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Journal articles on the topic 'Postprandial triglycerides'

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Published: 4 June 2021

Last updated: 26 July 2025

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1

Lomangino, Kevin. "Postprandial Triglycerides." Clinical Nutrition INSIGHT 33, no. 10 (2007): 9. http://dx.doi.org/10.1097/01.nmd.0000294127.80085.c5.

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Folwaczny, Alexander, Elisa Waldmann, Julia Altenhofer, Kerstin Henze, and Klaus G. Parhofer. "Postprandial Lipid Metabolism in Normolipidemic Subjects and Patients with Mild to Moderate Hypertriglyceridemia: Effects of Test Meals Containing Saturated Fatty Acids, Mono-Unsaturated Fatty Acids, or Medium-Chain Fatty Acids." Nutrients 13, no. 5 (2021): 1737. http://dx.doi.org/10.3390/nu13051737.

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Fasting and postprandial hypertriglyceridemia are causal risk factors for atherosclerosis. The prevalence of hypertriglyceridemia is approximately 25–30% and most hypertriglyceridemic patients suffer from mild to moderate hypertriglyceridemia. Data regarding dietary interventions on postprandial triglyceride metabolism of mildly to moderately hypertriglyceridemic patients is, however, sparse. In a randomized controlled trial, eight mildly hypertriglyceridemic patients and five healthy, normolipidemic controls received three separate standardized fat-meals containing either saturated fatty acid

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Bruinstroop, Eveline, Susanne E. la Fleur, Mariette T. Ackermans, et al. "The autonomic nervous system regulates postprandial hepatic lipid metabolism." American Journal of Physiology-Endocrinology and Metabolism 304, no. 10 (2013): E1089—E1096. http://dx.doi.org/10.1152/ajpendo.00614.2012.

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The liver is a key organ in controlling glucose and lipid metabolism during feeding and fasting. In addition to hormones and nutrients, inputs from the autonomic nervous system are also involved in fine-tuning hepatic metabolic regulation. Previously, we have shown in rats that during fasting an intact sympathetic innervation of the liver is essential to maintain the secretion of triglycerides by the liver. In the current study, we hypothesized that in the postprandial condition the parasympathetic input to the liver inhibits hepatic VLDL-TG secretion. To test our hypothesis, we determined the

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Hansen, John-Bjarne, José A. Fernández, Ann-Trude With Notø, Hiroshi Deguchi, Johan Björkegren, and Ellisiv B. Mathiesen. "The Apolipoprotein C-I Content of Very-Low-Density Lipoproteins Is Associated with Fasting Triglycerides, Postprandial Lipemia, and Carotid Atherosclerosis." Journal of Lipids 2011 (2011): 1–9. http://dx.doi.org/10.1155/2011/271062.

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Background. Experimental studies in animals suggest that apolipoprotein (apo) C-I is an important regulator of triglycerides in fasting and postprandial conditions and associated with carotid atherosclerosis.Methods. A cross-sectional study was conducted with 81 subjects, aged 56–80 years recruited from a population health survey. The participants underwent a fat tolerance test (1 g fat per Kg body weight) and carotid atherosclerosis was determined by ultrasound examination. VLDL particles, Sf 20–400, were isolated and their lipid composition and apoC-I content determined.Results. The carotid

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Georgopoulos, Angeliki, Omer Aras, and Michael Y. Tsai. "Codon-54 Polymorphism of the Fatty Acid-Binding Protein 2 Gene Is Associated with Elevation of Fasting and Postprandial Triglyceride in Type 2 Diabetes*." Journal of Clinical Endocrinology & Metabolism 85, no. 9 (2000): 3155–60. http://dx.doi.org/10.1210/jcem.85.9.6791.

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Abstract Patients with type 2 diabetes are frequently dyslipidemic or hypertriglyceridemic. To assess whether increased intestinal triglyceride input leads to elevated fasting and postprandial triglycerides in type 2 diabetes, we used the codon 54 polymorphism of the fatty acid-binding protein 2 gene, which results in the substitution of threonine (Thr) for alanine and is associated with increased intestinal input of triglyceride. Of the 287 diabetic patients screened, 108 (37.6%) were heterozygous and 31 (10.8%) were homozygous for the Thr-54 allele. Mean (±sem) fasting plasma triglyceride le

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Parhofer, Klaus G., P. Hugh R. Barrett, and Peter Schwandt. "Atorvastatin Improves Postprandial Lipoprotein Metabolism in Normolipidemic Subjects1." Journal of Clinical Endocrinology & Metabolism 85, no. 11 (2000): 4224–30. http://dx.doi.org/10.1210/jcem.85.11.6978.

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Atorvastatin is a potent HMG-CoA reductase inhibitor that decreases low-density lipoprotein (LDL) cholesterol and fasting triglyceride concentrations. Because of the positive association between elevated postprandial lipoproteins and atherosclerosis, we investigated the effect of atorvastatin on postprandial lipoprotein metabolism. The effect of 4 weeks of atorvastatin therapy (10 mg/day) was evaluated in 10 normolipidemic men (30 ± 2 yr; body mass index, 22 ± 3 kg/m2; cholesterol, 4.84 ± 0.54 mmol/L; triglyceride, 1.47 ± 0.50 mmol/L; high-density lipoprotein cholesterol, 1.17 ± 0.18 mmol/L; L

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Belahsen, Rekia, та Yves Deshaies. "Modulation of lipoprotein lipase activity in the rat by the β2-adrenergic agonist clenbuterol". Canadian Journal of Physiology and Pharmacology 70, № 12 (1992): 1555–62. http://dx.doi.org/10.1139/y92-223.

