Relevant bibliographies by topics / Positive end expiratory pressure (PEEP)

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Positive end expiratory pressure (PEEP)'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Positive end expiratory pressure (PEEP)"

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RAZZAQUE, Usman, RAHEEL AZHAR, TASSADAQ KHURSHID, Khalid Zaeem, and Syed Majid. "POSITIVE END EXPIRATORY PRESSURE (PEEP);." Professional Medical Journal 19, no. 01 (2012): 098–104. http://dx.doi.org/10.29309/tpmj/2012.19.01.1953.

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Introduction: Thoracic surgeries and aesthesia for lung resection has presented anaesthesiologists with certain uniquephysiological problems. These include placing (lateral decubitus position) in order to obtain optimal access for most operations on lungs, pleura,esophagus, and great vessels, opening the chest wall (open pneumothorax) and one lung ventilation anaesthesia. One lung ventilationanaesthesia and lateral decubitus position produces decrease in functional residual capacity and an obligatory right to left shunt that rangesfrom 15% to 40% leading to increase in ventilation perfusion (V
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Matuschak, G. M., M. R. Pinsky, and R. M. Rogers. "Effects of positive end-expiratory pressure on hepatic blood flow and performance." Journal of Applied Physiology 62, no. 4 (1987): 1377–83. http://dx.doi.org/10.1152/jappl.1987.62.4.1377.

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Positive end-expiratory pressure (PEEP) may impair extrapulmonary organ function. However, the effects of PEEP on the liver are unclear. We tested the hypothesis that at a constant cardiac output (CO), PEEP does not induce changes in hepatic blood flow (QL) and parenchymal performance. In splenectomized, close-chested canine preparations (group I, n = 6), QL was derived as hepatic outflow using electromagnetic flow probes (QLemf), and hepatic performance was defined by extraction and clearance of indocyanine green (ICG). In a noninvasive model (group II, n = 7), the effects of PEEP on hepatic
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Östberg, Erland, Arnar Thorisson, Mats Enlund, Henrik Zetterström, Göran Hedenstierna, and Lennart Edmark. "Positive End-expiratory Pressure and Postoperative Atelectasis." Anesthesiology 131, no. 4 (2019): 809–17. http://dx.doi.org/10.1097/aln.0000000000002764.

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Abstract Editor’s Perspective What We Already Know about This Topic What This Article Tells Us That Is New Background Positive end-expiratory pressure (PEEP) increases lung volume and protects against alveolar collapse during anesthesia. During emergence, safety preoxygenation preparatory to extubation makes the lung susceptible to gas absorption and alveolar collapse, especially in dependent regions being kept open by PEEP. We hypothesized that withdrawing PEEP before starting emergence preoxygenation would limit postoperative atelectasis formation. Methods This was a randomized controlled ev
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Mazzinari, Guido, Oscar Diaz-Cambronero, Jose Miguel Alonso-Iñigo, et al. "Intraabdominal Pressure Targeted Positive End-expiratory Pressure during Laparoscopic Surgery." Anesthesiology 132, no. 4 (2020): 667–77. http://dx.doi.org/10.1097/aln.0000000000003146.

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Abstract Background Pneumoperitoneum for laparoscopic surgery is associated with a rise of driving pressure. The authors aimed to assess the effects of positive end-expiratory pressure (PEEP) on driving pressure at varying intraabdominal pressure levels. It was hypothesized that PEEP attenuates pneumoperitoneum-related rises in driving pressure. Methods Open-label, nonrandomized, crossover, clinical trial in patients undergoing laparoscopic cholecystectomy. “Targeted PEEP” (2 cm H2O above intraabdominal pressure) was compared with “standard PEEP” (5 cm H2O), with respect to the transpulmonary
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Henning, R. J. "Effects of positive end-expiratory pressure on the right ventricle." Journal of Applied Physiology 61, no. 3 (1986): 819–26. http://dx.doi.org/10.1152/jappl.1986.61.3.819.

