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Academic literature on the topic 'Pool of Bethesda'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Pool of Bethesda"

1

Miles, M. "The Pool of Bethesda: Art in British Hospitals." Scottish Medical Journal 36, no. 2 (April 1991): 55–58. http://dx.doi.org/10.1177/003693309103600212.

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2

Connelly, Angela. "‘A pool of Bethesda’: Manchester‘s First Wesleyan Methodist Central Hall." Bulletin of the John Rylands Library 89, no. 1 (March 2012): 105–25. http://dx.doi.org/10.7227/bjrl.89.1.5.

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Methodist Central Halls were built in most British towns and cities. They were designed not to look like churches in order to appeal to the working classes. Entirely multi-functional, they provided room for concerts, plays, film shows and social work alongside ordinary worship. Some contained shops in order to pay for the future upkeep of the building. The prototype for this programme was provided in Manchester and opened on Oldham Street in 1886. This article offers a first analysis of it as a building type and looks at the wider social and cultural contribution of the building. It continues the narrative by discussing changing use and design during a twentieth century that witnessed the widespread contraction of Methodist congregations.

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3

Lee, Young Ho. "The Healing at the Pool of Bethesda (Jn 5:1-15)." Journal of Youngsan Theology 17 (December 31, 2009): 331. http://dx.doi.org/10.18804/jyt.2009.12.17.331.

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4

Verbruggen, Bert, Georges Rivard, Jerome Teitel, Irwin Walker, and Alan Giles. "A Detailed Comparison of the Performance of the Standard versus the Nijmegen Modification of the Bethesda Assay in Detecting Factor VIII:C Inhibitors in the Haemophilia A Population of Canada." Thrombosis and Haemostasis 79, no. 04 (1998): 872–75. http://dx.doi.org/10.1055/s-0037-1615080.

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SummaryThe Bethesda assay is widely used to monitor the development and progress of Factor VIII:C inhibitors. Factor VIII stability in the substrate plasma (normal pool) is compromised by pH shift and reduction in protein concentration. Preliminary study, by Verbruggen and colleagues (8), suggested a reduction in spuriously positive assay results may result from buffering the normal pool plasma substrate with imidazole to pH 7.4 and substituting Factor VIII deficient plasma for imidazole buffer in the control incubation mix. These laboratory findings have now been confirmed by the performance of both the standard and the modified Bethesda assays in parallel on 877 patient samples screened during the Factor VIII:C Inhibitor Surveillance Program instituted following the conversion of all Canadian haemophilia A patients to recombinant Factor VIII. Although this study does not address the question of the clinical significance of spurious positive assays, these laboratory findings do support the conclusions of Verbruggen and the modified assay has recently been endorsed by the Factor VIII/IX Subcommittee of the SSC.

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5

Baudino, Isabelle. "William Hogarth et la peinture baroque continentale : emprunts, satire et création dans The Pool of Bethesda." XVII-XVIII. Revue de la société d'études anglo-américaines des XVIIe et XVIIIe siècles 54, no. 1 (2002): 81–96. http://dx.doi.org/10.3406/xvii.2002.1640.

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6

Németh, István. "Between Christian and prophane Iconography: Depictions of the Pool of Bethesda in Netherlandish Art from circa 1400 to 1700." Acta Historiae Artium 44, no. 1 (November 1, 2003): 225–35. http://dx.doi.org/10.1556/ahista.44.2003.1-4.26.

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7

Miller, Connie H., S. Jean Platt, Thomas Abshire, Doreen Brettler, Paula Brockenstedt, Jorge DiPaola, Gita Massey, et al. "Experience with a Modified Nijmegen-Bethesda Method for Measurement of Inhibitors in Hemophilia Patients: The CDC Inhibitor Surveillance Pilot Project." Blood 110, no. 11 (November 16, 2007): 1156. http://dx.doi.org/10.1182/blood.v110.11.1156.1156.

