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Journal articles on the topic 'Porphyrin metabolism'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Thunell, S. "Porphyrins, porphyrin metabolism and porphyrias. I. Update." Scandinavian Journal of Clinical and Laboratory Investigation 60, no. 7 (2000): 509–40. http://dx.doi.org/10.1080/003655100448310.

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2

Straka, J. G., J. M. Rank, and J. R. Bloomer. "Porphyria and Porphyrin Metabolism." Annual Review of Medicine 41, no. 1 (1990): 457–69. http://dx.doi.org/10.1146/annurev.me.41.020190.002325.

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3

Hindmarsh, J. T. "The porphyrias: recent advances." Clinical Chemistry 32, no. 7 (1986): 1255–63. http://dx.doi.org/10.1093/clinchem/32.7.1255.

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Abstract Recent research has elucidated several of the hitherto poorly understood steps in heme synthesis. This review describes this metabolic pathway and pinpoints the enzymatic blockages in the various porphyrias. Recent advances in the understanding of the etiology of porphyria cutanea tarda are discussed, as are the abnormalities of porphyrin metabolism seen in chronic renal failure and in lead poisoning. An outline is given of the clinical and biochemical abnormalities seen in the porphyrias. Included is an algorithm to aid in the differential diagnosis of these diseases, and a brief rev

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4

Krivosheev, Alexander B., L. Ya Kupriyanova, and M. A. Kondratova. "DOUBLE PORPHYRIA: LITERATURE REVIEW AND ANALYSIS OF CLINICAL OBSERVATION." Russian Journal of Skin and Venereal Diseases 21, no. 2 (2018): 120–24. http://dx.doi.org/10.18821/1560-9588-2018-21-2-120-124.

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A brief review of the literature on the problem of double porphyria and analysis of its own observation is presented. For more than 10 years patient B was observed for more than 10 years with a verified diagnosis of acute intermittent porphyria, which manifested with acute pain abdominal syndrome, neurological disorders in the form of peripheral polyneuropathy and hemiparesis of lower extremities, and hypertension syndrome was also noted. The observed clinical symptoms corresponded to an acute porphyrin crisis in the manifestation and / or relapse of acute intermittent porphyria. The diagnosis

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5

Baytaeva, D. A., and S. S. Bessmel’tsev. "Porphyrin metabolism in secondary hepatic porphyria in patients with hereditary deficiency of glucose-6-phosphate dehydrogenase." Kazan medical journal 93, no. 3 (2012): 451–55. http://dx.doi.org/10.17816/kmj1865.

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Aim. The study the porphyrin metabolism during the development of secondary hepatic porphyria in patients with glucose-6-phosphate dehydrogenase deficiency. Methods. Examined were 148 male patients aged 5-19 years (median 12 years) with impaired activity of glucose-6-phosphate dehydrogenase in combination with β-thalassemia and without it. Qualitative and quantitative methods of examining the activity of this enzyme were used in order to verify the diagnosis. Taking into account the varying degree of glucose-6-phosphate dehydrogenase deficiency, the indices of metabolism of the enzyme and of t

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6

Schoenfeld, N., and R. Mamet. "Interference of ofloxacin with determination of urinary porphyrins." Clinical Chemistry 40, no. 3 (1994): 417–19. http://dx.doi.org/10.1093/clinchem/40.3.417.

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Abstract The second-generation quinolone ofloxacin interferes with the screening test of porphyrins. We observed a 20-fold increase in the porphyrin concentration measured in urine of an ofloxacin-treated patient, compared with drug-free normal urine. Two other fluorinated 4-quinolones tested, norfloxacin and ciprofloxacin, had a less marked effect (a twofold increase), whereas the first-generation quinolone, nalidixic acid, did not affect the measured porphyrin concentration at all. The interference is probably due to the overlap in the emission fluorescence spectra of ofloxacin and urinary p

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7

Paw, Barry H., Yvette Y. Yien, Raymond F. Robledo, et al. "Tmem14c Plays An Essential Role In Mitochondrial Heme Metabolism." Blood 122, no. 21 (2013): 427. http://dx.doi.org/10.1182/blood.v122.21.427.427.

