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Relevant bibliographies by topics / Positive inotrope

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Academic literature on the topic 'Positive inotrope'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

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Journal articles on the topic "Positive inotrope"

1

Urquhart, Robert A., and Kenneth J. Broadley. "The indirect negative inotropic effects of the P1-receptor agonist, L-phenylisopropyladenosine, in guinea-pig isolated cardiac preparations: comparison with cromakalim." Canadian Journal of Physiology and Pharmacology 70, no. 6 (1992): 910–15. http://dx.doi.org/10.1139/y92-122.

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The possible mechanisms of the indirect negative inotropic responses to the P1-receptor agonist, L-phenylisopropyladenosine (L-PIA) were evaluated in electrically paced (2 Hz, 5 ms pulse width, voltage 50% above threshold) left atria and papillary muscles of guinea pigs. The responses were compared in naive tissues (direct effects) or after prestimulation with submaximal concentrations of either cAMP-dependent positive inotropes (isoprenaline or forskoiin) or the cAMP-independent inotrope Bay K 8644. Cumulative concentration – response curves were obtained in naive or prestimulated preparation

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2

Sakai, Mari, Tokiko Suzuki, Kengo Tomita, et al. "Diminished responsiveness to dobutamine as an inotrope in mice with cecal ligation and puncture-induced sepsis: attribution to phosphodiesterase 4 upregulation." American Journal of Physiology-Heart and Circulatory Physiology 312, no. 6 (2017): H1224—H1237. http://dx.doi.org/10.1152/ajpheart.00828.2016.

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Dobutamine has been used in septic shock for many years as an only inotrope, but its benefit has been questioned. We weighed the effects of dobutamine and milrinone as inotropes in mice with cecal ligation and puncture (CLP)-induced polymicrobial sepsis. CLP-induced septic mice exhibited significant cardiac inflammation, as indicated by greatly increased mRNAs of proinflammatory cytokines and robust infiltration of inflammatory cells in the ventricular myocardium. Elevations of plasma cardiac troponin-I showed cardiac injury in CLP mice. Noninvasive echocardiographic assessment of cardiac func

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3

&NA;. "Positive inotrope confusion helped by classification system." Inpharma Weekly &NA;, no. 911 (1993): 10. http://dx.doi.org/10.2165/00128413-199309110-00022.

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4

Ahmad, Tariq, P. Elliott Miller, Megan McCullough, et al. "Why has positive inotropy failed in chronic heart failure? Lessons from prior inotrope trials." European Journal of Heart Failure 21, no. 9 (2019): 1064–78. http://dx.doi.org/10.1002/ejhf.1557.

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5

Bose, D., T. Kobayashi, R. A. Bouchard, and L. V. Hryshko. "Scattered light intensity fluctuation in the canine ventricular myocardium: correlation with inotropic drug effect." Canadian Journal of Physiology and Pharmacology 66, no. 9 (1988): 1232–38. http://dx.doi.org/10.1139/y88-203.

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Scattered light intensity fluctuation (SLIF) of coherent light by a strip of ventricular muscle during diastole is believed to be due to asynchronous cellular motion within the myocyte as a result of spontaneous release of Ca from the sarcoplasmic reticulum. Previous studies have shown a correlation between inotropic agents, such as ouabain and elevated extracellular Ca or decreased extracellular Na, and SLIF. The purpose of this study was to see if this correlation could be extended to other inotropic agents. The digitalis genin, ouabagenin, produces inotropy by increasing intracellular free

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6

Greenberg, Stanley S., Jeff Paul, and Alan Luisi. "General pharmacology of CK-2130: A new selective positive inotrope." Drug Development Research 21, no. 4 (1990): 301–24. http://dx.doi.org/10.1002/ddr.430210405.

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7

Sikkel, Markus B., Carl Hayward, Kenneth T. MacLeod, Sian E. Harding, and Alexander R. Lyon. "SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope." British Journal of Pharmacology 171, no. 1 (2013): 38–54. http://dx.doi.org/10.1111/bph.12472.

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8

Tocchetti, Carlo, Jeffrey Froehlich, Hector Valdivia, et al. "Nitroxyl: A positive inotrope enhancing SR CA2+ cycling in isolated myocytes." Journal of Molecular and Cellular Cardiology 42, no. 6 (2007): S109. http://dx.doi.org/10.1016/j.yjmcc.2007.03.246.

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9

Mazza, Rosa, Alfonsina Gattuso, Sandra Imbrogno, et al. "Selenoprotein T as a new positive inotrope in the goldfish, Carassius auratus." Journal of Experimental Biology 222, no. 11 (2019): jeb201202. http://dx.doi.org/10.1242/jeb.201202.

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10

Koch, Sheryl E., Michael Tranter, Nathan Robbins, et al. "Probenecid as a Noninjurious Positive Inotrope in an Ischemic Heart Disease Murine Model." Journal of Cardiovascular Pharmacology and Therapeutics 18, no. 3 (2012): 280–89. http://dx.doi.org/10.1177/1074248412469299.