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This study evaluated the effects of β2-adrenoceptor stimulation on some determinants of triglyceride metabolism. Male Sprague–Dawley rats were injected twice daily with clenbuterol (30 μg∙kg−1) for 7 days, or with an equivalent volume of vehicle. Serum triglycerides, hepatic triglyceride secretion rate, and lipoprotein lipase activity in white and brown adipose tissues as well as in red vastus lateralis muscle and heart were evaluated in the fasting state and following a fat-free, high-sucrose meal, 3 h after the last agonist injection. In rats killed in the fasting and postprandial states, cl

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Widdowson, William M., Anne McGowan, James Phelan, Gerard Boran, John Reynolds, and James Gibney. "Vascular Disease Is Associated With the Expression of Genes for Intestinal Cholesterol Transport and Metabolism." Journal of Clinical Endocrinology & Metabolism 102, no. 1 (2016): 326–35. http://dx.doi.org/10.1210/jc.2016-2728.

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Abstract Context: Intestinal cholesterol metabolism is important in influencing postprandial lipoprotein concentrations, and might be important in the development of vascular disease. Objective: This study evaluated associations between expression of intestinal cholesterol metabolism genes, postprandial lipid metabolism, and endothelial function/early vascular disease in human subjects. Design/Patients: One hundred patients undergoing routine oesophago-gastro-duodenoscopy were recruited. mRNA levels of Nieman-Pick C1-like 1 protein (NPC1L1), ABC-G5, ABC-G8, ABC-A1, microsomal tissue transport

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Picard, Frédéric, André Boivin, Josée Lalonde, and Yves Deshaies. "Resistance of adipose tissue lipoprotein lipase to insulin action in rats fed an obesity-promoting diet." American Journal of Physiology-Endocrinology and Metabolism 282, no. 2 (2002): E412—E418. http://dx.doi.org/10.1152/ajpendo.00307.2001.

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This study aimed to assess whether adipose lipoprotein lipase (LPL) becomes resistant to insulin in a nutritional model of resistance of glucose metabolism to insulin. Sprague-Dawley rats were fed for 4 wk chow or a purified high-sucrose, high-fat (HSHF) diet that induced overt insulin resistance. Rats were fasted for 24 h and then refed chow for 1, 3, or 6 h. The postprandial rise in insulinemia was similar in both dietary cohorts, whereas glycemia was higher in HSHF-fed than in chow-fed animals, indicating glucose intolerance and insulin resistance. In chow-fed rats, adipose LPL activity inc

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Cardona, Fernando, Sonsoles Morcillo, Montserrat Gonzalo-Marín, Lourdes Garrido-Sanchez, Manuel Macias-Gonzalez, and Francisco J. Tinahones. "Pro12Ala Sequence Variant of the PPARG Gene Is Associated with Postprandial Hypertriglyceridemia in Non-E3/E3 Patients with the Metabolic Syndrome." Clinical Chemistry 52, no. 10 (2006): 1920–25. http://dx.doi.org/10.1373/clinchem.2006.069690.

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Abstract Background: Postprandial hypertriglyceridemia, a component of the metabolic syndrome, has varied etiology and involves many genes related to triglyceride metabolism. Variations in these genes may affect postprandial hypertriglyceridemia in the context of the metabolic syndrome. Methods: We orally administered 60 g of fat overload to 74 patients with the metabolic syndrome. We then measured baseline concentrations of cholesterol, triglycerides, HDL cholesterol, apolipoprotein AI, apolipoprotein B, uric acid, and uric acid excretion; we also performed homeostasis model assessments of in

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Silveira, A. "Postprandial triglycerides and blood coagulation." Experimental and Clinical Endocrinology & Diabetes 109, no. 04 (2001): 527–32. http://dx.doi.org/10.1055/s-2001-15115.

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Jagla, A., and J. Schrezenmeir. "Postprandial triglycerides and endothelial function." Experimental and Clinical Endocrinology & Diabetes 109, no. 04 (2001): 533–47. http://dx.doi.org/10.1055/s-2001-15116.

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Hossain, Md Kamal, Farhena Ahmed, SM Suhrawardy, Mahmudul Hoque, Monira Khatun, and Nasrin Akter. "Study on Relationship Between Postprandial Triglycerides With Overweight and Obesity." Journal of Chittagong Medical College Teachers' Association 27, no. 2 (2017): 67–71. http://dx.doi.org/10.3329/jcmcta.v27i2.62368.

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Overweight and obesity are the major health problem in both developed and devloping countries. Various lipid abnormalities are observed in overweight and obese population. The objective of the study to evaluate relationship between postprandial triglycerides with overweight and obesity.The present case control study was carried out in the Department of Biochemistry, Chittagong Medical College. Samples were collected from population of different area of Chittagong City Corporation of different occupations, age from 35-60 years. The data were collected by a structured questionaries. BMI was calc

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Errim Ahl Cheikh, Kawtar Benkhaldoun, Saliha Chellak, and Abderrahmane Boukhira. "Diagnostic value of postprandial triglycerides testing in diabetic patients: Case series." GSC Advanced Research and Reviews 21, no. 3 (2024): 299–304. https://doi.org/10.30574/gscarr.2024.21.3.0503.