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Transmural cardiac pressures, stroke volume, right ventricular volume, and lung water content were measured in normal dogs and in dogs with oleic acid-induced pulmonary edema (PE) maintained on positive-pressure ventilation. Measurements were performed prior to and following application of 20 cmH2O positive end-expiratory pressure (PEEP). Colloid fluid was given during PEEP for ventricular volume expansion before and after the oleic acid administration. PEEP significantly increased pleural pressure and pulmonary vascular resistance but decreased right ventricular volume, stroke volume, and mea
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Brandolese, R., C. Broseghini, G. Polese, et al. "Effects of intrinsic PEEP on pulmonary gas exchange in mechanically-ventilated patients." European Respiratory Journal 6, no. 3 (1993): 358–63. http://dx.doi.org/10.1183/09031936.93.06030358.

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The aim of the study was to assess the impact of the intrinsic positive end-expiratory pressure (PEEPi) on pulmonary gas exchange in mechanically-ventilated patients, by comparing the effects of similar levels (0.8-0.9 kPa) of positive end-expiratory pressure (PEEP) and PEEPi. Ten patients with acute respiratory failure, without chronic airway disease, were studied with three ventilatory modes: 1) intermittent positive pressure ventilation with zero end-expiratory pressure (ZEEP mode); 2) continuous positive pressure ventilation with PEEP set by the ventilator (PEEP mode); and 3) intrinsic PEE
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Formenti, Paolo, Sara Miori, Andrea Galimberti, and Michele Umbrello. "The Effects of Positive End Expiratory Pressure and Lung Volume on Diaphragm Thickness and Thickening." Diagnostics 13, no. 6 (2023): 1157. http://dx.doi.org/10.3390/diagnostics13061157.

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Introduction: Diaphragm dysfunction is common in patients undergoing mechanical ventilation. The application of positive end-expiratory pressure (PEEP) and the varying end-expiratory lung volume cause changes in diaphragm geometry. We aimed to assess the impact of the level of PEEP and lung inflation on diaphragm thickness, thickening fraction and displacement. Methods: An observational study in a mixed medical and surgical ICU was conducted. The patients underwent a PEEP-titration trial with the application of three random levels of PEEP: 0 cmH2O (PEEP0), 8 cmH2O (PEEP8) and 15 cmH2O (PEEP15)
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Takeuchi, Muneyuki, Sven Goddon, Marisa Dolhnikoff, et al. "Set Positive End-expiratory Pressure during Protective Ventilation Affects Lung Injury." Anesthesiology 97, no. 3 (2002): 682–92. http://dx.doi.org/10.1097/00000542-200209000-00023.

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Background The most appropriate method of determining positive end-expiratory pressure (PEEP) level during a lung protective ventilatory strategy has not been established. Methods In a lavage-injured sheep acute respiratory distress syndrome model, the authors compared the effects of three approaches to determining PEEP level after a recruitment maneuver: (1) 2 cm H(2)O above the lower inflection point on the inflation pressure-volume curve, (2) at the point of maximum curvature on the deflation pressure-volume curve, and (3) at the PEEP level that maintained target arterial oxygen partial pre
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Risoe, C., C. Hall, and O. A. Smiseth. "Splanchnic vascular capacitance and positive end-expiratory pressure in dogs." Journal of Applied Physiology 70, no. 2 (1991): 818–24. http://dx.doi.org/10.1152/jappl.1991.70.2.818.

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We have investigated the effect of positive end-expiratory pressure ventilation (PEEP) on regional splanchnic vascular capacitance. In 12 anesthetized dogs hepatic and splenic blood volumes were assessed by sonomicrometry. Vascular pressure-diameter curves were defined by obstructing hepatic outflow. With 10 and 15 cmH2O PEEP portal venous pressure increased 3.1 +/- 0.3 and 5.1 +/- 0.4 mmHg (P less than 0.001) while hepatic venous pressure increased 4.9 +/- 0.4 and 7.3 +/- 0.4 mmHg (P less than 0.001), respectively. Hepatic blood volume increased (P less than 0.01) 3.8 +/- 0.9 and 6.3 +/- 1.4
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van den Berg, B., H. Stam, and JM Bogaard. "Effects of PEEP on respiratory mechanics in patients with COPD on mechanical ventilation." European Respiratory Journal 4, no. 5 (1991): 561–67. http://dx.doi.org/10.1183/09031936.93.04050561.

