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Journal articles on the topic 'Plurihormonal Pituitary Adenoma'

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Published: 3 June 2025

Last updated: 31 July 2025

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1

F. El Gharroudi, F. El jaafari, S. Rafi, G. El Mghari, and N. El Ansar. "Pituitary adenoma consecrating GH and ACTH: A rare case report." World Journal of Advanced Research and Reviews 18, no. 1 (2023): 1136–38. http://dx.doi.org/10.30574/wjarr.2023.18.1.0752.

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Plurihormonal Pituitary adenoma is defined as an adenoma that expresses more than one hormone on immunohistochemistry. The most common combination in these adenomas includes growth hormone (GH) and prolactin (PRL). We describe an interesting case of plurihormonal pituitary adenoma with double hormonal staining for adrenocorticotrophic hormone (ACTH) and GH in a patient who presented with acromegaly and subtle signs of Cushing disease due to a large pituitary macroadenoma

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2

F., El Gharroudi, El jaafari F., Rafi S., El Mghari G., and El Ansar N. "Pituitary adenoma consecrating GH and ACTH: A rare case report." World Journal of Advanced Research and Reviews 18, no. 1 (2023): 1136–38. https://doi.org/10.5281/zenodo.8179191.

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Plurihormonal Pituitary adenoma is defined as an adenoma that expresses more than one hormone on immunohistochemistry. The most common combination in these adenomas includes growth hormone (GH) and prolactin (PRL). We describe an interesting case of plurihormonal pituitary adenoma with double hormonal staining for adrenocorticotrophic hormone (ACTH) and GH in a patient who presented with acromegaly and subtle signs of Cushing disease due to a large pituitary macroadenoma

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3

Ndong, Charlène-ludwine Bifoume, Sana Rafi, Ghizlane El Mghari, and Nawal El Ansari. "Case Report A Rare Case of Plurihormonal Pituitary Adenoma Associating Cushing's Disease and Silent Prolactinoma." Scholars Journal of Medical Case Reports 10, no. 5 (2022): 422–24. http://dx.doi.org/10.36347/sjmcr.2022.v10i05.001.

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Plurihormonal pituitary adenomas are rare conditions that secrete two or more pituitary hormones. The association of prolactin with other hormones is described but associated with ACTH is uncommon. We report a rare case of mixed adenoma associating Cushing’s disease and silent prolactin adenoma in 21 years old male patient. This case highlights an unusual plurihormonal pituitary adenoma case with a rare association between ACTH and prolactin. It pathogeny is less clearly define.

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Kannan, Subramanian, Susan M. Staugaitis, Robert J. Weil, and Betul Hatipoglu. "A Rare Corticotroph-Secreting Tumor with Coexisting Prolactin and Growth Hormone Staining Cells." Case Reports in Endocrinology 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/529730.

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Pituitary adenomas can express and secrete different hormones. Expression of pituitary hormones in nonneoplastic pituitary cells is regulated by different transcription factors. Some pituitary adenomas show plurihormonal expression. The most commonly reported plurihormonal adenomas are composed of somatotrophs, lactotrophs, thyrotrophs and gonadotrophs. Pituitary adenomas composed of both corticotroph and somatolactotroph secreting cells are not common because transcription factors regulating the expression of these hormones are different. We report a rare case of pituitary adenoma with concom

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5

Hossain, Alisha, Emily Skutnik, Arjan Ahluwalia, et al. "A Rare Case of a Plurihormonal Pituitary Adenoma." Journal of the Endocrine Society 5, Supplement_1 (2021): A565. http://dx.doi.org/10.1210/jendso/bvab048.1152.

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Abstract Background: Pituitary adenomas are usually benign tumors that arise from adenophypophyseal cells and produce one or two types of hormones. Plurihormonal adenomas are a rare subtype that produce two or more hormones and represent less than 1% of all pituitary adenomas. Clinical Presentation: A 76-year-old female presented for evaluation of abnormal thyroid function test results. She was found to have an elevated free T4 of 1.92 ng/dL and total T4 of 14.4 ug/dL with an inappropriately normal TSH of 2.11 uIU/mL. Physical examination was significant for tachycardia, tremors, diaphoresis,

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6

Amir, Jumana, Marie Christine Guiot, and Natasha Garfield. "Plurihormonal pituitary adenoma cosecreting ACTH and GH: a rare cause of Cushing’s disease." BMJ Case Reports 15, no. 11 (2022): e251451. http://dx.doi.org/10.1136/bcr-2022-251451.