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Postprandial hypertriglyceridemia is now established as an important risk factor for cardiovascular disease (CVD). This metabolic abnormality is principally initiated by overproduction and/or decreased catabolism of triglyceride-rich lipoproteins (TRLs) and is a consequence of predisposing genetic variations and medical conditions such as obesity and insulin resistance. Accumulation of TRLs in the postprandial state promotes the retention of remnant particles in the artery wall. Because of their size, most remnant particles cannot cross the endothelium as efficiently as smaller low-density lip

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Keirns, Bryant H., Christina M. Sciarrillo, Samantha M. Hart, and Sam R. Emerson. "Comparison of a Standardized High-Fat Meal versus a High-Fat Meal Scaled to Body Mass for Measuring Postprandial Triglycerides: A Randomized Crossover Study." Metabolites 12, no. 1 (2022): 81. http://dx.doi.org/10.3390/metabo12010081.

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Post-meal triglycerides are an independent cardiovascular disease (CVD) risk factor, but the ideal high-fat meal formulation has yet to be standardized and is one challenge prohibiting widespread clinical adoption of postprandial triglyceride assessment. Two general approaches often used are giving individuals a high-fat meal scaled to body weight or a standardized high-fat meal containing a set fat bolus. A recent expert panel statement has endorsed the latter, specifying 75 g of fat as an appropriate fat dosage. Despite this recommendation, no study to date has tested whether there is a diff

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Freudenberger, Sadie, Bryant Keirns, Christina Sciarrillo, et al. "Body Composition Measures Associated With Postprandial Triglyceride Concentrations." Current Developments in Nutrition 6, Supplement_1 (2022): 441. http://dx.doi.org/10.1093/cdn/nzac057.007.

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Abstract Objectives A large increase in triglyceride (TG) concentrations following a high-fat meal (i.e., postprandial lipemia) is an independent cardiovascular disease risk factor. However, little is known regarding individual factors that are associated with or determine postprandial triglycerides. We aimed to identify body composition measures that are associated with postprandial triglyceride concentrations following a high-fat meal. Methods We conducted a secondary analysis of data from 5 previously conducted studies in our laboratory, each utilizing identical methods. Postprandial TG wer

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Zilversmit, D. B. "Atherogenic nature of triglycerides, postprandial lipidemia, and triglyceride-rich remnant lipoproteins." Clinical Chemistry 41, no. 1 (1995): 153–58. http://dx.doi.org/10.1093/clinchem/41.1.153.

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Abstract In addition to low-density lipoproteins, plasma chylomicrons and very-low-density lipoproteins (VLDL) contribute to atherogenesis. When triglyceride-rich particles bind to arterial endothelium and to deendothelialized areas, locally present lipoprotein lipase initiates triglyceride hydrolysis and decreases the size of the adhering particles. Additional changes in composition are brought about by the exchange of lipids between chylomicron/VLDL remnants and the cholesteryl ester-rich low- and high-density lipoproteins. These exchanges are mediated by lipid transfer proteins in plasma. A

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Weinstein, Marie Ritthamel, Katheryn Haugen, John Hewitt, and Diane Finan. "The Effects of Formula Change on Triglycerides and Free Fatty Acids in Preterm Infants." Journal of Pediatric Gastroenterology and Nutrition 6, no. 5 (1987): 780–83. http://dx.doi.org/10.1002/j.1536-4801.1987.tb09398.x.

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SummaryWe measured preprandial and 2‐h postprandial plasma triglycerides (TG) and free fatty acids (FFA) in 13 preterm infants weekly for 3 consecutive weeks to examine the effects of a premature, medium‐chain triglyceride (MCT)‐containing formula advancing to a term, long‐chain triglyceride (LCD‐containing formula. The infants were on premature formula for the first 2 weeks of the study and were switched to term formula 4–5 days before the last sampling. Significant increases were seen in preprandial and 2‐h postprandial TG when the infants were advanced from the premature to the term formula

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Wu, Ching-Lin, Tsung-Jen Yang, Min-Huan Wu, et al. "Walking Exercise Reduces Postprandial Lipemia but Does Not Influence Postprandial Hemorheological Properties and Oxidative Stress." Metabolites 12, no. 11 (2022): 1038. http://dx.doi.org/10.3390/metabo12111038.

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A higher postprandial triglycerides response and hemorheological abnormalities may increase the incidence of metabolic disorders and negatively interfere with the aging process. A single session of preprandial endurance exercise was found to be effective in reducing triglyceride levels after a high-fat diet. However, whether the exercise-induced reduction in postprandial triglyceride levels influences hemorheological indicators remains unknown. This study aims to investigate the effects of postprandial lipemia on hemorheological properties and oxidative stress. Eight healthy young male partici

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Ditschuneit, H. H., M. Flechtner-Mors, E. Hagel, and H. Ditschuneit. "Postprandial Lipoprotein Metabolism in Obese Patients with Moderate Hypertriglyceridaemia: Effects of Gemfibrozil." Journal of International Medical Research 20, no. 3 (1992): 197–210. http://dx.doi.org/10.1177/030006059202000301.

