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Journal articles on the topic 'Portosystemic'

Author: Grafiati

Published: 19 February 2023

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1

Conn, Harold O., Martin Rössle, Lewis Levy, and Franz X. Glocker. "Portosystemic myelopathy: Spastic paraparesis after portosystemic shunting." Scandinavian Journal of Gastroenterology 41, no. 5 (January 2006): 619–25. http://dx.doi.org/10.1080/00365520500318932.

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2

Uflacker, R., A. de O. Silva, L. A. d'Albuquerque, R. L. Piske, and G. S. Mourão. "Chronic portosystemic encephalopathy: embolization of portosystemic shunts." Radiology 165, no. 3 (December 1987): 721–25. http://dx.doi.org/10.1148/radiology.165.3.3685350.

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3

Leite, Andréa Farias de Melo, Américo Mota Jr., Francisco Abaeté Chagas-Neto, Sara Reis Teixeira, Jorge Elias Junior, and Valdair Francisco Muglia. "Acquired portosystemic collaterals: anatomy and imaging." Radiologia Brasileira 49, no. 4 (August 2016): 251–56. http://dx.doi.org/10.1590/0100-3984.2015.0026.

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Abstract Portosystemic shunts are enlarged vessels that form collateral pathological pathways between the splanchnic circulation and the systemic circulation. Although their causes are multifactorial, portosystemic shunts all have one mechanism in common-increased portal venous pressure, which diverts the blood flow from the gastrointestinal tract to the systemic circulation. Congenital and acquired collateral pathways have both been described in the literature. The aim of this pictorial essay was to discuss the distinct anatomic and imaging features of portosystemic shunts, as well as to provide a robust method of differentiating between acquired portosystemic shunts and similar pathologies, through the use of illustrations and schematic drawings. Imaging of portosystemic shunts provides subclinical markers of increased portal venous pressure. Therefore, radiologists play a crucial role in the identification of portosystemic shunts. Early detection of portosystemic shunts can allow ample time to perform endovascular shunt operations, which can relieve portal hypertension and prevent acute or chronic complications in at-risk patient populations.

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4

Mahros, AyaM, ReemM Mohammed, YoussefM Sewifee, NahedA Makhlof, HamdyM Ibrahim, and Ahmed Helmy. "Portosystemic collaterals." Journal of Current Medical Research and Practice 6, no. 2 (2021): 109. http://dx.doi.org/10.4103/jcmrp.jcmrp_90_18.

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5

Vulgamott, Jim C. "Portosystemic Shunts." Veterinary Clinics of North America: Small Animal Practice 15, no. 1 (January 1985): 229–42. http://dx.doi.org/10.1016/s0195-5616(85)50013-3.

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6

Rao, Prasanna K. S., Keyur A. Sheth, Raghunandan Nadig, Mallikarjun Patil, and Adarsh K. Channagiri. "Portosystemic Myelopathy: A Rare Neurological Presentation of Portosystemic Shunts." Journal of Clinical and Experimental Hepatology 2, no. 4 (December 2012): 393–95. http://dx.doi.org/10.1016/j.jceh.2012.10.005.

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7

Maddison, J. E., D. Yau, P. Stewart, and G. C. Farrell. "Cerebrospinal fluid γ-aminobutyric acid levels in dogs with chronic portosystemic encephalopathy." Clinical Science 71, no. 6 (December 1, 1986): 749–53. http://dx.doi.org/10.1042/cs0710749.

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1. Cerebrospinal fluid (CSF) γ-aminobutyric acid (GABA) levels were measured in a dog model of spontaneous chronic portosystemic encephalopathy. 2. Dogs with congenital portacaval shunts (intra- or extra-hepatic) develop neurological features of abnormal psychomotor behaviour and depressed consciousness that are consistent with the symptoms of chronic portosystemic encephalopathy in humans. In the five dogs studied, plasma ammonia was elevated, as was CSF tryptophan, both usual biochemical abnormalities in portosystemic encephalopathy. 3. CSF levels of GABA in five dogs with portosystemic encephalopathy (100 ± 13 pmol/ml) were not significantly different from those in five control dogs (96 ± 14 pmol/ml). CSF levels of GABA were not altered after ammonia infusion. 4. If enhanced GABA-ergic neurotransmission, due to influx of gut-derived GABA into the brain, is responsible for the pathophysiology of chronic portosystemic encephalopathy in this model, it is not reflected by increased levels of GABA in CSF.

