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1

DeCaprio, James A. "Merkel cell polyomavirus and Merkel cell carcinoma." Philosophical Transactions of the Royal Society B: Biological Sciences 372, no. 1732 (2017): 20160276. http://dx.doi.org/10.1098/rstb.2016.0276.

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Merkel cell polyomavirus (MCPyV) causes the highly aggressive and relatively rare skin cancer known as Merkel cell carcinoma (MCC). MCPyV also causes a lifelong yet relatively innocuous infection and is one of 14 distinct human polyomaviruses species. Although polyomaviruses typically do not cause illness in healthy individuals, several can cause catastrophic diseases in immunocompromised hosts. MCPyV is the only polyomavirus clearly associated with human cancer. How MCPyV causes MCC and what oncogenic events must transpire to enable this virus to cause MCC is the focus of this essay. This art
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2

Ciotti, Marco, Carla Prezioso, and Valeria Pietropaolo. "An overview on human polyomaviruses biology and related diseases." Future Virology 14, no. 7 (2019): 487–501. http://dx.doi.org/10.2217/fvl-2019-0050.

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In recent years, the Polyomaviridae family grew rapidly, thanks to the introduction of high-throughput molecular techniques. To date, 14 polyomaviruses have been identified in humans but the association with human diseases has been established only for few of them. BKPyV has been associated with nephropathy in kidney transplant patients and hemorrhagic cystitis in hematopoietic stem cell transplant patients; JCPyV to progressive multifocal leukoencephalopathy, mainly in HIV-positive patients; Merkel cell polyomavirus to Merkel cell carcinoma; Trichodysplasia spinulosa polyomavirus to the rare
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3

Feltkamp, Mariet C. W., Siamaque Kazem, Els van der Meijden, Chris Lauber, and Alexander E. Gorbalenya. "From Stockholm to Malawi: recent developments in studying human polyomaviruses." Journal of General Virology 94, no. 3 (2013): 482–96. http://dx.doi.org/10.1099/vir.0.048462-0.

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Until a few years ago the polyomavirus family (Polyomaviridae) included a dozen viruses identified in avian and mammalian hosts. Two of these, the JC and BK-polyomaviruses isolated a long time ago, are known to infect humans and cause severe illness in immunocompromised hosts. Since 2007 an unprecedented number of eight novel polyomaviruses were discovered in humans. Among them are the KI- and WU-polyomaviruses identified in respiratory samples, the Merkel cell polyomavirus found in skin carcinomas and the polyomavirus associated with trichodysplasia spinulosa, a skin disease of transplant pat
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4

Yang, June F., and Jianxin You. "Regulation of Polyomavirus Transcription by Viral and Cellular Factors." Viruses 12, no. 10 (2020): 1072. http://dx.doi.org/10.3390/v12101072.

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Polyomavirus infection is widespread in the human population. This family of viruses normally maintains latent infection within the host cell but can cause a range of human pathologies, especially in immunocompromised individuals. Among several known pathogenic human polyomaviruses, JC polyomavirus (JCPyV) has the potential to cause the demyelinating disease progressive multifocal leukoencephalopathy (PML); BK polyomavirus (BKPyV) can cause nephropathy in kidney transplant recipients, and Merkel cell polyomavirus (MCPyV) is associated with a highly aggressive form of skin cancer, Merkel cell c
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5

Mayberry, Colleen L., and Melissa S. Maginnis. "Taking the Scenic Route: Polyomaviruses Utilize Multiple Pathways to Reach the Same Destination." Viruses 12, no. 10 (2020): 1168. http://dx.doi.org/10.3390/v12101168.