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After an oral fat load of 1 g/kg body weight in 10 obese females with hyperlipoproteinaemia type IV, serum triglycerides concentrations were maximal at 4 h with a slight decline at 6 h, whereas serum cholesterol concentrations rose slightly at 4 h and 6 h. After 6 h, concentrations of triglycerides and cholesterol were significantly increased in chylomicrons and very low-density lipoprotein (VLDL), whereas cholesterol concentrations were decreased in high-density lipoprotein 2 (HDL2) plus HDL3. After oral treatment with 450 mg gemfibrozil twice daily for 28 days, triglyceride concentrations we

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Eisenberg, S. "Triglycerides, postprandial lipemia and hemostatic factors." Atherosclerosis 109, no. 1-2 (1994): 91. http://dx.doi.org/10.1016/0021-9150(94)93378-2.

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Talal, Lillian A., Huipei Wang, Brian T. Williams, Matthew J. Morris, and Peter J. Horvath. "Acute Effects of White Button and Shiitake Mushroom Powder Supplementation on Postprandial Lipemia and Glycemia Following a High-Fat Meal." International Journal of Nutrition 7, no. 2 (2023): 42–56. http://dx.doi.org/10.14302/issn.2379-7835.ijn-23-4430.

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Background To determine the acute effects on postprandial lipemia and glycemia by supplementing a high-fat meal with either white button (WB) or shiitake (SH) mushroom powder. Methods Nine healthy participants (4-male, 5-female, 23.3±1.3 years, 17.8±6% body fat, 56.2±11.4kg fat free mass) consumed a control hamburger. At one-week intervals, after consumption of a control meal, participants consumed hamburgers in random order, supplemented with 14g of either WB or SH mushroom powder. Peripheral blood for lipids (triglycerides, high-density lipoprotein HDL, low-density lipoprotein LDL), and gluc

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Versteeg, Ruth I., Dirk J. Stenvers, Dana Visintainer, et al. "Acute Effects of Morning Light on Plasma Glucose and Triglycerides in Healthy Men and Men with Type 2 Diabetes." Journal of Biological Rhythms 32, no. 2 (2017): 130–42. http://dx.doi.org/10.1177/0748730417693480.

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Ambient light intensity is signaled directly to hypothalamic areas that regulate energy metabolism. Observational studies have shown associations between ambient light intensity and plasma glucose and lipid levels, but human data on the acute metabolic effects of light are scarce. Since light is the main signal indicating the onset of the diurnal phase of physical activity and food intake in humans, we hypothesized that bright light would affect glucose and lipid metabolism. Therefore, we determined the acute effects of bright light on plasma glucose and lipid concentrations in 2 randomized cr

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Pathak, R., Prabin Adhikari, and UN Pathak. "Role of fasting and postprandial hypertriglyceridemia and its association with carotid intima media thickness in Type II diabetes patients." Journal of Advances in Internal Medicine 7, no. 1 (2018): 17–22. http://dx.doi.org/10.3126/jaim.v7i1.19578.

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Background and aim: Type 2 diabetes mellitus is associated with the development of premature arteriosclerosis and a higher cardiovascular morbidity and mortality. Diabetic dyslipidaemia is believed to play an important role in the pathogenesis of accelerated atherosclerosis in this condition. B-mode ultrasound enables non-invasive, direct visualization of the arterial wall. The aim of this study was to correlate intima media thickness of the carotid artery (CIMT) quantification as a reliable marker of atherosclerotic burden in diabetes patients.Method: It was a Cross Sectional Study done on 50

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Keirns, Bryant H., Christina M. Sciarrillo, Kara L. Poindexter, and Sam R. Emerson. "Daily Triglyceride Kinetics When Consuming a Realistic Western Diet in at-Risk Individuals across the Metabolic Spectrum: A Case Study." Obesities 1, no. 2 (2021): 107–12. http://dx.doi.org/10.3390/obesities1020010.

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High postprandial triglycerides are a strong cardiovascular disease risk factor. However, the extent triglycerides rise during daily living due to multiple meals remains poorly defined, especially in at-risk individuals. The aim of this case study was to quantify 24-h triglycerides in an individual with normal-weight obesity (NWO), metabolic syndrome (MetS), and type 2 diabetes (T2D) consuming a Western-style diet. In the morning, an indwelling catheter was inserted into a forearm vein and metabolic markers were measured hourly for 18-h and every 3-h for the last 6-h. The participants with NWO

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O’Hara, Crystal, Babajide Ojo, Sam R. Emerson, et al. "Acute Freeze-Dried Mango Consumption With a High-Fat Meal has Minimal Effects on Postprandial Metabolism, Inflammation and Antioxidant Enzymes." Nutrition and Metabolic Insights 12 (January 2019): 117863881986994. http://dx.doi.org/10.1177/1178638819869946.

