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1

Georg, Hertting, Spatz Hanns-Christof, and North Atlantic Treaty Organization. Scientific Affairs Division., eds. Modulation of synaptic transmission and plasticity in nervous systems. Berlin: Springer-Verlag, 1988.

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2

Hertting, Georg, and Hanns-Christof Spatz, eds. Modulation of Synaptic Transmission and Plasticity in Nervous Systems. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-73160-0.

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3

McMahon, Gale N., and Rich G. Buckner. Synaptic plasticity: Cell biology, regulation and role in disease. New York: Nova Biomedical, 2012.

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4

Synaptic plasticity: Dynamics, development and disease. Wien: Springer, 2012.

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5

Taupin, Philippe. The hippocampus: Neurotransmission and plasticity in the nervous system. New York: Nova Biomedical Books, 2007.

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6

The cerebellum: Brain for an implicit self. Upper Saddle River, N.J: FT Press, 2012.

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7

Nonsynaptic diffusion neurotransmission and late brain reorganization. New York: Demos, 1995.

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8

F, Kaiser Tim, and Peters Felix J, eds. Synaptic plasticity: New research. New York: Nova Science Publishers, 2008.

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9

Modulation Of Synaptic Transmission And Plasticity In Nervous Systems. Springer, 2012.

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10

Sala, Carlo, and Michael R. Kreutz. Synaptic Plasticity: Dynamics, Development and Disease. Springer, 2012.

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11

A, Wernig, ed. Plasticity of motoneuronal connections. Amsterdam: Elsevier, 1991.

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12

David, Evered, and Whelan Julie, eds. Plasticity of the neuromuscular system. Chichester: Wiley, 1988.

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13

Functional Plasticity and Genetic Variation: Insights into the Neurobiology of Alcoholism. Elsevier Science & Technology Books, 2010.

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14

Bates, David, and Nima Bassiri. Plasticity and Pathology: On the Formation of the Neural Subject. Fordham University Press, 2016.

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15

Plasticity and Pathology: On the Formation of the Neural Subject. Fordham University Press, 2016.

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16

Bates, David, and Nima Bassiri. Plasticity and Pathology: On the Formation of the Neural Subject. Fordham University Press, 2016.

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17

Kidd, Geoffrey. Understanding Neuromuscular Plasticity: A Basis for Clinical Rehabilitation. Singular Publishing Group, 1992.

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18

Christian, Hölscher, ed. Neuronal mechanisms of memory formation: Concepts of long-term potentiation and beyond. Cambridge: Cambridge University Press, 2001.

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19

Douglas, Fields R., ed. Beyond the synapse: Cell-cell signaling in synaptic plasticity. Cambridge: Cambridge University Press, 2008.

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20

Fields, R. Douglas. Beyond the Synapse: Cell-Cell Signaling in Synaptic Plasticity. Cambridge University Press, 2012.

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21

Schwenk, Karen L. The role of afferent input to the Superior Cervical Ganglion in the plasticity of cerebrovascular axons. 1995.

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22

Nitric oxide in brain development, plasticity, and disease. Amsterdam: Elsevier, 1998.

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23

Josef, Kittler, and Moss Stephen 1962-, eds. The dynamic synapse: Molecular methods in ionotropic receptor biology. Boca Raton: CRC/Taylor & Francis, 2006.

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24

(Editor), Josef T. Kittler, and Stephen J. Moss (Editor), eds. The Dynamic Synapse: Molecular Methods in Ionotropic Receptor Biology (Frontiers in Neuroscience). CRC, 2006.

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25

The dynamic synapse: Molecular methods in ionotropic receptor biology. Boca Raton, FL: CRC/Taylor & Francis, 2006.

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26

Nutt, David J., and Liam J. Nestor. The glutamate system and addiction. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198797746.003.0009.

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Glutamate is the primary excitatory neurotransmitter in the brain. Glutamate is involved in synaptic plasticity, particularly within dopamine systems of the brain that are involved in reward. Glutamate-dependent plasticity is involved in the development of substance addiction through its actions at NMDA receptors during long-term potentiation (LTP) related learning and memory processes. This plasticity within brain circuitry involved in learning and memory is sustained during substance abstinence and may provide a neural substrate for a vulnerability to addiction relapse. Medications that possess the efficacy to reduce glutamate tone in certain brain circuits may reduce craving, and ultimately, relapse in substance dependence. Further research is required, however, to show that the modulation of glutamate transmission in the brain confers clinical benefits in substance addiction.
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27

L, Pichler Florian, ed. Neural pathways research. New York: Nova Science Publishers, 2008.

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28

Spike Timing: Mechanisms and Function. Taylor & Francis Group, 2013.

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29

Protein kinase C and its brain substrates: Role in neuronal growth and plasticity. Amsterdam: Elsevier Science, 1991.

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30

Sajikumar, Sreedharan. Synaptic Tagging and Capture: From Synapses to Behavior. Springer, 2016.

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31

Sajikumar, Sreedharan. Synaptic Tagging and Capture: From Synapses to Behavior. Springer, 2014.

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32

R, Wolpaw Jonathan, Schmidt John T, and Vaughan Theresa M, eds. Activity-driven CNS changes in learning and development. New York, N.Y: New York Academy of Sciences, 1991.

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33

R, Wolpaw Jonathan, Schmidt John T, Vaughan Theresa M, State University of New York at Albany., and New York Academy of Sciences., eds. Activity-driven CNS changesin learning and development. New York, N.Y: New York Academy of Sciences, 1991.

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34

Yoshiaki, Iwamura, Rowe Mark, and International Union of Physiological Sciences. Congress, eds. Somatosensory processing: From single neuron to brain imaging. Amsterdam: Harwood Academic Publishers, 2001.

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35

(Editor), Mark Rowe, and Yoshiaki Iwamura (Editor), eds. Somatosensory Processing: From Single Neuron to Brain Imaging. CRC, 2001.

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36

(Foreword), Per Anderson, Michel Baudry (Editor), and Joel L. Davis (Editor), eds. Long-Term Potentiation, Vol. 1: A Debate of Current Issues. The MIT Press, 1991.

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37

Michel, Baudry, and Davis Joel 1948-, eds. Long-term potentiation: A debate of current issues. Cambridge, Mass: MIT Press, 1991.

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38

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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39

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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