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Dissertations / Theses on the topic 'Platelet activating factor'

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Published: 4 June 2021

Last updated: 25 July 2025

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1

Scarlett, Julie Anne. "Platelet activating factor in the horse." Thesis, University of Cambridge, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387750.

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2

Murphy, Christine Therese. "Mechanisms of stimulus-response coupling in platelet-activating factor stimulated platelets." Thesis, University of Bath, 1992. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304999.

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3

DENTAN, CHRISTINE. "Platelet-activating factor (paf), acetylhydrolase et atherosclerose." Paris 6, 1996. http://www.theses.fr/1996PA066772.

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L'atherosclerose est une pathologie des vaisseaux, multifactorielle, qui a un caractere immuno-inflammatoire chronique. Or, de plus en plus de donnees argumentent le role pro-atherogene et pro-thrombogene d'un puissant mediateur glycerophospholipidique, le platelet-activating factor (paf), qui est degrade specifiquement par l'acetylhydrolase. Cette hypothese est etayee par nos travaux qui ont mis en evidence la formation du paf dans un modele de cellules spumeuses, riches en esters de cholesterol, stimulees par phagocytose. Ces cellules, qui representent la principale composante cellulaire du

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4

Bonin, Fanny. "Cytoprotective effects of intracellular platelet activating factor acetylhydrolases." Thesis, University of Ottawa (Canada), 2003. http://hdl.handle.net/10393/26529.

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Platelet activating factor (PAF) is a biologically active phospholipid implicated in the developmental brain disorder Miller-Dieker Syndrome (MDS) and purported to be a primary mediator of cell death in HIV-dementia, ischemia, and epilepsy. As part of my honour's thesis, I demonstrated that PAF can elicit cell death independently of its G-protein coupled receptor (PAFR) in PC12 cells. In my M.Sc. research, I have sought to identify how PAF-mediated cell death is regulated in PC12 cells. PAF is inactivated in brain by two intracellular PAF-acetylhydrolases (PAF-AHs): PAF-AH I and PAF-AH II. PAF

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5

Gomez, Jorge. "Characterization and regulation of platelet activating factor receptors." Diss., The University of Arizona, 1990. http://hdl.handle.net/10150/185248.

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Platelet activating factor (PAF) is a potent mediator in a variety of inflammatory events. Determining whether PAF participates in the bronchial hyperresponsiveness characteristic of asthma is the long term obj ecti ve for which the studies described here represent an initial step. PAF is a potent agonist that causes contraction of guinea pig peripheral lung strips. To determine if specific receptor sites for PAF could be demonstrated in guinea pig lung membranes (GPLM), direct radioligand binding studies were performed with [³H]C₁₆-PAF (l-0-hexadecyl-2-acetyl-sn-glycero-3-phosphocholine) and

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6

Vanags, Daina M. "Adrenergic and serotonergic potentiation of platelet aggregation /." Title page, summary and contents only, 1993. http://web4.library.adelaide.edu.au/theses/09PH/09phv217.pdf.

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7

Kabbani, Nazir. "Chemical-genetic profiling of platelet-activating factor in yeast." Thesis, University of Ottawa (Canada), 2009. http://hdl.handle.net/10393/28189.

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The basic biological processes between the yeast Saccharomyces cerevisiae and mammals are highly conserved. Yeast posses many genes that are implicated in human diseases and have been successfully used as a model for the study of neurodegeneration. Platelet-Activating Factor (C16:0 PAF) causes neuronal cell death independent of its receptor and has been implicated in Alzheimer's disease. I hypothesized that yeast could be used as a model system for deciphering PAF receptor-independent signalling and have utilized genome-wide chemical genomic screening in yeast to further characterize the mole

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8

Rowe, Anna-Louise. "The role of platelet-activating factor in necrotising enterocolitis." Thesis, University of Birmingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.434713.

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9

Göggel, Rolf. "The mechanisms of platelet activating factor induced alterations in lung functions /." Konstanz : Hartung-Gorre, 2002. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=009757517&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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10

Bosnic, Anthony Martin. "Human antibody responses to a chlamydia-secreted protease factor : a thesis /." San Antonio : UTHSC, 2005. http://learningobjects.library.uthscsa.edu/cdm4/item%5Fviewer.php?CISOROOT=/theses&CISOPTR=14&REC=5.