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Plurihormonal pituitary adenomas are rare forms of pituitary adenomas that express more than one hormone. The most common association is with growth hormone (GH) and prolactin. Cosecretion of GH and adrenocorticotrophic hormone (ACTH) is rare with only 25 reported cases in literature. Most presented with features of GH excess, and only four presented with Cushing’s disease. We report a case of a woman in her 30s with recurrent plurihormonal pituitary macroadenoma cosecreting GH and ACTH, diagnosed during workup for polycystic ovarian syndrome, and both times presenting uniquely with Cushing’s

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Vora, TarangK, and Sudish Karunakaran. "Thyrotropic pituitary adenoma with plurihormonal immunoreactivity." Neurology India 65, no. 5 (2017): 1162. http://dx.doi.org/10.4103/neuroindia.ni_577_16.

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8

CAMARA, M'BALLOU, SANA RAFI, MGHARI GHIZLANE El, and ANSARI NAWAL El. "Mixed corticotropic and somatotropic pituitary adenoma: A case report." World Journal of Advanced Research and Reviews 17, no. 2 (2023): 735–38. https://doi.org/10.5281/zenodo.8109595.

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Multi-secreting pituitary adenomas are relatively rare. The mixed somatotropic and corticotropic pituitary adenoma is very rare. The most widely described association is that coupling hypersecretion of GH and prolactin. We report the case of a patient with a pituitary adenoma bi-secreting GH and ACTH. He was a 28-year-old patient admitted for pituitary tumor syndrome, cushing syndrome. The hormonal assessment found a dependent ACTH cushing syndrome. The other hypothalamohypophysial axes were without abnormality. The hypothesis of a pituitary adenoma was raised. The brain scan confirmed the dia

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Buurman, Hilke, and Wolfgang Saeger. "Subclinical adenomas in postmortem pituitaries: classification and correlations to clinical data." European Journal of Endocrinology 154, no. 5 (2006): 753–58. http://dx.doi.org/10.1530/eje.1.02107.

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Objective: The aim of this study was to examine pituitary adenomas in a series of postmortem pituitaries by use of modern technologies of immunostaining, to classify the adenomas according to the current WHO classification and to analyse the possible associations to the available clinical data. Methods: In this study, pituitaries of 3048 autopsy cases obtained from autopsy series of the years 1991–2004 were examined. Results: A total of 334 pituitary adenomas were found in 316 pituitaries. One hundred and thirty-two sparsely granulated prolactin cell adenomas (39.5%), 75 null cell adenomas (22

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Bovenkamp, Daniela, Alexander Micko, Jeremias Püls, et al. "Line Scan Raman Microspectroscopy for Label-Free Diagnosis of Human Pituitary Biopsies." Molecules 24, no. 19 (2019): 3577. http://dx.doi.org/10.3390/molecules24193577.

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Pituitary adenomas are neoplasia of the anterior pituitary gland and can be subdivided into hormone-producing tumors (lactotroph, corticotroph, gonadotroph, somatotroph, thyreotroph or plurihormonal) and hormone-inactive tumors (silent or null cell adenomas) based on their hormonal status. We therefore developed a line scan Raman microspectroscopy (LSRM) system to detect, discriminate and hyperspectrally visualize pituitary gland from pituitary adenomas based on molecular differences. By applying principal component analysis followed by a k-nearest neighbor algorithm, specific hormone states w

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11

Kostyleva, D. N., P. M. Khandaeva, A. M. Lapshina, et al. "Clinical case of plurihormonal pituitary adenoma (STH/ACTH/TSH/FSH/LH-secreting), diagnostic pitfalls." Problems of Endocrinology 70, no. 4 (2024): 24–31. http://dx.doi.org/10.14341/probl13349.

