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Journal articles on the topic 'Post cardiac arrest syndrome'

Author: Grafiati

Published: 4 June 2021

Last updated: 7 February 2022

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1

Dalessio, Linda. "Post–Cardiac Arrest Syndrome." AACN Advanced Critical Care 31, no. 4 (2020): 383–93. http://dx.doi.org/10.4037/aacnacc2020535.

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More than 356 000 out-of-hospital cardiac arrests occur in the United States annually. Complications involving post–cardiac arrest syndrome occur because of ischemic-reperfusion injury to the brain, lungs, heart, and kidneys. Post–cardiac arrest syndrome is a clinical state that involves global brain injury, myocardial dysfunction, macrocirculatory dysfunction, increased vulnerability to infection, and persistent precipitating pathology (ie, the cause of the arrest). The severity of outcomes varies and depends on precipitating factors, patient health before cardiac arrest, duration of time to

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2

Navarro-Vargas, José Ricardo, and José Luis Díaz. "Post cardiac arrest syndrome." Colombian Journal of Anesthesiology 42, no. 2 (2014): 107–13. http://dx.doi.org/10.1016/j.rcae.2014.01.004.

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3

Neumar, Robert W., Jerry P. Nolan, Christophe Adrie, et al. "Post–Cardiac Arrest Syndrome." Circulation 118, no. 23 (2008): 2452–83. http://dx.doi.org/10.1161/circulationaha.108.190652.

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4

Stub, Dion, Stephen Bernard, Stephen J. Duffy, and David M. Kaye. "Post Cardiac Arrest Syndrome." Circulation 123, no. 13 (2011): 1428–35. http://dx.doi.org/10.1161/circulationaha.110.988725.

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5

Navarro-Vargas, José Ricardo, and José Luis Díaz. "Post cardiac arrest syndrome☆." Colombian Journal of Anesthesiology 42, no. 2 (2014): 107–13. http://dx.doi.org/10.1097/01819236-201442020-00006.

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6

Bragina, N. V., T. G. Markova, and V. I. Gorbachev. "Post-cardiac arrest syndrome." Anesteziologiya i reanimatologiya, no. 4 (2021): 140. http://dx.doi.org/10.17116/anaesthesiology2021041140.

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7

Kang, Youngjoon. "Management of post-cardiac arrest syndrome." Acute and Critical Care 34, no. 3 (2019): 173–78. http://dx.doi.org/10.4266/acc.2019.00654.

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8

Biarent, Dominique, Christine Fonteyne, Ariane Willems, and Audrey Dupont. "Post-cardiac Arrest Syndrome in Children." Current Pediatric Reviews 9, no. 2 (2013): 125–33. http://dx.doi.org/10.2174/1573396311309020005.

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9

Tawfik, Mohamed Mohamed. "A Perspective on Post-Cardiac Arrest Syndrome." Anesthesia & Analgesia 128, no. 6 (2019): e114. http://dx.doi.org/10.1213/ane.0000000000004131.

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10

Soar, Jasmeet, and Jerry P. Nolan. "Mild hypothermia for post cardiac arrest syndrome." BMJ 335, no. 7618 (2007): 459–60. http://dx.doi.org/10.1136/bmj.39315.519201.be.

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11

McClelland, Heather. "Spreading the word – post-cardiac arrest syndrome." International Emergency Nursing 17, no. 4 (2009): 191–92. http://dx.doi.org/10.1016/j.ienj.2009.08.002.

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12

Miyazaki, Yusuke, and Fumito Ichinose. "Nitric Oxide in Post-cardiac Arrest Syndrome." Journal of Cardiovascular Pharmacology 75, no. 6 (2020): 508–15. http://dx.doi.org/10.1097/fjc.0000000000000765.

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13

Reynolds, Joshua C., and Benajmin J. Lawner. "Management of the Post-cardiac Arrest Syndrome." Journal of Emergency Medicine 42, no. 4 (2012): 440–49. http://dx.doi.org/10.1016/j.jemermed.2011.09.026.