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11

Huang, Yihui. "Platelet-activating factor and lysophosphatidylcholine in oxidized low density lipoprotein-mediated immune activation /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3450-9/.

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12

Foster, Adrian Paul. "Platelet activating factor : a putative mediator of equine cutaneous inflammation." Thesis, Royal Veterinary College (University of London), 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.519548.

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13

Court, Elaine N. "Platelet-activating factor : its actions and release in the lung." Thesis, University of Sunderland, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327832.

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14

Griffiths, Rachael. "The Regulation of Platelet Activating Factor Acetylhydrolase by Oxidized Phospholipids." VCU Scholars Compass, 2009. http://scholarscompass.vcu.edu/etd/1913.

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Platelet-activating factor acetylhydrolase (PAFAH) is elevated in atherosclerosis and may play a role in pathogenesis of this disease. Molecular mechanisms regulating the expression of this lipoprotein-associated PLA2 are indistinct. Mildy oxidized low density lipoprotein (oxLDL) and monocytes (the primary source of PAFAH) are co-localized in early atheromas. Monocytes are activated by oxidized phospholipids (oxPL) in the oxLDL particle. We hypothesized that oxPL-activated monocytes are the source of increased levels of PAFAH in atherosclerosis. We found that PAFAH expression is significantly

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15

Chase, Peter Burritt 1955. "The molecular pharmacology of a human platelet-activating factor receptor." Diss., The University of Arizona, 1996. http://hdl.handle.net/10150/290574.

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Platelet-activating factor (PAF) is a broadly bioactive family of phospholipids which contribute to the pathogenesis of numerous diseases as well as to many normal physiologic processes. The pleiotropic nature of PAFs actions may be due to the activation of several intracellular signaling pathways or the presence of PAF receptor subtypes. Therefore, to begin to understand the complex mechanisms by which PAF molecules induce cellular responses, a molecular approach was initiated to provide tools to investigate many of the issues surrounding PAF receptors. Using a strategy based upon homology cr

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16

管漢偉 and Hon-wai Michael Koon. "Potentiating effects of platelet activating factor on endothelin-1 induced rat arota vasoconstriction." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1998. http://hub.hku.hk/bib/B31221014.

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17

Koon, Hon-wai Michael. "Potentiating effects of platelet activating factor on endothelin-1 induced rat arota vasoconstriction /." Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20565689.

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18

Usman, Rukhsana. "Platelet activity and arachidonic acid metabolism: modulation by factors in plasma and cerebrospinal fluidand by diet." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1994. http://hub.hku.hk/bib/B31233934.

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19

Muller, Michael John. "Modulation of the response to cutaneous injury /." [St. Lucia, Qld.], 2000. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16146.pdf.

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20

Usman, Rukhsana. "Platelet activity and arachidonic acid metabolism : modulation by factors in plasma and cerebrospinal fluid and by diet /." [Hong Kong] : University of Hong Kong, 1994. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13787263.

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21

Bennett, Steffany Ann Larwill. "The role of platelet activating factor in tumour progression and neurodegeneration." Thesis, University of Ottawa (Canada), 1995. http://hdl.handle.net/10393/10728.

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Tumourigenesis is the result of increased cell number. Progressive neurodegenerative disorders are characterized by decreased cell number. These illnesses emphasize the life-threatening consequences of disrupting the delicate balance between cell growth, cell differentiation, and cell death. Exposure to chronic inflammation is a recognized risk factor for a variety of human cancers and progressive neurodegenerative diseases. This thesis represents an attempt to determine whether one endogenous proinflammatory agent, platelet activating factor (PAF), may participate in tumourigenesis and/or ne

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22

Teoh, Diane Marie. "Platelet-Activating Factor, PAF, and its effects on human neutrophil CD43." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0018/MQ49657.pdf.