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Objective: Postprandial fluxes in oxidative stress, inflammation, glucose, and lipids, particularly after a high-fat meal (HFM), have been implicated in the development of cardiovascular disease (CVD). The aim of this study is to determine whether acute freeze-dried mango consumption modulates the postprandial response to an HFM. We hypothesized that the addition of mango, which is a rich source of many bioactive components, to an HFM would lower postprandial triglycerides, glucose, and inflammation, and increase antioxidant enzymes, compared to a standard HFM alone. Methods: In a randomized c

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Olsen, A. K., E. M. Bladbjerg, P. Marckmann, L. F. Larsen, and A. K. Hansen. "The Göttingen minipig as a model for postprandial hyperlipidaemia in man: experimental observations." Laboratory Animals 36, no. 4 (2002): 438–44. http://dx.doi.org/10.1258/002367702320389116.

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Postprandial hyperlipidaemia is believed to be atherogenic. This study aimed to establish a minipig model to investigate determinants of postprandial lipid metabolism. In a randomized cross-over design seven minipigs were subjected to six different feeding regimens: intragastric fat loads of 1, 2, and 4 g fat (Intralipid®, 20%) kg-1 in two fractions 1.5 h apart (1/3 first, 2/3 second), 2 g fat (Intralipid®) kg-1 in one fraction, and 2 g olive oil kg-1 in two fractions, all after pre-feeding with standard diet, and finally 2 g fat (Intralipid®) kg-1 in two fractions without pre-feeding. Blood w

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Van de Wiel, Albert. "The Effect of Alcohol on Postprandial and Fasting Triglycerides." International Journal of Vascular Medicine 2012 (2012): 1–4. http://dx.doi.org/10.1155/2012/862504.

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Alcohol has a significant additive effect on the postprandial triglyceride peak when it accompanies a meal containing fat, especially saturated fat. This results from a decrease in the breakdown of chylomicrons and VLDL remnants due to an acute inhibitory effect of alcohol on lipoprotein lipase activity. Furthermore, alcohol increases the synthesis of large VLDL particles in the liver, which is the main source of triglycerides in the hypertriglyceridemia associated with chronic excessive alcohol intake. In case of chronic consumption, lipoprotein lipase activity seems to adapt itself. The effe

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BARRETT, HELEN L., MARLOES NITERT DEKKER, SUSAN J. DE JERSEY, MICHAEL C. D'EMDEN, LEONIE CALLAWAY, and H. DAVID MCINTYRE. "Maternal Postprandial Triglycerides in the Home Setting." Diabetes 67, Supplement 1 (2018): 1422—P. http://dx.doi.org/10.2337/db18-1422-p.

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Matsuo, Tatsuhiro, Chihiro Yokoyama, Takako Yamada, et al. "Effect of Simultaneous Intake of Medium-Chain Triglyceride and d-Allulose on Body Fat Accumulation in Rats Fed a High-Fat Diet." CURRENT TOPICS IN NUTRACEUTICAL RESEARCH 21, no. 3 (2023): 242–47. http://dx.doi.org/10.37290/ctnr2641-452x.21:242-247.

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Medium-chain triglycerides, lipids containing three 6−12 carbon medium-chain fatty acids, have antiobesity effects because they do not promote lipogenesis. d-Allulose, a low-calorie epimer of fructose commercially used as a low-calorie sweetener, suppresses hepatic lipogenesis and enhances postprandial fat oxidation. Therefore, we have explored whether a simultaneous intake of medium-chain fatty acids and d-allulose may exhibit a greater reduction in de novo lipogenesis and increase their antiobesity effects. To this end, 32 male Wistar rats were divided into four treatment groups of equal siz

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KASAI, Michio, Hideaki MAKI, Naohisa NOSAKA, et al. "Effect of Medium-chain Triglycerides on the Postprandial Triglyceride Concentration in Healthy Men." Bioscience, Biotechnology, and Biochemistry 67, no. 1 (2003): 46–53. http://dx.doi.org/10.1271/bbb.67.46.

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Kim, In-Sook, Soo-yeon Park, Min Ju Park, Kyeong Jin Kim, and Ji Yeon Kim. "Effect of Barley on Postprandial Blood Glucose Response and Appetite in Healthy Individuals: A Randomized, Double-Blind, Placebo-Controlled Trial." Nutrients 16, no. 22 (2024): 3899. http://dx.doi.org/10.3390/nu16223899.

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Background/Objectives: Barley dietary fiber (BDF), particularly β-glucan, has shown potential in modulating postprandial glycemic responses and improving metabolic health. This study aimed to assess the effects of Saechalssalbori (Hordeum vulgare L.), a glutinous barley variety rich in β-glucan, on postprandial blood glucose, insulin, glucagon, triglycerides, and appetite-related hormones in healthy adults. Methods: In this randomized, double-blind, placebo-controlled, crossover trial, healthy adults (n = 67) with fasting blood glucose levels below 126 mg/dL were assigned to consume either BDF

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Li, Xuan, Hiroaki Yamada, Sayo Morita, et al. "Effects of Free Linoleic Acid and Oleic Acid in Sesame Meal Extract as Pancreatic Lipase Inhibitors on Postprandial Triglyceridemia: A Randomized, Double-Blind, Placebo-Controlled, Crossover Study in Healthy Volunteers." Nutrients 15, no. 7 (2023): 1748. http://dx.doi.org/10.3390/nu15071748.