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14

Zima, Endre. "Sudden Cardiac Death and Post Cardiac Arrest Syndrome. An Overview." Journal of Critical Care Medicine 1, no. 4 (2015): 167–70. http://dx.doi.org/10.1515/jccm-2015-0031.

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Abstract A satisfactory neurologic outcome is the key factor for survival in patients with sudden cardiac death (SCD), however this is highly dependent on the haemodynamic status. Short term cardiopulmonary resuscitation and regained consciousness on the return of spontaneous circulation (ROSC) is indicative of a better prognosis. The evaluation and treatment of SCD triggering factors and of underlying acute and chronic diseases will facilitate prevention and lower the risk of cardiac arrest. Long term CPR and a prolonged unconscious status after ROSC, in the Intensive Care Units or Coronary C

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15

Morris, S. "N025 Post-cardiac arrest syndrome and therapeutic hypothermia." Canadian Journal of Cardiology 27, no. 5 (2011): S345—S346. http://dx.doi.org/10.1016/j.cjca.2011.08.026.

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16

Bascom, Karen, Richard R. Riker, and David B. Seder. "Heart Rate and the Post Cardiac Arrest Syndrome." Critical Care Medicine 44, no. 2 (2016): 448–49. http://dx.doi.org/10.1097/ccm.0000000000001495.

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17

Choi, Jong Ho. "Management of Brain Injury after Post-cardiac Arrest Syndrome." Korean Journal of Critical Care Medicine 24, no. 1 (2009): 1. http://dx.doi.org/10.4266/kjccm.2009.24.1.1.

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18

Nolan, Jerry P., Robert W. Neumar, Christophe Adrie, et al. "Post-cardiac arrest syndrome: Epidemiology, pathophysiology, treatment, and prognostication." Resuscitation 79, no. 3 (2008): 350–79. http://dx.doi.org/10.1016/j.resuscitation.2008.09.017.

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19

Bauer, Gerhard, Eugen Trinka, and Peter W. Kaplan. "EEG Patterns in Hypoxic Encephalopathies (Post–Cardiac Arrest Syndrome)." Journal of Clinical Neurophysiology 30, no. 5 (2013): 477–89. http://dx.doi.org/10.1097/wnp.0b013e3182a73e47.

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20

Bouglé, Adrien, Arthur Le Gall, Florence Dumas, et al. "ExtraCorporeal life support for Cardiac ARrest in patients with post cardiac arrest syndrome: The ECCAR study." Archives of Cardiovascular Diseases 112, no. 4 (2019): 253–60. http://dx.doi.org/10.1016/j.acvd.2018.11.005.

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21

Jevdjic, Jasna, Filip Zunic, and Bojan Milosevic. "New Therapeutic Concepts in Post-Resuscitation Care." Serbian Journal of Experimental and Clinical Research 18, no. 3 (2017): 251–55. http://dx.doi.org/10.1515/sjecr-2016-0037.

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Abstract After the return of spontaneous circulation (ROSC), as a result of global ischaemia due to cardiac arrest followed by reperfusion, a condition develops called post-cardiac arrest syndrome. It manifests, alongside the pathology that caused the cardiac arrest, as a systemic inflammatory response, including severe cardio-circulatory and neurological dysfunction, leading to a fatal outcome. Th e aim of post-resuscitation care is to reduce the consequences of circulatory arrest, reperfusion, and the inflammatory response of the body on vital organ functions. The basis of post-resuscitation

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22

Czerwińska-Jelonkiewicz, Katarzyna, Johannes Grand, Guido Tavazzi, et al. "Acute respiratory failure and inflammatory response after out-of-hospital cardiac arrest: results of the Post-Cardiac Arrest Syndrome (PCAS) pilot study." European Heart Journal: Acute Cardiovascular Care 9, no. 4_suppl (2020): S110—S121. http://dx.doi.org/10.1177/2048872619895126.