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Abstract A pilot project of a prospective surveillance system for coagulation factors VIII and IX inhibitors has been initiated at 9 U.S. Hemophilia Treatment Centers (HTCs). More than 500 patients have been enrolled. Risk factor and product exposure data are recorded at each HTC. A blood specimen is collected upon entry, annually, at product switch, or for clinical indication and tested centrally at the CDC, using a modified Nijmegen-Bethesda method. Plasma from specimens collected in 4.5mL evacuated tubes containing 3.2% sodium citrate is shipped overnight on cold packs. Specimens are heated to 56oC for 30 minutes to remove endogenous and infused FVIII and centrifuged. Specimens are initially screened for inhibitor using a single dilution of 3 parts patient plasma to 1 part normal pool plasma buffered with imidazole to pH 7.4 (BNPP). Specimens showing inhibition and those from previously positive patients are tested in multiple dilutions at 1 part patient dilution to 1 part BNPP. Dilution is in naturally FVIII-deficient plasma containing normal von Willebrand factor. After a 2-hour incubation at 37oC, FVIII remaining in the patient mixture is divided by FVIII remaining in a 1:1 mixture of BNPP and FVIII-deficient plasma and expressed as % residual activity (RA). %RA is converted to Nijmegen-Bethesda units (NBU) using a curve with one NBU equal to 50% RA. An inhibitor plasma of known titer is run with each assay as positive control. Split specimens shipped frozen and on cold packs showed a correlation of 0.998. 3:1 and 1:1 mixtures showed a correlation of 0.97. Almost 50% of the first 200 specimens received had measurable FVIII activity. The heating step was introduced to remove FVIII without damaging the antibody. 65 specimens went from >100% RA to a titer of 0–0.2 NBU after heating. Among 538 specimens tested for FVIII inhibitors, 435 (81%) were from patients with no previous history of inhibitor (shown below). 429 (98.6%) were < 0.5 NBU. The 6 specimens (1.4%) with > 0.6 NBU are under investigation as possible seroconversions. Nijmegen-Bethesda Units in Patients with No Previous Inhibitor NBU No. Pts. (%) NBU No. Pts. (%) NBU No. Pts. (%) 0 220 (50.6%) 0.4 3 (0.7%) 0.8 1 (0.2%) 0.1 127 (29.2%) 0.5 0 0.9 0 0.2 59 (13.6%) 0.6 0 1.0–4.0 2 (0.5%) 0.3 20 (4.6%) 0.7 2 (0.5%) 5.0–19.0 1 (0.2%) Among 121 specimens tested for factor IX (FIX) inhibitors, 113 were from patients with no previous history of inhibitor and all had NBU <0.2, using a comparable FIX inhibitor assay. Testing in a central laboratory by uniform methods will facilitate detection of new inhibitors and investigation of risk factors.

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8

Nutu, Ela. "Angels in America and Semiotic Cocktails of Sex, Bible and Politics." Biblical Interpretation 14, no. 1-2 (2006): 175–86. http://dx.doi.org/10.1163/156851506776145814.

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AbstractThis paper offers a reading of Mike Nichols' television adaptation of Tony Kushner's Angels in America with reference to biblical encounters with angels, whether direct, like those of Jacob and Elijah, or indirect, like that of the sick man by the Bethesda pool (John 5). Kushner's work is complex, and it addresses issues like the human condition, homosexuality, AIDS, race, religion and politics, while emphasising elements of choice and identity. For Kushner, it seems, 'angels' signify an absence rather than a presence of the divine, puzzles rather than answers (many of which refer to sex and gender identities), and turn-of-the-millennium angst. Kushner's 'Prior' character is declared a prophet by the messenger angel while dying of AIDS. Prior's encounter harbours echoes of Elijah's own encounter with an angel of the Lord while struggling with exhaustion and an apparent desire for death (1 Kings 19:1-9). Furthermore, unwilling to accept the role of prophet, Prior wrestles with the angel, and, in a similar vein to Jacob's experience (Gen. 32:22-32), this results in a ladder leading to heaven and a blessing. This paper explores the complex world of signifiers in Angels in America, while paying particular attention to the biblical elements present in the text.

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9

Sundue, Sharon Braslaw. "Confining the Poor to Ignorance? Eighteenth-Century American Experiments with Charity Education." History of Education Quarterly 47, no. 2 (May 2007): 123–48. http://dx.doi.org/10.1111/j.1748-5959.2007.00086.x.

