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Books on the topic 'Porphyrio'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 April 2022

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1

Der Horazkommentar des Pomponius Porphyrio: Untersuchungen zu seiner Terminologie und Textgeschichte. Stuttgart: Steiner, 2007.

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2

Diederich, Silke. Der Horazkommentar des Porphyrio im Rahmen der kaiserzeitlichen Schul- und Bildungstradition. Berlin: W. de Gruyter, 1999.

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3

Dobhal, M. P. Porphyrins - Spectral Data of Porphyrin Isomers and Expanded Porphyrins. Edited by V. Gupta, M. D. Lechner, and R. Gupta. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-47224-8.

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4

International Congress on Porphyrins and Porphyrias (2nd 1985 Paris, France). Porphyrins and porphyrias =: Porphyrines et porphyries : proceedings of the Second International Congress on Porphyrins and Porphyrias held in Paris (France), 19-22 June, 1985. London: J. Libbey, 1986.

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5

International, Congress on Porphyrins and Porphyrias (2nd 1985 Paris France). Porphyrins and porphyrias: Proceedings of the Second International Congress on Porphyrins and Porphyrias held in Paris (France) 19-22 June, 1985 = Porphyrines et porphyries. Paris, France: Editions INSERM, 1986.

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6

Duke, Stephen O., and Constantin A. Rebeiz, eds. Porphyric Pesticides. Washington, DC: American Chemical Society, 1994. http://dx.doi.org/10.1021/bk-1994-0559.

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7

Spourdalakēs, Chrēstos. Porphyriōn. Athēna: Ekdoseis Delphini, 1994.

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8

Gupta, R. Porphyrins. Edited by V. Gupta, M. D. Lechner, and R. Gupta. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-642-41556-2.

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9

Gupta, V., M. D. Lechner, and R. Gupta, eds. Porphyrins. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-41561-6.

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10

Dobhal, M. P. Porphyrins. Edited by V. Gupta, M. D. Lechner, and R. Gupta. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-41605-7.

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11

Gupta, R. Porphyrins. Edited by V. Gupta, M. D. Lechner, and R. Gupta. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-49340-3.

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12

Gouterman, Martin, Peter M. Rentzepis, and Karl D. Straub, eds. Porphyrins. Washington, DC: American Chemical Society, 1986. http://dx.doi.org/10.1021/bk-1986-0321.

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13

Porphyry. Porphyrii philosophi fragmenta. Stutgardiae: Teubner, 1993.

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14

David, Kessel, ed. Methods in porphyrin photosensitization. New York: Plenum Press, 1985.

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15

Porphyry. Die Sentenzen des Porphyrios. Frankfurt am Main: P. Lang, 1987.

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16

Moore, Michael R., Kenneth E. L. McColl, Claude Rimington, and Abraham Goldberg. Disorders of Porphyrin Metabolism. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-1277-2.

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17

Kessel, David, ed. Methods in Porphyrin Photosensitization. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2165-1.

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18

Ma, Shengqian, and Gaurav Verma, eds. Porphyrin-based Supramolecular Architectures. Cambridge: Royal Society of Chemistry, 2021. http://dx.doi.org/10.1039/9781839164934.

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19

Lüdtke-Buzug, Kerstin. Azosubstituierte Porphyrine. Wiesbaden: Springer Fachmedien Wiesbaden, 2017. http://dx.doi.org/10.1007/978-3-658-16313-6.

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20

Parker, Philip M., and James M. Parker. Porphyria: A bibliography and dictionary for physicians, patients, and genome researchers. Edited by Icon Group International Inc. San Diego, California: ICON Health Publications, 2007.

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21

Porphyry. Porphyry Introduction. Oxford: Oxford University Press, 2003.

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22

Porphyry: Red imperial porphyry : power and religion. Torino: U. Allemandi & C., 2012.

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23

Alessio, Enzo, ed. Non-Covalent Multi-Porphyrin Assemblies. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11677529.

