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Relevant bibliographies by topics / Pneumonie lobaire

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Academic literature on the topic 'Pneumonie lobaire'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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Journal articles on the topic "Pneumonie lobaire"

1

Ray, P. "Une pneumonie lobaire moyenne." Annales françaises de médecine d'urgence 1, no. 1 (December 6, 2010): 45–46. http://dx.doi.org/10.1007/s13341-010-0010-5.

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DE VUYST E, DE KEUKELEIRE T, LARIDON E, VANDENBROUCKE F, and LACOR P. "Bilaterale lobaire pneumonie bij een hiv-patiënt." Tijdschrift voor Geneeskunde 62, no. 12 (January 1, 2006): 916–17. http://dx.doi.org/10.2143/tvg.62.12.5002496.

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3

Kané, Bourama. "Aspect épidémiologique des Pneumopathies Aigues Communautaires de l'enfant dans le Service de Pediatrie de l'Hôpital du Mali." Mali Santé Publique 10, no. 1 (July 24, 2020): 64–70. http://dx.doi.org/10.53318/msp.v10i1.1665.

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Introduction : La pneumopathie aiguë, dite communautaire, est définie comme une infection respiratoire basse avec atteinte du parenchyme pulmonaire d'évolution aigue. Elle constitue un problème de santé publique dans le monde. La pneumonie constitue l'infection respiratoire la plus grave chez l'enfant. Le but de ce travail est d'étudier les pneumopathies aiguës communautaires cinq ans après l'introduction du vaccin antipneumococcique treize valences dans la vaccination de routine au Mali. Matériel et méthodes : Il s'agissait d'une étude rétrospective portant sur les dossiers médicaux des enfants dmois à 15 ans ayant été hospitalisés du 01 janvier au 31 décembre 2017 au service de pédiatrie de l'Hôpital du Mali pour gène respiratoire, toux, douleur thoracique. Ont été admis aussi les enfants référés par d'autres structures pour pneumopathie confirmées par une radiographie du thorax. Résultats : Nous avons observé une fréquence globale de 9,46%. La moyenne d'âge était de 3 ans avec des extrêmes d'un mois à 15 ans. Le sex ratio était de 1,44. Le motif d'hospitalisation le plus fréquent était la dyspnée (72.2%). La pneumopathie était associée à la malnutrition aigüe sévère chez 18.3% de nos enfants. Le diagnostic était dominé par la pneumonie franche lobaire aigue (PFLA) (57%) suivie de pleuro-pneumopathie (26.9%) et de broncho-pneumopathie (11.8%). L'association ceftriaxone gentamycine a été utilisée chez 95.7% des enfants. La durée moyenne d'hospitalisation était de 9 jours avec des extrêmes de 0 et 21 jours. Nous avons enregistré 60,2% de taux de guérison et 10,8% de taux de mortalité avec 28.1% chez les moins de 5 ans. Des complications ont été observées chez 28% de nos enfants nécessitant un drainage chirurgical. Conclusion : Le vaccin antipneumococcique a peu d'impact sur l'incidence de la pneumonie chez les enfants de moins de 5 ans. La détermination des causes et l'identification les sérotypes sont nécessaires pour réduire efficacement son incidence.

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Lazareva, Ya V., G. B. Sokolova, and I. P. Solovieva. "Caseating pneumonia: computed tomography, differentiated therapy." PULMONOLOGIYA, no. 3 (June 28, 2005): 52–58. http://dx.doi.org/10.18093/0869-0189-2005-0-3-52-58.

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We examined 50 patients with caseating pneumonias of various types and size. We selected CT types of caseating pneumonia (acinous, lobar, lobular, peribronchial), their structure and extension. Computed tomography is found to be the optimal radiological method to detect caseating pneumonic TB forms. Chemotherapy modes for caseating pneumonia patients are presented regarding to its CT types.

