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Books on the topic 'Platelet function'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

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1

Quinn, Martin, and Desmond Fitzgerald, eds. Platelet Function. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-917-2.

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2

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119375913.

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3

Gross, Peter Lawrence. Modification of rabbit platelet function in hypercholesterolemia. National Library of Canada, 1991.

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4

Scott, N. J. Investigations on human factor VIII and platelet function. North East London Polytechnic, 1985.

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5

Benedetto, C. Physiopathological aspects of prostanoids and platelet function in obstetrics and gynaecology. Brunel University, 1988.

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6

Islim, Ismail Fathalla Ali. Platelet function in essential hypertension and the effects of anthiypertensive treatment. University of Birmingham, 1995.

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7

Al-Maghrabi, Salahadin Al-Amin. Plasma lipid peroxides: Their measurement and relationship with selenium status and platelet function. University of Portsmouth, School of Pharmacy and Biomedical Science, 1995.

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8

Jacek, Hawiger, ed. Platelets: Receptors, adhesion, secretion. Academic, 1989.

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9

Latta, Eleanor Katrin. Effects on platelet function of chronic administration of ethanol to normo- and hypercholesterolaemic rabbits. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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10

Easton, Terese. The role of nitric oxide synthase in platelet membrane function in dyslexic adults compared to controls. University of Surrey, 1996.

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11

I, Barnhart Marion, and Lusher Jeanne M. 1935-, eds. Factor VIII/vWF and platelet formation and function in health and disease: A tribute to Marion I. Barnhart. New York Academy of Sciences, 1987.

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12

Marques, Marisa B. Quick guide to hemostasis. American Association for Clinical Chemistry, 2015.

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13

P, Watson Steve, and Authi Kalwant S, eds. Platelets: A practical approach. IRL Press at Oxford University Press, 1996.

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14

Nguyen, Augustin. Studies of the effects of ethanol on platelet functions. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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15

Prostaglandins and leukotrienes: Blood and vascular cell function. M. Dekker, 1985.

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16

Curry, Nicola, and Raza Alikhan. Normal platelet function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0281.

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The platelet is a small (2–4 µm in diameter), discoid, anucleate cell that circulates in the blood. In health, it plays a vital role in haemostasis, and in disease it contributes to disorders of bleeding and thrombosis. Platelets are produced from the surface of megakaryocytes in the bone marrow, under tight homeostatic control regulated by the cytokine thrombopoietin. Platelets have a lifespan of approximately 7–10 days, and usually circulate in the blood stream in a quiescent state. Intact, undamaged vessel walls help to maintain platelets in this inactive state by releasing nitric oxide, wh

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17

Platelet function: Assessment, diagnosis, and treatment. Humana Press, 2005.

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18

Quinn, Martin, Desmond Fitzgerald, and Dermott Cox. Platelet Function: Assessment, Diagnosis, and Treatment. Humana Press, 2007.

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19

Platelet Function: Assessment, Diagnosis, and Treatment. Humana Press, 2010.

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20

(Editor), Martin Quinn, Desmond Fitzgerald (Editor), and Dermott Cox (Editor), eds. Platelet Function: Assessment, Diagnosis, and Treatment (Contemporary Cardiology). Humana Press, 2005.

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21

Curry, Nicola, and Raza Alikhan. Platelet disorders. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0282.

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The term ‘platelet disorder’ covers a very large and heterogeneous group of diseases that have a multitude of causes. Platelet disorders are either inherited or acquired and are due to an abnormality of platelet number (quantitative disorder), an abnormality of platelet function (qualitative disorder), or a combination of both. This chapter addresses the causes, diagnosis, and management of platelet disorders.

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22

Von Willebrand Factor And the Mechanisms of Platelet Function. Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1997.

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23

Ruggerl, Zaverio M. Von Willebrand Factor and the Mechanisms of Platelet Function. Chapman & Hall, 1997.

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24

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Incorporated, John, 2018.

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25

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Incorporated, John, 2018.

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26

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Limited, John, 2018.

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27

Duttaroy, Asim K. Nutraceuticals and Human Blood Platelet Function: Applications in Cardiovascular Health. Wiley & Sons, Limited, John, 2018.

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28

Alikhan, Raza. Normal haemostatic function. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0283.

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Humans have evolved an intricate system that maintains blood in a fluid state. This relies on an intact vascular endothelium modulating vascular tone and forming a barrier between blood components and reactive subendothelial components. It also involves the production of inhibitors of both blood coagulation and platelet aggregation. In addition, haemostatic systems are primed to convert blood from its fluid state to a solid state, to allow the formation of a haemostatic plug, following vessel injury, to stem the flow of blood from or within a blood vessel. This chapter reviews the physiology o

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29

Ruggeri, Z. M. Von Willebrand Factor And The Mechanisms Of Platelet Function (Biotechnology Intelligence Unit). Edited by Z. M. Ruggeri. Springer, 1998.