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24

Chrysafis, Andreas C. Porphyra in Purple. UK: Evandia Publishing (UK)Ltd, 2008.

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25

Langendonk, Janneke G., and Timothy M. Cox. Porphyrias. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0043.

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The porphyrias are disorders caused by overproduction of metabolites involved in heme biosynthesis. The four acute porphyrias— acute intermittent porphyria (AIP), variegate porphyria (VP), hereditary coproporphyria (HCP), and Doss Porphyria—present with severe abdominal pain, often accompanied by agitation, hypertension, and tachycardia associated with neuropathy and sometimes paralysis. Painful and disabling neurovisceral attacks are due to excess production of the heme precursor ALA (delta-aminolevulinic acid).While 90% of individuals with an inherited defect in heme biosynthesis will never develop symptoms, acute attacks in those affected are provoked by drugs, fasting, and alcohol; in women of reproductive age, they usually occur in the progestagenic phase of the menstrual cycle. All other porphyrias are considered cutaneous porphyrias. They present with blisters or pain on light exposed areas, toxic porphyrins accumulate and give rise to skin symptoms. The cutaneous porphyrias (PCT, EPP, XLEPP, and HEP) do not present with acute neurovisceral attacks (e.g., abdominal pain). However, severe systemic complications can occur.
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26

Nordmann, Y. Porphyrins and Porphyria (Colloques INSERM). John Libbey & Co Ltd, 1986.

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27

Sybert, Virginia P. Metabolic Disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780195397666.003.0011.

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Porphyrias – Congenital Erythropoietic Porphyria – Erythropoietic Protoporphyria – Hereditary Coproporphyria – Porphyria Cutanea Tarda – Variegate Porphyria – Mucopolysaccharidoses – Hunter Syndrome – Other Metabolic Disorders – Acrodermatitis Enteropathica – Alkaptonuria – Biotinidase Deficiency – Familial Cutaneous Amyloidosis – Prolidase Deficiency
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28

R, Moore Michael, and Wintrobe Maxwell Myer 1901-, eds. Disorders of porphyrin metabolism. New York: Plenum Medical Book Co., 1987.

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29

Sybert, Virginia P. Metabolic Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190276478.003.0011.

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Chapter 11 covers Porphyrias (Congenital Erythropoietic Porphyria, Erythropoietic Protoporphyria, Hereditary Coproporphyria, Porphyria Cutanea Tarda, and Variegate Porphyria), Mucopolysaccharidoses (Hunter Syndrome), and Other Metabolic Disorders (Acrodermatitis Enteropathica, Alkaptonuria, Biotinidase Deficiency, Familial Cutaneous Amyloidosis, and Prolidase Deficiency). Each condition is discussed in detail, including dermatologic features, associated anomalies, histopathology, basic defect, treatment, mode of inheritance, prenatal diagnosis, and differential diagnosis.
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30

(Editor), Karl Kadish, Roger Guilard (Editor), and Kevin M. Smith (Editor), eds. The Porphyrin Handbook, Volumes 11-20, Volume 14: Medical Aspects of Porphyrins (The Porphyrin Handbook). Academic Press, 2002.

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31

(Editor), Karl Kadish, Roger Guilard (Editor), and Kevin M. Smith (Editor), eds. The Porphyrin Handbook, Volumes 11-20, Volume 14: Medical Aspects of Porphyrins (The Porphyrin Handbook). Academic Press, 2002.

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32

A, Sheldon Roger, ed. Metalloporphyrins in catalyticoxidations. New York: M. Dekker, 1994.

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33

A, Sheldon Roger, ed. Metalloporphyrins in catalytic oxidations. New York: M. Dekker, 1994.

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34

Aidalu de los Angeles Joubert-Castro. An evaluation of fast atom bombardment mass spectrometry as a technique for the analysis of geoporphyrins. 1987.

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35

Steensma, David P. Benign Hematology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0294.

