Log in

Relevant bibliographies by topics / Polyomavirus de Merkel / Dissertations / Theses

To see the other types of publications on this topic, follow the link: Polyomavirus de Merkel.

Dissertations / Theses on the topic 'Polyomavirus de Merkel'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 June 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 30 dissertations / theses for your research on the topic 'Polyomavirus de Merkel.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse dissertations / theses on a wide variety of disciplines and organise your bibliography correctly.

1

Laude, Hélène. "Rôle du Polyomavirus de Merkel dans les carcinomes à cellules de Merkel." Phd thesis, Université René Descartes - Paris V, 2012. http://tel.archives-ouvertes.fr/tel-00801219.

Full text

Abstract:

En 2008, le génome d'un nouveau virus a été caractérisé au sein d'un cancer cutané rare survenant préférentiellement chez l'immunodéprimé, le carcinome de Merkel. Ce nouveau virus appartenait à la famille des Polyomaviridae qui comprend des virus dont le caractère cancérigène chez l'animal est avéré depuis plus de 50 ans. Dénommé Polyomavirus de Merkel puisqu'il semblait lié à la survenue du cancer du même nom, il constituait le premier Polyomavirus impliqué de manière consistante dans un cancer humain. Cette implication reposant sur une étude unique limitée à 10 cas, l'objectif de notre trava

APA, Harvard, Vancouver, ISO, and other styles

2

Ho, Shek-yin, and 何碩然. "Detection of merkel cell polyomavirus in gynaecological diseases." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193567.

Full text

Abstract:

Merkel cell polyomavirus (MCPyV) is an oncogenic virus exist in about 80% of Merkel Cell Carcinoma (MCC), an aggressive human skin cancer. Evidence of MCPyV existing in other kind of skin neoplasms such as cutaneous squamous cell carcinomas (SCCs) has been reported. Since the major type of cervical cancer is SCCs, MCPyV may be associated with cervical cancer tumorigenesis. A Japanese research group has documented the presence of MCPyV DNA in both cervical SCCs and cervical adenocarcinomas (ACs) from Japanese patients. Nevertheless, the association between MCPyV and cervical cancer remains inco

APA, Harvard, Vancouver, ISO, and other styles

3

Adzahar, Noor Suhana Binti. "Effects of Merkel cell polyomavirus T antigen expression on cell transformation of Merkel cells." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/15589/.

Full text

Abstract:

Merkel cell carcinoma (MCC) is a rare but highly metastatic skin cancer that affects immunosuppressed individuals. The MCC tumour arises from mechanoreceptor merkel cells in the basal layer of the epidermis and is able to spread through the dermal lymphatic system. Merkel cell polyomavirus (MCPyV) has been detected in the majority of MCC tumour samples. Truncation mutations of the large tumour antigen (LT) are observed in the integrated genome rendering the virus replication defective. These replication-disabling mutations are only present in MCPyV isolates found in cancers and absent from vir

APA, Harvard, Vancouver, ISO, and other styles

4

Ferté-Chaudoy, Marion. "Virus host interactome du polyomavirus à cellules de Merkel." Thesis, Tours, 2017. http://www.theses.fr/2017TOUR3805/document.

Full text

Abstract:

Le polyomavirus à cellules de Merkel est aujourd’hui reconnu comme l’agent étiologique du carcinome à cellules de Merkel (CCM). Le cycle viral et les mécanismes de l’oncogenèse viro-induite sont peu connus et les connaissances se basent essentiellement sur les études menées notamment sur le polyomavirus SV40. L’objectif des travaux de thèse était d’identifier les interactions entre les protéines virales et les protéines cellulaires lors de l’infection ou dans le contexte du carcinome à cellules de Merkel (CCM). Pour identifier ces interactions, nous avons réalisé des cribles double hybride en

APA, Harvard, Vancouver, ISO, and other styles

5

Pallan, Lalit. "Investigating T cell immunity against the oncogenic Merkel cell polyomavirus." Thesis, University of Birmingham, 2017. http://etheses.bham.ac.uk//id/eprint/7165/.

