Relevant bibliographies by topics / Platelet function

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Platelet function'

Author: Grafiati
Published: 4 June 2021
Last updated: 10 March 2023

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Journal articles on the topic "Platelet function"

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Stolla, Moritz, Renata Grozovsky, Melissa M. Lee-Sundlov, Herve Falet, and Karin M. Hoffmeister. "Effects of Platelet Circulatory Age on Platelet Function." Blood 128, no. 22 (2016): 413. http://dx.doi.org/10.1182/blood.v128.22.413.413.

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Abstract The human body produces and removes 1011 platelets daily to maintain a normal steady-state platelet count. However, the regulatory mechanisms remain elusive. We have shown that platelets lacking sialic acid (desialylated platelets) are removed by the hepatic Ashwell-Morell receptor (AMR or asialoglycoprotein receptor type 2), thereby regulating platelet survival and hepatic TPO levels. Platelet counts and lifetime were increased in Asgr2-/- mice (AMR-null mice), compared to wild type (WT) mice. Platelet volume and immature platelet fraction (IPF) are decreased in AMR-null mice, consis
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Coxon, Carmen H., Alexander M. Lewis, Amanda J. Sadler, et al. "NAADP regulates human platelet function." Biochemical Journal 441, no. 1 (2011): 435–42. http://dx.doi.org/10.1042/bj20111175.

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Platelets play a vital role in maintaining haemostasis. Human platelet activation depends on Ca2+ release, leading to cell activation, granule secretion and aggregation. NAADP (nicotinic acid–adenine dinucleotide phosphate) is a Ca2+-releasing second messenger that acts on acidic Ca2+ stores and is used by a number of mammalian systems. In human platelets, NAADP has been shown to release Ca2+ in permeabilized human platelets and contribute to thrombin-mediated platelet activation. In the present study, we have further characterized NAADP-mediated Ca2+ release in human platelets in response to
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Margraf, Andreas, Claudia Nussbaum, and Markus Sperandio. "Ontogeny of platelet function." Blood Advances 3, no. 4 (2019): 692–703. http://dx.doi.org/10.1182/bloodadvances.2018024372.

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AbstractAlthough the hemostatic potential of adult platelets has been investigated extensively, regulation of platelet function during fetal life is less clear. Recent studies have provided increasing evidence for a developmental control of platelet function during fetal ontogeny. Fetal platelets feature distinct differences in reactive properties compared with adults. These differences very likely reflect a modified hemostatic and homeostatic environment in which platelet hyporeactivity contributes to prevent pathological clot formation on the one hand but still ensures sufficient hemostasis
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Feng, Shuju, Michael H. Kroll, Alpa M. Nick, Anil Sood, and Vahid Afshar-Kharghan. "Platelet Function in Ovarian Cancer." Blood 126, no. 23 (2015): 4656. http://dx.doi.org/10.1182/blood.v126.23.4656.4656.

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Abstract A significant number of patients with ovarian cancer develop venous thromboembolism that is associated with a worse prognosis. The etiology of an increased frequency of venous thrombosis in cancer patients is not clear, and various hypotheses, including the presence of hyperreactive platelets, have been postulated. Hyperreactive platelets have a lower threshold for aggregation, and hence there is a higher number of degranulated platelets in circulation and a higher concentration of platelet granular contents in plasma. We compared ADP- and collagen-induced platelet aggregation in pati
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Iwaszko-Simonik, A., and S. Graczyk. "Evaluation of platelet function in horses undergoing colic surgery using the PFA-100 platelet function analyser." Veterinární Medicína 60, No. 9 (2016): 476–82. http://dx.doi.org/10.17221/8438-vetmed.

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Fidler, Trevor P., Jesse W. Rowley, Claudia Araujo, et al. "Superoxide Dismutase 2 is dispensable for platelet function." Thrombosis and Haemostasis 117, no. 10 (2017): 1859–67. http://dx.doi.org/10.1160/th17-03-0174.

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SummaryIncreased intracellular reactive oxygen species (ROS) promote platelet activation. The sources of platelet-derived ROS are diverse and whether or not mitochondrial derived ROS, modulates platelet function is incompletely understood. Studies of platelets from patients with sickle cell disease, and diabetes suggest a correlation between mitochondrial ROS and platelet dysfunction. Therefore, we generated mice with a platelet specific knockout of superoxide dismutase 2 (SOD2-KO) to determine if increased mitochondrial ROS increases platelet activation. SOD2-KO platelets demonstrated decreas
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Sloand, J. A., and E. M. Sloand. "Studies on platelet membrane glycoproteins and platelet function during hemodialysis." Journal of the American Society of Nephrology 8, no. 5 (1997): 799–803. http://dx.doi.org/10.1681/asn.v85799.