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Kumar, Surinder, Arti Maria, Sanjeev R. Saigal, and Megha Maheshwari. "Mycoplasma pneumoniae as a cause of non-resolving pneumonia in a neonate." Journal of Medical Microbiology 59, no. 6 (June 1, 2010): 731–32. http://dx.doi.org/10.1099/jmm.0.017491-0.

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Mycoplasma pneumoniae is known to be the chief causative organism for community-acquired non-lobar pneumonia in children of 5–15 years of age. M. pneumoniae as an aetiological agent for pneumonia among neonates and infants has rarely been reported. We report here a case of persistent pneumonia due to M. pneumoniae in a 3-week-old neonate.

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Tian, Fang, Li-Ping Chen, Gang Yuan, Ai-Min Zhang, Yu Jiang, and Shuang Li. "Differences of TNF-α, IL-6 and Gal-3 in lobar pneumonia and bronchial pneumonia caused by mycoplasma pneumoniae." Technology and Health Care 28, no. 6 (November 17, 2020): 711–19. http://dx.doi.org/10.3233/thc-192011.

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OBJECTIVE: To investigate the differences of tumor necrosis factor-α (TNF-α), interleukin-6 (IL-6) and galectin-3 concentrations in lobar pneumonia and bronchopneumonia induced by mycoplasma pneumoniae (MP) in children and to explore these related factors predicting the severity of MP. METHODS: A total of 148 children with mycoplasma pneumoniae pneumonia (MPP) and 32 healthy controls were analyzed from March 2017 to August 2018 in our province. Clinical information was collected from the hospitalized MP patients. The 148 patients with MPP were divided into two groups: lobar pneumonia group and bronchial pneumonia group. The 32 healthy children were considered the control group. The concentrations of TNF-α, IL-6 and Gal-3 were examined in the serum of 148 children patients with MPP and 32 healthy children by double-antibody sandwich enzyme-linked immunosorbent assay (ELISA). RESULTS: The TNF-α, IL-6 and Gal-3 levels were obviously higher in both the lobar pneumonia and bronchial pneumonia groups, compared to those in the control group. Furthermore, these levels were significantly higher in the lobar pneumonia group, compared to the bronchial pneumonia group. After treatment, the levels of TNF-α, IL-6 and Gal-3 totally descended during the recovery period. CONCLUSION: There are differences in serum TNF-α, IL-6 and Gal-3 concentrations in lobar pneumonia and bronchial pneumonia caused by MP in children. In general, the TNF-α, IL-6 and Gal-3 levels were significantly higher in the lobar pneumonia group, when compared to the bronchial pneumonia group. This was because most lobar pneumonia cases are much more serious than bronchial pneumonia. Moreover, it has been proven that TNF-α, IL-6 and Gal-3 may play an important role in the pathogenesis development of MPP. At the same time, these are important issues in diagnosing MPP.

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Rani, Mamta, Rajesh K. Gupta, and S. Chhibber. "Protection against Klebsiella pneumoniae induced lobar pneumonia in rats with lipopolysaccharide and related antigens." Canadian Journal of Microbiology 36, no. 12 (December 1, 1990): 885–90. http://dx.doi.org/10.1139/m90-153.

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The immunoprotective role of lipopolysaccharide and related antigens from Klebsiella pneumoniae was studied in a lobar pneumonia model developed in rats. Various antigens were obtained by different chemical treatments of the lipopolysaccharide. All these antigens (purified lipopolysaccharide, reduced lipopolysaccharide, lipopolysaccharide – bovine serum albumin complex, and lipid A – bovine serum albumin complex were tested for pyrogenicity and the Shwartzman reaction. The lipopolysaccharide and the various related antigens were pyrogenic and elicited a positive Shwartzman reaction at high concentrations. However, at low concentrations, the same preparations did not show any side effects. All these antigens, on the other hand, were protective against bacterial challenge in Klebsiella pneumoniae induced lobar pneumonia in rats, as the bacterial colonization of lungs in the immunized animals was significantly lower when compared with the controls. The alveolar macrophages from these animals also showed significantly more uptake of Klebsiella pneumoniae as compared with those obtained from control animals. Key words: lipopolysaccharide, vaccine, pneumonia, protection.