Full text

Abstract:

Merkel cell polyomavirus (MCV) is a causative factor in Merkel cell cancer (MCC). This aggressive skin maligrancy is associated with UV-light exposure, ageing or immunosuppression, implying immune constraint of MCC development. We examined immune control over MCV in MCC patients by comparing immune parameters with donor groups who share risk factors alongside healthy controls. This showed MCC patients had frequent and strong MCV antibody responses but no differences in responses to other polyomaviruses suggesting no general defect in humoral immunity to these viruses. MCC patients had lower fr

APA, Harvard, Vancouver, ISO, and other styles

6

Stakaitytė, Gabrielė. "Merkel cell polyomavirus small T antigen’s role in cell motility." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/15538/.

Full text

Abstract:

Merkel cell carcinoma (MCC) is an aggressive skin cancer of neuroendocrine origin with a high likelihood of recurrence and metastasis. In 2008, Merkel cell polyomavirus (MCPyV) was discovered monoclonally integrated within the host genome of at least 80% of MCC tumours. MCPyV transforms and maintains MCC tumours via the expression of the large and small tumour (LT and ST) antigens. Specifically, ST is thought to be the major transforming factor in the tumourigenesis of MCC. Since the discovery of MCPyV, a number of mechanisms have been suggested to account for replication and tu- mour formatio

APA, Harvard, Vancouver, ISO, and other styles

7

MAZZIOTTA, CHIARA. "Merkel cell polyomavirus, a small DNA tumour virus, in humans." Doctoral thesis, Università degli studi di Ferrara, 2022. http://hdl.handle.net/11392/2489304.

Full text

Abstract:

Merkel cell polyomavirus (MCPyV) which causes an almost ubiquitous, asymptomatic infection, rarely causes an aggressive skin tumor with limited therapeutic options, i.e., Merkel cell carcinoma (MCC). Despite serum anti-MCPyV IgGs have been detected with a relatively high prevalence in healthy individuals, previous studies reported variable rates. Innovative immunoassays are needed to determine the MCPyV serology in humans. The main aim of the present thesis was to investigate the impact of oncogenic MCPyV infection in the healthy population. To this end, a new indirect ELISA assay using two sy

APA, Harvard, Vancouver, ISO, and other styles

8

Bachiri, Kamel. "Identification des interactions oncogéniques du Polyomavirus à cellules de Merkel." Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS113.

Full text

Abstract:

Le carcinome à cellules de Merkel (CCM) est un cancer de la peau agressif et de très mauvais pronostic. Ce cancer est lié à la présence du Polyomavirus à cellules de Merkel dans 80% des cas. Ce Polyomavirus exprime deux oncoprotéines virales : sT et LT tronquée. L'expression des deux antigènes T est suffisante à l'émergence du cancer et responsable de la perturbation de checkpoints de signalisation, de la modification du profil épigénétique et de l'évasion immunitaire. L'interactomique et la protéomique nous ont permis d'identifier de nombreux facteurs épigénétiques en relation avec les antigè

APA, Harvard, Vancouver, ISO, and other styles

9

Akhbari, Pouria. "Analysis of cellular transcriptomic changes induced by Merkel cell polyomavirus miRNA." Thesis, University of Bradford, 2017. http://hdl.handle.net/10454/15902.

Full text

Abstract:

Merkel cell carcinoma (MCC) is a highly aggressive skin cancer with rising global incidence. Merkel cell polyomavirus (MCV) was discovered in 2008 in 80% of MCC samples and since then a causal link between MCV and the majority of MCC cases has been established. microRNAs (miRNA, miR) are a family of small non-coding RNAs which play a key role in post-transcriptional regulation of gene expression and are considered significant players in disease and development in many species. Whilst the focus of MCV research has thus far been on the oncogenic MCV early proteins, large tumour (LT) and small tu

APA, Harvard, Vancouver, ISO, and other styles

10

Knight, Laura M. "Understanding the role of Merkel cell polyomavirus oncoproteins in the cellular transformation of mammalian Merkel cells." Thesis, University of Leeds, 2014. http://etheses.whiterose.ac.uk/9280/.