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23

Furno-de, Winter Agnès. "Synthese d'agonistes et d'antagonistes du paf (platelet activating factor) : etude pharmacotoxicologique." Paris 7, 1988. http://www.theses.fr/1988PA077055.

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24

Furno-de, Winter Agnès. "Synthèse d'agonistes et d'antagonistes du PAF, Platelet Activating Factor étude pharmacotoxicologique /." Grenoble 2 : ANRT, 1988. http://catalogue.bnf.fr/ark:/12148/cb37613711w.

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25

Noël, Cynthia Jenny. "Modulation orthostérique et allostérique du PAFR par des molécules synthétiques." Mémoire, Université de Sherbrooke, 2008. http://savoirs.usherbrooke.ca/handle/11143/3974.

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Le PAF (facteur d'activation des plaquettes) est un médiateur lipidique de l'inflammation très puissant impliqué dans plusieurs conditions pathophysiologiques.Le PAF agit principalement via un seul récepteur, le PAFR qui appartient à la famille des récepteurs couplés aux protéines G, les GPCRs. Le"two state model" assume que les GPCRs existent dans un état d'équilibre entre un état inactif (R) et un état actif (R*). L'isomérisation de R vers R* peut arriver de façon spontanée, c'est à dire indépendamment de la liaison d'un agoniste. Dans ces travaux de recherche, nous avons tenté de déterminer

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26

Qureshi, Shahryar Jamshed. "Nanomaterial Charge-Dependent Platelet Activating Factor Receptor Agonism in Human Epidermal Cells." Wright State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=wright1535391805311408.

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27

Marrache, Anne Marilise. "Platelet activating factor receptors : nuclear localization and signaling in microvascular endothelial cells." Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=82928.

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It has been postulated that intracellular binding sites for platelet activating factor (PAF) contribute to pro-inflammatory responses to PAF. Isolated nuclei from porcine cerebral microvascular endothelial cells (PCEC) produced PAF-molecular species in response to H2O2 . Using fluorescent-activated cell sorter analysis, we demonstrated the expression of PAF receptors (PAFR) on cell and nuclear surfaces of PCEC. Confocal microscopy studies performed on PCEC, Chinese hamster ovary cells stably overexpressing PAFR and on isolated nuclei from PCEC also showed a robust nuclear distribution o

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28

Appleyard, Caroline Barbara. "The synthesis and catabolism of Platelet Activating Factor in human colon mucosa." Thesis, University of Southampton, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359303.

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29

Rivier, François. "Le Platelet-Activating Factor (PAF) : effets physiopathologiques : implication dans la néphrotoxicité médicamenteuse." Paris 5, 1996. http://www.theses.fr/1996PA05P100.

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30

Dupuis, Fabienne. "Contribution à l'étude du role du platelet-activating factor dans l'hematopoi͏̈ese humaine." Limoges, 1997. http://www.theses.fr/1997LIMO104D.

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31

Cluzel, Jacques. "Étude métabolique et autoradiographique du Platelet-Activating Factor (PAF) dans la rétine." Clermont-Ferrand 1, 1994. http://www.theses.fr/1994CLF1PP01.

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32

Cluzel, Marc. "Etude de l'excrétion de Platelet Activating Factor (PAF) par les neutrophiles humains." Montpellier 2, 1990. http://www.theses.fr/1990MON20275.

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Le platelet activating factor (paf), un ether-phospholipide doue de nombreux effets biologiques est produit en grande quantite par les neutrophiles humains. Dans les conditions standard de stimulation (suspension cellulaire). La majorite du paf produit reste associee aux cellules, ce qui a fait douter du role du paf comme mediateur extracellulaire. Avec pour hypothese que le paf est excrete mais est ensuite recapture par les cellules, nous avons montre que l'excretion observee du paf est variable et peut atteindre 50% de la quantite totale de paf produite dans des conditions de stimulation ou

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33

Mukkamala, Muralikrishna. "Regulation of Platelet-Activating Factor Acetylhydrolase by Oxidized Phospholipids and Proinflammatory Cytokines." VCU Scholars Compass, 2008. http://scholarscompass.vcu.edu/etd/653.