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We studied the effects of positive end-expiratory pressure (PEEP) applied by the ventilator on respiratory mechanics in ventilated patients with chronic obstructive pulmonary disease (COPD). Airway pressures, relaxed expiratory flow-volume curves and end-expiratory volumes (EEV) were measured. In all patients investigated without PEEP applied by the ventilator, an intrinsic PEEP level (PEEPi) and a concavity in the flow-volume curve was present. Ventilator-PEEP caused a significant decrease in PEEPi in all patients (p less than 0.01). In patients in whom ventilator-PEEP exceeded PEEPi, signifi
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Dissertations / Theses on the topic "Positive end expiratory pressure (PEEP)"

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Madke, Gabriel Ribeiro. "Avaliação de dois diferentes niveis de peep no desempenho pós-operatório dos enxertos pulmonares em modelo suíno de transplante pulmonar unilateral esquerdo." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2011. http://hdl.handle.net/10183/148205.

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Objetivo: Avaliar os efeitos do uso de dois diferentes níveis de pressão expiratória final positiva (PEEP) em porcos submetidos a transplante pulmonar unilateral através de troca gasosa [pressão parcial de oxigênio (PaO2) e pressão parcial de dióxido de carbono (PaCO2)], hemodinâmica [freqüência cardíaca (FC), pressão arterial média (PAM) e pressão da artéria pulmonar (PAP)], parâmetros ventilatórios [pressão media das vias aéreas (Pmédia); complacência estática (Cst) e resistência das vias aéreas (Rest)], resposta inflamatória [interleuina 8 (IL-8)] e estresse oxidativo [substância reativa ao
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Lattuada, Marco. "Effect of Ventilatory Support on Abdominal Fluid Balance in a Sepsis Model." Doctoral thesis, Uppsala universitet, Klinisk fysiologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-207218.

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In patients affected by acute respiratory failure or acute respiratory distress syndrome (ARDS) the leading cause of death is failure of different vital organs other than the lungs, so called multiple organ dysfunction syndrome (MODS). The abdominal organs have a crucial role in the pathogenesis of this syndrome. There is a lack of knowledge regarding the mechanisms by which mechanical ventilation can affect the abdominal compartment. One hypothesis is that mechanical ventilation can interfere with abdominal fluid balance causing edema and inflammation. We addressed the question whether differ
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Mori, Audie Rollin Roldan. "Impacto de duas estratégias de titulação da PEEP em modelo suíno de síndrome do desconforto respiratório agudo: guiada por pressão esofágica versus guiada por tomografia de impedância elétrica." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5150/tde-27092017-114609/.

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INTRODUÇÃO: O uso de níveis elevados da pressão expiratória final positiva (PEEP) na Síndrome do desconforto respiratório agudo (SDRA), visando reduzir a quantidade de pulmão colapsado, tornando a ventilação mais homogênea, tem sido apontado por estudos clínicos randomizados e metaanálises como uma estratégia eficaz na melhora de alguns desfechos clínicos. Atualmente, não existe um método ideal para ajuste da PEEP na SDRA. Dois métodos distinguem-se pela racionalidade fisiológica e possibilidade de serem usados na prática clínica usual: ajuste da PEEP guiado por Pressão Esofágica (Pes) e ajust
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Perchiazzi, Gaetano. "Artificial Neural Networks (ANN) in the Assessment of Respiratory Mechanics." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4665.

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Johansson, Mats J. "Gas Exchange in the Normal Lung : Experimental studies on the effects of positive end-expiratory pressure and body position." Doctoral thesis, Linköpings universitet, Avdelningen för kardiovaskulär medicin, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-112364.

