropriate out-group for the other malarial parasites. Both parsimony and maximum-likelihood analyses produced similar phylogenetic trees. Life-history traits and parasite morpho
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Perkins, Susan L., and Jos J. Schall. "A molecular phylogeny of malarial parasites recovered from cytochrome b gene sequences." Journal of Parasitology 88, no. 5 (2002): 972–78. https://doi.org/10.5281/zenodo.13524290.

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(Uploaded by Plazi for the Bat Literature Project) A phylogeny of haemosporidian parasites (phylum Apicomplexa, family Plasmodiidae) was recovered using mitochondrial cytochrome b gene sequences from 52 species in 4 genera (Plasmodium, Hepatocystis, Haemoproteus, and Leucocytozoon), including parasite species infecting mammals, birds, and reptiles from over a wide geographic range. Leucocytozoon species emerged as an appropriate out-group for the other malarial parasites. Both parsimony and maximum-likelihood analyses produced similar phylogenetic trees. Life-history traits and parasite morpho
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Perkins, Susan L., and Jos J. Schall. "A molecular phylogeny of malarial parasites recovered from cytochrome b gene sequences." Journal of Parasitology 88, no. 5 (2002): 972–78. https://doi.org/10.5281/zenodo.13524290.

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(Uploaded by Plazi for the Bat Literature Project) A phylogeny of haemosporidian parasites (phylum Apicomplexa, family Plasmodiidae) was recovered using mitochondrial cytochrome b gene sequences from 52 species in 4 genera (Plasmodium, Hepatocystis, Haemoproteus, and Leucocytozoon), including parasite species infecting mammals, birds, and reptiles from over a wide geographic range. Leucocytozoon species emerged as an appropriate out-group for the other malarial parasites. Both parsimony and maximum-likelihood analyses produced similar phylogenetic trees. Life-history traits and parasite morpho
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Perkins, Susan L., and Jos J. Schall. "A molecular phylogeny of malarial parasites recovered from cytochrome b gene sequences." Journal of Parasitology 88, no. 5 (2002): 972–78. https://doi.org/10.5281/zenodo.13524290.

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Abstract:
(Uploaded by Plazi for the Bat Literature Project) A phylogeny of haemosporidian parasites (phylum Apicomplexa, family Plasmodiidae) was recovered using mitochondrial cytochrome b gene sequences from 52 species in 4 genera (Plasmodium, Hepatocystis, Haemoproteus, and Leucocytozoon), including parasite species infecting mammals, birds, and reptiles from over a wide geographic range. Leucocytozoon species emerged as an appropriate out-group for the other malarial parasites. Both parsimony and maximum-likelihood analyses produced similar phylogenetic trees. Life-history traits and parasite morpho
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Tewari, Rita, Solabomi A. Ogun, Ruwani S. Gunaratne, Andrea Crisanti, and Anthony A. Holder. "Disruption of Plasmodium berghei merozoite surface protein 7 gene modulates parasite growth in vivo." Blood 105, no. 1 (2005): 394–96. http://dx.doi.org/10.1182/blood-2004-06-2106.

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Abstract Merozoite invasion of red blood cells is crucial to the development of the parasite that causes malaria. Merozoite surface proteins (MSPs) mediate the first interaction between parasite and erythrocyte. In Plasmodium falciparum, they include a complex of products from at least 3 genes (msp1, msp6, and msp7), one of which, msp7, is part of a gene family containing 3 and 6 adjacent members in Plasmodium yoelii and Plasmodium falciparum, respectively. We have identified and disrupted msp7 in the Plasmodium berghei gene family. The protein is expressed in schizonts and colocalizes with MS
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Morgan, D. M. L., U. Bachrach, Y. G. Assaraf, E. Harari, and J. Golenser. "The effect of purified aminoaldehydes produced by polyamine oxidation on the development in vitro of Plasmodium falciparum in normal and glucose-6-phosphate-dehydrogenase-deficient erythrocytes." Biochemical Journal 236, no. 1 (1986): 97–101. http://dx.doi.org/10.1042/bj2360097.

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Purified aminoaldehydes produced by polyamine oxidation were toxic to the malarial parasite, Plasmodium falciparum, cultured in human erythrocytes. There was a profound effect on young ring forms, and, during maturation, parasites became more sensitive to the aldehydes. Oxidation of the aldehydes abolished the lethal effect. The plasmodia within glucose-6-phosphate-dehydrogenase (G6PD)-deficient erythrocytes were more sensitive to mono- and di-aldehydes than were parasites in normal erythrocytes. G6PD-deficient erythrocytes were also more sensitive to pretreatment with the dialdehyde produced
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Dissertations / Theses on the topic "Plasmodium parasites"

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Enweji, Nizar. "Dynamics of Resistant Plasmodium falciparum Parasites." Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-230224.