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According to numerous studies, the most common pituitary tumors are prolactinomas, reaching 60% of all clinically significant adenomas, the next in order are non-functional pituitary adenomas, somatotropinomas, corticotropinomas and thyrotropinomas. Plurigormonal tumors occur in less than 1% of all pituitary adenomas. The most common form of mixed secretion adenoma in this patient population, derived from the Pit-1 cell line, produces various combinations of hormones: growth hormone (GH), prolactin (PRL), thyroid-stimulating hormone (TSH). This article presents a patient with a plurihormonal t

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12

Dutta, Aditya, Nimisha Jain, Ashutosh Rai, et al. "The outcome of TSHoma from a tertiary care institute in India." Surgical Neurology International 12 (April 14, 2021): 161. http://dx.doi.org/10.25259/sni_877_2020.

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Background: Thyroid-stimulating hormone (TSH)-secreting pituitary adenoma (TSHoma) is the rarest functioning pituitary adenoma. Methods: A retrospective analysis of eight patients of TSHomas to highlight the presentations, diagnostic challenges, and treatment outcomes. Results: Median age at diagnosis was 42 years, median latency to diagnosis was 2.5 years, and thyrotoxic and compressive symptoms were the most common presenting symptoms. At presentation, three cases were plurihormonal, six cases were on medical treatment including thyroxine, and two cases were incidentally discovered. Imaging

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13

Asaad, W., N. V. Pachuashvili, L. S. Urusova, et al. "The role of immunohistochemical analysis in improving the diagnosis accuracy of neuroendocrine tumors of the pituitary gland." Endocrine Surgery 19, no. 1 (2025): 13–22. https://doi.org/10.14341/serg13007.

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BACKGROUND: The current clinical classification and treatment principles for pituitary adenomas are primarily based on the analysis of hormone levels in blood plasma. However, this approach does not account for the heterogeneity and plurihormonal nature of these tumors, which pose significant diagnostic challenges. Immunohistochemical (IHC) analysis is a crucial tool for studying the molecular characteristics of resected tumors, enabling the identification of features that may not be apparent through standard biochemical tests and clinical evaluation.AIM: To investigate the heterogeneity and h

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Khripun, I. A., S. V. Vorobyev, N. A. Kuzmenko, E. S. Engibaryan, and V. O. Perevalova. "Clinical aspects of the plurihormonal pituitary adenoma formation." South Russian Journal of Therapeutic Practice 5, no. 2 (2024): 98–102. http://dx.doi.org/10.21886/2712-8156-2024-5-2-98-102.

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Generally pituitary tumors have a benign growth, however, there are difficulties in diagnosis and treatment because of non-specific symptoms and the inability to predict the tumor growth. In clinical practice a hormonal activity of tumors has the significant role. To a greater extent, pituitary adenomas are prolactinomas, but the hypersecretion of prolactin could be combined with an excessive production of somatotropic hormone. In this case, the clinical picture of hyperprolactinemia is accompanied by acromegaly symptoms. The presented clinical case demonstrates the main reasons for the clinic

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15

Kendler, Ady, Dino Madonna, and Douglas Laske. "PLURIHORMONAL ECTOPIC PITUITARY ADENOMA OF THE SPHENOID SINUS." Journal of Neuropathology and Experimental Neurology 57, no. 5 (1998): 524. http://dx.doi.org/10.1097/00005072-199805000-00240.

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16

Paramita, Paul, Rao Shilpa, B. N. Nandeesh, T. C. Yasha, and Santosh Vani. "Delineating the Spectrum of Pituitary Adenoma Based on the WHO 2017 Classification." Neurology India 72, no. 1 (2024): 96–101. http://dx.doi.org/10.4103/neuroindia.ni_913_20.

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Background: The WHO 2017 classification of endocrine tumors incorporates lineage-specific transcription factors (TF) and hormone expression for the classification of pituitary adenoma (PA). There is paucity of reports describing the spectrum of PA based on this classification. Objective: The aim of this study was to delineate the spectrum of PA based on WHO 2017 classification of endocrine tumors. Materials and Methods: PA diagnosed in the year 2018 were studied. H and E and hormonal immunohistochemistry (IHC) for GH, PRL, ACTH, TSH, FSH, LH, CK, T-Pit and MIB-1 were performed and the results

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17

Saeger, Wolfgang, Dieter K. Lüdecke, Michael Buchfelder, Rudolf Fahlbusch, Hans-Jürgen Quabbe, and Stephan Petersenn. "Pathohistological classification of pituitary tumors: 10 years of experience with the German Pituitary Tumor Registry." European Journal of Endocrinology 156, no. 2 (2007): 203–16. http://dx.doi.org/10.1530/eje.1.02326.