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BACKGROUND: The principal function of the lung is gas exchange requiring adequate ventilation and perfusion at the level of the alveoli. The efficiency of gas exchange is depending on the distributions of regional ventilation (V) and pulmonary blood flow (Q) and their correlation. AIMS: To validate a high-resolution method to quantify regional V and to investigate the combined effect of positive end-expiratory pressure (PEEP) and body position on distributions of regional V and Q in the normal lung with mechanical ventilation. To assess the matching of V and Q by calculating ventilation-perfus
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Edmark, Lennart. "Reducing Atelectasis during General Anaesthesia – the Importance of Oxygen Concentration, End-Expiratory Pressure and Patient Factors : A Clinical Study Exploring the Prevention of Atelectasis in Adults." Doctoral thesis, Uppsala universitet, Centrum för klinisk forskning, Västerås, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-209714.

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Background: The use of pure oxygen during preoxygenation and induction of general anaesthesia is a major cause of atelectasis. The interaction between reduced lung volume, resulting in airway closure, and varying inspiratory fractions of oxygen (FIO2) in determining the risk of developing atelectasis is still obscure. Methods: In this thesis, computed tomography (in studies I and II during anaesthesia, in studies III and IV postoperatively) was used to investigate the area of atelectasis in relation to FIO2 and varying levels of continuous positive airway pressure (CPAP) or positive end-expira
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Zannin, Emanuela, Raffaele L. Dellaca, Peter Kostic, et al. "Optimizing positive end-expiratory pressure by oscillatory mechanics minimizes tidal recruitment and distension : an experimental study in a lavage model of lung injury." Uppsala universitet, Anestesiologi och intensivvård, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-188730.

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INTRODUCTION: It is well established that during mechanical ventilation of patients with acute respiratory distress syndrome cyclic recruitment/derecruitment and overdistension are potentially injurious for lung tissues. We evaluated whether the forced oscillation technique (FOT) could be used to guide the ventilator settings in order to minimize cyclic lung recruitment/derecruitment and cyclic mechanical stress in an experimental model of acute lung injury. METHODS: We studied six pigs in which lung injury was induced by bronchoalveolar lavage. The animals were ventilated with a tidal volume
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Gusman, Pablo Braga [UNESP]. "Distribuição regional de gás e tecido na síndrome da angústia respiratória aguda: consequências do efeito da pressão expiratória final positiva." Universidade Estadual Paulista (UNESP), 2007. http://hdl.handle.net/11449/100145.

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Made available in DSpace on 2014-06-11T19:30:29Z (GMT). No. of bitstreams: 0 Previous issue date: 2007-02-02Bitstream added on 2014-06-13T19:40:09Z : No. of bitstreams: 1 gusman_pb_dr_botfm.pdf: 625469 bytes, checksum: 27745b379b0a215dafab05d5182357ac (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>Desde sua primeira descrição, muito se investiu no melhor conhecimento da SARA, na identificação de sua etiologia, seus fatores de risco, seus mecanismos e mediadores, escolhendo-se métodos críticos de avaliação clínica que também pudessem definir o prognóstico. Com
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Waugh, Jonathan Byron. "The effects of Furosemide, Surfactant, and Positive End- Expiratory pressure on gas exchange and lung impedance in a PMA dog model of acute lung injury /." The Ohio State University, 1994. http://rave.ohiolink.edu/etdc/view?acc_num=osu148786139602517.

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Gusman, Pablo Braga. "Distribuição regional de gás e tecido na síndrome da angústia respiratória aguda: consequências do efeito da pressão expiratória final positiva /." Botucatu : [s.n.], 2007. http://hdl.handle.net/11449/100145.