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8

Izzo, Francesca, Edoardo Poggi, Francisco J. Pérez Duarte, and Filippo Cinti. "Laparoscopic portosystemic shunt attenuation in two cats." Journal of Feline Medicine and Surgery Open Reports 8, no. 1 (January 2022): 205511692210814. http://dx.doi.org/10.1177/20551169221081416.

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Case series summary Gradual attenuation of an extrahepatic portosystemic shunt using cellophane banding was achieved with a laparoscopic technique in two cats. The portosystemic shunts were treated via a right or left lateral laparoscopic approach. Ultrasonography or CT angiography were used to verify the results of surgery. The success of the procedure was confirmed by normalisation of serum bile acid concentrations and clinical signs at the final re-evaluation. Relevance and novel information The aim of this case series was to determine the feasibility and outcome of laparoscopy for portosystemic shunt attenuation in two cats. Laparoscopic portosystemic shunt attenuation appeared to be a feasible, safe and effective procedure in cats.

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9

Hüsing-Kabar, A., T. Meister, M. Köhler, W. Domschke, I. Kabar, C. Wilms, B. Hild, HH Schmidt, and HS Heinzow. "Is de novo hepatocellular carcinoma after transjugular intrahepatic portosystemic shunt increased?" United European Gastroenterology Journal 6, no. 3 (September 20, 2017): 413–21. http://dx.doi.org/10.1177/2050640617732886.

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Background Portal hypertension is a major complication of liver cirrhosis. Transjugular intrahepatic portosystemic shunt is effective in treatment of portal hypertension. However, decreased parenchymal portal venous flow after transjugular intrahepatic portosystemic shunt insertion favours ischaemic liver injury which has been discussed to induce hepatocarcinogenesis causing hepatocellular cancer. Aim This study aimed to explore the association between transjugular intrahepatic portosystemic shunt placement and the development of hepatocellular cancer. Methods A total of 1338 consecutive liver cirrhosis patients were included in this retrospective study between January 2004–December 2015. Data were analysed with regard to development of hepatocellular cancer during follow-up. Binary logistic regression and Kaplan-Meier analyses were conducted for the assessment of risk factors for hepatocellular cancer development. In a second step, to rule out confounders of group heterogeneity, case-control matching was performed based on gender, age, model of end-stage liver disease score and underlying cause of cirrhosis (non-alcoholic steatohepatitis, alcoholic liver disease and viral hepatitis). Results Besides established risk factors such as older age, male gender and underlying viral hepatitis, statistical analysis revealed the absence of transjugular intrahepatic portosystemic shunt insertion as a risk factor for hepatocellular cancer development. Furthermore, matched-pair analysis of 432 patients showed a significant difference ( p = 0.003) in the emergence of hepatocellular cancer regarding transjugular intrahepatic portosystemic shunt placement versus the non-transjugular intrahepatic portosystemic shunt cohort. Conclusion In patients with end-stage liver disease, transjugular intrahepatic portosystemic shunt insertion is significantly associated with reduced rates of hepatocellular cancer development.

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10

Durleshter, V. M., S. A. Gabriel’, N. V. Korochanskaya, A. Yu Buhtoyarov, P. V. Markov, D. S. Murashko, O. A. Oganesyan, L. G. Izmailova, M. A. Basenko, and Yu V. Horon'ko. "Transjugular intrahepatic portosystemic stent-shunt as minimally invasive method of portal hypertension correction in multi-disciplinary clinic." Annaly khirurgicheskoy gepatologii = Annals of HPB Surgery 25, no. 4 (December 17, 2020): 95–106. http://dx.doi.org/10.16931/1995-5464.2020495-106.

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Aim. Optimization of the tactics of management of patients with liver cirrhosis who underwent Transjugular Intrahepatic Portosystemic Shunt – TIPS based on own experience and literature data. Materials and methods. From 2014 to 2019 years 51 Transjugular Intrahepatic Portosystemic Shunt procedures were performed. Results. The indications for Transjugular Intrahepatic Portosystemic Shunt procedure were detailed. The tactics of treatment in post-operative period was assessed. Shot-term and long-term results of the treatment were discussed. Especial attention was put to persons who included in patient list of liver transplantation. Conclusion. Transjugular Intrahepatic Portosystemic Shunt allows obtaining stable decompression in portal system that reduces frequency of bleeding relapse from gastric and esophageal veins. The mortality was decreased, and patients can wait till liver transplantation.