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The major forms of benign hematologic conditions are anemia, neutropenia, transfusion reactions, Gaucher disease, and porphyria. Anemia is a sign of disease rather than a disease itself. Anemia results from 1 or more of 3 pathologic mechanisms: inadequate production of red blood cells (RBCs) by the bone marrow, blood loss, or premature destruction of RBCs. The major causes of neutropenia include hematologic neoplasm, metastatic neoplasm involving the marrow, irradiation, vitamin B12 deficiency and folate deficiency, drugs, infections, congenital or acquired primary disorders of hematopoiesis, autoimmune neutropenia, hypersplenism, hemodilution, and benign idiopathic neutropenia. The porphyrias are enzyme disorders that are autosomal dominant with low disease penetrance, except for congenital erythropoietic porphyria, which is autosomal recessive, and porphyria cutanea tarda, which may be acquired and is associated with hepatitis C and hemochromatosis.
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36

F, Merk Hans, ed. Porphyria. Basel: Karger, 1998.

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37

Deegan, Patrick. Porphyria. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0179.

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This chapter discusses six diseases caused by inborn errors of metabolism affecting the biosynthesis of haem. Haem is a tetracyclic metal-binding compound involved in oxygen transport (in haemoglobin and myoglobin) and redox reactions (e.g. in the cytochrome P450 system). Each of these conditions is caused by a single gene defect in one of the enzymes involved in the biosynthesis of haem. Inheritance is usually autosomal dominant with incomplete penetrance. The enzyme defect results in disease, not as a result of deficiency of the reaction product, but as a result of accumulation of precursors. Early, soluble precursors, 5-aminolaevulinic acid, and porphobilinogen (not porphyrins as such) are neurotoxic and, when present in great excess, as occurs when flux through the haem synthetic pathway is increased in response to particular medications or hormones, lead to acute neurovisceral crises. Later cyclical precursors (porphyrins) in the pathway are also water soluble and excreted in urine, but are susceptible to activation by electromagnetic radiation in the visible spectrum and are converted to free-radical metabolites that cause pain, inflammation, and tissue damage in the skin. The final haem precursors (also porphyrins) are hydrophobic and excreted in the bile and faeces and are also activated by light to toxic metabolites.
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38

Dierdorf, Stephen F. Porphyria. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0026.

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Heme, and iron containing compound that forms the nonprotein portion of hemoglobin, is essential to life. Heme synthesis requires eight enzymatic steps, and a deficiency in any one of the eight enzymes can lead to the accumulation of potentially toxic intermediates. Some forms of porphyria may be asymptomatic until the patient receives a triggering agent, and acute porphyrias can also be difficult to diagnose because of the nonspecific clinical features. The most serious of the clinical manifestations is severe neurologic dysfunction. An attack can be triggered by medications administered during the perioperative period, and failure to act promptly can result in mortality rates as high as 5%.
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39

Demetri Porphyrios. Academy Edns., 1992.

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40

Scully, Vincent, and Kenneth Powell. Porphyrios Associates. Papadakis Publisher, 2008.

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41

Kessel, David. Porphyrin Photosensitization. Springer, 2012.

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42

K, Silbergeld Ellen, Fowler Bruce A, and New York Academy of Sciences., eds. Mechanisms of chemical-induced porphyrinopathies. New York, N.Y: New York Academy of Sciences, 1987.

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43

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 2 (The Porphyrin Handbook). Academic Press, 1999.

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44

The Porphyrin Handbook Volume 8 (The Porphyrin Handbook). Academic Press, 1999.

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45

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 6 (The Porphyrin Handbook). Academic Press, 1999.

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46

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 1 (The Porphyrin Handbook). Academic Press, 1999.

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47

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 5 (The Porphyrin Handbook). Academic Press, 1999.

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48

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 7 (The Porphyrin Handbook). Academic Press, 1999.

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49

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 10 (The Porphyrin Handbook). Academic Press, 1999.

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50

(Editor), Karl Kadish, Kevin M. Smith (Editor), and Roger Guilard (Editor), eds. The Porphyrin Handbook Volume 10 (The Porphyrin Handbook). Academic Press, 1999.

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