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Books on the topic 'Platelets; Thrombocytopenia'

Author: Grafiati
Published: 5 June 2025
Last updated: 2 August 2025

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1

F, Décary, and Rock G, eds. Platelet membrane in transfusion medicine. Karger, 1988.

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2

A, Mousa Shaker, ed. Anticoagulants, antiplatelets, and thrombolytics. Humana Press, 2004.

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3

A, Mousa Shaker, ed. Anticoagulants, antiplatelets, and thrombolytics. Humana Press, 2004.

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4

Hewitt, John. Studies in antibody specificity against platelet glycoprotein IIb/IIIa in autoimmune thrombocytopenic purpura. University of Manchester, 1994.

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5

McCrae, Keith R. Thrombocytopenia. Taylor & Francis Group, 2006.

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6

Ishida, Yoji, and Yoshiaki Tomiyama. Autoimmune Thrombocytopenia. Springer, 2018.

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7

Ishida, Yoji, and Yoshiaki Tomiyama. Autoimmune Thrombocytopenia. Springer, 2017.

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8

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Haemostasis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0010.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII
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9

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Angela Theodoulou. Haemostasis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0010_update_001.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII
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10

Bell, Mckenzie G. Thrombocytopenia: Epidemiology, Potential Complications and Emerging Treatments. Nova Science Publishers, Incorporated, 2014.

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11

Greinacher, Andreas, and Theodore Warkentin. Heparin-Induced Thrombocytopenia. Taylor & Francis Group, 2003.

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12

Greinacher, Andreas, and Theodore Warkentin. Heparin-Induced Thrombocytopenia. Taylor & Francis Group, 2012.

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13

Greinacher, Andreas, and Theodore Warkentin. Heparin-Induced Thrombocytopenia. Taylor & Francis Group, 2001.

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14

Greinacher, Andreas, and Theodore Warkentin. Heparin-Induced Thrombocytopenia. Taylor & Francis Group, 2012.

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15

McCrae, Keith R. Thrombocytopenia (Basic and Clinical Oncology). Informa Healthcare, 2006.

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16

(Editor), Theodore Warkentin, and Andreas Greinacher (Editor), eds. Heparin-Induced Thrombocytopenia, Fourth Edition: Abridged, Special Edition (Fundamental and Clinical Cardiology). 4th ed. Informa Healthcare, 2007.

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17

Mousa, Shaker A. Anticoagulants, Antiplatelets, and Thrombolytics (Methods in Molecular Medicine). Humana Press, 2003.

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18

Anticoagulants, antiplatelets, and thrombolytics. Humana Press, 2003.

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19

Capodanno, Davide. Bivalirudin and argatroban. Edited by Raffaele DeCaterina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0052.

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The central role of thrombin in the process of clot formation makes it an important therapeutic target. Heparin is a potent anticoagulant, but has a number of limitations, in that—for example—it does not bind clot-bound thrombin, activates platelets, and may determine heparin-induced thrombocytopenia (HIT). Bivalirudin and argatroban, which belong to the class of intravenous direct thrombin inhibitors, overcome many of the limitations of heparin. Bivalirudin is currently indicated for patients undergoing percutaneous coronary intervention, patients with non-ST-segment elevation acute coronary
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20

Zaki, Maysaa El Sayed. Thrombocytopenia: Causes, Diagnosis and Treatment. Nova Science Publishers, Incorporated, 2012.

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21

Kothari, Jaimal, and Marie Scully. Thrombocytopenia in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0272.

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Thrombocytopenia is an extremely common finding in patients in intensive care. This can lead to serious morbidity and mortality from bleeding, and also uncertainty with regards to how to assess this risk in the wider context of the underlying pathology, which may be multifactorial, when specific blood products should be transfused, and the associated risks of carrying out interventional procedures. Haemodilution, platelet consumption, increased platelet destruction, reduced production, and increased sequestration are the main mechanisms that account for thrombocytopenia, and in the ICU setting
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22

Batchelor, Tracy, Joshua P. Klein, Andrés José María Ferreri, and Lisa M. DeAngelis, eds. Oxford Textbook of Neurohaematology. Oxford University PressOxford, 2024. http://dx.doi.org/10.1093/med/9780198884903.001.0001.