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Persistence of drug resistant Plasmodium falciparum is a major problem to management and control malaria in endemic areas. The focus of this thesis was to study the dynamics of resistant P. falciparum parasites. The study was performed in two African countries: 1) Sudan: Asar village in eastern Sudan, here we examined the persistence of drug sensitive and resistant P. falciparum genotypes among individuals with single-clone and multiple clones infection during the dry season. We genotyped microsatellite loci in the vicinity of the dihydrofolate reductase gene (dhfr) and the dihydropteroate syn
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Raja, Amber Iftikhar. "Characterisation of a Chemically Attenuated Blood-Stage Malaria Vaccine for the Rodent Parasite, Plasmodium yoelii." Thesis, Griffith University, 2016. http://hdl.handle.net/10072/366436.

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The development of a vaccine is essential for the elimination and eventual eradication of malaria. However, despite many years of effort, a successful malaria vaccine has not yet been achieved. Many of the subunit vaccine candidates tested in clinical trials have provided limited efficacy; therefore, efforts to develop an effective vaccine against malaria need to continue. The development of a whole parasite blood-stage vaccine would maximise the number of blood-stage antigens presented to the immune system, including conserved parasite antigens. Chemical treatment has been used to develop att
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Chappell, Lia Victoria Louise. "Novel approaches for transcriptome analysis in Plasmodium parasites." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648817.

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Hogg, Jonathan C. "Effects of Plasmodium infection on anopheline mosquito fecundity." Thesis, Keele University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.261487.

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Porter, Heidi S. "The effect of febrile temperature on Plasmodium falciparum /." Diss., CLICK HERE for online access, 2007. http://contentdm.lib.byu.edu/ETD/image/etd2225.pdf.

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Tay, Chwen Ling. "The targets and role of palmitoylation in Plasmodium parasites." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648641.

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Engel, Jessica Alexandra. "Investigating Plasmodium falciparum Histone Deacetylase 1 Complex Proteins." Thesis, Griffith University, 2017. http://hdl.handle.net/10072/367801.

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Malaria causes substantial morbidity and mortality worldwide. Although there has been a considerable decline in global malaria incidence and mortality rates since 2000, it is estimated that more than 400,000 deaths occurred in 2015 as a result of this parasitic disease. The lack of a broadly effective licensed vaccine and the threat of malaria parasite resistance to current drugs means there is an urgent need for the development of new therapies with novel parasite targets. With a renewed call for global malaria eradication, novel therapeutic strategies are crucial to continue progress achieve
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Lyons, E. J. "Diversity, structure and population genetics of the human malaria parasites Plasmodium falciparum and Plasmodium vivax." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.270206.

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Silva, Mafalda Lopes da. "Dissecting the molecular interaction between hepatocytes and Plasmodium liver parasites." Doctoral thesis, Universidade Nova de Lisboa. Instituto de Tecnologia Química e Biológica, 2011. http://hdl.handle.net/10362/8572.

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Dissertation presented to obtain the Ph.D degree in Biology<br>Malaria is one of the world´s leading causes of death, responsible for over 700,000 deaths per year, the majority of which are African children under 5 years of age. Malaria disease is caused by the transmission of an Apicomplexa parasite, Plasmodium, through the bit of a female Anopheles mosquito, and transmitted parasites quickly reach the mammalian host liver, where the first round of replication begins. Plasmodium sporozoites, once inside the liver, must invade and survive within hepatocytes until the first replicative s
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Ben, Abderrazak Souha. "Variabilité génétique des populations de "Plasmodium falciparum"." Montpellier 2, 1993. http://www.theses.fr/1993MON20013.

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Nous avons analyse par electrophorese d'isoenzymes (7 a 12 loci) le polymorphisme genetique de 5 populations (113 stocks) du plasmodium falciparum, l'agent de la forme la plus severe de paludisme. Les resultats ont ete interpretes en termes de genetique des populations, dans le but d'analyser la structure des populations naturelles de ce parasite. Un fort desequilibre de liaison apparait dans trois des populations etudiees, ainsi que dans l'echantillon global. Une quatrieme population montre egalement des indices de desequilibre, quoique dans une moindre mesure. Une seule serie n'a montre aucu
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Books on the topic "Plasmodium parasites"

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P, Waters Andrew, and Janse Chris J, eds. Malaria parasites, genomes and molecular biology. Caister Academic Press, 2004.

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Lee, Andrew Hojin. Investigating mutability and the plasmodium falciparum chloroquine resistance transporter in drug resistant malaria parasites. [publisher not identified], 2016.

