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Books on the topic 'Post-traumatic stress disorder PTSD Pregnancy'

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Published: 4 June 2021
Last updated: 1 February 2022

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1

Canada. Department of Veterans' Affairs. Post traumatic stress disorder (PTSD) and war-related stress. Ottawa, Ont: Veterans Affairs Canada, 2001.

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2

What nurses know-- PTSD. New York: Demos Health Pub., 2012.

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3

Craig, Gary. EFT for PTSD. Santa Rosa, CA: Energy Psychology, 2008.

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4

PTSD: My story, please listen! : post traumatic stress disorder. Bloomington, Ind: Authorhouse, 2010.

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5

CopShock: Surviving posttraumatic stress disorder (PTSD). Tucson, Ariz: Holbrook Street Press, 1999.

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6

Kates, Allen R. CopShock: Surviving posttraumatic stress disorder (PTSD). 2nd ed. Tucson, Ariz: Holbrook Street Press, 2008.

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7

J, Scott Michael. Counselling for post-traumatic stress disorder. 3rd ed. London: SAGE Publications, 2006.

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8

The chambers of memory: PTSD in the life stories of U.S. Vietnam veterans. Northvale, N.J: Jason Aronson, 1998.

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9

Katherine, Kirkland, ed. Why are you so scared?: A child's book about parents with PTSD. Washington, DC: Magination Press, 2012.

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10

Horowitz, Mardi Jon. Stress response syndromes: PTSD, grief, adjustment, and dissociative disorders. 5th ed. Lanham, Md: Jason Aronson, 2011.

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11

Lane, Timothy S. PTSD: Healing for bad memories. Greensboro, NC: New Growth Press, 2012.

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12

Stocker, Susan. Many faces of PTSD. Uniontown, Ohio: Holy Macro!, 2010.

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13

Pestonjee, D. M. A preliminary study of psychological aftereffects of post-traumatic stress disorder (PTSD) caused by earthquake: The Ahmedabad experience. Ahmedabad: Indian Institute of Management, 2001.

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14

PTSD and mild traumatic brain injury. New York: Guilford Press, 2012.

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15

At war with PTSD: Battling post traumatic stress disorder with virtual reality. Baltimore: Johns Hopkins University Press, 2012.

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16

1958-, Poijula Soili, ed. The PTSD workbook: Simple, effective techniques for overcoming traumatic stress symptoms. Oakland, Calif: New Harbinger Publications, 2002.

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17

Peraica, Ana. Victims symptom: (PTSD and culture). Amsterdam: Institute of Network Cultures, 2009.

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18

5 survivors: Personal stories of healing from PTSD and traumatic events. Center City, Minesota: Hazelden Publishing, 2011.

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19

Williams, Ruth, MA (Oxon), Dip. Psych. and Yule William, eds. Understanding post-traumatic stress: A psychosocial perspective on PTSD and treatment. Chichester: J. Wiley, 1997.

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20

David J. de L. Horne and Rod Watts. Coping with trauma: The victim and the helper. Bowen Hills, Qld: Australian Academic Press, 1994.

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21

Stress response syndromes: PTSD, grief, adjustment, and dissociative disorders. 5th ed. Lanham, Md: Jason Aronson, 2011.

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22

Ann, Hembree Elizabeth, and Rothbaum Barbara Olasov, eds. Prolonged exposure therapy for PTSD: Emotional processing of traumatic experiences : therapist guide. Oxford: Oxford University Press, 2007.

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23

Ayers, Susan, and Elizabeth Ford. Posttraumatic Stress During Pregnancy and the Postpartum Period. Edited by Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.18.

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Research on post-traumatic stress disorder (PTSD) in pregnancy and postpartum is relatively new but clearly demonstrates the importance of recognizing and treating women with PTSD at this time. Women with PTSD in pregnancy are at greater risk of pregnancy complications and health behaviors that have a negative impact on the woman and fetus. Approximately –3% of women develop PTSD after giving birth, and rates increase for women who have preterm or stillborn infants or life-threatening complications during pregnancy or labor. Models of the etiology of postpartum PTSD focus on the interaction among individual vulnerability, risk, and protective factors during and after birth. Research shows evidence for the role of previous psychiatric problems, depression in pregnancy, severe complications during birth, support, and women’s subjective experience of birth in postpartum PTSD. Very little research has examined screening or intervention. The chapter highlights key research topics that need addressing.
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24

Kornfield, Sara L., and C. Neill Epperson. PTSD and Women. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0013.

