Relevant bibliographies by topics / Polyomavirus de Merkel

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Polyomavirus de Merkel'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 June 2024

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Journal articles on the topic "Polyomavirus de Merkel"

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DeCaprio, James A. "Merkel cell polyomavirus and Merkel cell carcinoma." Philosophical Transactions of the Royal Society B: Biological Sciences 372, no. 1732 (2017): 20160276. http://dx.doi.org/10.1098/rstb.2016.0276.

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Merkel cell polyomavirus (MCPyV) causes the highly aggressive and relatively rare skin cancer known as Merkel cell carcinoma (MCC). MCPyV also causes a lifelong yet relatively innocuous infection and is one of 14 distinct human polyomaviruses species. Although polyomaviruses typically do not cause illness in healthy individuals, several can cause catastrophic diseases in immunocompromised hosts. MCPyV is the only polyomavirus clearly associated with human cancer. How MCPyV causes MCC and what oncogenic events must transpire to enable this virus to cause MCC is the focus of this essay. This art
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Ciotti, Marco, Carla Prezioso, and Valeria Pietropaolo. "An overview on human polyomaviruses biology and related diseases." Future Virology 14, no. 7 (2019): 487–501. http://dx.doi.org/10.2217/fvl-2019-0050.

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In recent years, the Polyomaviridae family grew rapidly, thanks to the introduction of high-throughput molecular techniques. To date, 14 polyomaviruses have been identified in humans but the association with human diseases has been established only for few of them. BKPyV has been associated with nephropathy in kidney transplant patients and hemorrhagic cystitis in hematopoietic stem cell transplant patients; JCPyV to progressive multifocal leukoencephalopathy, mainly in HIV-positive patients; Merkel cell polyomavirus to Merkel cell carcinoma; Trichodysplasia spinulosa polyomavirus to the rare
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Feltkamp, Mariet C. W., Siamaque Kazem, Els van der Meijden, Chris Lauber, and Alexander E. Gorbalenya. "From Stockholm to Malawi: recent developments in studying human polyomaviruses." Journal of General Virology 94, no. 3 (2013): 482–96. http://dx.doi.org/10.1099/vir.0.048462-0.

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Until a few years ago the polyomavirus family (Polyomaviridae) included a dozen viruses identified in avian and mammalian hosts. Two of these, the JC and BK-polyomaviruses isolated a long time ago, are known to infect humans and cause severe illness in immunocompromised hosts. Since 2007 an unprecedented number of eight novel polyomaviruses were discovered in humans. Among them are the KI- and WU-polyomaviruses identified in respiratory samples, the Merkel cell polyomavirus found in skin carcinomas and the polyomavirus associated with trichodysplasia spinulosa, a skin disease of transplant pat
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Yang, June F., and Jianxin You. "Regulation of Polyomavirus Transcription by Viral and Cellular Factors." Viruses 12, no. 10 (2020): 1072. http://dx.doi.org/10.3390/v12101072.

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Polyomavirus infection is widespread in the human population. This family of viruses normally maintains latent infection within the host cell but can cause a range of human pathologies, especially in immunocompromised individuals. Among several known pathogenic human polyomaviruses, JC polyomavirus (JCPyV) has the potential to cause the demyelinating disease progressive multifocal leukoencephalopathy (PML); BK polyomavirus (BKPyV) can cause nephropathy in kidney transplant recipients, and Merkel cell polyomavirus (MCPyV) is associated with a highly aggressive form of skin cancer, Merkel cell c
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Mayberry, Colleen L., and Melissa S. Maginnis. "Taking the Scenic Route: Polyomaviruses Utilize Multiple Pathways to Reach the Same Destination." Viruses 12, no. 10 (2020): 1168. http://dx.doi.org/10.3390/v12101168.

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Members of the Polyomaviridae family differ in their host range, pathogenesis, and disease severity. To date, some of the most studied polyomaviruses include human JC, BK, and Merkel cell polyomavirus and non-human subspecies murine and simian virus 40 (SV40) polyomavirus. Although dichotomies in host range and pathogenesis exist, overlapping features of the infectious cycle illuminate the similarities within this virus family. Of particular interest to human health, JC, BK, and Merkel cell polyomavirus have all been linked to critical, often fatal, illnesses, emphasizing the importance of und
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Duncavage, Eric J., Binh-Minh Le, David Wang, and John D. Pfeifer. "Merkel Cell Polyomavirus." American Journal of Surgical Pathology 33, no. 12 (2009): 1771–77. http://dx.doi.org/10.1097/pas.0b013e3181ba7b73.

