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1

Munthali, Mathildah T. The use of random amplified polymorphic DNA for identifying natural and tissue-culture induced variation in beet (beta vulgaris L.). University of Birmingham, 1992.

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2

Wyandt, Herman E. Human Chromosome Variation: Heteromorphism and Polymorphism. Springer Science+Business Media B.V., 2012.

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3

Wyandt, Herman E., and Vijay S. Tonk. Human Chromosome Variation: Heteromorphism and Polymorphism. Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-0896-9.

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4

Wyandt, Herman E., Golder N. Wilson, and Vijay S. Tonk. Human Chromosome Variation: Heteromorphism, Polymorphism and Pathogenesis. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-3035-2.

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5

Ivanovich, Galaziĭ Grigoriĭ, ред. Ėvoli͡u︡t͡s︡ionnai͡a︡ izmenchivostʹ vodnykh i nazemnykh zhivotnykh. Izd-vo "Nauka," Sibirskoe otd-nie, 1986.

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6

Single nucleotide polymorphisms: Methods and protocols. 2nd ed. Humana, 2009.

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7

Jonsson, Bjarni. Polymorphic segregation in arctic charr Salvelinus alpinus (L.) from Lake Vatnshlidarvatn, northern Iceland. 1996.

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8

Figueiredo, Jane Catherine. The clinical significance of family history, young age at diagnosis and polymorphic variation in breast cancer. 2006.

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9

Medjeral-Thomas, Nicholas, Anna Richards, and Matthew C. Pickering. Molecular basis of complement-mediated renal disease. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0333.

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Abnormal regulation of complement is intimately associated with C3 glomerulopathy and atypical haemolytic uraemic syndrome. Atypical haemolytic uraemic syndrome is characterized by renal thrombotic microangiopathy due to an inability to regulate complement activation along the renal endothelium. The development of thrombosis is critically dependent on the ability to activate C5. Eculizumab, a monoclonal anti-C5 antibody, is an effective therapy for this condition. C3 glomerulopathy refers to glomerular lesions characterized by accumulation of C3 in the absence of immunoglobulin. The prototypic
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10

Wilson, Golder N., Herman E. Wyandt, and Vijay S. Tonk. Human Chromosome Variation: Heteromorphism, Polymorphism and Pathogenesis. Springer, 2018.

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11

Wilson, Golder N., Herman E. Wyandt, and Vijay S. Tonk. Human Chromosome Variation: Heteromorphism, Polymorphism and Pathogenesis. Springer, 2017.

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12

XIth European Conference on Animal Blood Groups and Biochemical Polymorphism: Warsaw July 2nd–6th, 1968. Springer, 2014.

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13

Kwok, Pui-Yan. Single Nucleotide Polymorphisms: Methods and Protocols (Methods in Molecular Biology). Humana Press, 2002.

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14

May-Jean, King, ed. Human blood cells: Consequences of genetic polymorphisms and variations. Imperial College Press, 2000.

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15

1956-, Kwok Pui-Yan, ed. Single nucleotide polymorphisms: Methods and protocols. Humana Press, 2003.

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16

Minelli, Alessandro. Evolvability and Its Evolvability. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199377176.003.0007.

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No universally accepted notion of evolvability is available, focus being alternatively put onto either genetic or phenotypic change. The heuristic power of this concept is best found when considering the intricacies of the genotype→phenotype map, which is not necessarily predictable, expression of variation depending on the structure of gene networks and especially on the modularity and robustness of developmental systems. We can hardly ignore evolvability whenever studying the role of cryptic variation in evolution, the often pervious boundary between phenotypic plasticity and the expression
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17

Morell-Ducos, Fausto. COMT and morphine use in cancer pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0082.

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The landmark paper discussed in this chapter is ‘Genetic variation in the catechol-O-methyltransferase (COMT) gene and morphine requirements in cancer patients with pain’, published by Rakvåg et al. in 2008. Genetic variation contributes to differences in pain sensitivity and response to analgesics. Catecholamines are involved in the modulation of pain and are metabolized by catchol-O-methyltransferase (COMT). Genetic variability in the COMT gene may therefore contribute to differences in pain sensitivity and response to analgesics. It has been shown that a polymorphism in the COMT gene, Rs468
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18

Favre, Michel. Vereinheitlichung Vs. Graphies Multiples 1773-1822: Untersuchungen Zur Orthographischen Polymorphie in Franzosischen Theatertexten (Europaische Hochschulschriften: Reihe 13, Franzosische Sprac). Peter Lang Publishing, 2005.

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19

Walsh, Bruce, and Michael Lynch. Hitchhiking and Selective Sweeps. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198830870.003.0008.

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When a favorable allele increases in frequency, it alters the coalescent structure (the pattern of times back to a common ancestor) at linked sites relative to that under drift. This creates patterns of sequence polymorphism than can be used to potentially detect ongoing, or very recent, selection. This idea of a neutral allele hitchhiking up to high frequency when coupled to a favorable allele is the notion of a selective sweep, and this chapter reviews the considerable body of associated population-genetics theory on sweeps. Different types of sweeps leave different signatures, resulting in
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