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It is generally accepted that women are at greater risk of lifetime post-traumatic stress disorder (PTSD) than men. Both gender differences in trauma exposures and cognitive response to trauma as well as sex differences in neuroendocrine function are thought to contribute to the differences in prevalence of PTSD across the lifespan. For women, reproductive transitions such as puberty, pregnancy, and menopause are particularly relevant, as ovarian and stress hormones as well as neurosteroids exert profound effects on the central nervous system. Similarly, pregnancy and childbirth can be experienced as traumatic events leading to exacerbation or new-onset PTSD. This chapter reviews the relevant literature regarding PTSD in women to highlight the importance of considering gender and sex as risk and resilience factors. The chapter is organized according to reproductive stage, as PTSD symptoms and treatment implications vary across the female lifespan.
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25

Bremner, J. Douglas. Post-Traumatic Stress Disorder (PTSD). Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199988709.013.0027.

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26

Diamond, David J., and Martha O. Diamond. Understanding and Treating the Psychosocial Consequences of Pregnancy Loss. Edited by Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.30.

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This chapter reviews research on the psychological impact and treatment of pregnancy loss for women, men, and families. The psychological sequelae of pregnancy loss can include mild to severe grief, complicated grief, depressive disorders, post-traumatic stress disorder (PTSD), and other anxiety disorders. Effects on couples, men, and other family members, including the impact on subsequent pregnancies, parental attachment to subsequent children, and gender differences in how men and women cope and grieve, are discussed. The authors present a conceptual framework for understanding pregnancy loss, positing that reactions largely depend on deeply personal and often unconscious meanings of pregnancy and on how parenthood fits into personal identity and the achievement of adult developmental tasks. The authors discuss unique aspects of pregnancy loss that may explain its broad impact and describe ways in which psychotherapy with pregnancy loss patients differs from psychotherapy with the general population, especially regarding countertransference and attitudes toward self-disclosure.
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27

Nemeroff, Charles B., and Charles Marmar, eds. Post-Traumatic Stress Disorder. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.001.0001.

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Post-Traumatic Stress Disorder (PTSD) reviews current knowledge and controversies of PTSD. The history, nosology, epidemiology, pathophysiology, risk factors, diagnosis, and treatment of post-traumatic stress disorder are described in detail. Also covered are the evidence-based treatments for PTSD, including all psychopharmacological agents (such as antidepressants and antipsychotics) as well as individual psychotherapies. The book delves into a vast array of cases involving at-risk groups such as minorities and offers a closer look at the coverage of PTSD across the world. The authors present state-of-the-art findings in genetics, epigenetics, neurotransmitter function, and brain imaging to provide the most current and thorough review of this burgeoning field.
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28

War and PTSD (Post Traumatic Stress Disorder). PublishAmerica, 2002.

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29

Rosenthal, Michele. Heal Your PTSD. Brilliance Audio, 2015.

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30

Canada, Veterans Affairs, ed. Post-traumatic stress disorder (PTSD) and war-related stress. [Ottawa]: Veterans Affairs Canada, 2006.

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31

Lewis, Catherine F. Anxiety disorders including post traumatic stress disorder (PTSD). Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199360574.003.0035.

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Increasing numbers of studies of correctional populations have emphasized diagnosis with structured clinical instruments over the past two decades. These studies have primarily focused on serious mental illness (i.e., psychotic and mood disorders), substance use disorders, and personality disorders. The focus has made sense because of the need to identify the severely mentally ill who are incarcerated and to identify the most common disorders. Anxiety disorders include generalized anxiety disorder, social anxiety disorder, panic disorder, and specific phobias. One anxiety disorder that stands apart from others is PTSD, which is prevalent at much higher rates in both incarcerated men and women than in the community. Despite this fact, other anxiety disorders are often co-morbid and add to overall disease burden and impair ability to function. Individuals with a greater disease burden (i.e., number of diagnoses, symptom counts) have worse outcomes than those with uncomplicated disorders. These impaired outcomes include a deteriorating trajectory of illness, increased health service utilization, poor prognosis, and increased likelihood of morbidity and mortality. Thus, while anxiety disorders may not be the primary focus of the correctional system, they must be recognized as important. Unrecognized anxiety disorders can result in behavior that is disruptive and may appear to be volitional. They can also lead to overutilization of health services that are already facing substantial demands. Appropriate, available, and consistent assessment, diagnosis, and treatment that are well integrated can successfully intervene in the range of anxiety disorders that present in correctional settings.
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32

Bind, Rebecca Hannah, and Carmine M. Pariante. Psychoneuroimmunology of Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0021.