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Orientador: Luiz Antonio Vane<br>Banca: Yara Marcondes Machado Castiglia<br>Banca: José Reinaldo Cerqueira Braz<br>Banca: José Luiz Gomes do Amaral<br>Banca: Sílvia Regina Rios Vieira<br>Resumo: Desde sua primeira descrição, muito se investiu no melhor conhecimento da SARA, na identificação de sua etiologia, seus fatores de risco, seus mecanismos e mediadores, escolhendo-se métodos críticos de avaliação clínica que também pudessem definir o prognóstico. Com o objetivo de verificar se os efeitos da PEEP dependem da morfologia pulmonar, comparando sua resposta em três grupos de pacientes, descri
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Books on the topic "Positive end expiratory pressure (PEEP)"

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Martin-Loeches, Ignacio, and Antonio Artigas. Respiratory support with positive end-expiratory pressure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0094.

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Positive-end-expiratory pressure (PEEP) is the pressure present in the airway (alveolar pressure) above atmospheric pressure that exists at the end of expiration. The term PEEP is defined in two particular settings. Extrinsic PEEP (applied by ventilator) and intrinsic PEEP (PEEP caused by non-complete exhalation causing progressive air trapping). Applied (extrinsic) PEEP—is usually one of the first ventilator settings chosen when mechanical ventilation (MV) is initiated. Applying PEEP increases alveolar pressure and volume. The increased lung volume increases the surface area by reopening and
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Kreit, John W. Dynamic Hyperinflation and Intrinsic Positive End-Expiratory Pressure. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0010.

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Dynamic hyperinflation and intrinsic PEEP almost always occur in patients with severe obstructive lung disease, in whom slowing of expiratory flow prevents complete exhalation. Occasionally, patients without airflow obstruction develop dynamic hyperinflation when expiratory time, is excessively shortened by a rapid respiratory rate, a long set inspiratory time (TI), or both. Dynamic Hyperinflation and Intrinsic Positive End-Expiratory Pressure describes the causes of dynamic hyperinflation and the mechanisms of its adverse effects, including reduced cardiac output and blood pressure, pulmonary
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Sun, Crawford Ming. Study of blood-bone prostaglandin-like activity during positive end-expiratory pressure (PEEP) in anaesthetized dogs. 1986.

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Muders, Thomas, and Christian Putensen. Pressure-controlled mechanical ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0096.

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Beside reduction in tidal volume limiting peak airway pressure minimizes the risk for ventilator-associated-lung-injury in patients with acute respiratory distress syndrome. Pressure-controlled, time-cycled ventilation (PCV) enables the physician to keep airway pressures under strict limits by presetting inspiratory and expiratory pressures, and cycle times. PCV results in a square-waved airway pressure and a decelerating inspiratory gas flow holding the alveoli inflated for the preset time. Preset pressures and cycle times, and respiratory system mechanics affect alveolar and intrinsic positi
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Kiss, Thomas, and Paolo Pelosi. Lung recruitment techniques in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0120.

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Lung recruitment manoeuvres (RMs) have been suggested as a means of homogenizing the lung structure and distribution of the mechanical stress across the lungs. Such effects can be achieved provided enough pressure is applied for enough time at the airways, and maintained if adequate levels of positive end-expiratory pressure (PEEP) are used. When RMs effectively open atelectatic tissue, shear stress, and cyclic collapse/reopening are importantly reduced. The lung response to RMs is mainly determined by cause and severity of lung injury, and the position of the lungs with respect to the gravity
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Kreit, John W. Cardiovascular–Pulmonary Interactions. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0003.

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Intramural pressures within a tube or circuit determine the rate and direction of flow, whereas the transmural pressure of an elastic structure determines its volume. In Chapter 1, we applied these principles when talking about the pressure needed to overcome viscous forces and elastic recoil during ventilation. In this chapter, we use them to explain changes in blood flow between two portions of the circulatory system and changes in the volume and size of the heart chambers. Cardio–Pulmonary Interactions provides an overview of essential cardiovascular physiology as well as an in-depth discus
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Lucangelo, Umberto, and Massimo Ferluga. Pulmonary mechanical dysfunction in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0084.

