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Relevant bibliographies by topics / Post-Trascriptional gene silencing (PTGS)

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Academic literature on the topic 'Post-Trascriptional gene silencing (PTGS)'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Post-Trascriptional gene silencing (PTGS)"

1

Vaucheret, Hervé, Christophe Béclin, and Mathilde Fagard. "Post-transcriptional gene silencing in plants." Journal of Cell Science 114, no. 17 (2001): 3083–91. http://dx.doi.org/10.1242/jcs.114.17.3083.

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Post-transcriptional gene silencing (PTGS) in plants is an RNA-degradation mechanism that shows similarities to RNA interference (RNAi) in animals. Indeed, both involve double-stranded RNA (dsRNA), spread within the organism from a localised initiating area, correlate with the accumulation of small interfering RNA (siRNA) and require putative RNA-dependent RNA polymerases, RNA helicases and proteins of unknown functions containing PAZ and Piwi domains. However, some differences are evident. First, PTGS in plants requires at least two genes – SGS3 (which encodes a protein of unknown function co

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2

Tan, Huijuan, Bosheng Li, and Hongwei Guo. "The diversity of post-transcriptional gene silencing mediated by small silencing RNAs in plants." Essays in Biochemistry 64, no. 6 (2020): 919–30. http://dx.doi.org/10.1042/ebc20200006.

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Abstract In plants, post-transcriptional gene silencing (PTGS) tightly regulates development, maintains genome stability and protects plant against foreign genes. PTGS can be triggered by virus infection, transgene, and endogenous transcript, thus commonly serves as an RNA-based immune mechanism. Accordingly, based on the initiating factors, PTGS can be divided into viral-PTGS, transgene-PTGS, and endo-gene-PTGS. Unlike the intensely expressed invading transgenes and viral genes that frequently undergo PTGS, most endogenous genes do not trigger PTGS, except for a few that can produce endogenou

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3

Taochy, Christelle, Agnès Yu, Nicolas Bouché, et al. "Post-transcriptional gene silencing triggers dispensable DNA methylation in gene body in Arabidopsis." Nucleic Acids Research 47, no. 17 (2019): 9104–14. http://dx.doi.org/10.1093/nar/gkz636.

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Abstract Spontaneous post-transcriptional silencing of sense transgenes (S-PTGS) is established in each generation and is accompanied by DNA methylation, but the pathway of PTGS-dependent DNA methylation is unknown and so is its role. Here we show that CHH and CHG methylation coincides spatially and temporally with RDR6-dependent products derived from the central and 3′ regions of the coding sequence, and requires the components of the RNA-directed DNA methylation (RdDM) pathway NRPE1, DRD1 and DRM2, but not CLSY1, NRPD1, RDR2 or DCL3, suggesting that RDR6-dependent products, namely long dsRNA

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4

Sullivan, Jack M., Edwin H. Yau, Tiffany A. Kolniak, Lowell G. Sheflin, R. Thomas Taggart, and Heba E. Abdelmaksoud. "Variables and Strategies in Development of Therapeutic Post-Transcriptional Gene Silencing Agents." Journal of Ophthalmology 2011 (2011): 1–31. http://dx.doi.org/10.1155/2011/531380.

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Post-transcriptional gene silencing (PTGS) agents such as ribozymes, RNAi and antisense have substantial potential for gene therapy of human retinal degenerations. These technologies are used to knockdown a specific target RNA and its cognate protein. The disease target mRNA may be a mutant mRNA causing an autosomal dominant retinal degeneration or a normal mRNA that is overexpressed in certain diseases. All PTGS technologies depend upon the initial critical annealing event of the PTGS ligand to the target RNA. This event requires that the PTGS agent is in a conformational state able to suppor

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5

Mitsuhara, Ichiro, Naomi Shirasawa-Seo, Takayoshi Iwai, Shigeo Nakamura, Ryoso Honkura, and Yuko Ohashi. "Release From Post-transcriptional Gene Silencing by Cell Proliferation in Transgenic Tobacco Plants: Possible Mechanism for Noninheritance of the Silencing." Genetics 160, no. 1 (2002): 343–52. http://dx.doi.org/10.1093/genetics/160.1.343.