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This chapter reviews the evidence linking post-traumatic stress disorder (PTSD) with changes in immune function. The chapter starts with a brief explanation of the components of the immune system, including cytokines, and of the mechanisms linking psychological and psychiatric phenomena with changes in immune function (i.e., psychoneuroimmunology). Specific studies on PTSD are then described, including the potential neurobiological and health consequences of these immune changes and, finally, the effects of PTSD treatment on both symptomology and the immune system. While there is a consistent pattern of findings indicating increased immune activation in this condition, there is a paucity of research on the immunological correlates of PTSD, especially compared with the large number of immunological studies on depression and other psychiatric disorders.
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33

PTSD: A Short History. Johns Hopkins University Press, 2018.

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34

Koen, N., T. Amos, J. Ipser, and D. Stein. Antidepressants in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0034.

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This chapter discusses the use of antidepressants in treating symptoms of posttraumatic stress disorder (PTSD). Tricyclic antidepressants were the first psychotropic agents to be studied systematically and rigorously for the treatment of PTSD. While early studies focused both on the tricyclics and monoamine oxidase inhibitors (MAOIs), more recent work has centered on the selective serotonin reuptake inhibitors (SSRIs); and paroxetine and sertraline are currently approved by the U.S. Food and Drug Administration (FDA) for use in this disorder. However, given the relatively small effect sizes in SSRI trials of PTSD, there is a need for ongoing psychopharmacological research to understand underlying mechanisms of antidepressant efficacy and to optimize response to pharmacotherapy. Further data on pediatric PTSD and on medication prophylaxis are needed before routine antidepressant treatment can be endorsed in these contexts.
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35

Craig, Gary. EFT for PTSD. Energy Psychology Press, 2009.

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36

Shalev, Arieh Y., Anna C. Barbano, Wei Qi, and Charles R. Marmar. Prevention of Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0037.

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Post-traumatic stress disorder (PTSD) follows an exposure to traumatic events and as such its onset and early development are better charted then those of most other mental disorders. It is not surprising, therefore, that major efforts have been dedicated to preventing its occurrence before, during and after trauma exposure. This chapter discusses the rationale, desirability, feasibility and outcome of interventions designed to prevent PTSD. Several efficient interventions have been documented. Barriers to their early implementations, however, greatly reduce their effectiveness and require urgent attention.
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37

North, Carol S. Post-Traumatic Stress Disorder Associated with Disaster. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0011.

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This chapter provides an overview of disaster-related post-traumatic stress disorder (PTSD). The chapter begins by examining the special role that disasters have in contributing to our general understanding of PTSD. It further explores the critical roles played by both disaster trauma and exposure to it toward the development of PTSD, by applying nosology of the disorder and understanding the construction of criteria for its diagnosis. The chapter explains procedures and methods for assessment of disaster-related PTSD in individuals and in populations, reviews risk factors for PTSD after disasters, and describes the post-disaster course of PTSD. Finally, the chapter provides an operational approach with a mental health framework to address PTSD associated with exposure to disaster.
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38

Church, Dawson. EFT for PTSD: (Posttraumatic stress disorder). 2017.

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39

Jones, Christina, and Richard D. Griffiths. Post-Traumatic Stress Disorder Following Critical Illness. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0021.

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Post-traumatic stress disorder (PTSD) has been shown to be a significant problem for both patients and relatives after critical illness. For patients the recall of delusional memories from the period in ICU can be a powerful trigger for the development of PTSD. Such memories are described by patients as very vivid and difficult to separate from reality. Early recognition and treatment of PTSD, where needed, can reduce the long term effects. Chronic PTSD, where symptoms have been present for three months after the traumatic event, is associated with a number of long term health problems such as chronic pain. It can also have profound effects on relationships, financial status and overall wellbeing. The provision of an ICU diary has been shown to reduce the incidence of PTSD in patients and reduce the level of PTSD-related symptoms in family members. For the majority of patients this relatively simple intervention helps them to fill in memory gaps and combat any delusional memories they may recall.
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40

Wynn, Gary H., and David M. Benedek. Complementary and Alternative Medicine for PTSD. Oxford University Press, Incorporated, 2016.

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41

Shalev, Arieh Y., and Charles R. Marmar. Conceptual History of Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0001.

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This chapter illustrates the conceptual and historical background of our current understanding of post-traumatic stress disorder (PTSD) and the implication of that history for current and future research and practice. It follows two parallel paths, conceptual and chronological, showing a progression in defining, recognizing, and diagnosing PTSD, The chapter discusses a stable conceptual space within which key clinical features of PTSD are conserved across time and cultures. The chapter also considers recent conceptual and definitional challenges emanating from new computational tools and advances in neurobehavioral research.
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42

J, Egan Sylvia, ed. Post-traumatic stress disorder (PTSD): Causes, symptoms, and treatment. Hauppauge, N.Y: Nova Science Publishers, 2010.

