Relevant bibliographies by topics / Potentiel de plaque

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  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Potentiel de plaque'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Potentiel de plaque"

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Tiengo, Cesare, Jenny Deluca, Anna Belloni-Fortina, Roberto Salmaso, Flavia Galifi, and Mauro Alaibac. "Occurrence of Squamous Cell Carcinoma in an Area of Lichen Simplex Chronicus: Case Report and Pathogenetic Hypothesis." Journal of Cutaneous Medicine and Surgery 16, no. 5 (September 2012): 350–52. http://dx.doi.org/10.1177/120347541201600513.

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Background: Lichen simplex chronicus is a common skin disorder characterized by circumscribed, lichenified, pruritic plaque secondary to local repetitive trauma, notably rubbing and scratching. Objective: We describe a case of a squamous cell carcinoma arising in a patient with a long-lasting history of lichen simplex chronicus and discuss the potential role of the microenvironment in predisposing the malignant transformation. Conclusion: Here we propose a hypothesis in which rubbing and scratching contribute to an excess of inflammatory mediators, which in turn may lead to alterations in the processes of keratinocyte proliferation and differentiation. Renseignements de base: Le lichen simplex chronique est une affection cutanée courante caractérisée par une plaque pruritique lichénifiée circonscrite consécutive à des traumatismes locaux répétés, notamment le frottement et le grattage. Objectif: Nous décrivons un cas de carcinome squameux chez un patient présentant depuis longtemps des antécédents de lichen simplex chronique, et nous discutons du rôle potentiel du micro-environnement dans la prédisposition de la transformation maligne. Conclusion: Nous proposons ici une hypothèse selon laquelle le frottement et le grattage contribuent à un excès de médiateurs inflammatoires, lesquels peuvent à leur tour venir modifier les processus de prolifération et de différenciation des kératinocytes.
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Troquet, J., and Ph Lambin. "Répartition du potentiel électrocinétique imposée par un dipôle de courant excentré dans une plaque conductrice rectangulaire partiellement cloisonnée." Revue de Physique Appliquée 20, no. 6 (1985): 423–35. http://dx.doi.org/10.1051/rphysap:01985002006042300.

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Li, Hongbo, Hui Huang, Yun Luan, Niu Liu, Hui Gao, and Huijuan Sun. "Contrast-Enhanced Ultrasonography Characteristics in the Evaluation of Different Nature of Carotid Artery Plaques." Journal of Medical Imaging and Health Informatics 10, no. 3 (March 1, 2020): 688–92. http://dx.doi.org/10.1166/jmihi.2020.2919.

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To investigate the value of contrast-enhanced ultrasonography (CEUS) in the evaluation of neovascular density in plaques with different carotid atherosclerosis. 97 patients with carotid plaques were observed by conventional ultrasound from January 2016 to December 2018. The CEUS test was used to grade and quantify the new blood vessels in the plaque. The results showed of the 97 patients with 101 plaques, 49 were soft plaques, 45 were mixed plaques, and 7 were hard plaques. CEUS clearly showed the continuity of plaque fibrous caps and found plaque ulcers that were easily missed by conventional ultrasound. The lower the echo of the plaque, the more obvious contrast enhancement in the plaques. Quantitative analysis of CEUS showed the peak time of soft plaques was shorter and the peak intensity of soft plaques was higher compared with mixed plaques. The difference was statistically significant (P <0.01). The results displayed that carotid CEUS is a convenient and intuitive new technology that can better help analyze plaque morphology and determine and quantify the extent of neovascularization in plaque. It can be used for clinical evaluation of potential cerebrovascular risk and also provides an accurate and reproducible detection method for clinical efficacy evaluation.
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Hafiane, Anouar. "Vulnerable Plaque, Characteristics, Detection, and Potential Therapies." Journal of Cardiovascular Development and Disease 6, no. 3 (July 27, 2019): 26. http://dx.doi.org/10.3390/jcdd6030026.

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Plaque development and rupture are hallmarks of atherosclerotic vascular disease. Despite current therapeutic developments, there is an unmet necessity in the prevention of atherosclerotic vascular disease. It remains a challenge to determine at an early stage if atherosclerotic plaque will become unstable and vulnerable. The arrival of molecular imaging is receiving more attention, considering it allows for a better understanding of the biology of human plaque and vulnerabilities. Various plaque therapies with common goals have been tested in high-risk patients with cardiovascular disease. In this work, the process of plaque instability, along with current technologies for sensing and predicting high-risk plaques, is debated. Updates on potential novel therapeutic approaches are also summarized.
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Kovanen, P. T. "Mast cells and degradation of pericellular and extracellular matrices: potential contributions to erosion, rupture and intraplaque haemorrhage of atherosclerotic plaques." Biochemical Society Transactions 35, no. 5 (October 25, 2007): 857–61. http://dx.doi.org/10.1042/bst0350857.

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Mast cells are present in advanced human atherosclerotic plaques, where they are thought to exert multiple effects on their neighbouring cells and on the extracellular matrix of the plaque. Extensive efforts at delineating their role(s) in atherosclerotic plaques have unravelled mechanisms by which plaque mast cells may render advanced atherosclerotic plaques susceptible to erosion, rupture or intraplaque haemorrhage and so modulate their stability. In these mechanisms, the key effector molecules are mast-cell-derived neutral proteases and pro-inflammatory cytokines. These effector molecules are synthesized and stored in the cytoplasmic secretory granules of mast cells and, once the mast cells are activated to degranulate, are released into the microenvironment surrounding the activated mast cells. In the plaques, the key target cells are endothelial cells and smooth muscle cells and their pericellular matrices. In addition, the various components of the extracellular matrix of the plaques, notably collagen, are degraded when the released mast cell proteases activate matrix metalloproteinases in the plaques. By rendering the plaque susceptible to erosion, to rupture or to intraplaque haemorrhage, the mast cells may contribute to the onset of acute atherothrombotic complications of coronary atherosclerosis, such as myocardial infarction.
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Tang, Dalin, Chun Yang, Jie Zheng, Pamela K. Woodard, Jeffrey E. Saffitz, Gregorio A. Sicard, Thomas K. Pilgram, and Chun Yuan. "Quantifying Effects of Plaque Structure and Material Properties on Stress Distributions in Human Atherosclerotic Plaques Using 3D FSI Models." Journal of Biomechanical Engineering 127, no. 7 (July 29, 2005): 1185–94. http://dx.doi.org/10.1115/1.2073668.