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A great number of chemically diverse pancreatic lipase (PL) inhibitors have been identified to tackle obesity; however, very few of them have entered clinical studies. The ethanolic extract of sesame meal is a potent PL inhibitor, and its activity hinges exclusively on two free fatty acids: linoleic acid and oleic acid, which were proven to reduce postprandial triglyceride excursion in rats. Herein, to investigate the clinical efficacy of the sesame meal extract, in a crossover trial, 30 healthy volunteers were randomized to receive the sesame meal extract containing experimental food or place

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Shukla, Avikal. "Postprandial Lipid Abnormalities in Type 2 Diabetes Mellitus, A Study at NGMC, Kohalpur." Journal of Nepalgunj Medical College 13, no. 1 (2017): 21–24. http://dx.doi.org/10.3126/jngmc.v13i1.16407.

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Aim and Objectives: To study the postprandial lipid abnormalities in patients with type 2 diabetes mellitus.Material and Methods : Postprandial lipids were studied in 40 male type 2 diabetic subjects (age 49.75 ± 4.82 years) and 40 age and sex matched healthy controls (age 49.55 ± 4.82 years) after an oral fat challenge which consisted of a meal providing 729 kcal/m2 body surface area with 68 gm fat.Results: Average duration of diabetes among diabetic was 2.32 ± 3.03 years. The body mass index (cases 25.84 ± 4.52; controls 25.74 ± 5.0; p > 0.05) and waist-hip ratio (cases 1.06 ± 0.13; contr

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Louca, Panayiotis, Sarah E. Berry, Kate Bermingham, et al. "Postprandial Responses to a Standardised Meal in Hypertension: The Mediatory Role of Visceral Fat Mass." Nutrients 14, no. 21 (2022): 4499. http://dx.doi.org/10.3390/nu14214499.

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Postprandial insulinaemia, triglyceridaemia and measures of inflammation are thought to be more closely associated with cardiovascular risk than fasting measures. Although hypertension is associated with altered fasting metabolism, it is unknown as to what extent postprandial lipaemic and inflammatory metabolic responses differ between hypertensive and normotensive individuals. Linear models adjusting for age, sex, body mass index (BMI), visceral fat mass (VFM) and multiple testing (false discovery rate), were used to investigate whether hypertensive cases and normotensive controls had differe

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NOGAROTO, VIVIANE, MARCOS R. S. RODRIGUES, MARCELO R. VICARI, et al. "High Postprandial Triglycerides Serum Levels: Is Obesity a Good Predictor?" Anais da Academia Brasileira de Ciências 87, no. 1 (2015): 437–45. http://dx.doi.org/10.1590/0001-3765201520130380.

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The aim of this study was to analyze the correlation between triglyceride (TG) serum levels in obese and non-obese patients in a simulated postprandial state. Both groups showed TG levels < 150 mg/dL when fasting. After 12 h fasting, the subjects ingested a lipid overload diet and blood samples were collected. The variation between fasting and the postprandial TG peak levels were analyzed. The peak of postprandial TG levels occurred 4 h after the lipid overload in both groups. When the subjects were not fasting, the majority of non-obese subjects remained within the range of normal TG value

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Costabile, Giuseppina, Dominic Salamone, Giuseppe Della Pepa, et al. "Differential Effects of Two Isocaloric Healthy Diets on Postprandial Lipid Responses in Individuals with Type 2 Diabetes." Nutrients 16, no. 3 (2024): 333. http://dx.doi.org/10.3390/nu16030333.

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Background. High blood concentrations of triglycerides (TG) in the postprandial period have been shown to be more closely associated with the risk of cardiovascular disease (CVD) than fasting values in individuals with type 2 diabetes (T2D). Dietary changes are the primary determinants of postprandial lipid responses. Methods. We investigated the effects of an isocaloric multifactorial diet, rich in n-3 PUFA, MUFA, fiber, polyphenols, and vitamins, compared to an isocaloric diet, containing the same amount of MUFA, on the postprandial lipid response in T2D individuals. Following a randomized,

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Kurti, Stephanie P., Sara K. Rosenkranz, Morton Levitt, et al. "Does Moderate Intensity Exercise Attenuate the Postprandial Lipemic and Airway Inflammatory Response to a High-Fat Meal?" BioMed Research International 2015 (2015): 1–10. http://dx.doi.org/10.1155/2015/647952.

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We investigated whether an acute bout of moderate intensity exercise in the postprandial period attenuates the triglyceride and airway inflammatory response to a high-fat meal (HFM) compared to remaining inactive in the postprandial period. Seventeen (11 M/6 F) physically active (≥150 min/week of moderate-vigorous physical activity (MVPA)) subjects were randomly assigned to an exercise (EX; 60%VO2peak) or sedentary (CON) condition after a HFM (10 kcal/kg, 63% fat). Blood analytes and airway inflammation via exhaled nitric oxide (eNO) were measured at baseline, and 2 and 4 hours after HFM. Airw

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Lesser, Mary, Kasuen Mauldin, Lisa Sawrey-Kubicek, Virginia Gildengorin, and Janet King. "The Type of Dietary Fat in an Isocaloric Breakfast Meal Does Not Modify Postprandial Metabolism in Overweight/Obese Pregnant Women." Nutrients 11, no. 3 (2019): 490. http://dx.doi.org/10.3390/nu11030490.