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Members of the Polyomaviridae family differ in their host range, pathogenesis, and disease severity. To date, some of the most studied polyomaviruses include human JC, BK, and Merkel cell polyomavirus and non-human subspecies murine and simian virus 40 (SV40) polyomavirus. Although dichotomies in host range and pathogenesis exist, overlapping features of the infectious cycle illuminate the similarities within this virus family. Of particular interest to human health, JC, BK, and Merkel cell polyomavirus have all been linked to critical, often fatal, illnesses, emphasizing the importance of und
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6

Duncavage, Eric J., Binh-Minh Le, David Wang, and John D. Pfeifer. "Merkel Cell Polyomavirus." American Journal of Surgical Pathology 33, no. 12 (2009): 1771–77. http://dx.doi.org/10.1097/pas.0b013e3181ba7b73.

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7

Horváth, Katalin Barbara, Péter Pankovics, Zita Battyáni, Endre Kálmán, and Gábor Reuter. "A probable etiological role for Merkel cell polyomavirus in the development of Merkel cell carcinoma." Orvosi Hetilap 154, no. 3 (2013): 102–12. http://dx.doi.org/10.1556/oh.2013.29525.

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Approximately 20% of the tumours in humans are associated with contagious viral agents. Merkel cell carcinoma is a rare and highly aggressive tumour which may originate from the epidermal stratum basale, although the origin is still controversial. This tumour is most commonly found in elderly and immunocompromised patients in sun exposed areas, especially in the head and neck regions. Merkel cell carcinoma often causes a diagnostic challenge with a dramatically increasing incidence. In 2008, a DNA tumour virus, a polyomavirus (Merkel cell polyomavirus) was detected in Merkel cell carcinomas, a
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8

Nicol, Jérôme T. J., Rémy Robinot, Audrey Carpentier, et al. "Age-Specific Seroprevalences of Merkel Cell Polyomavirus, Human Polyomaviruses 6, 7, and 9, and Trichodysplasia Spinulosa-Associated Polyomavirus." Clinical and Vaccine Immunology 20, no. 3 (2013): 363–68. http://dx.doi.org/10.1128/cvi.00438-12.

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ABSTRACTSix new human polyomaviruses have been identified since 2008 (Merkel cell polyomavirus [MCPyV], human polyomavirus 6 [HPyV6], HPyV7, HPyV9, trichodysplasia spinulosa polyomavirus [TSPyV], and Malawi polyomavirus [MWPyV]). The presence of specific antibodies against MCPyV, HPyV6, HPyV7, HPyV9, and TSPyV in 828 Italian subjects aged 1 to 100 years was investigated by virus-like particle-based enzyme-linked immunosorbent assays (ELISAs). The findings indicate that all of these new polyomaviruses circulate widely in humans, with seroprevalences in adulthood ranging from 39.4% for HPyV9 to
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9

Moens, Ugo, Maria Ludvigsen, and Marijke Van Ghelue. "Human Polyomaviruses in Skin Diseases." Pathology Research International 2011 (September 12, 2011): 1–12. http://dx.doi.org/10.4061/2011/123491.

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Polyomaviruses are a family of small, nonenveloped viruses with a circular double-stranded DNA genome of ∼5,000 base pairs protected by an icosahedral protein structure. So far, members of this family have been identified in birds and mammals. Until 2006, BK virus (BKV), JC virus (JCV), and simian virus 40 (SV40) were the only polyomaviruses known to circulate in the human population. Their occurrence in individuals was mainly confirmed by PCR and the presence of virus-specific antibodies. Using the same methods, lymphotropic polyomavirus, originally isolated in monkeys, was recently shown to
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10

Clark, Paul, Gretchen V. Gee, Brandon S. Albright та ін. "Phosphoinositide 3′-Kinase γ Facilitates Polyomavirus Infection". Viruses 12, № 10 (2020): 1190. http://dx.doi.org/10.3390/v12101190.