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11

Ibukuro, Kenji, Toshitaka Tsukiyama, Koichi Mori, and Yoshihiro Inoue. "Transhepatic Portosystemic Shunts." American Journal of Roentgenology 175, no. 1 (July 2000): 153–57. http://dx.doi.org/10.2214/ajr.175.1.1750153.

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12

Han, Byoung Hee, Sung Bin Park, Mi Jin Song, Kyung Sang Lee, Young-Ho Lee, Sun Young Ko, and Yeon Kyung Lee. "Congenital Portosystemic Shunts." Journal of Ultrasound in Medicine 32, no. 1 (January 2013): 45–52. http://dx.doi.org/10.7863/jum.2013.32.1.45.

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13

Gaudiano, Frances. "Congenital portosystemic shunts." Veterinary Nursing Journal 22, no. 1 (January 2007): 18–21. http://dx.doi.org/10.1080/17415349.2007.11013544.

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14

Sokollik, Christiane, Robert H. J. Bandsma, Juan C. Gana, Meta van den Heuvel, and Simon C. Ling. "Congenital Portosystemic Shunt." Journal of Pediatric Gastroenterology and Nutrition 56, no. 6 (June 2013): 675–81. http://dx.doi.org/10.1097/mpg.0b013e31828b3750.

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15

Hsieh, Hung-Jen, Kun-Han Lue, Chih-Hao K. Kao, Yung-Hsiang Hsu, Ming-Chien Liu, and Pan-Fu Kao. "Portosystemic Collateral Circulation." Clinical Nuclear Medicine 34, no. 12 (December 2009): 958–59. http://dx.doi.org/10.1097/rlu.0b013e3181bed007.

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16

McLin, Valérie A., Stephanie Franchi Abella, Dominique Debray, Florent Guérin, Maurice Beghetti, Laurent Savale, Barbara E. Wildhaber, and Emmanuel Gonzales. "Congenital Portosystemic Shunts." Journal of Pediatric Gastroenterology and Nutrition 68, no. 5 (May 2019): 615–22. http://dx.doi.org/10.1097/mpg.0000000000002263.

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17

Berent, Allyson C., and Karen M. Tobias. "Portosystemic Vascular Anomalies." Veterinary Clinics of North America: Small Animal Practice 39, no. 3 (May 2009): 513–41. http://dx.doi.org/10.1016/j.cvsm.2009.02.004.

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18

Guérin, Florent, Stéphanie Franchi Abella, Valérie McLin, Oanez Ackermann, Muriel Girard, Jean Paul Cervoni, Laurent Savale, et al. "Congenital portosystemic shunts." Clinics and Research in Hepatology and Gastroenterology 44, no. 4 (September 2020): 452–59. http://dx.doi.org/10.1016/j.clinre.2020.03.004.

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19

Bilbao, José Ignacio, Mercedes Arias, Jesús María Longo, Pedro Luis Alejandre, María Teresa Betés, and Arlette María Elizalde. "Embolization of nonvariceal portosystemic collaterals in transjugular intrahepatic portosystemic shunts." Cardiovascular and Interventional Radiology 20, no. 2 (March 1997): 149–53. http://dx.doi.org/10.1007/s002709900125.

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20

Umek, Nejc, Domen Plut, Martina Krofič Žel, and Aleksandra Domanjko Petrič. "Radiologic Evaluation of Portosystemic Shunts in Humans and Small Animals: Review of the Literature with Clinical Case Reports." Diagnostics 13, no. 3 (January 28, 2023): 482. http://dx.doi.org/10.3390/diagnostics13030482.