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In intensive care units practitioners are confronted every day with mechanically-ventilated patients and should be able to sort out from all the data available from modern ventilators to tailored patient ventilatory strategy. Real-time visualization of pressure, flow and tidal volume provide valuable information on the respiratory system, to optimize ventilatory support and avoiding complications associated with mechanical ventilation. Early determination of patient–ventilator asynchrony, air-trapping, and variation in respiratory parameters is important during mechanical ventilation. A correc
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Kreit, John W. Respiratory Mechanics. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0001.

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Ventilation can occur only when the respiratory system expands above and then returns to its resting or equilibrium volume. This is just another way of saying that ventilation depends on our ability to breathe. Although breathing requires very little effort and even less thought, it’s nevertheless a fairly complex process. Respiratory Mechanics reviews the interaction between applied and opposing forces during spontaneous and mechanical ventilation. It discusses elastic recoil, viscous forces, compliance, resistance, and the equation of motion and the time constant of the respiratory system. I
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Kreit, John W. Acute Respiratory Distress Syndrome (ARDS). Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0012.

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Acute Respiratory Distress Syndrome reviews the definitions, causes, pathophysiology, and management of this relatively common, life-threatening disorder. This chapter describes how to ensure adequate tissue oxygen delivery while minimizing ventilator-induced lung injury and provides an in-depth review of how to determine the optimum level of positive end-expiratory pressure (PEEP). The first topic addressed is the precipitating factors and pathophysiology of acute respiratory distress syndrome. Next the chapter turns to mechanical ventilation, and covers the subjects of adequate oxygenation,
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Hedenstierna, Göran, and Hans Ulrich Rothen. Physiology of positive-pressure ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0088.

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During positive pressure ventilation the lung volume is reduced because of loss of respiratory muscle tone. This promotes airway closure that occurs in dependent lung regions. Gas absorption behind the closed airway results sooner or later in atelectasis depending on the inspired oxygen concentration. The elevated airway and alveolar pressures squeeze blood flow down the lung so that a ventilation/perfusion mismatch ensues with more ventilation going to the upper lung regions and more perfusion going to the lower, dependent lung. Positive pressure ventilation may impede the return of venous bl
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Book chapters on the topic "Positive end expiratory pressure (PEEP)"

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Citerio, G., C. Giussani, Hugo Sax, et al. "Intrinsic Positive End-Expiratory Pressure (PEEPi)." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_3179.

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Agostoni, P., E. Doria, M. Pepi, and G. Tamborini. "Effects of Positive End-Expiratory Pressure (PEEP) on Bronchial Blood Flow." In Current Topics in Rehabilitation. Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-3782-5_9.

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Zengin, Seniyye Ulgen, and Güniz Köksal. "Obesity and Positive End-Expiratory Pressure (PEEP)-Obesity and Recruitment Maneuvers During the Intraoperative Period." In Mechanical Ventilation in the Critically Ill Obese Patient. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-49253-7_14.

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Haider, M., and H. Schad. "Effect of Positive End-Expiratory Airway Pressure (PEEP) on Extravascular Thermal Lung Water Estimation in the Dog." In Practical Applications of Fiberoptics in Critical Care Monitoring. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75086-1_13.

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Fournell, A., T. W. L. Scheeren, and L. A. Schwarte. "Oxygenation of the Intestinal Mucosa in Anaesthetized Dogs is Attenuated by Intermittent Positive Pressure Ventilation (IPPV) with Positive End-Expiratory Pressure (PEEPO)." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5399-1_55.

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Lemaire, F., and C. Brun-Buisson. "Positive End Expiratory Pressure." In Mechanical Ventilation. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-87448-2_2.

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Ghavam, Sarvin, and Haresh Kirpalani. "Positive End-Expiratory Pressure." In Manual of Neonatal Respiratory Care. Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-2155-9_28.

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Pelosi, P., and D. Chiumello. "Use of Positive End-expiratory Pressure and Continuous Positive Airway Pressure." In Mechanics of Breathing. Springer Milan, 2002. http://dx.doi.org/10.1007/978-88-470-2916-3_24.

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Ranieri, V. M., L. Mascia, and R. Giuliani. "Positive End-Expiratory Pressure and Permissive Hypercapnia in ARDS." In Yearbook of Intensive Care and Emergency Medicine. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80053-5_36.