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Polyomaviruses are small, non-enveloped DNA tumor viruses that cause serious disease in immunosuppressed people, including progressive multifocal leukoencephalopathy (PML) in patients infected with JC polyomavirus, but the molecular events mediating polyomavirus entry are poorly understood. Through genetic knockdown approaches, we identified phosphoinositide 3′-kinase γ (PI3Kγ) and its regulatory subunit PIK3R5 as cellular proteins that facilitate infection of human SVG-A glial cells by JCPyV. PI3Kα appears less important for polyomavirus infection than PI3Kγ. CRISPR/Cas9-mediated knockout of
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11

Pietropaolo, Valeria, Carla Prezioso, and Ugo Moens. "Merkel Cell Polyomavirus and Merkel Cell Carcinoma." Cancers 12, no. 7 (2020): 1774. http://dx.doi.org/10.3390/cancers12071774.

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Viruses are the cause of approximately 15% of all human cancers. Both RNA and DNA human tumor viruses have been identified, with Merkel cell polyomavirus being the most recent one to be linked to cancer. This virus is associated with about 80% of Merkel cell carcinomas, a rare, but aggressive cutaneous malignancy. Despite its name, the cells of origin of this tumor may not be Merkel cells. This review provides an update on the structure and life cycle, cell tropism and epidemiology of the virus and its oncogenic properties. Putative strategies to prevent viral infection or treat virus-positive
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12

NAKAMURA, Tomoyuki, and Harutaka KATANO. "Merkel cell polyomavirus and Merkel cell carcinoma." Uirusu 59, no. 1 (2009): 37–42. http://dx.doi.org/10.2222/jsv.59.37.

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13

Minutilli, Ettore, and Antonino Mulè. "Merkel cell polyomavirus and cutaneous Merkel cell carcinoma." Future Science OA 2, no. 4 (2016): FSO155. http://dx.doi.org/10.4155/fsoa-2016-0064.

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14

Yang, June F., and Jianxin You. "Merkel cell polyomavirus and associated Merkel cell carcinoma." Tumour Virus Research 13 (June 2022): 200232. http://dx.doi.org/10.1016/j.tvr.2021.200232.

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15

Foulongne, Vincent, Nicolas Kluger, Olivier Dereure, Natalie Brieu, Bernard Guillot, and Michel Segondy. "Merkel Cell Polyomavirus and Merkel Cell Carcinoma, France." Emerging Infectious Diseases 14, no. 9 (2008): 1491–93. http://dx.doi.org/10.3201/eid1409.080651.

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16

Saggini, Andrea, Viviana Lora, Roberto Baldelli, Augusto Orlandi, and Carlo Cota. "Merkel Cell Polyomavirus–Positive Panniculitic Merkel Cell Carcinoma." American Journal of Dermatopathology 42, no. 2 (2020): 151–53. http://dx.doi.org/10.1097/dad.0000000000001404.

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17

Liu, Wei, Margo MacDonald, and Jianxin You. "Merkel cell polyomavirus infection and Merkel cell carcinoma." Current Opinion in Virology 20 (October 2016): 20–27. http://dx.doi.org/10.1016/j.coviro.2016.07.011.

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18

Gandhi, Rishi K., Arlene S. Rosenberg, and Stephen C. Somach. "Merkel cell polyomavirus: an update." Journal of Cutaneous Pathology 36, no. 12 (2009): 1327–29. http://dx.doi.org/10.1111/j.1600-0560.2009.01464.x.

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19

Du-Thanh, A., and B. Guillot. "Polyomavirus et tumeur de Merkel." Oncologie 15, no. 2 (2013): 91–96. http://dx.doi.org/10.1007/s10269-013-2257-y.

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20

Wong, Hannah H., and Jun Wang. "Merkel Cell Carcinoma." Archives of Pathology & Laboratory Medicine 134, no. 11 (2010): 1711–16. http://dx.doi.org/10.5858/2009-0165-rsr2.1.