Full text

Abstract:

Merkel cell carcinoma (MCC) is a highly aggressive skin cancer of neuroendocrine origin with a high propensity for metastasis through the dermal lymphatic system. In 2008, Merkel cell polyomavirus (MCPyV) was discovered monoclonally integrated within the host genome of more than 80% of MCC tumours. MCPyV is known to contribute to the transformation and maintenance of MCC tumour cells through the expression of the Large and Small Tumour (LT and ST) antigens. To date, a number of mechanisms by which MCPyV T antigen expression promotes cell transformation and viral replication have been elucidate

APA, Harvard, Vancouver, ISO, and other styles

11

Nicol, Jérôme. "Nouveaux polyomavirus : épidemiologie et partenaires cellulaires des protéines mineures de capside du polyomavirus à cellules de Merkel." Thesis, Tours, 2014. http://www.theses.fr/2014TOUR3803/document.

Full text

Abstract:

Les polyomavirus sont des virus très prévalents dans la population générale. Chez les sujets immunodéprimés, quatre polyomavirus sont associés à des pathologies. Parmi ces virus, le MCPyV est quant à lui responsable du carcinome à cellules de Merkel. Son implication dans un cancer humain a conduit à un regain d’intérêt pour la famille des Polyomaviridae. Au cours de ma thèse, nous nous somme intéressés à l’épidémiologie de six nouveaux polyomavirus humains (MCPyV, HPyV6, HPyV7, TSPyV, HPyV9 et MWPyV). Ces études ont montré que ces virus sont très répandus dans la population générale et que les

APA, Harvard, Vancouver, ISO, and other styles

12

Yoon, Rosa. "Merkel Cell Polyomavirus Small T Antigen Perturbs the Cellular DNA Damage Response." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17463129.

Full text

Abstract:

Merkel cell polyomavirus (MCPyV) small T antigen (ST) is expressed in the majority of Merkel cell carcinomas (MCC), a highly lethal and aggressive cancer of the skin. Since the discovery of MCPyV in 2008, the role of ST in the context of the virus and MCC has been under intense investigation. Much of our knowledge of polyomavirus ST comes from research on other polyomaviruses, including mouse polyomavirus (MPyV) and simian virus 40 (SV40). Both MPyV and SV40 ST contribute to transformation in part by binding to and inhibiting the cellular phosphatase PP2A. Likewise, MCPyV ST interacts with PP2

APA, Harvard, Vancouver, ISO, and other styles

13

Samimi, Mahtab. "Marqueurs pronostiques dans une cohorte historico-prospective de carcinomes de Merkel." Thesis, Tours, 2016. http://www.theses.fr/2016TOUR3801.

Full text

Abstract:

Le carcinome de Merkel est un cancer cutané de différenciation neuroendocrine rare, mais agressif, dont le facteur étiologique principal est le polyomavirus de Merkel (MCPyV). L’objectif de ce travail a été d’identifier des marqueurs virologiques et cellulaires pronostiques ou théranostiques à l’aide d’une cohorte historicoprospective de patients ayant un carcinome de Merkel. Les patients ayant des titres élevés d’anticorps dirigés contre la protéine de capside VP1 du MCPyV ont un pronostic favorable, tandis les anticorps dirigés contre les oncoprotéines virales reflètent l’évolution tumorale.

APA, Harvard, Vancouver, ISO, and other styles

14

Abdul-Sada, Hussein Katai. "Modulation of innate immune signaling by the small T antigen of Merkel cell polyomavirus, the causative agent of Merkel cell skin cancer." Thesis, University of Leeds, 2016. http://etheses.whiterose.ac.uk/13363/.