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The portal venous system is a network of vessels that carry blood from the capillary beds of the major abdominal organs to the liver. During embryology, the portal venous system can develop aberrantly, leading to vascular connections between the portal and systemic venous circulation known as portosystemic shunts. The purpose of this comparative review with a few short representative case reports was to present the similarities and differences in portosystemic shunts in humans and small animals and their radiologic evaluation. Aberrant vascular connections between the portal and systemic venous circulation enable portal blood to bypass metabolism and detoxification in the liver, leading to significant clinical implications. Portosystemic shunts are very rare in humans, but these connections are much more common in small animals, affecting up to 0.6% of small animals. Portosystemic shunts can be congenital or acquired and are divided into intrahepatic and extrahepatic types. Because of its ability to accurately assess abdominal structures, large vessels, and their flow dynamics without anesthesia, ultrasonography has become the first imaging modality employed for the diagnostic evaluation of portosystemic shunts in both humans and small animals. This is usually followed by contrast-enhanced computed tomographic angiography in order to better define the exact shunt anatomy and to plan treatment. It is important to understand the embryology, anatomy, pathology, and pathophysiology of portosystemic shunts in order to understand the findings of radiologic imaging and to initiate appropriate treatment.

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21

Nguyen, Thai Van, Thinh Tien Nguyen, Hoang Duc Dong, and Huy Quang Duong. "First Report of Fulminant Budd-Chiari Syndrome Treated by Direct Intrahepatic Portocaval Shunt in Vietnam." Case Reports in Gastroenterology 15, no. 3 (September 30, 2021): 877–84. http://dx.doi.org/10.1159/000518925.

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Budd-Chiari syndrome (BCS) is a rare disorder caused by hepatic venous outflow obstruction that can lead to acute liver failure proposing liver transplantation or transjugular intrahepatic portosystemic shunt. However, the transjugular intrahepatic portosystemic shunt is not always successful due to the entire hepatic vein thrombosis while transplantation is not unfailingly feasible. In these situations, the direct intrahepatic portosystemic shunt (DIPS) is a viable alternative that may ameliorate portal hypertension in these patients. We described a case of a 21-year-old male with fulminant hepatic failure owning to BCS with a 4-day history of abdominal pain and nausea. Laboratory workup, including viral, autoimmune etiologies JAK2 mutation, Factor V Leiden, antiphospholipid antibody syndrome, was negative. The patient’s clinical status worsened with hepatic encephalopathy stage II despite administering anticoagulation. Thus, the patient underwent urgently DIPS after unaccessible to the creation of a transjugular intrahepatic portosystemic shunt and impossible to transplantation. The patient’s health was improved and discharged. Fulminant Budd-Chiari is a rare disease to be demanding prompt treatment. While transplantation or transjugular intrahepatic portosystemic shunt is failed, the DIPS is considered an alternative candidate associated with clinical improvement.

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Timpanaro, Tiziana, Stefano Passanisi, Alessandra Sauna, Claudia Trombatore, Monica Pennisi, Giuseppe Petrillo, Pierluigi Smilari, and Filippo Greco. "Congenital Portosystemic Shunt: Our Experience." Case Reports in Pediatrics 2015 (2015): 1–5. http://dx.doi.org/10.1155/2015/691618.

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Introduction. Congenital portosystemic venous malformations are rare abnormalities in which the portal blood drains into a systemic vein and which are characterized by extreme clinical variability.Case Presentations. The authors present two case reports of a congenital extrahepatic portosystemic shunt (Type II). In the first patient, apparently nonspecific symptoms, such as headache and fatigue, proved to be secondary to hypoglycemic episodes related to the presence of a portosystemic shunt, later confirmed on imaging. During portal vein angiography, endovascular embolization of the portocaval fistula achieved occlusion of the anomalous venous tract. In the second patient, affected by Down’s syndrome, the diagnosis of a portosystemic malformation was made by routine ultrasonography, performed to rule out concurrent congenital anomalies. Because of the absence of symptoms, we chose to observe this patient.Conclusions. These two case reports demonstrate the clinical heterogeneity of this malformation and the need for a multidisciplinary approach. As part of a proper workup, clinical evaluation must always be followed by radiographic diagnosis.

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Chang, Ting, Hsin-Ling Ho, Shao-Jung Hsu, Ching-Chih Chang, Ming-Hung Tsai, Teh-Ia Huo, Hui-Chun Huang, Fa-Yauh Lee, Ming-Chih Hou, and Shou-Dong Lee. "Glucobrassicin Metabolites Ameliorate the Development of Portal Hypertension and Cirrhosis in Bile Duct-Ligated Rats." International Journal of Molecular Sciences 20, no. 17 (August 26, 2019): 4161. http://dx.doi.org/10.3390/ijms20174161.