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43

Greenberg, Jennifer H. Post-Traumatic Stress Disorder and Sexual Intimacy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190461508.003.0008.

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This chapter is designed to guide clinicians in assisting service members to navigate the return from combat with regards to post-traumatic stress disorder (PTSD) and intimacy. The service members (and, by extension, their spouses) who served during Operation Iraqi Freedom/Operation Enduring Freedom are struggling with not only physical wounds from combat but also psychological/invisible wounds, including PTSD and traumatic brain injury (TBI). The symptoms of PTSD and TBI, including fatigue, irritability, insomnia, depression and cognitive deficits, overlap, and as a result, it can be difficult for clinicians and patients alike to tease out the specific diagnoses. Thus, this chapter discusses PTSD and intimacy issues for service members and spouses and provides recommendations for designing therapeutic interventions. Three case studies illustrate these interventions. Suggestion resources are also included.
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44

Perkins, Elizabeth C., Shaun P. Brothers, and Charles B. Nemeroff. Animal Models for Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0024.

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Animal models of post-traumatic stress disorder (PTSD) provide a wellspring of biological information about this complex condition by providing the opportunity to manipulate trauma exposure and measure biological outcomes in a systematic manner that is not possible in clinical studies. Symptoms of PTSD may be induced in animals by physical (immobilization, foot shock, underwater stress) and psychological stressors (exposure to predator, social defeat, early life trauma) or a combination of both. In addition, genetic, epigenetic and transgenic models have been created by breeding animals with a behavioral propensity for maladaptive stress response or by directly manipulating genes that have been implicated in PTSD. The effect of stressors in animals is measured by a variety of means, including observation of behavior, measurement of structural alterations in the brain and of physiological markers such as HPA axis activity and altered gene expression of central nervous system neurotransmitter system components including receptors. By comparing changes observed in stress exposed animals to humans with PTSD and by comparing animal response to treatments that are effective in humans, we can determine the validity of PTSD animal models. The identification of a reliable physiological marker of maladaptive stress response in animals as well as standard use of behavioral cutoff criteria are critical to the development of a valid animal model of PTSD.
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45

Wynn, Gary H., and David M. Benedek. Clinical Manual for Management of PTSD. American Psychiatric Association Publishing, 2010.

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46

Guffanti, Guia, Milissa L. Kaufman, Lauren A. M. Lebois, and Kerry J. Ressler. Genetic Approaches to Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0026.

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Post-traumatic stress disorder (PTSD) is a debilitating psychiatric disorder with an estimated genetic component accounting for 30%–40% of the variance contributing to risk for the disease. This chapter starts with a review of the biological hypotheses and related genetic mechanisms currently proposed to be associated with PTSD and trauma-related disorders. It will follow with a description of the state-of-the-art on the methodologies and their application to map genetic loci and identify biomarkers associated with PTSD. Finally, we will review the latest results from genome-wide association studies of genetic variants as well as those derived from the emerging fields of epigenetics and gene expression.
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47

Rosenthal, Michele, and Mary Beth Williams. Heal Your PTSD: Dynamic Strategies That Work. Red Wheel/Weiser, 2015.

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48

Chapman, Alexander L., Kim L. Gratz, and Matthew T. Tull. Cognitive Behavioral Coping Skills Workbook for PTSD. New Harbinger Publications, 2017.

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49

1945-, Rosen Gerald M., and Frueh B. Christopher, eds. Clinician's guide to post traumatic stress disorder. Hoboken, N.J: John Wiley & Sons, 2010.

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50

Newman, Jennifer, and Charles R. Marmar. Executive Function in Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0015.

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This chapter discusses the role of executive function in post-traumatic stress disorder (PTSD), which is far from fully understood. Deficits are subtle and findings are often inconsistent. Impairments have been related to worsening of psychological symptoms, functioning, and quality of life. They can also negatively impact treatment. Functional imaging shows that neurocognitive deficits in PTSD may be related to an imbalance in brain connectivity, where emotion processing is enhanced and control is reduced. Structural findings show abnormalities in brain regions involved in higher-level functions. However, findings are often discrepant. Factors related to these inconclusive results are considered, including developmental course, premorbid functioning, and comorbidities such as traumatic brain injury, depression, substance use, attention deficit hyperactivity disorder, health behaviors, and medical concerns. Treatment implications, limitations of this work, and future directions are presented. The aim of future research is to advance scientific understanding of PTSD, neurocognitive impairments, and related conditions, with the goal of improving outcomes for those who encounter trauma.
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