Full text

Abstract:

Merkel cell Polyomavirus (MCPyV) is implicated in the pathogenesis of Merkel cell carcinoma (MCC), a rare and highly aggressive neuroendocrine skin cancer, through expression of two oncoproteins, small T antigen (sT) and large T antigen (LT). MCPyV sT expression is essential for cell transformation, however, the mechanisms by which sT may contribute towards MCC are poorly understood. Studies from our group identified that sT is an antagonist of NF-B signalling (Griffiths et al., 2013). This thesis focuses on dissecting the molecular basis of inhibition. Co-immunoprecipitation and co-immunoflu

APA, Harvard, Vancouver, ISO, and other styles

15

Hackmann, Johannes [Verfasser], and A. S. [Akademischer Betreuer] Yazdi. "Prävalenz und Penetranz des Merkel-Zell-Polyomavirus bei Merkel-Zell-Karzinomen, deren Metastasen und diversen Hauttumoren / Johannes Hackmann ; Betreuer: A. S. Yazdi." Tübingen : Universitätsbibliothek Tübingen, 2015. http://d-nb.info/1197057919/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Barros, Fabiana Mesquita. "Detecção dos poliomavírus humanos BK, JC, de células Merkel e TSV em fluídos orais de indivíduos HIV positivos." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/23/23154/tde-25062018-105900/.

Full text

Abstract:

Os poliomavírus compõem uma grande família de vírus que causam infecções primárias geralmente na infância, e se mantem em condições subclínicas. Em situações de imunossupressão podem causar algumas doenças. Os indivíduos com HIV/AIDS frequentemente apresentam deficiência imunológica e por isso podem exibir maior risco de doenças causadas pelos poliomavírus. A utilização da saliva no diagnóstico e acompanhamento de doenças infecciosas tem sido explorado na literatura. As vantagens de usar a saliva para rastreio se pautam especialmente na coleta não invasiva e segurança no manuseio. O presente e

APA, Harvard, Vancouver, ISO, and other styles

17

VILLANI, SONIA. "DETECTION OF HUMAN POLYOMAVIRUSES AND PAPILLOMAVIRUSES IN ORAL SPECIMENS OF IMMUNOCOMPETENT AND HIV POSITIVE SUBJECTS." Doctoral thesis, Università degli Studi di Milano, 2018. http://hdl.handle.net/2434/603748.

Full text

Abstract:

ABSTRACT The oral cavity of healthy subjects is colonized by several and different microorganisms, such as viruses, bacteria, fungi and protozoa. Many of these are commensal species, which are essential to maintain a health status when in equilibrium with the environment, but can became pathogenic and capable to eliciting disease when occur environmental changes or shifts in the composition of the oral microbiota. Despite there are numerous studies concerning the bacterial communities inhabiting the oral cavity, there are very few studies defining the oral viral community and their role, if a

APA, Harvard, Vancouver, ISO, and other styles

18

Griffiths, David Alun. "The role of the Merkel cell polyomavirus small T antigen in virus replication and MCC pathogenesis." Thesis, University of Leeds, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.590485.

Full text

Abstract:

Merkel cell carcinoma (MCC) is a highly aggressive human cancer and its incidence is increasing worldwide. In 2008 a novel human polyomavirus named Merkel cell polyomavirus (MeV) was isolated from MCC tumours. MCV is monoclonally integrated into the genomic DNA of 80% of these tumours and is now recognised as a causative factor in MCC pathogenesis. The Small Tumour (ST) antigens of mammalian polyomaviruses have been demonstrated to regulate viral promoter activation and contribute to cellular transformation. Mechanisms central to these putative functions are typically dependent on an interacti

APA, Harvard, Vancouver, ISO, and other styles

19

Theiß, Juliane [Verfasser], and Nicole [Akademischer Betreuer] Fischer. "Characterization of the Merkel cell polyomavirus encoded miRNA mcv-miR-M1 / Juliane Theiß. Betreuer: Nicole Fischer." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2016. http://d-nb.info/1084213206/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Kervarrec, Thibault. "Histogenesis of Merkel cell carcinoma." Thesis, Tours, 2019. http://www.theses.fr/2019TOUR3802.