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Patients suffering from liver cirrhosis are often complicated with the formation of portosystemic collateral vessels, which is associated with the progression of a splanchnic hyperdynamic circulatory state. Alleviating pathological angiogenesis has thus been proposed to be a feasible treatment strategy. Indole-3-carbinol (C9H9NO, I3C) and 3,3′-diindolymethane (DIM), formed by the breakdown of glucosinolate glucobrassicin, are prevalent in cruciferous vegetables and have anti-angiogenesis properties. We aimed to evaluate their influences on portal hypertension, the severity of mesenteric angiogenesis, and portosystemic collaterals in cirrhosis. Sprague-Dawley rats with common bile duct ligation (CBDL)-induced liver cirrhosis or sham operation (surgical control) were randomly allocated to receive I3C (20 mg/kg/3 day), DIM (5 mg/kg/day) or vehicle for 28 days. The systemic and portal hemodynamics, severity of portosystemic shunting, mesenteric angiogenesis, and mesenteric proangiogenic factors protein expressions were evaluated. Compared to vehicle, both DIM and I3C significantly reduced portal pressure, ameliorated liver fibrosis, and down-regulated mesenteric protein expressions of vascular endothelial growth factor and phosphorylated Akt. DIM significantly down-regulated pErk, and I3C down-regulated NFκB, pIκBα protein expressions, and reduced portosystemic shunting degree. The cruciferous vegetable byproducts I3C and DIM not only exerted a portal hypotensive effect but also ameliorated abnormal angiogenesis and portosystemic collaterals in cirrhotic rats.

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Park, Jonathan K., Sung-Ki Cho, Stephen Kee, and Edward W. Lee. "Vascular Plug-Assisted Retrograde Transvenous Obliteration of Portosystemic Shunts for Refractory Hepatic Encephalopathy: A Case Report." Case Reports in Radiology 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/391420.

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While balloon-assisted retrograde transvenous obliteration (BRTO) has been used for two decades in Asia for the management of gastric variceal bleeding, it is still an emerging therapy elsewhere. Given the shunt closure brought about by the procedure, BRTO has also been used for the management of portosystemic encephalopathy with promising results. Modified versions of BRTO have been developed, including plug-assisted retrograde transvenous obliteration (PARTO), where a vascular plug is deployed within a portosystemic shunt. To our knowledge, we present the first North American case of PARTO in the setting of a large splenorenal shunt for the management of portosystemic encephalopathy.

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Filik, Levent, and Sedat Boyacioglu. "Asymptomatic Aneurysmal Portosystemic Venous Shunt: A Case Report and Review of the Literature." Acta Medica (Hradec Kralove, Czech Republic) 49, no. 4 (2006): 241–44. http://dx.doi.org/10.14712/18059694.2017.140.

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Spontaneous intrahepatic portosystemic venous shunt (SIPSVS) is relatively rare and not well recognized. Herein, we report 75-year-old female of an aneurysmal portosystemic venous shunt detected by colour Doppler ultrasound in check-up examination. A direct vascular communication between left portal vein and middle hepatic vein was confirmed by CT-angiography. The cause of intrahepatic portosystemic venous shunt is disputed. This abnormality, mainly described in cirrhotic liver and rarely in healthy liver, is usually revealed by hepatic encephalopathy or glycoregulation disorders. However, with improvements in imaging the number of reports of SIPSVS identified incidentally in patients without definite symptoms increasing.

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26

Řeháková, Kristína, Ivana Uhríková, Leona Raušerová-Lexmaulová, Jana Lorenzová, Ladislav Stehlík, Eva Jánová, Ondřej Škor, and Jaroslav Doubek. "Association of increased erythrocyte osmotic resistance with haematological and histopathological findings in dogs with a congenital extrahepatic portosystemic shunt." Acta Veterinaria Brno 82, no. 4 (2013): 393–98. http://dx.doi.org/10.2754/avb201382040393.