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Background: Although the lungs are potentially highly susceptible to post-cardiac arrest syndrome injury, the issue of acute respiratory failure after out-of-hospital cardiac arrest has not been investigated. The objectives of this analysis were to determine the prevalence of acute respiratory failure after out-of-hospital cardiac arrest, its association with post-cardiac arrest syndrome inflammatory response and to clarify its importance for early mortality. Methods: The Post-Cardiac Arrest Syndrome (PCAS) pilot study was a prospective, observational, six-centre project (Poland 2, Denmark 1,

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23

Grundmann, Sebastian, Katrin Fink, Lyubomira Rabadzhieva, et al. "Perturbation of the endothelial glycocalyx in post cardiac arrest syndrome." Resuscitation 83, no. 6 (2012): 715–20. http://dx.doi.org/10.1016/j.resuscitation.2012.01.028.

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24

Chalkias, Athanasios. "Post-cardiac arrest syndrome: Mechanisms and evaluation of adrenal insufficiency." World Journal of Critical Care Medicine 1, no. 1 (2012): 4. http://dx.doi.org/10.5492/wjccm.v1.i1.4.

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25

Page, Michael. "They think it’s all over-managing post cardiac arrest syndrome." Journal of Paramedic Practice 4, no. 4 (2012): 195–200. http://dx.doi.org/10.12968/jpar.2012.4.4.195.

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26

Kelly, AM. "Post-Cardiac Arrest Syndrome: Time for a Bundle of Care?" Hong Kong Journal of Emergency Medicine 20, no. 4 (2013): 195–96. http://dx.doi.org/10.1177/102490791302000401.

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27

Markova, T. G., N. V. Bragina, and V. I. Gorbachev. "Prediction of Neurological Outcomes in Patients with Post-Cardiac Arrest Syndrome." Messenger of ANESTHESIOLOGY AND RESUSCITATION 18, no. 3 (2021): 72–78. http://dx.doi.org/10.21292/2078-5658-2021-18-3-72-78.

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Post-cardiac arrest syndrome is an extremely complex nosology, characterized by high mortality and the development of severe neurological disorders. Predicting the neurological outcome in this pathology is an urgent problem, since it allows determining the tactics of patient management and optimizing the scope of medical care, as well as preparing the patient's family members for expected results of treatment. Currently, clinical, laboratory and instrumental data are used as predictors of an unfavorable neurological outcome (e.g., pupillary responses, neuron-specific enolase levels, electroenc

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28

Anam, Ahmad Mursel, Raihan Rabbani, Farzana Shumy, et al. "Prolonged QTc: "Mind the Gap"." Bangladesh Critical Care Journal 2, no. 1 (2014): 44–45. http://dx.doi.org/10.3329/bccj.v2i1.19970.

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We report a case of drug induced torsades de pointes, following acquired long QT syndrome. The patient got admitted for shock with acute abdomen. The initial prolonged QT-interval was missed, and a torsadogenic drug was introduced post-operatively. Patient developed torsades de pointes followed by cardiac arrest. She was managed well and discharged without complications. The clinical manifestations of long QT syndromes, syncope or cardiac arrest, result from torsades de pointes. As syncope or cardiac arrest have more common differential diagnoses, even the symptomatic long QT syndrome are comm

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29

Krupičková, Petra, Zuzana Mormanová, Tomáš Bouček, Tomáš Belza, Jana Šmalcová, and Jan Bělohlávek. "Microvascular perfusion in cardiac arrest: a review of microcirculatory imaging studies." Perfusion 33, no. 1 (2017): 8–15. http://dx.doi.org/10.1177/0267659117723455.