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In 1996, the German Registry of Pituitary Tumors was founded by the Pituitary Section of the German Society of Endocrinology as a reference center for collection and consultant pathohistological studies of pituitary tumors. The experiences of the first 10 years of this registry based on 4122 cases will herein be reported. The data supplement former collections of the years 1970–1995 with 3480 surgically removed tumors or lesions of the pituitary region. The cases were studied using histology, immunostainings and in some cases also molecular pathology or electron microscopy. The adenomas were c

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18

Korkmaz, H. "Plurihormonal Pituitary Adenoma: Acromegaly Associated with Subclinical Cushing’s Disease." Acta Endocrinologica (Bucharest) 11, no. 3 (2015): 389–93. http://dx.doi.org/10.4183/aeb.2015.389.

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19

Mohyeldin, Ahmed, Laurence Katznelson, and Juan Fernandez-Miranda. "Somatotroph Adenomas have a Predilection to Invade the Cavernous Sinus and Resection of the Medial Wall of the Cavernous Sinus Offers the Highest Potential for Biochemical Remission in Acromegaly." Journal of the Endocrine Society 5, Supplement_1 (2021): A648—A649. http://dx.doi.org/10.1210/jendso/bvab048.1322.

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Abstract Recurrence and remission rates vary widely among different histological subtypes of pituitary adenoma. Invasion of the medial wall of the cavernous sinus is a known mechanism that may account for such failed clinical outcomes as its removal has long been considered unattainable. The use of modern endoscopic techniques allows for direct intraoperative evaluation of invasion and resection of the medial wall of the cavernous sinus with low morbidity when performed by highly experienced surgeons. In this retrospective study we evaluated 105 consecutive primary pituitary adenomas operated

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20

Berg, Kathryn K., Bernd W. Scheithauer, Ignacio Felix, et al. "Pituitary Adenomas that Produce Adrenocorticotropic Hormone and A-Subunit: Clinicopathological, Immunohistochemical, Ultrastructural, and Immunoelectron Microscopic Studies in Nine Cases." Neurosurgery 26, no. 3 (1990): 397–403. http://dx.doi.org/10.1227/00006123-199003000-00004.

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Abstract Eight surgical and one autopsy specimen of pituitary adenomas (six cases of Cushing's disease, two of Nelson's syndrome. and one of hypopituitarism) were studied by histochemical, immunohistocytological, and ultrastructural methods. Eight tumors showed the characteristic histochemical profile of corticotroph adenoma—amphophilic to basophilic, and periodic acid-Schiff-positive to some extent. In all tumors, immunohistochemical studies revealed adrenocorticotropic hormone (ACTH) and à-subunit in the cytoplasm of some adenoma cells. By electron microscopy, seven tumors were found to be m

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21

A, Ida Ilyani, Kang WH, Norasyikin AW, et al. "A RARE CASE OF DOUBLE ADRENOCORTICOTROPIC HORMONE-SECRETING PITUITARY ADENOMA." Journal of the ASEAN Federation of Endocrine Societies 34 (July 17, 2019): 40–41. https://doi.org/10.15605/jafes.034.s66.

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INTRODUCTIONWhen distinct pituitary hypersecretory manifestations coexist, the differential diagnoses include plurihormonal or multiple pituitary adenomas. We describe a rare case of Cushing’s disease and central diabetes insipidus caused by two non-contiguous pituitary adenomas, one located within the anterior pituitary and the other in the infundibulum. CASEA 14-year-old female presented with unexplained weight gain, central obesity, hirsutism, polyuria and polydipsia. Hormonal studies indicated Cushing’s disease, and magnetic resonance imaging showed a small focal area of delayed enhancemen

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Mori, Ryosuke, Naoko Inoshita, Junko Takahashi-Fujigasaki, et al. "Clinicopathological Features of Growth Hormone-Producing Pituitary Adenomas in 242 Acromegaly Patients: Classification according to Hormone Production and Cytokeratin Distribution." ISRN Endocrinology 2013 (January 21, 2013): 1–8. http://dx.doi.org/10.1155/2013/723432.