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Horváth, Katalin Barbara, Péter Pankovics, Zita Battyáni, Endre Kálmán, and Gábor Reuter. "A probable etiological role for Merkel cell polyomavirus in the development of Merkel cell carcinoma." Orvosi Hetilap 154, no. 3 (2013): 102–12. http://dx.doi.org/10.1556/oh.2013.29525.

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Approximately 20% of the tumours in humans are associated with contagious viral agents. Merkel cell carcinoma is a rare and highly aggressive tumour which may originate from the epidermal stratum basale, although the origin is still controversial. This tumour is most commonly found in elderly and immunocompromised patients in sun exposed areas, especially in the head and neck regions. Merkel cell carcinoma often causes a diagnostic challenge with a dramatically increasing incidence. In 2008, a DNA tumour virus, a polyomavirus (Merkel cell polyomavirus) was detected in Merkel cell carcinomas, a
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Nicol, Jérôme T. J., Rémy Robinot, Audrey Carpentier, et al. "Age-Specific Seroprevalences of Merkel Cell Polyomavirus, Human Polyomaviruses 6, 7, and 9, and Trichodysplasia Spinulosa-Associated Polyomavirus." Clinical and Vaccine Immunology 20, no. 3 (2013): 363–68. http://dx.doi.org/10.1128/cvi.00438-12.

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ABSTRACTSix new human polyomaviruses have been identified since 2008 (Merkel cell polyomavirus [MCPyV], human polyomavirus 6 [HPyV6], HPyV7, HPyV9, trichodysplasia spinulosa polyomavirus [TSPyV], and Malawi polyomavirus [MWPyV]). The presence of specific antibodies against MCPyV, HPyV6, HPyV7, HPyV9, and TSPyV in 828 Italian subjects aged 1 to 100 years was investigated by virus-like particle-based enzyme-linked immunosorbent assays (ELISAs). The findings indicate that all of these new polyomaviruses circulate widely in humans, with seroprevalences in adulthood ranging from 39.4% for HPyV9 to
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Moens, Ugo, Maria Ludvigsen, and Marijke Van Ghelue. "Human Polyomaviruses in Skin Diseases." Pathology Research International 2011 (September 12, 2011): 1–12. http://dx.doi.org/10.4061/2011/123491.

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Polyomaviruses are a family of small, nonenveloped viruses with a circular double-stranded DNA genome of ∼5,000 base pairs protected by an icosahedral protein structure. So far, members of this family have been identified in birds and mammals. Until 2006, BK virus (BKV), JC virus (JCV), and simian virus 40 (SV40) were the only polyomaviruses known to circulate in the human population. Their occurrence in individuals was mainly confirmed by PCR and the presence of virus-specific antibodies. Using the same methods, lymphotropic polyomavirus, originally isolated in monkeys, was recently shown to
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Clark, Paul, Gretchen V. Gee, Brandon S. Albright та ін. "Phosphoinositide 3′-Kinase γ Facilitates Polyomavirus Infection". Viruses 12, № 10 (2020): 1190. http://dx.doi.org/10.3390/v12101190.

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Polyomaviruses are small, non-enveloped DNA tumor viruses that cause serious disease in immunosuppressed people, including progressive multifocal leukoencephalopathy (PML) in patients infected with JC polyomavirus, but the molecular events mediating polyomavirus entry are poorly understood. Through genetic knockdown approaches, we identified phosphoinositide 3′-kinase γ (PI3Kγ) and its regulatory subunit PIK3R5 as cellular proteins that facilitate infection of human SVG-A glial cells by JCPyV. PI3Kα appears less important for polyomavirus infection than PI3Kγ. CRISPR/Cas9-mediated knockout of
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Dissertations / Theses on the topic "Polyomavirus de Merkel"

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Laude, Hélène. "Rôle du Polyomavirus de Merkel dans les carcinomes à cellules de Merkel." Phd thesis, Université René Descartes - Paris V, 2012. http://tel.archives-ouvertes.fr/tel-00801219.