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Cardiac arrest represents a leading cause of mortality and morbidity in developed countries. Extracorporeal cardiopulmonary resuscitation (ECPR) increases the chances for a beneficial outcome in victims of refractory cardiac arrest. However, ECPR and post-cardiac arrest care are affected by high mortality rates due to multi-organ failure syndrome, which is closely related to microcirculatory disorders. Therefore, microcirculation represents a key target for therapeutic interventions in post-cardiac arrest patients. However, the evaluation of tissue microcirculatory perfusion is still demanding

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30

Karanjia, Navaz, and Romergryko G. Geocadin. "Post–Cardiac Arrest Syndrome: Update on Brain Injury Management and Prognostication." Current Treatment Options in Neurology 13, no. 2 (2011): 191–203. http://dx.doi.org/10.1007/s11940-011-0112-2.

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31

Kelham, Matthew, Timothy N. Jones, Krishnaraj S. Rathod, et al. "An observational study assessing the impact of a cardiac arrest centre on patient outcomes after out-of-hospital cardiac arrest (OHCA)." European Heart Journal: Acute Cardiovascular Care 9, no. 4_suppl (2020): S67—S73. http://dx.doi.org/10.1177/2048872620974606.

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Background: Out-of-hospital cardiac arrest (OHCA) is a major cause of death worldwide. Recent guidelines recommend the centralisation of OHCA services in cardiac arrest centres to improve outcomes. In 2015, two major tertiary cardiac centres in London merged to form a large dedicated tertiary cardiac centre. This study aimed to compare the short-term mortality of patients admitted with an OHCA before-and-after the merger of services had taken place and admission criteria were relaxed, which led to managing OHCA in higher volume. Methods: We retrospectively analysed the data of OHCA patients pr

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32

Yeh, C., B. Camilotti, H. Hanif, et al. "P001: Proof-of-principle in a large animal pilot: cardiac arrest may be associated with acute, transient coagulopathy that may drive post-cardiac arrest syndrome." CJEM 22, S1 (2020): S64—S65. http://dx.doi.org/10.1017/cem.2020.209.

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Introduction: Many cardiac arrest survivors die later due to hemorrhage or thromboembolism, thought to be caused by acquired coagulopathy in post-cardiac arrest syndrome (PCAS) from shock and reperfusion injury. Understanding PCAS is a priority identified by the AHA for the prevention of complications in cardiac arrest survivors. Shock dysregulates both coagulation and fibrinolysis. The key effector enzyme thrombin (Th), is responsible for both up- and down-regulating coagulation and fibrinolysis. Measuring early Th activity may allow for predicting PCAS coagulopathy, and early medical interve

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33

Meyer, Martin Abild Stengaard, Sebastian Wiberg, Johannes Grand, et al. "Treatment Effects of Interleukin-6 Receptor Antibodies for Modulating the Systemic Inflammatory Response After Out-of-Hospital Cardiac Arrest (The IMICA Trial)." Circulation 143, no. 19 (2021): 1841–51. http://dx.doi.org/10.1161/circulationaha.120.053318.

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Background: Patients experiencing out-of-hospital cardiac arrest who remain comatose after initial resuscitation are at high risk of morbidity and mortality attributable to the ensuing post–cardiac arrest syndrome. Systemic inflammation constitutes a major component of post–cardiac arrest syndrome, and IL-6 (interleukin-6) levels are associated with post–cardiac arrest syndrome severity. The IL-6 receptor antagonist tocilizumab could potentially dampen inflammation in post–cardiac arrest syndrome. The objective of the present trial was to determine the efficacy of tocilizumab to reduce systemi

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34

Mai, Nguyen, Kathleen Miller-Rhodes, Sara Knowlden, and Marc W. Halterman. "The post-cardiac arrest syndrome: A case for lung–brain coupling and opportunities for neuroprotection." Journal of Cerebral Blood Flow & Metabolism 39, no. 6 (2019): 939–58. http://dx.doi.org/10.1177/0271678x19835552.