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Hemodialysis only partially corrects the defects in platelet function associated with uremia. Platelet contact with the artificial surfaces of the dialysis filter during hemodialysis can itself cause platelet activation, degranulation, and loss of platelet membrane glycoproteins. Although the transient platelet dysfunction that occurs after platelet contact with foreign surfaces during cardiopulmonary bypass has been well characterized, there has been no such investigation of hemodialysis. In this study of hemodialysis patients, bleeding times (BT) and the response of their platelets to thromb
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Coughlin, Shaun. "Protease-Activated Receptors and Platelet Function." Thrombosis and Haemostasis 82, no. 08 (1999): 353–56. http://dx.doi.org/10.1055/s-0037-1615853.

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IntroductionPlatelet activation is critical for normal hemostasis, and platelet-dependent arterial thrombosis underlies most myocardial infarctions. Thrombin is the most potent activator of platelets.1,2 For this reason, understanding the process by which thrombin activates platelets is necessary for understanding hemostasis and thrombosis and may yield novel anti-platelet therapies. This chapter focuses on our recent studies of the receptors that mediate activation of human platelets by thrombin.3,4 Thrombin signaling is mediated, at least in part, by a family of G protein-coupled protease-ac
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Mongirdienė, Aušra. "Possibilities of platelet function assays." Medicina 43, no. 10 (2007): 767. http://dx.doi.org/10.3390/medicina43100098.

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The aim of this article is to review various methods used and started to introduce into practice for determining platelet function, their possibilities, advantages, and disadvantages. Nowadays, in platelet function investigations, devices and their systems with unequal possibilities, operating in different principles, are used. However, because of a wide variety of platelets defects, none of them gives the accuracy of 100%. In order to avoid mistakes, several assays are used. Analysis of platelets function testing is represented in two ways: according to its investigation object and investigat
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Flaumenhaft, Robert, James R. Dilks, and Derek S. Sim. "Protein Palmitoylation in Platelet Function: Role in G Protein-Mediated Platelet Activation and Platelet Recruitment into Thrombi." Blood 106, no. 11 (2005): 650. http://dx.doi.org/10.1182/blood.v106.11.650.650.

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Abstract Protein palmitoylation represents the covalent linkage of a 16-carbon saturated fatty acid to a protein. This reversible post-translational modification directs protein-protein interactions as well as protein association with membranes and lipid rafts. Protein palmitoylation participates in ligand-induced signal transduction in several nucleated cells. Its role in platelet activation, however, has not previously been evaluated. We have found that platelets contain the palmitoyl transfer proteins GODZ and HIP14 as well as the palmitoyltransferase, acyl-protein thioesterase 1 (APT1). Th
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Dissertations / Theses on the topic "Platelet function"

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Hayman, Melissa Anne. "Genomic influences on platelet function." Thesis, Queen Mary, University of London, 2018. http://qmro.qmul.ac.uk/xmlui/handle/123456789/36221.

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The study of platelet messenger and micro-RNAs is of increasing interest owing to the fact that platelets contain the machinery to splice and translate mRNA into proteins in response to inhibitory or activating signals. However, the relatively small size (roughly 4000-5000 transcripts) and short half-life of the platelet transcriptome makes this a technically challenging aspect of platelet biology to investigate. The aims of these thesis investigations were therefore to optimise protocols for the isolation of platelets for downstream RNA analyses and function testing, to investigate the functi
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Arunima, Ghosh. "Role of CD36 in Platelet Function." Cleveland State University / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=csu1199991110.

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Vickers, James. "Endothelial cells and platelet function." Thesis, University of Nottingham, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336951.

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Radhakrishnan, Aparna. "Genetic variation studies of megakaryopoiesis, platelet formation and platelet function." Thesis, University of Cambridge, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708102.

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Cauwenberghs, Sandra. "Platelet responsiveness and function during storage implications for platelet transfusion therapy /." [Maastricht : Maastricht : Maastricht University] ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=12756.

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Davies, Anna. "Platelet function in the presence of Synthocytes : a novel platelet substitute." Thesis, University of Nottingham, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.324048.