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Platelet-Activating Factor Acetylhydrolase (PAFAH) is a monocyte-derived phospholipase A2 that catalyzes the hydrolysis of platelet-activating factor (PAF) and has been implicated in atherosclerosis. Although PAF and other proinflammatory stimuli are postulated to induce the enzyme, mechanisms controlling PAFAH expression are largely unknown at present. We have shown that PAFAH induction in monocytes is increased in response to oxidized phospholipids. The PAFAH 5' flanking region has at least 10 putative Stat elements, and IL-6 has been shown to be downstream from the prostaglandin receptor, E

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34

Lukashova, Viktoria. "Involvement of the Jak/STAT pathway in platelet-activating factor receptor signaling." Thèse, Sherbrooke : Université de Sherbrooke, 2003. http://savoirs.usherbrooke.ca/handle/11143/4173.

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35

Bücher, Karen. "Nachweis des Platelet-activating factor (PAF) in der bovinen Plazenta anhand der Expression des PAF-Rezeptors und der zugehörigen PAF-Azetylhydrolasen." Wettenberg : VVB Laufersweiler, 2005. http://deposit.d-nb.de/cgi-bin/dokserv?idn=978037847.

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36

Marr, Kathryn Anne. "Mediators of neutrophil activation and bronchoconstriction in equine chronic obstructive pulmonary disease." Thesis, Royal Veterinary College (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362393.

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37

Connor, David Ewan Clinical School St Vincent's Hospital Faculty of Medicine UNSW. "A study of the characterisation, procoagulant activity and Annexin V binding properties of platelet-derived microparticles." Awarded by:University of New South Wales, 2007. http://handle.unsw.edu.au/1959.4/37303.

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Platelet-derived microparticles, released as a result of platelet activation, promote coagulation through the surface exposure of phosphatidylserine, acting as the catalytic site for the conversion of prothrombin to thrombin by the activated coagulation factors X and V. Although elevated numbers of circulating platelet-derived microparticles can be detected in a number of clinical disorders, the methods for the detection of these microparticles are far from standardised. In addition, recent reports have also speculated that not all microparticles may expose phosphatidylserine, demonstrating th

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38

Lehmberg, Jens Matthias. "Die Mediatorfunktion von Bradykinin und Platelet-Activating Factor bei der globalen zerebralen Ischämie." Diss., lmu, 2002. http://nbn-resolving.de/urn:nbn:de:bvb:19-7775.

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39

Khoury, Joseph. "Endotoxin, platelet-activating factor, and sepsis-related cytokines: Effects on lung pericyte growth." Thesis, McGill University, 1997. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=95583.

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Gram-negative sepsis is an important cause of the acute respiratory distress syndrome (ARDS). Pulmonary hypertension (PH), which often develops in ARDS, results in part from the remodelling of the pulmonary microvasculature. Vascular pericytes (PC), dedifferentiated smooth muscle-like cells, are thought to playa role in the remodelling and neomuscularization of pulmonary microvessels. Endotoxin, lipopolysaccharide (LPS) from the outer membrane of gram-negative bacteria, activates macrophages, endothelial cells, and other cells to release potent cytokine and phospholipid mediators such as plate

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40

Khan, Nighat Murad. "An investigation of platelet-activating factor metabolism during normal and pre-eclamptic pregnancies." Thesis, University of Bath, 1993. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332312.

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41

Taylor, Matthew W. "Targeting Neurodegeneration in Alzheimer’s Disease Using Natural Products Derived from Maya Traditional Medicine." Thesis, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/30688.

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Alzheimer’s disease (AD) is a complex neurodegenerative disorder with limited treatment options. Previous research has shown that metabolism of the platelet activating factor (PAF) family of lipid second messengers is impaired in AD. While PAFs are known to exacerbate glutamate excitotoxicity, signal tau hyperphosphorylation, and mediate amyloid β neurotoxicity, it is not yet clear whether cognitive decline can be ascribed to activation of the G-protein-coupled PAF receptor (PAFR). Here, I assessed whether loss of PAFR would alter Morris water maze performance in the TgCRND8 (Tg) mouse model

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42

Dunn, Anita Marie 1956. "The role of neutrophils in systemic anaphylaxis in the rabbit." Thesis, The University of Arizona, 1989. http://hdl.handle.net/10150/276960.