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Abstract Merkel cell carcinoma is a rare, highly aggressive neuroendocrine cutaneous neoplasm with a variable clinical presentation. Histologically, it is a predominantly dermal-based lesion composed of monotonous small round cells with scanty cytoplasm, often difficult to differentiate from small round cell tumors, metastatic small cell carcinoma, blastic hematologic malignancies, and melanoma. The malignant cells express both epithelial and neuroendocrine immunohistochemical markers, a unique feature that helps differentiate this neoplasm from other entities. The pathogenesis of Merkel cell
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21

Peitsch, W. K. "Associations between Merkel cell carcinoma and Merkel cell polyomavirus." British Journal of Dermatology 173, no. 1 (2015): 7–8. http://dx.doi.org/10.1111/bjd.13925.

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22

Duncavage, Eric J., Barbara A. Zehnbauer, and John D. Pfeifer. "Prevalence of Merkel cell polyomavirus in Merkel cell carcinoma." Modern Pathology 22, no. 4 (2009): 516–21. http://dx.doi.org/10.1038/modpathol.2009.3.

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23

Levin, J., and J. Q. Del Rosso. "Prevalence of Merkel cell polyomavirus in Merkel cell carcinoma." Yearbook of Dermatology and Dermatologic Surgery 2010 (January 2010): 346–47. http://dx.doi.org/10.1016/s0093-3619(09)79385-2.

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24

XIA, YUN. "Abstract B046: Exploring the potential of commensal polyomavirus as a promising approach for breast cancer treatment." Cancer Research 84, no. 3_Supplement_1 (2024): B046. http://dx.doi.org/10.1158/1538-7445.advbc23-b046.

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Abstract Cancer is a leading cause of mortality worldwide, accounting for nearly one in six deaths. Our understanding of the immune system role in cancer control has increased dramatically in recent decades, facilitating the recent development of effective cancer immunotherapies, which have significantly improved the survival of cancer patients. However, current immunotherapies can induce overactive immune responses with severe even fatal outcomes. Moreover, immunotherapy may not work in every scenario, especially in ‘cold’ tumors that lack immune cell infiltration. Thus, more effective immuno
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25

Houben, Roland, Büke Celikdemir, Thibault Kervarrec, and David Schrama. "Merkel Cell Polyomavirus: Infection, Genome, Transcripts and Its Role in Development of Merkel Cell Carcinoma." Cancers 15, no. 2 (2023): 444. http://dx.doi.org/10.3390/cancers15020444.

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The best characterized polyomavirus family member, i.e., simian virus 40 (SV40), can cause different tumors in hamsters and can transform murine and human cells in vitro. Hence, the SV40 contamination of millions of polio vaccine doses administered from 1955–1963 raised fears that this may cause increased tumor incidence in the vaccinated population. This is, however, not the case. Indeed, up to now, the only polyomavirus family member known to be the most important cause of a specific human tumor entity is Merkel cell polyomavirus (MCPyV) in Merkel cell carcinoma (MCC). MCC is a highly deadly
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26

Iwasaki, Takeshi, Michiko Matsushita, Satoshi Kuwamoto, et al. "Usefulness of significant morphologic characteristics in distinguishing between Merkel cell polyomavirus–positive and Merkel cell polyomavirus–negative Merkel cell carcinomas." Human Pathology 44, no. 9 (2013): 1912–17. http://dx.doi.org/10.1016/j.humpath.2013.01.026.

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27

Imajoh, Masayuki, Yumiko Hashida, Ayuko Taniguchi, Mikio Kamioka, and Masanori Daibata. "Novel human polyomaviruses, Merkel cell polyomavirus and human polyomavirus 9, in Japanese chronic lymphocytic leukemia cases." Journal of Hematology & Oncology 5, no. 1 (2012): 25. http://dx.doi.org/10.1186/1756-8722-5-25.

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28

Foulongne, Vincent, Nicolas Kluger, Olivier Dereure, et al. "Merkel Cell Polyomavirus in Cutaneous Swabs." Emerging Infectious Diseases 16, no. 4 (2010): 685–87. http://dx.doi.org/10.3201/eid1604.091278.