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In 1738, the English evangelist George Whitefield traveled to the new colony of Georgia intending to establish “a house for fatherless children.” Inspired by both August Hermann Francke, the German Pietist who had great success educating and maintaining poor orphans in Halle, and by charity schools established in Great Britain, Whitefield's orphan house and charity school, named Bethesda, opened its doors early in 1740. For years, Whitefield devoted himself tirelessly to ensuring the success of the Bethesda school, preaching throughout Britain and North America on its behalf. Whitefield's preaching tour on behalf of his beloved Bethesda is well known for its role in catalyzing the religious revivals known collectively as the Great Awakening. The tour also marked an important shift in the history of education in America. News of the establishment of the orphanage at Bethesda coincided with new efforts to school the poor throughout the colonies. Drawing on both the British and German models of charity schooling that were highly influential for Whitefield, eighteenth-century Americans began or increased commitments to charity schooling for poor children. But the European models were not adopted wholesale. Instead, local administrators of the schooling experiments deviated from these models in a striking way. In America, elites offered some children the opportunity for extensive charity instruction, but not necessarily children at the bottom of the social hierarchy. This article will argue that the execution of these charity schooling programs was contingent upon local social conditions, specifically what appears to have been local elites' desire to maintain a certain social order and ensure a continued supply of cheap labor.

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10

Davies, Phil. "Role of multi-layer foam dressings with Safetac in the prevention of pressure ulcers: a review of the clinical and scientific data." Journal of Wound Care 25, Sup1 (January 1, 2016): S3—S23. http://dx.doi.org/10.12968/jowc.2016.25.sup1.s1.

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Background Despite the implementation of prevention strategies, pressure ulcers (PUs) continue to be a challenging health problem for patients (and their carers), clinicians and health-care providers. One area of growing interest is the use of prophylactic dressings (which were originally designed for the treatment of PUs and other wound types) as a component of standard prevention measures. Over the past few years, a large amount of scientific and clinical data relating to this subject has been published in peer-reviewed journals and presented at international meetings and conferences. A substantial proportion of these data relate to one group of dressings: multi-layer foam dressings with Safetac, which are manufactured by Mölnlycke Health Care (Gothenburg, Sweden). This evidence pool has influenced the experts involved in updating the Clinical Practice Guideline, produced by the National Pressure Ulcer Advisory Panel, European Pressure Ulcer Advisory Panel and Pan Pacific Pressure Injury Alliance, on the prevention and treatment of PUs. The updated Guideline, published in 2014, recommends that, as part of their PU prevention regimens, clinicians should consider applying prophylactic dressings to bony prominences in anatomical areas that are frequently subjected to friction and shear. Aims A literature review was undertaken to identify clinical data from the entire evidence hierarchy, as well as scientific data from laboratory studies, on the use of multi-layer foam dressings with Safetac in the prevention of pressure ulceration. Method The MEDLINE (National Library of Medicine, Bethesda, US) and EMBASE (Elsevier BV, Amsterdam, Netherlands) bibliographic databases were searched. In addition, abstract books and proceedings documents relating to national and international conferences were scanned in order to identify presentations (i.e. oral, e-posters and posters) of relevance to the review. Results Clinical and health economic experts have undertaken numerous studies, including randomised controlled trials, to assess the efficacy and cost-effectiveness of using multi-layer foam dressings with Safetac as a component of standard PU prevention strategies. The results of these studies indicate that the application of multi-layer foam dressings containing Safetac can reduce the occurrence of PUs on anatomical locations such as the sacrum and the heel, and underneath medical devices. Scientists have also developed and used laboratory methods to gain a better understanding of how prophylactic dressings work. The results of these studies indicate that the composition of foam dressings containing Safetac (i.e. their multi-layer structure) sets them apart from other dressings due to their ability to mediate the effects of physical forces (i.e. pressure, friction and shear) and control microclimate, all of which contribute to pressure ulceration. Conclusion The evidence pool clearly indicates that the prophylactic use of multi-layer foam dressings with Safetac as a component of standard prevention measures is beneficial to the clinician, the health-care provider and the patient. It should be noted that the findings outlined in this review may not be transferable to other products as their makeup and components are likely to differ significantly from those of multi-layer foam dressings with Safetac. As the importance of evidence-based practice and the need for cost-effective care continues to grow, clinicians and provider should carefully consider this point when selecting prophylactic dressings for PU prevention.

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Books on the topic "Pool of Bethesda"

1

Cubitt, Allan. The pool of Bethesda. London: Warner Chappell Plays, 1992.

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Cubitt, Allan. The pool of Bethesda. London: Warner Chappell Plays, 1992.