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Background: Atherosclerotic plaques may rupture without warning and cause acute cardiovascular syndromes such as heart attack and stroke. Methods to assess plaque vulnerability noninvasively and predict possible plaque rupture are urgently needed. Method: MRI-based three-dimensional unsteady models for human atherosclerotic plaques with multi-component plaque structure and fluid-structure interactions are introduced to perform mechanical analysis for human atherosclerotic plaques. Results: Stress variations on critical sites such as a thin cap in the plaque can be 300% higher than that at other normal sites. Large calcification block considerably changes stress/strain distributions. Stiffness variations of plaque components (50% reduction or 100% increase) may affect maximal stress values by 20–50 %. Plaque cap erosion causes almost no change on maximal stress level at the cap, but leads to 50% increase in maximal strain value. Conclusions: Effects caused by atherosclerotic plaque structure, cap thickness and erosion, material properties, and pulsating pressure conditions on stress/strain distributions in the plaque are quantified by extensive computational case studies and parameter evaluations. Computational mechanical analysis has good potential to improve accuracy of plaque vulnerability assessment.
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Teng, Zhongzhao, Aart J. Nederveen, Aad Van der Lugt, Jonathan H. Gillard, and Lambert Speelman. "MRI-based biomechanical parameters for carotid artery plaque vulnerability assessment." Thrombosis and Haemostasis 115, no. 03 (2016): 493–500. http://dx.doi.org/10.1160/th15-09-0712.

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SummaryCarotid atherosclerotic plaques are a major cause of ischaemic stroke. The biomechanical environment to which the arterial wall and plaque is subjected to plays an important role in the initiation, progression and rupture of carotid plaques. MRI is frequently used to characterize the morphology of a carotid plaque, but new developments in MRI enable more functional assessment of carotid plaques. In this review, MRI based biomechanical parameters are evaluated on their current status, clinical applicability, and future developments. Blood flow related biomechanical parameters, including endothelial wall shear stress and oscillatory shear index, have been shown to be related to plaque formation. Deriving these parameters directly from MRI flow measurements is feasible and has great potential for future carotid plaque development prediction. Blood pressure induced stresses in a plaque may exceed the tissue strength, potentially leading to plaque rupture. Multi-contrast MRI based stress calculations in combination with tissue strength assessment based on MRI inflammation imaging may provide a plaque stress-strength balance that can be used to assess the plaque rupture risk potential. Direct plaque strain analysis based on dynamic MRI is already able to identify local plaque displacement during the cardiac cycle. However, clinical evidence linking MRI strain to plaque vulnerability is still lacking. MRI based biomechanical parameters may lead to improved assessment of carotid plaque development and rupture risk. However, better MRI systems and faster sequences are required to improve the spatial and temporal resolution, as well as increase the image contrast and signal-to-noise ratio.
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Biasi, Giorgio M., Paolo M. Mingazzini, Lucia Baronio, Maria Rosa Piglionica, Stefano A. Ferrari, Tarek S. Elatrozy, and Andrew N. Nicolaides. "Carotid Plaque Characterization Using Digital Image Processing and its Potential in Future Studies of Carotid Endarterectomy and Angioplasty." Journal of Endovascular Therapy 5, no. 3 (August 1998): 240–46. http://dx.doi.org/10.1177/152660289800500309.

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Purpose: To corroborate the validity of a computerized methodology for evaluating carotid lesions at risk for stroke based on plaque echogenicity. Methods: The records of 96 carotid endarterectomy patients (59 men; median age 69.5 years, range 52 to 83) with stenoses > 50% were studied retrospectively. Forty-one patients (43%) had been symptomatic preoperatively. All patients had undergone computed tomography (CT) to detect infarction in the carotid territory and a duplex scan to measure carotid stenosis. Plaque echogenicity was analyzed by computer, expressing the echodensity in terms of the gray scale median (GSM). The incidence of CT-documented cerebral infarction was analyzed in relation to symptomatology, percent stenosis, and echodensity. Results: Symptoms correlated well with CT evidence of brain infarction: 32% of symptomatic patients had a positive CT scan versus 16% for asymptomatic plaques (p = 0.076). The mean GSM value was 56 ± 14 for plaques associated with negative CT scans and 38 ± 13 for plaques from patients with positive scans (p < 0.0001). However, there was no difference in the GSM value between plaques with > or < 70% stenosis. Furthermore, the incidence of CT infarction was 40% in the cerebral territory of carotid plaques with a GSM value < 50 and only 9% in those with a GSM > 50 (p < 0.001). Conclusions: Computerized analysis of plaque echogenicity appears to provide clinically useful data that correlates with the incidence of cerebral infarction and symptoms. This method of analyzing plaque echolucency could be used as a screening tool for carotid stent studies to identify high-risk lesions better suited to conventional surgical treatment.
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Ma, Wei, Xinyao Cheng, Xiangyang Xu, Furong Wang, Ran Zhou, Aaron Fenster, and Mingyue Ding. "Multilevel Strip Pooling-Based Convolutional Neural Network for the Classification of Carotid Plaque Echogenicity." Computational and Mathematical Methods in Medicine 2021 (August 18, 2021): 1–13. http://dx.doi.org/10.1155/2021/3425893.

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Carotid plaque echogenicity in ultrasound images has been found to be closely correlated with the risk of stroke in atherosclerotic patients. The automatic and accurate classification of carotid plaque echogenicity is of great significance for clinically estimating the stability of carotid plaques and predicting cardiovascular events. Existing convolutional neural networks (CNNs) can provide an automatic carotid plaque echogenicity classification; however, they require a fixed-size input image, while the carotid plaques are of varying sizes. Although cropping and scaling the input carotid plaque images is promising, it will cause content loss or distortion and hence reduce the classification accuracy. In this study, we redesign the spatial pyramid pooling (SPP) and propose multilevel strip pooling (MSP) for the automatic and accurate classification of carotid plaque echogenicity in the longitudinal section. The proposed MSP module can accept arbitrarily sized carotid plaques as input and capture a long-range informative context to improve the accuracy of classification. In our experiments, we implement an MSP-based CNN by using the visual geometry group (VGG) network as the backbone. A total of 1463 carotid plaques (335 echo-rich plaques, 405 intermediate plaques, and 723 echolucent plaques) were collected from Zhongnan Hospital of Wuhan University. The 5-fold cross-validation results show that the proposed MSP-based VGGNet achieves a sensitivity of 92.1%, specificity of 95.6%, accuracy of 92.1%, and F1-score of 92.1%. These results demonstrate that our approach provides a way to enhance the applicability of CNN by enabling the acceptance of arbitrary input sizes and improving the classification accuracy of carotid plaque echogenicity, which has a great potential for an efficient and objective risk assessment of carotid plaques in the clinic.
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Liu, Wu, Jenna May Kim, Benjamin K. Young, Ravinder Nath, Zhe Chen, Roy H. Decker, Melvin A. Astrahan, and Renelle Pointdujour-Lim. "Novel Eye Plaque Designs for Brachytherapy of Iris and Ciliary Body Melanoma and the First Clinical Application." Ocular Oncology and Pathology 5, no. 3 (October 11, 2018): 220–28. http://dx.doi.org/10.1159/000493269.