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Ji, Chenhui, Min Wang, and Xiaocheng Fan. "Water Extract of Gallnut Reduces the Injury of Alveolar Epithelial Cells Induced by Streptococcus pneumoniae by Up-Regulating miRNA-338-3p." Journal of Biomaterials and Tissue Engineering 11, no. 10 (October 1, 2021): 1969–76. http://dx.doi.org/10.1166/jbt.2021.2775.

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Streptococcus pneumoniae (S. pneumoniae) is the primary pathogen causing pneumonia, in addition to lobar pneumonia, meningitis, bronchitis, and other diseases. Inhibiting the apoptosis and inflammation of alveolar epithelial cells is essential for the treatment of pneumonia caused by S. pneumoniae. Traditional Chinese medicine has the characteristics of multiple components, multiple targets, and few adverse reactions. It is recognized by doctors and patients in the treatment of pneumonia and other diseases. We conducted this study to explore the effect of the water extract of gallnut on alveolar epithelial cells affected by S. pneumoniae. Studies have found that the water extract of gallnut can increase the optical density value, Bcl-2 protein expression, IL-10 content, and miRNA-338-3p levels of alveolar epithelial cells affected by S. pneumoniae. Additionally, it can reduce the rate of cell apoptosis, Bax protein expression, and IL-6 content. Further, its effect is dose-dependent: the higher the concentration of gallnut water extract, the more evident its effect on alveolar epithelial cells. Through nano PCR detection, it was found that overexpression of miRNA-338-3p can increase the activity of alveolar epithelial cells affected by S. pneumoniae and promote cell growth. Knockdown of miRNA-338-3p reduced the impact of the water extract of gallnut on the growth of alveolar epithelial cells and the expression of inflammatory factors affected by S. pneumoniae. Therefore, our findings suggest that the water extract of gallnut could inhibit the apoptosis of alveolar epithelial cells affected by S. pneumoniae by up-regulating the expression of miRNA-338-3p.

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Yang, Eun Ae, Mi Hyeon Gang, Sun Young You, Jin Hwan Kim, and Jae Ho Lee. "Clinical Characteristics of Children with Lobar Pneumonia Caused byMycoplasma pneumoniae." Pediatric Allergy and Respiratory Disease 22, no. 3 (2012): 256. http://dx.doi.org/10.7581/pard.2012.22.3.256.

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Shinozaki, Taro, Kotaro Sasahara, Eri Iwami, Aoi Kuroda, Tatsu Matsuzaki, Takahiro Nakajima, and Takeshi Terashima. "A Case of Influenza B and Mycoplasma pneumoniae Coinfection in an Adult." Case Reports in Infectious Diseases 2018 (November 25, 2018): 1–4. http://dx.doi.org/10.1155/2018/3529358.

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A 19-year-old woman was referred to our hospital because of a persistent fever and cough that lasted for over a week. Influenza B virus infection was diagnosed using the rapid test kit. Initially, the patient was diagnosed with influenza B infection associated with lobar pneumonia and treated with an anti-influenza virus drug and sulbactam/ampicillin. The patient’s fever persisted, and her respiratory condition worsened. On day 5, a computed tomography (CT) scan revealed an extension of the consolidation areas in the left lung and new opacities in the right lung. The antibiotic treatment was changed to meropenem and levofloxacin, and the patient’s physical condition gradually improved. A sputum sample revealed the presence of Mycoplasma pneumoniae-specific DNA. Both influenza B virus and M. pneumoniae infections were confirmed serologically. This was a case of coinfection with influenza B virus and M. pneumoniae in a healthy young woman. The M. pneumoniae pneumonia diagnosis was delayed because the predominant feature observed in the CT scan was dense consolidation. M. pneumoniae should be considered as one of the causative pathogens in influenza coinfection cases with CT scan images presenting dense consolidation.