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Garnham, P. C. C. Catalogue of the Garnham collection of malaria parasites and other haemosporidia. Cambridge University Press, 1986.

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Garnham, P. C. C. Catalogue of the Garnham Collection of malaria parasites and other haemosporidia. The Wellcome Trust, 1986.

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Garnham, P. C. C. Catalogue of the Garnham Collection of malaria parasites and other haemosporidia. Cambridge University Press London, 1988.

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Vladimirovna, Beĭer Tamara, Lysenko A. I͡A︡, and Vsesoi͡u︡znoe obshchestvo protozoologov, eds. Mali͡a︡riĭnye parazity mlekopitai͡u︡shchikh. Izd-vo "Nauka," Leningradskoe otd-nie, 1986.

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Iqbal, M. P. Investigation of the TFIIB and fibrillarin genes in the human malaria parasite plasmodium falciparum. UMIST, 1996.

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C, Frontali, ed. Molecular events in Plasmodium life cycle and factors affecting mosquito/parasite relationships: EEC symposium held at the Istituto superiore di sanità, Rome, October 30-November 1, 1986 : proceedings. Istituto superiore di sanità, 1987.

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Falkard, Brie. Investigations into the Metabolic Requirements for Lipoic Acid and Lipid Species During the Life Cycle of the Malarial Parasite Plasmodium Berghei. [publisher not identified], 2013.

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Special Programme for Research and Training in Tropical Diseases, Foundation for Innovative New Diagnostics, and Centers for Disease Control and Prevention (U.S.), eds. Malaria rapid diagnostic test performance: Results of WHO product testing of malaria RDTs : round 2 (2009). World Health Organization on behalf of the Special Programme for Research and Training in Tropical Diseases, 2010.

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Book chapters on the topic "Plasmodium parasites"

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Mir, Snober S., Subir Biswas, and Saman Habib. "Targeting Apicoplast Pathways in Plasmodium." In Apicomplexan Parasites. Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527633883.ch9.

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Prasad, G. Sridhar, Nicholas D. P. Cosford, and Sailen Barik. "Plasmodium Hsp90 as an Antimalarial Target." In Apicomplexan Parasites. Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527633883.ch23.

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Usluca, Selma. "Malaria and Molecular Diagnosis." In Molecular Approaches in Medicine. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359524.2.

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It is an endemic vector-borne parasitic disease caused by protozoan parasites of the genus Plasmodium in tropical and subtropical regions worldwide. In each endemic area, malaria is transmitted by a specific set of Anopheles species. Plasmodium consists of over 200 species, infecting mammals, birds, and reptiles, and malaria parasites generally tend to be host-specific. Plasmodium falciparum, Plasmodium vivax, Plasmodium malariae, Plasmodium ovale, and Plasmodium knowlesi are the five known species of the genus Plasmodium that causes malaria in humans. Of the five Plasmodium species that cause
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Lee, Kim-Sung, and Indra Vythilingam. "Plasmodium knowlesi: Emergent Human Malaria in Southeast Asia." In Parasites and their vectors. Springer Vienna, 2013. http://dx.doi.org/10.1007/978-3-7091-1553-4_2.

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Davioud-Charvet, Elisabeth, and Don Antoine Lanfranchi. "Subversive Substrates of Glutathione Reductases from Plasmodium falciparum-Infected Red Blood Cells as Antimalarial Agents." In Apicomplexan Parasites. Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527633883.ch20.

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Sanchez, Cecilia P., Judith Pfahler, Hernando A. del Portillo, and Michael Lanzer. "Transient Transfection of Plasmodium vivax Blood-Stage Parasites." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-026-7_10.

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Cui, Liwang, Namtip Trongnipatt, Jetsumon Sattabongkot, and Rachanee Udomsangpetch. "Culture of Exoerythrocytic Stages of the Malaria Parasites Plasmodium falciparum and Plasmodium vivax." In Host-Pathogen Interactions. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-204-5_18.

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Bozdech, Zbynek, Sachel Mok, and Archna P. Gupta. "DNA Microarray-Based Genome-Wide Analyses of Plasmodium Parasites." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-026-7_13.

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Ward, Martin, and Giovanni Benelli. "Mosquitoes, Plasmodium Parasites, and Cancer: Where from, Where to?" In Parasitology Research Monographs. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94075-5_14.

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Frimpong, Augustina, Frederica Dedo Partey, and Michael Fokuo Ofori. "Identification and Enumeration of Plasmodium falciparum Parasites by Light Microscopy." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2189-9_1.