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The aim of the study was to investigate changes in erythrocyte osmotic resistance in relation to haematological and histological changes in dogs with a congenital portosystemic shunt. Osmotic fragility tests with complete blood counts and liver histological examinations were performed in 12 dogs with single extrahepatic portosystemic shunt confirmed by surgical exploration. Laboratory results were compared with those from 30 healthy dogs. Dogs with portosystemic shunt had a significantly increased erythrocyte osmotic resistance (P < 0.01) with 5%, 50% and 90% haemolysis corresponding to 0.45%, 0.35% and 0.30% NaCl solution, respectively. Statistical analyses revealed no correlation between haematological indicators and the osmotic fragility test results. Increased osmotic resistance was significantly associated with hepatic lipogranulomas. Based on these results, dogs with a congenital portosystemic shunt have a significantly increased erythrocyte osmotic resistance suggesting impaired red blood cell deformability. Osmotic resistance test that until now was not studied in canine hepatopathies seems to be independent of routinely performed haematological tests.

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Staubli, Sergej E. L., Tobias Gramann, Christoph Schwab, David Semela, Lukas Hechelhammer, Daniel S. Engeler, Hans-Peter Schmid, Dominik Abt, and Livio Mordasini. "Life-Threatening Bleeding from Peristomal Varices after Cystoprostatectomy: Multimodal Approach in a Cirrhotic, Encephalopathic Patient with Severe Portal Hypertension." Case Reports in Urology 2015 (2015): 1–3. http://dx.doi.org/10.1155/2015/785010.

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The bleeding of peristomal varices due to a portosystemic shunt is rare but potentially life-threatening in cirrhotic patients with portal hypertension. The scarce case reports in the literature recommend transjugular intrahepatic portosystemic shunt (TIPS) to prevent further bleeding. We report on a 72-year-old man who was referred to our hospital because of life-threatening bleeding from peristomal varices, three years after radical cystoprostatectomy for invasive bladder cancer. CT imaging showed liver cirrhosis with a prominent portosystemic shunt leading to massively enlarged peristomal varices. TIPS was taken into consideration, but not possible due to hepatic encephalopathy (HE). Medical therapy with lactulose and the nonselective beta-blocker carvedilol was initiated to treat HE and portal hypertension. In a second step, the portosystemic shunt was percutaneously embolized. Here, we present a multimodal approach to treat intractable bleeding from peristomal varices in a patient with ileal conduit urinary diversion, not suitable for TIPS.

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Wancata, Lauren M., Christopher R. Connelly, and Susan L. Orloff. "Portal Hypertension: Current Surgical Management." Digestive Disease Interventions 06, no. 02 (June 2022): 075–85. http://dx.doi.org/10.1055/s-0042-1748021.

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AbstractPortal hypertension occurs due to multiple disorders of the liver and its circulation. It is defined as an elevated pressure gradient between the portal circulation and the systemic circulation. The primary clinical consequences of portal hypertension are ascites and the development of portosystemic varices, which may cause life threatening hemorrhage. Modern management of portal hypertension includes medical, endoscopic therapy and transjugular intrahepatic portosystemic shunt placement by interventional radiology. Historically, portal hypertension was treated through the creation of surgical portosystemic shunts. In this chapter, the physiology of portal hypertension and the approaches to its management will be discussed. Specifically, the chapter will focus on indications, technical considerations, and types of surgical shunts used to treat portal hypertension, as well as literature supporting these procedures. Three case reports describing recent successful surgical shunt creation will be presented. Although portosystemic surgical shunt creation is performed far less frequently as medical, endoscopic and radiologic interventions have improved, this chapter illustrates that this procedure is a vital tool to be used in the treatment of portal hypertension.

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Tonge, Mary Emily. "CPD article: Sedation and general anaesthesia of the portosystemic shunt patient." Companion Animal 26, no. 5 (May 2, 2021): 1–9. http://dx.doi.org/10.12968/coan.2020.0103.

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Sedation and general anaesthesia may be required in animals with confirmed or suspected portosystemic shunt for a variety of reasons, not limited to shunt attenuation, such as diagnostic, routine or emergency procedures. Veterinary surgeons should understand normal hepatic functions and processes in order to appreciate the implications of portosystemic shunts associated specifically with sedation and anaesthesia. The pathophysiological and physiological variations, and their effects on anaesthesia and sedation, are discussed, as is management of the peri-anaesthetic period, drug choice for sedation or premedication, induction and maintenance of anaesthesia and analgesia. Patient monitoring and problem solving are also discussed, in relation to situations commonly encountered in portosystemic shunt patients during anaesthesia and sedation.