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Abstract Red cells synthesize large amounts of heme during terminal differentiation. Central to this process is the transport and trafficking of heme synthesis intermediates within the cell. Despite the importance of transport during heme synthesis, the molecules involved in this process are largely unknown. In a screen for genes that are upregulated during erythroid terminal differentiation, we identified Tmem14c, a predicted multi-pass transmembrane protein as an essential component of the porphyrin metabolism pathway. Here, we report that Tmem14c facilitates the synthesis of mitochondrial p

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8

S. Thunell, P. Harper, A. Brock, N. "Porphyrins, porphyrin metabolism and porphyrias. II. Diagnosis and monitoring in the acute porphyrias." Scandinavian Journal of Clinical and Laboratory Investigation 60, no. 7 (2000): 541–60. http://dx.doi.org/10.1080/003655100448329.

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9

Baytaeva, D. A., та S. S. Bessmeltsev. "The metabolitic role of the iron porphyrin complex in the development of anemic syndrome in patients with minor form of β-thalassemia". Kazan medical journal 93, № 1 (2012): 7–11. http://dx.doi.org/10.17816/kmj2135.

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Aim. To determine the significance of the main indicators of metabolism of porphyrins and markers of iron metabolism in the development of anemic syndrome during β-thalassemia minor. Methods. Examined were 58 patients with β-thalassemia minor with a concomitant deficiency of the enzyme glucose-6-phosphate dehydrogenase and 150 patients β-thalassemia minor without the concomitant enzymopathy. Hemoglobin electrophoresis was used to verify the diagnosis of β-thalassemia. Evaluated were the main indicators of porphyrin metabolism in the erythrocytes and urine in comparison with the reserve pool of

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10

Harper, S. Thunell, P. "Porphyrins, porphyrin metabolism, porphyrias. III. Diagnosis, care and monitoring in porphyria cutanea tarda - suggestions for a handling programme." Scandinavian Journal of Clinical and Laboratory Investigation 60, no. 7 (2000): 561–80. http://dx.doi.org/10.1080/003655100448338.

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11

Buzzell, G. R., R. A. Hoffman, M. K. Vaughan, and R. J. Reiter. "Hypophysectomy prevents the castration-induced increase in porphyrin concentrations in the Harderian glands of the male golden hamster: a possible role for prolactin." Journal of Endocrinology 133, no. 1 (1992): 29–35. http://dx.doi.org/10.1677/joe.0.1330029.

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ABSTRACT The Harderian glands of golden hamsters contain high concentrations of porphyrin pigments, with female hamsters having considerably higher porphyrin concentrations than males. Castration of male hamsters leads to a rapid increase in porphyrin concentrations; testosterone treatment of females has the opposite effect, suggesting a central role for androgens in inhibiting the realization of high porphyrin concentrations by this organ. Previous studies in our laboratories have shown, however, that administration of a dopamine agonist to castrated hamsters prevents the normal increase in H

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12

Doss, M. O. "ALCOHOL AND PORPHYRIN METABOLISM." Alcohol and Alcoholism 35, no. 2 (2000): 109–25. http://dx.doi.org/10.1093/alcalc/35.2.109.

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13

Besur, Siddesh, Wehong Hou, Paul Schmeltzer, and Herbert Bonkovsky. "Clinically Important Features of Porphyrin and Heme Metabolism and the Porphyrias." Metabolites 4, no. 4 (2014): 977–1006. http://dx.doi.org/10.3390/metabo4040977.

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14

S. Thunell, P. Harper, A. Brun. "Porphyrins, porphyrin metabolism and porphyrias. IV. Pathophysiology of erythyropoietic protoporphyria - diagnosis, care and monitoring of the patient." Scandinavian Journal of Clinical and Laboratory Investigation 60, no. 7 (2000): 581–604. http://dx.doi.org/10.1080/003655100448347.

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15

Pavanelli, Giovana Memari, Sibele Sauzem Milano, Gabriela Sevignani, Juliana Elizabeth Jung, Vaneuza Araujo Moreira Funke, and Marcelo Mazza do Nascimento. "Furosemide-induced pseudoporphyria in a patient with chronic kidney disease: case report." Brazilian Journal of Nephrology 40, no. 3 (2018): 287–90. http://dx.doi.org/10.1590/2175-8239-jbn-2017-0029.

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ABSTRACT Introduction: Pseudoporphyria is a rare photodermatosis with characteristics similar to those of porphyria cutanea tarda, without, however, presenting abnormalities in porphyrin metabolism. Its etiology is related to chronic kidney disease, ultraviolet radiation and certain medications. The aim of the present study is to describe a case of furosemide-related pseudoporphyria in a patient with chronic kidney disease. Case description: A 76-year-old male patient with stage 4 chronic kidney disease and in continuous use of furosemide presented ulcerated lesions with peripheral erythema an

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16

Karki, Binod, Prakash Raj Pande, Arun Sharma, and Rajeeb Kumar Deo. "Acute intermittent porphyria presenting as recurrent limb weakness." Medical Journal of Shree Birendra Hospital 11, no. 2 (2013): 44–45. http://dx.doi.org/10.3126/mjsbh.v11i2.7910.