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29

Jaeger, T., J. Ring, and C. Andres. "Histological, Immunohistological, and Clinical Features of Merkel Cell Carcinoma in Correlation to Merkel Cell Polyomavirus Status." Journal of Skin Cancer 2012 (2012): 1–5. http://dx.doi.org/10.1155/2012/983421.

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Merkel cell carcinoma is a rare, but highly malignant tumor of the skin with high rates of metastasis and poor survival. Its incidence rate rises and is currently about 0.6/100000/year. Clinical differential diagnoses include basal cell carcinoma, cyst, amelanotic melanoma, lymphoma and atypical fibroxanthoma. In this review article clinical, histopathological and immunhistochemical features of Merkel cell carcinoma are reported. In addition, the role of Merkel cell polyomavirus is discussed.
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30

Rotondo, John Charles, Chiara Mazziotta, Carmen Lanzillotti, Mauro Tognon, and Fernanda Martini. "Epigenetic Dysregulations in Merkel Cell Polyomavirus-Driven Merkel Cell Carcinoma." International Journal of Molecular Sciences 22, no. 21 (2021): 11464. http://dx.doi.org/10.3390/ijms222111464.

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Merkel cell polyomavirus (MCPyV) is a small DNA virus with oncogenic potential. MCPyV is the causative agent of Merkel Cell Carcinoma (MCC), a rare but aggressive tumor of the skin. The role of epigenetic mechanisms, such as histone posttranslational modifications (HPTMs), DNA methylation, and microRNA (miRNA) regulation on MCPyV-driven MCC has recently been highlighted. In this review, we aim to describe and discuss the latest insights into HPTMs, DNA methylation, and miRNA regulation, as well as their regulative factors in the context of MCPyV-driven MCC, to provide an overview of current fi
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31

Touzé, Antoine, Julien Gaitan, Annabel Maruani, et al. "Merkel Cell Polyomavirus Strains in Patients with Merkel Cell Carcinoma." Emerging Infectious Diseases 15, no. 6 (2009): 960–62. http://dx.doi.org/10.3201/eid1506.081463.

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32

Wieland, Ulrike, Cornelia Mauch, Alexander Kreuter, Thomas Krieg, and Herbert Pfister. "Merkel Cell Polyomavirus DNA in Persons without Merkel Cell Carcinoma." Emerging Infectious Diseases 15, no. 9 (2009): 1496–98. http://dx.doi.org/10.3201/eid1509.081575.

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33

Lauttia, S., H. Sihto, H. Kavola, V. Koljonen, T. Böhling, and H. Joensuu. "Prokineticins and Merkel cell polyomavirus infection in Merkel cell carcinoma." British Journal of Cancer 110, no. 6 (2014): 1446–55. http://dx.doi.org/10.1038/bjc.2014.20.

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34

Rodig, Scott J., Jingwei Cheng, Jacek Wardzala, et al. "Improved detection suggests all Merkel cell carcinomas harbor Merkel polyomavirus." Journal of Clinical Investigation 122, no. 12 (2012): 4645–53. http://dx.doi.org/10.1172/jci64116.

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35

Varga, E., M. Kiss, K. Szabó, and L. Kemény. "Detection of Merkel cell polyomavirus DNA in Merkel cell carcinomas." British Journal of Dermatology 161, no. 4 (2009): 930–32. http://dx.doi.org/10.1111/j.1365-2133.2009.09221.x.

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36

Paolini, Francesca, Pietro Donati, Ada Amantea, Stefania Bucher, Emilia Migliano, and Aldo Venuti. "Merkel cell polyomavirus in Merkel cell carcinoma of Italian patients." Virology Journal 8, no. 1 (2011): 103. http://dx.doi.org/10.1186/1743-422x-8-103.