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30

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Jacek J. Hawiger (Editor), eds. Platelets: Receptors, Adhesion, Secretion, Part A, Volume 169: Volume 169: Platelets Part A (Methods in Enzymology). Academic Press, 1989.

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31

(Editor), John N. Abelson, Melvin I. Simon (Editor), and Jacek J. Hawiger (Editor), eds. Platelets: Receptors, Adhesion, Secretion, Part A, Volume 169: Volume 169: Platelets Part A (Methods in Enzymology). Academic Press, 1989.

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32

Kuiper, Gerhardus J. A. J. M., and Hugo ten Cate. Coagulation monitoring. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0266.

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Haemostasis is a dynamic process to stop bleeding after vessel wall damage. Platelets form a platelet plug via activation, adherence, and aggregation processes. The coagulation proteins are activated one-by-one, cascading towards fibrin polymerization, a process controlled by thrombin generation. Fibrinolysis is the process responsible for fibrin mesh degradation, which is also controlled by thrombin. Besides procoagulant proteins, anticoagulant proteins maintain a balance in the haemostatic system. Measuring platelet count and function can be done as part of the monitoring of haemostasis, whi

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33

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Haemostasis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0010.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII

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34

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Angela Theodoulou. Haemostasis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0010_update_001.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII

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35

Marques, Marisa B. Quick Guide to Coagulation Testing:. AACC Press, 2006.

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36

Mannucci, Pier Mannuccio. Platelet Function and Thrombosis: A Review of Methods Proceedings of a Postgraduate Course held at the Fondazione Lorenzini in Milan, Italy, February. ... in Experimental Medicine and Biology). Springer, 2013.

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37

(Editor), Stephen P. Watson, and Kalwant S. Authi (Editor), eds. Platelets: A Practical Approach. Oxford University Press, USA, 1996.

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38

Mannucci, Pier Mannuccio. Platelet Function and Thrombosis: A Review of Methods Proceedings of a Postgraduate Course held at the Fondazione Lorenzini in Milan, Italy, February 24-26, 1972. Springer, 2014.

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39

Mannucci, P. Platelet Function and Thrombosis: A Review of Methods Proceedings of a Postgraduate Course Held at the Fondazione Lorenzini in Milan, Italy, February 24-26 1972. Springer London, Limited, 2013.

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40

Truong, Anne. Erectile Dysfunction Fix: How to Regain Sexual Function, Libido and Treat Erectile Dysfunction Without Drugs or Surgery with Platelet Rich Plasma, the PRIAPUS SHOT®. Independently Published, 2018.

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41

Lusher, Jeanne M. Factor Viii/Vwf and Platelet Formation and Function in Health and Disease: A Tribute to Marion I. Barnhart (Annals of the New York Academy of Sciences). New York Academy of Sciences, 1987.

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42

Langelier, René. Platelets: Overview, Function and Disorders. Nova Science Publishers, Incorporated, 2019.

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43

Platelets: Overview, Function and Disorders. Nova Science Publishers, Incorporated, 2019.

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44

Carroll, Camilla Ma. The effects of the angiogenic growth factors, platelet derived growth factor and basic fibroblast growth factor, on the survival,perfusion and function of the latissmus dorsi muscle flap. 1997.

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45

Mannucci, Pier Mannuccio, and Maddalena Lettino. Bleeding and haemostasis disorders. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_003.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an

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46

Shrivastava, Seema, Beverley J. Hunt, and Anthony Dorling. Coagulopathies in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0135.

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Coagulation abnormalities are common in chronic kidney disease (CKD). Both haemorrhage and thrombosis are more common than in the general population. Haemorrhage, when it occurs, is associated with increased morbidity and mortality compared to that seen in non-uraemic patients. It is more likely spontaneously, but particularly in association with anti-platelet agents or anticoagulants. The increased risk of both arterial and venous thrombosis occurs in part because of the increase prevalence of traditional risk factors for thrombosis in CKD, in part because of the specific problems associated

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47

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0038.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone

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48

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0038_update_001.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone

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49

Pruthi, Rajiv K. Coagulation (Hemostasis and Thrombosis). Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0295.

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The coagulation system has 2 essential functions: to maintain hemostasis and to prevent and limit thrombosis. The procoagulant component of the hemostatic system prevents and controls hemorrhage. Vascular injury results in activation of hemostasis, which consists of vasospasm, platelet plug formation (platelet activation, adhesion, and aggregation), and fibrin clot formation (by activation of coagulation factors in the procoagulant system). The anticoagulant system prevents excessive formation of blood clots, and the fibrinolytic system breaks down and remodels blood clots. Quantitative abnorm

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50

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0070.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an

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