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Dissertations / Theses on the topic "Pneumonie lobaire"

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Vignet, Laure. "Enquête nîmoise à propos de 40 patients hospitalisés pour une pneumonie à pneumocoques, d'octobre 1991 à juillet 1992." Montpellier 1, 1993. http://www.theses.fr/1993MON11190.

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Champagne, Marie-Eve. "Ré-infections avec Streptococcus pneumoniae : effet sur les réponses immunes innée et acquise lors d'une pneumonie à pneumocoque." Master's thesis, Université Laval, 2007. http://hdl.handle.net/20.500.11794/19368.

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Gauthier, Jean-François. "Rôle des peptides n-formylés et des chimiokines dans le recrutement neutrophilique lors d'une pneumonie à pneumocoque." Thesis, Université Laval, 2007. http://www.theses.ulaval.ca/2007/24278/24278.pdf.

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4

Le, Thomas Isabelle. "Histoire naturelle de la colonisation nasopharyngée par Streptococcus pneumoniae chez l'enfant vivant en crèche fermée." Paris 5, 1998. http://www.theses.fr/1998PA05P205.

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Pasta, Franck. "Etude des mécanismes de l'hyper-recombinaison chez streptococcus pneumoniae." Toulouse 3, 1992. http://www.theses.fr/1992TOU30124.

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Lors de la transformation chez streptococcus pneumoniae, l'adn entrant est sous forme simple-brin et vient d'apparier au chromosome recepteur. Si une heterologie existe entre ces deux partenaires, il y a formation d'un heteroduplex. La reparation des heteroduplex par la bacterie, confere a certaines mutations un comportement particulier: baisse d'efficacite de transformation ou hyper-recombinaison. La premiere partie du travail est consacree a la mutation ami36. Quand on realise un croisement impliquant ami36 et une autre mutation du locus ami, on observe un exces de recombinants ami#+, eu egard a la distance qui separe les deux sites mutants. Il y a hyper-recombinaison. Une reparation du mesappariement a/g en paire c. G, semblait a l'origine de ce comportement. Ce travail montre que la reparation resulte d'une excision resynthese catalysee par la dna polymerase i. La seconde partie est consacree aux longues heterologies. Six insertions ont ete introduites dans le locus ami. Ces mutations induisent une hyper-recombinaison qui augmente avec la taille de l'heterologie et qui reste independante de la replication et de la dna polymerase i. Cette hyper-recombinaison resulterait d'une exclusion frequente des longues heterologies hors de la structure heteroduplex, et non pas d'une reparation specifique de l'heteroduplex. Ces deux mecanismes, (reparation des a/g et exclusion des longues heterologies), participent sans doute a la stabilite du patrimoine hereditaire

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Guenzi, Eric. "Identification d'un système protéi͏̈que à deux composantes impliqué dans la sensibilité à la pénicilline et l'induction de la compétence chez Streptococcus pneumoniae." Toulouse 3, 1993. http://www.theses.fr/1993TOU30108.

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Jusqu'a present, la resistance a la penicilline chez streptococcus pneumoniae a ete attribuee a la production de pbps (penicillin-binding proteins) modifiees, ayant une affinite reduite pour les antibiotiques beta-lactames. A present, nous avons determine la sequence nucleotidique d'un gene ciah et des regions adjacentes impliquees dans la resistance a la cefotaxime chez streptococcus pneumoniae c306. La sequence en acide amine obtenue a partir de celle-ci, montre que la proteine ciah est une proteine transmembranaire, appartenant a la famille des signal transducing histidine kinases. La substitution d'une pro230 a une thr, qui confere a la fois la resistance a la cefotaxime et une deficience totale dans la transformation est localisee pres du residu his226, le site de phosphorylation potentiel conserve au sein de cette famille. Le gene ciah fait partir d'un operon qui comprend un response regulator suppose etre implique dans la regulation de l'activite des pbps et l'induction des genes requis lors de l'etablissement de la competence. En plus, la sequence nucleotidique d'un fragment de 3. 1 kb a ete determinee. Celle-ci contient deux repetitions de 154 bp identifiees comme box ainsi que la partie 5 terminale du gene codant pour la proteine aspartate t-rna synthetase