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Background: While traditional eye plaque brachytherapy can be used for the treatment of iris melanoma, it faces challenges of poor patient tolerability due to cornea-plaque touch caused by radius of curvature mismatch and potential dosimetric inaccuracy from incomplete coverage. We present novel plaque designs and the first clinical application of the plaques for iris melanoma. Methods: Two dome-shaped plaques (EP2132 and EP1930) were designed to vault above the cornea to treat tumors of the iris and ciliary body. Image-based treatment planning of the first 2 clinical cases using the EP2132 plaque covered the tumor base plus a 2 mm margin and the involved ciliary body with at least 75 Gy to the tumor apex. Results: The tumors decreased in size following treatment. The patients tolerated the treatment well. There was no adverse event associated with the traditional iris plaques, such as decreased vision, pain, corneal edema, glaucoma, or cataract. Conclusion: The novel dome-shaped plaques for the treatment of iris melanoma provide effective dose distribution, improved surgical maneuverability, and increased tolerability for the patient. This plaque model can be used to treat iris melanoma of various sizes, configurations, and locations, including the ciliary body. The need for a customized plaque platform for each patient is minimized.
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Dissertations / Theses on the topic "Potentiel de plaque"

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Chasseraud, Maud. "Rôle potentiel du TRAIL dans le processus de calcification vasculaire et dans la déstabilisation de la plaque d'athérosclérose." Amiens, 2009. http://www.theses.fr/2009AMIED007.

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Les maladies cardiovasculaires, constituent la première cause de mortalité et de morbidité en France. L’athérosclérose, caractérisée par la formation au niveau de l’intima des artères de moyens et gros calibres de plaques de nature lipidique, est une composante-clé de ces maladies. Dans le développement de la pathologie, deux événements majeurs signes de complications peuvent survenir, la déstabilisation de la plaque qui peut conduire à une rupture de celle-ci, et l’apparition au niveau des plaques, de calcifications entraînant une rigidité vasculaire. Pour améliorer notre connaissance des mécanismes physiopathologiques à l’origine de ces complications, nous avons évalué l’implication potentielle du TRAIL (TNF-Related Apoptosis Inducing Ligand) et de l’OPG (ostéoprotégérine) dans l’athérosclérose et dans les processus de calcifications vasculaires. Dans un modèle de souris ApoE-/- rendues insuffisantes rénales chroniques par néphrectomie, nous avons montré grâce à la réalisation d’immuno-marquages sur des coupes de valves aortiques que l’IRC s’accompagnait d’une augmentation de l’expression au niveau artériel du TRAIL et de l’OPG. In vitro, nous avons évalué l’effet du TRAIL sur la capacité de minéraliser des cellules musculaires lisses et montré à l’aide de plusieurs modèles cellulaires que le TRAIL était un inducteur de la minéralisation. Ces données expérimentales obtenues dans un modèle animal et cellulaire, ont été prolongées par des études cliniques au cours desquelles nous avons mesuré les concentrations sériques du TRAIL et de l’OPG d’une part chez des patients insuffisants rénaux chroniques (IRC) et d’autre part chez des patients avec un syndrome coronarien aigu (SCA). Chez les patients IRC, en particulier chez des patients hémodialysés avec une IRC sévère, nous montrons une diminution significative des concentrations sériques de TRAIL qui s’accompagne d’une augmentation de l’OPG. A l’inverse de l’OPG, Les taux de TRAIL ne sont pas corrélés avec le score de calcification mesurés chez les patients hémodialysés. La diminution du TRAIL et l’augmentation de l’OPG sérique sont retrouvées chez les patients avec un SCA. Dans les deux populations, une corrélation inverse est mise en évidence entre les concentrations sériques de TRAIL et de CRP. Enfin, chez les patients présentant un SCA une corrélation inverse a été trouvée avec la MMP-9. En conclusion, l’ensemble de ces données mettent en évidence l’implication du couple OPG/TRAIL dans la physiopathologie de l’athérosclérose et des calcifications vasculaires
Cardiovascular disease constitutes the primary cause of morbidity and mortality in France. Atherosclerosis is a key contributor to cardiovascular disease. During the development of atherosclerosis, two major events signify that complications may arise, first of which is the destabilization of the atherosclerotic plaque, leading to plaque disruption and secondly, vascular calcifications leading to vascular rigidity. In order to improve our knowledge of molecular mechanism involved in both atherosclerosis and vascular calcification, we studied the potential implication of TRAIL and OPG. Through immuno-histochemical analysis of aortic valves taken from ApoE-/- mice, a model of chronic kidney disease (CKD), we were able to show that the occurrence of CKD correlates with an increase in the expression of TRAIL and OPG at the arterial level. In vitro, we alsio demonstrated that TRAIL dramatically increase the capacity of smooth muscle cells to mineralize. To extend these experimental observations, we carried out two clinical studies in which we have measured the concentrations of TRAIL and OPG in the serum of patients on haemodialysis presenting with calcifications and in patients presenting with an acute coronary syndrome. Our results clearly demonstrated in both populations studied, a significant decrease of seric TRAIL level, while OPG level were notably increased. In patients on haemodialysis, by contrast to OPG, TRAIL did not correlate with calcification score. A significant inverse relationship was found between TRAIL and CRP in both populations. Moreover, in patients with an acute coronary syndrome, we also demonstrated an inverse relationship between TRAIL and MMP-9 levels. Taken together, our data clearly suggest that TRAIL and OPG are involved in pathophysiological mechanism of atherosclerosis and vascular calcification
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Boudier, Jean-Louis. "Localisation des canaux sodium potentiel-dépendants à la jonction neuromusculaire par radioautographie des sites recepteurs aux toxines de scorpion." Aix-Marseille 2, 1991. http://www.theses.fr/1991AIX20652.

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Musarella, Magali. "Physiopathologie de la jonction neuromusculaire : étude de la souris med(motor endplate disease), un mutant pour le canal sodium potentiel-dépendant de type Nav1.6." Aix-Marseille 2, 2008. http://www.theses.fr/2008AIX20672.