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Abstract The Oxford Textbook of Neurohaematology is the first dedicated source of knowledge on the diverse neurological conditions associated with malignant and classical haematological diseases. The book is divided into three sections. In the first section, neurological conditions associated with malignant haematological diseases are covered. This section begins with chapters on primary haematological malignancies of the nervous system, including primary central nervous system lymphomas, vitreoretinal lymphoma, and other rare primary malignancies such as Hodgkin disease and lymphoproliferativ
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23

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0038.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone
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24

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0038_update_001.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone
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25

Revill, M. E. Measurement of IgG subclasses of platelet-associated immunoglobulin in HIV-related thrombocytopenia. 1994.

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26

Kesteven, Patrick. Haematology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0061.

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This chapter describes the common haematological abnormalities seen in rheumatic conditions with special reference to laboratory findings and diagnostic traps. The chapter is organized into sections dealing with red cell abnormalities (and the distinction between anaemia of chronic disease and iron deficiency); white cell abnormalities (neutrophilia and neutropenia); platelets (thrombocytosis, idiopathic thrombocytopenic purpura and thrombotic thrombocytopenic purpura); and finally coagulation abnormalities (lupus anticoagulant).
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27

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Clinical approach. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0001.

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History taking in patients with haematological disease - Physical examination - Splenomegaly - Lymphadenopathy - Unexplained anaemia - Patient with elevated haemoglobin - Elevated white blood cell (WBC) count - Reduced WBC count - Elevated platelet count - Reduced platelet count - Easy bruising - Recurrent thromboembolism - Pathological fracture - Raised ESR - Serum or urine paraprotein - Anaemia in pregnancy - Thrombocytopenia in pregnancy - Prolonged bleeding after surgery - Positive sickle test (HbS solubility test)
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28

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Hassan Al-Sader. Clinical approach. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0001_update_001.

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History taking in patients with haematological disease - Physical examination - Splenomegaly - Lymphadenopathy - Unexplained anaemia - Patient with elevated haemoglobin - Elevated white blood cell (WBC) count - Reduced WBC count - Elevated platelet count - Reduced platelet count - Easy bruising - Recurrent thromboembolism - Pathological fracture - Raised ESR - Serum or urine paraprotein - Anaemia in pregnancy - Thrombocytopenia in pregnancy - Prolonged bleeding after surgery - Positive sickle test (HbS solubility test)
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29

Webster, Michelle. Relative efficacy of intravenous immunoglobulin G in ameliorating thrombocytopenia induced by anti-platelet GPIIbIIIa versus GPIbalpha antibodies. 2006.

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30

Noris, Marina, and Tim Goodship. The patient with haemolytic uraemic syndrome/thrombotic thrombocytopenic purpura. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0174.

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The patient who presents with microangiopathic haemolytic anaemia, thrombocytopenia, and evidence of acute kidney injury presents a diagnostic and management challenge. Haemolytic uraemic syndrome (HUS) and thrombotic thrombocytopenic purpura (TTP) are two of the conditions that frequently present with this triad. They are characterized by low platelet count with normal or near-normal coagulation tests, anaemia, and signs of intravascular red cell fragmentation on blood films, and high LDH levels.HUS associated with shiga-like toxins produced usually by E.coli (typically O157 strains) may occu
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31

Misbah, Siraj. Immunological support. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0301.

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Until the 1980s, the use of immunoglobulin as a therapeutic agent was confined to replacement therapy in patients with primary or secondary antibody deficiency. Its role as an effective immunomodulator was discovered serendipitously, when IV immunoglobulin (IVIg) was shown to consistently increase the platelet count in a child with antibody deficiency and immune thrombocytopenic purpura. Since then, the use of high-dose IVIg as an immunomodulator has become established as an important therapeutic option in many immune-mediated diseases. This chapter reviews the therapeutic indications for immu
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32

McDonald, Vickie, and Marie Scully. Anticoagulants and antithrombotics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0051.