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Abstract Transgenic tobacco plants that overproduce luciferase (Luc) frequently exhibit post-transcriptional gene silencing (PTGS) of luc. The silencing was observed over five generations and found not to be inherited but acquired by the next generation at a certain frequency. Luc imaging analysis of silenced plants revealed Luc activity only in proliferating tissues such as shoot meristem and developing flower. The luc gene expression has been recovered from silencing before development of germ cells, excluding a possible recovery from the PTGS at meiosis. A systemic silencing signal transfer

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6

Singh, S., A. Green, P. Stoutjesdijk, and Q. Liu. "Inverted-repeat DMA: a new gene-silencing tool for seed lipid modification." Biochemical Society Transactions 28, no. 6 (2000): 925–27. http://dx.doi.org/10.1042/bst0280925.

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Post-transcriptional gene silencing (PTGS) has been successfully used to modify seed lipids in oilseed crops like soybean, canola and sunflower. Conventionally, PTGS has been induced by transforming the plants with either antisense or co-suppression constructs targeted against key seed lipid biosynthesis genes. A major drawback of this approach has been the recovery of only a modest proportion of silenced individuals from large populations of transgenic plants. In this report we show that inverted-repeat DNA constructs containing an intron encoding RNA with a hairpin structure can induce PTGS

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7

Meng, Chunying, Jun Chen, Jinrong Peng, and Sek-Man Wong. "Host-induced avirulence of hibiscus chlorotic ringspot virus mutants correlates with reduced gene-silencing suppression activity." Journal of General Virology 87, no. 2 (2006): 451–59. http://dx.doi.org/10.1099/vir.0.81578-0.

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Post-transcriptional gene silencing (PTGS) and virus-encoded gene-silencing suppressors are defence and counterdefence strategies developed by host and pathogens during evolution. Using a green fluorescence protein-based transient suppression system, the coat protein (CP) of Hibiscus chlorotic ringspot virus (HCRSV) was identified as a strong gene-silencing suppressor. CP suppressed sense RNA-induced but not dsRNA-induced local and systemic PTGS. This is different from another virus in the genus Carmovirus, Turnip crinkle virus (TCV), the CP of which strongly suppresses dsRNA-induced PTGS. HCR

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8

Youngson, Neil A., Pin-Chun Lin, and Shih-Shun Lin. "The convergence of autophagy, small RNA and the stress response – implications for transgenerational epigenetic inheritance in plants." BioMolecular Concepts 5, no. 1 (2014): 1–8. http://dx.doi.org/10.1515/bmc-2013-0032.

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AbstractRecent discoveries in eukaryotes have shown that autophagy-mediated degradation of DICER and ARGONAUTE (AGO), the proteins involved in post-transcriptional gene silencing (PTGS), can occur in response to viral infection and starvation. In plants, a virally encoded protein P0 specifically interacts with AGO1 and enhances degradation through autophagy, resulting in suppression of gene silencing. In HeLa cells, DICER and AGO2 protein levels decreased after nutrient starvation or after treatment to increase autophagy. Environmental exposures to viral infection and starvation have also rece

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9

Teycheney, Pierre-Yves, and Mark Tepfer. "Virus-specific spatial differences in the interference with silencing of the chs-A gene in non-transgenic petunia." Journal of General Virology 82, no. 5 (2001): 1239–43. http://dx.doi.org/10.1099/0022-1317-82-5-1239.

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Potyviruses, such as potato virus Y and tobacco etch virus, as well as cucumber mosaic cucumovirus, interfere with post-transcriptional gene silencing (PTGS). When RedStar-type Petunia hybrida cultivars, whose flowers have alternating white and pigmented sectors, were infected with these viruses, each virus induced a different pattern of restoration of floral anthocyanin pigmentation. Local reversion to coloured phenotypes in the white sectors, which occurred through interference with PTGS of the chalcone synthase A (chs-A) gene, was correlated with locally increased levels of chs-A mRNA and v

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10

Jan, Fuh-Jyh, Carmen Fagoaga, Sheng-Zhi Pang, and Dennis Gonsalves. "A single chimeric transgene derived from two distinct viruses confers multi-virus resistance in transgenic plants through homology-dependent gene silencing." Journal of General Virology 81, no. 8 (2000): 2103–9. http://dx.doi.org/10.1099/0022-1317-81-8-2103.

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We showed previously that 218 and 110 bp N gene segments of tomato spotted wilt virus (TSWV) that were fused to the non-target green fluorescent protein (GFP) gene were able to confer resistance to TSWV via post-transcriptional gene silencing (PTGS). N gene segments expressed alone did not confer resistance. Apparently, the GFP DNA induced PTGS that targetted N gene segments and the incoming homologous TSWV for degradation, resulting in a resistant phenotype. These observations suggested that multiple resistance could be obtained by replacing the GFP DNA with a viral DNA that induces PTGS. The

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