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Les souris med (motor endplate disease) sont des mutants dépourvus du canal sodium potentiel-dépendant de type Nav1. 6 qui est le type majoritaire exprimé aux nœuds de Ranvier (NdR) des axones myélinisés du SNC et du SNP. Les mutants med présentent une paralysie musculaire progressive à partir du 10ème jour de vie postnatale (P10) aboutissant à la mort à la fin de la 3ème semaine. La conduction axonale le long des nerfs périphériques est normale, cependant une stimulation de ces derniers est incapable d’induire une contraction musculaire. A la jonction neuromusculaire (JNM), l’exocytose des neurotransmetteurs n’est pas affectée en elle-même tout comme la capacité des fibres musculaires à se contracter sous l’action d’une stimulation directe. Ainsi, l’origine du phénotype med correspond à un défaut de neurotransmission sans que les acteurs cellulaires et/ou moléculaires impliqués aient été identifiés. Le premier objectif de ces travaux de thèse a donc été de déterminer quel(s) partenaire(s) cellulaire(s) et/ou moléculaire(s) de la JNM sont à l’origine de ce défaut de neurotransmission. L’analyse des JNM par des approches associant immunocytochimie sur muscles entiers, microscopie confocale avec reconstruction 3D et quantification, a permis d’obtenir les résultats suivants : 1) Aux NdR des nerfs périphériques le canal sodium de type Nav1. 2 persiste jusqu’au stade terminal de la pathologie, alors qu’il n’est normalement plus exprimé à partir de P7, se substituant ainsi à Nav1. 6 et permettant une conduction axonale normale. 2) Le phénotype med ne peut être expliqué par une dénervation physique (à P19, 78% de JNM sont innervées chez la souris med, contre 73% chez la souris sauvage) malgré des altérations touchant à la fois les éléments pré- et postsynaptiques. Les vésicules synaptiques sont redistribuées/accumulées dans les terminaisons présynaptiques dès P10, puis au stade terminal de la pathologie, les axones préterminaux bourgeonnent et la taille des plaques motrices est réduite. 3) Parmi les altérations des JNM, le fait le plus remarquable est l’absence de partenaire glial pour un nombre important de synapses, allant de 1/3 à 2/3 des JNM dépourvues de cellules de Schwann terminales (TSC) suivant le type de muscle étudié. Les TSC sont essentielles au développement des JNM, à leur maintien et à leur réparation après lésion. Des données plus récentes montrent que ces cellules gliales modulent la neurotransmission et ont conduit à réconsidérer cette synapse périphérique comme d’ailleurs les synapses centrales non plus comme des systèmes à deux mais à trois partenaires. Le deuxième objectif a donc été de réévaluer le rôle des TSC dans le phénotype med. 1) L’absence de TSC d’un nombre important de JNM est observable dès le début du phénotype (P10) et précède la survenue des altérations pré- et postsynaptiques. Ce déficit en TSC ne peut être expliqué par un mécanisme d’apoptose (détectée par la methode TUNEL) accrue et dependrait plus d’un défaut de prolifération et/ou de migration. 2) Nous avons mis en évidence par immunocytochimie et hybridation in situ qu’à la JNM de souris sauvage, les TSC sont le seul partenaire qui exprime le canal sodium potentiel-dépendant de type Nav1. 6 et que l’arborisation terminale en est dépourvue. Afin de déterminer si ces canaux sont exprimés à la membrane des TSC et fonctionnels, nous avons enregistré les courants potentiel-dépendants en patch-clamp de ces cellules in situ. 3) Enfin nous avons montré que c’est entre P10 et P13, l’âge correspondant à l’apparition du phénotype med, que les TSC de souris sauvages commencent à répondre à la libération de neurotransmetteurs (obtenue par stimulation à haute fréquence du nerf afférent ou par application locale d’acétylcholine ou d’ATP) par une élévation du taux de calcium
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Pugh, Karen. "Protease-activated receptor-1 as potential target in atherosclerotic plaque stabilisation." Thesis, University of Strathclyde, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431780.

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Thomas, Didier. "Intérêt des potentiels évoqués auditifs de latence intermédiaire dans le diagnostic de la sclérose en plaques." Bordeaux 2, 1994. http://www.theses.fr/1994BOR2M034.

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Bicknell, Colin David. "Embolic potential of human carotid atherosclerotic plaques during endoluminal intervention." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422309.

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Salle, Jean-Yves. "Etude des potentiels evoques moteurs au cours des poussees de sclerose en plaques traitees par la methylprednisolone." Limoges, 1989. http://www.theses.fr/1989LIMO0176.

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Schiro, Andrew. "The use of microparticles and inflammatory cytokines as potential biomarkers for plaque instability in patients with carotid disease." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/the-use-of-microparticles-and-inflammatory-cytokines-as-potential-biomarkers-for-plaque-instability-in-patients-with-carotid-disease(95ea501f-60d4-442b-b6c8-02e705266dd0).html.

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Aim: Endothelial microparticles (EMPs) are released from dysfunctional endothelial cells. We hypothesised that patients with unstable carotid plaque have higher levels of circulating microparticles compared to patients with stable plaques, and this may correlate with serum markers of plaque instability and inflammation. Method: Circulating EMPs and inflammatory markers were measured in twenty healthy controls and seventy patients undergoing carotid endarterectomy. EMPs were quantified using flow cytometry. Bioplex assays profiled systemic inflammatory and bone-related proteins. Immunohistological analysis detailed the contribution of differentially-regulated systemic markers to plaque pathology. Alizarin red staining showed calcification. Results: EMPs were significantly higher in patients with carotid stenosis (greater than or equal to 70%) compared to controls, with no differences between asymptomatic vs symptomatic patients. Asymptomatic patients with unstable plaques exhibited higher levels of EMPs compared to those with stable plaques, with a similar trend observed in symptomatic patients. CXCL9 and SCGF-β were significantly elevated in asymptomatic patients with unstable plaques, with IL-16 and macrophage inhibitory factor significantly elevated in the stable plaque group. CXCL9, CTACK and SCGF-β were detected within all plaques, suggesting a contribution to both localised and systemic inflammation. Osteopontin and osteoprotegerin were significantly elevated in the symptomatic vs asymptomatic group, while osteocalcin was higher in asymptomatic patients with stable plaque. All plaques exhibited calcification, which was significantly greater in asymptomatic patients. This may impact on plaque stability. Conclusions: Circulatory EMP, CXCL9 and SCGF-β levels are raised in asymp-tomatic patients with unstable plaques, which could be important in identifying patients at most benefit from intervention.
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Petit, Jérôme. "Irm et sclerose en plaques : interet dans le diagnostic positif, comparaison avec les autres examens complementaires ; etude retrospective de 146 cas." Nancy 1, 1994. http://www.theses.fr/1994NAN11165.

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Rico-Lamy, Audrey. "Caractérisation fonctionnelle du système moteur au stade précoce de la sclérose en plaques : approche par stimulation magnétique transcrânienne et imagerie fonctionnelle par résonnance magnétique." Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX20724.