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Naidoo, Pumersha, Narisha Maharaj, Vanesha Naidu, and Jaynund Maharajh. "An unusual case of intrahepatic portosystemic venous shunt." South African Journal of Radiology 17, no. 2 (June 11, 2013): 57–58. http://dx.doi.org/10.4102/sajr.v17i2.244.

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Intrahepatic portosystemic venous shunts are rare vascular anomalies that may be detected in asymptomatic patients, given the recent advances in radiological imaging techniques. Accurate shunt evaluation and classification can be performed with ultrasound and multi-detector computed tomography. We report an unusual case of an intrahepatic portosystemic venous shunt with an incidental finding of neurofibroma.

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Lillegard, J. B., A. M. Hanna, T. J. McKenzie, C. R. Moir, M. B. Ishitani, and D. M. Nagorney. "A Single-Institution Review of Portosystemic Shunts in Children: An Ongoing Discussion." HPB Surgery 2010 (May 6, 2010): 1–6. http://dx.doi.org/10.1155/2010/964597.

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Purpose. Review the safety and long-term success with portosystemic shunts in children at a single institution. Methods. An IRB-approved, retrospective chart review of all children ages 19 and undergoing surgical portosystemic shunt from January 1990–September 2008. Results. Ten patients were identified, 8 females and 2 males, with a mean age of 15 years (range 5–19 years). Primary diagnoses were congenital hepatic fibrosis (5), hepatic vein thrombosis (2), portal vein thrombosis (2), and cystic fibrosis (1). Primary indications were repeated variceal bleeding (6), symptomatic hypersplenism (2), and significant liver dysfunction (2). Procedures performed were distal splenorenal bypass (4), side-to-side portocaval shunt (3), proximal splenorenal shunt (2), and an interposition H-graft portocaval shunt (1). There was no perioperative mortality and only minor morbidity. Seventy percent of patients had improvement of their symptoms. Eighty percent of shunts remained patent. Two were occluded at a median follow-up of 50 months (range 0.5–13.16 years). Two patients underwent subsequent liver transplantation. Two patients died at 0.5 and 12.8 years postoperatively, one from multisystem failure with cystic fibrosis and one from post-operative transplant complications. Conclusions. The need for portosystemic shunts in children is rare. However, in the era of liver transplantation, portosystemic shunts in selected patients with well-preserved liver function remains important. We conclude that portosystemic shunts are safe and efficacious in the control of variceal hemorrhage and symptoms related to hypersplenism.

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Trupac, Paola, and Petja Fister. "CONGENITAL PORTOSYSTEMIC VENOUS SHUNTS." Slovenska pediatrija, revija pediatrov Slovenije in specialistov šolske ter visokošolske medicine Slovenije 28, no. 1 (2021): 10–15. http://dx.doi.org/10.38031/slovpediatr-2021-1-02en.

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33

Park, J. H., S. H. Cha, J. K. Han, and M. C. Han. "Intrahepatic portosystemic venous shunt." American Journal of Roentgenology 155, no. 3 (September 1990): 527–28. http://dx.doi.org/10.2214/ajr.155.3.2117349.

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Tyagi, Ravi, Ana Gonzalez, Divya Meher Surabhi, and Rakesh Navuluri. "Transjugular Intrahepatic Portosystemic Shunt." Contemporary Diagnostic Radiology 45, no. 18 (August 31, 2022): 1–7. http://dx.doi.org/10.1097/01.cdr.0000856520.01226.68.

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Yamada, Ryusaku. "Transjugular Intrahepatic Portosystemic Shunt." Japanese Journal of Gastroenterological Surgery 27, no. 1 (1994): 163–66. http://dx.doi.org/10.5833/jjgs.27.163.

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Lee, Woong Hee, Young Tong Kim, Sung Shick Jou, and Hyeong Cheol Shin. "Congenital Intrahepatic Portosystemic Shunts." Journal of the Korean Radiological Society 59, no. 6 (2008): 405. http://dx.doi.org/10.3348/jkrs.2008.59.6.405.