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Systemic inflammation and multi-organ failure represent hallmarks of the post-cardiac arrest syndrome (PCAS) and predict severe neurological injury and often fatal outcomes. Current interventions for cardiac arrest focus on the reversal of precipitating cardiac pathologies and the implementation of supportive measures with the goal of limiting damage to at-risk tissue. Despite the widespread use of targeted temperature management, there remain no proven approaches to manage reperfusion injury in the period following the return of spontaneous circulation. Recent evidence has implicated the lung

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35

Mayasi, Yunis, and Romergryko G. Geocadin. "Updates on the Management of Neurologic Complications of Post–Cardiac Arrest Resuscitation." Seminars in Neurology 41, no. 04 (2021): 388–97. http://dx.doi.org/10.1055/s-0041-1731310.

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AbstractSudden cardiac arrest (SCA) is one of the leading causes of mortality and morbidity in the United States, and survivors are frequently left with severe disability. Of the 10% successfully resuscitated from SCA, only around 10% of these live with a favorable neurologic outcome. Survivors of SCA commonly develop post–cardiac arrest syndrome (PCAS). PCAS is composed of neurologic, myocardial, and systemic injury related to inadequate perfusion and ischemia–reperfusion injury with free radical formation and an inflammatory cascade. While targeted temperature management is the cornerstone o

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36

Jou, Christopher, Rian Shah, Andrew Figueroa, and Jignesh K. Patel. "The Role of Inflammatory Cytokines in Cardiac Arrest." Journal of Intensive Care Medicine 35, no. 3 (2018): 219–24. http://dx.doi.org/10.1177/0885066618817518.

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Introduction: Post-cardiac arrest syndrome (PCAS) is characterized by systemic ischemia/reperfusion injury, anoxic brain injury, and post-arrest myocardial dysfunction superimposed on a precipitating pathology. The role of inflammatory cytokines in cardiac arrest remains unclear. Aims: We aimed to describe, with an emphasis on clinical applications, what is known about the role of inflammatory cytokines in cardiac arrest. Data Sources: A PubMed literature review was performed for relevant articles. Only articles in English that studied cytokines in patients with cardiac arrest were included. R

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37

Cour, Martin, Didier Bresson, Romain Hernu, and Laurent Argaud. "SOFA score to assess the severity of the post-cardiac arrest syndrome." Resuscitation 102 (May 2016): 110–15. http://dx.doi.org/10.1016/j.resuscitation.2016.03.001.

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38

Bro-Jeppesen, John, Pär I. Johansson, Christian Hassager, et al. "Endothelial activation/injury and associations with severity of post-cardiac arrest syndrome and mortality after out-of-hospital cardiac arrest." Resuscitation 107 (October 2016): 71–79. http://dx.doi.org/10.1016/j.resuscitation.2016.08.006.

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39

Choi, Yoon Hee, Dong Hoon Lee, Je Hyeok Oh, et al. "Inter-Hospital Transfer after Return of Spontaneous Circulation Shows no Correlation with Neurological Outcomes in Cardiac Arrest Patients Undergoing Targeted Temperature Management in Cardiac Arrest Centers." Journal of Clinical Medicine 9, no. 6 (2020): 1979. http://dx.doi.org/10.3390/jcm9061979.

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This study evaluated whether inter-hospital transfer (IHT) after the return of spontaneous circulation (ROSC) was associated with poor neurological outcomes after 6 months in post-cardiac-arrest patients treated with targeted temperature management (TTM). We used data from the Korean Hypothermia Network prospective registry from November 2015 to December 2018. These out-of-hospital cardiac arrest (OHCA) patients had either received post-cardiac arrest syndrome (PCAS) care at the same hospital or had been transferred from another hospital after ROSC. The primary endpoint was the neurological ou

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40

Nagase, Midori, Atsushi Sakurai, Atsunori Sugita, et al. "Oxidative stress and abnormal cholesterol metabolism in patients with post-cardiac arrest syndrome." Journal of Clinical Biochemistry and Nutrition 61, no. 2 (2017): 108–17. http://dx.doi.org/10.3164/jcbn.17-30.

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41

Tamura, Tomoyoshi, Masaru Suzuki, Kei Hayashida, et al. "Hydrogen gas inhalation alleviates oxidative stress in patients with post-cardiac arrest syndrome." Journal of Clinical Biochemistry and Nutrition 67, no. 2 (2020): 214–21. http://dx.doi.org/10.3164/jcbn.19-101.