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La sclérose en plaques est la maladie non tumorale du système nerveux la plus fréquente du sujet jeune. Elle est définie comme une atteinte démyélinisante inflammatoire multifocale et chronique de la substance blanche du système nerveux central d’origine dysimmunitaire. Le handicap moteur est l’un des risques évolutifs principaux de cette maladie. Mieux caractériser les mécanismes du dysfonctionnement du système moteur (atteinte de la voie motrice centrale efférente, altération du réseau moteur cortical à l’origine du mouvement) de même que les mécanismes compensatoires qui le limitent est un enjeu important dans la compréhension et la prévention de l’apparition du handicap. L’étude de ces mécanismes au stade précoce de la maladie permet de mieux comprendre les liens entre l’atteinte tissulaire, les mécanismes de compensation et les conséquences fonctionnelles. Nos travaux ont permis au stade précoce de la SEP de mieux caractériser l’atteinte fonctionnelle de la voie motrice centrale, de montrer l’influence de l’atteinte encéphalique diffuse sur la réorganisation corticale du système moteur et enfin de démontrer l’existence d’une plasticité cérébrale fonctionnelle basale du réseau moteur non dominant corrélée avec les changements d’activation observés lors du mouvement au sein du même réseau
Multiple sclerosis (MS) is a chronic inflammatory demyelinating disorder of the central nervous system which is frequently responsible for motor disability. Better assessment of pathophysiological process implicated in motor system dysfunction (cortico-spinal tract or more diffuse tissue damage) as well as the compensatory mechanises are critical in the understanding and the prevention of disability. Their study in the early stage of the disease will allow to a better understanding of the links between tissue injury, compensatory mechanisms and functional consequences. We have better characterized functional dysfunction of the central motor pathway at the early stage of multiple sclerosis. We have also evidenced the influence of diffuse brain injury on functional motor cortical reorganization. Last, we have demonstrated the existence of a basal functional plasticity at rest of the nondominant motor network that is correlated with its functional plasticity during action
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Books on the topic "Potentiel de plaque"

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Arabian plate hydrocarbon geology and potential: A plate tectonic approach. Tulsa, Okla., U.S.A: American Association of Petroleum Geologists, 1991.

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Volozh, I︠U︡ A. Orenburgskiĭ tektonicheskiĭ uzel: Geologicheskoe stroenie i neftegazonosnostʹ = Orenburg tectonic junction : geological structure and petroleum potential. Moskva: Nauchnyĭ mir, 2013.

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Office, General Accounting. Defense acquisitions: Antiarmor Munitions Master Plan does not identify potential excesses or support planned procurements : report to the Chairman, Subcommittee on Defense, Committee on Appropriations, House of Representatives. Washington, D.C. (P.O. Box 37050, Washington, D.C. 20013): The Office, 2000.

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Oldstone, Michael B. A. Viruses, Plagues, and History. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190056780.001.0001.

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“Viruses, Plagues, & History” focuses on the effects of viral diseases on human history. Written by an eminent internationally respected virologist, it couples the fabric of history with major concepts developed in virology, immunology, vaccination, and accounts by people who first had, saw and acted at the times these events occurred. Much of the preventive and therapeutic progress (vaccines, antiviral drugs) has been made in the last 60 years. Many of those who played commanding roles in the fight to understand, control and eradicate viruses and viral diseases are (were) personally known to the author and several episodes described in this book reflect their input. The book records the amazing accomplishments that led to the control of lethal and disabling viral diseases caused by Smallpox, Yellow Fever, Measles, Polio, Hepatitis A, B and C, and HIV. These six success stories are contrasted with viral infections currently out of control—COVID-19, Ebola virus, Lassa Fever virus, Hantavirus, West Nile virus, and Zika. Influenza, under reasonable containment at present, but with the potential to revert to a world-wide pandemic similar to 1918–1919 where over 50 million people were killed. The new platforms to develop inhibitory and prophylactic vaccines to limit these and other viral diseases is contrasted to the anti-vaccine movement and the false prophets of autism.
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Renata, Dmowska, and Ekström Göran, eds. Shallow subduction zones: Seismicity, mechanics, and seismic potential. Basel: Birkhäuser Verlag, 1993.

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DMOWSKA and ECKSTRÖM. Shallow Subduction Zones: Seismicity, Mechanics and Seismic Potential Part 1 (Pageoph Topical Volumes). Birkhauser, 1993.

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Saraiya, Ami, Deep Joshipura, and Alice Gottlieb. Psoriasis treatment. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198737582.003.0026.

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Psoriasis is an immune-mediated skin disease that is associated with various factors, including genetics, stress, infections, and environmental triggers. Numerous treatment options exist for plaque psoriasis including topical therapy, phototherapy, systemic therapy, and biological therapy. In order to select a treatment for a patient, a clinician must consider many aspects. First, one must assess the impact and burden of the disease on a patient as well as a patient’s expectations from therapy. Other important factors to consider include the severity of skin disease, location of psoriatic plaques, comorbidities and presence of psoriatic arthritis, efficacy of different treatments, potential side-effects, safety, and cost. In this chapter, an evidence-based review is presented on the treatment armamentarium for psoriasis as well as new biological treatments and those under investigation. In order to guide practitioners, several treatment algorithms are provided and others are referenced from the literature.
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Veech, Richard L., and M. Todd King. Alzheimer’s Disease. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0026.

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Deficits in cerebral glucose utilization in Alzheimer’s disease (AD) arise decades before cognitive impairment and accumulation of amyloid plaques and neurofibrillary tangles in brain. Addressing this metabolic deficit has greater potential in treating AD than targeting later disease processes – an approach that has failed consistently in the clinic. Cerebral glucose utilization requires numerous enzymes, many of which have been shown to decline in AD. Perhaps the most important is pyruvate dehydrogenase (PDH), which links glycolysis with the Krebs cycle and aerobic metabolism, and whose activity is greatly suppressed in AD. The unique metabolism of ketone bodies allows them to bypass the block at pyruvate dehydrogenase and restore brain metabolism. Recent studies in mouse genetic models of AD and in a human Alzheimer’s patient showed the potential of ketones in maintaining brain energetics and function. Oral ketone bodies might be a promising avenue for treatment of Alzheimer’s disease.
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Gardner, Hunter H. Pestilence and the Body Politic in Latin Literature. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780198796428.001.0001.

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Lucretius, Vergil, and Ovid developed important conventions of the Western plague narrative as a response to the breakdown of the Roman res publica in the mid-first century CE and the reconstitution of stabilized government under the Augustan Principate (31 BCE–14 CE). Relying on the metaphoric relationship between the human body and the body politic, these authors use largely fictive representations of epidemic disease to address the collapse of the social order and suggest remedies for its recovery. Plague as such functions frequently in Roman texts to enact a drama in which the concerns of the individual must be weighed against those of the collective. In order to understand the figurative potential of plague, this book evaluates the reality of epidemic disease in Rome, in light of twentieth-century theories of plague discourse, those of Artaud, Foucault, Sontag, and Girard, in particular. Pestilence and the Body Politic in Latin Literature identifies consistent features of the outbreaks described by Roman epic poets, charting the emergence of Golden-Age imagery, emphasis on bodily dissolution, and poignant accounts of broken familial bonds. Such features are expressed through Roman idioms that provocatively recall the discourse of civil strife that characterized the last century of the Roman Republic. The final chapters examine key moments in the resurgence of Roman plague topoi, beginning with early imperial poets (Lucan, Seneca, and Silius Italicus), and concluding with discussion of late antique Christian poetry, paintings of the late Italian Renaissance, and Anglo-American novels and films.
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Solymosi, Reka, and Kate J. Bowers. The Role of Innovative Data Collection Methods in Advancing Criminological Understanding. Edited by Gerben J. N. Bruinsma and Shane D. Johnson. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190279707.013.35.