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En 2008, le génome d'un nouveau virus a été caractérisé au sein d'un cancer cutané rare survenant préférentiellement chez l'immunodéprimé, le carcinome de Merkel. Ce nouveau virus appartenait à la famille des Polyomaviridae qui comprend des virus dont le caractère cancérigène chez l'animal est avéré depuis plus de 50 ans. Dénommé Polyomavirus de Merkel puisqu'il semblait lié à la survenue du cancer du même nom, il constituait le premier Polyomavirus impliqué de manière consistante dans un cancer humain. Cette implication reposant sur une étude unique limitée à 10 cas, l'objectif de notre trava
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Ho, Shek-yin, and 何碩然. "Detection of merkel cell polyomavirus in gynaecological diseases." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193567.

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Merkel cell polyomavirus (MCPyV) is an oncogenic virus exist in about 80% of Merkel Cell Carcinoma (MCC), an aggressive human skin cancer. Evidence of MCPyV existing in other kind of skin neoplasms such as cutaneous squamous cell carcinomas (SCCs) has been reported. Since the major type of cervical cancer is SCCs, MCPyV may be associated with cervical cancer tumorigenesis. A Japanese research group has documented the presence of MCPyV DNA in both cervical SCCs and cervical adenocarcinomas (ACs) from Japanese patients. Nevertheless, the association between MCPyV and cervical cancer remains inco
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Adzahar, Noor Suhana Binti. "Effects of Merkel cell polyomavirus T antigen expression on cell transformation of Merkel cells." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/15589/.

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Merkel cell carcinoma (MCC) is a rare but highly metastatic skin cancer that affects immunosuppressed individuals. The MCC tumour arises from mechanoreceptor merkel cells in the basal layer of the epidermis and is able to spread through the dermal lymphatic system. Merkel cell polyomavirus (MCPyV) has been detected in the majority of MCC tumour samples. Truncation mutations of the large tumour antigen (LT) are observed in the integrated genome rendering the virus replication defective. These replication-disabling mutations are only present in MCPyV isolates found in cancers and absent from vir
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Ferté-Chaudoy, Marion. "Virus host interactome du polyomavirus à cellules de Merkel." Thesis, Tours, 2017. http://www.theses.fr/2017TOUR3805/document.

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Le polyomavirus à cellules de Merkel est aujourd’hui reconnu comme l’agent étiologique du carcinome à cellules de Merkel (CCM). Le cycle viral et les mécanismes de l’oncogenèse viro-induite sont peu connus et les connaissances se basent essentiellement sur les études menées notamment sur le polyomavirus SV40. L’objectif des travaux de thèse était d’identifier les interactions entre les protéines virales et les protéines cellulaires lors de l’infection ou dans le contexte du carcinome à cellules de Merkel (CCM). Pour identifier ces interactions, nous avons réalisé des cribles double hybride en
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Pallan, Lalit. "Investigating T cell immunity against the oncogenic Merkel cell polyomavirus." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7165/.

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Merkel cell polyomavirus (MCV) is a causative factor in Merkel cell cancer (MCC). This aggressive skin maligrancy is associated with UV-light exposure, ageing or immunosuppression, implying immune constraint of MCC development. We examined immune control over MCV in MCC patients by comparing immune parameters with donor groups who share risk factors alongside healthy controls. This showed MCC patients had frequent and strong MCV antibody responses but no differences in responses to other polyomaviruses suggesting no general defect in humoral immunity to these viruses. MCC patients had lower fr
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Stakaitytė, Gabrielė. "Merkel cell polyomavirus small T antigen’s role in cell motility." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/15538/.