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Smout, Jonathan Daniel. "Platelet function in atherosclerotic vascular disease." Thesis, University of Newcastle upon Tyne, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417517.

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Rolf, Michael George. "P2X₁ receptors and human platelet function." Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621241.

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Peacock, Lesley. "Isomeric fatty acids and platelet function." Thesis, University of Aberdeen, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253708.

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1. <i>Cis</i> unsaturated fatty acids were shown to inhibit porcine platelet aggregation in response to both collagen and thrombin. Fatty acids with a <i>trans</i> double bond had an anti-aggregatory effect on collagen-induced aggregation but this was significantly less than that observed with the <i>cis</i> equivalent and was diminished as the dose of agonist increased. Thrombin-induced platelet aggregation was unchanged or slightly potentiated by <i>trans</i> isomers. 2. Both the <i>cis</i> and <i>trans</i> isomeric acids inhibited collagen-induced TXB<sub>2</sub> production. The <i>trans</i
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Tymkewycz, Paulina M. "Biologically active lipids and platelet function." Thesis, University of Edinburgh, 1987. http://hdl.handle.net/1842/30866.

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Books on the topic "Platelet function"

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Quinn, Martin, and Desmond Fitzgerald, eds. Platelet Function. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-917-2.

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Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119375913.

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Gross, Peter Lawrence. Modification of rabbit platelet function in hypercholesterolemia. National Library of Canada, 1991.

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Scott, N. J. Investigations on human factor VIII and platelet function. North East London Polytechnic, 1985.

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Benedetto, C. Physiopathological aspects of prostanoids and platelet function in obstetrics and gynaecology. Brunel University, 1988.

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Islim, Ismail Fathalla Ali. Platelet function in essential hypertension and the effects of anthiypertensive treatment. University of Birmingham, 1995.

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Al-Maghrabi, Salahadin Al-Amin. Plasma lipid peroxides: Their measurement and relationship with selenium status and platelet function. University of Portsmouth, School of Pharmacy and Biomedical Science, 1995.

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Jacek, Hawiger, ed. Platelets: Receptors, adhesion, secretion. Academic, 1989.

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Latta, Eleanor Katrin. Effects on platelet function of chronic administration of ethanol to normo- and hypercholesterolaemic rabbits. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Easton, Terese. The role of nitric oxide synthase in platelet membrane function in dyslexic adults compared to controls. University of Surrey, 1996.

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Book chapters on the topic "Platelet function"

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Gawaz, Meinrad. "Platelets and Inflammation." In Platelet Function. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-917-2_5.

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López, José A., Ian del Conde, and Jing-Fei Dong. "Platelet Function Under Flow." In Platelet Function. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-917-2_9.

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Holme, S. "Platelet Function Preservation." In Coagulation and Blood Transfusion. Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-3900-1_7.

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Lancé, Marcus D. "Platelet Function Monitoring." In Patient Blood Management in Cardiac Surgery. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-15342-7_9.

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Lordkipanidzé, Marie, and Paul Harrison. "Platelet Function Tests." In Platelets in Thrombotic and Non-Thrombotic Disorders. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47462-5_38.

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Annamalai, Aniyizhai. "Platelet Function Disorders." In Medical Management of Psychotropic Side Effects. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-51026-2_27.

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Feinstein, M. B., S. P. Halenda, and G. B. Zavoico. "Calcium and Platelet Function." In Calcium and Cell Physiology. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70070-5_18.

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McNaughton, L., A. Radomski, G. Sawicki, and Marek W. Radomski. "Regulation of Platelet Function." In Nitric Oxide. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57077-3_11.

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Zucker-Franklin, Dorothea. "Platelet Structure and Function." In Thrombopoiesis and Thrombopoietins. Humana Press, 1997. http://dx.doi.org/10.1007/978-1-4612-3958-1_2.

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Harrison, Paul. "Tests of Platelet Function." In Practical Hemostasis and Thrombosis. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9781444306286.ch5.

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Conference papers on the topic "Platelet function"

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Kantak, Ameya, Yuri Lvov, David K. Mills, James G. Spaulding, and Steven A. Jones. "Platelet Function Assessment in a Microfabricated Device." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-33135.