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Environmental criminology emphasizes the importance of situational factors associated with increased risk in terms of crime opportunities. One branch of research in this field is oriented toward strengthening the scientific approach to understanding the link between exposure to risk and crime. To achieve this, we need data about how potential victims and potential offenders spend their time, and what places they visit as part of their daily activities. This chapter lays out the potential of novel data sets and then considers in detail two of these new approaches. The first approach involves utilizing advances in technology and sensing to develop bespoke surveys created with specific research studies in mind. The second makes use of existing “big data” or “open-access data” sources on people’s everyday interactions with the environment, and combines multiple data sources to make inferences about routine activities and their link to perception of crime and place.
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Book chapters on the topic "Potentiel de plaque"

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Zargaryan, Stepan. "On the Asymptotic Behavior of Solutions of a System of Integral Equations of Mixed Boundary Value Problem of Plane Elasticity in a Neighborhood of Corner Points of the Contour." In Potential Theory, 351–62. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0981-9_44.

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Gianturco, Sandra H., and William A. Bradley. "A Cellular Basis for the Potential Atherogenicity of Triglyceride-Rich Lipoproteins." In Pathobiology of the Human Atherosclerotic Plaque, 513–23. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3326-8_33.

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Nestoridis, V. "Compactifications of the plane and extensions of the disc algebra." In Complex Analysis and Potential Theory, 61–75. Providence, Rhode Island: American Mathematical Society, 2012. http://dx.doi.org/10.1090/crmp/055/05.

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Conway, John B. "Potential Theory in the Plane." In Functions of One Complex Variable II, 301–83. New York, NY: Springer New York, 1995. http://dx.doi.org/10.1007/978-1-4612-0817-4_9.

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Markey, Sean, Sarah-Patricia Breen, Kelly Vodden, and Jen Daniels. "Identity and commitment to place." In The Theory, Practice, and Potential of Regional Development, 102–22. Abingdon, Oxon ; New York, NY : Routledge, 2019. | Series: Regions and cities ; 136: Routledge, 2019. http://dx.doi.org/10.4324/9781351262163-6.

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Volberg, A. L. "On the Harmonic Measure of Self-Similar Sets on the Plane." In Harmonic Analysis and Discrete Potential Theory, 267–80. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4899-2323-3_22.

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Bird, Peter, Yan Y. Kagan, and David D. Jackson. "Plate Tectonics and Earthquake Potential of Spreading Ridges and Oceanic Transform Faults." In Plate Boundary Zones, 203–18. Washington, D. C.: American Geophysical Union, 2013. http://dx.doi.org/10.1029/gd030p0203.

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Yanushauskas, A. I. "Oblique Derivative Problem with Direction of Differentiation Going into the Tangent Plane." In The Oblique Derivative Problem of Potential Theory, 113–67. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-1674-9_3.

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Yu, Guang, Steven G. Wesnousky, and Göran Ekström. "Slip Partitioning along Major Convergent Plate Boundaries." In Shallow Subduction Zones: Seismicity, Mechanics and Seismic Potential Part 1, 183–210. Basel: Birkhäuser Basel, 1993. http://dx.doi.org/10.1007/978-3-0348-5846-5_2.

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Miyata, M., N. Kurita, and I. Nakamura. "Turbulent Plane Jet Excited Mechanically by an Oscillating Thin Plate in the Potential Core." In Turbulent Shear Flows 7, 209–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-76087-7_16.

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Conference papers on the topic "Potentiel de plaque"

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Landric, C. "Fracture de l’angle mandibulaire déplacé chez l’enfant : ou réduire ? Quand retirer le matériel ?" In 66ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2020. http://dx.doi.org/10.1051/sfco/20206603007.

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Chez l’enfant, les fractures alvéolo-dentaire sont plus fréquentes que les fractures basales car l’os est plus élastique que chez l’adulte. Cependant la présence de germes fragilise la mandibule (Cole 2009). Concernant les fractures squelettiques maxillo-faciales, 55% concernent les condyles mais seulement 8% concernent l’angle. La prise en charge entre l’adulte et l’enfant diffère du fait du risque d’accident d’évolution des germes et des troubles de croissance secondaires. Le cas présenté à été pris en charge au Centre Hospitalier de Pau. Il concerne un enfant de 8 ans ayant présenté une fracture comminutive de l’angle mandibulaire gauche avec plaie submandibulaire et sublinguale suite à un accident domestique. L’examen clinique dans ce contexte fut difficile. L’examen tomodensitométrique avait mis en évidence une fracture déplacée de l’angle mandibulaire gauche passant par le germe de 37. Il a bénéficié d’une exploration cervicale ( vasculaire, nerveuse et glandulaire) et d’une ostéosynthèse basilaire et sur la ligne oblique externe par voie d’abord mixte selon les plaies. Une plaque de 1,5mm sur la ligne oblique et sur le rebord basilaire ont été posées avec des vis mono corticale de 5 mm. Un contrôle clinique et radiologique a été réalisé a 15 jours et 2 mois sans complications post opératoire. L’ablation du matériel à été réalisé à 3 mois en reprenant les mêmes voie d’abord. Le contrôle a 1 mois et 6 mois après dépose du matériel n’a pas mis en évidence d’accident dévolution sur le germe de 37. La réduction de la fracture de l’angle déplacé chez l’adulte se fait par une plaque rigide sur la ligne oblique externe associée ou non à une ostéosynthèse basilaire. Chez l’enfant les auteurs recommandent une réduction basilaire pour ne pas atteindre les germes des dents définitives avec abord cervical, endobuccal ou mixte (Zimmermann 2006). Consolidation osseuse chez l’adulte 6 semaines, 3 semaines chez l’enfant car le potentiel ostéogénique est supérieur à celui de l’adulte. Si le condyle est reconnu comme centre du potentiel de croissance, la mandibule répond à une croissance adaptative et maintenir une ostéosynthèse rigide après le délai de consolidation pourrait entraîner des troubles de la croissance (Wheeler 2011). Chez l’adulte l’ablation du matériel d’ostéosynthèse n’est pas systématique, et est réalisé en moyenne à 6 mois a 1 an post opératoire. Chez l’enfant la moyenne est a 3 mois post opératoire (Bakathir 200). Pour l’instant les plaques résorbables ne montrent pas entière satisfaction, du fait de la réaction inflammatoire importante qu’elles induisent.
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Polieieva, Yu S., and Yu A. Vasik. "THE ROLE OF PAUSES IN THE TEMPORAL PLANE OF INSTITUTIONAL DISCOURSE." In PHILOLOGICAL SCIENCES AND TRANSLATION STUDIES: EUROPEAN POTENTIAL. Baltija Publishing, 2021. http://dx.doi.org/10.30525/978-9934-26-110-7-40.