Full text

Abstract:

Le carcinome à cellules de Merkel (CCM) est un cancer neuroendocrine de la peau rare et agressif. Dans environ 80 % des cas, l'intégration génomique du polyomavirus à cellules de Merkel (MCPyV) est retrouvée au sein de ces tumeurs et la surexpression des deux oncoprotéines virales est considérée comme le principal déterminant oncogénique des tumeurs viro-positives. Cependant, la nature de la cellule au sein de laquelle cette intégration virale se produit est à ce jour inconnue. L'objectif de ce travail était de déterminer la cellule d'origine du CCM.Dans une première partie, une cohorte multic

APA, Harvard, Vancouver, ISO, and other styles

21

Martel-Jantin, Claire. "Mode de transmission, variabilité génétique et intégration virale et cellulaire du nouveau polyomavirus humain associé au carcinome à cellules de Merkel." Paris 7, 2013. http://www.theses.fr/2013PA077077.

Full text

Abstract:

Le Polyornavirus des cellules de Merkel (MCPyV) est un nouveau Polyomavirus humain récemment identifié dans des tissus tumoraux de patients atteints d'un carcinome à cellules de Merkel (MCC). Le MCC est un carcinome neuro-endocrinien de la peau, agressif, touchant principalement les patients d'origine caucasienne, âgés, parfois immunodéprimés (VIH, greffés, hémopathie maligne). Dans le cadre d'une collaboration multicentrique associant plusieurs services de dermatologie et de laboratoires d'anatomopathologie, notre Unité a développé différentes études sur ce nouveau virus oncogène. 1) Détectio

APA, Harvard, Vancouver, ISO, and other styles

22

Czech-Sioli, Manja [Verfasser], and Nicole [Akademischer Betreuer] Fischer. "Einfluss der Ubiquitin spezifischen Protease 7 auf den Lebenszyklus des Merkel Zell Polyomavirus / Manja Czech-Sioli ; Betreuer: Nicole Fischer." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2020. http://d-nb.info/1209676192/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

Robles, Hellín Claudia 1980. "Role of human polyomaviruses in lymphoproliferative disorders and bladder cancer." Doctoral thesis, Universitat Pompeu Fabra, 2014. http://hdl.handle.net/10803/319715.

Full text

Abstract:

Polyomaviruses are suspected to cause cancer in humans although to date Merkel cell polyomavirus (MCPyV) is the only proven human carcinogen. Potential associations of up to nine polyomaviruses with 468 lymphoproliferative disorders, 1135 bladder cancer and 359 chronic lymphocytic leukemia (CLL) subjects using three case-control studies in Spain has been explored. Viral exposure was measured as seroreactivity against these viruses by virus-like-particles enzyme linked immunosorbent assay and fluorescent bead-based multiplex serology technology. In explored lymphomas, only diffuse large b-cell

APA, Harvard, Vancouver, ISO, and other styles

24

Albertini, Silvia. "The Everlasting Fight between Host and Pathogens: Unveiling Common Strategies Adopted by Small DNA Tumour Viruses to Dampen Innate Immunity." Doctoral thesis, Università del Piemonte Orientale, 2018. http://hdl.handle.net/11579/104067.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Pedergnana, Vincent. "Contrôle génétique de la réponse à l’infection par des virus oncogènes en population endémique." Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05S022.

Full text

Abstract:

La recherche de facteurs génétiques de susceptibilité aux infections virale dans des populations générales exhaustives endémiques est une approche originale en épidémiologie génétique. Nos travaux de thèse nous ont permis d’établir, dans une population endémique pour deux virus oncogènes MCPyV et HHV-8 au Cameroun et dans une population endémique pour le VHC en Egypte, plusieurs arguments forts en faveur d’une susceptibilité génétique aux infections par les virus oncogènes humains définies par la séropositivité/ séronégativité vis-à-vis du virus impliqué. Concernant l’infection par le MCPyV, d

APA, Harvard, Vancouver, ISO, and other styles

26

Cibulka, Jakub. "Příprava expresních vektorů a virových mutant pro studium minoritních strukturních proteinů polyomavirů." Master's thesis, 2013. http://www.nusl.cz/ntk/nusl-322115.