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Almonds provide a satiating, healthy source of fat and fiber. The postprandial metabolic and satiety response to 2 ounces of nuts or dairy was assessed in 18 overweight/obese women during late pregnancy. Serum glucose, triglycerides, insulin, c-peptide, leptin, ghrelin, and lipoprotein particles were measured prior to and during a 5-h postprandial period following the consumption of an isocaloric breakfast meal with equivalent amounts of fat from either nuts or dairy on two separate mornings. Satiety was assessed by visual analogue scale (VAS) questionnaires and ad libitum food intake at the e

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Wang, Ying I., John Schulze, Nadine Raymond, et al. "Endothelial inflammation correlates with subject triglycerides and waist size after a high-fat meal." American Journal of Physiology-Heart and Circulatory Physiology 300, no. 3 (2011): H784—H791. http://dx.doi.org/10.1152/ajpheart.01036.2010.

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A rise in postprandial serum triglycerides (PP-sTG) can potentiate inflammatory responses in vascular endothelial cells (ECs) and thus serves as an independent risk factor for predicting increased cardiovascular morbidity. We examined postprandial triglyceride-rich lipoproteins (PP-TGRLs) in subjects ranging from normal to hypertriglyceridemic for their capacity to alter EC acute inflammatory responses. Cultured human aortic ECs (HAECs) were conditioned with PP-TGRLs isolated from human serum at the peak after a moderately high-fat meal. VLDL particle size increased postprandially and varied d

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Sofra, Xanya. "Balancing hormones improves Type 2 diabetes." Journal of Diabetes, Metabolic Disorders & Control 9, no. 1 (2022): 16–25. http://dx.doi.org/10.15406/jdmdc.2022.09.00232.

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Treating physicians have consistently recommended exercise to either prevent diabetes or de-escalate symptomatology. Diabetic complications render physical activity undesirable or unattainable. Diabetes has been often associated with hypothyroidism, weight gain, fatigue, accumulation of white adipose tissue, and inadequate supply of brown fat to generate energy. Accumulated toxicity results in hormonal imbalance, increased hunger, chronic pain, and wounds on extremities associated with diabetic neuropathy. Recent research with an effortless exercise method has demonstrated enhanced fitness and

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Poindexter, Kara, Nicholas Koemel, Madison Dixon, et al. "Determinants of Postprandial Triglycerides Across the Spectrum of Aging." Current Developments in Nutrition 5, Supplement_2 (2021): 44. http://dx.doi.org/10.1093/cdn/nzab033_044.

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Abstract Objectives Elevated postprandial triglycerides (TG) are a risk factor for cardiovascular disease, and older adults exhibit greater postprandial lipemia (PPL) compared to younger adults. However, determinants of PPL, especially in older populations, remain poorly defined. This cross-sectional study examined the influence of body composition, lifestyle behaviors, and metabolic risk factors for PPL across the aging spectrum. Methods We recruited individuals evenly distributed between the ages of 50–89 years (50% male; 50% female). Participants completed diet and physical activity questio

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de Morais Junior, Alcides C., Raquel M. Schincaglia, Marisa Passarelli, Gustavo D. Pimentel, and João F. Mota. "Acute Epigallocatechin-3-Gallate Supplementation Alters Postprandial Lipids after a Fast-Food Meal in Healthy Young Women: A Randomized, Double-Blind, Placebo-Controlled Crossover Study." Nutrients 12, no. 9 (2020): 2533. http://dx.doi.org/10.3390/nu12092533.

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A high-fat fast-food meal negatively impacts postprandial metabolism even in healthy young people. In experimental studies, epigallocatechin-3-gallate (EGCG), a bioactive compound present in green tea, has been described as a potent natural inhibitor of fatty acid synthase. Thus, we sought to evaluate the effects of acute EGCG supplementation on postprandial lipid profile, glucose, and insulin levels following a high-fat fast-food meal. Fourteen healthy young women 21 ± 1 years and body mass index 21.4 ± 0.41 kg/m2 were enrolled in a randomized, double-blind, placebo-controlled crossover study

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Poindexter, Kara, Christina Sciarrillo, Janice Hermann, and Sam Emerson. "Fasting and Postprandial Triglycerides Across Older Adulthood: A Cross-sectional Study." Current Developments in Nutrition 4, Supplement_2 (2020): 66. http://dx.doi.org/10.1093/cdn/nzaa040_066.

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Abstract Objectives Elevated postprandial triglycerides (TG) are a risk factor for cardiovascular disease (CVD). Although some evidence suggests that older adults exhibit greater postprandial TG than younger adults, it is unknown how postprandial lipid tolerance changes across the spectrum of older adulthood. This cross-sectional study examines postprandial TG responses across the spectrum of aging, as well as factors that may modify this response. Methods We are recruiting individuals into four age categories (age 50–59, 60–69, 70–79, 80–89 years), with an equal number of participants and sex

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Greenfield, Jerry R., Katherine Samaras, Chris S. Hayward, Donald J. Chisholm, and Lesley V. Campbell. "Beneficial Postprandial Effect of a Small Amount of Alcohol on Diabetes and Cardiovascular Risk Factors: Modification by Insulin Resistance." Journal of Clinical Endocrinology & Metabolism 90, no. 2 (2005): 661–72. http://dx.doi.org/10.1210/jc.2004-1511.