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Hidajat, N., T. Vogl, H. Stobbe, J. Schmidt, C. Wex, R. Lenzen, T. Berg, P. Neuhaus, and R. Felix. "TRANSJUGULAR INTRAHEPATIC PORTOSYSTEMIC SHUNT." Acta Radiologica 41, no. 5 (September 2000): 474–78. http://dx.doi.org/10.1034/j.1600-0455.2000.041005474.x.

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Sanyal, Arun J., and Mitchell L. Shiffman. "Transjugular intrahepatic portosystemic shunts." Current Opinion in Gastroenterology 11, no. 3 (May 1995): 228–32. http://dx.doi.org/10.1097/00001574-199505000-00008.

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Skeens, J., C. Semba, and M. Dake. "TRANSJUGULAR INTRAHEPATIC PORTOSYSTEMIC SHUNTS." Annual Review of Medicine 46, no. 1 (February 1995): 95–102. http://dx.doi.org/10.1146/annurev.med.46.1.95.

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Cwikiel, W. "Transjugular intrahepatic portosystemic shunt." Acta Paediatrica 91, no. 4 (January 2, 2007): 374–75. http://dx.doi.org/10.1111/j.1651-2227.2002.tb01657.x.

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Guérin, Florent, Thomas Blanc, Frédéric Gauthier, Stephanie Franchi Abella, and Sophie Branchereau. "Congenital portosystemic vascular malformations." Seminars in Pediatric Surgery 21, no. 3 (August 2012): 233–44. http://dx.doi.org/10.1053/j.sempedsurg.2012.05.006.

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Burroughs, Andrew, and David Patch. "Transjugular Intrahepatic Portosystemic Shunt." Seminars in Liver Disease 19, no. 04 (1999): 457–73. http://dx.doi.org/10.1055/s-2007-1007132.

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Eagle, Kim, Michael A. Sadler, and Robert S. Shapiro. "Transjugular Intrahepatic Portosystemic Shunt." New England Journal of Medicine 330, no. 3 (January 20, 1994): 182. http://dx.doi.org/10.1056/nejm199401203300306.

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Ochs, Andreas. "Transjugular Intrahepatic Portosystemic Shunt." Digestive Diseases 23, no. 1 (2005): 56–64. http://dx.doi.org/10.1159/000084726.

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MADDISON, JE. "Canine congenital portosystemic encephalopathy." Australian Veterinary Journal 65, no. 8 (August 1988): 245–49. http://dx.doi.org/10.1111/j.1751-0813.1988.tb14310.x.

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Kamath, PS, and MA McKusick. "Transvenous intrahepatic portosystemic shunts." Gastroenterology 111, no. 6 (December 1996): 1700–1705. http://dx.doi.org/10.1016/s0016-5085(96)70035-5.

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Stern, Mark A., and Richard Moseley. "Transjugular intrahepatic portosystemic shunts." Gastroenterology 114, no. 1 (January 1998): 228. http://dx.doi.org/10.1016/s0016-5085(98)70664-x.

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Zemel, Gerald, Barry T. Katzen, Gary J. Becker, James F. Benenati, and D. Skip Sallee. "PERCUTANEOUS TRANSJUGULAR PORTOSYSTEMIC SHUNT." Southern Medical Journal 84, Supplement (September 1991): 77. http://dx.doi.org/10.1097/00007611-199109001-00281.

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Kimmey, Michael B., Chairman David A. Burnett, David L. Carr-Locke, Anthony J. DiMarino, Dennis M. Jensen, Ronald Katon, Bruce V. MacFadyen, Martin W. Scobey, Theodore N. Stein, and Steven M. Steinberg. "Transjugular intrahepatic portosystemic shunt." Gastrointestinal Endoscopy 39, no. 6 (January 1993): 889–91. http://dx.doi.org/10.1016/s0016-5107(93)70317-x.

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Patidar, Kavish R., Malcolm Sydnor, and Arun J. Sanyal. "Transjugular Intrahepatic Portosystemic Shunt." Clinics in Liver Disease 18, no. 4 (November 2014): 853–76. http://dx.doi.org/10.1016/j.cld.2014.07.006.

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