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Huang, Xueying, Chun Yang, Jie Zheng, Richard Bach, David Muccigrosso, Pamela K. Woodard, and Dalin Tang. "Sudden Death in Coronary Artery Disease are Associated With High 3D Critical Plaque Wall Stress: A 3D Multi-Patient FSI Study Based on Ex Vivo MRI of Coronary Plaques." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14501.

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Atherosclerotic plaque rupture is the primary cause of cardiovascular clinical events such as heart attack and stroke. It is commonly believed that plaque rupture may be linked to critical mechanical conditions. Image-based computational models of vulnerable plaques have been introduced seeking critical mechanical indicators which may be used to identify potential sites of rupture [1–5]. A recent study by Tang et al. [4] using in vivo MRI-based 3D fluid-structure interaction (FSI) models for human carotid plaques with and without rupture reported that higher critical plaque wall stress (CPWS) values were associated with plaques with rupture, compared to those without rupture. However, existing computational plaque models are mostly for carotid plaques based on MRI data. Comparable similar studies for coronary plaques are lacking in the current literature. In this study, 3D computational multi-component models with FSI were constructed to identified 3D critical plaque wall stress, critical flow shear stress (CFSS) based on ex vivo MRI data of coronary plaques acquired from 10 patients. The patients were split into 2 groups: patients died in carotid artery disease (CAD, Group 1, 6 patients) and non CAD (Group 2, 4 patients). The possible link between CPWS and death in CAD was investigated by comparing the CPWS values from the two groups.
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Debattista, Maria, Andrew Fletcher, and Claire Capewell. "P-124 Preferred place of care – root cause analysis." In People, Partnerships and Potential, 16 – 18 November 2016, Liverpool. British Medical Journal Publishing Group, 2016. http://dx.doi.org/10.1136/bmjspcare-2016-001245.147.

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H. Al-Hadidy, Aboosh, and Adnan A.M. Aqrawi. "Hydrocarbon Potential of the Upper Permian Chia Zairi Formation in Northwest Iraq." In Third Arabian Plate Geology Workshop. Netherlands: EAGE Publications BV, 2011. http://dx.doi.org/10.3997/2214-4609.20144050.

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Rabie, A., R. Husain, and M. Al-Mukhaizeem and A.M. Al-Fares. "Integrated Formation Evaluation and Production Potential of Pre-Jurassic Formations in Kuwait." In Third Arabian Plate Geology Workshop. Netherlands: EAGE Publications BV, 2011. http://dx.doi.org/10.3997/2214-4609.20144086.

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Teng, Zhongzhao, Gador Canton, Chun Yuan, Marina Ferguson, Chun Yang, Xueying Huang, Jie Zheng, Pamela K. Woodard, and Dalin Tang. "Predicting Human Carotid Plaque Site of Rupture Using 3D Critical Plaque Wall Stress and Flow Shear Stress: A 3D Multi-Patient FSI Study Based on In Vivo MRI of Plaques With and Without Prior Rupture." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19080.

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Atherosclerotic plaque rupture is the primary cause of cardiovascular clinical events such as heart attack and stroke. Image-based computational models of vulnerable plaques have been introduced seeking critical mechanical indicators which may be used to identify potential sites of rupture [1–5]. Models derived from 2D ex vivo and in vivo magnetic resonance images (MRI) have shown that 2D local critical stress values rather than global maximum stress values correlated better with plaque vulnerability, as defined by histopathological and morphological analyses [5]. A recent study by Tang et al. [4] using in vivo MRI-based 3D fluid-structure interaction (FSI) models for ruptured human carotid plaques, reported that mean plaque wall stress (PWS) values from ulcer nodes were 86% higher than mean PWS values from all non-ulcer nodes (p<0.0001). This study extends the “critical stress” concept to 3D and uses 3D FSI models based on in vivo MRI data of human atherosclerotic carotid plaques with and without prior rupture to identify 3D critical plaque wall stress (CPWS), critical flow shear stress (CFSS), and to investigate their associations with plaque rupture.
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Faisal Haider, Mohammad, Victor Giurgiutiu, Bin Lin, and Lingyu Yu. "Simulation of Lamb Wave Propagation Using Excitation Potentials." In ASME 2017 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/pvp2017-66074.

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Acoustic emission (AE) can be used to measure energy associated with inelastic deformation such as slip, twinning, and microcracking, etc. in a structure. By obtaining AE information during a damage process, the failure indication can be detected. Therefore, better understanding of AE from a damage process is essential for proper damage detection. Elastic waves emission from a damage process due to energy release can be generalized by excitation potentials. There are two types of potentials exists in a plate for straight crested Lamb waves: pressure potential and shear potential. Theoretical formulation showed that due to excitation potentials the elastic waves in a plate followed the Raleigh-Lamb wave equation. The total energy released from damage can be decomposed as pressure potential and shear potential. Each potential has contribution to different wave modes. A numerical simulation was conducted to identify different wave modes due to excitation potentials. Out of plane displacement was calculated numerically on top of the plate at 500 mm distance from excitation point in each of 2mm, 6mm and 12 mm thick stainless steel plate. There were large losses in peak signal amplitude of anti-symmetric fundamental mode (A0) with increasing plate thickness from 2mm to 12 mm.
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Johnson, Louise. "P-212 Everything in place: a good death is a well-planned death." In People, Partnerships and Potential, 16 – 18 November 2016, Liverpool. British Medical Journal Publishing Group, 2016. http://dx.doi.org/10.1136/bmjspcare-2016-001245.233.

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De Schryver, Thomas, Jan Kips, Abigail Swillens, and Patrick Segers. "Effects of Plaque on Carotid Wall Deformation Studied Using a Finite Element Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206449.

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It is well established that atherosclerosis alters the biomechanics of the arterial wall. In more advanced stages of the disease, atherosclerosis leads to formation of plaques, which can cause life-threatening events such as stroke or myocardial infarction when they rupture. The carotid artery is of special interest when screening patients for cardiovascular disease because of its superficial nature, which makes it easy to study with ultrasound. In addition, as it is particularly susceptible to plaque formation, it is a potential source of stroke. A lot of research effort is dedicated to the identification of plaques prone to rupture (the vulnerable plaque), using invasive (for coronary) and non-invasive ultrasound techniques for superficial arteries such as the carotid. As these vulnerable plaques can not be distinguished from stable plaques on a pure morphological basis other indices have to be found. It is commonly believed that detailed analysis of stresses and strains in the vessel wall may provide the necessary information to make this distinction.
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Reports on the topic "Potentiel de plaque"

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Sass, Bruce M., Ryan L. Fimmen, Eric A. Foote, Victor S. Magar, and Upal Ghosh. Characterization of Contaminant Migration Potential Through In-Place Sediment Caps. Fort Belvoir, VA: Defense Technical Information Center, April 2009. http://dx.doi.org/10.21236/ada512810.