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Acute intermittent porphyria is a rare autosomal dominant condition of porphyrin metabolism resulting from the half-normal Hydroxy methyl bilane synthase activity. The disorder can present with protean manifestation ranging from acute abdomen, psychosis to gross peripheral neuropathy thus making the diagnosis challenging and misled most of the times. A case of Acute intermittent porphyria with extreme neurological involvement in form of acute onset quadriparesis is presented in this paper.Medical Journal of Shree Birendra Hospital; July-December 2012/vol.11/Issue2/44-45 DOI: http://dx.doi.org/

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17

Tenhunen, R. "Disorders of Porphyrin and Heme Metabolism." Scandinavian Journal of Clinical and Laboratory Investigation 48 (1988): 79–80. http://dx.doi.org/10.3109/00365518809168516.

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18

Jirsa, Milan, Michaela Summerova, Vratislav Nemecek, Jirina Kaslikova, Eva Krejcova, and Stefan Vitko. "Porphyrin metabolism in chronic renal failure." Journal of Hepatology 36 (April 2002): 272. http://dx.doi.org/10.1016/s0168-8278(02)80985-0.

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19

RANK, JEFFREY M., JAMES G. STRAKA, and JOSEPH R. BLOOMER. "Liver in disorders of porphyrin metabolism." Journal of Gastroenterology and Hepatology 5, no. 5 (1990): 573–85. http://dx.doi.org/10.1111/j.1440-1746.1990.tb01443.x.

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20

O'Reilly, F. M., C. Darby, J. Fogarty, et al. "Porphyrin metabolism in hepatitis C infection." Photodermatology, Photoimmunology & Photomedicine 12, no. 1 (1996): 31–33. http://dx.doi.org/10.1111/j.1600-0781.1996.tb00241.x.

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21

Zaccaro, Mar�a Cristina, Carmen Salazar, Gloria Zulpa de Caire, M�nica Storni de Cano, and Ana Mar�a Stella. "Lead toxicity in cyanobacterial porphyrin metabolism." Environmental Toxicology 16, no. 1 (2001): 61–67. http://dx.doi.org/10.1002/1522-7278(2001)16:1<61::aid-tox70>3.0.co;2-l.

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22

Tenhunen, R. "Disorders of Porphyrin and Heme Metabolism." Scandinavian Journal of Clinical and Laboratory Investigation 48, sup190 (1988): 79–80. http://dx.doi.org/10.1080/00365518809168516.

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23

el-Sharabasy, MMH, AM el-Waseef, MM Hafez, and SA Salim. "Porphyrin metabolism in some malignant diseases." British Journal of Cancer 65, no. 3 (1992): 409–12. http://dx.doi.org/10.1038/bjc.1992.83.

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24

Yasuda, Gen, Hidehisa Satta, Yumiko Ikeda, et al. "Porphyrin metabolism abnormalities and treatment in a uremic patient with porphyria cutanea tarda." Journal of Japanese Society for Dialysis Therapy 26, no. 4 (1993): 485–90. http://dx.doi.org/10.4009/jsdt1985.26.485.

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25

Amemiya, Morimasa, Eiji Kusano, Koji Ueno, Keisuke Kotoda, Masao Kondo, and Yasushi Asano. "Porphyrin metabolism in patients on maintenance hemodialysis." Nihon Toseki Igakkai Zasshi 28, no. 9 (1995): 1225–30. http://dx.doi.org/10.4009/jsdt.28.1225.

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26

Abdel-Aziz, A. F., M. M. H. El-Sharabasy, M. A. El-Far, and Wafaa M. Yousef. "Porphyrin metabolism in rats intaking chemical carcinogens." Cancer Letters 130, no. 1-2 (1998): 77–81. http://dx.doi.org/10.1016/s0304-3835(98)00117-7.

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27

Wilde, Annegret, Sandra Mikolajczyk, Ali Alawady, Heiko Lokstein, and Bernhard Grimm. "Thegun4gene is essential for cyanobacterial porphyrin metabolism." FEBS Letters 571, no. 1-3 (2004): 119–23. http://dx.doi.org/10.1016/j.febslet.2004.06.063.