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Humbert, Odile. "Recombinaison entre séquences partiellement divergentes et réparation des mésappariements chez Streptococcus pneumoniae." Toulouse 3, 1994. http://www.theses.fr/1994TOU30215.

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Le systeme hex assure la reparation generalisee des mesappariements chez streptococcus pneumoniae. Il intervient pour corriger les erreurs de replication et agit en recombinaison homologue entre sequences non-identiques pour eliminer des recombinants potentiels. Des systemes apparentes existent dans tout le monde vivant. Par leur role antirecombinogene, ils pourraient etre impliques dans le controle de la fidelite des echanges genetiques, constituant une barriere a la recombinaison tant intrachromosomique que interespece. Cette hypothese est appuyee par des donnees obtenues pour le systeme mut d'escherichia coli. Mais notre etude sur l'action du systeme hex en recombinaison entre sequences partiellement divergentes (homeologues) remet en cause l'universalite de cette proposition. Les resultats obtenus montrent que le systeme hex se revele incapable d'inhiber la recombinaison entre sequences divergentes de 5%. Cette absence d'action correspond a un blocage complet (saturation) de hex du a l'exces de mesappariements presents sur l'heteroduplex forme entre sequences homeologues. La presence d'un fragment divergent unique suffit a saturer hex. Cette inhibition est egalement retrouvee en recombinaison heterospecifique. La saturation de hex est corrigee par surproduction, dans la cellule, de l'une ou l'autre des proteines du systeme, hexa ou hexb, alors que leur role dans la reparation n'est pas equivalent. Ce resultat paradoxal est explique dans un modele. L'analyse de recombinants obtenus avec de l'adn divergent dans des receptrices hex#+ et hex#- montre que la saturation n'est pas un phenomene du tout ou rien, par lequel la reparation serait soit complete, soit non initiee. En effet, de plus petites sequences sont integrees dans une souche hex#+, ce qui apparait comme le resultat d'une correction par cycles successifs, initiee aux extremites du fragment donneur vers les mesappariements les plus proches. Cette analyse a precise le modele de correction des mesappariements par le systeme hex. Un autre aspect de mon travail m'a amenee a developper, a la fois, une methode de classification des streptocoques exploitant le phenomene de saturation de hex, et un test d'identification du pneumocoque par une sonde nucleique correspondant a une sequence particuliere de cet organisme, l'element box

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Floc'h, Erwan. "Contribution à l'étude de l'épidémiologie de Streptococcus pneumoniae résistant aux bêta-lactamines." Paris 5, 1998. http://www.theses.fr/1998PA05P010.

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Dintilhac, Agnès. "Caractérisation du système ADC, perméase de type ABC impliquée dans la transformation de Streptococcus pneumoniae, et définition d'un nouveau groupe de transporteurs d'ions métalliques." Toulouse 3, 1996. http://www.theses.fr/1996TOU30333.