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Conference papers on the topic "Plasmodium parasites"

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K, Senthil, Yuktha Varshika J, Gopinath M, Pooja Sri V, Vishwa S, and Monikaa R. "Genomic Insights into the Classification and Identification of Plasmodium Parasites." In 2024 First International Conference on Innovations in Communications, Electrical and Computer Engineering (ICICEC). IEEE, 2024. https://doi.org/10.1109/icicec62498.2024.10808865.

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Kumar, V. Bhuvana, S. Sampoornamma, and M. Hymavathi. "Identifying Plasmodium Parasites in Blood Smears with Deep Learning Techniques." In 2025 2nd International Conference on Research Methodologies in Knowledge Management, Artificial Intelligence and Telecommunication Engineering (RMKMATE). IEEE, 2025. https://doi.org/10.1109/rmkmate64874.2025.11042660.

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S, Shashikiran, Srinivas Babu N, Sunitha HD, Sandip Mondal, Syed Sadaqathulla, and Pramod K S. "Plasmodium Vivax Parasite Detection using Transfer Learning Techniques." In 2024 Asia Pacific Conference on Innovation in Technology (APCIT). IEEE, 2024. http://dx.doi.org/10.1109/apcit62007.2024.10673683.

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Salem, Mostafa, Saad Abouzahir, Hosni Ghedira, Abdulmotaleb El Saddik, and Mohammad Yaqub. "PlasmoStage: A Hierarchical Deep Learning Framework for Plasmodium Parasite Staging in Malaria." In 2025 IEEE Medical Measurements & Applications (MeMeA). IEEE, 2025. https://doi.org/10.1109/memea65319.2025.11067957.

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Nugroho, Hanung Adi, A. Fatan D. Marsiano, Khampaserth Xaphakdy, et al. "Multithresholding Approach for Segmenting Plasmodium Parasites." In 2019 11th International Conference on Information Technology and Electrical Engineering (ICITEE). IEEE, 2019. http://dx.doi.org/10.1109/iciteed.2019.8929995.

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Nugroho, Hanung Adi, Julisa Bana Abraham, Aina Hubby Azzira, et al. "Performance of Convolutional Neural Network in Detecting Plasmodium Parasites." In 2019 IEEE 9th International Conference on System Engineering and Technology (ICSET). IEEE, 2019. http://dx.doi.org/10.1109/icsengt.2019.8906303.

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Kehrer, Jessica. "Clearing of hemozoin in Plasmodium parasites enables STED Nanoscopy." In European Microscopy Congress 2020. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.emc2020.703.

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Prananda, Alifia Revan, Hanung Adi Nugroho, and Igi Ardiyanto. "Enumeration of Plasmodium Parasites on Thin Blood Smear Digital Microscopic Images." In 2019 5th International Conference on Science in Information Technology (ICSITech). IEEE, 2019. http://dx.doi.org/10.1109/icsitech46713.2019.8987492.

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Aladago, Maxwell Mbailla, Lorenzo Torresani, and Elena V. Rosca. "Semantic Segmentation of the Growth Stages of Plasmodium Parasites using Convolutional Neural Networks." In 2019 IEEE AFRICON. IEEE, 2019. http://dx.doi.org/10.1109/africon46755.2019.9133937.

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Luther, Ed, and Howard M. Shapiro. "Use of DNA base pair composition for the specefic identification of malaria parasites (plasmodium)." In 2014 40th Annual Northeast Bioengineering Conference (NEBEC). IEEE, 2014. http://dx.doi.org/10.1109/nebec.2014.6972861.

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Reports on the topic "Plasmodium parasites"

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Piper, Robert C. Parasite Lactate Dehydrogenase for Diagnosis of Plasmodium Falciparum. Phase II. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/adb230017.

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Prachumsri, Jetsumon. Proteomic Study of Human Malaria Parasite Plasmodium Vivax Liver Stages for Development of Vaccines and Drugs. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada494445.

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McElwain, Terry, Eugene Pipano, Guy Palmer, Varda Shkap, Stephen Hines, and Douglas Jasmer. Protection of Cattle Against Babesiosis: Immunization with Recombinant DNA Derived Apical Complex Antigens of Babesia bovis. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7612835.bard.

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Bovine babesiosis caused by Babesia bovis continues to be a significant deterrent to global livestock production. Current control methods have both biological and technical drawbacks that have stimulated research on improved methods of vaccination. This BARD project has focused on characterization of candidate Babesia bovis vaccine antigens located in the apical complex, a unique group of subcellular organelles - including rhoptries, micronemes, and spherical bodies - involved in the invation of erythrocytes. Spherical bodies and rhoptries were partially purified and their contents characteriz
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