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Although platelets are small and simple in shape, they are complicated in their physiologhy. Their alpha granules and dense bodies secrete a large number of agents that are involved in haemostasis, and the glycoproteins on their surfaces form the linkages with proteins like fibrinogen, fibronectin, collagen and von Willebrand factor that are necessary for adhesion and aggregation (Frojmovic, 1998). Diseases such as heart attack (Meade, 1992), stroke (Harker, 1998) and eclampsia (Schindler et al., 1990), can be the result of pathologies in platelets. Although devices have been recently develope
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Imaoka, T., and T. Asaji. "FUNCTION OF PLATELET 47K PHOSPHOPROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644634.

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Secretion of platelet granule constituents is closely associated with the phosphorylation of a cytosol polypeptide that we called P47 of Mr 47,000 (Imaoka, T. and Haslam, R.J., J. Biol. Chem. 258, 11404, 1983), by protein kinase C. Since the identity and function remains to be known, we purified protein kinase C, unphosphorylated and phosphorylated P47 to homogeneity from human platelets. Then precise phosphorylation reaction of P47 in vitro and a biological function of P47 were studied. Protein kinase C catalysed the phosphorylation reaction of P47 protein, platelet myosin light chain, histon
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Wilkinson, D., P. Vowden, L. Gilka, S. M. Rajah, and R. C. Kester. "PLATELET FUNCTION IN VASOSPASTIC DISORDERS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643476.

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If vasospastic disorders are associated with abnormal eicosanoid metabolism then disturbed platelet function may be observed. We have measured platelet aggregation (PAG) at 37°C to ADP (8 1:1 sequential dilutions from 10μM), collagen (2mg/l) and adrenaline (5μM) in 37 patients with Raynaud's syndrome (21 primary (RD), 9 secondary to scleroderma (RP) and 7 vibration white finger disease (VWF)) and 10 normal subjects (N). In addition we have determined the platelet aggregate ratio (PAR). PAG was expressed as the percentage fall in optical density after the addition of the aggregant. We also reco
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Asaji, T., E. Murakami, N. Takekoshi, S. Matsui, and T. Imaoka. "EFFECT OF ATRIAL NATRIURETIC POLYPEPTIDES ON PLATELET FUNCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644872.

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Atrial natriuretic polypeptides (ANP) have been shown to possess a potent diuretic and natriuretic activity, and medicated to patients with heart insufficiency as a drug to be mediated by cGMPaccumulation in glomeruli. A existence of receptors for ANP have recently beenreported in human platelet. But, whether ANP has a direct effect on platelet function remains to be known.Single stimulation of ANP in any concentration did not induce aggregation in neither platelet rich plasma, nor washed platelets. Also no effect of pretreatment with ANP was observed against aggregation triggered by known med
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Weisenberger, H., and W. Haarmann. "CORRELATION BETWEEN INHIBITION OF PLATELET PHOSPHODIESTERASES (PDEs) AND PLATELET FUNCTION TESTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643433.

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A systematic investigation of over 100 PDE inhibiting pyrimidopyrimidines was performed regarding a possible correlation between influence on platelet function tests and inhibition of platelet PDEs. Basically, the compounds tested were congeners of Dipyridamole but lack substitution in position 6. The concentration necessary for a 50% inhibition of PDE activity in platelet homogenates ranged between 0.000045 and 35 μmoles/L.The inhibition of platelet PDEs was measured in freeze-thaw homogenates of human platelets using 3H-cAMP as substrate. Intraplatelet cAMP changes were measured by prelabell
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Chen, W. J., Z. Chen, and C. X. Bao. "HIP2: A NEW MONOCLONAL ANTI-HUMAN PLATELET ANTIBODY WITH STIMULANT FUNCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643505.

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A monoclonal anti-human platelet antibody(MoAb), HIP2 (IgG3) which induced irrevesible aggregation of platelet in association with the release of serotonin and thromboxane B2 formation is described. Indirect immunofluorescence assay (IFS) showed that the antibody binded to platelets and megakryocytes, and gave a weak reaction with aortic, liver and capillary endothelial cells. Electrophoresis of radiolabelled antigen showed that HIP2 recognized platelet membrane glycoprotein lib (130KD). The purified HIP2 MoAb induced aggregation with normal PRP, not with thrombasthenia platelets, formalin-fix
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Grotemeyer, K.-H., I. Pilawa, and H. P. Schlake. "ABNORMAL PLATELET FUNCTION MEASURED IN RELATIVES OF STROKE PATIENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643407.