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Streptococcus pneumoniae est capable de transformer naturellement. Pour transformer les bacteries doivent etre dans un etat physiologique particulier appele competence. La competence est une forme de differenciation cellulaire reversible qui apparait au cours de la phase exponentielle de croissance. Son developpement s'accompagne d'un changement global du metabolisme cellulaire, et s'effectue de maniere synchrone pour l'ensemble des cellules d'une culture. Il est induit par l'accumulation extracellulaire d'une pheromone, le csp (competence stimulting peptide). Il est egalement module par des changements d'environnement tels que des modifications de concentration de certains ions, de temperature ou de ph. Afin d'identifier des genes impliques dans le phenomene de transformation, une mutagenese a ete realisee par insertion dans le chromosome d'un gene de resistance a l'erythromycine. Elle a permis de generer un mutant presentant une deficience pour un test de transformation en milieu gelose. L'analyse de la region mutee a revele une organisation typique des operons codant pour des transporteurs de type abc (atp-binding-cassette). Ce locus, appele adc (adhesine competence), presente des homologies avec des operons codant pour des adhesines de streptocoques. Un des genes du locus adc, le gene adca, coderait pour une lipoproteine adca comportant un site de fixation potentiel des metaux. Adca, avec quatorze autres lipoproteines, dont les adhesines homologues, pourraient constituer une nouvelle famille de proteines extracytoplasmiques affines du substrat, la famille 9, specifique du transport des metaux. L'analyse de la croissance d'un mutant adc en milieu synthetique gelose a revele la necessite de zinc pour une croissance optimale des cellules mutees. L'addition de zinc ameliore le taux de croissance et la transformabilite du mutant adc dans le milieu liquide c. Le zinc, dont l'exigence pour la transformation de s. Pneumoniae etait jusqu'alors inconnue, est requis immediatement apres le contact des cellules avec le csp.

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Magdelaine, François. "Etude de l'activité bactéricide de cinq associations d'antibiotiques sur cinq souches de "S. Pneumoniae" en vue d'une application thérapeutique dans le traitement des méningites à pneumocoque résistant à la péniciline G." Paris 5, 1993. http://www.theses.fr/1993PA05P061.

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Books on the topic "Pneumonie lobaire"

1

Ambulatory lobar pneumonia. [S.l: s.n., 1985.

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Newton, Pippa. Pneumonia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0129.

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Pneumonia is defined as acute infection of the pulmonary parenchyma, presenting with consistent symptoms and signs and associated with new radiographic shadowing. It may be acute or chronic in onset and involve either one area of a lung (e.g. lobar pneumonia) or be multifocal in nature. It may be community acquired or hospital acquired. Community- acquired pneumonia is defined as pneumonia occurring in an individual with no recent contact with a healthcare setting, or in a patient admitted to hospital with development of symptoms and/or signs of pneumonia within 48 hours of admission. Hospital-acquired pneumonia or nosocomial pneumonia occurs when a patient develops symptoms or signs of pneumonia after 48 hours of admission to a healthcare setting or in the context of a long-term nursing home resident. A subtype of nosocomial pneumonia is ventilator-associated pneumonia, defined as pneumonia occurring at least 48–72 hours post intubation.

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C, McCallum D., ed. Lobar pneumonia in a child aged three, high and prolonged pyrexia after all physical evidence of the pneumonia had disappeared. [S.l: s.n., 1985.

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Book chapters on the topic "Pneumonie lobaire"

1

Mitsuyama, Rei. "Lobar Pneumonia." In Essential Radiology Review, 121–22. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26044-6_32.

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Gooch, Jan W. "Lobar Pneumonia." In Encyclopedic Dictionary of Polymers, 905. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_14134.

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Davis, James W., Dana Forman, La Scienya M. Jackson, James W. Davis, Javier Garau, David N. O’Dwyer, Elisa Vedes, et al. "Lobar Pneumonia." In Encyclopedia of Intensive Care Medicine, 1337. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1839.

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Zinserling, Vsevolod. "Lobar Pneumonia." In Infectious Pathology of the Respiratory Tract, 79–88. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-66325-4_10.

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"Lobar Pneumonia." In High-Yield Imaging: Chest, 122–24. Elsevier, 2010. http://dx.doi.org/10.1016/b978-1-4160-6161-8.00052-7.