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28

Goerz, G., K. Bolsen, and H. Merk. "Influence of chloroquine on the porphyrin metabolism." Archives of Dermatological Research 277, no. 2 (1985): 114–17. http://dx.doi.org/10.1007/bf00414107.

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29

Smirnov, I. V., A. A. Levina, I. V. Kuznetsov, et al. "Hemin arginate as a porphyrin metabolism corrector." Pharmaceutical Chemistry Journal 34, no. 5 (2000): 223–25. http://dx.doi.org/10.1007/bf02524623.

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30

Yasuda, Gen, Yumiko Ikeda, Hidehisa Satta, Hiroshi Shionoiri, Masao Ishii, and Yoshio Ikezawd. "Porphyrin Metabolism Abnormalities and Its Treatment in a Uremic Patient with Porphyria cutanea tarda." Nephron 63, no. 2 (1993): 235–36. http://dx.doi.org/10.1159/000187193.

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31

Burmeister, Veronika, and R. Swaminathan. "The blister of porphyria cutanea tarda: A Scanning and Transmission Electron Microscopic study." Proceedings, annual meeting, Electron Microscopy Society of America 44 (August 1986): 334–35. http://dx.doi.org/10.1017/s0424820100143298.

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Porphyria cutanea tarda (PCT) is a disorder of porphyrin metabolism which occurs most often during middle age. The disease is characterized by excessive production of uroporphyrin which causes photosensitivity and skin eruptions on hands and arms, due to minor trauma and exposure to sunlight. The pathology of the blister is well known, being subepidermal with epidermodermal separation, it is not always absolutely clear, whether the basal lamina is attached to the epidermis or the dermis. The purpose of our investigation was to study the attachment of the basement membrane in the blister by com

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32

Kondo, M. "Change of porphyrin metabolism of a arsenicpoisoned patient." SANGYO EISEIGAKU ZASSHI 40, Special (1998): 451. http://dx.doi.org/10.1539/sangyoeisei.kj00001990274.

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33

Fukushima, Masao, Takuo Fujisawa, and Toshiyuki Katagi. "Tomato Metabolism and Porphyrin-Catalyzed Oxidation of Pyriproxyfen." Journal of Agricultural and Food Chemistry 53, no. 13 (2005): 5353–58. http://dx.doi.org/10.1021/jf0503816.

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34

Hill, R. H. "Effects of polyhalogenated aromatic compounds on porphyrin metabolism." Environmental Health Perspectives 60 (May 1985): 139–43. http://dx.doi.org/10.1289/ehp.8560139.

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35

McColl, K. E., G. G. Thompson, E. el Omar, M. R. Moore, and A. Goldberg. "Porphyrin metabolism and haem biosynthesis in Gilbert's syndrome." Gut 28, no. 2 (1987): 125–30. http://dx.doi.org/10.1136/gut.28.2.125.

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36

Meserve, Lee A. "Harderian glands: Porphyrin metabolism, behavioral and endocrine effects." Trends in Endocrinology & Metabolism 4, no. 3 (1993): 111–12. http://dx.doi.org/10.1016/1043-2760(93)90089-w.

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37

Fontanellas, A., J. A. Herrero, J. I. Trobo, et al. "Abnormalities of heme biosynthesis in experimental acute renal failure." Journal of the American Society of Nephrology 7, no. 4 (1996): 628–32. http://dx.doi.org/10.1681/asn.v74628.

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Several abnormalities of porphyrin metabolism have been described in patients with end-stage renal failure. Because the heme biosynthetic pathway in acute renal failure has not been studied hitherto, an experimental model was therefore induced in 30 dogs by ligation and transection of both ureters. Forty-eight h after this procedure, anemia and uremia developed, erythrocyte aminolevulinate dehydratase activity decreased, and plasma porphyrins increased in these 30 dogs, whereas seven sham-operated animals did not exhibit any alteration of these parameters. Uremic plasma showed a capacity to in

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38

Groß, U., M. Honcamp, E. Daume, M. Frank, B. Düsterberg, and M. Doss. "Hormonal Oral Contraceptives, Urinary Porphyrin Excretion and Porphyrias." Hormone and Metabolic Research 27, no. 08 (1995): 379–83. http://dx.doi.org/10.1055/s-2007-979983.