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Merkel cell carcinoma (MCC) is an aggressive skin cancer of neuroendocrine origin with a high likelihood of recurrence and metastasis. In 2008, Merkel cell polyomavirus (MCPyV) was discovered monoclonally integrated within the host genome of at least 80% of MCC tumours. MCPyV transforms and maintains MCC tumours via the expression of the large and small tumour (LT and ST) antigens. Specifically, ST is thought to be the major transforming factor in the tumourigenesis of MCC. Since the discovery of MCPyV, a number of mechanisms have been suggested to account for replication and tu- mour formatio
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MAZZIOTTA, CHIARA. "Merkel cell polyomavirus, a small DNA tumour virus, in humans." Doctoral thesis, Università degli studi di Ferrara, 2022. http://hdl.handle.net/11392/2489304.

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Merkel cell polyomavirus (MCPyV) which causes an almost ubiquitous, asymptomatic infection, rarely causes an aggressive skin tumor with limited therapeutic options, i.e., Merkel cell carcinoma (MCC). Despite serum anti-MCPyV IgGs have been detected with a relatively high prevalence in healthy individuals, previous studies reported variable rates. Innovative immunoassays are needed to determine the MCPyV serology in humans. The main aim of the present thesis was to investigate the impact of oncogenic MCPyV infection in the healthy population. To this end, a new indirect ELISA assay using two sy
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Bachiri, Kamel. "Identification des interactions oncogéniques du Polyomavirus à cellules de Merkel." Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS113.

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Le carcinome à cellules de Merkel (CCM) est un cancer de la peau agressif et de très mauvais pronostic. Ce cancer est lié à la présence du Polyomavirus à cellules de Merkel dans 80% des cas. Ce Polyomavirus exprime deux oncoprotéines virales : sT et LT tronquée. L'expression des deux antigènes T est suffisante à l'émergence du cancer et responsable de la perturbation de checkpoints de signalisation, de la modification du profil épigénétique et de l'évasion immunitaire. L'interactomique et la protéomique nous ont permis d'identifier de nombreux facteurs épigénétiques en relation avec les antigè
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Akhbari, Pouria. "Analysis of cellular transcriptomic changes induced by Merkel cell polyomavirus miRNA." Thesis, University of Bradford, 2017. http://hdl.handle.net/10454/15902.

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Merkel cell carcinoma (MCC) is a highly aggressive skin cancer with rising global incidence. Merkel cell polyomavirus (MCV) was discovered in 2008 in 80% of MCC samples and since then a causal link between MCV and the majority of MCC cases has been established. microRNAs (miRNA, miR) are a family of small non-coding RNAs which play a key role in post-transcriptional regulation of gene expression and are considered significant players in disease and development in many species. Whilst the focus of MCV research has thus far been on the oncogenic MCV early proteins, large tumour (LT) and small tu
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Knight, Laura M. "Understanding the role of Merkel cell polyomavirus oncoproteins in the cellular transformation of mammalian Merkel cells." Thesis, University of Leeds, 2014. http://etheses.whiterose.ac.uk/9280/.

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Merkel cell carcinoma (MCC) is a highly aggressive skin cancer of neuroendocrine origin with a high propensity for metastasis through the dermal lymphatic system. In 2008, Merkel cell polyomavirus (MCPyV) was discovered monoclonally integrated within the host genome of more than 80% of MCC tumours. MCPyV is known to contribute to the transformation and maintenance of MCC tumour cells through the expression of the Large and Small Tumour (LT and ST) antigens. To date, a number of mechanisms by which MCPyV T antigen expression promotes cell transformation and viral replication have been elucidate
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Books on the topic "Polyomavirus de Merkel"

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Franceschi, Silvia, Hashem B. El-Serag, David Forman, Robert Newton, and Martyn Plummer. Infectious Agents. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190238667.003.0024.

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Eleven infectious agents (seven viruses, three parasites, and one bacterium) have been classified by the International Agency for Research on Cancer as carcinogenic to humans for one or more cancer sites: hepatitis B virus; hepatitis C virus; thirteen types of human papillomavirus (HPV); human immunodeficiency virus type 1 (HIV-1); human T-cell leukemia virus type 1; Epstein-Barr virus; Kaposi sarcoma herpesvirus; Helicobacter pylori; Opisthorchis viverrini; Clonorchis sinensis; and Schistosoma haematobium. Other infectious agents, such as Merkel cell polyomavirus, Plasmodium falciparum, and c
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Book chapters on the topic "Polyomavirus de Merkel"

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Fischer, Nicole, and Adam Grundhoff. "Merkel Cell Polyomavirus." In Cancers in People with HIV and AIDS. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0859-2_8.