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37

Álvarez-Argüelles, Marta E., Susana Rojo Alba, Blanca Vivanco Allende, and Santiago Melón García. "Merkel cell carcinoma caused by Merkel cell polyomavirus following trauma." Enfermedades Infecciosas y Microbiología Clínica 38, no. 3 (2020): 138–39. http://dx.doi.org/10.1016/j.eimc.2019.05.001.

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38

Álvarez-Argüelles, Marta E., Susana Rojo Alba, Blanca Vivanco Allende, and Santiago Melón García. "Merkel cell carcinoma caused by Merkel cell polyomavirus following trauma." Enfermedades infecciosas y microbiologia clinica (English ed.) 38, no. 3 (2020): 138–39. http://dx.doi.org/10.1016/j.eimce.2019.05.010.

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39

High, W. A. "Detection of Merkel cell polyomavirus DNA in Merkel cell carcinomas." Yearbook of Dermatology and Dermatologic Surgery 2011 (January 2011): 329–30. http://dx.doi.org/10.1016/j.yder.2011.01.001.

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40

Silling, Steffi, Alexander Kreuter, Thilo Gambichler, Thomas Meyer, Eggert Stockfleth, and Ulrike Wieland. "Epidemiology of Merkel Cell Polyomavirus Infection and Merkel Cell Carcinoma." Cancers 14, no. 24 (2022): 6176. http://dx.doi.org/10.3390/cancers14246176.

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Merkel cell polyomavirus (MCPyV) is a ubiquitous virus replicating in human dermal fibroblasts. MCPyV DNA can be detected on healthy skin in 67–90% of various body sites, and intact virions are regularly shed from the skin. Infection occurs early in life, and seropositivity increases from 37 to 42% in 1- to 6-year-olds to 92% in adults. Merkel cell carcinoma (MCC) is a rare but very aggressive neuroendocrine tumor of the skin. It develops mainly on sun-exposed areas as a fast-growing, reddish nodule. Two MCC entities exist: about 80% of MCC are MCPyV-associated. Tumorigenesis is driven by vira
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41

Zampetti, Anna, Claudio Feliciani, Guido Massi, and Antonello Tulli. "Updated Review of the Pathogenesis and Management of Merkel Cell Carcinoma." Journal of Cutaneous Medicine and Surgery 14, no. 2 (2010): 51–61. http://dx.doi.org/10.2310/7750.2010.09017.

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Background:Merkel cell carcinoma is a rare, aggressive, malignant cutaneous tumor of the elderly or immunosuppressed individuals that usually appears on sun-exposed areas of the body. Its pathogenesis is still debated, and, currently, no standardized treatment exists.Objective:To provide a current updated review of the most relevant data concerning the pathogenesis and management of Merkel cell carcinoma.Methods:Using relevant MeSH terms, we performed a review of the literature on these subjects from 1980 to June 2009.Results and Conclusion:The current management of Merkel cell carcinoma is ba
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42

Seyffert, Jennifer, Alysa Herman, Brian Katz, and Daniel Rivlin. "Bilateral symmetric Merkel cell carcinomas of the dorsal hands: multiple primary tumors or early metastasis?" International Journal of Research in Dermatology 7, no. 5 (2021): 730. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20213352.

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<p>Merkel cell carcinoma (MCC) is an aggressive and uncommon neuroendocrine tumor which clinically presents as a rapidly growing solitary nodule or plaque located in sun exposed areas on the head, neck, and extremities. Merkel cell carcinomas can be UV-induced or result from viral infection with the Merkel cell polyomavirus (MCV). We present a unique case of bilateral symmetric Merkel cell carcinomas located on the dorsal hands and briefly discuss options for genomic investigation to delineate if the tumors are multiple primary tumors or result of metastatic disease.</p>
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43

Viscidi, Raphael P., Dana E. Rollison, Vernon K. Sondak, et al. "Age-Specific Seroprevalence of Merkel Cell Polyomavirus, BK Virus, and JC Virus." Clinical and Vaccine Immunology 18, no. 10 (2011): 1737–43. http://dx.doi.org/10.1128/cvi.05175-11.