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Coagulation is best thought of using the cell-based model of coagulation. Patients commenced on heparin therapy should have their platelet count monitored early because of the risk of heparin-induced thrombocytopenia, which can occur on any type or dose of heparin. Emergency reversal of warfarin should be with prothrombin complex concentrate (containing factors II, VII, IX, and X) and not fresh frozen plasma. New oral anticoagulants have the advantage of predictable pharmacokinetics and do not require routine monitoring, but optimal reversal strategies for these agents are not clear. Thromboly
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33

Syed, Almas, Robert Evans Heithaus, and Chet R. Rees. Elimination of Post-Procedural Bleeding After Placement of Tunneled Dialysis Catheters. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0047.

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This chapter discusses how to prevent postoperative bleeding with the placement of tunneled dialysis catheters. Tunneled intravenous chest catheters, particularly dialysis catheters, sometimes result in oozing or frank bleeding from the incisions during the first 24 hours after placement. This problem is exacerbated by the large diameter and stiffness of these catheters. Moreover, patients requiring these catheters frequently suffer from abnormal hemostasis profiles—an elevated international normalized ratio, thrombocytopenia, or abnormal platelet function due to uremia is commonly seen. The t
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34

Qu, Lirong, and Darrell J. Triulzi. Blood product therapy in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0267.

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Transfusions are among the most common medical procedures in the intensive care unit. Several randomized controlled trials (RCT) indicate that restrictive red cell transfusion practice using a haemoglobin of <7g/dL is safe in critically-ill patients. Although similar RCT are not available for plasma or platelet transfusion guidelines, a large body of observational studies suggest that plasma transfusion for an invasive procedure has not been shown to be of benefit in patients with INR <2.0. Similarly, in thrombocytopenic patients, the target platelet count for bleeding or for an invasive
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35

Curry, Nicola, and Raza Alikhan. Bruising and bleeding. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0038.

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Bruising is extremely common and a normal response to injury. Perception of what is a normal level of bruising is subjective and it can be difficult to differentiate a patient with ‘normal’ bruising from a patient who has bruising due to a mild bleeding disorder. Patients with bruising/bleeding can be categorized into two clinical groups: those who have no other symptoms, in whom the cause is likely to be either a normal response to injury, or an isolated platelet disorder or clotting factor deficiency; and those who have additional symptoms, in whom haematological disease (e.g. thrombocytopen
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36

Wiles, Kate, and Catherine Nelson-Piercy. Pre-eclampsia and related disorders. Edited by Norbert Lameire and Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0296_update_001.

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Pre-eclampsia is a pregnancy-specific condition diagnosed by new-onset hypertension and proteinuria after 20 weeks’ gestation. The incidence of pre-eclampsia means that it is both the most prevalent cause of acute kidney injury (AKI) in pregnancy and the commonest glomerular disease in the world. This chapter outlines the diagnosis and management of pre-eclampsia. Particular emphasis is given to the post-partum disease course as this is when a specialist nephrology opinion may be sought. HELLP (haemolysis, elevated liver enzymes, low platelets) syndrome is a variant of severe pre-eclampsia. Th
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37

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0070.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an
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38

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_001.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an
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39

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_002.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an
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40

Sternbach, Marion. Apheresis in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0268.

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This chapter describes therapeutic plasma exchange, as well as cytapheresis for hyperleukocytosis and essential thrombocythemia, as well as harvesting haematological stem cells (HSC) for transplantation. Instrumentation and techniques are mostly density centrifugation, much less column adsorption for antibodies or membrane filtration for noxious molecules. Pathophysiology of apheresis is dealt with in great detail with emphasis on prevention and treatment of side effects, much more critical in the intensive care unit (ICU) setting. Main manifestations are: hypocalcaemia due to chelation by ant
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