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42

Jones, Alan E., Nathan I. Shapiro, J. Hope Kilgannon, and Stephen Trzeciak. "Goal-directed hemodynamic optimization in the post-cardiac arrest syndrome: A systematic review." Resuscitation 77, no. 1 (2008): 26–29. http://dx.doi.org/10.1016/j.resuscitation.2007.10.021.

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43

Álvarez-Fernández, Jesús A., and Raquel Pérez-Quintero. "Use of transcranial Doppler ultrasound in the management of post-cardiac arrest syndrome." Resuscitation 80, no. 11 (2009): 1321–22. http://dx.doi.org/10.1016/j.resuscitation.2009.07.011.

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44

Jahandiez, V., M. Cour, B. Pillot, N. Gallo-Bona, M. Ovize, and L. Argaud. "Mitochondrial cyclophilin D-dependent effects of therapeutic hypothermia in post-cardiac arrest syndrome." Archives of Cardiovascular Diseases Supplements 9, no. 2 (2017): 157. http://dx.doi.org/10.1016/s1878-6480(17)30387-7.

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45

Mongardon, Nicolas, Virginie Lemiale, Didier Borderie, et al. "Plasma thioredoxin levels during post-cardiac arrest syndrome: relationship with severity and outcome." Critical Care 17, no. 1 (2013): R18. http://dx.doi.org/10.1186/cc12492.

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46

Matsumoto, Gaku, Fumiaki Iwase, Junichi Inoue, et al. "289: PREDICTION OF NEUROLOGIC PROGNOSIS FOR POST-CARDIAC ARREST SYNDROME WITH BISPECTRAL INDEX." Critical Care Medicine 46, no. 1 (2018): 127. http://dx.doi.org/10.1097/01.ccm.0000528308.93604.61.

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47

Zima, Endre, Boldizsár Kiss, Anna Párkányi, et al. "Analysis and application of FORECAST (Forecast of Mortality Risk Early in the post Cardiac Arrest Syndrome Therapy) pilot score for estimate early mortality in post cardiac arrest syndrome patient population." Resuscitation 142 (September 2019): e98. http://dx.doi.org/10.1016/j.resuscitation.2019.06.237.

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48

Lam, Kin-Kwan, Fei-Lung Lau, Wai-Kwong Chan, and Wing-Nam Wong. "Effect of Severe Acute Respiratory Syndrome on Bystander Willingness to Perform Cardiopulmonary Resuscitation (CPR)–Is Compression–Only Preferred to Standard CPR?" Prehospital and Disaster Medicine 22, no. 4 (2007): 325–29. http://dx.doi.org/10.1017/s1049023x00004957.

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AbstractObjective:The effect of the severe acute respiratory syndrome (SARS) outbreak on the willingness of laypersons to provide bystander cardiopulmonary resuscitation (CPR) using standard CPR (SCPR) or compression-only CPR (CCPR) was evaluated.The preferred type of SCPR in the post-SARS era was assessed.Methods:A descriptive study was conducted through telephone interviews. Persons who attended a CPR coursefrom January 2000 through February 2003 answered a structured questionnaire. The respondents' willingnessto perform SCPR or CCPR during a witnessed cardiac arrest of an average adult stra

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49

Lu, Jing, Lijun Liu, Jianliang Zhu, and Xinying Guo. "Factors Influencing the Quality of Standardized Treatment for Patients with Post-Cardiac Arrest Syndrome." Korean Circulation Journal 47, no. 4 (2017): 455. http://dx.doi.org/10.4070/kcj.2016.0437.

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50

Polderman, Kees H., and Joseph Varon. "Cool hemodynamics – The intricate interplay between therapeutic hypothermia and the post-cardiac arrest syndrome." Resuscitation 85, no. 8 (2014): 975–76. http://dx.doi.org/10.1016/j.resuscitation.2014.06.002.

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