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The aim of this study was to clarify the relationship between the histological features of GH-producing adenomas surgically resected at the Toranomon Hospital and the clinical features of the patients. Histological examinations, including immunohistochemistry for anterior pituitary hormones and cytokeratin (CK), were performed on 242 consecutively excised GH-producing pituitary adenomas. Immunohistochemistry showed 45% of the adenomas to be monohormonal and 55% to be plurihormonal, producing GH-PRL (77%), GH-TSH (13%), and GH-PRL-TSH (10%). One-fourth of the monohormonal GH adenomas had a dot-

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23

Rachmasari, Kharisa. "Abstract #807046: Cyclical Plurihormonal Pituitary Adenoma: The Ups and Downs." Endocrine Practice 26 (May 2020): 231–32. http://dx.doi.org/10.1016/s1530-891x(20)39795-0.

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24

Al-Sharim, M., B. W. Scheithauer, E. Horvath, et al. "Plurihormonal gonadotroph cell pituitary adenoma: report of a unique case." Clinical Neuropathology 28, no. 05 (2009): 182–87. http://dx.doi.org/10.5414/npp28182.

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Raverot, Gerald, Alexandre Vasiljevic, Emmanuel Jouanneau, and Hélène Lasolle. "Confirmation of a new therapeutic option for aggressive or dopamine agonist-resistant prolactin pituitary neuroendocrine tumors." European Journal of Endocrinology 181, no. 2 (2019): C1—C3. http://dx.doi.org/10.1530/eje-19-0359.

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Recent publications suggested that pasireotide could be a good therapeutic option in some dopamine-resistant or aggressive prolactinomas. We discussed the two published cases and describe another case of poorly differentiated plurihormonal PIT-1-positive adenoma with moderate SSTR2 expression and intense STTR5 expression successfully treated with PAS-LAR 40 mg/month.

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Malik, Maham, Brihant Sharma, Ayushi Sood, Omar Syed, Khyati Khattar, and Brian Jameson. "PSUN346 TSHoma: A Rare Cause of Pituitary Adenoma." Journal of the Endocrine Society 6, Supplement_1 (2022): A574—A575. http://dx.doi.org/10.1210/jendso/bvac150.1192.

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Abstract Introduction The TSH-producing adenoma (TSHoma) prevalence in the general population is 1 to 2 cases per million, accounting for less than 2% of all pituitary adenomas. Plurihormonal tumors account for about 30% of these cases, with the most common co-secreted hormone being GH. We present the case of a patient diagnosed with TSH adenoma many years after presentation. Case Presentation A 58 year old male with a history of incidentally diagnosed pituitary microadenoma on MRI in 2007, presented in 2014 following abnormal thyroid function testing (TFT), only reported symptoms being occasi

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Zada, Gabriel, Whitney W. Woodmansee, Shakti Ramkissoon, Jordan Amadio, Vania Nose, and Edward R. Laws. "Atypical pituitary adenomas: incidence, clinical characteristics, and implications." Journal of Neurosurgery 114, no. 2 (2011): 336–44. http://dx.doi.org/10.3171/2010.8.jns10290.

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Object The 2004 WHO classification of pituitary adenomas now includes an “atypical” variant, defined as follows: MIB-1 proliferative index greater than 3%, excessive p53 immunoreactivity, and increased mitotic activity. The authors review the incidence of this atypical histopathological subtype and its correlation with tumor subtype, invasion, and surgical features. Methods The records of 121 consecutive patients who underwent transsphenoidal surgery for pituitary adenomas during an 18-month period were retrospectively reviewed for evidence of atypical adenomas. Results Eighteen adenomas (15%)

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Roca, Elena, Pier Paolo Mattogno, Teresa Porcelli, et al. "Plurihormonal ACTH-GH Pituitary Adenoma: Case Report and Systematic Literature Review." World Neurosurgery 114 (June 2018): e158-e164. http://dx.doi.org/10.1016/j.wneu.2018.02.120.

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Trivedi, Priti, and Sanjiban Patra. "A unique coexistence of a plurihormonal pituitary adenoma with granulomatous hypophysitis." Indian Journal of Pathology and Microbiology 66, no. 3 (2023): 618. http://dx.doi.org/10.4103/ijpm.ijpm_439_21.