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Forst, H., J. Racenberg, K. Peter, and K. Messmer. "Right Ventricular Performance and Positive End-Expiratory Pressure Ventilation." In Current Concepts in Critical Care. Springer London, 1988. http://dx.doi.org/10.1007/978-1-4471-1443-7_8.

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Conference papers on the topic "Positive end expiratory pressure (PEEP)"

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Ito, Y., M. Vedrenne-Cloquet, J. C. Hotz, A. K. Bhalla, C. J. Newth, and R. G. Khemani. "Positive End-expiratory Pressure (PEEP) Targeting End-Expiratory Transpulmonary Pressure Compared With PEEP by the Low PEEP/FIO2 Table in Children With Acute Respiratory Distress Syndrome." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6920.

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Petersson, J., M. Ax, J. Frey, A. Sanchez-Crespo, SG Lindahl, and M. Mure. "Positive End-Expiratory Pressure (PEEP) Redistributes Regional Blood Flow and Ventilation Differently in Supine and Prone Humans." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3090.

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MacIntyre, Neil R., John Davies, Rita Hanel Hanel, Trisha Oura, Ian Robertson, and Don Thrall. "Carbon Monoxide And Acetylene Uptake In Acutely Injured Lungs At Different Levels Of Positive End Expiratory Pressure (PEEP)." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a6353.

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4

Cereda, Maurizio, Kiarash Emami, Yi Xin, et al. "Positive End Expiratory Pressure (PEEP) And Surfactant Administration Decrease Airspace Dilatation In Ventilated Rats After Pulmonary Saline Lavage." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5445.

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Xin, Yi, Maurizio Cereda, Kiarash Emami, et al. "Combined Effects Of Positive End-Expiratory Pressure (PEEP) And Surfactant Administration On Lung Dynamic Compliance In Surfactant Depleted Rats." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3061.

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Mansfield, Hunter, Jason Shenoi, and Sindhura Sridhar. "EXPEDITING ESOPHAGEAL MANOMETRY THROUGH CREATION OF A SLEEVE FOR A NASOGASTRIC TUBE." In 2023 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2023. http://dx.doi.org/10.1115/dmd2023-0629.

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Abstract:
Abstract Acute Respiratory Distress Syndrome (ARDS) is an acute inflammatory condition that causes impaired oxygenation and is almost always treated with mechanical ventilation. Positive End-Expiratory Pressure (PEEP) is an extrinsic pressure applied in ventilation to maintain alveolar opening. Esophageal manometry is a useful tool in titrating PEEP to ensure adequate ventilation is being provided in cases of increased chest wall pressure. However, few pulmonologists or critical care specialists utilize esophageal manometry routinely because of the required additional procedure, sparse trainin
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KO, TUO-YU, and Kuo-Chin Kao. "Comparison between positive end expiratory pressure guided by stress index and low PEEP/FiO2 table in moderate to severe ARDS." In ERS Congress 2024 abstracts. European Respiratory Society, 2024. http://dx.doi.org/10.1183/13993003.congress-2024.pa2611.

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Nickel, A., J. L. Allen, J. M. McDonough, and H. B. Panitch. "An Active Simulation Model of Intrinsic Positive End Expiratory Pressure (PEEPi) During Mechanical Ventilation." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5238.

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9

Ribeiro, B. M., M. R. Tucci, M. Victor Júnior, et al. "The Role of FIO2 in Lung Perfusion Distribution During Mechanical Ventilation of Supine Healthy Swines Using Low Positive End Expiratory Pressure (PEEP)." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5285.

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Milesi, Ilaria, Raffaele Dellacà, Simona Cacciatore, Luca Barbano, Roberto Porta, and Michele Vitacca. "Effects of posture on tidal Expiratory Flow Limitation (EFLT) and on minimum PEEP(Positive End Expiratory Pressure) able to abolish it in Chronic Obstructive Pulmonary Disease (COPD) patients." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2138.

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