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ABSTRACTWe produced capsids of Merkel cell polyomavirus (MCPyV) in a baculovirus expression system and developed a virus-like particle (VLP) enzyme-linked immunosorbent assay (ELISA). To determine age-specific seroprevalence, serum samples were collected from 947 individuals attending hospital outpatient clinics and ranging in age from 1 to 93 years. To evaluate the association between exposure to MCPyV and Merkel cell cancer (MCC), plasma samples were obtained from 33 MCC patients and 37 controls. MCPyV seroprevalence was 45% in children under 10 years of age, increased to 60% in the next dec
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44

Yang, Rong, Eunice E. Lee, Jiwoong Kim, et al. "Characterization of ALTO-encoding circular RNAs expressed by Merkel cell polyomavirus and trichodysplasia spinulosa polyomavirus." PLOS Pathogens 17, no. 5 (2021): e1009582. http://dx.doi.org/10.1371/journal.ppat.1009582.

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Circular RNAs (circRNAs) are a conserved class of RNAs with diverse functions, including serving as messenger RNAs that are translated into peptides. Here we describe circular RNAs generated by human polyomaviruses (HPyVs), some of which encode variants of the previously described alternative large T antigen open reading frame (ALTO) protein. Circular ALTO RNAs (circALTOs) can be detected in virus positive Merkel cell carcinoma (VP-MCC) cell lines and tumor samples. CircALTOs are stable, predominantly located in the cytoplasm, and N6-methyladenosine (m6A) modified. The translation of MCPyV cir
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45

Mangana, Joanna, Piotr Dziunycz, Katrin Kerl, Reinhard Dummer, and Antonio Cozzio. "Prevalence of Merkel Cell Polyomavirus among Swiss Merkel Cell Carcinoma Patients." Dermatology 221, no. 2 (2010): 184–88. http://dx.doi.org/10.1159/000315067.

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46

Amber, Kyle, Michael P. McLeod, and Keyvan Nouri. "The Merkel Cell Polyomavirus and Its Involvement in Merkel Cell Carcinoma." Dermatologic Surgery 39, no. 2 (2013): 232–38. http://dx.doi.org/10.1111/dsu.12079.

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47

Wu, Karen N., Peter A. McCue, Adam Berger, Joseph R. Spiegel, Zi-Xuan Wang, and Agnieszka K. Witkiewicz. "Detection of Merkel Cell Carcinoma Polyomavirus in Mucosal Merkel Cell Carcinoma." International Journal of Surgical Pathology 18, no. 5 (2010): 342–46. http://dx.doi.org/10.1177/1066896910371638.

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48

Samimi, Mahtab, Charlotte Gardair, Jérome T. J. Nicol, Francoise Arnold, Antoine Touzé, and Pierre Coursaget. "Merkel Cell Polyomavirus in Merkel Cell Carcinoma: Clinical and Therapeutic Perspectives." Seminars in Oncology 42, no. 2 (2015): 347–58. http://dx.doi.org/10.1053/j.seminoncol.2014.12.021.

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49

Woodhouse, B., T. J. Robb, J. I. Hearn, et al. "Merkel cell polyomavirus is uncommon in New Zealand Merkel cell carcinomas." British Journal of Dermatology 179, no. 5 (2018): 1197–98. http://dx.doi.org/10.1111/bjd.16903.

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50

Carter, Joseph J., Kelly G. Paulson, Greg C. Wipf, et al. "Association of Merkel Cell Polyomavirus–Specific Antibodies With Merkel Cell Carcinoma." JNCI: Journal of the National Cancer Institute 101, no. 21 (2009): 1510–22. http://dx.doi.org/10.1093/jnci/djp332.

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