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Gidley, Philip, Seokjoon Kwon, and Upal Ghosh. Characterization of Contaminant Migration Potential Through In-Place Sediment Caps. Fort Belvoir, VA: Defense Technical Information Center, June 2011. http://dx.doi.org/10.21236/ada553531.

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Ebner, M. A. The potential pyrophoricity of BMI-SPEC and aluminum plate spent fuels retrieved from underwater storage. Office of Scientific and Technical Information (OSTI), August 1996. http://dx.doi.org/10.2172/378675.

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P. H. Titus, S. Avasaralla, A. Brooks, R. Hatcher. NSTX Disruption Simulations of Detailed Divertor and Passive Plate Models by Vector Potential Transfer from OPERA Global Analysis Results. Office of Scientific and Technical Information (OSTI), September 2010. http://dx.doi.org/10.2172/988890.

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Carruth, William D. Evaluation of In-Place Asphalt Recycling for Airfield Applications. Engineer Research and Development Center (U.S.), July 2021. http://dx.doi.org/10.21079/11681/41142.

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Over the last few decades, in-place recycling of asphalt pavements has seen increased use by the highway industry, primarily to take a dvantage of potential cost and logistical savings compared to conventional reconstruction. More recently, the U.S. Navy and Federal Aviation Administration have allowed recycling to be used on airfields with lighter traffic. This report contains a discussion of in-place recycling design considerations obtained from a literature review of its use in the highway industry. Observations developed from a review of airfield pavement projects that have utilized recycling is also included. A structural analysis was performed using the Pavement-Transportation Computer Assisted Structural Engineering (PCASE) tool to determine typical stiffness values that recycled layers must achieve to support various types of military aircraft traffic for different pavement structures. Overall, in-place recycling is recommended for consideration as a rehabilitati on technique for military airfield pavements, and further investigation is recommended before it is implemented it into design guidance.
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Nelson, Arthur, Robert Hibberd, and Kristina Currans. Transit Impacts on Jobs, People and Real Estate. Transportation Research and Education Center (TREC), 2021. http://dx.doi.org/10.15760/trec.258.

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This report is comprised of five substantive elements. The first is crafting a scientifically sound framework for identifying landscapes within the metropolitan areas we studied. The second is applying those Place Typologies and spatial analysis to economic and demographic change for the transit system in each metropolitan area. The third is analyzing how real estate markets respond to transit system proximity with special reference to the Place Typologies. Fourth, this is followed by specialized studies into how urban form and society are shaped by transit systems. The fifth is providing an overall perspective of our research as well as a framework for unlocking the potential to leverage economic benefits of transit to advance social well-being.
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Keane, Claire, Karina Doorley, and Dora Tuda. COVID-19 and the Irish welfare system. ESRI, June 2021. http://dx.doi.org/10.26504/bp202201.

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COVID-19 had, and continues to have, a strong negative effect on incomes in Ireland due to widespread job losses as the measures put in place to slow the spread of the disease resulted in severe economic restrictions. Despite the existence of unemployment supports, additional income supports were introduced to protect incomes. As public health restrictions lift and the economy recovers, we face the withdrawal of such supports. We examine these supports and the role they played in supporting incomes. By profiling those who benefitted most from the new schemes, we highlight the groups most at risk of significant income losses as they wind down. We consider what gaps in the social welfare system necessitated the introduction of such schemes in the first place, along with potential future policy changes to ensure that the social welfare system can provide adequate income protection and financial incentives to work as we emerge from the COVID-19 crisis.
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Sumpter, Cameron. Lab-in-Field Experiments for the Reintegration of Violent Extremists: The Promise of Prosocial Evaluation. RESOLVE Network, January 2021. http://dx.doi.org/10.37805/rve2021.3.

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When an inmate leaves prison following a sentence for terrorism offenses, their reintegration will depend on whether they can function as a relatively social member of their community. Obstacles such as stigmatization exist for all former convicts, but among steadfast extremists these barriers will be mutual, if they continue to perceive the ingroup-outgroup dichotomy that fed their extremism in the first place. A simple but effective means for determining the likelihood that returning prisoners will act prosocially towards the ‘other’ could be the use of so-called lab-in-field games, which provide small incentives to learn how individuals behave in a given situation, rather than just eliciting their sentiment. This chapter outlines the potential for such an approach. It draws on field research conducted in Indonesia in 2018, which involved interviews with 28 former convicted terrorists, regarding their practical experiences with reintegration and interactions in the community.
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Salazar, Lina, Ana Claudia Palacios, Michael Selvaraj, and Frank Montenegro. Using Satellite Images to Measure Crop Productivity: Long-Term Impact Assessment of a Randomized Technology Adoption Program in the Dominican Republic. Inter-American Development Bank, September 2021. http://dx.doi.org/10.18235/0003604.

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This study combines three rounds of surveys with remote sensing to measure long-term impacts of a randomized irrigation program in the Dominican Republic. Specifically, Landsat 7 and Landsat 8 satellite images are used to measure the causal effects of the program on agricultural productivity, measured through vegetation indices (NDVI and OSAVI). To this end, 377 plots were analyzed (129 treated and 248 controls) for the period from 2011 to 2019. Following a Differencein-Differences (DD) and Event study methodology, the results confirmed that program beneficiaries have higher vegetation indices, and therefore experienced a higher productivity throughout the post-treatment period. Also, there is some evidence of spillover effects to neighboring farmers. Furthermore, the Event Study model shows that productivity impacts are obtained in the third year after the adoption takes place. These findings suggest that adoption of irrigation technologies can be a long and complex process that requires time to generate productivity impacts. In a more general sense, this study reveals the great potential that exists in combining field data with remote sensing information to assess long-term impacts of agricultural programs on agricultural productivity.
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Hickling, Sophie. Tackling Slippage. Institute of Development Studies (IDS), September 2019. http://dx.doi.org/10.19088/slh.2020.004.

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This issue of Frontiers of CLTS explores current thinking and practice on the topic of tackling slippage of open defecation free (ODF) status. It looks at how slippage is defined and identified, and at different patterns of slippage that are seen after ODF is declared. Although a considerable amount has been written on how to establish strong Community-Led Total sanitation (CLTS) programmes that prevent slippage from happening, this issue looks at how to reverse slippage that has already taken place. Note however, that at a certain level, strategies used to reverse slippage and those used in advance to set a programme up for success to prevent slippage occurring overlap. From the literature, there is little documented evidence on how slippage can be reversed; evidence and guidance tend to focus on prevention. This review begins to address this gap. Implementers are encouraged to use the proposed patterns of slippage framework and slippage factors section to understand the type and extent of slippage experienced, then use the examples in the section on tackling slippage to identify potential slippage responses. In addition to a review of current literature,1 in depth interviews were carried out with key informants at global, regional and country level. Key informants were selected purposively to identify experiences and innovations in tackling slippage from across the sector.
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