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Dissertations / Theses on the topic "Positive inotrope"

1

Weintraub, Joe͏̈lle. "Les hétérosides cardiotoniques : propriétés pharmacologiques et mécanisme de leur action inotrope positive." Paris 5, 1988. http://www.theses.fr/1988PA05P057.

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2

Chamberland, Caroline. "Action inotrope positive de l'apéline liée à l'augmentation de l'amplitude du courant sodique dans les myocytes cardiaque de chien." Mémoire, Université de Sherbrooke, 2008. http://savoirs.usherbrooke.ca/handle/11143/3965.

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L'apéline est l'agoniste du récepteur APJ-R (putative angiotensin II receptor like), récepteur couplé aux protéines G. Exprimée dans le coeur de plusieurs espèces dont l'homme, l'apéline joue un rôle important dans le système cardiovasculaire. L'apéline a un effet inotrope positif sur le coeur. Szokodi et al. ont démontré que cet effet était dû à l'activation de la cascade PLC-PKC et que les échangeurs sodium-hydrogène (NHE) et sodium-calcium (NCX) étaient impliqués. Ils ont également démontré que l'augmentation de la force contractile du myocarde n'était pas due à l'augmentation du courant ca

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3

ALEMANNI, MATTEO. "The modulation of SERCA pump activity as a tool for management of hearth failure." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2009. http://hdl.handle.net/10281/7462.

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The current positive inotropic interventions for the management of heart failure (HF) are all associated to an unchanged or increased long term mortality, which limit their use. Istaroxime (IST) is a novel positive inotrope able to inhibit Na+, K+ ATPase and to stimulate SERCA pump. We employed IST as a tool to assess the role of a mild SERCA stimulation as a positive inotropic intervention. In a guinea pig model of HF, where the sarcoplasmic reticulum (SR) function was deranged, IST was able to restore an optimal SR function, increase the SR Ca2+ content and to improve all the EC coupling par

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4

Razafinjatovo, Jean-Jacques. "Inhibiteurs de phosphodiestérases et autres inotropes positifs apparentes." Strasbourg 1, 1988. http://www.theses.fr/1988STR15067.

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5

Weisselberg, Tilman Wilhelm. "Die Wirkung von Stickstoffmonoxid auf Isoprenalin-induzierte positiv inotrope Effekte beim hypertrophierten Herzen der Ratte." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963216902.

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6

Bell, David. "Receptors for positive inotropic peptides and associated mechanisms of contractile coupling in isolated ventricular cardiomyocytes." Thesis, Queen's University Belfast, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.359022.

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7

Quiniou, Gilles. "Les nouveaux inotropes positifs dans le traitement de l'insuffisance cardiaque, à propos d'une étude ouverte au centre hospitalier de la côte basque sur l'Enoxinome oral." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25216.

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8

Marciniak, Gilbert. "Nouveaux heterocycles doues d'activites antiangineuses, inotropes positives ou antagonistes des canaux calciques : synthese, modelisation et relations structure-activite." Université Louis Pasteur (Strasbourg) (1971-2008), 1987. http://www.theses.fr/1987STR13142.

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Recherche par modelisation moleculaire sur ordinateur d'une structure commune a cinq inotrpes positifs connus ; synthese et proprietes pharmacologiques de nouveaux inotropes positifs (pyridyl-quinoles) ; preparation et pharmacologie de composes apparentes a la perhexiline ; synthese et activite biologique de dihydropyridines

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Marciniak, Gilbert. "Nouveaux hétérocycles doués d'activités antiangineuses, inotropes positives ou antagonistes des canaux calciques synthèse, modélisation et relations structure-activité /." Grenoble 2 : ANRT, 1987. http://catalogue.bnf.fr/ark:/12148/cb37607708r.

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10

Kania, Sebastian Martin Albert [Verfasser], Rolf [Akademischer Betreuer] Wachter, Wolfram-Hubertus [Gutachter] Zimmermann, and Wolfgang [Gutachter] Krick. "Der positiv inotrope Effekt von Insulin am menschlichen Myokard / Sebastian Martin Albert Kania. Betreuer: Rolf Wachter. Gutachter: Wolfram-Hubertus Zimmermann ; Wolfgang Krick." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2016. http://d-nb.info/1105759962/34.

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Books on the topic "Positive inotrope"

1

1929-, Braunwald Eugene, ed. Newer positive inotropic agents. American Heart Association, 1986.

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2

Gy, Papp J., ed. A symposium, positive inotropy by calcium sensitization: An evolving approach for the treatment of end-stage heart failure. Excerpta Medica, 1998.

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3

Arrigo, Mattia, and Alexandre Mebazaa. Positive inotropes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0035.

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Inotropic agents are substances used to improve cardiac output and end-organ perfusion in severe forms of acute heart failure. However, inappropriate use of inotropic agents may be associated with severe adverse effects and death. Despite clear indications to restrict their use to acute heart failure patients presenting with signs of end-organ hypoperfusion, the current use of inotropes is very frequent and often unnecessary. This chapter reviews mechanisms of action of current and future inotropes (including catecholamines, phosphodiesterase-III inhibitors, calcium sensitizers, cardiac myosin

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4

Cardiac Glycoside Receptors and Positive Inotropy: Evidence for More Than One Receptor? Springer, 1986.