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Donangelo, Ines, Song-Guang Ren, Tamar Eigler, Clive Svendsen, and Shlomo Melmed. "Sca1+ murine pituitary adenoma cells show tumor-growth advantage." Endocrine-Related Cancer 21, no. 2 (2014): 203–16. http://dx.doi.org/10.1530/erc-13-0229.

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The role of tumor stem cells in benign tumors such as pituitary adenomas remains unclear. In this study, we investigated whether the cells within pituitary adenomas that spontaneously develop in Rb+/− mice are hierarchically distributed with a subset being responsible for tumor growth. Cells derived directly from such tumors grew as spheres in serum-free culture medium supplemented with epidermal growth factor and basic fibroblast growth factor. Some cells within growing pituitary tumor spheres (PTS) expressed common stem cell markers (Sca1, Sox2, Nestin, and CD133), but were devoid of hormone

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Cross, Emily, Richard Moreland, and Seth Wallack. "Feline Pituitary-Dependent Hyperadrenocorticism and Insulin Resistance Due to a Plurihormonal Adenoma." Topics in Companion Animal Medicine 27, no. 1 (2012): 8–20. http://dx.doi.org/10.1053/j.tcam.2011.12.001.

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Morinaga, Yusuke, Kouhei Nii, Kimiya Sakamoto, Ritsurou Inoue, Takafumi Mitsutake, and Hayatsura Hanada. "Case of Acromegaly Caused by Rathke Cleft Cyst Mimicking Plurihormonal Pituitary Adenoma." World Neurosurgery 126 (June 2019): 570–75. http://dx.doi.org/10.1016/j.wneu.2019.02.163.

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Egensperger, R., B. Scheithauer, E. Horvath, et al. "Cushing's disease due to plurihormonal adrenocorticotropic hormone and gonadotropin-producing pituitary adenoma." Acta Neuropathologica 102, no. 4 (2001): 398–403. http://dx.doi.org/10.1007/s004010100376.

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Nachankar, Amit S., Vikram Singh Shekhawat, Sachin Modi, Mandeep Sharma, Ashish Kumar, and Mantha Santosha. "Plurihormonal pituitary neuroendocrine tumor causing acromegaly with subclinical Cushing’s disease." IJEM Case Reports 2, no. 3 (2024): 96–100. http://dx.doi.org/10.4103/ijemcr.ijemcr_3_24.

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ABSTRACT Macrosomatotropinoma is the most common cause of acromegaly. Plurihormonal pituitary neuroendocrine tumor causing acromegaly often has growth hormone (GH) and prolactin excess. Here, we present a case of acromegaly with adrenocorticotrophic hormone (ACTH) excess due to macrosomatocorticotropinoma. A 32 years old man presented with left-sided progressive painless loss of vision. Clinically, he had acromegaloid features with overt diabetes mellitus (DM) without any discriminatory features of Cushing’s disease or hypertension. The hormonal evaluation revealed elevated insulin-like growth

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Robinson, Kathleen, and Brian O’Neill. "A Case of TSH-Secreting Pituitary Adenoma With Unusual Positive Staining for SF1." Journal of the Endocrine Society 5, Supplement_1 (2021): A911—A912. http://dx.doi.org/10.1210/jendso/bvab048.1861.

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Abstract Background: TSH-secreting pituitary adenomas are the rarest functional pituitary tumors. While they are often plurihormonal, SF1 positivity (indicating gonadotroph lineage) is unusual. Here we present the rare case of a patient with TSH-secreting pituitary adenoma with staining positive for TSH, GH, prolactin, SSTR2A, PIT-1, and SF1. Clinical Case: Patient is a 42 year-old male with a history of hypogonadotropic hypogonadism (lost to follow-up), who presented with a 5 day history of nausea and vomiting. Vitals were notable for tachycardia to 108 BPM, with BMI 14.8 kg/m2. On physical e

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Chin, Sang Ouk, Jin-Kyung Hwang, Sang Youl Rhee, et al. "Acromegaly due to a Macroinvasive Plurihormonal Pituitary Adenoma and a Rectal Carcinoid Tumor." Endocrinology and Metabolism 30, no. 3 (2015): 389. http://dx.doi.org/10.3803/enm.2015.30.3.389.