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3

Schlenther, Boyd Stanley. To convert the poor people in America: The Bethesda Orphanage and the thwarted zeal of the Countess of Huntingdon. [Georgia: Georgia Historical Quarterly, 1994.

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4

Cubitt, Allan. The Pool of Bethesda. Josef Weinberger Plays, 1992.

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5

Thomkins, Floyd. By the Pool of Bethesda. Genesis One Twenty-Six, 1992.

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6

Hausleben, Mr Paul John. The Pool in Bethesda: Adventures of the Quiet Stranger in the Black Hat. God Bless the Keg Publishing, 2019.

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Book chapters on the topic "Pool of Bethesda"

1

"A.C. Petty, G.L. Daniels and P. Tippett, Vox Sang, 66, 216-224 (1994). 11. K.E. Coyne, S.E. Hall, E.S. Thompson, M.A. Arce, T. Kinoshita, T. Fujita, D.J. Anstee, W. Rosse and D.M. Lublin, J. Immun. 149. 2906-2913 (1992). 12. A.C. Petty, G.L. Daniels, D.J. Anstee and P. Tippett, Vox Sang., 65, 309-315 (1993). 13. M.J. Telen, N. Rao, E.S. Thompson and D.M. Lublin, (abs) Transfusion, 32, suppl 47S (1992). 14. G. Daniels, Vox Sang., 56, 205-211 (1989). 15. M.J. Telen, in Blood Groups:Ch/Rq. Kn/McC/Yk, Cromer. J.M. Moulds and B. Laird-Fryer, eds. American Association of Blood Banks, Bethesda MD, (1992) pp. 45-63. 16. D.M. Lublin, E.S. Thompson, A.M. Green, C. Levene and M.J. Telen, J. Clin. Invest., 87, 1945-1952 (1991). 17. D.M. Lublin, G. Mallinson, M.E. Reid, J. Poole, E.S. Thompson, B.R. Ferdman, M.J. Telen, D.J. Anstee and M.J.A. Tanner, (abs) Transfusion, 32, suppl 47S (1992). 18. P.D. Issitt, Transf. Med. Rev., 3, 1-12 (1989). 19. C. Lomas, W. Grassman, D. Ford, J. Watt, A. Gooch, J. Jones, M. Beolet, D. Stern, M. Wallace and P. Tippett, Transfusion in press. 20. P. Agre and J-P. Cartron, Blood, 78, 551-563 (1991). 21. J-P. Cartron and P. Agre, Seminars Haemat., 30, 193-208 (1993). 22. D.J. Anstee and M.J.A. Tanner, in Baillieres’s Clinical Haematology. M.J.A. Tanner and D.J. Anstee, eds. Bailliere Tindall, London (1993) pp. 401-422. 23. N.D. Avent, K. Ridgwell, W.J. Mawby, M.J.A. Tanner, D.J. Anstee and B. Kumpel, Biochem. J., 256 1043-1046 (1988). 24. C. Bloy, D. Blanchard, W. Dahr, K. Beyreuther, C. Salmon and J-P. Cartron, Blood, 72, 661-666 (1988). 25. A.M. Saboori, B.L. Smith and P. Agre, Proc. Natl. Acad. Sci. USA, 85, 4042-4045 (1988). 26. N.D. Avent, K. Ridgwell, M.J.A. Tanner and D.J. Anstee, Biochem. J., 271.821-825 (1990). 27. B. Cherif-Zahar, C. Bloy, C. Le Van Kim, D. Blanchard, P. Bailly, P. Hermand, C. Salmon, J-P. Cartron, Y. Colin, Proc. Natl. Acad. Sci. USA, 87, 6243-6247 (1990). 28. I. Mouro, Y. Colin, B. Cherif-Zahar, J-P. Cartron and C. Le Van Kim, Nature Genet., 5, 62-65 (1993). 29. K. Ridgwell, N.K. Spurr, B. Laguda, C. MacGeoch, N.D. Avent and M.J.A. Tanner, Biochem. J., 287, 223-228 (1992). 30. G. Mallinson, D.J. Anstee, N.D. Avent, K. Ridgwell, M.J.A. Tanner, G.L. Daniels, P. Tippett and A.E.G. von dem Borne, Transfusion, 30, 222-225 (1990)." In Transfusion Immunology and Medicine, 199. CRC Press, 1995. http://dx.doi.org/10.1201/9781482273441-17.

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