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5

Schwarte, Lothar A., Stephan A. Loer, J. K. Götz Wietasch, and Thomas W. L. Scheeren. Cardiovascular drugs in anaesthetic practice. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0019.

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Anaesthetists should be familiar with currently available cardiovascular drugs used to maintain cardiovascular stability and achieve haemodynamic goals in surgical patients. The first part of this chapter summarizes antihypertensive agents, and the second part discusses positive inotropic drugs and vasopressors, which can be used perioperatively. Selection of vasoactive agents should be guided by the therapeutic goal (e.g. decreasing or increasing blood pressure or blood flow) and the underlying pathophysiology. Choice of catecholamines in a given situation should be based on the desired effec

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6

Erdmann, Erland, R. Jacob, and W. Schaper. Cardiac Glycoside Receptors and Positive Inotropy: Evidence for More Than One Receptor? - Symposium, Munich, October 26-29 1983. Steinkopff, Dietrich, 2011.

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7

Erdmann, Erland, R. Jacob, and W. Schaper. Cardiac Glycoside Receptors and Positive Inotropy: Evidence for more than one receptor? Symposium, Munich, October 26-29, 1983. Steinkopff, 2011.

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8

Erdmann, E., R. Jacob, and W. Schaper. Cardiac Glycoside Receptors and Positive Inotropy: Evidence for More Than One Receptor? Symposium, Munich, October 26-29 1983. Steinkopff, Dietrich, 2013.

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9

Muders, Thomas, and Christian Putensen. Pressure-controlled mechanical ventilation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0096.

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Beside reduction in tidal volume limiting peak airway pressure minimizes the risk for ventilator-associated-lung-injury in patients with acute respiratory distress syndrome. Pressure-controlled, time-cycled ventilation (PCV) enables the physician to keep airway pressures under strict limits by presetting inspiratory and expiratory pressures, and cycle times. PCV results in a square-waved airway pressure and a decelerating inspiratory gas flow holding the alveoli inflated for the preset time. Preset pressures and cycle times, and respiratory system mechanics affect alveolar and intrinsic positi

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Book chapters on the topic "Positive inotrope"

1

Schuster, H. P. "Vasodilatatoren und positiv-inotrope Pharmaka." In Intensivmedizin. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-97929-3_32.

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2

Schuster, H. P. "Vasodilatatoren und positiv-inotrope Pharmaka." In Intensivmedizin. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-97183-9_32.

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3

Richardt, G., and A. Schömig. "Therapie — Nichtglykosidische positiv-inotrope Substanzen." In Aktuelle Therapieprinzipien in Kardiologie und Angiologie. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77021-0_19.

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4

Hauf, G. F., and E. Grom. "Positiv-inotrope Substanzen in der Kardiologie." In Herzkrankheiten. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18649-3_39.

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5

Scholz, Hasso. "Introduction: Mechanisms of positive inotropic effects." In Inotropic Stimulation and Myocardial Energetics. Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-07908-9_1.

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6

Honerjäger, P. "Therapie mit positiv inotropen Substanzen." In Medikamentöse Behandlung des Postinfarktpatienten nach CAST. Steinkopff, 1991. http://dx.doi.org/10.1007/978-3-642-85414-9_13.

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7

Gralinski, Michael, Liomar A. A. Neves, and Olga Tiniakova. "Positive Inotropic Activity (Cardiac Glycosides)." In Drug Discovery and Evaluation: Pharmacological Assays. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-05392-9_9.

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8

Gralinski, Michael, Liomar A. A. Neves, and Olga Tiniakova. "Positive Inotropic Activity (Cardiac Glycosides)." In Drug Discovery and Evaluation: Pharmacological Assays. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27728-3_9-1.

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9

Erdmann, E., and M. Böhm. "Positive inotropic stimulation in the normal and insufficient human myocardium." In Inotropic Stimulation and Myocardial Energetics. Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-07908-9_12.

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10

Schmitz, Wilhelm, C. Kohl, J. Neumann, H. Scholz, and J. Scholz. "On the mechanism of positive inotropic effects of alpha-adrenoceptor agonists." In Inotropic Stimulation and Myocardial Energetics. Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-07908-9_4.

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Conference papers on the topic "Positive inotrope"

1

Haarmann, W., and H. Weisenberger. "LACK OF CORRELATION BETWEEN INTRAPLATELET cAMP INCREASE AND SYSTEMIC CIRCULATORY EFFECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644532.

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Compounds inhibiting platelet function by acting on platelet cAMP metabolism usually also have effects on the circulatory system, i.e. they decrease systemic blood pressure (bp) and are positive inotropic. For several compounds selected because of their distinct platelet inhibitory effects, the influence on these parameters in animals and on the cAMP metabolism in human platelets was determined.Inotropic effects and bp were measured via an indwelling catheter in anestetised cats after i.v. application of the test compounds. The inhibition of platelet PDEs was measured in freeze-thaw homogenate

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