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Villa, Alessandro, Mariarosaria Cervasio, Marialaura Del Basso De Caro, and Luigi Maria Cavallo. "A rare case of ACTH-LH plurihormonal pituitary adenoma: letter to the editor." Acta Neurochirurgica 156, no. 7 (2014): 1389–91. http://dx.doi.org/10.1007/s00701-014-2027-7.

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Maraqa, Sima, Lina Bushnaq, and Julie M. Silverstein. "ODP286 A Case of Synchronous Growth Hormone and Adrenocorticotrophic Hormone Secretion in a Patient with an Incidental Pituitary Adenoma." Journal of the Endocrine Society 6, Supplement_1 (2022): A480. http://dx.doi.org/10.1210/jendso/bvac150.997.

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Abstract The most common type of co-secreting functional pituitary adenoma is a tumor-producing growth hormone and prolactin. Other types of co-existing functioning adenomas are extremely rare. We present a 68-year-old male patient with a pituitary adenoma presenting with acromegaly and Cushing's disease (CD). A 68-year-old male was referred for an incidental pituitary macroadenoma found on magnetic resonance imaging (MRI) for his chronic nasal congestion. He denied headaches or blurry vision. He reported an increase in shoe size, worsening hypertension and diabetes, and a 40-pounds weight gai

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Schwehr, Megan, and Mayumi Endo. "Pleurihormonal Pituitary Adenomas Presenting as Acromegaly." Journal of the Endocrine Society 5, Supplement_1 (2021): A602. http://dx.doi.org/10.1210/jendso/bvab048.1227.

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Abstract Background: Pleurihormonal pituitary adenomas represent 10-15% of all functioning pituitary adenomas1. One of the rarest combinations is ACTH-GH co-secretion with concomitant acromegaly and Cushing’s disease. Clinical Case: 30-year-old female with type 2 diabetes (A1c 11.4%) developed rapidly progressive peripheral neuropathy. Labs revealed a transaminitis. Hepatic ultrasound revealed fatty infiltration of the liver consistent with steatosis. Patient endorsed life-long “chicken legs”, buffalo hump, and easy bruising. She continued to have worsening paresthesias and stated her teeth we

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Clarke, Michelle J., Dana Erickson, M. Regina Castro, and John L. D. Atkinson. "Thyroid-stimulating hormone pituitary adenomas." Journal of Neurosurgery 109, no. 1 (2008): 17–22. http://dx.doi.org/10.3171/jns/2008/109/7/0017.

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Object Thyroid-stimulating hormone (TSH)–secreting pituitary adenomas are rare, representing < 2% of all pituitary adenomas. Methods The authors conducted a retrospective analysis of patients with TSH-secreting or clinically silent TSH-immunostaining pituitary tumors among all pituitary adenomas followed at their institution between 1987 and 2003. Patient records, including clinical, imaging, and pathological and surgical characteristics were reviewed. Twenty-one patients (6 women and 15 men; mean age 46 years, range 26–73 years) were identified. Of these, 10 patients had a history of clini

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Manrique, Helard Andres, and Miguel Eduardo Pinto. "TSH, GH, and Prolactin Producing Pituitary Adenoma: An Unusual Case Presentation." Journal of the Endocrine Society 5, Supplement_1 (2021): A616. http://dx.doi.org/10.1210/jendso/bvab048.1256.

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Abstract Background: Pituitary adenomas are benign and frequent tumors, some of them with tendency to be recurrent or invasive e. several pituitary tumors are positive for secretion of one or more hormones of the same adenohypophysial cell lineage. However, some cases have a subclinical course and are underestimated because they have plurihormonal secretion of different adenohypophyseal cells lineage. Clinical Case: A 38 years old women with chronic headaches, who is in fertility treatment for 12 month ago presents to the clinic with unremarkable physical examination. Her laboratory studies in

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Sarmiento, J. Manuel, Matt S. Wi, Zhe Piao, Kritsanapol Boon-Unge, and Eric S. Stiner. "Two collision sellar lesions: Rathke's cleft cyst with non-functional pituitary adenoma and Rathke's cleft cyst with plurihormonal adenoma." journal of Cancer Therapeutics and Research 2, no. 1 (2013): 14. http://dx.doi.org/10.7243/2049-7962-2-14.