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Schrama, David, and Jürgen C. Becker. "Merkel Cell Polyomavirus." In Cancer Associated Viruses. Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0016-5_18.

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Fischer, Nicole, and Adam Grundhoff. "Merkel Cell Polyomavirus (MCV)." In Encyclopedia of AIDS. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-9610-6_8-1.

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Sofia, Asioli, and Costantino Ricci. "Merkel Cell Polyomavirus (MCPyV)." In Encyclopedia of Pathology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-28845-1_5057-1.

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Asioli, Sofia, and Costantino Ricci. "Merkel Cell Polyomavirus (MCPyV)." In Endocrine Pathology. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-62345-6_5057.

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Fischer, Nicole, and Adam Grundhoff. "Merkel Cell Polyomavirus (MCV)." In Encyclopedia of AIDS. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7101-5_8.

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Liu, Wei, and Jianxin You. "Merkel Cell Polyomavirus and Human Merkel Cell Carcinoma." In Viruses and Human Cancer. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-57362-1_12.

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Lowe, Garrett C., Jerry D. Brewer, and Jeremy S. Bordeaux. "Epidemiology and Genetics (Including High Risk Patients, Polyomavirus, Prognostic Factors)." In Merkel Cell Carcinoma. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6608-6_2.

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Rozenberg, Flore. "Merkel Cell Polyomavirus: Pathogenesis and Host Immune Response." In Viruses and Human Cancer. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0870-7_14.

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Shahzad, Naveed, Usman Shah Gilani, Menahil Mahmood, Fareeda Tasneem, Muhammad Farhan Ul Haque, and Iqra Hussain. "Ubiquitous Merkel Cell Polyomavirus: Causative Agent of the Rare Merkel Cell Carcinoma." In Human Viruses: Diseases, Treatments and Vaccines. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71165-8_17.

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Conference papers on the topic "Polyomavirus de Merkel"

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Rollison, Dana E., Anna R. Giuliano, Vernon K. Sondak, et al. "Abstract 3772A: Merkel cell polyomavirus infection in Merkel cell carincoma." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3772a.

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Prakash, Varsha, Sharon Wu, Alex Farrell, et al. "559 Molecular characterization of Merkel cell carcinoma and association with Merkel cell polyomavirus." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0559.

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Shi, Hao. "Abstract 3562: Merkel cell polyomavirus t-antigen regulates aggresome formation and autophagy in merkel cell carcinoma." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3562.

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Speel, Ernst-Jan M., Andreia Schmidt, M. Baumann, et al. "Abstract 5352: A high percentage of Merkel cell carcinomas is biologically associated with Merkel cell polyomavirus." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5352.

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Rollison, Dana E., Anna R. Giuliano, Jane L. Messina, et al. "Abstract 950: Merkel cell polyomavirus infection in cutaneous squamous cell carcinomas." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-950.

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Kumar, S., H. Xie, H. Shi, et al. "PO-254 Merkel cell polyomavirus T-antigen regulate microRNAs post-transcriptionally through DICER in merkel cell carcinoma." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.769.

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Haugg, Anke M., Dorit Rennspiess, Axel zur Hausen, et al. "Abstract 557: Fluorescencein situhybridization versus qPCR as means to detect Merkel cell polyomavirus in Merkel cell carcinoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-557.

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Sihto, Harri, Heli Kukko, Virve Koljonen, et al. "Abstract C237: Merkel cell polyomavirus infection-related Merkel cell carcinomas have intratumoral lymphocyte infiltrates and favorable survival." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-c237.

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Feng, Huichen, Masahiro Shuda, Hyun Jin Kwun, Xi Liu, Yuan Chang, and Patricks Moore. "Abstract PR5: Human Merkel cell polyomavirus causes Merkel cell carcinoma: Implication of viral etiology in human cancers." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-pr5.

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Frey, Alexander, Curtis Perry, Kathryn Clulo, Kelly Olino, and Jeffrey Ishizuka. "1134 A novel mRNA therapeutic vaccine for polyomavirus associated merkel cell carcinoma." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.1134.

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