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Moderate alcohol consumption protects against type 2 diabetes and cardiovascular disease. Because humans spend most of their time in the postprandial state, we examined the effect of 15 g alcohol on postprandial metabolic factors in 20 postmenopausal women over 6 h. We measured 1) glucose, insulin, lipids, C-reactive protein, and adiponectin levels; 2) augmentation index by applanation tonometry; and 3) energy expenditure and substrate oxidation by indirect calorimetry. Subjects received low carbohydrate (LC; visits 1 and 2) and high carbohydrate (HC; visits 3 and 4) high fat meals with and wi

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Tucker, Wesley J., Brandon J. Sawyer, Catherine L. Jarrett, et al. "High-intensity interval exercise attenuates but does not eliminate endothelial dysfunction after a fast food meal." American Journal of Physiology-Heart and Circulatory Physiology 314, no. 2 (2018): H188—H194. http://dx.doi.org/10.1152/ajpheart.00384.2017.

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We investigated whether two different bouts of high-intensity interval exercise (HIIE) could attenuate postprandial endothelial dysfunction. Thirteen young (27 ± 1 yr), nonexercise-trained men underwent three randomized conditions: 1) four 4-min intervals at 85–95% of maximum heart rate separated by 3 min of active recovery (HIIE 4 × 4), 2) 16 1-min intervals at 85–95% of maximum heart rate separated by 1 min of active recovery (HIIE 16 × 1), and 3) sedentary control. HIIE was performed in the afternoon, ~18 h before the morning fast food meal (1,250 kcal, 63g of fat). Brachial artery flow-med

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Sanders, T. A. B., G. J. Miller, Tamara de Grassi, and Najat Yahia. "Postprandial Activation of Coagulant Factor VII by Long-chain Dietary Fatty Acids." Thrombosis and Haemostasis 76, no. 03 (1996): 369–71. http://dx.doi.org/10.1055/s-0038-1650585.

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SummaryFactor VII coagulant activity (FVIIc) is associated with an increased risk of fatal ischaemic heart disease (IHD). Several reports have suggested that dietary fat intake or hypertriglyceridaemia are associated with elevated levels of FVII. This study demonstrates that an intake of long-chain fatty acids sufficient to induce postprandial lipaemia in healthy subjects leads to a substantial elevation in both FVIIc and the concentration of FVII circulating in the activated form. Such an increase in FVIIc could not be induced by medium-chain triglycerides. These results suggest that the cons

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Keirns, Bryant H., Samantha M. Hart, Christina M. Sciarrillo, Kara L. Poindexter, Stephen L. Clarke, and Sam R. Emerson. "Postprandial Triglycerides, Flow-Mediated Dilation, and the Inflammatory Cytokine Milieu in Metabolically Healthy Obesity: A Cross-Sectional Pilot Study." Obesities 1, no. 1 (2021): 58–71. http://dx.doi.org/10.3390/obesities1010006.

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The cardiovascular disease (CVD) risk of metabolically healthy obesity (MHO) remains controversial. We sought to further characterize the CVD risk profile in MHO by evaluating postprandial triglycerides, vascular function, and systemic inflammatory markers. Control individuals that were normal-weight and metabolically healthy (Con), MHO, and metabolic syndrome (MetS) were recruited (n = 10–11/group). Each participant underwent an abbreviated fat tolerance test, fasting and postprandial flow-mediated dilation (FMD), and had a panel of inflammatory cytokines measured. MHO displayed postprandial

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Mena-Vázquez, Natalia, Rocío Redondo-Rodríguez, José Rioja, et al. "Postprandial Hyperlipidemia: Association with Inflammation and Subclinical Atherosclerosis in Patients with Rheumatoid Arthritis." Biomedicines 10, no. 1 (2022): 133. http://dx.doi.org/10.3390/biomedicines10010133.

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Objective: To describe postprandial lipidemia in patients with rheumatoid arthritis (RA) and to analyze its association with subclinical atherosclerosis and inflammatory activity. Methods: Observational study of 80 cases of RA and 80 sex- and age-matched controls. We excluded individuals with dyslipidemia. Postprandial hyperlipidemia (PPHL) was defined as postprandial triglycerides >220 mg/dL and/or postprandial ApoB48 levels >75th percentile (>p75). Plasma lipids, cholesterol, triglycerides, ApoB48, and total ApoB were evaluated at baseline and after a meal. Other variables analyzed

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Mirghani, Hyder Osman. "Effect of dates on blood glucose and lipid profile among patients with type 2 diabetes." World Journal of Diabetes 15, no. 6 (2024): 1079–85. http://dx.doi.org/10.4239/wjd.v15.i6.1079.

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Poor fruit and vegetable consumption is one of the 10 major risk factors for mortality. There is a misconception regarding the consumption of dates among patients with diabetes. This manuscript assessed the effects of date consumption on fasting and postprandial blood glucose, glycated hemoglobin, total cholesterol, triglycerides, low-density lipoproteins, high-density lipoproteins, and microbial markers. Four literature databases were searched for relevant articles. Of the 595 studies retrieved, 24 assessed the effects of dates on glycemic control and lipids. Overall, the evidence suggests th

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