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Luk, Cynthia, Kalman Kovacs, Fabio Rotondo, Eva Horvath, Michael Cusimano, and Gillian Booth. "Plurihormonal Pituitary Adenoma Immunoreactive for Thyroid-Stimulating Hormone, Growth Hormone, Follicle-Stimulating Hormone, and Prolactin." Endocrine Practice 18, no. 5 (2012): e121-e126. http://dx.doi.org/10.4158/ep12033.cr.

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Singh, Sonum. "Abstract #763568: A Rare Plurihormonal Pituitary Adenoma: A Case Report and Review of the Literature." Endocrine Practice 26 (May 2020): 194–95. http://dx.doi.org/10.1016/s1530-891x(20)39404-0.

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Pereira, Bernardo Dias, Luísa Raimundo, Ozgur Mete, Ana Oliveira, Jorge Portugal, and Sylvia L. Asa. "Monomorphous Plurihormonal Pituitary Adenoma of Pit-1 Lineage in a Giant Adolescent with Central Hyperthyroidism." Endocrine Pathology 27, no. 1 (2015): 25–33. http://dx.doi.org/10.1007/s12022-015-9395-2.

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Iglesias, Pedro, Betina Biagetti, Marta Araujo-Castro, et al. "Clinical Significance of T2-Weighted Sequence Intensity on Magnetic Resonance Imaging in Clinically Non-Functioning Pituitary Adenomas." Experimental and Clinical Endocrinology & Diabetes 131, no. 12 (2023): 631–38. http://dx.doi.org/10.1055/a-2197-3566.

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Abstract Background Little is known about the relationship between signal intensity patterns on T2-weighted magnetic resonance imaging (MRI) in non-functioning pituitary adenomas (NFPAs). Objective In this study, the clinical, hormonal, histological features, and therapeutic responses were evaluated according to the T2 signal intensity in NFPAs. Methods This retrospective and multicenter study included a group of 166 NFPA patients (93 men, 56%, mean age 58.5 ±14.8 yr). Results: Approximately half of the tumors (n=84, 50.6%) were hyperintense, while 34.3% (n=57) and 15.1% (n=25) were iso- and h

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Третяк, О. Е., Л. В. Щекатурова, Г. А. Кидалова, and Д. Г. Когут. "Newly revealed diabetes mellitus as a manifestation of plurihormonal pituitary adenoma (GH/ACTH-producing). Case report." Clinical endocrinology and endocrine surgery, no. 4(56) (December 1, 2016): 71–76. http://dx.doi.org/10.24026/1818-1384.4(56).2016.87364.

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SHIMATSU, AKIRA, HIROYUKI MURABE, YOSHIO NAKAMURA, HARUO MIZUTA, CHIHIRO IHARA, and KAZUWA NAKAO. "Long-Term Treatment with Bromocriptine of a Plurihormonal Pituitary Adenoma Secreting Thyrotropin, Growth Hormone and Prolactin." Endocrine Journal 46, no. 1 (1999): 159–65. http://dx.doi.org/10.1507/endocrj.46.159.

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Coculescu, Mihail. "Evolution under Complex Therapy of Acromegaly Due to a Pituitary Plurihormonal Adenoma with Colocalisation of GH and FSH." Acta Endocrinologica (Bucharest) 2, no. 3 (2006): 337–48. http://dx.doi.org/10.4183/aeb.2006.337.

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Andersen, Marianne. "MANAGEMENT OF ENDOCRINE DISEASE: GH excess: diagnosis and medical therapy." European Journal of Endocrinology 170, no. 1 (2014): R31—R41. http://dx.doi.org/10.1530/eje-13-0532.

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Acromegaly is predominantly caused by a pituitary adenoma, which secretes an excess of GH resulting in increased IGF1 levels. Most of the GH assays used currently measure only the levels of the 22 kDa form of GH. In theory, the diagnostic sensitivity may be lower compared with the previous assays, which have used polyclonal antibodies. Many GH-secreting adenomas are plurihormonal and may co-secrete prolactin, TSH and α-subunit. Hyperprolactinaemia is found in 30–40% of patients with acromegaly, and hyperprolactinaemia may occasionally be diagnosed before acromegaly is apparent. Although trans-

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