Full text

Abstract:

Polyomaviruses are small non-enveloped DNA viruses infecting birds and mammals, including human. Their capsid consists of the major capsid protein, VP1, and two minor capsid proteins, VP2 and VP3. The VP2 and VP3 proteins are supposed to have an important function in the transport of viral genome into the cell nucleus, which is a key step to facilitate viral replication. VP2 and VP3 proteins of mouse polyomavirus and SV40 have an ability to bind and disrupt cellular membranes. This feature is believed to be involved in the transport of viral genome into the nucleus. Plasmids carrying genes of

APA, Harvard, Vancouver, ISO, and other styles

27

Nwogu, N., James R. Boyne, S. J. Dobson, et al. "Cellular sheddases are induced by Merkel cell polyomavirus small tumour antigen to mediate cell dissociation and invasiveness." 2018. http://hdl.handle.net/10454/16579.

Full text

Abstract:

yes<br>Merkel cell carcinoma (MCC) is an aggressive skin cancer with a high propensity for recurrence and metastasis. Merkel cell polyomavirus (MCPyV) is recognised as the causative factor in the majority of MCC cases. The MCPyV small tumour antigen (ST) is considered to be the main viral transforming factor, however potential mechanisms linking ST expression to the highly metastatic nature of MCC are yet to be fully elucidated. Metastasis is a complex process, with several discrete steps required for the formation of secondary tumour sites. One essential trait that underpins the ability of ca

APA, Harvard, Vancouver, ISO, and other styles

28

Vochyánová, Klára. "Příprava monoklonálních protilátek proti proteinu VP2 lidských polyomavirů." Master's thesis, 2013. http://www.nusl.cz/ntk/nusl-322097.

Full text

Abstract:

Aim of this diploma thesis was to prepare two protein antigens and two monoclonal antibodies, all based on VP2 minor protein of human polyomaviruses BK virus and Merkel Cell Polyomavirus. One monoclonal antibody was being prepared against unique part of VP2 protein (N-terminal epitope, not present in VP3 protein). A cell line producing such monoclonal antibody has never been established before due to low immunogenicity of the epitope. Our approach was successful in terms of mouse immunization, however, serious problems with hybridoma line stability appeared later during the preparation process

APA, Harvard, Vancouver, ISO, and other styles

29

Akhbari, Pouria, Desmond J. Tobin, Krzysztof Poterlowicz, W. Roberts, and James R. Boyne. "MCV-miR-M1 targets the host-cell immune response resulting in the attenuation of neutrophil chemotaxis." 2018. http://hdl.handle.net/10454/15651.

Full text

Abstract:

Yes<br>Virus-encoded miRNAs are emerging as key regulators of persistent infection and host-cell immune evasion. Merkel cell polyomavirus (MCPyV), the predominant aetiological agent of Merkel cell carcinoma (MCC), encodes a single miRNA, MCV-miR-M1, which targets the oncogenic MCPyV large T antigen (LT). MCV-miR-M1 has previously been shown to play an important role in establishment of long-term infection, however, the underlying mechanism is not fully understood. A key unanswered question is whether, in addition to auto-regulating LT, MCV-miR-M1 also targets cellular transcripts to orchestra

APA, Harvard, Vancouver, ISO, and other styles

30

Sauerová, Pavla. "Studium vlastností genových produktů Polyomaviru karcinomu Merkelových buněk : Příprava protilátek a konstrukce expresních vektorů." Master's thesis, 2013. http://www.nusl.cz/ntk/nusl-322774.

Full text

Abstract:

Merkel cell polyomavirus (MCPyV) is a recently discovered human virus, having it's genome often integrated in a genome of Merkel carcinoma cells. Although this type of carcinoma is not so usual, it is very aggressive and it's incidence has been rising in last few years. It is not surprising that this virus is nowadays in the centre of scientific interest, as well as other pathogens and mechanisms affecting human life. Because the virus was discovered not so long ago, its research has been at the whole beginning. This diploma thesisaims to contribute to the study of this virus from the molecula

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!