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39

Krijt, Jan, Hana Krijtová, and Jaroslav Sanitrák. "Effect of Tiagabine and Topiramate on Porphyrin Metabolism in an in vivo Model of Porphyria." Pharmacology & Toxicology 89, no. 1 (2008): 15–22. http://dx.doi.org/10.1111/j.1600-0773.2001.890103.x.

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40

Krijt, Jan, Hana Krijtova, and Jaroslav Sanitrak. "Effect of Tiagabine and Topiramate on Porphyrin Metabolism in an in vivo Model of Porphyria." Pharmacology and Toxicology 89, no. 1 (2001): 15–22. http://dx.doi.org/10.1034/j.1600-0773.2001.d01-130.x.

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41

Gomi, Hiroko. "Type of Impaired Porphyrin Metabolism Caused by Hepatitis C Virus Is Not Porphyria Cutanea Tarda but Chronic Hepatic Porphyria." Archives of Dermatology 133, no. 9 (1997): 1170. http://dx.doi.org/10.1001/archderm.1997.03890450122020.

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42

Gomi, H. "Type of impaired porphyrin metabolism caused by hepatitis C virus is not porphyria cutanea tarda but chronic hepatic porphyria." Archives of Dermatology 133, no. 9 (1997): 1170–71. http://dx.doi.org/10.1001/archderm.133.9.1170.

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43

Hodgins, R., and R. B. Van Huystee. "Porphyrin Metabolism in Chill Stressed Maize (Zea mays L.)." Journal of Plant Physiology 125, no. 3-4 (1986): 325–36. http://dx.doi.org/10.1016/s0176-1617(86)80154-7.

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44

Buchet, J. P., R. Lauwerys, A. Hassoun, et al. "Effect of Aluminum on Porphyrin Metabolism in Hemodialyzed Patients." Nephron 46, no. 4 (1987): 360–63. http://dx.doi.org/10.1159/000184390.

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45

Mikulecky, M. "Porphyrin metabolism in Gilbert's, Dubin-Johnson, and Rotor's syndromes." Gut 28, no. 11 (1987): 1548–49. http://dx.doi.org/10.1136/gut.28.11.1548.

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46

Tseilikman, V. E., V. E. Ryabinin, A. S. Popova, L. I. Krupitskaya, and A. I. Sinitsky. "Variability of Microsomal Oxidation and Porphyrin Metabolism in Rats." Bulletin of Experimental Biology and Medicine 150, no. 2 (2010): 178–79. http://dx.doi.org/10.1007/s10517-010-1098-6.

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47

Smirnov, I. V., L. T. Minina, I. V. Kuznetsov, et al. "Experimental safety evaluation of the porphyrin metabolism corrector arhem." Pharmaceutical Chemistry Journal 45, no. 6 (2011): 341–43. http://dx.doi.org/10.1007/s11094-011-0629-4.

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48

Martinez, María del Carmen, Silvina Fernanda Ruspini, Susana Graciela Afonso, Roberto Meiss, Ana Maria Buzaleh, and Alcira Batlle. "Experimental Protoporphyria: Effect of Bile Acids on Liver Damage Induced by Griseofulvin." BioMed Research International 2015 (2015): 1–10. http://dx.doi.org/10.1155/2015/436319.

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The effect of bile acids administration to an experimental mice model of Protoporphyria produced by griseofulvin (Gris) was investigated. The aim was to assess whether porphyrin excretion could be accelerated by bile acids treatment in an attempt to diminish liver damage induced by Gris. Liver damage markers, heme metabolism, and oxidative stress parameters were analyzed in mice treated with Gris and deoxycholic (DXA), dehydrocholic (DHA), chenodeoxycholic, or ursodeoxycholic (URSO). The administration of Gris alone increased the activities of glutathione reductase (GRed), superoxide dismutase

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49

Murphy, Gillian. "Type of Impaired Porphyrin Metabolism Caused by Hepatitis C Virus Is Not Porphyria Cutanea Tarda but Chronic Hepatic Porphyria-Reply." Archives of Dermatology 133, no. 9 (1997): 1171. http://dx.doi.org/10.1001/archderm.1997.03890450122021.

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50

Herrick, A., A. Mclellan, M. J. Brodie, K. E. L. Mccoll, M. R. Moore, and A. Goldberg. "EFFECT OF HAEM ARGINATE THERAPY ON PORPHYRIN METABOLISM AND MIXED FUNCTION OXYGENASE ACTIVITY IN ACUTE HEPATIC PORPHYRIA." Lancet 330, no. 8569 (1987): 1178–79. http://dx.doi.org/10.1016/s0140-6736(87)91320-1.

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