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Platelets are assumed to play a crucical role in stroke. Abnormal reactivity of platelets has been observed in patients suffering from stroke but it seems unclear whether or not this abnormal reactivity represents a basic factor of stroke. If abnormal platelet reactivity is more but a consequence of stroke it should be present before stroke happens.In families with stroke patients stroke is more frequent as usual. In order to obtain more information we determined platelet reactivity in healthy persons with a family history without vascular events and healthy persons with a history showing stro
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Yasunaga, K. "HEREDITARY PLATELET FUNCTION DISORDERS IN JAPAN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644876.

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Nationwide surveys of hereditary platelet function disorders in Japan were carried out in 1976, 1981, and 1986. Information on 271 cases received in the 1986 survey was analyzed with that for 103 other cases reported in earlier surveys but not in 1986, making a total of 374 cases. The mortality rate was 6.8% of 162 cases in 1976, 6.6% of 213 cases in 1981, and 5.9% of 374 cases in 1986. Bleeding symptoms appeared at age 12 years in 56.8% of patients and the most common were epistaxis and purpura. Of the 295 cases 49.5% were isolated cases, 20.3% had siblings with confirmed bleeding tendencies,
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Feige, T., C. Freiburg, L. Donner та M. Elvers. "The role of platelet mitochondrial function for platelet mediated Aβ40 aggregation". У 65th Annual Meeting of the Society of Thrombosis and Haemostasis Research. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728208.

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Bodzenta-Lukaszvk, A., K. Krupiński, and M. Bielawiec. "PLATELET FUNCTION AND KALLIKREIN SYSTEM IN PATIENTS WITH ESSENTIAL HYPERTENSION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644258.

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Since the pathogenesis of hypertension is still discussed the aim of this study was to investigate behaviour of platelets and kallikrein system in patients suffering from this disease. In 30 patients with essential hypertension, aged 23-31 years and 20 normotensive healthy subjects, aged 21-35 years the following parameters of platelet function were studied: platelet aggregation induced with ADP,platelet activating factor (PAF) and arachidonic acid (AA) according Born's method, plasma beta-thromboglobulin (Beta-TG) and platelet factor 4 (PF4) , plasma thromboxane B2 (TXB2) and cyclic AMP using
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Reports on the topic "Platelet function"

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Tsokos, George. Complement Activation Alters Platelet Function. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada612004.

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Tsokos, George. Complement Activation Alters Platelet Function. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612005.

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Tsokos, George. Complement Activation Alters Platelet Function. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada612741.

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Upchurch, G. R., Valeri Jr., Khuri C. R., Rohrer S. F., Welch M. J., and G. N. The Effect of Heparin on Fibrinolytic Activity and Platelet Function. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada360274.

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Spiess, Bruce D. Studies of the Effects of Perfluorocarbon Emulsions on Platelet Number and Function in Models of Critical Battlefield Injury. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612698.

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Khuri, Shukri F., Miguel Josa, Trevoc C. Axford, Samar Assousa, and Gina Ragno. Hematologic Changes During and Following Cardiopulmonary Bypass and Their Relationship to Non-Surgical Blood Loss. 1. Platelet Function and the Bleeding Time. Defense Technical Information Center, 1990. http://dx.doi.org/10.21236/ada360145.

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Viksna, Ludmila, Oksana Kolesova, Aleksandrs Kolesovs, Ieva Vanaga, and Seda Arutjunana. Clinical characteristics of COVID-19 patients (Latvia, Spring 2020). Rīga Stradiņš University, 2020. http://dx.doi.org/10.25143/fk2/hnmlhh.

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Data include following variables: Demographics, epidemiological history, comorbidities, diagnosis, complications, and symptoms on admission to the hospital. Also, body’s temperature and SpO2. Blood cells: white cells count (WBC), neutrophils (Neu), lymphocytes (Ly), eosinophils (Eo) and monocytes (Mo), percentages of segmented and banded neutrophils, erythrocytes (RBC), platelet count (PLT), hemoglobin (Hb), and hematocrit (HCT); Inflammatory indicators: erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP); Tissue damage indicators: alanine aminotransferase (ALT), lactate dehydrog
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Michelson, A. D., M. R. Barnard, H. B. Hechtman, H. Macgregor, and R. J. Connolly. In Vivo Tracking of Platelets: Circulating Degranulated Platelets Rapidly Lose Surface P-Selectin but Continue to Circulate and Function. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada360258.

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Valeri, C. R., G. Ragno, H. MacGregor, and L. E. Pivacek. The Effect of Disinfection on Viability and Function of Baboon Red Blood Cells and Platelets. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada360325.

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