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Iqbal, Farrukh. "Lobar Pneumonia." In 100 Case Histories in Clinical Medicine for MRCP (Part 1), 41. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10001_14.

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"Lobar Pneumonia." In Thoracic Imaging, edited by Michael Galanski. Stuttgart: Georg Thieme Verlag, 2010. http://dx.doi.org/10.1055/b-0034-74039.

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Dravid, NV. "Lobar Pneumonia." In Manual of Autopsy Pathology, 10. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10461_3.

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Little, Brent P., and Travis S. Henry. "Atelectasis, Pneumonia, and Aspiration." In Chest Imaging, 71–76. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199858064.003.0013.

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Atelectasis and pneumonia are commonly encountered in the inpatient setting, particularly in the intensive care units (ICUs) where patients are intubated and seriously ill, and often subject to variety of co-morbidities. The two entities are often confused as they have overlapping imaging appearances and may coexist. Atelectasis represents incomplete expansion of the lung parenchyma, with associated loss of volume –whereas pneumonia is an infection of the parenchyma and not typically associated with volume loss. Recognition of the characteristic imaging findings of these diseases allows a confident diagnosis to be made in many cases, and a helpful differential diagnosis to be offered in others. Clues to lobar or total lung atelectasis include volume loss, mediastinal shift, fissural and hilar displacement, and a homogeneous opacity with straight borders obscuring adjacent structures (e.g., hemidiaphragm or heart border). Pneumonia may manifest with many different patterns – often nonspecific for a given organism; however, the pattern may help to narrow the differential diagnostic considerations. Aspiration most commonly manifests with dependent centrilobular nodules and/or consolidation. Chest radiographs may change rapidly in patients affected by aspiration or the more severe forms of atelectasis (i.e. lobar or whole lung) and the patient’s symptoms typically change just as rapidly, frequently developing hypoxemia and respiratory distress. While atelectasis in the inpatient setting is a common finding, outpatients who present with lobar atelectasis should be suspected to have an obstructing tumor until proven otherwise (except for patients with CF or asthma).

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Benson, Ryo E. C. "Lung Cancer: Atelectasis and Consolidation." In Chest Imaging, 269–73. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199858064.003.0047.

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The chapter titled atelectasis and consolidation discusses these specific manifestations of lung cancer. Patients with lung cancer can present with postobstructive atelectasis and/or pneumonia secondary to centrally obstructive neoplasms. Typical central primary lung cancers are squamous cell and small cell carcinomas. Atelectasis may be sublobar, lobar or may involve the entire lung. Lobar atelectasis may exhibit the S-sign of Golden or the luftsichel sign, which suggest underlying malignancy and require further evaluation with chest CT or bronchoscopy. Central lung cancers may also manifest with postobstructive lipoid pneumonia, typically without active infection. In addition, some adenocarcinomas may manifest with imaging features of consolidation due to replacement of alveolar airspaces by tumor. Therefore, consolidations in adults should be followed to complete radiographic resolution to exclude underlying malignancy.

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Conference papers on the topic "Pneumonie lobaire"

1

Suzuki, Jun, Hiroshi Sekiguchi, and Jay H. Ryu. "Pulmonary Hydatid Disease Presenting As Community-Acquired Lobar Pneumonia." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6909.

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Deepika, N., P. Vinupritha, and D. Kathirvelu. "Classification of Lobar Pneumonia by Two Different Classifiers in Lung CT Images." In 2018 International Conference on Communication and Signal Processing (ICCSP). IEEE, 2018. http://dx.doi.org/10.1109/iccsp.2018.8524384.

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Patel, S. R., A. Ammar, R. A. Khan, and M. R. Weatherly. "A Triple Threat: Acute Pulmonary Emboli, Pneumococcal Pneumonia and Congenital Bronchiectasis with Auto-Pneumonectomy and Compensatory Hyper-Lobar Expansion." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6450.

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