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The objective of this study was to determine whether neutrophils play a significant role in anaphylaxis or in the response to the anaphylactic mediator platelet activating factor (PAF) in the rabbit. Vinblastine and anti-neutrophil antibodies were compared as neutrophil depleting agents, and 0.35 mg/kg vinblastine was selected as optimal for efficiency and specificity of depletion. Anaphylaxis was induced in sensitized rabbits by intravenous antigen challenge. Neutrophil depletion to 399 ± 101 cells/mm³ blood (14 ± 3%) did not significantly inhibit the physiologic and hematologic events associ

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43

Min, Jung-Hyun. "Enzyme mechanism, substrate specificity, and lipoprotein association of human plasma platelet-activating factor acetylhydrolase /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/9254.

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44

Iqbal, Shehzad Mustafa. "The role of cytokines in regulating platelet-activating factor (PAF) synthesis in endothelial cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ64961.pdf.

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45

Lohmeyer, Matthias. "The mechanism of action of antitumour lipid agents related to platelet-activating factor (PAF)." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319528.

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46

Hing-lun, Lee. "Hemodynamic effects of endothelin-1 and platelet-activating factor after nitric oxide synthase inhibition in the rat." Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21903967.

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47

Syrett, Andrew J. "Bioactive Glycerophospholipids and Their Role in Modulating Neuronal Vulnerability Following Cerebral Ischemia." Thesis, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20536.

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Stroke is a devastating and debilitating condition resulting from a blockage or hemorrhage in the vasculature of the brain. Despite extensive research, the etiology and pathophysiology of the disease at the level of the cell membrane are poorly understood, and effective treatment has been elusive. Though much research has shown marked increases in lipid metabolism following stroke, the impact of these changes have often been overlooked given the technical challenges associated with identifying regionally specific changes in degenerating tissue. The advent of lipidomics – a systems biology appr

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48

De, Leon Ellen Jane Biotechnology &amp Biomolecular Sciences Faculty of Science UNSW. "Engineering antibodies against complex platelet antigens using phage display technology." Awarded by:University of New South Wales, 2007. http://handle.unsw.edu.au/1959.4/37009.

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Platelets are small anucleate cell fragments found in blood whose physiological role is important in maintaining haemostasis. In vivo, platelet surface glycoproteins mediate the mechanistic roles of platelets, and polymorphic changes to these glycoproteins have been observed to have significant effects on the platelet cellular function and such changes may include over-expression, under-expression and antigenicity of the protein. Human platelet antigens (HPA) are a result of polymorphic differences in platelet surface glycoproteins which have been found to be variably expressed in the populati

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49

Nunez, Danielle. "Étude de la plaquette sanguine : phosphorylation des proteines et recepteur du platelet activating factor (paf)." Paris 7, 1990. http://www.theses.fr/1990PA077072.

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I) Effet du PAF sur les réactions de phosphorylation dans la plaquette. Le PAF, après s'être lie a son récepteur, stimule, comme la thrombine, la phosphorylation de la chaine légère de la Myosine (P20) et celle d'une protéine de poids moléculaire apparent 47 KD (P47) dont la nature et la fonction restent discutées. Nous avons montré que la P47 n'est pas l'actine dont elle diffère par le point isoélectrique. La phosphorylation de la p47 est en relation avec la sécrétion: en présence de l'Ionophore A23187 et l'EDTA l'agrégation est inhibée alors que la sécrétion et la phosphorylation de la p47 n

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50

Herd, Caroline Mary. "Inflammatory mediators : the involvement of histamine, serotonin and platelet activating factor in anaphylactoid-type reactions." Title page, summary and table of contents only, 1990. http://hdl.handle.net/2440/19111.

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1v.<br>Title page, contents and abstract only. The complete thesis in print form is available from the University Library.<br>Thesis (Ph.D.)--University of Adelaide, 1990<br>Thesis (Ph.D.)--University of Adelaide, Dept. of Clinical and Experimental Pharmacology, 1990

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